Relevant bibliographies by topics / Peripheral circulation Regulation

Academic literature on the topic 'Peripheral circulation Regulation'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Peripheral circulation Regulation"

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Sparks, H. V. "Learning the regulation of peripheral blood flow." Advances in Physiology Education 277, no. 6 (December 1999): S164. http://dx.doi.org/10.1152/advances.1999.277.6.s164.

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Students can learn a great deal about the peripheral circulation when teaching is based on five building blocks: hemodynamic principles, neurohumoral control, and three elements of local control of blood flow (metabolic, myogenic, and paracrine). Study of a particular special circulation starts with the application of these building blocks in the context of the function of that tissue. For example, control of skin blood flow is largely concerned with regulation of body temperature (neurohumoral control) and the response to injury (paracrine control). Regulation of coronary blood flow is almost entirely a matter of meeting the metabolic needs of the myocardium (metabolic control). By mixing and matching the five building blocks and keeping in mind the special functions of a particular tissue, students can master the peripheral circulation efficiently.
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Al-Shammari Mohammed Jasim Ismael, Hayder Yousif Falih, and Sagalaeva Irina Vladimirovna. "Hemodynamic criteria of the circulatory system in ethnic groups of students with different types of autonomic regulation of the heart rate." journal of the college of basic education 25, no. 104 (September 25, 2019): 312–20. http://dx.doi.org/10.35950/cbej.v25i104.4651.

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Under physiological conditions, the first years of university studies of the students of Arabic and African subgroups with moderate parasympathetic autonomous regulation) MPAR( and self-regulation )SR( were characterized by toughness, low effectiveness of the system of blood circulation, increased peripheral vascular resistance, vascular type of self-regulation of blood circulation )TSC (; Indian and Latino-American subgroups with MPAR SR revealed the weakness and low efficiency of the circulatory system, the optimal general peripheral blood circulation )GPBC( and cardiovascular type of self-regulation of blood circulation )TSC( were revealed in Indian and Latino-American subgroups with moderate parasympathetic autonomous regulation self-regulation )MPAR SR (and subgroups with prounonced parasympathetic autonomous regulation self-regulation)PPAR SR( showed high endurance of the circulatory system. The Russian subgroup with moderate parasympathetic autonomous regulation self-regulation) MPAR SR (has the highest endurance of the circulatory system and current functional fatigue, the most marked in the subgroup with prounonced parasympathetic autonomous regulation )PPAR (increased general peripheral blood circulation and cardiovascular type of self-regulation of blood circulation.
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Upchurch, Gilbert R. "Nitric oxide and the regulation of the peripheral circulation." Journal of Vascular Surgery 34, no. 2 (August 2001): 379. http://dx.doi.org/10.1067/mva.2001.115818.

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&NA;. "NITRIC OXIDE AND THE REGULATION OF THE PERIPHERAL CIRCULATION." Shock 14, no. 2 (August 2000): 246. http://dx.doi.org/10.1097/00024382-200014020-00031.

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Slavnoe, N. V., V. V. Markov, N. A. Kovpan, V. M. Rudichenko, and G. N. Terekhova. "Peripheral circulation regulation in patients with the hypothalamic syndrome neuroendocrine metabolic form." Problems of Endocrinology 39, no. 6 (December 15, 1993): 17–20. http://dx.doi.org/10.14341/probl11928.

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Peripheral circulation and regulating hormonal (renin- angiotensin-aldosterone system) and electrolytic (plasma sodium and potassium) factors were studied in 102 patients with the hypothalamic syndrome neuroendocrine metabolic form administered pathogenetic therapy with antiserotonin and dopaminergic drugs as well as routine therapy. Blood plasma sodium vasopressin and aldosterone levels were found increased, arterial vessel reactivity in the forearm reduced, and venous circulation disordered in these patients. Routine therapy failed to normalize electrolytes and hormonal parameters and was conducive to a still more marked reduction of arterial vessel reactivity. Peritol therapy resulted in a reduction of vasopressin concentration and normalization of blood plasma sodium and aldosterone, as well as in improvement of the myogenic mechanisms of vascular tone regulation and normalization of venous circulation parameters. A course of parlodel therapy lead to normalization of blood plasma levels of vasopressin, aldosterone, and sodium but no changes in the regional vessels were observed.
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Manasyan, S. G., S. Yu Ermolov, A. G. Apresyan, and A. V. Arutyunyan. "Modified methods of polygepatography and peripheral arterial tonometry in the assessment of peripheral circulation regulation." "Arterial’naya Gipertenziya" ("Arterial Hypertension") 27, no. 6 (March 18, 2022): 683–95. http://dx.doi.org/10.18705/1607-419x-2021-27-6-683-695.

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Objective. The purpose of the work was to assess the application of modified ways of polyhepatography (PHG) and peripheral arterial tonometry (PAT) in the evaluation of regulation of peripheral circulation (capillary blood flow).Design and methods. We included 150 people, divided into four groups. The first group (n = 40) includes patients with stage II hypertension, moderate and high risk of cardiovascular complications. The second group (n = 40) includes patients with stable forms of coronary heart disease in combination with hypertension. The third group (n = 40) includes patients with chronic liver diseases. The fourth group (n = 30) consisted of subjects without anamnestic and objective data of pathology. All subjects underwent a comprehensive clinical and laboratory examination, an assessment of intrahepatic hemodynamics by the PGG method, an assessment of the endothelial function by the PAT method. A modified method of PAT was used to evaluate the central reaction of the peripheral blood flow regulation system (endothelium-independent vasodilation).Results. Endothelial dysfunction was found in patients with cardiovascular pathology and in patients with chronic liver diseases. A modified method of PAT showed a multidirectional reaction of peripheral blood flow to the test with local ischemia. A number of features were identified in the study groups when assessing disorders of intrahepatic microcirculation. Patients of group I had multidirectional disorders of arteriovenous inflow (45% cases, confidence interval (CI) from 27 % to 63 %) and outflow (37,5 %, CI from 22 % to 56 %) in the liver, while in patients of group II and group III, disorders of arteriovenous inflow were more common, 85 % (CI from 70 % to 95%) and 90% (82 % to 94%), respectively. Rheographic signs of bile passage disorders were more common in groups II and III. A significant negative relationship was established between endothelial dysfunction and the severity of intrahepatic microcirculation disorders (r = –0,35, p < 0,001).Conclusions. Modified methods of peripheral arterial tone and PHG enable assessment of local and central mechanisms of blood flow regulation at the microvascular level in patients with hypertension, coronary heart disease and chronic liver diseases. The relationship between impaired endothelial function and intrahepatic microcirculation allows us to consider the liver as a target organ in cardiovascular pathology.
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Andersson, Rune, and Per Bjerle. "Peripheral Circulation, Particularly Heat Regulation Reactions, in Patients with Amyloidosis and Polyneuropathy." Acta Medica Scandinavica 199, no. 1-6 (April 24, 2009): 191–96. http://dx.doi.org/10.1111/j.0954-6820.1976.tb06715.x.

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Zadorozhnia, V., O. Kuchkovsky, and O. Kovaleva. "Vegetative status and adaptation peculiarities possibilities in student youth depending on blood circulation self-regulation type." Visnyk of Lviv University. Biological series, no. 83 (December 25, 2020): 83–97. http://dx.doi.org/10.30970/vlubs.2020.83.10.

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Vegetative status and adaptive capabilities peculiarities of aged 19 to 21 girls depending on their blood circulation self-regulation type were studied in this work. The study was conducted at the Biology Faculty of Zaporizhzhia National University, Zaporizhzhia. Such primary indices as heart rate and blood pressure were studied in all test individuals. Based on the obtained data, the blood circulation self-regulation type in each subject was determined separately, which allowed to form three groups (individuals with cardiovascular, vascular and mixed blood circulation self-regulation types). The assessment scheme included cardiovascular system indices calculation such as average dynamic arterial pressure, specific peripheral resistance, cardiac output, cardiac index, stroke volume, external myocardial function, myocardial stress index, myocardial efficiency criterion, autonomic regulation index (vegetative Kerdo’s index) and adaptive potential. The results were processed by variation statistics methods, and the obtained data were subjected to correlation analysis. Deviations from the reference values were revealed in systemic hemodynamics ave­rage group parameters analysis in female students with different blood circulation regulation types. It was recorded that the average dynamic pressure exceeded the normal upper limit by 2.5 % and 6.2 %, respectively, in persons with mixed and vascular regulation type. Specific peripheral resistance indices obtained from data analysis in girls with different blood circulation self-regulation types revealed that this index did not exceed the normal in the groups with mixed and vascular type, in contrast to the group with the cardiac type. The specific peripheral resistance was lower by 5.2 % than the lower limit of the reference value for this index in the latter group. Statistically significant differences were identified in cardiac output, cardiac index and stroke volume parameters in girls with different self-regulation circulatory types. Specific trends were identified in the average group and individual myocardial function indices in girls depending on the self-regulation type. Statistical significance was found between myocardial stress indices in girls with different self-regulation types. Both the average group and individual vegetative Kerdo’s index indices fluctuated within eytony in the mixed type persons group. The mean group autonomic index values in girls with vascular type indicated pronounced vagotonia. We found that the largest percentage of all respondents had satisfactory adaptation. The largest number of girls with satisfactory adaptation had a vascular self-regulation type (83.33 %) and a mixed type (81.82 %), a smaller percentage of girls from these groups (16.68 % and 18.18 %, respectively) had adaptation mechanisms functional stress. In the group with the cardiac type, almost half (46.15 %) had adaptation mechanisms functional stress, and the rest (53.85 %) had satisfactory adaptation. Thus, the differences in cardiovascular system indices, the autonomic nervous system sympathetic and parasympathetic parts influences ratio on the cardiovascular system were determined, as well as the adaptive potential in 19-21 years old girls can be attributed to blood circulation compensatory-adaptive reactions.
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Sheriff, D. D. "PASSIVE REGULATION OF CARDIAC OUTPUT BY THE ELASTIC CHARACTERISTICS OF THE PERIPHERAL CIRCULATION." Medicine & Science in Sports & Exercise 30, Supplement (May 1998): 217. http://dx.doi.org/10.1097/00005768-199805001-01240.

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Thomas, Gail D. "Neural control of the circulation." Advances in Physiology Education 35, no. 1 (March 2011): 28–32. http://dx.doi.org/10.1152/advan.00114.2010.

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The purpose of this brief review is to highlight key concepts about the neural control of the circulation that graduate and medical students should be expected to incorporate into their general knowledge of human physiology. The focus is largely on the sympathetic nerves, which have a dominant role in cardiovascular control due to their effects to increase cardiac rate and contractility, cause constriction of arteries and veins, cause release of adrenal catecholamines, and activate the renin-angiotensin-aldosterone system. These effects, as well as the control of sympathetic outflow by the vasomotor center in the medulla and the importance of sensory feedback in the form of peripheral reflexes, especially the baroreflexes, are discussed in the context of cardiovascular regulation.
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Dissertations / Theses on the topic "Peripheral circulation Regulation"

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Ahmed, Badreldeen Ibrahim. "A study of peripheral circulation dynamics and regulation in non-pregnant, pregnant and in preeclamptic women using Applied Potential Tomography (APT)." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324863.

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Choo, Hui C. "Peripheral blood flow changes in response to post-exercise cold water immersion." Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2014. https://ro.ecu.edu.au/theses/1012.

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A reduction in body temperature is considered to be the primary mechanism by which cold water immersion (CWI) enhances short-term (h) recovery and improves exercise capacity in the heat. However, improvement in exercise performance may be optimised at a given cooling magnitude. Water temperature and immersion duration influence the magnitude of cooling in the core body, muscle and skin. Given the role of blood flow in convective heat flux, substrate delivery and metabolic waste clearance, it is important to understand the influence of different water temperatures on compartmental distribution of limb blood flow during CWI. Therefore, the purpose of this study was to compare blood flow changes in the common femoral artery, vastus lateralis muscle, and thigh skin induced by 5 min of post-exercise water immersion at 8°C, 14°C, 35°C or passive rest. In a randomised manner, nine recreationally active men performed exhaustive cycling in a climate control chamber (32.8 ± 0.4°C and 32 ± 5%rh), followed by 5 min of water immersion at 8.6 ± 0.2°C (WI8), 14.6 ± 0.3°C (WI14), 35.0 ± 0.4°C (WI35) or passive rest (CON). The exercise task involved 25 min of cycling at a power output equivalent to first ventilatory threshold, followed by high-intensity intermittent cycling (30 s at 90% of peak power output to 30 s at 70 W). Measurement of blood flow in thigh skin (laser Doppler flowmetry), vastus lateralis muscle (near infrared spectroscopy), and common femoral artery (Doppler ultrasound), heart rate, mean arterial pressure, skin, muscle, rectal, and mean body temperatures were obtained prior to exercise and up to 60 min post-immersion. Both WI14 and WI8 reduced mean body, calf and thigh skin, and muscle temperatures, compared with WI35 and CON (p0.05). Relative to pre-immersion, differences were observed in the magnitude of reduction between skin, muscle, and common femoral blood flow. Decreases in muscle and skin blood flow were similar (p>0.05), but to a lesser extent when compared with femoral blood flow (p Therefore, 5 min of CWI at 8°C and 14°C effectively reduced temperatures, when compared with CON and WI35. Although WI8 was more effective than WI14 in reducing mean body temperature, there was no influence on the decreases in skin, muscle and femoral blood flow. Furthermore, WI8 did not result in significant reduction in muscle blood flow compared to WI35, despite significant muscle cooling. Given that mean arterial blood pressure was elevated, it is possible hydrostatic effects during WI35, coupled with shivering thermogenesis during WI8 confounded extent of muscle blood flow reduction in the present study. As such, influence of hydrostatic pressure per se on peripheral blood flow cannot be ruled out although blood flow changes were similar between WI35 and CON. Additionally, current findings indicate unknown vascular beds, other than measured sites in the vastus lateralis muscle and thigh skin, contribute to overall changes in the limb blood flow. It appears that vasoconstriction in skin and muscle vasculatures are associated with the interaction between suppressed vasodilatory substances (e.g. nitric oxide) and altered baroreflex mediated sympathetic nerve activity. However, underlying mechanisms warrant further investigation.
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Gould, Dianna Jane. "Mechanisms of peripheral nervous control of rat iris arterioles." Phd thesis, 1996. http://hdl.handle.net/1885/142974.

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Books on the topic "Peripheral circulation Regulation"

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Kadowitz, Philip J., and Dennis B. McNamara, eds. Nitric Oxide and the Regulation of the Peripheral Circulation. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0.

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Kadowitz, Philip J., and Dennis B. Mcnamara. Nitric Oxide and the Regulation of the Peripheral Circulation. Springer, 2012.

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McNamara, Dennis B., and Philip J. Kadowitz. Nitric Oxide and the Regulation of the Peripheral Circulation. Birkhauser Verlag, 2012.

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Nitric Oxide and the Regulation of the Peripheral Circulation (Nitrogen Oxides in Biology and Medicine). Birkhauser, 2000.

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(Editor), Philip J. Kadowitz, and Dennis B. McNamara (Editor), eds. Nitric Oxide and the Regulation of the Peripheral Circulation (Nitric Oxide in Biology and Medicine). Birkhäuser Boston, 2000.

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Anders, Nobin, Owman Christer, Arneklo-Nobin Birgitta, Lunds universitet. Dept. of Histology., Lunds universitet. Dept. of Surgery., and Eric K. Fernström Symposium (10th : 1986 : Örenäs Castle, Glumslöv, Sweden), eds. Neuronal messengers in vascular function: Focus on peripheral interaction between neuropeptides and classical transmitters. Amsterdam: Elsevier Science Publishers, Biomedical Division, 1987.

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Book chapters on the topic "Peripheral circulation Regulation"

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Kerslake, D. McK, and K. E. Cooper. "Factors Concerned in the Regulation of Skin Blood Flow." In Ciba Foundation Symposium - Peripheral Circulation in Man, 115–21. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470715185.ch10.

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Lautt, W. Wayne, and M. Paula Macedo. "Nitric Oxide and the Hepatic Circulation." In Nitric Oxide and the Regulation of the Peripheral Circulation, 243–58. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_15.

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Bauer, Philippe, Zsuzsanna Rozsa, and D. Neil Granger. "Nitric Oxide and the Gastrointestinal Circulation." In Nitric Oxide and the Regulation of the Peripheral Circulation, 259–69. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_16.

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Kaye, Alan D., Bobby D. Nossaman, and Philip J. Kadowitz. "Intravenous Anesthetics in the Pulmonary Circulation." In Nitric Oxide and the Regulation of the Peripheral Circulation, 131–47. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_9.

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Cassin, Sidney. "Nitric Oxide and the Perinatal Pulmonary Circulation." In Nitric Oxide and the Regulation of the Peripheral Circulation, 185–96. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_12.

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Leffler, Charles W. "Nitric Oxide in Control of the Cerebral Circulation." In Nitric Oxide and the Regulation of the Peripheral Circulation, 113–27. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_8.

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Nossaman, Bobby D., Alan D. Kaye, and Philip J. Kadowitz. "Nitric Oxide and the Pulmonary Circulation in the Adult." In Nitric Oxide and the Regulation of the Peripheral Circulation, 197–223. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_13.

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Ignarro, Louis J. "Nitric Oxide in the Regulation of Blood Flow: A Historical Overview." In Nitric Oxide and the Regulation of the Peripheral Circulation, 1–12. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_1.

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Hurford, William E., Wolfgang Steudel, and Warren M. Zapol. "Inhaled Nitric Oxide Therapy for Acute Respiratory Failure." In Nitric Oxide and the Regulation of the Peripheral Circulation, 148–65. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_10.

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Haddad, Elie, Lesley J. Millatt, and Roger A. Johns. "Clinical Applications of Inhaled Nitric Oxide." In Nitric Oxide and the Regulation of the Peripheral Circulation, 166–84. Boston, MA: Birkhäuser Boston, 2000. http://dx.doi.org/10.1007/978-1-4612-1326-0_11.

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Conference papers on the topic "Peripheral circulation Regulation"

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Romita, L., and L. Bocchi. "Modeling of thermal regulation of peripheral circulation." In 2014 8th International Symposium on Medical Information and Communication Technology (ISMICT). IEEE, 2014. http://dx.doi.org/10.1109/ismict.2014.6825232.

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Libby, Peter, Stephen J. C. Warner, and Louis K. Birinyi. "THE VESSEL WALL AS A SOURCE OF VASORHGOLATORY AND IMMDNOSTIMOLATORY CYTOKINES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643982.

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The cytokines Interleukin-1 (IL-1) and Tumor Necrosis Factor (TNF, also known as cachectin) exhibit multiple effects on circulating blood cells and cells of the blood vessel wall. For example, these mediators elicit a coordinated Drogram of functions of endothelial cells (EC) that promotes blood coagulation and thrombosis, and lead to clot stabilization. Furthermore, IL-1 and TNF promote adherence to vascular endothelium of leukocytes of many classes.Thus, these cytokines are likely to be involved in signaling the pathologic changes in blood vessels that characterize a number of inflammatory or infectious processes. These two cytokines were originally isolated frcm activated human mononuclear phagocytes, hence their comnon designation as monokines and the terminology "interleukin". However, recent findings have broadened this concept considerably. It is now clear that many cell types can produce IL-1-1ike activity.Several groups showed that human vascular EC can secrete material that stimulates proliferation of thymocytes incubated with suboptima1 doses of the mitogenic lectin phytohemagglutinin, a typical acitivty of IL-1 (thymocyte costimulation).Two related but distinct genes cloned frcm human peripheral blood monocytes encode IL-1 molecules. In human blood monocytes stimulated with bacterial lioopolysaccharide (LPS) IL-1 beta (pi ∼ 7) is the major form expressed while IL-1 alpha (pi ∼ 5) is the less abundant species secreted by human monocytes under these conditions. We found that EC and smooth muscle cells (SMC) isolated from adult human vessels can express these same IL-1 genes. LPS, a standard stimulus to IL-1 secretion in the monocyte, caused accumulation of IL-1 beta mRNA in both vascular cell types. Endothelial cells frcm adult human vessels also contained IL-1 alpha mRNA when treated with LPS in the presence of cycloheximide and LPS-stimulated smooth muscle cells contained RNA that hybridized with an IL-1 alpha cDNA probe as well. Although both vascular cell types can transcribe these IL-1 genes, the time course of this response differs. LPS induced IL-1 beta mRNA production by SMC maximally at 4-6 hr., whereas maximal IL-1 induction by LPS in EC occured 1 day after initiation of the exposure. Actinanycin D (1 ug/ml) blocked 3H-uridine incorporation into macromolecules by > 95% in both EC & SMC, and prevented the LPS-induced increases in IL-1 mRNA levels in these cells. This result suggests that this potentially injurious stimulus causes IL-1 mRNA accumulation by an increase in rates of transcription. These LPS-induced increases in IL-1 mRNA levels corresponded to production of biologically active IL-1 determined as thymocyte costimulation activity. Interestingly, gel filtration experiments revealed a molecular weight of around 22kD for both SMC and EC-derived IL-1 secreted into culture medium in response to LPS. This molecular weight contrasts with the 17 kD species which is the fully processed product secreted frcm activated human monocytes. A possible explanation for this disparity is that the vascular cells secrete a partially processed intermediate form of mature IL-1. Other stimuli for IL-1 mRNA accumulation and secretion of biological activity include TNF and IL-1 itself. Recombinant human INF (≥ 10 ng/ml) increased IL-1 beta mRNA levels in EC & SMC, and caused the EC & SMC to release IL-1-1 ike thymocyte costimulation activity. Of interest is the recent observation that IL-1 itself can stimulate expression of IL-rl genes in vascular wall cells. Both IL-1 aloha and beta can increase IL-1 beta mRNA content in EC & SMC. Hris observation was confirmed with homogenous IL-1 prepared by recombinant DNA technologies (rIL-1). These findings raise the possibility of a novel positive feedback loop in vascular pathophysiology. We also found that rIL-1 alpha or beta also induced the production of prostaglandin E2 (PGE2) by both vascular SMC & EC. This prostanoid, induced by IL-1, inhibits thymocyte _ proliferation. Thus, IL-1 not only induced its own expression but increased production of this immunosuppressive prostanoid. This mechanism provides a potential negative control loop in regulation of the local immune response in blood vessels. Vie conclude that these cells of the blood vessel wall are a source of the potent vasoregulatory and immune mediators IL-1 alpha and beta. Since IL-1 influences the thrombotic, hemostatic, and fibrinolytic functions of endothelium, as well as other responses to acute injury, our findings suggest novel local control mechanisms that may be important in a variety of pathologic states.
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