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Relevant bibliographies by topics / Peptide effects/adrenal cortex

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Academic literature on the topic 'Peptide effects/adrenal cortex'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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Journal articles on the topic "Peptide effects/adrenal cortex"

1

Hinson, JP, and S. Kapas. "Actions of vasoactive intestinal peptide on the rat adrenal zona glomerulosa." Journal of Endocrinology 161, no. 1 (1999): 51–57. http://dx.doi.org/10.1677/joe.0.1610051.

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Previous studies, by this group and others, have shown that vasoactive intestinal peptide (VIP) stimulates aldosterone secretion, and that the actions of VIP on aldosterone secretion by the rat adrenal cortex are blocked by beta adrenergic antagonists, suggesting that VIP may act by the local release of catecholamines. The present studies were designed to test this hypothesis further, by measuring catecholamine release by adrenal capsular tissue in response to VIP stimulation. Using intact capsular tissue it was found that VIP caused a dose-dependent increase in aldosterone secretion, with a c

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2

Thomson, LM, S. Kapas, M. Carroll, and JP Hinson. "Autocrine role of adrenomedullin in the human adrenal cortex." Journal of Endocrinology 170, no. 1 (2001): 259–65. http://dx.doi.org/10.1677/joe.0.1700259.

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Previous studies from our laboratory have reported that adrenomedullin is synthesised in rat zona glomerulosa cells. In the present studies, it was found that the human adrenocortical cell line H295R expresses the gene encoding adrenomedullin, and that immunoreactive adrenomedullin is released into the culture medium. Furthermore, it was found that secretion of adrenomedullin is regulated by angiotensin II and forskolin. Studies on the actions of adrenomedullin and calcitonin gene-related peptide (CGRP) revealed a stimulatory effect of adrenomedullin, but not of CGRP, on aldosterone and cortis

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3

Hinson, J. P., L. A. Cameron, and S. Kapas. "Neuropeptide Y modulates the sensitivity of the rat adrenal cortex to stimulation by ACTH." Journal of Endocrinology 145, no. 2 (1995): 283–89. http://dx.doi.org/10.1677/joe.0.1450283.

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Abstract Neuropeptide Y (NPY) has been identified in nerves supplying the adrenal cortex of several mammalian species, although its function in this tissue is unknown. The present studies, employing adrenocortical cells prepared by collagenase digestion, have shown that NPY, in the absence of other stimulants, has no effect on steroid secretion by the rat adrenal over a range of peptide concentrations (10−11 to 10 −6 mol/l). However, in the presence of physiological concentrations of ACTH, which are submaximal for the stimulation of aldosterone secretion, NPY (10−6 mol/l) significantly enhance

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4

Coll, Anthony P., Martin Fassnacht, Steffen Klammer, et al. "Peripheral administration of the N-terminal pro-opiomelanocortin fragment 1–28 to Pomc−/− mice reduces food intake and weight but does not affect adrenal growth or corticosterone production." Journal of Endocrinology 190, no. 2 (2006): 515–25. http://dx.doi.org/10.1677/joe.1.06749.

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Pro-opiomelanocortin (POMC) is a polypeptide precursor that undergoes extensive processing to yield a range of peptides with biologically diverse functions. POMC-derived ACTH is vital for normal adrenal function and the melanocortin α-MSH plays a key role in appetite control and energy homeostasis. However, the roles of peptide fragments derived from the highly conserved N-terminal region of POMC are less well characterized. We have used mice with a null mutation in the Pomc gene (Pomc−/−) to determine the in vivo effects of synthetic N-terminal 1–28 POMC, which has been shown previously to po

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5

Andreis, P. G., G. Neri, T. Prayer-Galetti, et al. "Effects of Adrenomedullin on the Human Adrenal Glands: An in Vitro Study." Journal of Clinical Endocrinology & Metabolism 82, no. 4 (1997): 1167–70. http://dx.doi.org/10.1210/jcem.82.4.3854.

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Abstract Numerous lines of evidence indicate that adrenal medulla exerts a paracrine control on the secretory activity of the cortex by releasing catecholamines and several regulatory peptides. Adrenomedullin (ADM) is contained in adrenal medulla of several mammalian species, including humans. Thus, we investigated whether human ADM1–52 exerts a modulatory action on steroid secretion of human adrenal cortex in vitro. Dispersed adrenocortical cells (obtained from the gland tail deprived of chromaffin cells) and adrenal slices (including both capsule and medulla) were employed. ADM specifically

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Kapas, S., A. Martinez, F. Cuttitta, and JP Hinson. "Local production and action of adrenomedullin in the rat adrenal zona glomerulosa." Journal of Endocrinology 156, no. 3 (1998): 477–84. http://dx.doi.org/10.1677/joe.0.1560477.

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This study was designed to investigate the synthesis and action of adrenomedullin in the rat adrenal gland. The results obtained from in situ hybridization and immunocytochemical studies suggest that adrenomedullin is synthesized not only in the medulla, but also within the zona glomerulosa of the rat adrenal cortex. Findings from in situ hybridization and binding studies also suggested that specific adrenomedullin receptors are expressed in the zona glomerulosa, and that low levels are present in the inner zones of the cortex. The Kd of the zona glomerulosa adrenomedullin receptor (5.5 nmol/l

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Hinson, J. P., S. Kapas, C. D. Orford, and G. P. Vinson. "Vasoactive intestinal peptide stimulation of aldosterone secretion by the rat adrenal cortex may be mediated by the local release of catecholamines." Journal of Endocrinology 133, no. 2 (1992): 253–58. http://dx.doi.org/10.1677/joe.0.1330253.

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ABSTRACT The effects of vasoactive intestinal peptide (VIP) on adrenocortical function were investigated using several different preparations of adrenocortical tissue. VIP caused a significant increase in perfusion medium flow rate and in aldosterone and corticosterone secretion by the isolated perfused rat adrenal gland, with a threshold of 1 pmol in 200 μl, but did not affect basal steroid secretion by collagenase-dispersed adrenocortical cells at any concentration used, from 10 pmol/l to 10 μmol/l. The presence of VIP (100 nmol/l) had no significant effect on the response of zona glomerulos

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Karteris, Emmanouil, Rachel J. Machado, Jing Chen, Sevasti Zervou, Edward W. Hillhouse, and Harpal S. Randeva. "Food deprivation differentially modulates orexin receptor expression and signaling in rat hypothalamus and adrenal cortex." American Journal of Physiology-Endocrinology and Metabolism 288, no. 6 (2005): E1089—E1100. http://dx.doi.org/10.1152/ajpendo.00351.2004.

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Although starvation-induced biochemical and metabolic changes are perceived by the hypothalamus, the adrenal gland plays a key role in the integration of metabolic activity and energy balance, implicating feeding as a major synchronizer of rhythms in the hypothalamic-pituitary-adrenal (HPA) axis. Given that orexins are involved in regulating food intake and activating the HPA axis, we hypothesized that food deprivation, an acute challenge to the systems that regulate energy balance, should elicit changes in orexin receptor signaling at the hypothalamic and adrenal levels. Food deprivation indu

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Delarue, C., JM Conlon, I. Remy-Jouet, A. Fournier, and H. Vaudry. "Endothelins as local activators of adrenocortical cells." Journal of Molecular Endocrinology 32, no. 1 (2004): 1–7. http://dx.doi.org/10.1677/jme.0.0320001.

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Besides the classical corticotropic hormones, ACTH and angiotensin II, various regulatory peptides produced by the adrenal gland are thought to participate in the control of corticosteroid secretion. Here, we review the evidence that endothelins (ETs) synthesized within the adrenal cortex may act as autocrine and/or paracrine factors to regulate adrenocortical cell activity. The expression of ETs has been detected in normal, hyperplastic and neoplastic adrenocortical cells. The occurrence of ET receptors has been described in the different zones of the cortex. ETs stimulate the secretion of bo

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Giuliani, Luisa, Livia Lenzini, Michele Antonello, et al. "Expression and Functional Role of Urotensin-II and Its Receptor in the Adrenal Cortex and Medulla: Novel Insights for the Pathophysiology of Primary Aldosteronism." Journal of Clinical Endocrinology & Metabolism 94, no. 2 (2009): 684–90. http://dx.doi.org/10.1210/jc.2008-1131.

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Abstract Context: The involvement of urotensin II, a vasoactive peptide acting via the G protein-coupled urotensin II receptor, in arterial hypertension remains contentious. Objective: We investigated the expression of urotensin II and urotensin II receptor in adrenocortical and adrenomedullary tumors and the functional effects of urotensin II receptor activation. Design: The expression of urotensin II and urotensin II receptor was measured by real time RT-PCR in aldosterone-producing adenoma (n = 22) and pheochromocytoma (n = 10), using histologically normal adrenocortical (n = 6) and normal

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Dissertations / Theses on the topic "Peptide effects/adrenal cortex"

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Hansell, D. J. "Action of ACTH peptides on the adrenal gland." Thesis, University of Oxford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233492.

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Dunn, Stacey Leanne. "Fetal growth restriction : effects on the adrenal cortex in postnatal lambs /." Title page and abstract only, 2004. http://web4.library.adelaide.edu.au/theses/09SB/09sbd9231.pdf.

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Supornsilchai, Vichit. "Effects of endocrine disruptors on adrenocortical and leydig cell steroidogenesis /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-276-7/.

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MacAuley, Iain Alasdair Somerled. "An analysis of the effects of oncogenes and growth factors on rat adrenal cortex cells." Thesis, University of British Columbia, 1987. http://hdl.handle.net/2429/27438.

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The process of oncogenic transformation in vitro has been examined in an attempt to define the molecular mechanisms of carcinogenesis. Transformation in Ki-MSV-infected rat adrenal cortex cells appears to be a multistep process (Auersperg et al., 1981), as does the process of transformation in other nonestablished cells (Land et at., 1983b). The Ki-MSV-infected adrenal cortex cells initially express a partially transformed phenotype and after further passaging progress to a highly transformed phenotype (Calderwood and Auersperg, 1984). Examination of Ki-MSV-infected adrenal cortex cells indica

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Bertke, Alexander. "Social Buffering By Unfamiliar Adult Males in Periadolescent Guinea Pigs: The Effects on HPA Axis Activity And Fos Induction In The Medial Prefrontal Cortex." Wright State University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1559558966039455.

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Leboulenger, François. "Contribution à l'étude de l'activité de la corticosurrénale des amphibiens anoures, une glande endocrine sous contrôle multifactoriel." Rouen, 1986. http://www.theses.fr/1986ROUES027.

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Mise en évidence des fluctuations saisonnières et journalières des taux circulants de corticostéroïdes. Analyse des paramètres de la réponse corticosurrénalienne à l'ACTH et à différents peptides issus de la maturation de la proopiomélanocortine. Mise en évidence de la co-localisation du VIP et de peptides opiacés dans les granules de sécrétion des cellules chromaffines, rôle de ces peptides dans la stimulation de la stéroïdogenèse. Structure de la proenképhaline A d'amphibien. Rôle de l'acétylcholine via des récepteurs muscariniques utilisant les prostaglandines comme second messager

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Costa, Marcia Helena Soares. "Estudo da expressão dos receptores do peptídeo insulinotrópico dependente de glicose (GIPR) e do hormônio luteinizante (LHCGR) em tumores e hiperplasias do córtex adrenal." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-11092007-134837/.

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Introdução: Os receptores do peptídeo insulinotrópico dependente de glicose (GIPR) e do hormônio luteinizante (LHCGR) são receptores acoplados à proteína G com amplo padrão de expressão tecidual. A expressão anômala destes receptores tem sido descrita em casos de hiperplasia adrenal macronodular independente de ACTH (AIMAH) e em alguns adenomas, resultando em aumento da secreção hormonal (cortisol, andrógenos e aldosterona) pelo cortex adrenal. O papel destes receptores em outras formas de hiperplasia, como a doença adrenocortical nodular pigmentosa primária (PPNAD), aumento da adrenal associa

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Khan, Tanweera S. "New Diagnostic and Therapeutic Approaches in Adrenocortical Cancer." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4243.

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Netchitailo, Pierre. "La corticostéroïdogénèse chez un amphibien anoure : mécanismes intracellulaires et contrôle multifactoriel." Rouen, 1987. http://www.theses.fr/1987ROUES037.

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Zhao, Huifang. "Improved Methods of Sepsis Case Identification and the Effects of Treatment with Low Dose Steroids: A Dissertation." eScholarship@UMMS, 2011. https://escholarship.umassmed.edu/gsbs_diss/529.

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Sepsis is the leading cause of death among critically ill patients and the 10th most common cause of death overall in the United States. The mortality rates increase with severity of the disease, ranging from 15% for sepsis to 60% for septic shock. Patient with sepsis can present varied clinical symptoms depending on the personal predisposition, causal microorganism, organ system involved, and disease severity. To facilitate sepsis diagnosis, the first sepsis consensus definitions was published in 1991 and then updated in 2001. Early recognition of a sepsis patient followed with timely and app

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Books on the topic "Peptide effects/adrenal cortex"

1

Yuuki, Inoue, and Watanabe Kouki, eds. Adverse effects of steroids. Nova Science Publishers, 2008.

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2

(Editor), Philip W. Harvey, David Everett (Editor), and Christopher Springall (Editor), eds. Adrenal Toxicology (Target Organ Toxicology Series). Informa Healthcare, 2008.

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Book chapters on the topic "Peptide effects/adrenal cortex"

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Mulrow, Patrick J., Roberto Franco-Saenz, K. Atarashi, Masao Takagi, and Mari Takagi. "Effect of Atrial Peptides on the Adrenal Cortex." In Atrial Hormones and Other Natriuretic Factors. Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4614-7529-3_9.

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Höllt, V., and G. Horn. "Nicotine Induces Opioid Peptide Gene Expression in Hypothalamus and Adrenal Medulla of Rats." In Effects of Nicotine on Biological Systems. Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-7457-1_38.

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Kochakian, Charles D. "The Effects on Enzymes of Adrenal Cortex, Diet, Œstrogens, and Experimental Diabetes." In Ciba Foundation Symposium - Steroid Hormones and Enzymes (Book II of Colloquia on Endocrinology, Vol. 1). John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470718742.ch12.

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Bergenstal, D. M., Charles Huggins, and Thomas L.-Y. Dao. "Metabolic Effects of Adrenalectomy in Man." In Ciba Foundation Symposium - The Human Adrenal Cortex (Book II of Colloquia on Endocrinology, Vol. 8). John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470715215.ch9.

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Mach, R. S., and J. Fabre. "Clinical and Metabolic Effects of Aldosterone." In Ciba Foundation Symposium - The Human Adrenal Cortex (Book II of Colloquia on Endocrinology, Vol. 8). John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470715215.ch6.

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Miyamoto, Hirokuni, Fumiko Mitani, Kuniaki Mukai, and Yuzuru Ishimura. "Effects of ACTH and Angiotensin II on the Novel Cell Layer Without Corticosteroid-Synthesizing Activity in Rat Adrenal Cortex." In Oxygen Homeostasis and Its Dynamics. Springer Japan, 1998. http://dx.doi.org/10.1007/978-4-431-68476-3_29.

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Sherlock, Mark, and Mark Gurnell. "Disorders of the adrenal cortex." In Oxford Textbook of Medicine, edited by Mark Gurnell. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0249.

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Three classes of steroid hormone are produced by the adrenal cortex after uptake of precursor cholesterol from the plasma—mineralocorticoids, glucocorticoids, and sex steroids—with classical endocrine feedback loops controlling their secretion. Glucocorticoids have more diverse and extensive roles than mineralocorticoids, regulating sodium and water homeostasis, glucose and carbohydrate metabolism, inflammation, and stress. These effects are mediated by the interaction of cortisol with ubiquitous glucocorticoid receptors, and the induction or repression of target gene transcription (via glucocorticoid response elements, GREs). Adrenocortical diseases are relatively uncommon, but they have detrimental clinical consequences and can be treated effectively. Hormonal deficiency or excess is usually the result of abnormal secretion.

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Auchus, Richard J., and Keith L. Parker. "The Adrenal Glands." In Textbook of Endocrine Physiology. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199744121.003.0016.

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The basic function of the adrenal glands is to protect the organism against acute and chronic stress, a concept popularized as the fight-or-flight response for the medulla and as the alarm reaction for the cortex. The steroid hormones of the cortex and the catecholamines of the medulla probably developed as protection against immediate stress or injury and more prolonged deprivation of food and water. In acute stress, catecholamines mobilize glucose and fatty acids for energy and prepare the heart, lungs, and muscles for action, while glucocorticoids protect against overreactions from the body’s responses to stress. In the more chronic stress of food and fluid deprivation, adrenocortical steroid hormones stimulate gluconeogenesis to maintain the supply of glucose and increase sodium reabsorption to maintain body fluid volume. Based on the widespread effects of its secreted products in multiple tissues, adrenal dysfunction is associated with protean manifestations. Diseases associated with adrenocortical hypofunction are relatively rare, while those associated with adrenocortical hyperfunction are slightly more common. However, both of these conditions are life threatening if untreated, and a high index of suspicion must therefore be maintained. Subtle increases in cortisol secretion or tissue sensitivity to glucocorticoids may be involved in many of the devastating effects of chronic stress, including visceral obesity, hypertension, diabetes mellitus, dyslipidemia, infertility, and depression. Moreover, exogenous glucocorticoids are widely used to treat numerous diseases and, when used in supraphysiological doses, can induce all of the manifestations of glucocorticoid excess. Perhaps because the adrenal medulla accounts for only 10 % of total sympathetic nervous activation, we can live quite well without it, and syndromes due to hypofunction are not clinically significant. However, conditions of excess catecholamine output due to tumors called pheochromocytomas are a rare but potentially life-threatening cause of secondary hypertension.

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Ziegler, CG, JW Brown, AV Schally, et al. "Neuropeptide Hormone Receptor Expression in Human Adrenal Tumors and Cell Lines: Antiproliferative Effects of Peptide Analogues." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part1.p2.p1-65.

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McCarty, Richard. "Stress-Sensitive Brain Circuits." In Stress and Mental Disorders: Insights from Animal Models. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780190697266.003.0005.

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A series of forebrain, midbrain, and brainstem nuclei exert stimulatory or inhibitory effects on sympathetic outflow from the intermediolateral cell column in thoracic and lumbar areas of the spinal cord. Some of these brain areas contain cell bodies that serve as command neurons that can simultaneously activate sympathetic outflow to multiple target tissues. CRF-containing cell bodies in the paraventricular nuclei receive multiple direct and indirect inputs from various brain areas that participate in the regulation of the HPA axis. Hormones of the adrenal cortex have been shown to exert damaging structural effects on the structure of neurons in the hippocampus and amygdala. The immune system is stress-responsive, and circulating immune cells and proinflammatory cytokines are able to penetrate the blood-brain barrier during exposure to stressors. Brain microglia appear to serve as neuroimmune sensors of stress. Optogenetic and chemogenetic techniques have been essential tools in probing the functions of stress-responsive brain circuits and their impact on behavior.

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Conference papers on the topic "Peptide effects/adrenal cortex"

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Tanable, A., Y. Yatomi, T. Ohashi, H. Oka, T. Kariya, and S. Kume. "EFFECTS OF HUMAN ATRIAL NATRIURETIC PEPTIDES ON SECRETION REACTION IN HUMAN PLATELETS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644873.

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Human atrial natriuretic peptide (h-ANP) has vasodilating and natriuretic properties, and inhibits smooth muscle contraction, renal renin secretion and adrenal aldosterone release. Although Schiffrin has reported that human platelets have receptors for ANP, its effects in platelets are not established in vivo. We therefore investigated the influence of h-ANP on secretion reaction in human platelets. Eight healthy subjects, males, aged 20 to 24 years, donated blood for the study. Citrated platelet-rich plasma (PRP) was incubated with or without h-ANP at 37 C for 2.5 minutes. The samples of 0.5

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