Relevant bibliographies by topics / Periodontitis Research

Academic literature on the topic 'Periodontitis Research'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Periodontitis Research"

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Nishihara, Tatsuji. "Basic research on macrophages in periodontitis." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 60, no. 4 (December 28, 2018): 167–72. http://dx.doi.org/10.2329/perio.60.167.

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Khomich, S., B. Kritsky, and A. Khomich. "Morphological research of chronic apical periodontitis." International Journal of Oral and Maxillofacial Surgery 38, no. 5 (May 2009): 573. http://dx.doi.org/10.1016/j.ijom.2009.03.615.

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Kordiyak, Olena J. "Periodontal Destruction and Regeneration in Experimental Models: Combined Research Approaches." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 5, no. 5 (October 24, 2020): 28–34. http://dx.doi.org/10.26693/jmbs05.05.028.

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Chronic periodontitis is a common dental disease, resulting in destruction of gingival tissue, periodontal ligament, cementum, alveolar bone and, consequently- teeth loss in the adult population. Experimental animal models have enabled the study of periodontal disease pathogenesis and are used to test new therapeutic approaches for treating the disease The purpose of this review study was to draw the evidence from animal models, required for future assessment of destructional and regenerative processes in periodontal tissues. Material and methods: a rat experimental periodontitis models of ligature, streptozotocin, and immune complexes induced periodontitis, periodontal defect, altered functional loading, stress exposures and surgically created chronic acid reflux esophagitis models. Histomorphomorphological/-metrical, immunohisto (-cyto)chemical and histopathological analysis, micro-computed tomography, scanning and transmission electron microscopy, polarizing light and confocal microscopy, spectrophotometry, radiographic and biomechanical analysis, descriptive histology and computer-assisted image analysis. Results and discussion. Scaling and root planing may not always be effective in preventing periodontal disease progression, and, moreover, with currently available therapies, full regeneration of lost periodontal tissues after periodontitis cannot be achieved. However, in 70.5% of the results of experimental studies reported, irrespective of the defect type and animal model used, beneficial outcome for periodontal regeneration after periodontal ligament stem cell implantation, including new bone, new cementum and new connective tissue formation, was recorded. Therefore, platelet-rich fibrin combined with rat periodontal ligament stem cells provides a useful instrument for periodontal tissue engineering. Conclusion. There is sufficient evidence from preclinical animal studies suggesting that periodontal tissue engineering would provide a valuable tool for periodontal regeneration. Further elaboration of the developed in preclinical studies experimental techniques should justify progress to clinical studies and subsequent medical application
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Natto, Zuhair S., Randa H. Abu Ahmad, Lina T. Alsharif, Hanan F. Alrowithi, Duaa A. Alsini, Hetaf A. Salih, and Nabil F. Bissada. "Chronic Periodontitis Case Definitions and Confounders in Periodontal Research: A Systematic Assessment." BioMed Research International 2018 (November 28, 2018): 1–9. http://dx.doi.org/10.1155/2018/4578782.

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Case definitions and criteria of periodontal diseases are not yet consistent worldwide. This can affect the accuracy of any comparison made between two studies. This study determines which are the most common chronic periodontitis case definitions as well as confounding variables that have been reported worldwide in periodontal literature. A systematic assessment on periodontal disease classification and confounders was conducted using all publications in MEDLINE, EMBASE, SCOPUS, and Google Scholar between 1965 and October 2017. Screening of eligible studies and data extraction were conducted in duplicate and independently by two reviewers. The search protocol produced 4,218 articles. Out of these, 492 potentially relevant articles were selected for review. Only 351 studies fulfilled the selection criteria. Combination of probing depth and clinical attachment loss was the most common chronic periodontitis case definitions used (121, studies, 34.5%). CPI/CPITN was the most common classification used. Age was the most common confounder studied in periodontal research (303 studies, 86.3%), followed by gender (268 studies, 76.4%) and race (138 studies, 39.3%). Albumin and creatinine were the least common variables studied (1 or 2 studies each). Different case definitions affect the prevalence and treatment consequences of periodontitis. We need to standardize periodontitis case definitions worldwide to avoid difficulties in case diagnosis and prognosis. Further studies need to be done to assess the association between periodontitis and several potential confounders.
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Ali, Syed Muhammad, and Syed Mahmood Haider. "PERIODONTAL STATUS." Professional Medical Journal 25, no. 12 (December 8, 2018): 1961–65. http://dx.doi.org/10.29309/tpmj/18.4382.

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Thalassemia is a worldwide disease. 5-8% of Pakistani population (approx. 8–10 millions) are carrying thalassemia genes. It causes various abnormalities in different organs of patients. In Pakistan (to the best of our knowledge) no research work on oral and maxillofacial manifestation has been carried out, therefore present study was under taken. Setting: Abbasi Shaheed Hospital Karachi with collaboration of Husaini Institute of Blood Diseases Karachi, Fatimid Foundation and National institute of Oral Diseases Karachi Pakistan. Period: 2 years June 2013 to June 2015. Objectives: To assess the prevalence of periodontal disease and compare with age in thalassemia patient. Study Design: The sample consisted of 200 patients with Thalassemia Major, 120 males and 80 females aged 5 to 35years, with the mean age18.1. The sample was divided into three subgroups according to age. Clinical examination were carried out to assess the prevalence of change statistical differences in frequencies of percentage between the three groups. Aims of study: The aim of this study was to evaluate the frequency of periodontal status of Thalassemic patients and correlate with age from population of Karachi. Methods and Materials: 200 diagnosed β – thalassemia major patients, were randomlyselected form the Patients who were registered for their treatment. Their clinical examination and percentage of recorded feature were calculated and cross tabulated with age and (Ramfjords PDI Index) were used for evaluation of periodontitis and result were deduced. Results: Themost prominent features recorded were periodontitis in 82.5 normal patients were 17.5, Mild periodontitis 45.5% %; Moderate periodontitis 30 %; Severe periodontitis 7%. Conclusion: The increase in severity of Periodontistis increases with age and duration, necessitate regular surveillance and counselling in order to reassure the patient, reduce Periodontitis in thalassemia patients and improve the overall condition and life style of Patients.
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Zholdoshev, Ch, K. Kuttubaeva, R. Alymbaev, E. Suerkulov, and T. Abdyshev. "Indicators of Microbiological Research in Generalized Chronic Periodontitis." Bulletin of Science and Practice 7, no. 10 (October 15, 2021): 231–35. http://dx.doi.org/10.33619/2414-2948/71/25.

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The state of microflora in generalized chronic periodontitis was studied according to the data of KSMA the Department of Therapeutic Dentistry for the period of 2020. A total of 102 patients were examined.
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Ycaza Béjar, María Gracia, and Carlos Xavier Ycaza Reynoso. "Control of glucose levels in patients with periodontitis in the ucsg periodontics clinics semester a-2019." Journal of America health 3, no. 1 (January 6, 2020): 31–39. http://dx.doi.org/10.37958/jah.v3i1.25.

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Objective: Diabetes is considered a modifier of periodontal disease; it is important to consider HbA1c in patients with periodontal disease in order to determine the degree of progression found by patients in the periodontics clinics I and II of the UCSG Semester A 2019. Materials and Methods: cross-sectional, longitudinal and analytical research, measures prevalence of the exposure and effect of diabetes and periodontitis with a sample of 36 patients who are diagnosed with Periodontitis and who have a history of diabetes for 6 weeks, were examined in the clinical laboratory, After data collection, where all the results obtained from the research work were entered to perform the statistics obtained. Grade A decreased considerably from 44.44% to 16.67%, the percentage of patients in grade B remained at 50% while patients with grade C increased from 5.56% to 33.33%. Discussion: Of 36 patients 17 are diabetic, 13 at high risk of diabetes and 6 healthy. The age range with the highest prevalence was 50-59 years and the most prevalent gender in diabetes was 30.56% in men; After performing the HbA1c test, the diagnosis of periodontitis referring to Grade changes completely, keeping Grade B at 50% but Grade C increases to 33.33% and Grade A 16.67%.
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Lopez, Rodrigo, and Vibeke Baelum. "Classifying periodontitis among adolescents: implications for epidemiological research." Community Dentistry and Oral Epidemiology 31, no. 2 (March 14, 2003): 136–43. http://dx.doi.org/10.1034/j.1600-0528.2003.00022.x.

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Tonoyan, Lilit, Marlène Chevalier, Séverine Vincent-Bugnas, Robert Marsault, and Alain Doglio. "Detection of Epstein–Barr Virus in Periodontitis: A Review of Methodological Approaches." Microorganisms 9, no. 1 (December 29, 2020): 72. http://dx.doi.org/10.3390/microorganisms9010072.

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Periodontitis, an inflammatory condition that affects the structures surrounding the tooth eventually leading to tooth loss, is one of the two biggest threats to oral health. Beyond oral health, it is associated with systemic diseases and even with cancer risk. Obviously, periodontitis represents a major global health problem with significant social and economic impact. Recently, a new paradigm was proposed in the etiopathogenesis of periodontitis involving a herpesviral–bacterial combination to promote long-term chronic inflammatory disease. Periodontitis as a risk factor for other systemic diseases can also be better explained based on viral–bacterial etiology. Significant efforts have brought numerous advances in revealing the links between periodontitis and Epstein–Barr virus (EBV), a gamma herpesvirus ubiquitous in the adult human population. The strong evidence from these studies may contribute to the advancement of periodontitis research and the ultimate control of the disease. Advancing the periodontitis research will require implementing suitable methods to establish EBV involvement in periodontitis. This review evaluates and summarizes the existing methods that allow the detection and diagnosis of EBV in periodontitis (also applicable in a more general way to other EBV-related diseases), and discusses the feasibility of the application of innovative emerging technologies.
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Dietrich, Th, and K. Hoffmann. "A Comprehensive Index for the Modeling of Smoking History in Periodontal Research." Journal of Dental Research 83, no. 11 (November 2004): 859–63. http://dx.doi.org/10.1177/154405910408301107.

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Cigarette smoking is both a strong and common risk factor for chronic periodontitis. It is a multi-dimensional exposure that is difficult to model accurately. We propose a new comprehensive smoking index (CSI) that accounts for intensity, duration, and recency of smoking and allows for estimation of the half-life of the smoking effect. Using NHANES III data from 12,623 subjects aged 20+ yrs, we compared the performance of the CSI with that of various conventional approaches using multiple logistic regression models of chronic periodontitis. The estimate of the smoking effect’s half-life was 1.5 yrs (95% CI, 0.5–2.5 yrs). Use of the new index resulted in best model fit and the highest Wald statistic for the smoking effect on chronic periodontitis. The results suggest that use of the CSI may be a more comprehensive, efficient, and parsimonious approach to the modeling of smoking history in periodontal research.
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Dissertations / Theses on the topic "Periodontitis Research"

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Strout, Stephen Lewis. "Association between systemic lupus erythematosus and periodontitis." [Gainesville, Fla.] : University of Florida, 2004. http://purl.fcla.edu/fcla/etd/UFE0004857.

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Thesis (M.S.)--University of Florida, 2004.
Typescript. Title from title page of source document. Document formatted into pages; contains 43 pages. Includes Vita. Includes bibliographical references.
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Malamis, Dimitrios. "Systemic levels of inflammatory mediators in periodontitis." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1436961245.

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Голубчук, О. О. "Порівняльна дія мазі альтанової на тканини пародонта щурів із спонтанним пародонтитом." Thesis, Сумський державний університет, 2016. http://essuir.sumdu.edu.ua/handle/123456789/44945.

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Мета роботи – порівняти дію мазі альтанової на тканини пародонта щурів із спонтанним пародонтитом (СП). Досліди виконані на 35 білих щурах. Вони були поділені на групи: перша – інтактні, друга – тварини із СП, третя – щури із СП, яких лікували альтановою маззю 2%, четверта – щури із СП, яких лікували метилурациловою маззю 10%. Лікувальні пов’язки з препаратами накладали на ясна нижньої щелепи під ефірним рауш-наркозом, після чого щурів 2 год. утримували від приймання їжі та води протягом 5 днів. Стан тканин пародонту оцінювали візуально за наступними показниками: набряк, гіперемія ясен, наявність пародонтальних кишень, рухливість зубів. Для визначення дистрофії тканин пародонту виявляли ступінь оголення коренів молярів за методом А.В. Ніколаєвої і Є.В. Розовської.
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Motta, Raphael Jurca Gonçalves da. "Estudo clínico-patológico da distribuição de linfócitos citotóxicos e linfócitos T regulatórios na doença periodontal." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/58/58131/tde-04092017-074045/.

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O objetivo deste estudo foi analisar a expressão e o padrão de distribuição de linfócitos citotóxicos (LCs) e linfócitos T regulatórios (LTregs) no tecido gengival de pacientes com doença periodontal através de análise imunoistoquímica. Foram selecionados 30 pacientes (10 por grupo) com diagnóstico de periodontite agressiva (PA), periodontite crônica (PC) e gengiva clinicamente saudável (controle); dos quais foi colhida uma amostra de tecido gengival. A distribuição das células (epitélio e córion) foi identificada usando os imunomarcadores CD56, CD57, Granzima B e Perforina (LCs); CD4, CD25, FOXP3 (LTregs). A imunoexpressão foi avaliada, utilizando representação de imagem por meio de um sistema computadorizado, constituído por microscópio de luz, adaptado a uma câmera de alta resolução. Contagens independentes de 10 campos separados para cada caso foram feitas. Two-way ANOVA e posterior teste de Fisher foram utilizados para observar diferenças entre os diagnósticos e os marcadores; e teste t de Student para observar diferenças entre epitélio e córion (p<0.05). Os resultados indicaram que pacientes com PA e PC apresentaram um número significantemente maior de células CD56+ e CD57+, em relação ao grupo controle, porém sem diferenças entre si; um número significantemente maior de células CD56+ e CD57+ foi observado em relação às células Granzima B+ e Perforina+ em todos os pacientes. Em relação aos LTregs, o número de células CD25+ e FOXP3+, foi significativamente diferente entre PA, PC e controle, aparecendo em maior número na PC. Células CD4+ foram observadas em número similar em pacientes com PA e PC, diferindo significantemente do grupo controle; em pacientes com PA e PC, foi observado um número significantemente maior de CD4+, em relação às células CD25+ e FOXP3+. Pacientes com PA e PC tem maior número de LCs no tecido gengival em relação ao grupo controle sugerindo a participação destas células na patogênese da PA e PC. Pacientes com PA apresentaram menor número de LTregs no tecido gengival em comparação aos pacientes com PC, sugerindo que estas células podem estar envolvidas no mecanismo de regulação do processo inflamatório e reabsorção óssea.
This project aims to observe the expression and distribution of cytotoxic lymphocytes (LCs) and regulatory T lymphocytes (LTregs) in gingival tissue from periodontal disease affected patients through immunohistochemical analysis. 30 patients (10 per group) diagnosed with aggressive periodontitis (PA), chronic periodontitis (PC) and clinically healthy gingiva (control) were selected; from which a sample of gingival tissue was collected. The distribution of cells (epithelium and chorion) was identified using the immunomarkers CD56, CD57, Granzyme B, Perforin (LCs); CD4, CD25, FOXP3 (LTregs). The immunoexpression was assessed using image representation by a computer system, comprising a light microscope adapted to a high resolution camera. Independent counts of 10 separate fields for each case were done. Two-way ANOVA and posterior Fisher´s Test were used to observe differences between diagnostics and immunomarkers; and unpaired Student t test to observe differences between epithelium and chorium (p<0.05). The results indicates that patients with PA and PC presented a significantly higher number of CD56+ and CD57+ cells, in relation to control, but without differences between each other; a significantly higher number of CD56+ and CD57+ cells was observed in relation to Granzyme B and Perforine cells in all patients. Related to the LTregs, the number of CD25+ and FOXP3+ cells was significantly different between PA, PC and control, appearing in greater number in PC. CD4+ cells were observed in similar number in patients with PA and PC, it was observed a significantly higher number of CD4+, in relation to CD25+ and FOXP3+ cells. Patients with PA and PC have a greater number of LCs in gingival tissue in relation to control group - suggesting the participation of this cells in the pathogenesis of PA and PC. Patients with PA presented less LTregs in gingival tissue when compared to PC patients, suggesting that this cells may be involved in the regulatory mechanism of the inflammatory process and bone resorption.
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Berenguer, Gaston. "Lamina dura bundle bone or radiographic artifact /." [Gainesville, Fla.] : University of Florida, 2005. http://purl.fcla.edu/fcla/etd/UFE0010577.

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Thesis (M.S.)--University of Florida, 2005.
Typescript. Title from title page of source document. Document formatted into pages; contains 45 pages. Includes Vita. Includes bibliographical references.
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Aaron, Gavin Malcolm. "The prevalence of non-carious cervical lesions in modern and ancient american skulls lack of evidence for an occlusal etiology /." [Gainesville, Fla.] : University of Florida, 2004. http://purl.fcla.edu/fcla/etd/UFE0004872.

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Thesis (M.S.)--University of Florida, 2004.
Typescript. Title from title page of source document. Document formatted into pages; contains 34 pages. Includes Vita. Includes bibliographical references.
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Frey, Christof. "Trend Ölziehen - Evidenz und Risiko der Lipidpneumonie." 2020. https://diu.qucosa.de/id/qucosa%3A70986.

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Ziel dieser Publikation soll es sein, der/m Behandler/in fundiertes Grundwissen bzgl. des Ölziehens zu vermitteln. Darüber hinaus wird die aktuelle Studienlage dargestellt, es werden Trends aufgezeigt und das Risiko der Lipidpneumonie erläutert. Die Autoren kommen zu dem Ergebnis, dass es derzeit geringe Evidenz für einen positiven Nutzen des Ölziehens gibt, sodass weiterführende Studien auf diesem Gebiet notwendig sind. Über das sehr geringe Risiko einer Lipidpneumonie bei nicht korrekter Anwendung sollten die Patientinnen und Patienten aufgeklärt werden.
This publication should provide the practitioner with sound basic knowledge about oil pulling. In addition, the authors present the current study situation, show trends and explain the risk of lipoid pneumonia. The limited evidence to date from clinical trials suggest that oil pulling may have beneficial effects, therefore more studies are necessary to prove beneficial effects on orodental hygiene. Furthermore, patients should be informed about the risk of lipoid pneumonia if used incorrectly.
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Books on the topic "Periodontitis Research"

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Gmür, Rudolf. Value of new serological probes for the study of putative periodontal pathogens: A survey after five years of application. Chicago: Quintessence Pub. Co., 1995.

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Dumitrescu, Alexandrina L. Understanding periodontal research. Heidelberg: Springer, 2012.

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Parker, Philip M., and James N. Parker. Gingivitis: A medical dictionary, bibliography and annotated research guide to Internet references. San Diego, CA: ICON Health Publications, 2004.

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Parker, Philip M., and James N. Parker. Bleeding gums: A medical dictionary, bibliography, and annotated research guide to Internet references. San Diego, CA: ICON Health Publications, 2004.

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Health), Joint Symposium on Clinical Trial Design and Analysis in Periodontics (1996 National Institutes of. Proceedings of the 1996 Joint Symposium on Clinical Trial Design and Analysis in Periodontics. Chicago, Ill. (Suite 800, 737 N. Michigan Ave., Chicago, Ill. 60611): American Academy of Periodontology, 1997.

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Parker, Philip M., and James N. Parker. Gum disease: A medical dictionary, bibliography, and annotated research guide to Internet references. San Diego, CA: ICON Health Publications, 2004.

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Parker, Philip M., and James N. Parker. Peridontal disease: A medical dictionary, bibliography and annotated research guide to Internet references. San Diego, CA: ICON Health Publications, 2004.

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Mistry, Surajit, Debabrata Kundu, and Agathi Spiropoulou. Advanced Research in Periodontitis. DI Press, 2022.

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Santi-Rocca, Julien. Periodontitis: Advances in Experimental Research. Springer International Publishing AG, 2022.

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Newman, Hubert N. Inflammation and Immunology in Chronic Inflammatory Periodontal Disease: Proceedings of the 11th Meeting of the British Inflammation Research Associa. Science Reviews, 1992.

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Book chapters on the topic "Periodontitis Research"

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Hbibi, Abdelhadi, Amal Bouziane, Badiaa Lyoussi, Mimoun Zouhdi, and Driss Benazza. "Aggregatibacter actinomycetemcomitans: From Basic to Advanced Research." In Periodontitis, 45–67. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_3.

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Santi-Rocca, Julien. "Advances in Experimental Research About Periodontitis: Lessons from the Past, Ideas for the Future." In Periodontitis, 1–15. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_1.

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Schaefer, Arne S. "Complementary Experimental Methods in Genetics Open Up New Avenues of Research to Elucidate the Pathogenesis of Periodontitis." In Periodontitis, 209–27. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96881-6_11.

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Dumitrescu, Alexandrina L. "Assessing Confounding and Effect Modification of Periodontitis–Systemic Disease Associations." In Understanding Periodontal Research, 645–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28923-1_20.

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Bascones-Martinez, Antonio, and Jerian González-Febles. "Epigenetics and Periodontitis: A Source of Connection to Systemic Diseases." In Translational Oral Health Research, 19–31. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78205-8_3.

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Xu, Caixia, Xiaokun Yang, Zhipeng Wang, Pengyong Han, Xiaoguang Li, and Zhengwei Li. "Research on the Potential Mechanism of Rhizoma Drynariae in the Treatment of Periodontitis Based on Network Pharmacology." In Intelligent Computing Theories and Application, 207–19. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-13829-4_17.

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N. Alawaji, Yasmine. "Periodontitis, Its Associations, and Prevention." In Dentistry. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.109015.

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The ultimate goal of studying associations with diseases is to plan, implement, and evaluate preventive strategies. Today, after reviewing the body of evidence, one needs to ask: What has not been learned yet regarding periodontitis associations and its prevention? Current recommendations to prevent periodontitis are mostly limited to individual patient care while population-based approaches are nearly absent. Current strategies are not only time-consuming and costly but can be also ineffective to combat disease burden in populations. To initiate and sustain successful outcomes, prevention needs to be applied at multiple levels. Interventions need to target unhealthy behaviors along with their associated social and physical environmental constraints. The chapter presents highlights from current research on associations with periodontitis, its limitations, and the need to understand pathways linking periodontitis with its exposures over the life course. Finally, a suggested multilevel strategy for periodontitis prevention was outlined.
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Jebisha, Dr L., Dr R. Sathish Muthukumar, Dr C. Sreeja, and Dr Merlin Jayaraj. "ROLE OF IMMUNE CELLS IN PERIODONTAL DISEASE." In Emerging Trends in Oral Health Sciences and Dentistry. Technoarete Publishers, 2022. http://dx.doi.org/10.36647/etohsd/2022.01.b1.ch016.

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Periodontitis is intricate & multi-factorial lesion. It is characterized by a group of inflammatory conditions in the periodontium which is caused by bacteria. Chronic inflammation is precise by B and T cells by the initiation in cytokine secretion, then osteoclastogenesis is modulated. The objective of the article briefly reviews the recent research and the consequence in B cell & T lymphocytes subsets, and the expression of cytokine seen in the morbific of the periodontitis. Gingival homeostasis is maintained by T reg (T regulatory), CD8+ T, and tissue-resident γδ, T cells plays a vital role in gingival hemostasis. Clinically healthy periodontium has a low percentage of memory B cells, evade bone damage due to subclinical inflammation. The RANKL -dependent method makes an alveolar bone loss and causes periodontitis. Stimulation of T and B lymphocytes subunits is one that causes either the seditious lesion will alleviate in chronic gingivitis or it may advance to a tissue destructive periodontitis.
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Balagangadharan, Manovijay, and Mythili Swaminathan. "Determination of Relationship between Chronic Periodontitis and Metabolic Syndrome." In Recent Developments in Medicine and Medical Research Vol. 9, 142–47. Book Publisher International (a part of SCIENCEDOMAIN International), 2021. http://dx.doi.org/10.9734/bpi/rdmmr/v9/2849f.

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Roy, Jenny Susan, Sajith Sebastian, Nandini Manjunath, and Joanne Mary Sajith. "Determination of Salivary Glycoconjugates and Salivary Ros Levels in Chronic Periodontitis." In New Horizons in Medicine and Medical Research Vol. 7, 114–49. Book Publisher International (a part of SCIENCEDOMAIN International), 2022. http://dx.doi.org/10.9734/bpi/nhmmr/v7/2653c.

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Conference papers on the topic "Periodontitis Research"

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Sodikova, Shoira. "PERIODONTITIS IN PREGNANT WOMEN." In SCIENTIFIC PRACTICE: MODERN AND CLASSICAL RESEARCH METHODS. European Scientific Platform, 2021. http://dx.doi.org/10.36074/logos-26.02.2021.v3.15.

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2

Yoo, Wook-Sung, Hwapyeong Song, and Hyun-Duck Kim. "Collaborative Research on Rapid Periodontitis Test." In 2022 IEEE 46th Annual Computers, Software, and Applications Conference (COMPSAC). IEEE, 2022. http://dx.doi.org/10.1109/compsac54236.2022.00070.

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3

Gofur, Nanda Rachmad Putra, Nurdiana, Kusworini Handono, and Handono Kalim. "Immune aging marker associated with periodontitis in systemic lupus erythematosus patients." In INTERNATIONAL CONFERENCE ON BIOINFORMATICS AND NANO-MEDICINE FROM NATURAL RESOURCES FOR BIOMEDICAL RESEARCH: 3rd Annual Scientific Meeting for Biomedical Sciences. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5109978.

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4

Pina, C., S. Cunha, R. Silva, J. C. Sousa, and I. Lopes Cardoso. "Antimicrobial susceptibilities of Porphyromonas and Prevotella species isolated from periodontitis infections in the north of Portugal." In MICROBES IN APPLIED RESEARCH - Current Advances and Challenges. WORLD SCIENTIFIC, 2012. http://dx.doi.org/10.1142/9789814405041_0099.

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5

Sherina, Natalia, Khaled Amara, Natalie Sippl, Daniel RamsköLd, Johanna Steen, Lena Israelsson, Monika Hansson, et al. "08.41 cloning of gingival tissue b cells from an acpa+ ra patient with periodontitis." In 37th European Workshop for Rheumatology Research 2–4 March 2017 Athens, Greece. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2016-211055.41.

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6

Gawron, Katarzyna, Grzegorz Bereta, Zuzanna Nowakowska, Katarzyna Łazarz-Bartyzel, Anna B. Montgomery, Magdalena Marczuk, Anna Kowalska, et al. "07.14 Novel polymorphism of peptidylarginine deiminase from p. gingivalis augments bacterial pathogenicity and severity of periodontitis." In 37th European Workshop for Rheumatology Research 2–4 March 2017 Athens, Greece. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2016-211054.14.

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7

Sayed, Ahmed, Ahmed Mahmoud, Eros Chaves, Richard Crout, Kevin Sivaneri, and Osama Mukdadi. "Assessment of Gingival Inflammation Using Ultrasound Imaging." In ASME 2012 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/imece2012-89627.

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Abstract:
Gingivitis is the most common gingival inflammation in the oral cavity, and the most prevalent periodontal disease affecting 90% of the population in all age groups. Recently, a few research groups have investigated the possibility of using ultrasound in dentistry, particularly in diagnosing bony destruction in the more severe form of periodontal disease called periodontitis. This work investigates the feasibility of using ultrasound imaging to quantitatively assess gingival tissue inflammation. Signal and image processing of ultrasound data have been performed to quantitatively assess gingival tissue. A number of gingival scans were conducted in vitro to render ultrasound images of high-spatial and contrast resolutions. For each sample the B-mode images were matched with almost the same slices in histology. Results show that ultrasound scans for tissues with gingivitis exhibited low intensity of reflections (hypo echoic) at the inflamed tissues, while healthy dense epithelium layers exhibited higher reflections (hyper echoic). Histological diagnosis revealed good agreement with the ultrasound results indicating the usefulness of such ultrasound imaging in diagnosing gingivitis. In addition, a new design for an intraoral linear array ultrasound probe is demonstrated and utilized in our clinic in vivo. Analysis of the echogenicity patterns of the resultant images demonstrates the potential of using such a new probe in gingival health assessment, which would be feasible and clinical relevant for patient evaluations clinically.
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8

Lestari, Citra, Eryati Darwin, Deddi Prima Putra, and Netti Suharti. "The Effect of α-Mangosteen on Runt-Related Transcription Factor 2 and Tartrate-Resistant Acid Phosphatase 5b Expressions on Bone Remodeling in Periodontitis (An Experimental Research on Wistar Rats)." In 1st International Conference on Health Sciences and Biotechnology (ICHB 2021). Paris, France: Atlantis Press, 2022. http://dx.doi.org/10.2991/ahsr.k.220303.007.

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9

Courbon, G., M. Rinaudo-Gaujous, V. Blasco-Baque, I. Auger, R. Caire, L. Mijola, L. Vico, S. Paul, and H. Marotte. "P164 Porphyromonas gingivalis experimentally induces periodontis and an anti-CCP2-associated arthritis in the rat." In 39th European Workshop for Rheumatology Research, 28 February–2 March 2019, Lyon, France. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2019.146.

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