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Books on the topic 'Peripheral'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 August 2024

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1

Maitland, G. D. Peripheral manipulation. 3rd ed. Oxford: Butterworth-Heinemann, 1999.

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2

Liu, Ningfei, ed. Peripheral Lymphedema. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3484-0.

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3

Bakker, Saskia, Doris Hausen, and Ted Selker, eds. Peripheral Interaction. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29523-7.

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4

Heldman, Bruce R. Peripheral visions. Rocky Mount, NC: New Moon Pub., 1995.

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5

1927-, Dyck Peter James, ed. Peripheral neuropathy. 3rd ed. Philadelphia: Saunders, 1993.

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6

Corporation, Intel. Peripheral components. Santa Clara, Calif: Intel Corporation, 1992.

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7

National Housing and Town Planning Council., ed. Peripheral estates. London: National Housing and Town Planning Council, 1987.

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8

Gamalinda, Eric. Peripheral vision. Quezon City: New Day Publishers, 1992.

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9

Corporation, Intel. Peripheral components. Mt. Prospect, IL: Intel Corporation, 1993.

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10

Fowler, Karen Joy. Peripheral vision. Eugene, OR: Pulphouse, 1990.

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11

Bateson, Mary C. Peripheral Visions. New York: HarperCollins, 2008.

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12

1927-, Dyck Peter James, ed. Peripheral neuropathy. 3rd ed. Philadelphia: W. B. Saunders, 1993.

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13

1927-, Dyck Peter James, and Thomas P. K. 1926-2008, eds. Peripheral neuropathy. 4th ed. Philadelphia: Saunders, 2005.

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14

Corporation, Intel. Peripheral components. [Santa Clara]: Intel Corporation, 1996.

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15

-P, Hartung H., ed. Peripheral neuropathies. London: Baillière Tindall, 1996.

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16

1927-, Dyck Peter James, ed. Peripheral neuropathy. 3rd ed. Philadelphia: W. B. Saunders, 1993.

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17

1927-, Dyck Peter James, and Thomas P. K. 1926-, eds. Peripheral neuropathy. 4th ed. Philadelphia: Elsevier Saunders, 2005.

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18

Media, inc Concept. Peripheral lines. Irvine, CA: Distributed by Concept Media, 2008.

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19

Goldstar. Micro peripheral IC data book: Micro peripheral. Seoul: Goldstar, 1992.

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20

Slavin, Konstantin V. Peripheral nerve stimulation. Basel: Karger, 2011.

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21

Admir, Hadzic, Vloka Jerry D, and New York School of Regional Anesthesia., eds. Peripheral nerve blocks. New York: McGraw-Hill Health Professions Division, 2004.

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22

Berthiaume, Joseph. Peripheral. Independently Published, 2019.

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23

Ryland, John. Peripheral. World Castle Publishing, LLC, 2022.

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24

Peripheral. World Castle Publishing, LLC, 2022.

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25

William, Gibson. Peripheral. Penguin Books, Limited, 2015.

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26

Wong, Wes. Peripheral. 1First Comics, Inc., 2019.

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27

Ward, Zeth. Peripheral. CreateSpace Independent Publishing Platform, 2011.

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28

Gibson, William. Peripheral. Penguin Books, Limited, 2014.

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29

Gibson, William. Peripheral. Penguin Books, Limited, 2022.

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30

Ryland, John. Peripheral. World Castle Publishing, LLC, 2022.

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31

Peripheral. Independently Published, 2021.

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32

Corporation, Intel. Peripheral Components, 1993 (Peripheral Components). Intel Corporation (CA), 1993.

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33

van der Burg, Jorien M. M., N. Ahmad Aziz, and Maria Björkqvist. Peripheral Pathology. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199929146.003.0014.

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Clinicians and researchers have previously focused on the neurologic and psychiatric aspects of Huntington’s disease (HD). However, it is becoming evident that many neurodegenerative disorders are also complicated by pathology in tissues outside the brain. Although many clinical features of HD can be ascribed to neuronal loss and dysfunction, there is accumulating evidence indicating a role for the pathology of non-neuronal tissues in the disease process. Mutant huntingtin is expressed throughout the body and may induce pathology in parallel in both the brain and other organs. Insights into peripheral pathology in HD have the potential of improving knowledge of key pathogenic mechanisms. This chapter describes peripheral manifestations of HD, including weight loss, muscle wasting, and cardiac dysfunction, and discusses how these might constitute targets for drug treatment as well as offering disease modeling systems and potential sources of biomarkers.

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34

Timperley, Jonathan, and Sandeep Hothi. Peripheral oedema. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0014.

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Peripheral oedema is a palpable swelling caused by increased interstitial fluid in soft tissues, and can be due to local or systemic disease. Fluid distribution between capillaries and the interstitium is governed by Starling forces. The lymphatic system returns excess fluid and protein from the extracellular, interstitial space to the bloodstream. Thus, interstitial oedema may arise from factors that increase capillary pressure or permeability, factors that reduce plasma colloid osmotic pressure, factors that impede lymphatic drainage, or a combination of these causes. This topic addresses the diagnosis of peripheral oedema.

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35

Scarpa, Raffaele, Francesco Caso, Luisa Costa, Rosario Peluso, Nicola Matteo Dario Di Minno, and Antonio Del Puente. Peripheral arthritis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0010.

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Clinical presentation of peripheral arthritis in patients with psoriatic arthritis (PsA), has been described by Moll and Wright who classified it into four subsets: symmetrical polyarthritis, asymmetrical oligoarthritis, distal interphalangeal (DIP) arthritis and arthritis mutilans. In the symmetrical polyarthritis subset, the distribution of articular involvement is similar to rheumatoid arthritis and this has for many years justified the inappropriate use of the terminology ‘rheumatoid-like form’, at present completely abandoned. Oligoarthritis is characterized by asymmetrical involvement of few joints (less than four), which include scattered DIP or proximal interphalangeal (PIP) joints and/or metatarsophalangeal joints. DIP arthritis may occur with symmetrical or asymmetrical features, and it is often in strict association with onycopathy. The arthritis mutilans pattern is characterized by osteolysis of phalanx and metacarpals and it is very rare, occurring in less than 1% of patients with established form of arthritis. In 15-20% of the cases the arthritis may precede the onset of the psoriatic skin rash. Consequently, psoriatic arthritis ‘sine psoriasis’ should not be considered a rare clinical finding. In this subset articular involvement is clinically expressed, while cutaneous is apparently absent. Laboratory tests and imaging are relevant for differential diagnosis which in some presentations may represent a diagnostic challenge. The outcome of peripheral patterns of PsA patients is related not only to the spectrum of peripheral phenotypes, but also to early diagnosis, and metabolic aspects, which may affect excess in morbidity and mortality.

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36

Birn, Anne-Emanuelle, and Raúl Necochea López, eds. Peripheral Nerve. Duke University Press, 2020. http://dx.doi.org/10.1515/9781478012221.

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37

Birn, Anne-Emanuelle, and Raúl Necochea López, eds. Peripheral Nerve. Duke University Press, 2020. http://dx.doi.org/10.1215/9781478012221.

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38

Armada-Da-Silva, Paulo, ed. Peripheral Neuropathy. InTech, 2014. http://dx.doi.org/10.5772/57082.

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39

Hengeveld, Elly, and Kevin Banks. Peripheral Manipulation. Elsevier - Health Sciences Division, 2005.

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40

Boesen, Elisabeth, Fabienne Lentz, Michel Margue, Denis Scuto, and Renée Wagener, eds. Peripheral Memories. transcript Verlag, 2012. http://dx.doi.org/10.1515/transcript.9783839421161.

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41

Peripheral Neuropathy. Elsevier, 2005. http://dx.doi.org/10.1016/b978-0-7216-9491-7.x5001-7.

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42

Peripheral Neuropathy. Johns Hopkins University Press, 2016. http://dx.doi.org/10.1353/book.49293.

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43

Boesen, Elisabeth, Fabienne Lentz, Michel Margue, Denis Scuto, and Renée Wagener, eds. Peripheral Memories. transcript-Verlag, 2012. http://dx.doi.org/10.14361/transcript.9783839421161.

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44

Cornblath, David R., and Richard A. C. Hughes. Peripheral neuropathy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0013.

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Disorders of peripheral nerves are one of the most common neurological problems today and include the increasing number of people with diabetes worldwide and those with inherited neuropathy, toxic neuropathy, carpal tunnel syndrome, inflammatory neuropathy, radiculopathies, and, increasingly, traumatic nerve injuries. Neuropathic pain is a growing problem without solution. In this chapter, ten landmark papers in peripheral nerve disorders have been selected, covering Bell’s palsy, Charcot-Marie-Tooth disease, carpal tunnel syndrome, paraneoplastic neuropathy, neurophysiology, familial amyloid polyneuropathy, chronic inflammatory demyelinating polyradiculoneuropathy, toxic neuropathy, diabetic neuropathy, and Guillain–Barré syndrome. These important papers set the stage for many subsequent advances in the field but may be forgotten now, so they are brought to the reader’s attention.

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45

Hunyor, Stephen. Peripheral Circulation. Elsevier, 1985.

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46

Hartung, H. P. Peripheral Neuropathies. Elsevier, 1996.

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47

Peripheral Components. McGraw-Hill, 1996.

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48

Key, Bart. Peripheral Sur. BookBaby, 2019.

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49

Patterson, Zabet. Peripheral Vision. The MIT Press, 2015. http://dx.doi.org/10.7551/mitpress/9950.001.0001.

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50

Dolan, Martin. Peripheral Vision. Recent Work Press, 2018.

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