Relevant bibliographies by topics / Pediatric growth

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Pediatric growth'

Author: Grafiati
Published: 18 May 2024

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Journal articles on the topic "Pediatric growth"

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Gallagher, Heather. "Pediatric growth faltering." JAAPA 36, no. 11 (November 2023): 1–6. http://dx.doi.org/10.1097/01.jaa.0000979524.39905.7f.

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ABSTRACT Pediatric growth faltering (GF), previously known as failure to thrive and now also called pediatric malnutrition and weight faltering, is a common clinical finding in primary care. Most pediatric GF cases are caused by inadequate caloric intake, not organic disease states. Evaluation requires clinicians to obtain detailed nutritional, medical, psychosocial, and family histories; take accurate anthropometric measurements; and perform a careful physical examination. Evaluation findings should be analyzed to determine whether targeted diagnostic workup, specialty referral, or a trial of nutritional counseling is indicated. Management includes caregiver education about childhood nutrition and frequent monitoring of growth parameters. A multidisciplinary approach that includes nutritionist, developmental therapist, and other specialty team member involvement is desirable.
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Coutant, R. "Pediatric growth hormone deficiency." Archives de Pédiatrie 28, no. 8 (February 2022): 28/8S1. http://dx.doi.org/10.1016/s0929-693x(22)00035-5.

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Dimeglio, Alain. "Growth in Pediatric Orthopaedics." Journal of Pediatric Orthopaedics 21, no. 4 (July 2001): 549–55. http://dx.doi.org/10.1097/01241398-200107000-00026.

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DeAngelis, Catherine, Ralph Feigin, Thomas DeWitt, Lewis R. First, Ethan A. Jewett, Robert Kelch, Russell W. Chesney, Holly J. Mulvey, Jimmy L. Simon, and Errol R. Alden. "Final Report of the FOPE II Pediatric Workforce Workgroup." Pediatrics 106, Supplement_E1 (November 1, 2000): 1245–55. http://dx.doi.org/10.1542/peds.106.se1.1245.

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From the inception of the Future of Pediatric Education II (FOPE II) Project, it was acknowledged that any discussion of pediatric education would need to encompass a review of the pediatric workforce. This report looks at the current trends in pediatric workforce and draws some conclusions regarding future growth and composition. In addition to looking at demographic trends, ranging from geography to gender, the report explores influences including managed care, telemedicine, and others. Models for determining workforce needs are described and scenarios and projections are discussed. Pediatrics 2000;106(suppl):1245–1255;pediatric workforce.
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Thakkar, Nirav, and James W. Schroeder. "Pediatric Maxillary Sinus Growth Curve." Otolaryngology–Head and Neck Surgery 145, no. 2_suppl (August 2011): P241. http://dx.doi.org/10.1177/0194599811415823a348.

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Ferguson, Anne, and David M. Sedgwick. "Growth Failure in Pediatric IBD." Journal of Pediatric Gastroenterology and Nutrition 18, no. 4 (May 1994): 504. http://dx.doi.org/10.1097/00005176-199405000-00021.

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Chang, Johnny T., Clinton S. Morrison, John R. Styczynski, William Mehan, Stephen R. Sullivan, and Helena O. Taylor. "Pediatric Orbital Depth and Growth." Journal of Craniofacial Surgery 26, no. 6 (September 2015): 1988–91. http://dx.doi.org/10.1097/scs.0000000000001974.

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Goolsby, S. L. P., C. L. Banks, B. B. Eubanks, and L. Christie. "Growth After Pediatric Heart Transplant." Journal of the American Dietetic Association 97, no. 9 (September 1997): A25. http://dx.doi.org/10.1016/s0002-8223(97)00408-2.

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de Broux, E., C. H. Huot, S. Chartrand, and C. Chartrand. "Growth after pediatric heart transplantation." Transplantation Proceedings 33, no. 1-2 (February 2001): 1735–37. http://dx.doi.org/10.1016/s0041-1345(00)02662-2.

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DeBoer, Scott, and Michael Seaver. "Pediatric Growth and Development Revisited." Journal of Emergency Nursing 42, no. 5 (September 2016): e1-e2. http://dx.doi.org/10.1016/j.jen.2016.07.005.

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Dissertations / Theses on the topic "Pediatric growth"

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Amendola, Richard Lee. "Graph-based segmentation of the pediatric trachea in MR images to model growth." Thesis, University of Iowa, 2012. https://ir.uiowa.edu/etd/2810.

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The upper airways are a major site of pediatric airway obstruction with its accompanying morbidity and mortality. The simplest approach to provide a stable airway is to perform a tracheotomy but it is a long recovery with its own complications. Other surgical procedures to reconstruct the airway require significant experience. The long-term objectives of this project are to develop a greater understanding of congenital abnormalities of the larynx and trachea. The objective of this thesis is to create a process to automatically segment and measure the pediatric trachea from MR images. Using 3DSlicer and ITK and program was created to perform the measurements. The software tool was optimized to produce similar results to that of CT image measurements from Pulmonary Workstation. The program was tested on a pediatric population and showed a significant correlation between cross-sectional area and age or height of the individual.
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Saha, Amal Kumar. "Studies on paediatric growth problems in North Bengal districts of India and development of a fuzzy object-oriented knowledge based system for treatment planning." Thesis, University of North Bengal, 1998. http://hdl.handle.net/123456789/190.

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Buhl, Juliane [Verfasser], and Peter [Akademischer Betreuer] Angel. "The senescence-associated secretory phenotype regulates the growth behavior of pediatric pilocytic astrocytoma / Juliane Buhl ; Betreuer: Peter Angel." Heidelberg : Universitätsbibliothek Heidelberg, 2019. http://d-nb.info/1191760510/34.

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Karlsson, Videhult Frida. "Effects of early probiotic supplementation in a pediatric setting : Focus on body composition, metabolism and inflammation." Doctoral thesis, Umeå universitet, Pediatrik, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-119835.

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We aimed to determine the short- and long-term effects on growth, body composition, metabolic and inflammatory markers following supplementation with the probiotic Lactobacillus paracasei ssp. paracasei F19 (LF19) during weaning. Methods: One-hundred and seventy-nine healthy, infants in Umeå city, Västerbotten County were randomised to daily intake of cereals with (n=89) or without (n=90) LF19 108 colony-forming units from 4 to 13 months of age. Weight, length, head circumference and body composition, assessed by skinfold thickness, were examined at 4, 5.5, 6.5, 9, 12 and 13 months of age. Venous blood was drawn at 5.5 and 13 months. In all, 171 infants completed the intervention and were invited to a follow-up at 8-9 years of age between 2009 and 2011, 120 children participated. Weight, height, sagittal abdominal diameter and body composition (using Dual Energy X-ray Absorptiometry-scan) were measured. Data on weight and height at 4 years were collected from medical records. The families filled out a 4-day food record and a food frequency questionnaire, physical activity was assessed using a pedometer for 7 days. At 5.5, 13 months and 8-9 years of age we analysed the serum blood lipid profile. At 8-9 years fasting glucose, insulin, aspartate and alanine transaminases were analysed in serum. Homeostatic Model Assessment index was calculated. At follow-up serum adiponectin, high-sensitivity C-reactive protein and plasma C-peptide, ghrelin, gastric inhibitory polypeptide, glucagon-like peptide 1, glucagon, insulin, leptin, plasminogen activator inhibitor-1, resistin and visfatin were analysed. For characterisation of the plasma metabolome, a subgroup (n=40) was analysed at 5.5 and 13 months of age by gas chromatography time-of-flight mass spectrometry (GC-TOF/MS) analysis and in all (n=112) children at the follow-up using untargeted GC-GC/MS. Results: There were no differences between the LF19 and placebo group regarding body weight, length/height at any assessment from 4 months to 8-9 years of age; nor were there any differences between the groups in body composition. In the LF19 group 19 % were overweight/obese, the corresponding number was 21 % in the placebo group (p=0.78). Analysed metabolic and inflammatory markers, both during the intervention and the follow-up did not differ between the two groups. At 13 months of age lower levels of palmitic acid and palmitoleic acid (both p<0.04) and higher levels of putrescine (p<0.01) were seen in the LF19 compared to the placebo group. These differences did not persist at 8-9 years of age. At that age, we found statistically stronger models when comparing overweight/obese and normal weight children as well as in relation to sex. Conclusion: Early intervention with the probiotic LF19 at the time of weaning exerted transient effects on the metabolome. In a long-term perspective, we found neither benefit nor harm on growth, body composition, metabolic or inflammatory markers following supplementation with LF19 during weaning.
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Tassone, Evelyne. "Extracellular matrix-degrading enzymes and control of fibroblast growth factor-2 (FGF-2) signaling in pediatric glioma cell lines." Doctoral thesis, Università degli studi di Padova, 2012. http://hdl.handle.net/11577/3422194.

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The main purpose of my research project was to investigate the role of two extracellular matrix-degrading enzymes, heparanase (HPSE) and membrane-type 1 matrix metalloproteinase (MT1-MMP), in pediatric gliomas. I spent the first two years of my PhD program in Dott. Maurizio Onisto’s laboratory (University of Padua). Then I continued my work at New York University School of Medicine, under the supervision of Prof. Paolo Mignatti, whose experimental work focuses on the molecular mechanisms of proteolysis-independent signaling by MT1-MMP and its physiological inhibitor, tissue inhibitor of metalloproteinase-2 (TIMP-2). Gliomas, the most common primary brain tumors, comprise a heterogeneous group of neoplasms that originate from glial cells. Despite recent advances in the management of these tumors, children affected by gliomas, particularly the more aggressive forms, have a poor prognosis. Gliomas can diffusely penetrate throughout the brain, even though they remain localized in this organ. One of the most important events during glioma cell invasion is extracellular matrix (ECM) degradation, a complex mechanism that involves both glycosidic and proteolytic enzymes. HPSE is an endo-β-D-glucuronidase secreted in the ECM, where it cleaves the heparan sulfate side chains of both soluble and membrane-bound proteoglycans. MT1-MMP, a cell membrane-bound proteinase with an extracellular catalytic domain and a short cytoplasmic tail, has been implicated in the proteolytic degradation of extracellular and transmembrane proteins. High levels of HPSE and MT1-MMP are present in a variety of aggressive malignancies, a finding that highlights their important role in cancer invasion and metastasis. In this study we characterized pediatric glioma cell lines derived from different types of gliomas: two glioblastoma multiforme, one anaplastic astrocytoma, one diffuse astrocytoma and one pilocytic astrocytoma. In addition, we used a human breast adenocarcinoma cell line to examine the role of MT1-MMP, because these cells do not express this proteinase and thus represent an ideal model for the regulation of its expression. The data reported here show that MT1-MMP controls activation of intracellular signaling by fibroblast growth factor-2 (FGF-2) and FGF-2 binding to the breast adenocarcinoma cells. We found no clear correlation between HPSE, MT1-MMP or FGF-2 expression and the aggressiveness of the pediatric astrocytoma cells. Gene silencing of HPSE in a pediatric glioblastoma cell line does not affect vascular endothelial growth factor (VEGF) expression or cell proliferation, but upregulates matrix metalloproteinase-2 (MMP-2) and MT1-MMP expression. Moreover, ERK1/2 activation by FGF-2 does not correlate with MT1-MMP expression and is modified by an MMP inhibitor in these pediatric glioma cells. Finally, TIMP-2 controls ERK1/2 activation in all glioma cells. Taken together, the results show that MT1-MMP does not have the same effects in breast carcinoma and pediatric glioma cells, indicating a different and more complex control mechanism of intracellular signaling. This initial characterization of these unique pediatric astrocytoma cell lines provides new insights into the knowledge of this poorly studied group of tumors.
L’obiettivo principale del mio progetto di ricerca è stato analizzare il ruolo di due enzimi che degradano la matrice extracellulare, l’“heparanase” (HPSE) e la “membrane-type 1 matrix metalloproteinase” (MT1-MMP), nei gliomi pediatrici. Ho trascorso i primi due anni di Dottorato nel laboratorio del Dott. Maurizio Onisto (Università di Padova). Ho poi continuato il mio lavoro presso la New York University School of Medicine, sotto la supervisione del Prof. Paolo Mignatti, il cui lavoro sperimentale è focalizzato sull’approfondimento dei meccanismi molecolari alla base dell’attivazione del segnale intracellulare da parte di MT1-MMP e del suo inibitore fisiologico, il “tissue inhibitor of metalloproteinases-2” (TIMP-2). I gliomi, i più comuni tumori cerebrali primari, comprendono un gruppo eterogeneo di neoplasie che originano dalle cellule gliali. Nonostante i recenti progressi raggiunti nel trattamento e nel controllo di tali tumori, la prognosi dei bambini affetti da glioma, ed in particolare dalle sue forme più aggressive, rimane tuttora infausta. Pur essendo confinati nell’organo nel quale originano, i gliomi possono invadere tutte le aree del cervello. Uno degli eventi più importanti che caratterizzano l’invasività dei gliomi è costituito dalla degradazione della matrice extracellulare, un complesso meccanismo che coinvolge enzimi sia glicosidici sia proteolitici. HPSE è una endo-β-D-glucuronidasi secreta nella matrice extracellulare, nella quale taglia le catene di eparan solfato dei proteoglicani solubili e legati alla membrana. MT1-MMP, una proteasi legata alla membrana e composta da un dominio catalitico extracellulare e da una piccola coda citoplasmatica, è coinvolta nella degradazione proteolitica di proteine extracellulari e di membrana. Elevati livelli di HPSE e MT1-MMP sono stati riscontrati in numerosi tipi di tumore e tale evidenza sottolinea il ruolo chiave che essi svolgono nell’invasività tumorale e nella formazione di metastasi. In questo studio sono state caratterizzate cinque linee cellulari di glioma pediatrico derivanti da diversi tipi di glioma: due glioblastomi multiformi, un astrocitoma anaplastico, un astrocitoma diffuso ed un astrocitoma pilocitico. Con lo scopo iniziale di esaminare il ruolo di MT1-MMP nell’attivazione del segnale indotto dall’FGF-2, è stata inoltre utilizzata una linea cellulare di carcinoma mammario, la quale non esprime MT1-MMP e perciò rappresenta un modello ideale per studiare la regolazione della sua espressione. I dati riportati mostrano che, nelle cellule di carcinoma mammario, MT1-MMP regola l’attivazione del segnale intracellulare da parte del “fibroblast growth factor-2” (FGF-2) e controlla il legame di questo fattore di crescita alla superficie delle cellule. Nelle cellule di astrocitoma pediatrico non è stata identificata alcuna chiara correlazione tra espressione di HPSE, MT1-MMP o FGF-2 ed aggressività tumorale. I risultati inoltre dimostrano che il silenziamento genico di HPSE in una linea cellulare di glioblastoma pediatrico non influenza l’espressione del “vascular endothelial growth factor” (VEGF) o la proliferazione cellulare, ma determina la sovraespressione della “matrix metalloproteinase-2” (MMP-2) e di MT1-MMP. Inoltre, nelle cellule di glioma, l’attivazione di ERK1/2 da parte di FGF-2 non correla con l’espressione di MT1-MMP e risulta modificata dal trattamento con un inibitore di MMP. Infine, in tutte le cellule di glioma, anche TIMP-2 regola l’attivazione del segnale intracellulare. In conclusione, i risultati ottenuti mostrano che MT1-MMP non ha gli effetti nelle cellule di carcinoma mammario e di glioma pediatrico, indicando l’esistenza di un differente e più complesso meccanismo di controllo del segnale intracellulare. La caratterizzazione delle linee cellulari di astrocitoma pediatrico presentata in questa tesi offre una più completa conoscenza di questo gruppo di tumori ancora poco studiati.
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Lundgren, Maria. "Born Small for Gestational Age : Impact of Linear Catch-up Growth." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl.[distributör], 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3563.

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Appleman, Stephanie S. M. D. "Bone Disease in TPN-dependent Infants and Children with Intestinal Failure." University of Cincinnati / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1320326652.

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Forslund, Marianne. "The neurodevelopment potential in the preterm infant a longitudinal follow-up study on growth and development from birth to nine years of age /." Lund : Dept. of Pediatrics, University of Lund, 1992. http://books.google.com/books?id=hNhqAAAAMAAJ.

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Hanlon, Harriet Wehner. "Differences in female and male development of the human cerebral cortex from birth to age 16." Diss., This resource online, 1994. http://scholar.lib.vt.edu/theses/available/etd-10192006-115609/.

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Li, Yi. "A Generalization of AUC to an Ordered Multi-Class Diagnosis and Application to Longitudinal Data Analysis on Intellectual Outcome in Pediatric Brain-Tumor Patients." Digital Archive @ GSU, 2009. http://digitalarchive.gsu.edu/math_diss/1.

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Receiver operating characteristic (ROC) curves have been widely used in evaluation of the goodness of the diagnostic method in many study fields, such as disease diagnosis in medicine. The area under the ROC curve (AUC) naturally became one of the most used variables in gauging the goodness of the diagnosis (Mossman, Somoza 1991). Since medical diagnosis often is not dichotomous, the ROC curve and AUC need to be generalized to a multi-dimensional case. The generalization of AUC to multi-class case has been studied by many researchers in the past decade. Most recently, Nakas & Yiannoutsos (2004) considered the ordered d classes ROC analysis by only considering the sensitivities of each class. Hence, their dimension is only d. Cha (2005) considered more types of mis-classification in the ordered multiple-class case, but reduced the dimension of Ferri, at.el. from d(d-1) to 2(d-1). In this dissertation we are trying to adjust and calculate the VUS for an ordered multipleclass with Cha’s 2(d-1)-dimension method. Our methodology of finding the VUS is introduced. We present the method of adjusting and calculating VUS and their statistical inferences for the 2(d-1)-dimension. Some simulation results are included and a real example will be presented. Intellectual outcomes in pediatric brain-tumor patients were investigated in a prospective longitudinal study. The Standard-Binet Intelligence Scale-Fourth Edition (SB-IV) Standard Age Score (SAS) and Composite intelligence quotient (IQ) score are examined as cognitive outcomes in pediatric brain-tumor patients. Treatment factors, patient factors and time since diagnosis are taken into account as the risk factors. Hierarchical linear/quadratic models and Gompertz based hierarchical nonlinear growth models were applied to build linear and nonlinear longitudinal curves. We use PRESS and Volume Under the Surface (VUS) as the criterions to compare these two methods. Some model interpretations are presented in this dissertation.
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Books on the topic "Pediatric growth"

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Wales, Jerry. Pediatric endocrinology and growth. 2nd ed. Edinburgh: Saunders, 2003.

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Loche, Sandro. Pediatric neuroendocrinology. Basel: Karger, 2010.

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Sandro, Loche, ed. Pediatric neuroendocrinology. Basel: Karger, 2010.

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Wales, Jerry. Color atlas of pediatric endocrinology and growth. London: Mosby-Wolfe, 1996.

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Rekers-Mombarg, Lyset. Idiopathic short stature: Growth and growth hormone treatment. Amsterdam: Thesis Publishers, 1998.

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Sandro, Loche, ed. Pediatric neuroendocrinology. Basel: Karger, 2010.

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1942-, Docherty David, and Canadian Society for Exercise Physiology., eds. Measurement in pediatric exercise science. Champaign, IL: Human Kinetics, 1996.

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Babler, Elizabeth K. Clinical handbook of pediatric endocrinology. St. Louis, Missouri: Quality Medical Publishing, Inc., 2013.

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1928-, Raimondi Anthony J., Choux M, and Di Rocco C, eds. The Pediatric spine. New York: Springer-Verlag, 1989.

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Marks, David Ryan. Accommodation for head growth in pediatric cochlear implantation. [New Haven: s.n.], 1989.

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Book chapters on the topic "Pediatric growth"

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Triantafyllou, Panagiota, and Stephanie Roberts. "Pediatric Body Growth." In Pediatric Dentistry, 25–35. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-78003-6_3.

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Gevers, Evelien F., and Mehul T. Dattani. "Growth and Growth Factors." In Yearbook of Pediatric Endocrinology, 41–60. Basel: KARGER, 2009. http://dx.doi.org/10.1159/000239771.

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Rosenbloom, Arlan L., and Jaime Guevara-Aguirre. "Growth Hormone Insensitivity." In Pediatric Endocrinology, 31–59. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73782-9_2.

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Ong, Ken K. "Intrauterine Growth Retardation." In Pediatric Obesity, 103–12. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60327-874-4_8.

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Rosenbloom, Arlan L. "Growth Hormone Insensitivity." In Pediatric Endocrinology, 29–53. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-60761-395-4_2.

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Styne, Dennis M. "Disorders of Growth." In Pediatric Endocrinology, 47–90. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-18371-8_5.

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Styne, Dennis M. "Disorders of Growth." In Pediatric Endocrinology, 55–119. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-09512-2_5.

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Coutant, Régis. "Growth and Growth Factors." In Yearbook of Pediatric Endocrinology 2005, 33–46. Basel: KARGER, 2005. http://dx.doi.org/10.1159/000088696.

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Cianfarani, Stefano. "Growth and Growth Factors." In Yearbook of Pediatric Endocrinology 2012, 45–59. Basel: S. KARGER AG, 2012. http://dx.doi.org/10.1159/000341203.

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Bouhours-Nouet, Natacha, and Régis Coutant. "Growth and Growth Factors." In Yearbook of Pediatric Endocrinology 2006, 33–46. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000094104.

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Conference papers on the topic "Pediatric growth"

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Roszelle, Breigh N., Benjamin T. Cooper, Ning Yang, Steven Deutsch, and Keefe B. Manning. "The Challenges of Developing a Pediatric Ventricular Assist Device From a Fluid Dynamics Perspective." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19549.

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As a medical device proves successful in adult patients, it is anticipated that a similar solution for pediatrics may be developed. However, in many cases this task has proved to be much more complex than simply scaling the device down for a miniature adult patient. Pediatric patients present a unique set of characteristics and constraints not seen in adults. These include a large range of sizes from infants to adolescents, the possible growth of the patient during use, possible anatomical deformities and a body that has not fully matured.
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Ramos-Homs, Amy. "Synthesis of Bone Scaffold for Pediatric Bone Defects Using 3D Printing." In MME Undergraduate Research Symposium. Florida International University, 2022. http://dx.doi.org/10.25148/mmeurs.010560.

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Pediatric bone defects, requiring surgical interventions and implants, include malignant and nonmalignant bone tumors and trauma fractures. Malignant bone tumors (MBT), such as Osteosarcoma and Ewing sarcoma, are aggressive primary cancers that affect growing adolescent bones (10- to 19-year-olds) and require complex reconstruction due to large bone excision during surgical interventions. Pediatric bone fractures requiring surgical interventions peak in 10- to 14-year-olds and are a major public health concern in the US with an impact on patients, parents, and healthcare costs of approx. 350 billion. These diseases require bone tissue replacement in changing bones. Bone reconstruction and medical implant design for growing pediatric bones have unique challenges due to active growth and there is a greater need for active, resorbable, and patient-specific implants to prevent growth impediments. The current available pediatric implant is limited in addressing these needs and is primarily addressed by static metallic implants designed for adults. We plan to work towards the design and synthesis of a bone scaffold by modifying a CAD model considering the size of the porosity in the structure of the pediatric bone. This modified model will be 3D printed and subjected to tests to evaluate the strength and composition of the scaffold. Afterwards, the scaffold is used for cell culture in hopes of eliciting cellular response for bone formation and cell regeneration, since a key factor to assess is whether the scaffold will grow with the bone, or the bone will grow with the scaffold. This is done to support the attachment of cells on the surface of the bone to actively support bone modeling processes under structural changes of growing bones.
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Lerner, Amy L., Janet L. Kuhn, and Scott J. Hollister. "Stress-Growth Relationships in the Developing Rabbit Distal Femur." In ASME 1996 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1996. http://dx.doi.org/10.1115/imece1996-1271.

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Abstract Several pediatric orthopaedic conditions appear to be related to mechanical influences on bone growth. In this study we tested the hypothesis that there is a relationship between bone growth rates and mechanical stresses which are present in the growth plate during normal development. The model selected was the rabbit distal femur, which exhibits a dramatic growth plate curvature resulting from variations in bone growth rates.
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Bochicchio, Mario A., Lucia Vaira, Antonella Longo, Antonio Malvasi, and Andrea Tinelli. "FPGT: An online system for customized fetal and pediatric growth tracking." In 2014 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2014. http://dx.doi.org/10.1109/bhi.2014.6864393.

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Daartey, Abel, Rameswari Chilamakuri, and Saurabh Agarwal. "Adavosertib targets Cell Cycle Regulator WEE1 and Inhibits Pediatric Neuroblastoma Growth." In ASPET 2024 Annual Meeting Abstract. American Society for Pharmacology and Experimental Therapeutics, 2024. http://dx.doi.org/10.1124/jpet.048.934960.

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Mooney, Marie Renee. "Abstract B15: Pediatric glioblastoma cell lines maintain response to developmental growth factors." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-b15.

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Chen, Joshua, Christopher Pool, Einat Slonimsky, Tonya King, Sandeep Pradham, and Meghan Wilson. "Anatomic Parameters and Growth of the Pediatric Skull Base: Endonasal Access Implications." In 31st Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1743774.

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Reincke, J., V. C. Stark, D. Diaz-Gil, Y. Von Kodolitsch, R. Kozlik-Feldmann, J. Olfe, P. Wiegand, T. Zeller, and T. S. Mir. "Transforming Growth Factor β Level in Healthy Pediatric Children: Strong Impact of Age." In The 54th Annual Meeting of the German Society for Pediatric Cardiology (DGPK). Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1742984.

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Amendola, Richard L., Joseph M. Reinhardt, Yutaka Sato, Miriam B. Zimmerman, Henry R. Diggelmann, and Deborah Kacmarynski. "Graph-based segmentation of the pediatric trachea in MR images to model growth." In SPIE Medical Imaging, edited by John B. Weaver and Robert C. Molthen. SPIE, 2013. http://dx.doi.org/10.1117/12.2006290.

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Oostveen, Ellie, H. Bentouhami, M. Hagendorens, W. De Backer, and J. Weyler. "Nonuniform growth in pediatric lung function between 4 and 14 years of age." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.oa4559.

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Reports on the topic "Pediatric growth"

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Su, Huimin, Hui Mao, Yangyang Zhang, Heng Yin, Jiayi Hong, Yifei Song, Yuxuan Yang, et al. The effect of long-term pediatric tuina on physical growth in children: A protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2024. http://dx.doi.org/10.37766/inplasy2024.2.0116.

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Wang, Xiaoyu. Pediatric tuina in treating recurrent respiratory tract infection in children: a systematic review and meta‑analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2023. http://dx.doi.org/10.37766/inplasy2023.4.0075.

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Abstract:
Review question / Objective: Is pediatric tuina an effective treatment for recurrent respiratory tract infection in children? Condition being studied: Recurrent respiratory tract infection (RRTI) is a common disease in children, which refers to the recurrence of upper and lower respiratory tract infections within a year, exceeding the prescribed number of times. It is more common in infants under 3 years old. The disease is easy to relapse and lasts for a long time, affecting the normal growth and development of children and physical and mental health, easily causing other diseases, leading to a variety of chronic wasting diseases, and damaging the function of organs and the immune system. Immunotherapy and nutritional therapy are commonly used in Western medicine. At present, the treatment of RRTI in children with traditional Chinese medicine has achieved a certain effect, and the treatment mainly includes internal treatment and external treatment. Tuina therapy is one of the common therapies for the treatment of RRTI in children with traditional Chinese medicine. Because of its advantages, there are many literature reports on tuina treatment of this disease, with a good total effective rate, but whether its therapeutic effect is higher than other therapies has not been determined as a whole. This study used the method of systematic review to collect the published clinical research literature on the treatment of RRTI in children at home and abroad for systematic review, so as to provide a reference for clinical research.
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