Relevant bibliographies by topics / Periodontal cells

Academic literature on the topic 'Periodontal cells'

Author: Grafiati
Published: 15 June 2024

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Periodontal cells.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Journal articles on the topic "Periodontal cells":

1

Nokhbehsaim, Marjan, Anna Damanaki, Andressa Vilas Boas Nogueira, Sigrun Eick, Svenja Memmert, Xiaoyan Zhou, Shanika Nanayakkara, et al. "Regulation of Ghrelin Receptor by Periodontal Bacteria In Vitro and In Vivo." Mediators of Inflammation 2017 (2017): 1–11. http://dx.doi.org/10.1155/2017/4916971.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Ghrelin plays a major role in obesity-related diseases which have been shown to be associated with periodontitis. This study sought to analyze the expression of the functional receptor for ghrelin (GHS-R1a) in periodontal cells and tissues under microbial conditions in vitro and in vivo. The GHS-R1a expression in human periodontal cells challenged with the periodontopathogen Fusobacterium nucleatum, in gingival biopsies from periodontally healthy and diseased individuals, and from rats with and without ligature-induced periodontitis was analyzed by real-time PCR, immunocytochemistry, and immunofluorescence. F. nucleatum induced an initial upregulation and subsequent downregulation of GHS-R1a in periodontal cells. In rat experimental periodontitis, the GHS-R1a expression at periodontitis sites was increased during the early stage of periodontitis, but significantly reduced afterwards, when compared with healthy sites. In human gingival biopsies, periodontally diseased sites showed a significantly lower GHS-R1a expression than the healthy sites. The expression of the functional ghrelin receptor in periodontal cells and tissues is modulated by periodontal bacteria. Due to the downregulation of the functional ghrelin receptor by long-term exposure to periodontal bacteria, the anti-inflammatory actions of ghrelin may be diminished in chronic periodontal infections, which could lead to an enhanced periodontal inflammation and tissue destruction.
2

Mancini, Leonardo, Adriano Fratini, and Enrico Marchetti. "Periodontal Regeneration." Encyclopedia 1, no. 1 (January 13, 2021): 87–98. http://dx.doi.org/10.3390/encyclopedia1010011.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Periodontal regeneration is a technique that aims to regenerate the damaged tissue around periodontally compromised teeth. The regenerative process aims to use scaffolds, cells, and growth factors to enhance biological activity.
3

Nighojkar, Urvashi Rajeev, Dr Priya A. Lele, and Mudita Agrawal. "Scope of Stem Cells in Periodontal Regeneration." International Journal of Scientific Research 2, no. 5 (June 1, 2012): 428–31. http://dx.doi.org/10.15373/22778179/may2013/145.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Karunanithi Arulvizhi M, Arunagiri. "Stem Cells in Periodontal Regenerations - A Review." International Journal of Science and Research (IJSR) 13, no. 2 (February 5, 2024): 589–94. http://dx.doi.org/10.21275/sr24206105401.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Gonçalves, Gabriela Sumie Yaguinuma, Tayna Natsumi Takakura, Anderson Catelan, Rosalinda Tanuri Zaninotto Venturim, Carolina dos Santos Santinoni, and Christine Men Martins. "Tratar ou extrair? Tratamento de lesão endoperiodontal, um relato de caso clínico." ARCHIVES OF HEALTH INVESTIGATION 9, no. 6 (April 20, 2020): 535–40. http://dx.doi.org/10.21270/archi.v9i6.4814.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Introdução: Lesões endoperiodontais são lesões originadas de produtos inflamatórios encontrados tanto em periodonto quanto em polpa. Tais lesões podem se originar devido a uma infecção pulpar ou periodontal. Visando o prognóstico favorável, é imprescindível o conhecimento da etiologia, realização do correto diagnóstico e elaboração do plano de tratamento que envolve o tratamento endodôntico precedido do tratamento periodontal. Objetivo: O propósito do presente trabalho foi de relatar um caso clínico de lesão endoperiodontal e o tratamento realizado. Relato de caso clínico: Paciente gênero feminino, 51 anos, compareceu à clínica com uma fístula na região do dente 46, procedeu-se com exame radiográfico, rastreamento de fístula, testes endodônticos e avaliação periodontal. Foi diagnosticada lesão endoperiodontal. Executou-se, então, o tratamento endodôntico em sessões múltiplas, utilizando hidróxido de cálcio como medicação intracanal e o tratamento periodontal concomitante; finalizou-se endodontia obturando-se os canais radiculares. Conclusão: Observou-se, no controle, que a associação de tratamentos foi eficaz e houve melhora significativa do quadro, constatando-se silêncio clínico e sucesso do tratamento. Realizar o tratamento conservador a despeito da exodontia foi a melhor escolha para a paciente. Descritores: Endodontia; Periodontia; Polpa Dentária; Periodonto. Referências Sunitha VR, Emmadi P, Namasivayam A, Thyegarajan R, Rajaraman V. The periodontal - endodontic continuum A review. J Conserv Dent. 2008;11(2):54-62. Betancourt P, Elgueta R, Fuentes R. Treatment of endo-periodontal lesion using leukocyte-platelet-rich fibrin - a case report. Colomb Med. 2017;48(4):204-7. Lopes HP, Siqueira JF. Endodontia: Biologia e Técnica. Rio de Janeiro: Medsi-Guanabara Koogan; 2015. Lindhe J, Karring T, Lang NP. Tratado de periodontia clínica e implantologia oral. Rio de Janeiro: Guanabara Koogan; 2010. Anand V, Govila V, Gulati M. Endo-perio lesion part II (the treatment) - a review. 2012;3(1):10-6. Rotstein I, Simon JH. Diagnosis, prognosis and decision-making in the treatment of combined periodontal-endodontic lesions. J Periodontol. 2004;34:165-203. Parolia A, Gait TC, Porto ICCM, Mala K. Endo-perio lesion: a dilemma from 19th until 21st century. J Interdisp Dent. 2013;3(1):2-11. Kim E, Song JS, Jung IY, Lee SJ, Kim S. Prospective clinical study evaluating endodontic microsurgery outcomes for cases with lesions of endodontic origin compared with cases with lesions of combined periodontal-endodontic origin. J Endod. 2008;34(5):546-51. Heasman PA. An endodontic conundrum: the association between pulpal infection and periodontal disease. Br Dent J. 2014;216(6):275-9. Schmidt JC, Walter C, Amato M, Weiger R. Treatment of periodontal-endodontic lesions--a systematic review. J Clin Periodontol. 2014; 41(8):779-90. Jivoinovici R, Suciu I, Dimitriu B, Perlea P, Bartok R, Malita M, Ionescu C. Endo-periodontal lesion--endodontic approach. J Med Life. 2014;7(4):542-44. Estrela C. Endodontia laboratorial e clínica, Série Abeno: Odontologia Essencial - Parte Clínica. São Paulo: Artes Médicas; 2013. Vera J, Siqueira JF Jr, Ricucci D, Loghin S, Fernández N, Flores B et al. One-versus two-visit endodontic treatment of teeth with apical periodontitis: a histobacteriologic study. J Endod. 2012;38(8):1040-52. Mohammadi Z, Dummer PMH. Properties and applications of calcium hydroxide in endodontics and dental traumatology. Inter Endod J. 2011;44(8):697-730. Batista VES, Olian DA, Mori GG. Diffusion of hydroxyl ions from calcium hydroxide and aloe vera pastes. Braz Dent J. 2014;25(3):212-16. Pereira TC, da Silva Munhoz Vasconcelos LR, Graeff MSZ, Ribeiro MCM, Duarte MAH, de Andrade FB. Intratubular decontamination ability and physicochemical properties of calcium hydroxidepastes. Clin Oral Investig. 2019;23(3):1253-62. Andolfatto C, da Silva GF, Cornélio AL, Guerreiro-Tanomaru JM, Tanomaru-Filho M, Faria G, Bonetti-Filho I, Cerri PS. Biocompatibility of intracanal medications based on calcium hydroxide. ISRN Dent. 2012;2012:904963. Duque TM, Prado M, Herrera DR, Gomes BPFA. Periodontal and endodontic infectious/inflammatory profile in primary periodontal lesions with secondary endodontic involvement after a calcium hydroxide-based intracanal medication. Clin Oral Investig. 2019;23(1):53-63. Kim D, Kim E. Antimicrobial effect of calcium hydroxide as an intracanal medicament in root canal treatment: a literature review - Part I. In vitro studies. Restor Dent Endod. 2014; 39(4):241-52. Adl A, Motamedifar M, Shams MS, Mirzaie A. Clinical investigation of the effect of calcium hydroxide intracanal dressing on bacterial lipopolysaccharide reduction from infected root canals. Aust Endod J. 2015;41(1):12-6. Hilton TJ, Ferracane JL, Mancl L; Northwest Practice-based Research Collaborative in Evidence-based Dentistry (NWP). Comparison of CaOH with MTA for direct pulp capping: a PBRN randomized clinical trial. J Dent Res. 2013;92(7 Suppl):16S-22S. Labban N, Yassen GH, Windsor LJ, Platt JA. The direct cytotoxic effects of medicaments used in endodontic regeneration on human dental pulp cells. Dent Traumatol. 2014;30(6):429-34. McIntyre PW, Wu JL, Kolte R, Zhang R, Gregory RL, Bruzzaniti A, Yassen GH. The antimicrobial properties, cytotoxicity, and differentiation potential of double antibiotic intracanal medicaments loaded into hydrogel system. Clin Oral Investig. 2019;23(3):1051-59. Bergenholtz, G., Hasselgren, G. Endodontics and periodontics. In: Lindhe, K., Karring, T., Lang, N. Clinical periodontology and implant dentistry. Copenhagen:Munksgaard; 2015. Harrington GW, Steiner DR, Ammons WF. The periodontal-endodontic controversy. Periodontol 2000. 2002;30:123-30. Fernandes LA, Martins TM, Almeida JM, Nagata MJ, Theodoro LH, Garcia VG, Bosco AF. Experimental periodontal disease treatment by subgingival irrigation with tetracycline hydrochloride in rats. J Appl Oral Sci. 2010;18(6):635-40. Storrer CM, Bordin GM, Pereira TT. How to diagnose and treat periodontal endodontic lesions? 2012;9(4):427-33. Verma PK, Srivastava R, Gupta KK, Srivastava A. Combined endodontic periodontal lesions: A clinical dilema. J Interdiscip Dent. 2011;1(2):119-24. Oh SL, Fouad AF, Park SH. Treatment strategy for guided tissue regeneration in combined endodontic-periodontal lesions: case report and review. J Endod. 2009;35(10):1331-36. Malli R, Lele P, Vishakha. Guided tissue regeneration in communicating periodontal and endodontic lesions - a hope for the hopeless. J Indian Soc Periodontol. 2011;15(4):410-13. Ghezzi C, Virzì M, Schupbach P, Broccaioli A, Simion M. Treatment of combined endodontic-periodontic lesions using guided tissue regeneration: clinical case and histology. Int J Periodontics Restorative Dent. 2012;32(4):433-9. Sun J, Liu Q. [Bio-Oss collagen bone grafting in the treatment of endodontic-periodontic lesion]. Nan Fang Yi Ke Da Xue Xue Bao. 2009;29(9):1905-6. Sharma R, Hegde V, Siddharth M, Hegde R, Manchanda G, Agarwal P. Endodontic-periodontal microsurgery for combined endodontic-periodontal lesions: An overview. J Conserv Dent. 2014;17(6):510-16. Li Y, Wang X, Xu J, Zhou X, Xie K. [The clinical study on the use of diode laser irradiation in the treatment of periodontal-endodontic combined lesions]. Hua Xi Kou Qiang Yi Xue Za Zhi. 2012;30(2):161-64, 168. Narang S, Narang A, Gupta R. A sequential approach in treatment of perio-endo lesion. J Indian Soc Periodontol. 2011;15(2):177-80. Pereira AL, Orzechowski PR, Filho SB, Cortelli JR. Subepithelial connective tissue graft: an alternative application for treating endoperiodontal lesions. Gen Dent. 2013;61(2):50-3. Yoneda M, Motooka N, Naito T, Maeda K, Hirofuji T. Resolution of furcation bone loss after non-surgical root canal treatment: application of a peptidase-detection kit for treatment of type I endoperiodontal lesion. J Oral Sci. 2005; 47(3):143-47. Shenoy N, Shenoy A. Endo-perio lesions: diagnosis and clinical considerations. Indian J Dent Res. 2010;21(4):579-85. Gerritsen AE, Allen PF, Witter DJ, Bronkhorst EM, Creugers NH. Tooth loss and oral health-related quality of life: a systematic review and meta-analysis. Health Qual Life Outcomes. 2010;8:126.
6

Fabila-Plata, Miriam Susana, and Rosa Diana Hernández Palacios. "Tratamiento de la enfermedad periodontal con células troncales de origen pulpar, en un adulto mayor. Caso clínico." Casos y Revisiones de Salud 3, no. 1 (July 1, 2021): 32–39. http://dx.doi.org/10.22201/fesz.26831422e.2021.3.1.4.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Introducción. La enfermedad periodontal (EP) es un padecimiento de tipo inflamatorio crónico y con frecuencia causa pérdida dental en los adultos mayores. Su tratamiento se orienta a la regeneración de los tejidos periodontales a través de la eliminación de los agentes infecciosos presentes en el periodonto y en la sustitución del hueso alveolar perdido. Caso clínico. Paciente femenina de 61 años de edad que acudió a consulta estomatológica por presentar movilidad dental. En la valoración bucal se observó enfermedad periodontal. Se realizaron las tres fases de la terapia periodontal con aplicación de células troncales mesenquimales (Mesenchymal Stem Cells, MSC) de origen pulpar, mostrando disminución de la profundidad al sondaje y aumento en el nivel de inserción clínica. Conclusiones. Los hallazgos sugieren que la terapia periodontal regenerativa con MSC de origen pulpar representan una opción para el tratamiento de los defectos óseos ocasionados por la EP en los adultos mayores; sin embargo, es necesario llevar a cabo ensayos clínicos controlados y aumentar el tamaño de la muestra para recomendar su aplicación en la práctica clínica.
7

Song, In Seok, Yoon Sic Han, Joo-Hee Lee, Soyoun Um, Hui Young Kim, and Byoung Moo Seo. "Periodontal Ligament Stem Cells for Periodontal Regeneration." Current Oral Health Reports 2, no. 4 (August 27, 2015): 236–44. http://dx.doi.org/10.1007/s40496-015-0060-0.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Aditya, Vangara, and Kharidhi Laxman Vandana. "Management of endo-perio lesion with autologous stem cell therapy." Saudi Journal of Oral Sciences 11, no. 1 (January 2024): 54–59. http://dx.doi.org/10.4103/sjoralsci.sjoralsci_79_23.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
The objective of periodontal therapy is the regeneration of tooth-supporting tissues. Various treatment modalities, such as the use of bone grafting materials, guided tissue regeneration, and delivery of enamel matrix derivatives or growth factors, are applied with large variability in regenerative outcomes. However, a case report was done by utilization of autologous dental pulp stem cells and periodontal ligament stem cell niches in the treatment of bone loss associated with endodontically and periodontally involved teeth. An autologous periodontal ligament stem cells niche adherent to the root and dental pulpal stem cells from dental pulp directly into the selected osseous defect following extraction of the impacted tooth in the same patient. The results of this case reported that the dental pulpal stem cells and periodontal ligament stem cells niche with gelatin sponge resulted in a significant amount of bone fill and reduction in probing pocket depth.
9

Hosokawa, Yoshitaka, Ikuko Hosokawa, Satoru Shindo, Kazumi Ozaki, and Takashi Matsuo. "IL-4 Modulates CCL11 and CCL20 Productions from IL-1β-Stimulated Human Periodontal Ligament Cells." Cellular Physiology and Biochemistry 38, no. 1 (2016): 153–59. http://dx.doi.org/10.1159/000438617.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Background/Aims: IL-4 is a multifunctional cytokine that is related with the pathological conditions of periodontal disease. However, it is uncertain whether IL-4 could control T cells migration in periodontal lesions. The aim of this study was to examine the effects of IL-4 on CCL11, which is a Th2-type chemokine, and CCL20, which is related with Th17 cells migration, productions from human periodontal ligament cells (HPDLCs). Methods: CCL20 and CCL11 productions from HPDLCs were monitored by ELISA. Western blot analysis was performed to detect phosphorylations of signal transduction molecules in HPDLCs. Results: IL-1β could induce both CCL11 and CCL20 productions in HPDLCs. IL-4 enhanced CCL11 productions from IL-1β-stimulated HPDLCs, though IL-4 inhibited CCL20 production. Western blot analysis showed that protein kinase B (Akt) and signal transducer and activator of transcription (STAT)6 pathways were highly activated in IL-4/IL-1β-stimulated HPDLCs. Akt and STAT6 inhibitors decreased CCL11 production, but enhanced CCL20 production in HPDLCs stimulated with IL-4 and IL-1β. Conclusions: These results mean that IL-4 enhanced Th2 cells migration in periodontal lesion to induce CCL11 production from HPDLCs. On the other hand, IL-4 inhibits Th17 cells accumulation in periodontally diseased tissues to inhibit CCL20 production. Therefore, IL-4 is positively related with the pathogenesis of periodontal disease to control chemokine productions in periodontal lesions.
10

Kramer, P. R., S. Nares, S. F. Kramer, D. Grogan, and M. Kaiser. "Mesenchymal Stem Cells Acquire Characteristics of Cells in the Periodontal Ligament in vitro." Journal of Dental Research 83, no. 1 (January 2004): 27–34. http://dx.doi.org/10.1177/154405910408300106.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Mesenchymal stem cells differentiate into multiple types of cells derived from mesenchyme. Periodontal ligament cells are primarily derived from mesenchyme; thus, we expected mesenchymal stem cells to differentiate into periodontal ligament. Using a combination of immunohistochemistry and in situ hybridization on co-cultures of mesenchymal stem cells and periodontal ligament, we observed a significant increase in mesenchymal stem cells’ expression of osteocalcin and osteopontin and a significant decrease in expression of bone sialoprotein, characteristics of periodontal ligament in vivo. Increased osteopontin and osteocalcin and decreased bone sialoprotein expression was detected within 7 days and maintained through 21 days of co-culture. We conclude that contact or factors from periodontal ligament induced mesenchymal stem cells to obtain periodontal-ligament-like characteristics. Importantly, analysis of the data suggests the feasibility of utilizing mesenchymal stem cells in clinical applications for repairing and/or regenerating periodontal tissue.
More sources
You might also be interested in the extended bibliographies on the topic 'Periodontal cells' for particular source types:
Journal articles Dissertations / Theses

Dissertations / Theses on the topic "Periodontal cells":

1

Worapamorn, Wilairat. "Cell-surface proteoglycan expression by periodontal cells /." St. Lucia, Qld, 2001. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe16097.pdf.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Gay, Isabel C. "Isolation and characterization of human periodontal ligament stem cells." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2007. http://www.mhsl.uab.edu/dt/2007m/gay.pdf.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Stoianovici, Charles. "Directing Mesenchymal Stem Cells for Periodontal Regeneration." VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5335.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Background: Directing autogenous Mesenchymal Stem Cell (MSC) to defect sites has a great promise in bone regeneration. We designed a MSC specific, bone affinity peptide (E7HA7) by conjugating E7 with a polyglutamate hydroxyapatite (HA) binding motif. We sought to characterize the in-vivo releasing pattern and bioactivity of E7HA7. Methods: HA discs were coated with fluorescent labeled peptides E7HA7, E7HA2 or E7 were subcutaneously implanted in Sprague Dawley rats. In an ectopic bone formation model was used to test the in-vivo bioactivity of E7HA7 conjugated to DBM. Results: E7HA7 showed slower peptide release from scaffolds in comparison to other groups, being statistically significant at week 2 compared to E7, and to E7HA2 at week 4 and 8. In ectopic model, the medians for new bone formation in each group were: iDBM=0.041mm3, iDBM-E7=0.071mm3, aDBM=0.138mm3, and aDBM-E7=0.192mm3. Conclusions: Conjugation of E7 to polyglutamate bone binding domain showed slow releasing kinetics and osteoinductive potential.
4

Winning, Lewis. "The osteogenic potential of periodontal ligament stem cells." Thesis, Queen's University Belfast, 2018. https://pure.qub.ac.uk/portal/en/theses/the-osteogenic-potential-of-periodontal-ligament-stem-cells(e5fdef0e-d55b-42b6-acb5-75a617b43edd).html.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Campbell, Lauren Dee. "The role of CD4+ T cells in periodontal disease." Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8241/.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Introduction: Periodontal disease (PD) is the most common bone destructive chronic inflammatory disease in humans. Severe PD affects 8-15% of the population and impacts on the ability to chew and appearance, reduces quality of life, and is responsible for a substantial proportion of dental care costs. A dysbiotic oral biofilm is necessary but insufficient for development of PD. Rather, a dysregulated immune response to the disease-associated biofilm results in destruction of tooth supporting structures and eventual tooth loss. Despite the apparent involvement of the immune system in PD, clinical management focuses solely on the mechanical removal of the oral biofilm – with partial success and frequent recurrence. Therefore, a better understanding of the immune response in PD could highlight potential novel preventative and therapeutic strategies. T cells are present at sites of PD; however, there remains ambiguity regarding whether these T cells are protective or destructive in PD. The aim of these studies was to characterize CD4+ T cells in a P. gingivalis-induced murine model of PD. Results: P. gingivalis-infected mice displayed subtle changes in their CD4+ T cell compartment, predominantly in the draining lymph nodes (dLNs). Such changes included a suggested increase in T follicular helper cells, a trend towards a decrease in regulatory T cells and a trend towards increased production of IFN-γ. Elevated levels of IFN-γ were also noted in gingival CD8+ T cells and splenocytes, with similar trends in CD8+ T cells from dLNs. The transcriptome of CD4+ T cells isolated from gingivae and dLNs of P. gingivalis–infected suggested minimal changes in gene expression following infection; however, identified a profile of the mucosal oral CD4+ T cell compared with CD4+ T cells of the dLN. To investigate the response of CD4+ T cells specific for P. gingivalis, the bacteria were genetically manipulated to express ovalubumin (OVA) peptide 323-339. However, these OVA peptide expressing P. gingivalis failed to induce a response in OVA-specific T cells, both in vitro and in vivo. Conclusion: These data imply that CD4+ T cells do not substantially change upon P. gingivalis infection in a murine model. IFN-γ production, however, was elevated both locally and systemically. Together, the data presented in this thesis and data previously published warrant further investigations into the role of IFN-γ in PD and may point to IFN-γ as a biomarker or biological target for adjunctive PD therapy.
6

Åsman, Björn. "Juvenile periodontitis generation of free oxygen radicals and elastase by peripheral PMN cells /." Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1988. http://catalog.hathitrust.org/api/volumes/oclc/18171198.html.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Engman, Sara. "Expression and Regulation of the Cell Surface Proteins CD47 and SIRPα in Resident Periodontal Cells." Thesis, Umeå universitet, Institutionen för odontologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-129258.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Periodontal disease is an inflammatory disorder affecting the supporting tissues of the tooth. The inflammation triggers a destruction of the connective- and bone tissue surrounding the tooth, a process that is not fully elucidated. It is known that periodontitis shares features with other inflammatory disease like Crohn's disease and rheumatoid arthritis. The cell surface proteins signal regulatory protein alpha (SIRPα) and cluster of differentiation 47 (CD47) are important for the progression of the inflammation in rheumatoid arthritis and Crohn's disease. It has also been shown that lack of SIRPα or CD47 render in reduced number of osteoclast. The aim of this study was to investigate if cells from the periodontium (human gingival fibroblasts) from periodontally healthy individuals express SIRPα and CD47 and if the expression of these membrane proteins is regulated under inflammatory conditions.   We demonstrate, by using quantitative rt-qPCR, that human gingival fibroblasts express both CD47 and SIRPα mRNA. The expression of SIRPα was positively regulated (2-fold) by the pro-inflammatory cytokines tumor necrosis factor alpha (TNF-α) and interleukin-1-beta (IL-1β). TNF-α caused a 2-fold up-regulation of CD47 in human gingival fibroblasts. Neither CD47 nor SIRPα were time-dependently regulated by the two pro-inflammatory cytokines.   We here conclude that SIRPα and CD47 gene expression are up-regulated in human gingival fibroblasts cultured under inflammatory conditions. These findings indicate that SIRPα and CD47 may play a role in periodontal disease.
8

Bou, Chebel Najib. "Periodontal bacterial-DNA initiated immuno-inflammatory responses in human osteoblastic cells." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/97.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Periodontitis is a chronic inflammatory disease initiated by gram negative anaerobic bacteria. These bacteria possess pathogen-associated molecular patterns (PAMPs) that interact with various receptors including Toll like receptors (TLRs). Bacterial DNA (bDNA) is one of the PAMPs mainly recognized by TLR9. Interaction of bDNA and its receptors leads to activation of inflammatory signaling pathways potentially resulting in periodontal bone destruction. The aim of this study was to determine the production of IL- 6 and IL-8 in response to periodontal bDNA from human osteoblastic cells (MG-63). MG- 63 cells were stimulated in duplicate for 20 hours with 100ng/μl of bDNA from various pathogens including Porhyromonas gingivalis, Esherichia coli, Streptococcus sanguinis, Aggregatibacter actinomycetemcomitans as well as heat killed whole bacteria (1:100). E.coli LPS (10ng/μl) was used as a positive control in each experiment. To block TLR9 signaling, further experiments were carried out by treating MG-63 cells with chloroquine (10ng/μl) for 2 hours at 37ºC prior to stimulations. Cytokine levels were determined using enzyme linked-immunosorbent assay. Although IL-6 and IL-8 production was increased in response to periodontal bDNA in MG-63 cells, the results were not significant compared to unstimulated controls. As expected, E.coli DNA, E.coli LPS and heat killed whole bacteria stimulated significantly increased cytokine production (p<0.05). Blocking TLR9 with chloroquine did not affect the amount of cytokine production in bDNA stimulated cells suggesting that TLR9 may not be operant in triggering IL-6 and IL-8 production from MG- 63 cells. In conlusion, periodontal bDNA did not trigger significantly increased IL-6 and IL-8 production from MG-63 cells. Considering the involvement of several inflammatory mediators in periodontal bone destruction, further studies are warranted to assess the production of other cytokines in response to periodontal bDNA in human osteoblastic cells.
9

Moore, Edward Andrew. "Cell attachment and spreading on physical barriers used in periodontal guided tissue regeneration /." Oklahoma City : [s.n.], 2002. http://library.ouhsc.edu/epub/theses/Moore-William-A.pdf.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Wescott, David Clark, and n/a. "Osteogenic gene expression by human periodontal ligament cells under cyclic mechanical tension." University of Otago. School of Dentistry, 2008. http://adt.otago.ac.nz./public/adt-NZDU20081202.131453.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Background and objectives: The most widely accepted tooth movement model is defined by the pressure-tension hypothesis. An orthodontic force applied to a tooth generates areas of compression and tension in the periodontal ligament (PDL), which are transmitted to the alveolar bone. Areas of tissue exposed to tensile strain undergo bone deposition, whereas areas of tissue exposed to compressive strain undergo bone resorption. We propose that human PDL cells in monolayer culture exposed to tensile mechanical strain would express multiple genes involved in osteogenesis. Materials and Methods: Human PDL cells were isolated and cultured from premolar teeth that were extracted for orthodontic reasons. These cells were plated on control and experimental Uniflex[TM] plates. Using a Flexercell FX4000 strain unit, PDL cells on experimental plates were exposed to a 12% uni-axial cyclic strain for 6 seconds out of every 90 seconds over a 24 hour period. RNA was extracted from the PDL cells at 6 hours, 12 hours and 24 hours. The differential expression of 78 genes implicated in osteoblast differentiation and bone metabolism was analysed using real-time reverse transcriptase polymerase chain reaction (RT-PCR) array technology. Results: Of the 78 genes tested, sixteen genes showed statistically significant (p<0.05) changes in expression in response to the mechanical strain regime. Eight genes were up-regulated (ALPL, BMP2, BMP6, COL2A1, ICAM1, PHEX, SOX9, and VEGFA) and eight genes were down-regulated (ANXA5, BMP4, COL11A1, COL3A1, EGF, ITGB1, MSX and SMAD1). Conclusions: This study has demonstrated that cultured human PDL cells express multiple osteogenic genes under tensile strain, which suggests that PDL cells may have a potential role in osseous remodeling during tooth movement. Key Words: Tooth movement, human PDL cells, tensile mechanical strain, osteogenic genes, real-time RT-PCR array, and Flexercell FX4000.
More sources

Books on the topic "Periodontal cells":

1

sman, Bjo rn A. Juvenile periodontitis: Generation of free oxygen radicals and elastase by peripheral PMN cells. Stockholm: Kongl. Carolinska Medico Chirurgiska Institutet, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Lin, Deborah G. Storage conditions of avulsed teeth affect the phenotype of cultured human periodontal ligament cells. [Toronto: Faculty of Dentistry, University of Toronto, 1999.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Larjava, Hannu. Oral wound healing: Cell biology and clinical management. Chichester, West Sussex: John Wiley & Sons, 2012.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Fardal, Øystein. Initial attachment of fibroblast-like cells to periodontally diseased root surfaces in vitro. [Toronto]: Faculty of Dentistry, University of Toronto, 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Soory, Mena. Concepts of Periodontal Regeneration and Regenerative Medicine: Mechanisms That Modulate Cells and Matrices. Nova Science Publishers, Incorporated, 2015.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

Devlin, Hugh, and Rebecca Craven. Bone. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759782.003.0004.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Bone in relation to dentistry is the topic of this chapter. This chapter describes the mineral, cells, vascular and matrix components of bone. Throughout the chapter, the clinical relevance of these features and how they interact in health and disease are emphasized. The later parts of the chapter describe bone healing, bone grafts, healing of the extraction socket, orthodontic tooth movement, periodontal bone loss in chronic periodontitis, and the effect of bisphosphonates. A final section summarizes age changes in bone and bone cells.
7

Devlin, Hugh, and Rebecca Craven. Diabetes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759782.003.0007.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Diabetes in relation to dentistry is the topic of this chapter. The incidence of diabetes is increasingly rapidly, hand-in-hand with the increase in obesity. Obesity predisposes patients to an increased insulin resistance, i.e. reduces their ability to increase the glucose transport into adipocytes, muscle, and liver cells. The pancreas responds by producing more insulin but when it can no longer produce enough to overcome the insulin resistance, the blood glucose rises. Diabetes is characterized by raised blood glucose. We describe the devastating long-term effects of diabetes, in particular the microvascular and macrovascular medical complications. The dental complications include an increased severity of periodontal disease, oral candidiasis, and dry mouth but in those who are poorly controlled the impaired defence mechanisms can lead to severe head and neck infections and osteomyelitis. A final summary lists the important clinical recommendations for treatment of diabetic patients.
8

Larjava, Hannu. Oral Wound Healing: Cell Biology and Clinical Management. Wiley & Sons, Incorporated, John, 2012.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

Larjava, Hannu. Oral Wound Healing: Cell Biology and Clinical Management. Wiley & Sons, Limited, John, 2013.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
10

Larjava, Hannu. Oral Wound Healing: Cell Biology and Clinical Management. Wiley & Sons, Incorporated, John, 2012.

Find full text
APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Periodontal cells":

1

Takeuchi, Hiroki, and Atsuo Amano. "Invasion of Gingival Epithelial Cells by Porphyromonas gingivalis." In Periodontal Pathogens, 215–24. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_21.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Matsushita, Kenji. "Analysis of Interaction Between Porphyromonas gingivalis and Endothelial Cells In Vitro." In Periodontal Pathogens, 225–33. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_22.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Hasebe, Akira, Ayumi Saeki, and Ken-ichiro Shibata. "Lipoprotein Extraction from Microbial Membrane and Lipoprotein/Lipopeptide Transfection into Mammalian Cells." In Periodontal Pathogens, 195–204. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_19.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Imai, Kenichi. "Analysis of the Interaction Between HIV and Periodontopathic Bacteria That Reactivates HIV Replication in Latently Infected Cells." In Periodontal Pathogens, 207–14. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0939-2_20.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Pagni, Giorgio. "Stem Cells for Periodontal Regeneration." In Dental Stem Cells: Regenerative Potential, 165–86. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33299-4_9.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Kao, Richard T., and Mark C. Fagan. "Regeneration of Intrabony Defects Utilizing Stem Cells Allograft." In Advances in Periodontal Surgery, 101–15. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-12310-9_7.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Tobita, Morikuni, and Hiroshi Mizuno. "Adipose-Derived Stem Cells for Periodontal Tissue Regeneration." In Adipose-Derived Stem Cells, 461–70. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-61737-960-4_34.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Wang, Songlin, Gang Ding, Fulan Wei, and Yi Liu. "Bioengineering of Roots and Periodontal Tissues." In Stem Cells in Craniofacial Development and Regeneration, 485–99. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118498026.ch27.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Calenic, Bogdan, and Ken Yaegaki. "DNA Damage Caused by Oral Malodorous Compounds in Periodontal Cells In Vitro: Novel Carcinogenic Pathway." In Studies on Periodontal Disease, 77–84. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9557-4_5.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Maeda, Hidefumi, Shinsuke Fujii, Satoshi Monnouchi, Naohisa Wada, and Akifumi Akamine. "Differentiation of Periodontal Ligament Stem/Progenitor Cells: Roles of TGF-β1." In Stem Cells and Cancer Stem Cells, Volume 4, 51–58. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-2828-8_5.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Periodontal cells":

1

Gao, Zhen, and Xiaoting Luo. "Biological Effect of Titanium's Surface Roughness on Periodontal Ligament Cells." In 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5162449.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Si-Eun Kim, Soo-Hyuk Uhm, Doo-Hoon Song, Chong-Kwan Kim, Kwang-Mahn Kim, Kyoung-Nam Kim, and Jeon-Geon Han. "Enhanced funtion of human periodontal ligament cells cultured on nanoporous titanium surfaces." In 2012 IEEE 39th International Conference on Plasma Sciences (ICOPS). IEEE, 2012. http://dx.doi.org/10.1109/plasma.2012.6383859.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Lee, Jaeyul, Jaeseok Park, Muhammad Faizan Shirazi, Hosung Jo, Pilun Kim, Ruchire Eranga H. Wijesinghe, Mansik Jeon, and Jeehyun Kim. "Imaging of periodontal tissue using swept-source optical coherence tomography for measurement of gingival sulcus depth (Conference Presentation)." In Imaging, Manipulation, and Analysis of Biomolecules, Cells, and Tissues XVIII, edited by Daniel L. Farkas, James F. Leary, and Attila Tarnok. SPIE, 2020. http://dx.doi.org/10.1117/12.2547280.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Yining Wang, Haibin Xia, Yan Zhao, and Tao Jiang. "Three-Dimensional Culture of Human Periodontal Ligament Cells on Highly Porous Polyglycolic Acid Scaffolds in vitro." In 2005 IEEE Engineering in Medicine and Biology 27th Annual Conference. IEEE, 2005. http://dx.doi.org/10.1109/iembs.2005.1615573.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Mohamed Zaid, Hagar, Khaled Abdel-ghaffar, Tarek Hessin El-bialy, Mamdouh Farid, and Fatma Hamed M. El-demerdash. "Evaluation Of Regenerative Potential Of Pulp -derived Stem Cells And Gingival-derived Stem Cells In The Regeneration Of Periodontal Defects (experimental Study)." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0741.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Hessin El-bialy, Tarek, Jacqueline Crossman, Ali Saleem, Khaled Abdel-ghaffar, Mamdouh Farid, and Elham Fawzi. "Effect Of Using Gingival Stem Cells And Therapeutic Ultrasound On Periodontal Ligament During Orthodontic Treatment In Beagle Dogs." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0060.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Valverde, Mariana Bonilla, and Rodrigo Mora Rodriguez. "miRNA networks regulating gene expression in response to tension or compression forces in the cells of the periodontal ligament." In 2022 IEEE 4th International Conference on BioInspired Processing (BIP). IEEE, 2022. http://dx.doi.org/10.1109/bip56202.2022.10032478.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Santos, Karoline dos, Karina Ruiz, and Catharina Sacramento. "Evaluation of 5-Azacytidine as an inducer of osteoblastic / cementoblastic differentiation of progenitor cells from the periodontal ligament of humans." In Congresso de Iniciação Científica UNICAMP. Universidade Estadual de Campinas, 2019. http://dx.doi.org/10.20396/revpibic2720192363.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Andriani, Ika, Atiek Driana Rahmawati, Maulida Nurhasanah, and M. Ihza Humanindito. "The Effects of Antimicrobial Peptide Gel on Angiogenesis and Fibroblast Cells in Periodontal Tissue Regeneration in a Periodontitis Rats Model Exposed by Nicotine." In 4th International Conference on Sustainable Innovation 2020–Health Science and Nursing (ICoSIHSN 2020). Paris, France: Atlantis Press, 2021. http://dx.doi.org/10.2991/ahsr.k.210115.036.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Ataf Abdel-ghaffar, Khaled, Tarek El-bialy, Ali Saleem, Mamdouh Farid, and Elham Fawzi. "Title: The Use Of Low Intensity Pulsed Ultrasound (lipus) And Gingival Mesenchymal Stem Cells (gmscs) For The Treatment Of Severe Periodontal Defects In Dogs." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0729.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Periodontal cells":

1

Zhang, Yuhao, Wenheng Zhao, Liyang Jia, Nan Xu, Yan Xiao, and Qiyan Li. The application of stem cells in tissue engineering for periodontal defects in randomized controlled trial: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2022. http://dx.doi.org/10.37766/inplasy2022.1.0036.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Hu, Jing, Zeyue Ouyang, Yue Guo, and YunZhi Feng. Clinical application and efficacy of mesenchymal stem cells in the regeneration of periodontal defects: a systematic review and meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2023. http://dx.doi.org/10.37766/inplasy2023.5.0097.

Full text
APA, Harvard, Vancouver, ISO, and other styles

To the bibliography