Relevant bibliographies by topics / Pediatric. paediatric traumatic brain injury

Academic literature on the topic 'Pediatric. paediatric traumatic brain injury'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Contents

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Pediatric. paediatric traumatic brain injury.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Pediatric. paediatric traumatic brain injury"

1

Coulter, Ian C., and Rob J. Forsyth. "Paediatric traumatic brain injury." Current Opinion in Pediatrics 31, no. 6 (December 2019): 769–74. http://dx.doi.org/10.1097/mop.0000000000000820.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Medani, Samah, and Shruti Agrawal. "Neuroprotection in paediatric traumatic brain injury." Paediatrics and Child Health 31, no. 6 (June 2021): 233–39. http://dx.doi.org/10.1016/j.paed.2021.03.002.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Porter, David, and Kevin Morris. "Traumatic brain injury in the paediatric population." Paediatrics and Child Health 23, no. 5 (May 2013): 212–19. http://dx.doi.org/10.1016/j.paed.2013.02.005.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Thango, Nqobile S., Ursula K. Rohlwink, Lindizwe Dlamini, M. Phophi Tshavhungwe, E. Banderker, Shamiel Salie, J. M. N. Enslin, and Anthony A. Figaji. "Brain interstitial glycerol correlates with evolving brain injury in paediatric traumatic brain injury." Child's Nervous System 37, no. 5 (February 13, 2021): 1713–21. http://dx.doi.org/10.1007/s00381-021-05058-2.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Hornyak, J. E., V. S. Nelson, and E. A. Hurvitz. "The use of methylphenidate in paediatric traumatic brain injury." Pediatric Rehabilitation 1, no. 1 (January 1997): 15–17. http://dx.doi.org/10.3109/17518429709060937.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Savage, Ronald C., Roberta DePompei, Janet Tyler, and Marilyn Lash. "Paediatric traumatic brain injury: A review of pertinent issues." Pediatric Rehabilitation 8, no. 2 (April 2005): 92–103. http://dx.doi.org/10.1080/13638490400022394.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Poomthavorn, P., W. Maixner, and M. Zacharin. "Pituitary function in paediatric survivors of severe traumatic brain injury." Archives of Disease in Childhood 93, no. 2 (November 6, 2007): 133–37. http://dx.doi.org/10.1136/adc.2007.121137.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Armstrong, Kira, and Kimberly A. Kerns. "The assessment of parent needs following paediatric traumatic brain injury." Pediatric Rehabilitation 5, no. 3 (January 2002): 149–60. http://dx.doi.org/10.1080/1363849021000039353.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Bressan, Silvia, Amit Kochar, Ed Oakley, Meredith Borland, Natalie Phillips, Sarah Dalton, Mark D. Lyttle, et al. "Traumatic brain injury in young children with isolated scalp haematoma." Archives of Disease in Childhood 104, no. 7 (March 4, 2019): 664–69. http://dx.doi.org/10.1136/archdischild-2018-316066.

Full text
Abstract:
ObjectiveDespite high-quality paediatric head trauma clinical prediction rules, the management of otherwise asymptomatic young children with scalp haematomas (SH) can be difficult. We determined the risk of intracranial injury when SH is the only predictor variable using definitions from the Pediatric Emergency Care Applied Research Network (PECARN) and Children’s Head Injury Algorithm for the Prediction of Important Clinical Events (CHALICE) head trauma rules.DesignPlanned secondary analysis of a multicentre prospective observational study.SettingTen emergency departments in Australia and New Zealand.PatientsChildren <2 years with head trauma (n=5237).InterventionsWe used the PECARN (any non-frontal haematoma) and CHALICE (>5 cm haematoma in any region of the head) rule-based definition of isolated SH in both children <1 year and <2 years.Main outcome measuresClinically important traumatic brain injury (ciTBI; ie, death, neurosurgery, intubation >24 hours or positive CT scan in association with hospitalisation ≥2 nights for traumatic brain injury).ResultsIn children <1 year with isolated SH as per PECARN rule, the risk of ciTBI was 0.0% (0/109; 95% CI 0.0% to 3.3%); in those with isolated SH as defined by the CHALICE, it was 20.0% (7/35; 95% CI 8.4% to 36.9%) with one patient requiring neurosurgery. Results for children <2 years and when using rule specific outcomes were similar.ConclusionsIn young children with SH as an isolated finding after head trauma, use of the definitions of both rules will aid clinicians in determining the level of risk of ciTBI and therefore in deciding whether to do a CT scan.Trial registration numberACTRN12614000463673.
APA, Harvard, Vancouver, ISO, and other styles
10

Parry, Louise, Arthur Shores, Caroline Rae, Allan Kemp, Mary-Clare Waugh, Ray Chaseling, and Pamela Joy. "An Investigation of Neuronal Integrity in Severe Paediatric Traumatic Brain Injury." Child Neuropsychology 10, no. 4 (December 2004): 248–61. http://dx.doi.org/10.1080/09297040490909279.

Full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Pediatric. paediatric traumatic brain injury"

1

Ward, Heather Jean, and n/a. "Prospective Memory: Early Developmental Trajectory and Effects of Paediatric Traumatic Brain Injury on its Functioning." Griffith University. School of Psychology, 2005. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20050804.154501.

Full text
Abstract:
Very little is known about the effects of paediatric traumatic brain injury (TBI) on prospective memory, the memory for future intentions such as remembering to post a letter in the morning or do homework. The main aim of this thesis was to redress that shortcoming in the literature. To investigate the effects of paediatric TBI on prospective memory as reliably and fully as possible, the study of children and adolescents with brain injuries was preceded by a developmental study. Given that the process of recovery from brain injury is imposed on the ongoing process of development, it is important to understand more about the normal developmental trajectory of prospective memory first of all. Study 1 compared the prospective-memory performance of 88 normally developing children, adolescents and young adults. The main task was computerised, and its design was influenced by a prefrontal-lobe model because prospective memory is believed to be mediated by the prefrontal regions of the brain. Variables associated with prefrontal-lobe capacity were manipulated: the cognitive demand of an ongoing task, and the importance of the prospective task. Results of Study 1 found that children remembered to respond to fewer prospective cues than adolescents or adults, but that adolescents and adults remembered similarly. Further, the differences between the children's performance and the adolescents' and adults' widened as the cognitive demand of the ongoing task increased. However, the effects of increasing the cognitive demand did not vary between the adolescents and adults. It made no difference to anyone's performance whether the importance of remembering the prospective cues was stressed or not. On the other hand, performance on executive functions, as measured by the Self-Ordered Pointing Task (SOPT), the Stroop Colour Word Interference Test (Stroop), and the Tower of London (TOL), which are also believed to be affected by prefrontal capacity, produced the same age effects as were produced on the computerised prospective-memory task. Further, performance on the SOPT and Stroop predicted performance on the high-demand level of the prospective-memory task. Study 2 compared 34 children and adolescents with TBI with the non-injured children and adolescents from Study 1 on the same tasks. Results revealed that overall those with TBI had poorer prospective-memory performance than their non-injured peers. However, a different pattern of impairment was evident in the children than in the adolescents. Specifically, the children with TBI performed similarly to their non-injured peers, but the adolescents with TBI were significantly worse than the non-injured adolescents. This trend was most noticeable as the cognitive demand of the ongoing task increased. Further, the age and injury effects were reflected in the performances on the executive-function tests, and the TOL predicted performance on the high-demand, prospective-memory task in those with TBI. Study 3 aimed to examine the ecological validity of Study 2, by investigating whether the impairments in prospective memory in young people with TBI measured quantitatively, were matched with qualitative data. Twelve parents of children and adolescents with mild to severe TBI were interviewed about whether or not their children's injuries impacted on their memory (retrospective and prospective) in everyday life. Results showed that in general most children suffered memory losses as a result of their brain injuries, and that prospective-memory loss caused particular hardships for the children and their families. Taken together, the results of the current research revealed that the development of prospective memory reaches a peak of maturity in adolescence, and that adolescents with TBI show greater decrements in prospective memory than adolescents without TBI, but that this pattern is not evident in children, where those with TBI were not significantly different from those without. These findings give support to the prefrontal-lobe model of prospective memory by showing that prefrontal maturity, which reaches a peak during adolescence, reflects the prospective-memory performance of healthy adolescents, and prefrontal injury, which is very common with TBI, shows the effects of deficits more during adolescence than in earlier years when the prefrontal regions are not yet fully developed. Study 3 showed that impairments in prospective memory that result from TBI translate into disabilities in the real world. As a follow up it is recommended that rehabilitation strategies be designed to assist young people with prospective-memory impairments adjust better to school and the demands of everyday living. The prefrontal-lobe model should guide the design of such strategies.
APA, Harvard, Vancouver, ISO, and other styles
2

Ward, Heather Jean. "Prospective Memory: Early Developmental Trajectory and Effects of Paediatric Traumatic Brain Injury on its Functioning." Thesis, Griffith University, 2005. http://hdl.handle.net/10072/367932.

Full text
Abstract:
Very little is known about the effects of paediatric traumatic brain injury (TBI) on prospective memory, the memory for future intentions such as remembering to post a letter in the morning or do homework. The main aim of this thesis was to redress that shortcoming in the literature. To investigate the effects of paediatric TBI on prospective memory as reliably and fully as possible, the study of children and adolescents with brain injuries was preceded by a developmental study. Given that the process of recovery from brain injury is imposed on the ongoing process of development, it is important to understand more about the normal developmental trajectory of prospective memory first of all. Study 1 compared the prospective-memory performance of 88 normally developing children, adolescents and young adults. The main task was computerised, and its design was influenced by a prefrontal-lobe model because prospective memory is believed to be mediated by the prefrontal regions of the brain. Variables associated with prefrontal-lobe capacity were manipulated: the cognitive demand of an ongoing task, and the importance of the prospective task. Results of Study 1 found that children remembered to respond to fewer prospective cues than adolescents or adults, but that adolescents and adults remembered similarly. Further, the differences between the children's performance and the adolescents' and adults' widened as the cognitive demand of the ongoing task increased. However, the effects of increasing the cognitive demand did not vary between the adolescents and adults. It made no difference to anyone's performance whether the importance of remembering the prospective cues was stressed or not. On the other hand, performance on executive functions, as measured by the Self-Ordered Pointing Task (SOPT), the Stroop Colour Word Interference Test (Stroop), and the Tower of London (TOL), which are also believed to be affected by prefrontal capacity, produced the same age effects as were produced on the computerised prospective-memory task. Further, performance on the SOPT and Stroop predicted performance on the high-demand level of the prospective-memory task. Study 2 compared 34 children and adolescents with TBI with the non-injured children and adolescents from Study 1 on the same tasks. Results revealed that overall those with TBI had poorer prospective-memory performance than their non-injured peers. However, a different pattern of impairment was evident in the children than in the adolescents. Specifically, the children with TBI performed similarly to their non-injured peers, but the adolescents with TBI were significantly worse than the non-injured adolescents. This trend was most noticeable as the cognitive demand of the ongoing task increased. Further, the age and injury effects were reflected in the performances on the executive-function tests, and the TOL predicted performance on the high-demand, prospective-memory task in those with TBI. Study 3 aimed to examine the ecological validity of Study 2, by investigating whether the impairments in prospective memory in young people with TBI measured quantitatively, were matched with qualitative data. Twelve parents of children and adolescents with mild to severe TBI were interviewed about whether or not their children's injuries impacted on their memory (retrospective and prospective) in everyday life. Results showed that in general most children suffered memory losses as a result of their brain injuries, and that prospective-memory loss caused particular hardships for the children and their families. Taken together, the results of the current research revealed that the development of prospective memory reaches a peak of maturity in adolescence, and that adolescents with TBI show greater decrements in prospective memory than adolescents without TBI, but that this pattern is not evident in children, where those with TBI were not significantly different from those without. These findings give support to the prefrontal-lobe model of prospective memory by showing that prefrontal maturity, which reaches a peak during adolescence, reflects the prospective-memory performance of healthy adolescents, and prefrontal injury, which is very common with TBI, shows the effects of deficits more during adolescence than in earlier years when the prefrontal regions are not yet fully developed. Study 3 showed that impairments in prospective memory that result from TBI translate into disabilities in the real world. As a follow up it is recommended that rehabilitation strategies be designed to assist young people with prospective-memory impairments adjust better to school and the demands of everyday living. The prefrontal-lobe model should guide the design of such strategies.
Thesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Psychology
Full Text
APA, Harvard, Vancouver, ISO, and other styles
3

Trenchard, Sian Olivia. "Traumatic brain injury in a paediatric population." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/traumatic-brain-injury-in-a-paediatric-population(cf299afa-75ff-4a58-9684-6b332d25715e).html.

Full text
Abstract:
This thesis examined neuropsychological and psychological outcomes following paediatric traumatic brain injury (TBI). The introductory chapter provides an overview of the paediatric TBI literature, giving definitions of key terms and concepts and providing a description of the epidemiology of childhood head injury. Key models relevant to paediatric TBI are introduced, including developmental neurological, cognitive and psychological perspectives. This is followed by a discussion of factors pertinent to outcome after TBI, followed by a description of outcomes relating to cognitive, behavioural, psychological, adaptive and family functioning domains. Existing research demonstrates that poor outcomes are frequently observed in paediatric TBI populations across these domains and difficulties are persistent over time, particularly where children have sustained severe head injury. Thus, research has turned its focus to the prediction of outcomes which can assist clinicians in the identification of those individuals who will require rehabilitation in order to promote their long-term recovery. Whilst the literature has identified injury and demographic factors that can assist in this process, little attention has been given to the potential utility of psychological screening assessment. Given the prevalence of neuropsychological and psychosocial problems after paediatric TBI and lack of empirical data considering factors predictive of difficulty at the post-acute phase, this research aimed to consider the clinical utility of completing a pre-discharge screening assessment in children and adolescents with TBI. Specific areas of consideration included the potential impact of injury severity on neuropsychological functioning, psychosocial impairment and return to full-time schooling. The study design comprised a prospective case series of 11 children and adolescents with TBI (aged 7-15 years), who were assessed both pre- and post-discharge (3-6 month follow-up). Domains of intellectual, emotional, behavioural, and adaptive functioning, health-related quality of life and parenting stress were assessed at both time-points. Clinically significant findings were demonstrated in domains of neuropsychological and psychosocial functioning, particularly for those with a severe TBI. Specifically, ratings of self-reported emotional distress, and parental perceptions of child health-related quality of life were found to be within clinical ranges at pre- and post-discharge for more than half of the participants. The majority of participants with severe injury required further neuropsychological assessment and interventions relating to emotional and/or behavioural management. The post-discharge functioning of this cohort provided preliminary evidence for the clinical utility of cognitive and psychosocial screening after paediatric TBI. The observed level of clinical need, particularly in the severely-injured group indicated that screening was a useful tool for early identification of difficulties, and provided an opportunity for timely intervention. Without screening, children and adolescents with TBI may be discharged to the community without appropriate support in place; raising long-term concerns for the child, family, and the wider social and economic systems. Despite this, further research which explicates these findings within larger samples is required. The discussion chapter reviews these findings in relation to the wider literature, followed by consideration of this study's limitations. The thesis concludes with a description of the clinical implications of the findings and suggested future directions.
APA, Harvard, Vancouver, ISO, and other styles
4

Lalani, Sanam Jivani. "Effects of Traumatic Brain Injury on Pediatric Brain Volume." BYU ScholarsArchive, 2018. https://scholarsarchive.byu.edu/etd/6924.

Full text
Abstract:
This study investigated the effects of lesion presence within larger brain networks (e.g., default mode network (DMN), salience network (SN), and mentalizing network (MN)) in the chronic phase of a pediatric traumatic brain injury (TBI) and the effect on social function. We compared children with a TBI to children with an orthopedic injury (OI) with three different aims. The first aim was to determine whether network volume differed by group (e.g., TBI vs. OI). Second, investigate if lesion presence in a sub component region of the network resulted in total network volume loss for that network. Finally, learn whether network volume would predict outcome on the Behavior Assessment System for Children, Second Edition (BASC-2). Approximately 184 participants (65% male; 70% Caucasian) between the ages of 6-17 years completed testing and a structural MRI scan in the chronic stage (at least one-year post-injury) of the injury. Injury severity included complicated mild, moderate, and severe TBI. Radiological findings were analyzed using recommendations from the Common Data Elements' core (presence or absence of a lesion) and supplementary (lesion type and location) recommendations. Volumetrics for all participants were obtained with FreeSurfer to quantify total network volumes for the DMN, SN, and MN. The parent of each participant completed a behavioral measure for externalizing and internalizing behaviors. Three sets of statistical analyses were completed, including multivariate analysis of covariance, analysis of covariance, and multiple regression, for each of the three aims of the study, respectively. There were significant differences in total DMN volume between the two groups and participants with lesions solely in the MN had lower total MN volume. Moreover, lower total MN volume was associated with worse functioning on measures of externalizing and internalizing behaviors. The larger implications, including developmental and social implications, of these findings are discussed.
APA, Harvard, Vancouver, ISO, and other styles
5

Sheppard-Jones, Nicolas. "Ocular impairment in pediatric mild traumatic brain injury." Thesis, McGill University, 2014. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=121355.

Full text
Abstract:
Mild Traumatic Brain Injuries (mTBI) and concussions are complex injuries with high incidence rates in children and adolescents. There currently exists no 'gold standard' for the diagnosis of concussions, and detection and monitoring are made challenging by highly variable clinical presentations. There is growing evidence that mTBI is associated with oculomotor impairment in adults, and that this type of deficit may serve as a marker for the injury. The literature indicates the question has never been addressed in pediatric mTBI. The research presented here sought to address this knowledge gap by first evaluating smooth pursuit and fixational eye movement integrity in a cohort of children and adolescents suffering from mTBI, and then comparing their performances to control participants not having sustained a head injury. The research yielded mixed findings. On the one hand, we found that fixational eye movements are not impaired in pediatric mTBI; measures of fixational eye movement integrity were comparable across groups. On the other hand, selective deficits in smooth pursuit eye movements were found. Synchronization of eye movement with target motion was significantly poorer for mTBI patients. Their abilities to trace target trajectory accurately and to match target velocity, however, were not found to be impaired. It remains unclear whether the observed deficits were caused by disrupted function of the smooth pursuit system proper, by damage to areas that modulate smooth pursuit through top-down influence, or by a combination of both. These preliminary results suggest that select smooth pursuit paradigms could play a role for diagnosing pediatric mTBI, and reinforce the need for further studies in this novel area of research.
Les Traumatismes Crâniens Légers (TCL) et les commotions cérébrales sont des blessures complexes auxquelles les enfants et les adolescents sont particulièrement à risque. Il n'existe pas actuellement d'outil objectif pour le diagnostic et le monitorage de ces blessures, qui sont difficiles à gérer en raison de la grande hétérogénéité clinique qui les caractérise. Un nombre grandissant d'études indique que les TCL peuvent engendrer une dysfonction au niveau des mouvements oculaires chez les adultes, et que ces troubles visuels pourraient servir de marqueurs efficaces pour la détection. La question n'a jamais été posée chez les enfants. Ce projet tente d'apporter une réponse préliminaire, d'abord en évaluant l'intégrité des Mouvements de Poursuite Visuelle (MPV) et de fixation chez des enfants et adolescents atteints d'un TCL, puis en comparant leur performance à celle de sujets contrôles n'ayant pas subi de blessure à la tête. Les résultats obtenus sont mixtes. Aucune dysfonction au niveau des mouvements de fixation n'a été décelée; toutes les mesures utilisées pour évaluer les capacités de fixation étaient comparables par groupe. En revanche, des troubles sélectifs ont été détectés au niveau des MPV. Les patients atteints de TCL éprouvaient en moyenne plus de difficulté à synchroniser le mouvement de leurs yeux avec le mouvement d'une cible. La précision et la vélocité du mouvement ne semblaient pas toutefois affectées. Les résultats ne permettent pas de trancher sur la nature exacte du trouble observé, ce dernier pouvant être causé à la fois par une dysfonction au niveau des circuits visuo-moteurs propre, et par une dysfonction au niveau de structures de plus haut niveau modulant les MPV. Ces résultats préliminaires indiquent que l'évaluation des MPV pourrait contribuer au diagnostic et au monitorage de TCL pédiatriques, et renforcent le besoin d'investigations additionnelles dans ce domaine.
APA, Harvard, Vancouver, ISO, and other styles
6

Rohlwink, Ursula Karin. "Paediatric traumatic Brain Injury: The relationship between Intracranial Pressure and Brain Oxygenation." Master's thesis, University of Cape Town, 2009. http://hdl.handle.net/11427/2889.

Full text
Abstract:
Introduction: Intracranial pressure (ICP) monitoring is a cornerstone of care for patients with severe traumatic brain injury (TBI). The primary goal of ICP treatment is to preserve brain oxygenation, and since brain oxygenation is usually not measured, the control of ICP is used as a surrogate marker. However studies indicating that cerebral hypoxia/ischemia may occur in the face of adequate ICP and cerebral perfusion pressure (CPP) suggest that the interaction between ICP and brain oxygenation is poorly understood and warrants further investigation. This is of particular importance in the context of children in whom the interpretation of relationships between intracranial factors is even more complex due to changing physiological norms with age. To date little scientific data exists in children and treatment threshold values are often extrapolated from adult guidelines. This study aims to better understand the relationship between ICP and brain oxygenation measured as brain tissue oxygen tension (PbtO2) in a large paediatric cohort suffering from severe TBI. Specifically analysis 1) investigated ICP and PbtO2 profiles over time following TBI, 2) examined the relationship between ICP and PbtO2 from time-linked paired observations, 3) explored various critical thresholds for ICP and PbtO2, and 4) interrogated digital data trends depicting the relationship between ICP and PbtO2. The level of agreement between hourly recorded and high frequency electronic data for ICP and PbtO2 was also evaluated. Method: Paired ICP and PbtO2 data from 75 children with severe TBI were tested with correlation and regression. Additional analyses controlled for mean arterial pressure (MAP), arterial partial pressure of oxygen (PaO2), CPP, arterial partial pressure of carbon dioxide (PaCO2) and haemoglobin (Hb) using multivariate logistic regression analysis and general estimating equations. Various thresholds for ICP were examined; these included age-related thresholds to account for the potential influence of age. Receiver-operating curves (ROCs) were used to graphically demonstrate the relationships between various thresholds of ICP and various definitions of low PbtO2. These were constructed for pooled and individual patient data. Interrogation of electronically recorded data allowed for case illustrations examining the relationship between ICP and PbtO2 at selected time points. Hourly and electronic data were compared using Bland and Altman plots and by contrasting the frequency of ICP and PbtO2 perturbations recorded with each system. 5 Result: Analyses using over 8300 hours of paired observations revealed a weak relationship between ICP and PbtO2, with an initially positive but weak slope (r = 0.05) that trended downwards only at higher values of ICP. Controlling for inter-individual differences, as well as MAP, CPP, PaO2, PaCO2 and Hb did not strengthen this association. This poor relationship was further reflected in the examination of threshold ICP values with ROCs, no singular critical ICP threshold for compromised brain oxygenation was discernible. Using age-based thresholds did not improve this relationship and individual patient ROCs demonstrated inter-individual heterogeneity in the relationship between ICP and PbtO2. However, it was clear that in individual patients ICP did exhibit a strong negative relationship with PbtO2 at particular time points, but various different relationships between the 2 variables were also demonstrated. A high level of agreement was found between hourly and electronic data. Conclusion: These results suggest that the relationship between ICP and PbtO2 is highly complex. Although the relationship in individual children at specific time points may be strong, pooled data for the entire cohort of patients, and even for individual patients, suggest only a weak relationship. This is likely because several other factors affect PbtO2 outside of ICP, and some factors affect both independently of each other. These results suggest that more study should be directed at optimising ICP thresholds for treatment in children. The use of complimentary monitoring modalities may assist in this task. Depending on the adequacy of measures of brain perfusion, metabolism or oxygenation, it is possible that targeting a range of ICP values in individual patients may be appropriate; however this would require detailed investigation.
APA, Harvard, Vancouver, ISO, and other styles
7

Peng, Jin. "Follow-up Appointment Adherence after Pediatric Traumatic Brain Injury." The Ohio State University, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu1520418824950075.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Hughes, Keith G. "Predictors of family functioning following pediatric traumatic brain injury." Connect to resource, 1996. http://rave.ohiolink.edu/etdc/view.cgi?acc%5Fnum=osu1261056181.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Sukhina, Alona. "Developments on Post-Traumatic Brain Injury-Induced Hypothalamic Pituitary Dysfunction: A Pediatric Case." Thesis, The University of Arizona, 2018. http://hdl.handle.net/10150/626895.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Moran, Lisa M. "Do post-concussive symptoms discriminate injury severity in pediatric mild traumatic brain injury?" The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1250198689.

Full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Books on the topic "Pediatric. paediatric traumatic brain injury"

1

Roberta, DePompei, ed. Pediatric traumatic brain injury: Proactive intervention. 2nd ed. Australia: Delmar/Thomson Learning, 2003.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Roberta, DePompei, ed. Pediatric traumatic brain injury: Proactive intervention. San Diego, Calif: Singular Pub., 1994.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Anderson, Vicki, and Keith Owen Yeates, eds. Pediatric Traumatic Brain Injury. Cambridge: Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511676383.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

R, Hooper Stephen, ed. Pediatric traumatic brain injury. Thousand Oaks, Calif: Sage Publications, 1994.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Catroppa, Cathy, Vicki Anderson, Miriam H. Beauchamp, and Keith Owen Yeates. New Frontiers in Pediatric Traumatic Brain Injury. New York, NY : Routledge, 2016.: Routledge, 2016. http://dx.doi.org/10.4324/9780203868621.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Blosser, Jean L., and Roberta DePompei. Pediatric Traumatic Brain Injury: Proactive Intervention. 2nd ed. Singular, 2002.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Snow, Jeffrey H. Pediatric traumatic brain injury. 1994.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

1958-, Anderson Vicki, and Yeates Keith Owen, eds. Pediatric traumatic brain injury. Cambridge: Cambridge University Press, 2010.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

Huh, Jimmy, Ramesh Raghupathi, and Bridgette D. Semple, eds. Long-Term Consequences of Pediatric Traumatic Brain Injury. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-83250-303-4.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Blosser, Jean L., and Roberta DePompei. Pediatric Traumatic Brain Injury: Proactive Intervention, Third Edition. Plural Publishing, Inc., 2019.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Book chapters on the topic "Pediatric. paediatric traumatic brain injury"

1

Saha, Snigdha, and Stephen Honeybul. "Contemporary Management of Paediatric Head Injuries." In Traumatic Brain Injury, 123–35. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-78075-3_12.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Awaad, Yasser M. "Traumatic Brain Injury." In Absolute Pediatric Neurology, 687–704. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78801-2_27.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Ammar, Ahmed, and Stephen Honeybul. "Ethical Issues in Paediatric Traumatic Brain Injury." In Traumatic Brain Injury, 327–34. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-78075-3_32.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Hoersting, Angela, and Jodi E. Mullen. "Traumatic Brain Injury." In Nursing Care of the Pediatric Neurosurgery Patient, 255–316. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-49319-0_8.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Hoersting, Angela, and Jodi E. Mullen. "Traumatic Brain Injury." In Nursing Care of the Pediatric Neurosurgery Patient, 193–248. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-32554-0_7.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Carroll, Christopher P., Vijay M. Ravindra, and Mario J. Cardoso. "Pediatric Traumatic Brain Injury." In Pediatric Trauma Care, 167–88. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-08667-0_14.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Backhaus, Samantha. "Pediatric Traumatic Brain Injury." In Encyclopedia of Clinical Neuropsychology, 2615–20. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_264.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Backhaus, Samantha. "Pediatric Traumatic Brain Injury." In Encyclopedia of Clinical Neuropsychology, 1–7. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_264-2.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Abecasis, Francisco. "Traumatic Brain Injury – Pediatric." In Neurovascular Sonography, 197–207. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96893-9_13.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Backhaus, Samantha. "Pediatric Traumatic Brain Injury." In Encyclopedia of Clinical Neuropsychology, 1890–95. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_264.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Pediatric. paediatric traumatic brain injury"

1

Fuchs, Franklin, Omar Kamal, Hanao Li, Mihye Ahn, and So Young Ryu. "Pediatric Patient Traumatic Brain Injury Prediction1." In 2020 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2020. http://dx.doi.org/10.1109/bibm49941.2020.9313568.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Dennis, Emily L., Faisal Rashid, Talin Babikian, Richard Mink, Christopher Babbitt, Jeffrey Johnson, Christopher Giza, Robert Asarnow, and Paul Thompson. "Altered network topology in pediatric traumatic brain injury." In 13th International Symposium on Medical Information Processing and Analysis, edited by Jorge Brieva, Juan David García, Natasha Lepore, and Eduardo Romero. SPIE, 2017. http://dx.doi.org/10.1117/12.2285245.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Souza, Leandro Cândido de, Ricardo Santos de Oliveira, Francisco de Assis Carvalho do Vale, and Matheus Fernando Manzolli Ballestero. "Epidemiology of Pediatric Traumatic Brain Injury in Brazil." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.630.

Full text
Abstract:
Background: Pediatric traumatic brain injury (TBI) is a serious social and economic problem. Emerging countries have 89% of the cases worldwide and lack relevant epidemiological studies on the subject. Objectives: Characterize the demographic, social and economic profiles of the pediatric population suffering TBI in Brazil. Methods: Data on the cases of pediatric TBI in Brazil between 2008 and 2020 were collected through the computer department of the Unified Health System (DATASUS) maintained by the Brazilian Ministry of Health. Results: There are about 28,836 hospital admissions due to pediatric TBI per year and an incidence of 45.11 admissions /100,000/year. The in-hospital mortality rate was 1.47/100,000/year, and the case fatality rate was 3.26%. The average annual cost of hospital expenses was US$ 12.311.759, with the average admission cost having a value of US $417. The 15–19 age group was the most frequently admitted to hospital for pediatric TBI and had the highest number of in- hospital deaths; in addition, more males were affected by this trauma compared to females at a rate of 2.31:1. Ethnic populations that are social minorities are more susceptible to a poor prognosis of TBI. Conclusion: Pediatric TBI should be recognized as an important public health problem in Brazil, as it is responsible for considerable social and economic costs. Public policies that reduce the causes of this type of trauma in the pediatric population are urgently needed in Brazil and other emerging countries.
APA, Harvard, Vancouver, ISO, and other styles
4

BRAGA, L. W. "TBI.01. Traumatic Brain Injury in Pediatric and Adolescent Populations." In I International Symposium in Neuroscience Meeting. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/isnm-sine3.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Ryan, E., I. Okafor, C. Blackburn, M. Barrett, T. Bolger, and E. Molloy. "G349(P) Spectrum of paediatric traumatic brain injury presenting to tertiary paediatric emergency departments." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.339.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Ryan, Emer, Eimear Duff, Dean Huggard, Mark Bates, Derek G. Doherty, Ashanty M. Melo, and Eleanor Molloy. "GP123 Altered systemic inflammatory response in paediatric mild traumatic brain injury." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.188.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Noufi, Camille, Adam C. Lammert, Daryush D. Mehta, James R. Williamson, Gregory Ciccarelli, Douglas Sturim, Jordan R. Green, Thomas F. Campbell, and Thomas F. Quatieri. "Vocal Biomarker Assessment Following Pediatric Traumatic Brain Injury: A Retrospective Cohort Study." In Interspeech 2019. ISCA: ISCA, 2019. http://dx.doi.org/10.21437/interspeech.2019-1200.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Lee, Sanghun, Seung Chul Lee, Kyoung Jun Song, Ki Jeong Hong, Kyung Won Kang, and Seong Chun Kim. "PW 0697 Epidemiology and outcomes of pediatric traumatic brain injury in korea." In Safety 2018 abstracts. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/injuryprevention-2018-safety.356.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Zavaliangos-Petropulu, Artemis, Emily L. Dennis, Greg Ver Steeg, Talin Babikian, Richard Mink, Christopher Babbitt, Jeffrey Johnson, Christopher C. Giza, Robert F. Asarnow, and Paul M. Thompson. "Variable clustering reveals associations between subcortical brain volume and cognitive changes in pediatric traumatic brain injury." In 12th International Symposium on Medical Information Processing and Analysis, edited by Eduardo Romero, Natasha Lepore, Jorge Brieva, and Ignacio Larrabide. SPIE, 2017. http://dx.doi.org/10.1117/12.2256977.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Ryan, Emer, Lynne Kelly, Eimear Duff, Turlough Bolger, and Eleanor Molloy. "OC15 Paediatric mild traumatic brain injury is associated with systemic inflammasome activation and pubertal scoring." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.15.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Pediatric. paediatric traumatic brain injury"

1

Peñaloza, Blanca. Does paediatric home care improve health outcomes in children? SUPPORT, 2017. http://dx.doi.org/10.30846/1701133.

Full text
Abstract:
Paediatric home care for ill children has been developed for different diseases and with different models as an alternative to care based in hospitals. In this summary we present evidence for home care for children with acute physical conditions, home rehabilitation for children with traumatic brain injury, and home chemotherapy.
APA, Harvard, Vancouver, ISO, and other styles
2

Shujaa, Asaad Suliman, and Qasem Almulihi. Is Hypertonic Saline an Effective Alternative to Mannitol in the Treatment of TBI in Adult and Pediatric Patients? A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0010.

Full text
Abstract:
Review question / Objective: Evaluate and compare the effectiveness of hypertonic saline and mannitol in patients with traumatic brain injury. Rationale: Traumatic brain injury is one of the main reasons for death and disability worldwide. Generally, the frequency of traumatic brain injury in Europe is >2,000 per million yearly; guidelines suggest more conservative interventions, e.g., raising of the upper body, cerebrospinal fluid drainage, and the use of hypertonic saline or mannitol before executing decompressive craniectomy. It is still uncertain whether hypertonic saline is better than mannitol in managing pediatric and adult patients with traumatic brain injury. The present systemic review and meta-analysis aimed to evaluate the effect of hypertonic saline compared to mannitol for managing TBI in traumatic brain injury. Eligibility criteria: Studies were included based on the described eligibility criteria using PICOS: P (Population); I (Intervention); C (Control); O (Outcome); S (Studies); only clinical trials and cohort studies published in English were selected.
APA, Harvard, Vancouver, ISO, and other styles
3

Newman-Toker, David E., Susan M. Peterson, Shervin Badihian, Ahmed Hassoon, Najlla Nassery, Donna Parizadeh, Lisa M. Wilson, et al. Diagnostic Errors in the Emergency Department: A Systematic Review. Agency for Healthcare Research and Quality (AHRQ), December 2022. http://dx.doi.org/10.23970/ahrqepccer258.

Full text
Abstract:
Objectives. Diagnostic errors are a known patient safety concern across all clinical settings, including the emergency department (ED). We conducted a systematic review to determine the most frequent diseases and clinical presentations associated with diagnostic errors (and resulting harms) in the ED, measure error and harm frequency, as well as assess causal factors. Methods. We searched PubMed®, Cumulative Index to Nursing and Allied Health Literature (CINAHL®), and Embase® from January 2000 through September 2021. We included research studies and targeted grey literature reporting diagnostic errors or misdiagnosis-related harms in EDs in the United States or other developed countries with ED care deemed comparable by a technical expert panel. We applied standard definitions for diagnostic errors, misdiagnosis-related harms (adverse events), and serious harms (permanent disability or death). Preventability was determined by original study authors or differences in harms across groups. Two reviewers independently screened search results for eligibility; serially extracted data regarding common diseases, error/harm rates, and causes/risk factors; and independently assessed risk of bias of included studies. We synthesized results for each question and extrapolated U.S. estimates. We present 95 percent confidence intervals (CIs) or plausible range (PR) bounds, as appropriate. Results. We identified 19,127 citations and included 279 studies. The top 15 clinical conditions associated with serious misdiagnosis-related harms (accounting for 68% [95% CI 66 to 71] of serious harms) were (1) stroke, (2) myocardial infarction, (3) aortic aneurysm and dissection, (4) spinal cord compression and injury, (5) venous thromboembolism, (6/7 – tie) meningitis and encephalitis, (6/7 – tie) sepsis, (8) lung cancer, (9) traumatic brain injury and traumatic intracranial hemorrhage, (10) arterial thromboembolism, (11) spinal and intracranial abscess, (12) cardiac arrhythmia, (13) pneumonia, (14) gastrointestinal perforation and rupture, and (15) intestinal obstruction. Average disease-specific error rates ranged from 1.5 percent (myocardial infarction) to 56 percent (spinal abscess), with additional variation by clinical presentation (e.g., missed stroke average 17%, but 4% for weakness and 40% for dizziness/vertigo). There was also wide, superimposed variation by hospital (e.g., missed myocardial infarction 0% to 29% across hospitals within a single study). An estimated 5.7 percent (95% CI 4.4 to 7.1) of all ED visits had at least one diagnostic error. Estimated preventable adverse event rates were as follows: any harm severity (2.0%, 95% CI 1.0 to 3.6), any serious harms (0.3%, PR 0.1 to 0.7), and deaths (0.2%, PR 0.1 to 0.4). While most disease-specific error rates derived from mainly U.S.-based studies, overall error and harm rates were derived from three prospective studies conducted outside the United States (in Canada, Spain, and Switzerland, with combined n=1,758). If overall rates are generalizable to all U.S. ED visits (130 million, 95% CI 116 to 144), this would translate to 7.4 million (PR 5.1 to 10.2) ED diagnostic errors annually; 2.6 million (PR 1.1 to 5.2) diagnostic adverse events with preventable harms; and 371,000 (PR 142,000 to 909,000) serious misdiagnosis-related harms, including more than 100,000 permanent, high-severity disabilities and 250,000 deaths. Although errors were often multifactorial, 89 percent (95% CI 88 to 90) of diagnostic error malpractice claims involved failures of clinical decision-making or judgment, regardless of the underlying disease present. Key process failures were errors in diagnostic assessment, test ordering, and test interpretation. Most often these were attributed to inadequate knowledge, skills, or reasoning, particularly in “atypical” or otherwise subtle case presentations. Limitations included use of malpractice claims and incident reports for distribution of diseases leading to serious harms, reliance on a small number of non-U.S. studies for overall (disease-agnostic) diagnostic error and harm rates, and methodologic variability across studies in measuring disease-specific rates, determining preventability, and assessing causal factors. Conclusions. Although estimated ED error rates are low (and comparable to those found in other clinical settings), the number of patients potentially impacted is large. Not all diagnostic errors or harms are preventable, but wide variability in diagnostic error rates across diseases, symptoms, and hospitals suggests improvement is possible. With 130 million U.S. ED visits, estimated rates for diagnostic error (5.7%), misdiagnosis-related harms (2.0%), and serious misdiagnosis-related harms (0.3%) could translate to more than 7 million errors, 2.5 million harms, and 350,000 patients suffering potentially preventable permanent disability or death. Over two-thirds of serious harms are attributable to just 15 diseases and linked to cognitive errors, particularly in cases with “atypical” manifestations. Scalable solutions to enhance bedside diagnostic processes are needed, and these should target the most commonly misdiagnosed clinical presentations of key diseases causing serious harms. New studies should confirm overall rates are representative of current U.S.-based ED practice and focus on identified evidence gaps (errors among common diseases with lower-severity harms, pediatric ED errors and harms, dynamic systems factors such as overcrowding, and false positives). Policy changes to consider based on this review include: (1) standardizing measurement and research results reporting to maximize comparability of measures of diagnostic error and misdiagnosis-related harms; (2) creating a National Diagnostic Performance Dashboard to track performance; and (3) using multiple policy levers (e.g., research funding, public accountability, payment reforms) to facilitate the rapid development and deployment of solutions to address this critically important patient safety concern.
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Pediatric. paediatric traumatic brain injury' for particular source types:
Journal articles Dissertations / Theses Books
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography