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Journal articles on the topic 'Peripheral vasculature'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Civelli, Valerie. "Drug Coated Balloon Angioplasty in Peripheral Vasculature: Review of Literature." Clinical Cardiology and Cardiovascular Interventions 2, no. 4 (December 16, 2019): 01–03. http://dx.doi.org/10.31579/2641-0419/034.

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2

WORCESTER, SHARON. "Estrogen Drop Affects Peripheral Vasculature." Ob.Gyn. News 40, no. 8 (April 2005): 28. http://dx.doi.org/10.1016/s0029-7437(05)70207-x.

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3

Mulvany, Michael J. "PERIPHERAL VASCULATURE IN ESSENTIAL HYPERTENSION." Clinical and Experimental Pharmacology and Physiology 23, s1 (November 1996): s6—s10. http://dx.doi.org/10.1111/j.1440-1681.1996.tb03034.x.

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4

Thom, S., A. Hughes, G. Martin, H. Nielsen, P. Inkpen, M. Schachter, and P. Sever. "Peptides in human peripheral vasculature." Regulatory Peptides 22, no. 4 (September 1988): 434. http://dx.doi.org/10.1016/0167-0115(88)90211-x.

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5

Douek, Philippe C. "MultiHance in MRA of peripheral vasculature." European Radiology Supplements 14, S7 (August 2004): O55—O60. http://dx.doi.org/10.1007/s10406-004-0065-6.

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6

Douek, Philippe C. "MultiHance in MRA of peripheral vasculature." European Radiology Supplements 15, S5 (December 2005): e17-e23. http://dx.doi.org/10.1007/s10406-005-0162-1.

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7

Reilly, Dermot F., Elizabeth J. Westgate, and Garret A. FitzGerald. "Peripheral Circadian Clocks in the Vasculature." Arteriosclerosis, Thrombosis, and Vascular Biology 27, no. 8 (August 2007): 1694–705. http://dx.doi.org/10.1161/atvbaha.107.144923.

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8

Hughes, A., G. Martin, S. Thom, and P. Sever. "Dopaminergic Mechanisms in Human Peripheral Vasculature." Journal of Hypertension 3, no. 6 (December 1985): 664–65. http://dx.doi.org/10.1097/00004872-198512000-00024.

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9

Schmiedl, Udo P., Chun Yuan, Hanh V. Nghiem, Thomas C. Winter, and Patrick C. Freeny. "MR angiography of the peripheral vasculature." Seminars in Ultrasound, CT and MRI 17, no. 4 (August 1996): 404–11. http://dx.doi.org/10.1016/s0887-2171(96)90026-8.

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10

Woods, Bartholomew O’Beirne. "Clinical Evaluation of the Peripheral Vasculature." Cardiology Clinics 9, no. 3 (August 1991): 413–27. http://dx.doi.org/10.1016/s0733-8651(18)30280-7.

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11

Schnall, M. D., G. A. Holland, R. A. Baum, C. Cope, M. L. Schiebler, and J. P. Carpenter. "MR angiography of the peripheral vasculature." RadioGraphics 13, no. 4 (July 1993): 920–30. http://dx.doi.org/10.1148/radiographics.13.4.8356278.

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12

Fadel, Paul J. "Baroreflex control of the peripheral vasculature." Medicine & Science in Sports & Exercise 39, Supplement (May 2007): 49. http://dx.doi.org/10.1249/01.mss.0000272470.79816.ab.

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13

Penman, A., J. F. Talbot, E. L. Chuang, A. C. Bird, and G. R. Serjeant. "Peripheral retinal vasculature in normal Jamaican children." British Journal of Ophthalmology 78, no. 8 (August 1, 1994): 615–17. http://dx.doi.org/10.1136/bjo.78.8.615.

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14

WORCESTER, SHARON. "Decline in Estrogen Levels Affects Peripheral Vasculature." Family Practice News 35, no. 9 (May 2005): 5. http://dx.doi.org/10.1016/s0300-7073(05)70619-8.

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15

Lehrman, E. D., A. N. Plotnik, T. Hope, and D. Saloner. "Ferumoxytol-enhanced MRI in the peripheral vasculature." Clinical Radiology 74, no. 1 (January 2019): 37–50. http://dx.doi.org/10.1016/j.crad.2018.02.021.

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16

Criqui, Michael H., and Joachim H. Ix. "Highs and Lows in the Peripheral Vasculature." Journal of the American College of Cardiology 59, no. 4 (January 2012): 408–9. http://dx.doi.org/10.1016/j.jacc.2011.10.861.

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17

Auerbach, Eric G., and Edward T. Martin. "Magnetic resonance imaging of the peripheral vasculature." American Heart Journal 148, no. 5 (November 2004): 755–63. http://dx.doi.org/10.1016/j.ahj.2004.04.045.

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18

Hata, Toshiyuki, Kenji Kanenishi, Uiko Hanaoka, Kenta Yamamoto, Nobuhiro Mori, and Takahito Miyake. "SlowflowHD for Detection of Small Fetal Peripheral Vasculature." Donald School Journal of Ultrasound in Obstetrics and Gynecology 13, no. 4 (2019): 155–58. http://dx.doi.org/10.5005/jp-journals-10009-1603.

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19

van PROOSDU, MARC P., LEO J. SCHULTZE KOOL, and J??RG W. OESTMANN. "Superimposed Peripheral Pulmonary Vasculature in Anthropomorphic Chest Phantoms." Investigative Radiology 29, no. 4 (April 1994): 466–68. http://dx.doi.org/10.1097/00004424-199404000-00014.

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20

Mulvany, M. J., and C. Aalkjaer. "Calcium Metabolism and Structure in the Peripheral Vasculature." Journal of Cardiovascular Pharmacology 12, Supplement (1988): 134. http://dx.doi.org/10.1097/00005344-198800125-00024.

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21

Mulvany, M. J., and C. Aalkjaer. "Calcium Metabolism and Structure in the Peripheral Vasculature." Journal of Cardiovascular Pharmacology 12 (1988): 134. http://dx.doi.org/10.1097/00005344-198806125-00024.

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22

Calver, Alison, Joe Collier, and Patrick Vallance. "Dilator actions of arginine in human peripheral vasculature." Clinical Science 81, no. 5 (November 1, 1991): 695–700. http://dx.doi.org/10.1042/cs0810695.

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1. l-Arginine is the physiological precursor for the formation of endothelium-derived nitric oxide. The synthesis of nitric oxide is stereospecific: d-arginine is not a substrate for nitric oxide synthase. It is possible that the provision of excess l-arginine substrate might increase the vascular synthesis of nitric oxide. We have examined this possibility by studying the effects of local infusion of l-and d-arginine in the forearm resistance bed and the superficial dorsal hand veins of healthy subjects. 2. Drugs were either infused locally into a vein on the back of the hand and then the vein diameter was measured using a linear displacement technique, or into the brachial artery and then the forearm blood flow was measured by venous occlusion plethysmography. 3. In the superficial hand veins, l- and d-arginine free base and l- and d-arginine hydrochloride (all four preparations at a dose of 5 μmol/min) all caused a significant increase in venous diameter. The responses of the l-and d-enantiomers did not differ significantly from one another. 4. In the forearm resistance bed, l- and d-arginine free base and l-arginine hydrochloride were without effect at doses of 10 and 40 μmol/min. However, at doses of 160 μmol/min all three preparations of arginine caused a significant increase in forearm blood flow compared with control values. The responses to the three preparations of arginine did not differ significantly from one another. 5. These results show that arginine in high dose is a vasodilator in both human resistance vessels and superficial veins in vivo. The response to arginine was not stereospecific: both the l- and d-enantiomers had the same effect. The dilator effect of high-dose arginine showed neither arterio-nor veno-selectivity. 6. This suggests that the hypotensive effect of systemic infusions of l-arginine in man is mediated by peripheral vasodilatation. It is not possible to ascribe the actions of arginine supplementation in this study to activation of the l-arginine/nitric oxide pathway through the provision of excess substrate.

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23

Brengelmann, George L. "Stressed and unstressed volumes of the peripheral vasculature." Canadian Journal of Anesthesia/Journal canadien d'anesthésie 65, no. 9 (June 7, 2018): 1072–73. http://dx.doi.org/10.1007/s12630-018-1155-6.

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24

Murarka, Shishir, and Richard R. Heuser. "Chronic total occlusions in peripheral vasculature: techniques and devices." Expert Review of Cardiovascular Therapy 7, no. 10 (October 2009): 1283–95. http://dx.doi.org/10.1586/erc.09.107.

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25

Dedania, Vaidehi S., Cem Ozgonul, and Cagri G. Besirli. "Peripheral Persistent Fetal Vasculature: A Report of Three Cases." Ophthalmic Surgery, Lasers and Imaging Retina 49, no. 9 (September 1, 2018): e83-e88. http://dx.doi.org/10.3928/23258160-20180907-12.

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26

FADEL, PAUL J. "Arterial Baroreflex Control of the Peripheral Vasculature in Humans." Medicine & Science in Sports & Exercise 40, no. 12 (December 2008): 2055–62. http://dx.doi.org/10.1249/mss.0b013e318180bc80.

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27

Richards, Oliver C., Summer M. Raines, and Alan D. Attie. "The Role of Blood Vessels, Endothelial Cells, and Vascular Pericytes in Insulin Secretion and Peripheral Insulin Action." Endocrine Reviews 31, no. 3 (June 1, 2010): 343–63. http://dx.doi.org/10.1210/er.2009-0035.

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The pathogenesis of type 2 diabetes is intimately intertwined with the vasculature. Insulin must efficiently enter the bloodstream from pancreatic β-cells, circulate throughout the body, and efficiently exit the bloodstream to reach target tissues and mediate its effects. Defects in the vasculature of pancreatic islets can lead to diabetic phenotypes. Similarly, insulin resistance is accompanied by defects in the vasculature of skeletal muscle, which ultimately reduce the ability of insulin and nutrients to reach myocytes. An underappreciated participant in these processes is the vascular pericyte. Pericytes, the smooth muscle-like cells lining the outsides of blood vessels throughout the body, have not been directly implicated in insulin secretion or peripheral insulin delivery. Here, we review the role of the vasculature in insulin secretion, islet function, and peripheral insulin delivery, and highlight a potential role for the vascular pericyte in these processes.

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28

Wells, Celia, Ziya Zhang, Christy Chan, Amy Brito, and Roopa Kohli-Seth. "Impact of a Peripheral Vascular Access Service on Device Use." American Journal of Critical Care 30, no. 4 (July 1, 2021): 295–301. http://dx.doi.org/10.4037/ajcc2021425.

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Background More than 1 billion peripheral vascular access devices are inserted annually worldwide with potential complications including infection, thrombosis, and vasculature damage. Vasculature damage can necessitate the use of central catheters, which carry additional risks such as central catheter–associated bloodstream infections. To address these concerns, one institution used expert nurses and a consult request system with algorithms embedded in the electronic medical record. Objectives To develop a uniform process for catheter insertion by means of a peripheral vascular access service dedicated to selecting, placing, and maintaining all inpatient peripheral catheters outside of the intensive care units. Methods Descriptive analysis and χ2 analysis were done to describe the impact of the peripheral vascular access service. Results In 2018, 6246 consults were reviewed. Of these, 26% did not require vascular access. Similarly, in 2019, 7861 consults were reviewed, and 35.3% did not require vascular access. Use of central catheters decreased from 21% in 2017 to 17% in 2018 and 2019. Conclusions The peripheral vascular access service allowed patients to receive appropriate peripheral vascular access devices and avoid unnecessary peripheral catheter placements. This may have preserved patients’ peripheral vasculature and thus prevented premature central catheter placement and contributed to an overall decrease in central catheter days. With the peripheral vascular access service, peripheral vascular access devices were selected, placed, and maintained by experts with a standardized process that promoted a culture of quality and patient safety.

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29

Jin, Zhaoyang, Ling Xia, Minming Zhang, and Yiping P. Du. "Background-Suppressed MR Venography of the Brain Using Magnitude Data: A High-Pass Filtering Approach." Computational and Mathematical Methods in Medicine 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/812785.

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Conventional susceptibility-weighted imaging (SWI) uses both phase and magnitude data for the enhancement of venous vasculature and, thus, is subject to signal loss in regions with severe field inhomogeneity and in the peripheral regions of the brain in the minimum-intensity projection. The purpose of this study is to enhance the visibility of the venous vasculature and reduce the artifacts in the venography by suppressing the background signal in postprocessing. A high-pass filter with an inverted Hamming window or an inverted Fermi window was applied to the Fourier domain of the magnitude images to enhance the visibility of the venous vasculature in the brain after data acquisition. The high-pass filtering approach has the advantages of enhancing the visibility of small veins, diminishing the off-resonance artifact, reducing signal loss in the peripheral regions of the brain in projection, and nearly completely suppressing the background signal. The proposed postprocessing technique is effective for the visualization of small venous vasculature using the magnitude data alone.

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30

Sawyer, Iain, Sarah-Jane Smillie, Jennifer V. Bodkin, Elizabeth Fernandes, Kevin T. O'Byrne, and Susan D. Brain. "The Vasoactive Potential of Kisspeptin-10 in the Peripheral Vasculature." PLoS ONE 6, no. 2 (February 9, 2011): e14671. http://dx.doi.org/10.1371/journal.pone.0014671.

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31

Spiliopoulos, S., P. Kitrou, K. Katsanos, and D. Karnabatidis. "FD-OCT and IVUS intravascular imaging modalities in peripheral vasculature." Expert Review of Medical Devices 14, no. 2 (January 13, 2017): 127–34. http://dx.doi.org/10.1080/17434440.2017.1280391.

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32

Fitzgerald, R. S., G. A. Dehghani, J. S. Sham, M. Shirahata, and W. A. Mitzner. "Peripheral chemoreceptor modulation of the pulmonary vasculature in the cat." Journal of Applied Physiology 73, no. 1 (July 1, 1992): 20–29. http://dx.doi.org/10.1152/jappl.1992.73.1.20.

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The present study was undertaken to determine whether stimulation of the carotid and aortic bodies (cb and ab) could affect the pulmonary vasculature. Our hypothesis was that each promoted vasodilation and thus could modulate the pulmonary vasoconstrictor response to hypoxia. The experimental design of the first set of experiments took advantage of the facts that 1) the ab, but not the cb, increases its neural output in response to CO, whereas both respond to a decreased arterial PO2 (hypoxic hypoxia, HH) and 2) the aortic nerves in cats are easily transected. Hence, both cb and ab sent neural activity to the brain stem when the intact cat was exposed to 10% O2 in N2. Only the ab sent information during CO hypoxia (COH intact). Only the cb did so during HH in the cat in which the aortic nerves had been transected, removing the aortic body (HH abr); neither ab nor cb did so during COH abr. Fifteen anesthetized paralyzed artificially ventilated cats were fit with catheters in the femoral artery and vein, right and left atria, left ventricle, and pulmonary artery and with an aortic flow probe. In the HH intact and HH abr conditions, there was a significant rise in cardiac output, whereas pulmonary arterial pressure (Ppa) rose initially but then leveled off while cardiac output continued to rise. During the 15-min exposure to HH, pulmonary vascular resistance [PVR = (Ppa - Pla)/cardiac output, where Pla is left atrial pressure] rose initially and then decreased significantly at 2–3 min. In response to COH, PVR showed only a significant decrease. In the second set of experiments, seven cats were instrumented as above and had loops placed in the common carotid arteries for selectively perfusing the cbs. In response to a brief infusion of venous blood mixed with 0.3–0.5 micrograms NaCN, which selectively stimulated only the cb, aortic flow remained relatively constant while heart rate and Ppa - alveolar pressure difference decreased significantly; so also did PVR. These data are consistent with the hypothesis that stimulation of the ab and cb singly or together can provoke a significant pulmonary vasodilation in the anesthetized paralyzed artificially ventilated cat.

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33

Byard, Roger W., Boleslaw Lach, and David N. Preston. "Peripheral nerve and vasculature involvement in myophosphorylase deficiency (mcardle’s disease)." Pathology 23, no. 1 (1991): 62–65. http://dx.doi.org/10.3109/00313029109061442.

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34

Stekiel, Thomas A., Zeljko J. Bosnjak, and William J. Stekiel. "Effects of General Anesthetics on Regulation of the Peripheral Vasculature." Seminars in Cardiothoracic and Vascular Anesthesia 7, no. 3 (September 2003): 311–31. http://dx.doi.org/10.1177/108925320300700307.

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35

Kalliokoski, Riikka J., Kari K. Kalliokoski, Maila Penttinen, Ilkka Kantola, Aila Leino, Jorma S. Viikari, Olli Simell, Pirjo Nuutila, and Olli T. Raitakari. "Structural and functional changes in peripheral vasculature of Fabry patients." Journal of Inherited Metabolic Disease 29, no. 5 (August 12, 2006): 660–66. http://dx.doi.org/10.1007/s10545-006-0340-x.

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36

Johnson, Nicholas B., Katherine A. Rodby, Amir Vafa, Daniel Katz, and Samantha Minc. "28 (CR). Bullet Embolism To the Peripheral Vasculature, Two Cases." Annals of Vascular Surgery 29, no. 4 (May 2015): 635. http://dx.doi.org/10.1016/j.avsg.2015.04.030.

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37

McNulty, J. A., L. M. Fox, P. L. Shaw, V. E. Alones, B. S. Klausen, R. S. Swenson, and A. J. Castro. "Pineal Gland Transplants into the Cerebral Hemisphere of Newborn Rats: A Study of the Blood Brain Barrier and Innervation." Journal of Neural Transplantation and Plasticity 2, no. 2 (1991): 113–24. http://dx.doi.org/10.1155/np.1991.113.

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Pineal glands from neonatal (0-1 day) Long-Evans black-hooded rats were transplanted into the cerebral hemispheres of litter mates for periods of 1 to 5.5 months. Grafts exhibited differentiated pinealocytes that were intensely immunoreactive for serotonin. Transplant vasculature was permeable to endogenous IgG, comprised fenestrated endothelia with wide pericapillary spaces typical ofin situglands, and had a volume density intermediate to that of surrounding cortex andin situpineals. Along the periphery, transplant capillaries tended to have continuous endothelia similar to those of host cortex. This peripheral zone was impermeable to endogenous IgG and appeared to increase in size in older grafts. The presence of noradrenergic-like fibers within the perivascular compartment suggested that transplants were innervated by peripheral sympathetic neurons from the superior cervical ganglia. In animals which had been superior cervical ganglionectomized, noradrenergic-like fibers were absent or degenerating. Neural regulation of transplant metabolic activity was suggested by the increased frequency of pinealocyte synaptic ribbons in denervated grafts. These findings are consistent with the hypothesis that factors from both graft and host influence vasculature physiology and differentiation in neural transplants. Furthermore, grafts appeared to receive appropriate neural input from the peripheral sympathetic system.

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38

Bond, R. F., C. G. Scott, L. H. Krech, and C. H. Bond. "Systemic and local effects of endotoxin on canine gracilis muscle vascular conductance." American Journal of Physiology-Heart and Circulatory Physiology 258, no. 5 (May 1, 1990): H1498—H1506. http://dx.doi.org/10.1152/ajpheart.1990.258.5.h1498.

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To define the site and mechanism of action that endotoxin has on the peripheral vasculature, an in situ constant-flow double-canine gracilis muscle (GM) preparation was utilized. During systemic endotoxemia, one GM was innervated and the other was denervated during a 30-min intravenous infusion of 2 mg/kg endotoxin. Significantly increased vascular conductance (URP) in the denervated GM (106 +/- 26%) occurred compared with the innervated GM (50 +/- 7%), which suggests that decompensation is not totally dependent on neural depression. During local endotoxemia, with both GMs either intact or denervated, one GM was infused intra-arterially for 30 min with a dose of endotoxin calculated to provide a blood concentration similar to that achieved during systemic endotoxemia, whereas the other GM was infused with the vehicle. The URPs did not change significantly in either the saline or endotoxin GMs. Therefore, endotoxin does not act directly on peripheral vasculature or totally through depression of the autonomic nervous system. It apparently interacts with a systemically dependent mechanism to release a vasodepressor substance that is transported to the peripheral vasculature causing relaxation of vascular tone.

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39

Ng, Ella S. M., and Paul Kubes. "The physiology of S-nitrosothiols: carrier molecules for nitric oxide." Canadian Journal of Physiology and Pharmacology 81, no. 8 (August 1, 2003): 759–64. http://dx.doi.org/10.1139/y03-078.

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Recent work has demonstrated that inhalation of nitric oxide (NO) can impact the peripheral vasculature, suggesting that an NO-stabilizing moiety may exist in vivo. One possibility is the formation of S-nitrosothiols, which extend the half-life of NO manyfold. In this review, we provide evidence that S-nitrosothiols exist in the vasculature, particularly during NO inhalation. The potential biochemical pathways that have been proposed for the formation of these products are also summarized. Finally, we highlight the limited evidence for the role that these potent vasodilating molecules may play as physiologically and therapeutically important regulators of the vascular system.Key words: inhaled NO, S-nitroso-albumin, peripheral circulation.

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40

Marteslo, Jeffrey P., Mina S. Makary, Hooman Khabiri, Vince Flanders, and Joshua D. Dowell. "Intravascular Ultrasound for the Peripheral Vasculature—Current Applications and New Horizons." Ultrasound in Medicine & Biology 46, no. 2 (February 2020): 216–24. http://dx.doi.org/10.1016/j.ultrasmedbio.2019.10.010.

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41

Singer, Michael, Min Sagong, Jano van Hemert, Laura Kuehlewein, Darren Bell, and SriniVas R. Sadda. "Ultra-widefield Imaging of the Peripheral Retinal Vasculature in Normal Subjects." Ophthalmology 123, no. 5 (May 2016): 1053–59. http://dx.doi.org/10.1016/j.ophtha.2016.01.022.

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42

DuPRE, CONNIE T., and RUTH-MARIE E. FINCHER. "Holt-Oram Syndrome Associated With Hypoplastic Peripheral Vasculature and Midsystolic Click." Southern Medical Journal 86, no. 4 (April 1993): 453–56. http://dx.doi.org/10.1097/00007611-199304000-00017.

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43

Malishevskaya, T. N., T. N. Kiseleva, Yu E. Filippova, A. S. Vlasova, I. V. Nemtsova, V. V. Vasilchenko, and M. S. Zaytsev. "Structural and functional features of peripheral vasculature in patients with glaucoma." Vestnik oftal'mologii 136, no. 5 (2020): 67. http://dx.doi.org/10.17116/oftalma202013605167.

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44

Yucel, E. Kent. "Magnetic resonance angiography and the peripheral vasculature: how useful is it?" Nature Clinical Practice Cardiovascular Medicine 2, no. 3 (March 2005): 136–37. http://dx.doi.org/10.1038/ncpcardio0131.

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45

Hoffmann, K. L., A. K. W. Wood, P. H. McCarthy, K. A. Griffiths, D. L. Evans, and R. W. Gill. "Sonographic Observations of the Peripheral Vasculature of the Equine Thoracic Limb." Anatomia, Histologia, Embryologia: Journal of Veterinary Medicine Series C 28, no. 5-6 (December 1999): 281–89. http://dx.doi.org/10.1046/j.1439-0264.1999.00206.x.

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46

Schainfeld, Robert M. "Potential emerging therapeutic strategies to prevent restenosis in the peripheral vasculature." Catheterization and Cardiovascular Interventions 56, no. 3 (June 17, 2002): 421–31. http://dx.doi.org/10.1002/ccd.10211.

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47

Reardon, Lindsay, Andrew O. Maree, and Michael de Moor. "Moyamoya Disease with Peripheral Pulmonary Artery Stenoses and Coronary Artery Fistulae." Case Reports in Medicine 2009 (2009): 1–2. http://dx.doi.org/10.1155/2009/840904.

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Moyamoya is a progressive disorder of the cerebral vasculature. Our report describes a rare case of Moyamoya disease with distal peripheral pulmonary artery stenoses and coronary fistulae in a 12-year-old Caucasian female patient.

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48

Michel, R. P., J. B. Gordon, and K. Chu. "Development of the pulmonary vasculature in newborn lambs: structure-function relationships." Journal of Applied Physiology 70, no. 3 (March 1, 1991): 1255–64. http://dx.doi.org/10.1152/jappl.1991.70.3.1255.

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Our objectives were 1) to describe the quantitative light microscopy and ultrastructure of newborn lamb lungs and 2) to correlate hemodynamic changes during normoxia and hypoxia with the morphology. By light microscopy, we measured the percent muscle thickness (%MT) and peripheral muscularization of pulmonary arteries and veins from 25 lambs aged less than 24 h, 2-4 days, 2 wk, and 1 mo. At the same ages, lungs were isolated and perfused in situ and, after cyclooxygenase blockade with indomethacin, total, arterial (delta Pa), middle (delta Pm), and venous pressure gradients at inspired O2 fractions of 0.28 (mild hyperoxia) and 0.04 (hypoxia) were determined with inflow-outflow occlusion. During mild hyperoxia, delta Pa and delta Pm fell significantly between 2-4 days and 2 wk, whereas during hypoxia, only delta Pm fell. The %MT of all arteries (less than 50 to greater than 1,000 microns diam) decreased, and peripheral muscularization of less than 100-microns-diam arteries fell between less than 4 days and greater than 2 wk. Our data suggest that 1) the %MT of arteries determines normoxic pulmonary vascular resistance, because only arterial and middle segment resistance fell, 2) peripheral muscularization is a major determinant of hypoxic pulmonary vasoconstriction, because we observed a fall with age in peripheral muscularization of less than 100-micron-diam arteries and in delta Pm with hypoxia, and 3) the arterial limit of the middle segment defined by inflow-outflow occlusion lies in 100- to 1,000-microns-diam arteries.

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49

Durdana, Shazia, and Ahmad Rizwan. "Peripheral symmetrical gangrene due to severe Plasmodium falciparum malaria: a case report." Tropical Doctor 50, no. 3 (May 21, 2020): 251–53. http://dx.doi.org/10.1177/0049475520925378.

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We present a patient with severe Plasmodium falciparum malaria of seven days’ duration who developed an altered sensorium of one day. During hospital admission, peripheral symmetrical gangrene of hands and feet followed, despite normal limb vasculature.

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Giron, Alec, Cameron Cox, and Brendan MacKay. "Techniques for Imaging Vascular Supply of Peripheral Nerves." Journal of Brachial Plexus and Peripheral Nerve Injury 16, no. 01 (January 2021): e24-e30. http://dx.doi.org/10.1055/s-0041-1731280.

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Abstract:

AbstractFew studies have been developed to map the vascular structures feeding peripheral nerves, with the majority using cadaveric models and inadequate sample sizes. Preliminary evidence, while limited, indicates that the mapping of these vessels may allow or preclude certain procedures in nerve reconstruction due to the location of essential arterial inflow to the vasa nervorum. This review evaluates the evidence regarding historical, current, and emerging techniques for visualizing these vascular structures in vivo and considers their potential application in peripheral nerve vasculature.

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