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Relevant bibliographies by topics / Peripheral vasculature

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Academic literature on the topic 'Peripheral vasculature'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Peripheral vasculature"

1

Civelli, Valerie. "Drug Coated Balloon Angioplasty in Peripheral Vasculature: Review of Literature." Clinical Cardiology and Cardiovascular Interventions 2, no. 4 (December 16, 2019): 01–03. http://dx.doi.org/10.31579/2641-0419/034.

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WORCESTER, SHARON. "Estrogen Drop Affects Peripheral Vasculature." Ob.Gyn. News 40, no. 8 (April 2005): 28. http://dx.doi.org/10.1016/s0029-7437(05)70207-x.

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Mulvany, Michael J. "PERIPHERAL VASCULATURE IN ESSENTIAL HYPERTENSION." Clinical and Experimental Pharmacology and Physiology 23, s1 (November 1996): s6—s10. http://dx.doi.org/10.1111/j.1440-1681.1996.tb03034.x.

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Thom, S., A. Hughes, G. Martin, H. Nielsen, P. Inkpen, M. Schachter, and P. Sever. "Peptides in human peripheral vasculature." Regulatory Peptides 22, no. 4 (September 1988): 434. http://dx.doi.org/10.1016/0167-0115(88)90211-x.

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Douek, Philippe C. "MultiHance in MRA of peripheral vasculature." European Radiology Supplements 14, S7 (August 2004): O55—O60. http://dx.doi.org/10.1007/s10406-004-0065-6.

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Douek, Philippe C. "MultiHance in MRA of peripheral vasculature." European Radiology Supplements 15, S5 (December 2005): e17-e23. http://dx.doi.org/10.1007/s10406-005-0162-1.

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Reilly, Dermot F., Elizabeth J. Westgate, and Garret A. FitzGerald. "Peripheral Circadian Clocks in the Vasculature." Arteriosclerosis, Thrombosis, and Vascular Biology 27, no. 8 (August 2007): 1694–705. http://dx.doi.org/10.1161/atvbaha.107.144923.

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Hughes, A., G. Martin, S. Thom, and P. Sever. "Dopaminergic Mechanisms in Human Peripheral Vasculature." Journal of Hypertension 3, no. 6 (December 1985): 664–65. http://dx.doi.org/10.1097/00004872-198512000-00024.

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Schmiedl, Udo P., Chun Yuan, Hanh V. Nghiem, Thomas C. Winter, and Patrick C. Freeny. "MR angiography of the peripheral vasculature." Seminars in Ultrasound, CT and MRI 17, no. 4 (August 1996): 404–11. http://dx.doi.org/10.1016/s0887-2171(96)90026-8.

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Woods, Bartholomew O’Beirne. "Clinical Evaluation of the Peripheral Vasculature." Cardiology Clinics 9, no. 3 (August 1991): 413–27. http://dx.doi.org/10.1016/s0733-8651(18)30280-7.

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Dissertations / Theses on the topic "Peripheral vasculature"

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Kneale, Barry J. "The influence of gender on forearm resistance vessel function." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312451.

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Hunt, Julie. "The impact of blood flow restricted exercise on the peripheral vasculature." Thesis, Loughborough University, 2014. https://dspace.lboro.ac.uk/2134/14595.

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Distortion to hemodynamic, ischemic and metabolic stimuli during low load resistance exercise with blood flow restriction (BFR) may influence regional vascular adaptation. This thesis investigated the acute response and chronic adaptations of the peripheral vasculature to low load resistance exercise with BFR. The methodology utilised Doppler ultrasound, strain gauge plethysmography and muscle biopsy for insightful measures of the vasculature at different regions of the arterial tree. Short term (4-6 weeks) localised low load (30-40% 1RM) resistance exercise with BFR increased brachial (3.1%) and popliteal (3.3%) artery maximal diameter (in response to ischemic exercise), forearm (29%) and calf (24%) post-occlusive blood flow, and calf filtration capacity (14%). These findings indicate potential vascular remodelling at the conduit (chapters 3, 4) resistance (chapter 4) and capillary (chapter 4) level of the vascular tree. Regional, rather than systemic, factors are responsible for these adaptations as evidenced by an absent response in the contralateral control limb. Transient improvements in popliteal artery FMD% occurred at week 2 before increased maximal diameter at week 6, suggesting functional changes precede structural remodelling (chapter 4). Maximal brachial artery diameter and forearm post-occlusive blood flow returned to baseline values after a 2 week detraining period, signifying rapid structural normalisation after stimulus removal (chapter 3). Enhanced capillarity, despite low training loads, could be explained by augmentation of VEGF (~7 fold), PGC-1α (~6 fold) and eNOS (~5 fold) mRNA, and upregulation VEGFR-2 (~5 fold) and HIF-1α (~2.5 fold) mRNA with BFR (chapter 5). This indicates a targeted angiogenic response potentially mediated through enhanced metabolic, ischemic and shear stress stimuli. Large between subject variability in the level of BFR was observed during upper and lower limb cuff inflation protocols. Adipose tissue thickness and mean arterial pressure were the largest independent determinants of upper and lower limb BFR, respectively (Chapter 6). In conclusion, this thesis demonstrates that low load resistance exercise with BFR induces adaptation in the conduit, resistance and capillary vessels. The mediators of this response are likely to be the hemodynamic and chemical signals elicited by repeated bouts of BFR resistance exercise, although confirmation of these mechanisms is required. The functional significance of these adaptations is unknown and warrants further investigation.

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Aubdool, Aisah Aniisah. "The role of the Transient Receptor Potential Ankyrin-1 in the peripheral vasculature." Thesis, King's College London (University of London), 2014. https://kclpure.kcl.ac.uk/portal/en/theses/the-role-of-the-transient-receptor-potential-ankyrin1-in-the-peripheral-vasculature(c632b315-f86c-47f8-be01-55e1904550e1).html.

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The phenomenon of cold-induced vasodilatation (CIVD) was discovered by Sir Thomas Lewis (1930) and has been extensively investigated as it is involved in protecting against local cold-induced injury. The mechanisms underlying this well-established protective response remain unclear. The non-selective cation channel, transient receptor potential ankyrin-1 (TRPA1) is expressed in a subset of sensory neurons and acts as a polymodal membrane channel for cold sensitivity, but this remains a controversial issue in the literature. Additionally, the role of TRPA1 as a vascular cold sensor is currently unknown. Previous studies in our group have shown that TRPA1 plays an important role in regulating peripheral blood vessel tone, with little information available on the downstream signalling mechanism. The aims of this PhD project were to investigate the effects of TRPA1 activation by an exogenous agonist cinnamaldehyde and local cold exposure on peripheral vascular responses in murine skin in vivo. Using a combination of pharmacological antagonists and genetically modified mice, topical application of cinnamaldehyde (10%) was shown to increase blood flow in a TRPA1-dependent manner in the mouse ear model. This response was further shown to be dependent on the release of the potent microvascular vasodilator calcitonin gene-related peptide (CGRP), highlighting the involvement of a neurogenic component. This study provides novel evidence demonstrating the relative contribution of neuronal nitric-oxide synthase (nNOS)-derived nitric oxide and reactive oxygen species, downstream of TRPA1 activation by cinnamaldehyde. These findings highlight the prominent role of TRPA1 in mediating peripheral vasodilatation. The project further progressed to the development and characterisation of a local cold model in the mouse hindpaw in vivo. Local cold exposure was shown to cause a rapid and transient vasoconstriction, followed by a prolonged vasodilatation phase to return blood flow to baseline, an essential physiological function for protecting against local cold-induced injury. The activation of TRPA1 in the peripheral vasculature was shown to drive this cold-induced vascular response at 10°C. This model enabled the determination of the relative role of sympathetic nerves, post-junctional α2-adrenergic receptors and reactive oxygen species in the local cold-induced vasoconstriction. This study provides novel evidence showing that local cold exposure causes an increase in intracellular superoxide production in a TRPA1-dependent manner, which activates the Rho-kinase-mediated pathways and induces cold-induced α2C-adrenergic vasoconstriction. The neuropeptide CGRP was subsequently shown to have a prominent role in the vasodilator phase. This study provide novel evidence of a major involvement of TRPA1 in mediating cold-induced vasoconstriction in vivo, with a new perspective of the underlying mechanisms mediating the protection against local-cold induced injury.

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hsueh, Hsueh-Wen, and 薛學文. "Studies on Peripheral Nerves and Vasculatures in Peripheral Artery Disease." Thesis, 2019. http://ndltd.ncl.edu.tw/handle/z3g853.

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碩士
國立臺灣大學
臨床醫學研究所
107
Background: Peripheral artery disease (PAD) is a common disorder, affecting 3-10% in general population. It impaired the functional capacity and the quality of life. Intermittent claudication is the most famous symptoms of PAD, but there were only 10~20% patients with PAD presented this pathognomonic symptom. More patients present atypical symptoms or are asymptomatic. The discrepancy of the blood flow change and functional capacity also indicates the possibility of other mechanisms underlying the PAD. PAD-related neuropathy is one possible mechanism, but less investigated in the past. This study would explore the PAD-related neuropathy pathogenesis and the nerve damage change with the integrated examinations. Methods: Patients, aged at least 20 years old, below the 90 years old, with diagnosis of PAD by the computed topography angiography or digital substraction angiography would be enrolled in this study. The data of clinical stage, questionaaires for quality of life (QOL) and walking ability, nerve conduction studies and nerve/vascular sonography would be collected. The analysis would be performed for (1)the comparison between the PAD and normal groups and (2) correlation between each parameters. Results: Nineteen patients (mean age: 69.9 ± 8.9 years; male) were recruited for further analysis. Among all patients, 12 (63.2%) patients had diabetes mellitus, and 13 (68.4%) patients had hyperlipidemia. The questionnaires of quality of life were correlated to the Fontaine stage. The abnormality rate of nerve conduction study swas 85.7%. The compound motor action ptoential of tibial and peroneal nerve correlated to the Fontane stage, QOL, and walking impairment questionnaire. The cross sectional area and nerve height index were similar between the PAD group and normal group. The resistance index of the finger digital artery, peak systolic velocity of the proximal foot, peak systolic velocity of the distal foot, and mean velocity of the distal foot were siginifcantly different between the PAD and control groups. However, only the PSV of the distal foot correlated well to the Fontane stage, questionnaire of QOL, and walking impairment questionnaire. However, no significant correlation was noted between the ultrasound and nerve conduction studies. Conclusion: This study showed the nerve conduction study and the flow velocity of the very distal artery of the foot could reflect the severity of PAD. However, the interaction between the very distal artery and nerve needs further study.

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Books on the topic "Peripheral vasculature"

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Kahn, S. Lowell. Balloon Anchor Techniques for Sheath, Guide Catheter, and Stent Advancement and to Facilitate Chronic Total Occlusion Traversal. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0061.

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Advancement of a sheath or guide catheter into a small, diseased or angled branch vessel such as the superior mesenteric artery or renal artery can be difficult. Similarly, there are times when placement of a sheath up and over a sharply angulated aortic bifurcation can present a challenge. Obtaining a sheath position at or beyond a stenotic or occlusive lesion may be critical for delivering a stent, particularly with the inherent risk of dislodgment associated with balloon-expandable stents. The use of balloons as anchors has been described most commonly in the coronary vasculature, but it can have an important role in peripheral and visceral applications. This chapter discusses the utility of balloons as an anchor to advance a sheath or stent to a target location.

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Covic, Adrian, Mugurel Apetrii, Luminita Voroneanu, and David J. Goldsmith. Vascular calcification. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.

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Vascular calcification (VC) is a common feature of patients with advanced CKD and it could be, at least in part, the cause of increased cardiovascular mortality in these patients. From a morphologic point of view, there are at least two types of pathologic calcium phosphate deposition in the arterial wall—namely, intima calcification (mostly associated with atherosclerotic plaques) and media calcification (associated with stiffening of the vasculature, resulting in significantly adverse cardiovascular outcomes). Although VC was viewed initially as a passive phenomenon, it appears to be a cell-mediated, dynamic, and actively regulated process that closely resembles the formation of normal bone tissue, as discovered recently. VC seems to be the result of the dysregulation of the equilibrium between promoters and inhibitors. The determinants are mostly represented by altered calcium and phosphorus metabolism, secondary hyperparathyroidism, vitamin D excess, high fibroblast growth factor 23, and high levels of indoxyl sulphate or leptin; meanwhile, the inhibitors are vitamin K, fetuin A, matrix G1a protein, osteoprotegerin, and pyrophosphate. A number of non-invasive imaging techniques are available to investigate cardiac and vascular calcification: plain X-rays, to identify macroscopic calcifications of the aorta and peripheral arteries; two-dimensional ultrasound for investigating the calcification of carotid arteries, femoral arteries, and aorta; echocardiography, for assessment of valvular calcification; and, of course, computed tomography technologies, which constitute the gold standard for quantification of coronary artery and aorta calcification. All these methods have a series of advantages and limitations. The treatment/ prevention of VC is currently mostly around calcium-mineral bone disease interventions, and unproven. There are interesting hypotheses around vitamin K, Magnesium, sodium thiosulphate and other potential agents.

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Waldmann, Carl, Neil Soni, and Andrew Rhodes. Obstetric emergencies. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199229581.003.0031.

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Pre-eclampsia 518Eclampsia 520HELLP syndrome 522Postpartum haemorrhage 524Amniotic fluid embolism 526Pre-eclampsia is a common complication of pregnancy, UK incidence is 3–5%, with a complex hereditary, immunological and environmental aetiology.Abnormal placentation is characterized by impaired myometrial spiral artery relaxation, failure of trophoblastic invasion of these arterial walls and blockage of some vessels with fibrin, platelets and lipid-laden macrophages. There is a 30–40%, reduction in placental perfusion by the uterine arcuate arteries as seen by Doppler studies at 18–24 weeks gestation. Ultimately the shrunken, calcified, and microembolized placenta typical of the disease is seen. The placental lesion is responsible for fetal growth retardation and increased risks of premature labour, abruption and fetal demise. Maternal systemic features of this condition are characterized by widespread endothelial damage, affecting the peripheral, renal, hepatic, cerebral, and pulmonary vasculatures. These manifest clinically as hypertension, proteinuria and peripheral oedema, and in severe cases as eclamptic convulsions, cerebral haemorrhage (the most common cause of death due to pre-eclampsia in the UK), pulmonary oedema, hepatic infarcts and haemorrhage, coagulopathy and renal dysfunction....

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Banerjee, Amitava, and Kaleab Asrress. Screening for cardiovascular disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0351.

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Screening involves testing asymptomatic individuals who have risk factors, or individuals who are in the early stages of a disease, in order to decide whether further investigation, clinical intervention, or treatment is warranted. Therefore, screening is classically a primary prevention strategy which aims to capture disease early in its course, but it can also involve secondary prevention in individuals with established disease. In the words of Geoffrey Rose, screening is a ‘population’ strategy. Examples of screening programmes are blood pressure monitoring in primary care to screen for hypertension, and ultrasound examination to screen for abdominal aortic aneurysm. The effectiveness and feasibility of screening are influenced by several factors. First, the diagnostic accuracy of the screening test in question is crucial. For example, exercise ECG testing, although widely used, is not recommended in investigation of chest pain in current National Institute for Health and Care Excellence guidelines, due to its low sensitivity and specificity in the detection of coronary artery disease. Moreover, exercise ECG testing has even lower diagnostic accuracy in asymptomatic patients with coronary artery disease. Second, physical and financial resources influence the decision to screen. For example, the cost and the effectiveness of CT coronary angiography and other new imaging modalities to assess coronary vasculature must be weighed against the cost of existing investigations (e.g. coronary angiography) and the need for new equipment and staff training and recruitment. Finally, the safety of the investigation is an important factor, and patient preferences and physician preferences should be taken into consideration. However, while non-invasive screening examinations are preferable from the point of view of patients and clinicians, sometimes invasive screening tests may be required at a later stage in order to give a definitive diagnosis (e.g. pressure wire studies to measure fractional flow reserve in a coronary artery). The WHO’s principles of screening, first formulated in 1968, are still very relevant today. Decision analysis has led to ‘pathways’ which guide investigation and treatment within screening programmes. There is increasing recognition that there are shared risk factors and shared preventive and treatment strategies for vascular disease, regardless of arterial territory. The concept of ‘vascular medicine’ has gained credence, leading to opportunistic screening in other vascular territories if an individual presents with disease in one territory. For example, post-myocardial infarction patients have higher incidence of cerebrovascular and peripheral arterial disease, so carotid duplex scanning and measurement of the ankle–brachial pressure index may be valid screening approaches for arterial disease in other territories.

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Book chapters on the topic "Peripheral vasculature"

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Feliciano, David V. "Peripheral Vasculature." In Acute Care Surgery, 656–74. New York, NY: Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-69012-4_40.

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Bernard, C., A. Tedgui, and D. Payen. "The Peripheral Vasculature." In Update in Intensive Care and Emergency Medicine, 267–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-84827-8_19.

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Bilbao, Juan M., and Robert E. Schmidt. "The Peripheral Nerve Vasculature." In Biopsy Diagnosis of Peripheral Neuropathy, 111–22. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-07311-8_6.

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Leiner, Tim, and Jeffrey H. Maki. "MRA of the Peripheral Vasculature." In Clinical Blood Pool MR Imaging, 93–114. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-77861-5_9.

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Fail, Peter S., and Vinod Nair. "Imaging of the Peripheral Vasculature and Carotid Arteries." In Atlas of Cardiovascular Computed Tomography, 205–20. London: Springer London, 2017. http://dx.doi.org/10.1007/978-1-4471-7357-1_12.

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White, Brian, Peter Gonzalez, Gerard A. Malanga, and Venu Akuthota. "Physical Examination of the Peripheral Nerves and Vasculature." In Nerve and Vascular Injuries in Sports Medicine, 41–59. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-76600-3_4.

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Balogh, Péter. "Developmental Relationship and Convergence Between the Formation of Lymphoid Organs and Lymphatic Vasculature." In Developmental Biology of Peripheral Lymphoid Organs, 49–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-14429-5_6.

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Niessen, Wiro, Alexander Montauban van Swijndregt, Bernard Elsman, Onno Wink, Max Viergever, and Willem Mali. "Enhanced Artery Visualization in Blood Pool MRA: Results in the Peripheral Vasculature." In Lecture Notes in Computer Science, 340–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/3-540-48714-x_26.

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Riederer, Stephen J. "The Application of Highly Accelerated MR Acquisition Techniques to Imaging the Peripheral Vasculature." In IFMBE Proceedings, 169–73. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-00846-2_42.

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Syed, Maaz B. J., Jakub Kaczynski, and David E. Newby. "18F-Sodium Fluoride Positron Emission Tomography/Computed Tomography Imaging of the Peripheral Vasculature." In Sodium Fluoride PET/CT in Clinical Use, 85–94. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-23577-2_11.

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Conference papers on the topic "Peripheral vasculature"

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Ermilov, Sergey, Richard Su, Mario Zamora, Travis Hernandez, Vyacheslav Nadvoretsky, and Alexander Oraevsky. "Optoacoustic angiography of peripheral vasculature." In SPIE BiOS, edited by Alexander A. Oraevsky and Lihong V. Wang. SPIE, 2012. http://dx.doi.org/10.1117/12.911629.

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Vasudevan, Srikanth, Doe Kumsa, Pavel Takmakov, Cristin G. Welle, and Daniel X. Hammer. "Real time imaging of peripheral nerve vasculature using optical coherence angiography." In SPIE BiOS, edited by Steen J. Madsen, Victor X. D. Yang, E. Duco Jansen, Qingming Luo, Samarendra K. Mohanty, and Nitish V. Thakor. SPIE, 2016. http://dx.doi.org/10.1117/12.2213160.

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Hoi, J. W., M. A. Khalil, H. K. Kim, and A. H. Hielscher. "Contact-free diffuse optical tomography system for dynamic imaging of peripheral vasculature." In Biomedical Optics. Washington, D.C.: OSA, 2014. http://dx.doi.org/10.1364/biomed.2014.bm3a.72.

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Giordano, M., C. Neukirchen, M. Bertram, W. Mali, M. A. Viergever, and E. J. Vonken. "Perfusion estimation in the peripheral vasculature using C-arm X-ray systems." In 2009 IEEE Nuclear Science Symposium and Medical Imaging Conference (NSS/MIC 2009). IEEE, 2009. http://dx.doi.org/10.1109/nssmic.2009.5401583.

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Wang, Roy, Rudolph L. Gleason, and Luke Brewster. "Diameter Constriction Reduces Intramural Circumferential Stress Gradient in the Vein Under Arterial Pressures." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80797.

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Coronary and peripheral artery diseases are a leading cause of morbidity and mortality in developed countries. For severe cases, surgical intervention to bypass the disease using autologous vessels continues to be the preferred choice of treatment. These bypass vessels are typically obtained from the venous vasculature. Despite the superior long-term patency of veins over synthetic grafts, one-year failure rates approach 30–40% in both the coronary and peripheral systems [1–2]. Still, bypass surgery remains the recommended therapy for most persons with severe arterial blockages [3]. As the number of bypass procedures increase and patients receiving bypasses live longer, improving the lifetime of bypass grafts is increasingly important.

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Torrents-Barrena, Jordina, Gemma Piella, Narcis Masoller, Eduard Gratacos, Elisenda Eixarch, Mario Ceresa, and Miguel A. Gonzalez Ballester. "Automatic Segmentation Of the Placenta and its Peripheral Vasculature in Volumetric Ultrasound for TTTS Fetal Surgery." In 2019 IEEE 16th International Symposium on Biomedical Imaging (ISBI). IEEE, 2019. http://dx.doi.org/10.1109/isbi.2019.8759296.

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Park, Seungman, Catherine Whittington, Mervin C. Yoder, Sherry Voytik-Harbin, and Bumsoo Han. "Effects of Collagen Microstructure on the Transport Properties of Vascularized Engineered Tissues." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80817.

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When vascular perfusion is compromised within the body, a number of physiological phenomena can occur, including the progression of disease states (e.g. peripheral vascular disease), impaired wound healing, and organ/tissue transplant failure. Therefore, there is a need for an engineered tissue construct that promotes therapeutic vasculogenesis by enhancing the formation of functional, stabilized vessel networks that have the ability to integrate with the host vasculature. In order to restore tissue structure and function, a matrix-based delivery system, which combines a biopolymer and endothelial precursor cells, is necessary to ensure the localization and guidance of vessel formation and stabilization.

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Mookerjee, Ashis, Ahmed M. Al-Jumaily, Andrew Lowe, and Berend E. Westerhof. "Gender-Specific Transfer Functions Might Give More Accurate Estimates of Central Pressure." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19207.

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This study investigates whether individualized transfer functions for estimating central pressures can be derived based on statistical information on the physical properties of the forearm vasculature. Physical parameters of the uniform thin walled artery model are assigned based on clinical measurements and invasive pressure measurements (n = 20) are used to calculate the effective peripheral load. The calculated load is parameterized by a Windkessel model and the variation of the Windkessel parameters is analyzed with gender, age and height. Whilst there is high spread and poor correlation of the data with age and height, there is clear clustering of data with gender. This suggests that model parameters are significantly different in either gender, and gender-specific transfer functions might be more appropriate for accurate reconstruction of central pressure.

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Holton, Andrea D., Brigitta C. Brott, Edward G. Walsh, Ramakrishna Venugopalan, Alan M. Shih, Roy Koomullil, Yasushi Ito, and Andreas S. Anayiotos. "Comparative Computational Fluid Dynamics and Experimental Phase-Contrast MRI: Evaluations of In-Stent Restenosis." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-59355.

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While angiography and other translesional catheter-based assessments of stented peripheral vasculature are currently used in clinical applications, a quantitative non-invasive imaging modality would improve the treatment of intermediate levels of in-stent restenosis (ISR). The use of magnetic resonance imaging (MRI), in metal stents has been limited due to magnetic susceptibility artifacts and radiofrequency shielding effects. However, MRI compatible materials such as nickel-titanium alloys used in stents have shown superior lumen visibility. In this study, we used phase contrast MRI in a flow phantom of three different geometries of stenosis: a) 90% axisymmetric, b) 75% axisymmetric and c) 50% asymmetric. The velocity distribution was obtained at 3 different locations inside the stent. This was compared with an equivalent computational fluid dynamics (CFD) model of the same stenotic geometries.

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Chen, Jia-Jiun, Chi-Shiun Chiang, Ji-Hong Hong, and Chih-Kuang Yeh. "Characterize the vasculatures distribution of murine tumors between center and peripheral regions based on doppler ultrasound and immunofluorescent analysis." In 2011 IEEE International Ultrasonics Symposium (IUS). IEEE, 2011. http://dx.doi.org/10.1109/ultsym.2011.0566.

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