Academic literature on the topic 'Phalloideae'

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Journal articles on the topic "Phalloideae"

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Alvarado, Pablo, Antonia Gasch-Illescas, Sylvie Morel, Magda Bou Dagher-Kharrat, Gabriel Moreno, José Luis Manjón, Xavier Carteret, et al. "Amanita Section Phalloideae Species in the Mediterranean Basin: Destroying Angels Reviewed." Biology 11, no. 5 (May 18, 2022): 770. http://dx.doi.org/10.3390/biology11050770.

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In Europe, amatoxin-containing mushrooms are responsible for most of the deadly poisonings caused by macrofungi. The present work presents a multidisciplinary revision of the European species of Amanita sect. Phalloideae based on morphology, phylogeny, epidemiology, and biochemistry of amatoxins and phallotoxins. Five distinct species of this section have been identified in Europe to date: A. phalloides, A. virosa, A. verna, the recently introduced North American species A. amerivirosa, and A. vidua sp. nov., which is a new name proposed for the KOH-negative Mediterranean species previously described as A. verna or A. decipiens by various authors. Epitypes or neotypes are selected for species lacking suitable reference collections, namely A. verna and A. virosa. Three additional taxa, Amanita decipiens, A. porrinensis, and A. virosa var. levipes are here considered later heterotypic synonyms of A. verna, A. phalloides, and A. amerivirosa, respectively.
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Davison, E. M., D. Giustiniano, F. Busetti, G. M. Gates, and K. Syme. "Death cap mushrooms from southern Australia: additions to Amanita (Amanitaceae, Agaricales) section Phalloideae Clade IX." Australian Systematic Botany 30, no. 4 (2017): 371. http://dx.doi.org/10.1071/sb17032.

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The following three similar Amanita spp. are described: Amanita djarilmari E.M.Davison, A. gardneri E.M.Davison from the south-west of Western Australia and A. millsii E.M.Davison & G.M.Gates (=A. sp. 10 ZLY-2014 HKAS 77322 in KUN) from Tasmania. All have a white- or pale-coloured pileus and white universal veil, but differ in the shape of the bulb, spore shape, and structure of the universal veil. All are from subgenus Lepidella section Phalloideae. Phylogenetic analysis showed that these species cannot be separated on the basis of data derived from nuclear ribosomal internal transcribed-spacer sequences. They can be separated in a multi-locus phylogeny of the 28S nuclear ribosomal large-subunit rRNA region, RNA polymerase-II region, β-tubulin region and translation elongation-factor 1-α region. Amanita djarilmari, A. gardneri, A. millsii and two other previously described species in section Phalloideae from southern Australia (A. eucalypti and A. marmorata) cluster in Clade IX. These, together with other species in this clade, segregate into two lineages, namely, Clade IX A, with a white or pale pileus, and Clade IX B, with a brown pileus. Solvent extraction, followed by liquid-chromatography tandem-mass spectrometry of A. djarilmari, A. eucalypti, A. gardneri and A. marmorata basidiomes did not detect the highly toxic amatoxins α-amanitin and β-amanitin, but did detect the phallotoxins phallacidin and phalloidin.
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CODJIA, JEAN EVANS I., SEPAS SARAWI, BORIS A. OLOU, MEIKE PIEPENBRING, ZHU L. YANG, and NOUROU S. YOROU. "Amanita stubbosa, a new non amatoxin nor phallotoxin-containing species within Amanita sect. Phalloideae (Amanitaceae, Agaricales), from West Africa." Phytotaxa 592, no. 2 (April 20, 2023): 267–78. http://dx.doi.org/10.11646/phytotaxa.592.3.5.

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Amanita stubbosa, a new species in subclade III of A. sect. Phalloideae, is described from West Africa based on morphological and molecular evidence. The species is characterized by a white pileus covered with small white broadly adherent squamules or greyish to whitish erected, pyramidal, subconical, patchy universal veil, a slightly striate pileal margin, a marginate basal bulb, and broadly ellipsoid to ellipsoid, weakly amyloid basidiospores. Phylogenetic analyses based on three gene regions (nrLSU, RPB2, and TEF1-α) revealed that A. stubbosa is a distinct species closely related to A. bulbulosa. Moreover, A. stubbosa was reported to lack the most notorious toxins (amatoxins and phallotoxins). A detailed description, illustrations, and comparisons with morphologically and phylogenetically closely related species are provided.
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Jr., Orson K. Miller, Don E. Hemmes, and George Wong. "Amanita marmorata subsp. Myrtacearum: A New Subspecies in Amanita Section Phalloideae from Hawaii." Mycologia 88, no. 1 (January 1996): 140. http://dx.doi.org/10.2307/3760795.

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Parnmen, Sittiporn, Nattakarn Nooron, Pilailuk Akkapaiboon Okada, Sujitra Sikaphan, Onanong Pringsulaka, Sutheewan Binchai, and Achariya Rangsiruji. "Characterization of Lethal Toxin-Producing Genes in Amanita brunneitoxicaria and PCR-based Detection of Deadly Poisonous Amanitas in the Section Phalloideae." Trends in Sciences 20, no. 3 (January 18, 2023): 4301. http://dx.doi.org/10.48048/tis.2023.4301.

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A recent discovery of Amanita brunneitoxicaria revealed that this mushroom and A. exitialis are the main causative agents of fatal mushroom poisoning in Thailand. Poisonous amatoxins present are bicyclic octapeptides encoded by the MSDIN family and macrocyclized by prolyl oligopeptidase B enzyme. Diversity of the MSDIN toxin-associated genes in A. brunneitoxicaria however, remains unexplored. Therefore, this study aimed to characterize the MSDIN family members in A. brunneitoxicaria in comparison with A. exitialis based on whole genome sequencing, followed by gene annotation using bioinformatic tools. Identification of conserved core peptides of the MSDIN family revealed the presence of α-amanitin and β-amanitin as endogenous toxins in A. brunneitoxicaria. In addition to the major amatoxins, amanexitide and phallacidin were also found in A. exitialis. Phylogenetic analyses of the MSDIN family members showed that an unknown peptide present in A. brunneitoxicaria is clustered with phalloidin. On the basis of PCR detection, a modified forward primer was incorporated into the PCR reactions to obtain clear and distinctive DNA fragments of approximately 300 bp from three deadly poisonous mushrooms of A. brunneitoxicaria, A. exitialis and A. fuliginea in the section Phalloideae. Hence, they could be distinguished from other edible and less toxic species in the same genus. Such rapid and accurate identification of the target region of the MSDIN family from the lethal mushrooms is relevant as means to reduce mortality. HIGHLIGHTS Amanita brunneitoxicaria and A. exitialis are responsible for most cases of fatal mushroom poisoning in Thailand During wild mushroom foraging, A. brunneitoxicaria is often mistaken for the edible species of A. vaginata A. brunneitoxicaria and other lethal aminitas contain amatoxin-producing genes encoded by the MSDIN family PCR-based detection of the target region of the MSDIN family can be used to distinguish the lethal aminitas in the section Phalloideae This rapid detection is useful for timely and appropriate medical treatment of mushroom intoxication GRAPHICAL ABSTRACT
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Weiß, Michael, Zhu-Liang Yang, and Franz Oberwinkler. "Molecular phylogenetic studies in the genus Amanita." Canadian Journal of Botany 76, no. 7 (July 1, 1998): 1170–79. http://dx.doi.org/10.1139/b98-129.

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A group of 49 Amanita species that had been thoroughly examined morphologically and anatomically was analyzed by DNA sequence comparison to estimate natural groups and phylogenetic relationships within the genus. Nuclear DNA sequences coding for a part of the ribosomal large subunit were determined and evaluated using neighbor-joining with bootstrap analysis, parsimony analysis, conditional clustering, and maximum likelihood methods. Sections Amanita, Caesarea, Vaginatae, Validae, Phalloideae, and Amidella were substantially confirmed as monophyletic groups, while the monophyly of section Lepidella remained unclear. Branching topologies between and within sections could also partially be derived. Subgenera Amanita and Lepidella were not supported. The Mappae group was included in section Validae. Grouping hypotheses obtained by DNA analyses are discussed in relation to the distribution of morphological and anatomical characters in the studied species.Key words: fungi, basidiomycetes phylogeny, Agaricales, Amanita systematics, large subunit rDNA, 28S.
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Fraiture, André, Mario Amalfi, Olivier Raspé, Ertugrul Kaya, Ilgaz Akata, and Jérôme Degreef. "Two new species of Amanita sect. Phalloideae from Africa, one of which is devoid of amatoxins and phallotoxins." MycoKeys 53 (June 6, 2019): 93–125. http://dx.doi.org/10.3897/mycokeys.53.34560.

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Two new species of Amanitasect.Phalloideae are described from tropical Africa (incl. Madagascar) based on both morphological and molecular (DNA sequence) data. Amanitabweyeyensissp. nov. was collected, associated with Eucalyptus, in Rwanda, Burundi and Tanzania. It is consumed by local people and chemical analyses showed the absence of amatoxins and phallotoxins in the basidiomata. Surprisingly, molecular analysis performed on the same specimens nevertheless demonstrated the presence of the gene sequence encoding for the phallotoxin phallacidin (PHA gene, member of the MSDIN family). The second species, Amanitaharkonenianasp. nov. was collected in Tanzania and Madagascar. It is also characterised by a complete PHA gene sequence and is suspected to be deadly poisonous. Both species clustered together in a well-supported terminal clade in multilocus phylogenetic inferences (including nuclear ribosomal partial LSU and ITS-5.8S, partial tef1-α, rpb2 and β-tubulin genes), considered either individually or concatenated. This, along with the occurrence of other species in sub-Saharan Africa and their phylogenetic relationships, are briefly discussed. Macro- and microscopic descriptions, as well as pictures and line drawings, are presented for both species. An identification key to the African and Madagascan species of Amanitasect.Phalloideae is provided. The differences between the two new species and the closest Phalloideae species are discussed.
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Méndez-Navarro, Jorge, Nayeli X. Ortiz-Olvera, Margarita Villegas-Ríos, Luis J. Méndez-Tovar, Karin L. Andersson, Rosalba Moreno-Alcantar, Víctor E. Gallardo-Cabrera, et al. "Hepatotoxicity from ingestion of wild mushrooms of the genus Amanita section Phalloideae collected in Mexico City: two case reports." Annals of Hepatology 10, no. 4 (October 2011): 568–74. http://dx.doi.org/10.1016/s1665-2681(19)31529-7.

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Fleming, Vaughan. "Battarraea phalloides." Mycologist 13, no. 1 (February 1999): 14. http://dx.doi.org/10.1016/s0269-915x(99)80067-6.

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Visser, Miranda, Willemien F. J. Hof, Astrid M. Broek, Amanda van Hoek, Joyce J. de Jong, Daan J. Touw, and Bart G. J. Dekkers. "Unexpected Amanita phalloides-Induced Hematotoxicity—Results from a Retrospective Study." Toxins 16, no. 2 (January 29, 2024): 67. http://dx.doi.org/10.3390/toxins16020067.

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Introduction: Amanita phalloides poisoning is a serious health problem with a mortality rate of 10–40%. Poisonings are characterized by severe liver and kidney toxicity. The effect of Amanita phalloides poisonings on hematological parameters has not been systematically evaluated thus far. Methods: Patients with suspected Amanita phalloides poisonings were retrospectively selected from the hospital database of the University Medical Center Groningen (UMCG). Medical data—including demographics; liver, kidney, and blood parameters; treatment; and outcomes—were collected. The severity of the poisoning was scored using the poison severity score. Results: Twenty-eight patients were identified who were admitted to the UMCG with suspected Amanita phalloides poisoning between 1994 and 2022. A time-dependent decrease was observed for hemoglobin and hematocrit concentrations, leukocytes, and platelets. Six out of twenty-eight patients developed acute liver failure (ALF). Patients with ALF showed a higher increase in liver enzymes, international normalized ratios, and PSS compared to patients without ALF. Conversely, hemoglobin and platelet numbers were decreased even further in these patients. Three out of six patients with ALF died and one patient received a liver transplant. Conclusion: Our study shows that Amanita phalloides poisonings may be associated with hematotoxicity in patients. The quantification of hematological parameters is of relevance in intoxicated patients, especially in those with ALF.
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Dissertations / Theses on the topic "Phalloideae"

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BENVENUTO, VINCENT. "Intoxication phalloidienne : aspect botanique, toxicologique, clinique, therapeutique et preventif." Saint-Etienne, 1989. http://www.theses.fr/1989STET6215.

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CHAUD, JEAN-PAUL. "L'intoxication phalloidienne : a propos de quatre observations recensees dans le departement des alpes-maritimes." Nice, 1988. http://www.theses.fr/1988NICE6539.

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Joos, Luc. "Effets de la phalloidine et de la viroisine sur Acanthamoeba castelanii après perméabilisation." Thèse, Université du Québec à Trois-Rivières, 1985. http://depot-e.uqtr.ca/6051/1/000555862.pdf.

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HEURTEBIZE, PASCALE. "Intoxication phalloidienne : a propos de cinq cas traites par ceftazidime et silymarine." Besançon, 1990. http://www.theses.fr/1990BESA3095.

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Perrier, Anne-Lise. "Syndrome phalloïdien : à propos d'une intoxication chez une femme enceinte à treize semaines d'aménorrhée." Montpellier 1, 1998. http://www.theses.fr/1998MON11025.

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Lourenço, Ana Luísa Martins. "Insuficiência hepática aguda provocada por Amanita phalloides." Master's thesis, 2014. http://hdl.handle.net/10451/23839.

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Trabalho Final do Curso de Mestrado Integrado em Medicina, Faculdade de Medicina, Universidade de Lisboa, 2014
There are certain mushroom species that are able to induce acute liver failure. Amanita phalloides is responsible for most poisonings. This paper analyses the pathogenesis, clinical features, therapeutic strategies and selection criteria for transplantation in the acute liver failure associated to ingestion of Amanita phalloides. The toxins of Amanita phalloides are capable of inhibiting RNA polymerase II resulting in a deficient protein synthesis and cell necrosis. The most affected organ is the liver, however, the kidney is also involved. After an asymptomatic phase, the clinical picture is characterised by gastrointestinal symptoms, followed by liver and kidney involvement and toxicity may progress to death if correct treatment isn´t established. The treatment consists of supportive measures, detoxification, hepatoprotective therapies and, in the most severe cases, transplantation can be indicated.
Existem determinadas espécies de cogumelos que podem causar insuficiência hepática aguda, sendo que a Amanita phalloides é a responsável pela grande maioria dos casos. Este trabalho vai analisar o mecanismo de acção das toxinas existentes na Amanita phalloides, a sintomatologia, as estratégias terapêuticas, os critérios de selecção para transplante hepático e o prognóstico. A insuficiência hepática provocada por esta espécie é secundária à capacidade das toxinas de se ligarem à ARN polimerase II e bloquearem a produção proteica, levando rapidamente à morte celular. O órgão mais afectado é o fígado, sendo que o rim também é vulnerável à acção das toxinas. Após um período assintomático, o quadro clínico é caracterizado por sintomas gastrointestinais. De seguida, verifica-se um envolvimento hepático e renal, que pode culminar na morte do doente se a terapêutica correcta não for instituída. O tratamento inclui medidas de suporte, desintoxicação, hepatoprotecção e, nalguns casos, transplante hepático.
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Garcia, Juliana Cristina Venera. "Discovery of an effective antidote for Amanita Phalloides poisoning." Doctoral thesis, 2015. https://repositorio-aberto.up.pt/handle/10216/83801.

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Rodrigues, Daniela Ferreira. "In vitro evaluation of antidotes for Amanita Phalloides intoxications." Master's thesis, 2018. https://hdl.handle.net/10216/117302.

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Rodrigues, Daniela Ferreira. "In vitro evaluation of antidotes for Amanita Phalloides intoxications." Dissertação, 2018. https://hdl.handle.net/10216/117302.

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Garcia, Juliana Cristina Venera. "Discovery of an effective antidote for Amanita Phalloides poisoning." Tese, 2015. https://repositorio-aberto.up.pt/handle/10216/83801.

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Books on the topic "Phalloideae"

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Bastien, Pierre. J'ai dû manger des amanites mortelles. Paris: Maison rustique, 1985.

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Kern, Armin. Erzeugung von Antikörpern gegen Toxine des Grünen Knollenblätterpilzes, Amanita phalloides, mittels niedermolekularer Lipopeptid-Toxin-Konjugate. [s.l.]: [s.n.], 1988.

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Wieland, Theodor. Moderne Naturstoffchemie Am Beispiel des Pilzgiftstoffes Phalloidin: Vorgetragen in der Sitzung Vom 25. April 1981. Springer London, Limited, 2013.

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Freda, Benjamin J., and Gregory L. Braden. Other toxic acute tubulointerstitial nephritis. Edited by Adrian Covic. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0085.

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Chronic kidney disease (CKD) is often the result of incomplete recovery of renal function from a variety of causes of acute tubulointerstitial injury. Exposure to ethylene glycol, chlorinated hydrocarbons, paraquat, or toxic mushrooms often causes severe acute kidney injury (AKI), leading to chronic tubulointerstitial nephritis (TIN) and CKD, including end-stage renal disease. Ethylene glycol intoxication often leads to chronic TIN and CKD from direct renal tubular toxicity and from interstitial calcium oxalate deposition. Chlorinated hydrocarbon exposure can cause dialysis-dependent AKI, but only rarely causes CKD from interstitial calcium deposition. Paraquat intoxication causes dose-dependent AKI and often Fanconi syndrome in up to 50% of patients, but only 15% of these patients survive, so CKD is rarely seen as a complication. The toxic mushrooms Cortinarius and Amanita phalloides often cause delayed AKI leading to CKD, chronic dialysis, or renal transplantation.
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Book chapters on the topic "Phalloideae"

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Vidal, C., and W. R. Külpmann. "Phalloidine." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_2396-1.

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Vidal, C., and W. R. Külpmann. "Phalloidine." In Springer Reference Medizin, 1858. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_2396.

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Wieland, Theodor. "Non-Toxic Peptides from Amanita phalloides." In Springer Series in Molecular Biology, 181–206. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71295-1_9.

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BIBEROĞLU, Serap. "Mantar Zehirlenmeleri." In Kırmızı Alan: Acilde Kritik Vakalar. İstanbul Üniversitesi–Cerrahpaşa Üniversite Yayınevi, 2024. http://dx.doi.org/10.5152/9921.

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Mantar zehirlenmeleri tüm dünyada olduğu gibi ülkemizde de sık görülen ve toksininine göre çok farklı bulgularla karşılaşılan geniş klinik sonuçlara yol açan bir zehirlenmedir. Sağlık merkezlerine başvuran tüm zehirlenmelerinin %1,5-3,4’ünü mantarla zehirlenmelerin oluşturduğu bildirilmektedir. Amanita phalloides (A. phalloides) zehirlenmeye yol açan ve ölümlerin büyük bölümüne sebep olan mantar türüdür. Acil servise mantar zehirlenmesi nedeniyle gelen olgularda en sık görülen şikayetler; bulantı, kusma, karın ağrısı, ishal, baş dönmesi, şuur kaybı ve ensefalopatidir. Etkiler tüketilmesinden sonra 3 saat içinde gelişiyorsa ‘erken mantar zehirlenmesi’dir. Erken mantar zehirlenmesi ile ortaya çıkan gastrointestinal semptomlar muskarine-allenik norlösine ve gastrointestinal sitoksinlere bağlıdır. Geç belirti gösteren mantar zehirlenmelerinde 24 saat sonra semptomların varlığı gelişir. Bu dönemde görülen gastrointestinal bulgular orellinorellaninine ikincildir. Karaciğerle ilgili bulgular amatoksingiromitrin etkisine bağlı görülür. Böbrekleri etkileyen toksin ise ise orellin, orellanin ve allenik norlösine ikincil gelişebilir. Acil Serviste tedavide ilk yaklaşım tetkiklerin alınmasıyla damar yolu, hidrasyon ve gastrik lavaj yoluyla gastrointestinal dekontaminasyondur. Eğer tüketilen mantarın zehirli olmadığı kanıtlanamıyorsa 0.5-1 gr/kg aktif kömür oral yolla verilir. Parenteral sıvı ve elektrolit desteği uygulanır. Antiemetikler kullanılır. Anti-diyareyik tedavi uygulanmaz çünkü toksine maruz kalınan zaman uzatılmak istenilmez. Kolinerjik zehirlenme, disülfiram benzeri reaksiyon, akut rabdomiyoliz, akut gastroenterit ve gecikmiş böbrek yetmezliği, gecikmiş gastroenterit ve karaciğer toksisitesi, gecikmiş gastroenterit, nöbetler ve karaciğer toksisitesi, gecikmiş böbrek yetmezliği ve rabdomiyoliz, eritromelalji gibi tablolarla karşımıza çıkmaktadır. Ülkemizde mantar zehirlenmelerine ilkbahar ve sonbahar mevsimlerinde bölgesel olarak sıklıkla karşılaşılmaktadır. Bu yüzden acil servise başvuran GİS semptomlara sahip hastalarda mutlaka toplama mantar tüketim öyküsü sorgulanmalı, hızla tetkikleri alınıp hidrasyona başlanılmalıdır. Ulusal Zehir Danışma Merkezi’ne bildirilmelidir.
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Conference papers on the topic "Phalloideae"

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Wachulski, Marcin F., Diana Kamińska-Gocał, Maciej Dądalski, Piotr Socha, and Jan J. Mulawka. "Prognosis for children with acute liver failure due to Amanita phalloides poisoning." In Photonics Applications in Astronomy, Communications, Industry, and High-Energy Physics Experiments 2011, edited by Ryszard S. Romaniuk. SPIE, 2011. http://dx.doi.org/10.1117/12.904482.

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Stöckert, Petra, Andreas Brosig, Vlad Pavel, Martina Müller-Schilling, and Stephan Schmid. "Therapeutic plasma exchange as a bridge to recovery for acute liver failure due to Amanita phalloides poisoning." In 40. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag, 2024. http://dx.doi.org/10.1055/s-0043-1777529.

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