Relevant bibliographies by topics / Pertussis

Academic literature on the topic 'Pertussis'

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Published: 4 June 2021
Last updated: 14 March 2024

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Journal articles on the topic "Pertussis"

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Schneider, Ferenc, Éva Stánitz, Judit Kalácska, Tünde Tompity, and Beáta Gábor. "Whooping cough in an urban high school in Hungary. Conclusions of a local pertussis outbreak." Orvosi Hetilap 150, no. 33 (August 1, 2009): 1557–61. http://dx.doi.org/10.1556/oh.2009.28655.

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A pertussis a védőoltás bevezetésével fokozatosan visszaszoruló fertőző betegség lett, de teljes eradikációja nem lehetséges. Ezt mutatják a nemzetközi és a hazai adatok is. Az ezredforduló után a pertussisos megbetegedések lassú növekedése figyelhető meg. Célkitűzés: Az adolescens pertussis klinikumának bemutatása egy helyi járvány kapcsán, ezzel egyidejűleg a pertussis jelenlétének demonstrálása a hazai populációban. Módszer: Az első felismert 18 éves beteg környezetében retrospektív adatgyűjtéssel felderített, tartósan köhögő betegektől vett vérmintából emelkedő pertussis-antitoxin-IgG meghatározásával igazolták a pertussisetiológiát. Eredmények: A járványügyi munka 17 további, szerológiai vizsgálattal megerősített megbetegedést derített fel a felismert beteg környezetében. Következtetések: Tartós köhögés hátterében pertussist kell keresni. A védőoltással szerzett pertussis elleni immunitás a gyermekkor végére kialszik, a kórokozó a hazai populációban is cirkulál, a pertussisemlékeztető oltás beillesztése a 11 éves korúak oltásába indokolt. Serdülő- és felnőttkorban a pertussis enyhébben és atípusosan zajlik, de elhúzódó köhögés esetén gondolni kell rá.
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Tjahjowargo, Sendy, and Hartono Gunardi. "Perbandingan Efektivitas dan Keamanan Vaksin Pertusis Aselular dan Whole-cell." Sari Pediatri 18, no. 5 (March 29, 2017): 403. http://dx.doi.org/10.14238/sp18.5.2017.403-8.

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Latar belakang. Imunisasi merupakan upaya pencegahan terbaik terhadap berbagai penyakit infeksi. Vaksin difteri, tetanus, pertusis whole-cell (DTwP) dapat menimbulkan kejadian ikutan pasca imunisasi (KIPI) yang mengkhawatirkan orangtua. Vaksin difteri, tetanus, pertusis aselular (DTaP) memiliki KIPI lebih ringan, tetapi diduga kurang efektif. Tujuan. Mengetahui efektivitas vaksin DTaP dibandingkan dengan vaksin DTwP.Metode. Penelusuran literatur elektronik PubMed dan Cochrane dengan kata kunci “DTaP/acellular pertussis”, “DTwP/whole-cell pertussis”, “children”, “pertussis”, “vaccine” and “safety/efficacy/effectiveness” dalam 10 tahun terakhir (2006 – 2016). Hasil. Terdapat dua studi meta-analisis yang membandingkan efektivitas pemberian imunisasi DTwP dan DTaP serta satu studi kasus terkontrol yang membandingkan efek perlindungan jangka panjang pemberian imunisasi DTaP dengan DTwP. Efektivitas vaksin pertusis aselular berkisar 74% (IK95%, 51–86%) – 97% (IK95%, 91–99%). Efektivitas vaksin pertusis whole-cell sebesar 94% (IK95%, 88–97%; p<0,0001). Estimasi effect size vaksin pertusis aseluer untuk melindungi terhadap penyakit pertusis sesuai kriteria WHO adalah sebesar 84% (IK95%, 81–87%); sedangkan untuk vaksin pertusis whole cell adalah 94% (IK95%, 88–97%). KIPI vaksin DTaP lebih ringan dan jarang dibandingkan vaksin DTwPKesimpulan. Vaksin DTaP dan DTwP mempunyai efektivitas yang sebanding. Vaksin DTwP mempunyai effect size yang lebih besar untuk melindungi terhadap penyakit pertusis dan perlindungan jangka panjang yang lebih baik dibandingkan vaksin DTaP. Vaksin DTaP mempunyai KIPI yang lebih ringan dan jarang dibandingkan vaksin DTwP.
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Zadorozhnaya, V. I., A. P. Podavalenko, and N. I. Operchuk. "Risk of an Intensification of Pertussis Epidemic and its Vaccinal Prevention Process in Ukraine." Epidemiology and Vaccine Prevention 14, no. 1 (February 20, 2015): 78–82. http://dx.doi.org/10.31631/2073-3046-2015-14-1-78-82.

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The pertussis epidemic process is characterized by cyclical ups 2-5 years, high level of child and town population morbidity. Longterm pertussis morbidity dynamics in Ukraine tends to increase. Low coverage of a pertusiss immunization of the children's population, increase of immunocompromised individuals in the population, deterioration of social, economic and ecological factors are adverse conditions for a current of the pertussis epidemic process. The first priority is to maintain an appropriate level of routine immunization of children with increased attention to immunocompromised persons.
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Kilgore, Paul E., Abdulbaset M. Salim, Marcus J. Zervos, and Heinz-Josef Schmitt. "Pertussis: Microbiology, Disease, Treatment, and Prevention." Clinical Microbiology Reviews 29, no. 3 (March 30, 2016): 449–86. http://dx.doi.org/10.1128/cmr.00083-15.

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SUMMARYPertussis is a severe respiratory infection caused byBordetella pertussis, and in 2008, pertussis was associated with an estimated 16 million cases and 195,000 deaths globally. Sizeable outbreaks of pertussis have been reported over the past 5 years, and disease reemergence has been the focus of international attention to develop a deeper understanding of pathogen virulence and genetic evolution ofB. pertussisstrains. During the past 20 years, the scientific community has recognized pertussis among adults as well as infants and children. Increased recognition that older children and adolescents are at risk for disease and may transmitB. pertussisto younger siblings has underscored the need to better understand the role of innate, humoral, and cell-mediated immunity, including the role of waning immunity. Although recognition of adult pertussis has increased in tandem with a better understanding ofB. pertussispathogenesis, pertussis in neonates and adults can manifest with atypical clinical presentations. Such disease patterns make pertussis recognition difficult and lead to delays in treatment. Ongoing research using newer tools for molecular analysis holds promise for improved understanding of pertussis epidemiology, bacterial pathogenesis, bioinformatics, and immunology. Together, these advances provide a foundation for the development of new-generation diagnostics, therapeutics, and vaccines.
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Conly, John M., and B. Lynn Johnston. "The Role of the Aceullular Pertussis Vaccine and the Comeback of 'Pertussis Pete'?" Canadian Journal of Infectious Diseases 12, no. 1 (2001): 15–17. http://dx.doi.org/10.1155/2001/839183.

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Pertussis or whooping cough is an acute infectious disease of the respiratory tract caused principally byBordetella pertussisand less commonly byBordetella parapertussis(1). Until two decades ago, pertussis in adults was a medical curiosity (2-4), but with the purification of specificBordetellaspecies antigens, the development of reliable enzyme immunoassays allowing accurate serological diagnosis and better understanding of the duration of immunity from vaccination, it has been clearly demonstrated thatB pertussisis a common cause of prolonged cough in adults. Indeed, its incidence has been increasing gradually over the past decade in both adults and adolescents. Given the recognition of the importance of pertussis as a cause of prolonged cough in adults and the advent of the new acellular pertussis vaccines, it is timely to review current concepts of the pathogenesis of pertussis, its epidemiology in adults and the utility of the anticipated impact of the acellular vaccine.
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Daniels, Heather L., and Camille Sabella. "Bordetella pertussis (Pertussis)." Pediatrics in Review 39, no. 5 (May 2018): 247–57. http://dx.doi.org/10.1542/pir.2017-0229.

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Pascual, F. Brian, Candace L. McCall, Aaron McMurtray, Tony Payton, Forrest Smith, and Kristine M. Bisgard. "Outbreak of Pertussis Among Healthcare Workers in a Hospital Surgical Unit." Infection Control & Hospital Epidemiology 27, no. 6 (June 2006): 546–52. http://dx.doi.org/10.1086/506232.

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Background.In September 1999, a pertussis outbreak was detected among surgical staff of a 138-bed community hospital. Patients were exposed toBordetella pertussisduring the 3-month outbreak period.Objective.To describe the outbreak among surgical staff, to evaluate implemented control measures, and to determine whether nosocomial transmission occurred.Methods.Clinical pertussis was defined as acute cough illness with a duration of 14 days or more without another apparent cause; persons with positive culture, PCR, or serologic test results were defined as having laboratory-confirmed pertussis. Surgical healthcare workers (HCWs) were interviewed regarding pertussis symptoms, and specimens were obtained for laboratory analysis. Patients exposed toB. pertussisduring an ill staff member's 3-week infectious period were interviewed by phone to determine the extent of nosocomial spread.Participants.A total of 53 HCWs assigned to the surgical unit and 146 exposed patients. HCWs with pertussis were defined as case subjects; HCWs without pertussis were defined as non-case subjects.Results.Twelve (23%) of 53 HCWs had clinical pertussis; 6 cases were laboratory confirmed. The median cough duration in the 12 case subjects was 27 days (range, 20-120 days); 10 (83%) had paroxysms. Eleven (92%) of 12 case subjects and 28 (86%) of 41 non-case subjects received antibiotic treatment or prophylaxis. Seven case subjects (58%) reported they always wore a mask when near patients. Of 146 patients potentially exposed to pertussis from the 12 case subjects, 120 (82%) were interviewed; none reported a pertussis-like illness.Conclusions.Surgical staff transmittedB. pertussisamong themselves; self-reported data suggests that these HCWs did not transmitB. pertussisto their patients, likely because of mask use, cough etiquette, and limited face-to-face contact. Control measures might have helped limit the outbreak once pertussis was recognized.
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Kandola, Kami, Amy Lea, Wanda White, and Maria Santos. "A Comparison of Pertussis Rates in the Northwest Territories: Pre- and Postacellular Pertussis Vaccine Introduction in Children and Adolescents." Canadian Journal of Infectious Diseases and Medical Microbiology 16, no. 5 (2005): 271–74. http://dx.doi.org/10.1155/2005/642315.

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INTRODUCTION: During the past decade, a trend toward increasing cases ofBordetella pertussisin older children and adults has been witnessed in Canada. The National Advisory Committee on Immunization now recommends that the adult formulation of the acellular pertussis (adult dTap) vaccine combined with diphtheria and tetanus toxoids be substituted for diphtheria and tetanus toxoids alone for the 14- to 16-year-old booster dose. In October 2000, the government of the Northwest Territories was one of the first to adopt adult dTap into their territorial immunization program free of charge.OBJECTIVE: To evaluate the effect of the acellular pertussis vaccine in children and adolescents on the epidemiology of pertussis in the Northwest Territories.METHODS: Pertussis is a reportable disease in the Northwest Territories, and data on the incidence rates of pertussis are available from 1989 to 2004. The present study reviews pertussis cases during three four-year periods: the whole-cell vaccine era (1993 to 1996); the preadult dTap era (1997 to 2000); and the postadult dTap era (2001 to 2004).RESULTS: The incidence of pertussis decreased from 18.0 cases per 10,000 population in 1993 to 0.2 cases per 10,000 population in 2004. The number of cases decreased from 186 to 129 to 19 cases in the three chronological time periods (ie, whole-cell vaccine era, preadult dTap era and postadult dTap era, respectively), with the most substantial reduction coming with the introduction of postadult dTap.CONCLUSIONS: There appears to be a decrease in the incidence of pertussis with the targeted introduction of adult dTap in the Northwest Territories.
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Zhang, Xuqing, Sara E. Hester, Mary J. Kennett, Alexia T. Karanikas, Liron Bendor, David E. Place, and Eric T. Harvill. "Interleukin-1 Receptor Signaling Is Required To Overcome the Effects of Pertussis Toxin and for Efficient Infection- or Vaccination-Induced Immunity againstBordetella pertussis." Infection and Immunity 79, no. 1 (October 25, 2010): 527–41. http://dx.doi.org/10.1128/iai.00590-10.

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ABSTRACTInterleukin-1 receptor-deficient (IL-1R−/−) mice are healthy despite being colonized by commensal microbes but are defective in defenses against specific pathogens, suggesting that IL-1R-mediated effects contribute to immune responses against specific pathogenic mechanisms. To better define the role of IL-1R in immunity to respiratory infections, we challenged IL-1R−/−mice withBordetella pertussisandBordetella parapertussis, the causative agents of whooping cough. Following inoculation withB. pertussis, but notB. parapertussis, IL-1R−/−mice showed elevated bacterial numbers and more extensive inflammatory pathology than wild-type mice. AcellularB. pertussisvaccines were not efficiently protective againstB. pertussisin IL-1R−/−mice.B. pertussis-stimulated dendritic cells from IL-1R−/−mice produced higher levels of tumor necrosis factor alpha (TNF-α) and IL-6 than wild-type cells. Moreover, elevated levels of gamma interferon (IFN-γ) and TNF-α but lower levels of IL-10 were detected duringB. pertussisinfection in IL-1R−/−mice. SinceB. parapertussisdid not cause severe disease in IL-1R−/−mice, we hypothesized that the extreme requirement for IL-1R involves pertussis toxin (Ptx), which is expressed only byB. pertussis. An isogenic Ptx-deficientB. pertussisstrain had only a modest phenotype in wild-type mice but was completely defective in causing lethal disease in IL-1R−/−mice, indicating that the particular virulence ofB. pertussisin these mice requires Ptx. Ptx contributes to IL-1β induction byB. pertussis, which is involved in IL-10 induction through IL-1R signaling. IL-10 treatment reducedB. pertussisnumbers in IL-1R−/−mice, suggesting that the lower IL-10 responses partially account for the uncontrolled inflammation and bacterial growth in these mice.
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Aase, Audun, Tove Karin Herstad, Samuel Merino, Merete Bolstad, Synne Sandbu, Hilde Bakke, and Ingeborg S. Aaberge. "Immunization of Teenagers with a Fifth Dose of Reduced DTaP-IPV Induces High Levels of Pertussis Antibodies with a Significant Increase in Opsonophagocytic Activity." Clinical and Vaccine Immunology 18, no. 8 (June 15, 2011): 1269–74. http://dx.doi.org/10.1128/cvi.05067-11.

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ABSTRACTWaning vaccine-induced immunity againstBordetella pertussisis observed among adolescents and adults. A high incidence of pertussis has been reported in this population, which serves as a reservoir forB. pertussis. A fifth dose of reduced antigen of diphtheria-tetanus-acellular-pertussis and inactivated polio vaccine was given as a booster dose to healthy teenagers. The antibody activity againstB. pertussisantigens was measured prior to and 4 to 8 weeks after the booster by different assays: enzyme-linked immunosorbent assays (ELISAs) of IgG and IgA against pertussis toxin (PT) and filamentous hemagglutinin (FHA), IgG against pertactin (PRN), opsonophagocytic activity (OPA), and IgG binding to liveB. pertussis. There was a significant increase in the IgG activity against PT, FHA, and PRN following the booster immunization (P< 0.001). The prebooster sera showed a geometric mean OPA titer of 65.1 and IgG binding to live bacteria at a geometric mean concentration of 164.9 arbitrary units (AU)/ml. Following the fifth dose, the OPA increased to a titer of 360.4, and the IgG concentration against live bacteria increased to 833.4 AU/ml (P< 0.001 for both). The correlation analyses between the different assays suggest that antibodies against FHA and PRN contribute the most to the OPA and IgG binding.
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Dissertations / Theses on the topic "Pertussis"

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Rambow-Larsen, Amy Alison. "Assembly and secretion of pertussis toxin by bordetella pertussis." Cincinnati, Ohio : University of Cincinnati, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=ucin1066417734.

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RAMBOW-LARSEN, AMY ALISON. "ASSEMBLY AND SECRETION OF PERTUSSIS TOXIN BY BORDETELLA PERTUSSIS." University of Cincinnati / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1066417734.

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Hegerle, Nicolas. "Evolution of Bordetella pertussis and Bordetella parapertussis under acellular Pertussis vaccine pressure : What future for whooping cough and Pertussis vaccination ?" Paris 7, 2014. http://www.theses.fr/2014PA077038.

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La coqueluche est une maladie respiratoire dangereuse pour les nouveaux nés non vaccinés. Celle-ci est causée par Bordetella pertussis et Bordetella parapertussis, deux bactéries à gram négatif dont le seul hôte connu est l'homme. L'introduction de la vaccination contre B. Pertussis, le pathogène majoritairement responsable de cette maladie, à grandement impacté l'épidémiologie de la coqueluche ainsi que les populations de B. Pertussis circulant dans la population humaine. Les premier vaccins introduit dans les 1950 et utilisés pour la primo vaccination et le rappel enfant, dit à germes entier, ont permit de contrôler les isolats circulants semblables aux souches vaccinales. L'introduction des vaccines acellulaire, ne ciblant que quelques antigènes bactériens, pour le rappel adolescent puis pour la primo-vaccination et le rappel adulte a changé la donne. En plus d'un changement de l'immunité induite par le vaccin on a assisté à une augmentation de l'immunité vaccinale au sein de la population. Quelques années après l'introduction des vaccins acellulaires nous avons mis en évidence l'augmentation de la prévalence d'isolat n'exprimant plus un des antigènes vaccinaux alors que la population restait inchangée par rapport à l'ère post-germe entier au niveau génétique. La caractérisation phénotypique des ces isolats nous a permit de montrer qu'ils n'étaient pas plus virulent dans différents modèles in vitro ou in vivo mais qu'ils pouvaient présenter un avantage sélectif dans population immunisée par des vaccins acellulaires ciblant cet antigène en question. Bien que les vaccins restent efficaces, la surveillance doit continuer
Whooping cough is an acute respiratory disease life threatening for unvaccinated young children. It is caused by Bordetella pertussis and Bordetella parapertussis, two gram-negative bacteria restricted to humans. The introduction of vaccination against B. Pertussis in the 1950s greatly changed the epidemiology of pertussis as well as the bacterial population itself. Whole cell vaccines were first introduced for children immunization and first booster and enabled to control isolates similar to strains included in the vaccines. Acellular pertussis vaccines, only targeting few bacterial antigens, were later introduced for adolescent booster vaccination before being generalized to the whole population, including adults and new-borns. In addition to a change in the type of vaccine-induced immunity, B. Pertussis also had to face increased herd immunity. Few years alter acellular pertussis vaccine introduction we demonstrated a temporal increase in the prevalence of isolates lacking the production of one vaccine antigen, pertactin, while the population remained quite monomorphic at the genetic level as compare to the post-whole cell vaccine era (allelic stability of known virulence factors). We characterized these isolates at the phenotypic level and demonstrated that they remain as virulent in different in vitro and in vivo models of host pathogen interaction while they might present a selective advantage in an acellular immunized background targeting pertactin. Although vaccines remain effective, surveillance must continue to follow the evolution of these isolate prevalence and the possible impact of pertactin loss on vaccine effectiveness
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Phillips, Linda Jane. "Immunization against Bordetella pertussis." Thesis, Kansas State University, 1985. http://hdl.handle.net/2097/9871.

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Smith, Maureen. "Studies on the modification of insulin secretion by pertussis vaccine and pertussis toxin." Thesis, University of Strathclyde, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.303295.

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Smith, Colin J. "Genetic studies with Bordetella pertussis." Thesis, University of Glasgow, 1986. http://theses.gla.ac.uk/1505/.

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Lewandowski, Anna Zofia. "Antigenic variation in Bordetella pertussis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ60480.pdf.

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Sidey, Fiona M. "Metabolic effects of Bordetella pertussis." Thesis, University of Strathclyde, 1987. http://oleg.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=20352.

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The present work confirmed that B. pertussis infection or pertussis toxin produce hypoglycaemia in mice. The hypoglycaemia was associated with hyperinsulinaemia, and both were abolished by destruction of the pancreatic β cells with alloxan. Impaired glucose counterregulatory mechanisms may also contribute to pertussis-induced hypoglycaemia, as the hypoglycaemic action of insulin was prolonged in pertussis infected mice. On the other hand, impaired responsiveness to lower doses of insulin was found. Pertussis-induced hyperinsulinaemia had two components. First, the increase in serum insulin in response to food intake was both greater and more prolonged in pertussis-infected mice. Second, infected or pertussis toxin-treated animals, unlike controls, showed a marked increase in serum insulin in response to certain stresses, such as ether, histamine, anoxia and 2-deoxyglucose. However, other stresses (LPS, cold and hypoxia) did not cause hyperinsulinaemia in pertussis infected mice. Stress-induced hyperinsulinaemia was also seen in normal mice receiving the a2- adrenoceptor blocking drug idazoxan. Stress-induced hyperinsulinaemia in a2 adrenoceptor blocked mice, but not in pertussis-treated mice, was prevented by β adrenoceptor blockade using propranolol. Adrenal demedullation or ganglionic blockade (using hexamethonium) in normal mice also allowed stress induced hyperinsulinaemia. Thus, adrenal medullary catecholamines may normally serve to prevent stress induced hyperinsulinaemia, which becomes unmasked when they are absent or when their action is prevented. Stress-induced hyperinsulinaemia in pertussis treated mice was unlikely to involve autonomic, cholinergic oropioid mechanisms as it was not blocked by hexamethonium, atropine or naloxone. Human infants with pertussis showed no hypoglycaemia compared with non-pertussis controls, although their plasma insulin concentrations were slightly but significantly raised. It remains possible that hyperinsulinaemia with resultant profound hypoglycaemia might occur in susceptible patients following exposure to pertussis-toxin (either during the disease or following vaccination).
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Ladant, Daniel. "L'adenyl cyclase de bordetella pertussis." Paris 7, 1989. http://www.theses.fr/1989PA077200.

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Bordetella pertussis, l'agent de la coqueluche secrete une adenyl cyclase qui constitue l'un des principaux facteurs de virulence de cet organisme. Cet enzyme bacterien presente l'originalite d'etre active par la calmoduline, une proteine regulatrice eucaryote. Nous avons purifie l'adenyl cyclase a l'homogeneite; elle possede l'activite specifique la plus elevee connue pour cette classe d'enzymes. Elle est constituee d'une chaine polypeptidique de 43-50 kilodaltons qui fixe une molecule de calmoduline avec une grande affinite. Par proteolyse limitee du complexe adenyl cyclase/calmoduline, nous avons identifie, dans la molecule d'adenyl cyclase, deux domaines d'interaction avec la calmoduline. Le clonage du gene de l'adenyl cyclase de b. Pertussis nous a permis d'etudier et de caracteriser la proteine exprimee chez e. Coli. La determination de la sequence nucleotidique du gene a revele l'existence d'une phase ouverte de 1706 acides amines: l'adenyl cyclase se situe dans la partie n-terminale; la partie c-terminale presente une homologie de sequence avec l'hemolysine d'e. Coli. Ces donnees suggerent que l'adenyl cyclase pourrait etre synthetisee sous forme d'un precurseur commun, adenyl cyclase-hemolysine. Un tel precurseur, d'un poids moleculaire apparent de 200 kilodaltons a ete identifie dans les souches virulentes de b. Pertussis; nous avons montre que, dans certaines conditions de culture, cette forme d'adenyl cyclase de 200 kilodaltons peut etre secretee par b. Pertussis sans proteolyse. Enfin, nous avons mis en evidence une parente antigenique entre les adenyl cyclases calmoduline-dependantes de b. Pertussis et de cerveau de rat. L'hypothese d'une origine evolutive commune pour ces deux enzymes est ainsi serieusement etayee
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Goard, Jody Ruth. "An Ecological Perspective on Pertussis." ScholarWorks, 2016. https://scholarworks.waldenu.edu/dissertations/2537.

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In 2012, 48,277 cases of pertussis were diagnosed in the United States. Pertussis, otherwise known as whooping cough, is a highly contagious, often debilitating, sometimes deadly, vaccine-preventable disease with an increasing incidence and death rate in the U.S, which may be due to vaccine exemptions. The purpose of this project was to determine if a relationship exists between immunization policies and immunization exemption rates, immunization exemption rates and pertussis rates, and immunization policies and pertussis rates in each state. Bronfenbrenner's bio-ecological framework was used to guide the project. Publically available data from the Centers for Disease Control and Prevention (CDC), schools of public health, state health departments, and public health officials were retrieved for this cross-sectional, ecological comparison study. Spearman's r product-moment correlation coefficient was used to investigate the relationship between the variables. States with lenient vaccine laws had higher exemption rates (r = .359, p < .01), and states with higher exemption rates had higher pertussis rates (r = .470, p < .01). Finally, states with lenient vaccine laws had higher pertussis rates (r = .111, p = 0.439). This project should be added to the literature used to inform and educate the public as well as influence policy makers. As a result of this study, arguments for eliminating non-medical vaccine exemptions should be strengthened. As policies are changed, social change should follow in the form of decreased immunization exemption rates and decreased pertussis rates.
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Books on the topic "Pertussis"

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Ronald, Sekura, Moss Joel, Vaughan Martha 1926-, National Institute of Child Health and Human Development (U.S.), National Heart, Lung, and Blood Institute., and Pertussis Toxin Conference (1984 : National Institutes of Health), eds. Pertussis toxin. Orlando: Academic Press, 1985.

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Fedele, Giorgio, and Clara Maria Ausiello, eds. Pertussis Infection and Vaccines. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-33249-5.

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International Symposium on Pertussis (6th 1990 Bethesda, Md.). Abstrasts and program [of] Sixth International Symposium on Pertussis [held at] Jack Masur Auditorium, Warren Grant Magnuson Clinical Center, National Institutes of Health, Bethesda, Maryland, September 26-28, 1990. Bethesda, Md: Dept. of Health and Human Services, United States Public Health Service, Food and Drug Administration, 1990.

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C, Wardlaw A., and Parton Roger, eds. Pathogenesis and immunity in pertussis. Chichester: Wiley, 1988.

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Agarwal, P. Kaia. Immunogold investigations of pertussis toxin. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Center for Biologics Evaluation and Research (U.S.), ed. Proceedings of the Sixth International Symposium on Pertussis, Jack Masur Auditorium, Warren Grant Magnuson Clinical Center, National Institutes of Health, Bethesda, Maryland, September 26-28, 1990. Bethesda, Md: Dept. of Health and Human Services, United States Public Health Service, Food and Drug Administration, 1990.

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on pertussis. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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Cawthorn, Els. The effect of pertussis toxin on insulin secretion from obese (fa/fa) Zucker rats. Ottawa: National Library of Canada, 1991.

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Cawthorn, Els. The effect of pertussis toxin on insulin secretion from obese (fa/fa) Zucker rats. Charlottetown: University of Prince Edward Island, 1991.

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T, Perkins F., International Association of Biological Standardization., and World Health Organization, eds. Proceedings of the Fourth International Symposium on Pertussis: A joint meeting of the International Association of Biological Standardization and the World Health Organization held at the Executive Board room of the World Health Organization, Geneva, Switzerland 25.-27. Sept. 1984. Basel: S. Karger, 1985.

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Book chapters on the topic "Pertussis"

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Tan, Tina Q. "Pertussis and Pertussis Syndrome." In Introduction to Clinical Infectious Diseases, 67–73. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-91080-2_6.

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Domachowske, Joseph, and Manika Suryadevara. "Pertussis and Pertussis Syndrome." In Clinical Infectious Diseases Study Guide, 43–46. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50873-9_8.

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Al-Shaalan, Mohammad. "Pertussis." In Textbook of Clinical Pediatrics, 1017–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_87.

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Mortimer, Edward A. "Pertussis." In Bacterial Infections of Humans, 529–43. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5327-4_27.

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Domachowske, Joseph. "Pertussis." In Vaccines, 257–66. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-58414-6_21.

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Guo, Yinglin, Lili Liu, and Bailu Liu. "Pertussis." In Radiology of Infectious Diseases: Volume 2, 187–94. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9876-1_18.

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Gilchrist, M. J. R., and C. C. Linnemann. "Pertussis." In Laboratory Diagnosis of Infectious Diseases, 403–10. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3898-0_42.

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Mortimer, Edward A. "Pertussis." In Bacterial Infections of Humans, 499–511. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4757-1211-7_23.

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Neumann, G., H. H. Feucht, W. Becker, and M. Späth. "Pertussis." In Gynäkologische Infektionen, 67–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-05268-2_18.

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Tiwari, Tejpratap S. P. "Pertussis." In Emerging Infections 8, 257–75. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815592.ch13.

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Conference papers on the topic "Pertussis"

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Silva, Flávio, Luiz Pinto, Alexandre Saisse, Luciano Gomes, and Salvatore De Simone. "Bordetella pertussis: mapeamento e caracterização dos epitopos da toxina pertussis e pertactina." In I Seminário Anual Científico e Tecnológico em Imunobiológicos. Instituto de Tecnologia em Imunobiológicos, 2013. http://dx.doi.org/10.35259/isi.sact.2013_28799.

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Nicolai, Ambra, Raffaella Nenna, Antonella Frassanito, Alessandra Pierangeli, Federica Mileto, Carolina Scagnolari, Enrica Mancino, et al. "Respiratory viruses andbordetella pertussisco-infections: A frequent occurrence in children hospitalized withbordetella pertussis." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa1275.

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Böhme, M., H. Riemenschneider, K. Voigt, E. Balogh, L. Sanftenberg, and A. Bergmann. "Pertussis-Impfstatus bei Medizinstudierenden: Eine internationale Multicenterstudie." In Prävention in Lebenswelten – 54. Jahrestagung der DGSMP – Die DGSMP Jahrestagung in Dresden findet statt unter Beteiligung des MDK Sachsen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1667720.

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Yakubu, Aisha Aliyu, Farah Aini Abdullah, Ahmad Izani Md Ismail, and Yazariah Mohd Yatim. "Dynamical analysis of pertussis with maternally derived immunity." In PROCEEDINGS OF INTERNATIONAL CONFERENCE ON ADVANCES IN MATERIALS RESEARCH (ICAMR - 2019). AIP Publishing, 2020. http://dx.doi.org/10.1063/5.0018472.

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Raba, Ali, and Ibraheem Krebit. "P393 Pertussis Infection; recent concerns and new recommendations." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.739.

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Shimizu, Kenichiro, Masayuki Ohtsuka, Ken Kikuchi, and Keiichi Hiramatsu. "Nation-wide Surveillance Of Pertussis In Japanese Adults." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5483.

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Matsuda, T. "Early Detection of Bordetella Pertussis and Bordetella Parapertussis Infection with Pertussis Antibody Ig-M, Ig-A, and IgM/IgA Ratio." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6161.

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Brass, L. F., D. R. Manning, and M. J. Woolkalis. "G PROTEIN REGULATORS OF PHOSPHOLIPASE C AND ADENYLATE CYCLASE IN PLATELETS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644630.

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The hydrolysis of polyphosphoinositides (PI) by phospholipase C during platelet activation produces two key intracellular messengers, inositol triphosphate and diacylglycerol. This process is thought to be regulated by a guanine nucleotide binding protein referred to as Gp. Although the evidence that Gp exists is compelling, to date it has not been isolated. Uncertainty about its identity has been compounded by variations between tissues in the susceptibility of Gp to pertussis toxin and by reconstitution studies which show that pertussis toxin-inhibited PI hydrolysis can be restored by purified Gi, the pertussis toxin-sensitive G protein which inhibits adenylate cyclase. Therefore, it remains unclear whether Gp represents a new G protein or a second role for Gj. When platelets permeabilized with saponin were incubated with pertussis toxin and 32P-NAD, a single 42 kDa protein was 32P-ADP-ribosylated which co-migrated with the purified a subunit of Gi. Preincubating the platelets with an agonist inhibited labeling of this protein by dissociating the G protein into subunits. The extent of inhibition correlated with the number of toxin-sensitive functions caused by the agonist. Labeling was abolished by thrombin, which inhibited cAMP formation and caused toxin-inhibitable PI hydrolysis. Labeling was partially inhibited by vasopressin and platelet activating factor, which caused toxin-inhibitable PI hydrolysis, but had no effect on cAMP formation and by epinephrine, which inhibited cAMP formation, but did not cause PI hydrolysis. Labeling was unaffected by the TxA2 analog U46619, which neither caused toxin-sensitive PI hydrolysis nor inhibited cAMP formation. These observations suggest that the 42 kDa band may contain a subunits from both Gp and Gi and, in fact, 2D electrophoresis resolved the 42 kDa protein band into two proteins with distinct pi. However, those agonists linked functionally only to Gp or only to Gi decreased the labeling of both proteins. Therefore, our data suggest (1) that Gj and Gp are the same protein and (2) that whether a aiven platelet agonist affects adenylate cyclase or phospholipase C or both depends upon factors extrinsic to the G protein.
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Hendrick, Louise, and Mary Ward. "P365 Pertussis vaccination: Should we be doing something different?" In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.711.

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Schiavoni, Ilaria, Pasqualina Leone, and Giorgio Fedele. "Investigating the mechanisms of Bordetella pertussis-induced airway inflammation." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4632.

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Reports on the topic "Pertussis"

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Rust, William L. Elucidation of Pertussis Toxin-Sensitive Migration Signaling in Human Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, May 2001. http://dx.doi.org/10.21236/ada395881.

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Rust, William L. Elucidation of Pertussis Toxin-Sensitive Migration Signaling in Human Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, May 2002. http://dx.doi.org/10.21236/ada406136.

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Lewin, Simon, Sebastián García Martí, Agustín Ciapponi, Shaun Treweek, and Andy Oxman. What are the effects of interventions to improve childhood vaccination coverage? SUPPORT, 2016. http://dx.doi.org/10.30846/16081605.

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Routine vaccination during childhood is considered to be the single most effective way of controlling many infectious diseases, including measles, polio, diphtheria, pertussis and tetanus, and reducing child mortality and morbidity. However, not all children receive their recommended vaccinations. Different approaches that aim to increase childhood vaccination coverage include health education, monetary incentives for clients, provider oriented interventions, system interventions such as integration, home visits and reminders for parents.
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McDonough, E. A., C. P. Barrozo, K. L. Russell, and D. Metzgar. A Multiplex PCR for Detection of Mycoplasma pneumoniae, Chlamydophila pneumoniae, Legionella pneumophila, and Bordetella pertussis in Clinical Specimens. Fort Belvoir, VA: Defense Technical Information Center, January 2005. http://dx.doi.org/10.21236/ada432554.

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Corbacho, Ana, Steve Brito, and Rene Osorio Rivas. Does Birth Underregistration Reduce Childhood Immunization?: Evidence from the Dominican Republic. Inter-American Development Bank, December 2013. http://dx.doi.org/10.18235/0011512.

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Birth registration is not only a fundamental human right, but also a requirement for obtaining additional documents, proving legal identity, and accessing a number of government benefits. Yet, little is known about the effects of birth under-registration on access to health care. Using data from the Dominican Republic, this paper is the first to shed light on the causal impact of the lack of birth registration on childhood immunization, one of the key components of public services in many developing countries. Controlling for potential endogeneity and standard socioeconomic determinants of immunization, this paper finds that children between 0 and 59 months of age that do not have birth certificates are behind by nearly one vaccine (out of a total of nine) compared to those that have birth certificates. The results are robust to several robustness tests and threats to the exclusion restriction of the instrumental variables. Birth under-registration specifically reduces the probability of vaccination against polio, diphtheria, pertussis, and tetanus--once leading causes of child morbidity and infant mortality. In addition, untimely vaccination costs governments billions per year in treatment and rehabilitation.
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Gidengil, Courtney, Matthew Bidwell Goetz, Margaret Maglione, Sydne J. Newberry, Peggy Chen, Kelsey O’Hollaren, Nabeel Qureshi, et al. Safety of Vaccines Used for Routine Immunization in the United States: An Update. Agency for Healthcare Research and Quality (AHRQ), May 2021. http://dx.doi.org/10.23970/ahrqepccer244.

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Objective. To conduct a systematic review of the literature on the safety of vaccines recommended for routine immunization in the United States, updating the 2014 Agency for Healthcare Research and Quality (AHRQ) report on the topic. Data sources. We searched MEDLINE®, Embase®, CINAHL®, Cochrane CENTRAL, Web of Science, and Scopus through November 9, 2020, building on the prior 2014 report; reviewed existing reviews, trial registries, and supplemental material submitted to AHRQ; and consulted with experts. Review methods. This report addressed three Key Questions (KQs) on the safety of vaccines currently in use in the United States and included in the Centers for Disease Control and Prevention’s (CDC) recommended immunization schedules for adults (KQ1), children and adolescents (KQ2), and pregnant women (KQ3). The systematic review was supported by a Technical Expert Panel that identified key adverse events of particular concern. Two reviewers independently screened publications; data were extracted by an experienced subject matter expert. Studies of vaccines that used a comparator and reported the presence or absence of adverse events were eligible. We documented observed rates and assessed the relative risks for key adverse events. We assessed the strength of evidence (SoE) across the existing findings from the prior 2014 report and the new evidence from this update. The systematic review is registered in PROSPERO (CRD42020180089). Results. A large body of evidence is available to evaluate adverse events following vaccination. Of 56,608 reviewed citations, 189 studies met inclusion criteria for this update, adding to data in the prior 2014 report, for a total of 338 included studies reported in 518 publications. Regarding vaccines recommended for adults (KQ1), we found either no new evidence of increased risk for key adverse events with varied SoE or insufficient evidence in this update, including for newer vaccines such as recombinant influenza vaccine, adjuvanted inactivated influenza vaccine, and recombinant adjuvanted zoster vaccine. The prior 2014 report noted a signal for anaphylaxis for hepatitis B vaccines in adults with yeast allergy and for tetanus, diphtheria, and acellular pertussis vaccines. Regarding vaccines recommended for children and adolescents (KQ2), we found either no new evidence of increased risk for key adverse events with varied SoE or insufficient evidence, including for newer vaccines such as 9-valent human papillomavirus vaccine and meningococcal B vaccine. The prior 2014 report noted signals for rare adverse events—such as anaphylaxis, idiopathic thrombocytopenic purpura, and febrile seizures—with some childhood vaccines. Regarding vaccines recommended for pregnant women (KQ3), we found no evidence of increased risk for key adverse events with varied SoE among either pregnant women or their infants following administration of tetanus, diphtheria, and acellular pertussis vaccines during pregnancy. Conclusion. Across this large body of research, we found no new evidence of increased risk since the prior 2014 report for key adverse events following administration of vaccines that are routinely recommended. Signals from the prior report remain unchanged for rare adverse events, which include anaphylaxis in adults and children, and febrile seizures and idiopathic thrombocytopenic purpura in children. There is no evidence of increased risk of adverse events for vaccines currently recommended in pregnant women. There remains insufficient evidence to draw conclusions about some rare potential adverse events.
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Woods, Rachel, Alison Zhong, and Madelyn Vincent. Factors Associated with Influenza & Tdap Vaccine Uptake in Pregnant Patients at the UT Family Medicine Clinic in Memphis. University of Tennessee Health Science Center, 2021. http://dx.doi.org/10.21007/com.lsp.2020.0003.

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INTRODUCTION: Given the increased risk for infections among pregnant patients and newborns, vaccination against influenza (>50,000,000 annual US cases affecting all ages) and pertussis (>15,000 annual US cases disproportionately affecting newborns) are recommended among pregnant patients in order to protect them and their babies via passive immunity to cover a newborn’s window of vaccine ineligibility. Though flu and Tdap vaccination rates among pregnant patients have been trending upwards nationally, there is still room for improvement to achieve optimal rates. OBJECTIVES: The primary objectives were to study factors that affect the vaccination rates at the University of Tennessee Family Medicine Clinic at Memphis (UTFMC-M), compare those rates with national pregnancy flu/Tdap vaccination rates, and to generate recommendations based off observed factors associated with vaccine uptake to improve flu/Tdap vaccination rates in UTFMC-M pregnant patients. METHODS: This was a retrospective chart review of UTFMC-M patients who were pregnant from September 1, 2019-April 24, 2020 (included 2019-2020 flu season) (n=465). Variables studied included demographic data (race, age, insurance), immunization history (vaccine status, history of physician encouragement), and prenatal history (parity, number of prenatal visits, trimester at first visit, high risk clinic (HRC) admittance status). Vaccination status was based on ACIP recommendations (Flu shot eligible = any gestational age; Tdap eligible = ≥27 weeks). Positive HRC admittance was noted for patients with ≥2 visits to the UTFMC-M HRC, a clinic that specializes in high risk pregnant patient care. RESULTS: The patient sample was predominantly black (84.3%) and insured by Medicaid programs (88%). Among eligible UTFMC-M pregnant patients, 50.1% were flu-vaccinated (n=465); 73.8% were Tdap-vaccinated (n=317); and 52.1% were Flu+Tdap-vaccinated (n=317). No significant associations were found between vaccine uptake and HRC status, parity, and age. However, statistically significant relationships were found between vaccine uptake and physician encouragement (positive relationship with flu shot: X2(1, N = 465) =131, p < 0.001, Tdap: X2 (6, N = 465) =476, p < 0.001), number of prenatal visits (flu shot group median 8 visits, Tdap group median 9 visits vs. unvaccinated group median 4 visits; p < 0.001), and early trimester age at first prenatal visit (X2(6, N = 465) =47.635 , p CONCLUSION: 2019-2020 UTFMC-M vaccination rates were on par with 2018-2019 US flu vaccine rates and higher than 2018-2019 US Tdap and Flu+Tdap rates. There were statistically significant relationships between vaccine uptake at UTFMC-M and physician encouragement, number of prenatal visits, and early trimester age at first prenatal visit but no significant relationships with UTFMC-M HRC admittance, parity, or age. Recommendations following from our observations to address further vaccine rate improvement include: continue vaccine encouragement, continue booking multiple visits (8 for flu, 9 for Tdap), prioritize Tdap vaccine higher for late trimester intake patients, and focus on flu vaccine encouragement and education.
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