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Journal articles on the topic 'Pc 5'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 February 2022

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1

Abernethy, Amy P. "PC-FACS." Journal of Pain and Symptom Management 37, no. 5 (May 2009): 934–40. http://dx.doi.org/10.1016/s0885-3924(09)00448-5.

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Abernethy, Amy P. "PC-FACS." Journal of Pain and Symptom Management 38, no. 2 (August 2009): 322–26. http://dx.doi.org/10.1016/s0885-3924(09)00626-5.

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3

Abernethy, Amy P. "PC-FACS." Journal of Pain and Symptom Management 40, no. 1 (July 2010): 155–61. http://dx.doi.org/10.1016/s0885-3924(10)00449-5.

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4

Abernethy, Amy P. "PC-FACS." Journal of Pain and Symptom Management 41, no. 4 (April 2011): 812–17. http://dx.doi.org/10.1016/s0885-3924(11)00151-5.

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Abernethy, Amy P. "PC-FACS." Journal of Pain and Symptom Management 41, no. 6 (June 2011): 1111–15. http://dx.doi.org/10.1016/s0885-3924(11)00237-5.

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6

Zhukovsky, Donna S. "PC-FACS." Journal of Pain and Symptom Management 45, no. 1 (January 2013): 150–56. http://dx.doi.org/10.1016/s0885-3924(12)00830-5.

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7

Zhukovsky, Donna S. "PC-FACS." Journal of Pain and Symptom Management 45, no. 2 (February 2013): 310–16. http://dx.doi.org/10.1016/s0885-3924(13)00036-5.

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8

di. "Pilotprojekt Tablet-PC." Pflegezeitschrift 71, no. 1-2 (January 2018): 59. http://dx.doi.org/10.1007/s41906-018-0372-5.

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9

Hudson, M. K., R. E. Denton, M. R. Lessard, E. G. Miftakhova, and R. R. Anderson. "A study of Pc-5 ULF oscillations." Annales Geophysicae 22, no. 1 (January 1, 2004): 289–302. http://dx.doi.org/10.5194/angeo-22-289-2004.

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Abstract. A study of Pc-5 magnetic pulsations using data from the Combined Release and Radiation Effects Satellite (CRRES) was carried out. Three-component dynamic magnetic field spectrograms have been used to survey ULF pulsation activity for the approximate fourteen month lifetime of CRRES. Two-hour panels of dynamic spectra were examined to find events which fall into two basic categories: 1) toroidal modes (fundamental and harmonic resonances) and 2) poloidal modes, which include compressional oscillations. The occurence rates were determined as a function of L value and local time. The main result is a comparable probability of occurence of toroidal mode oscillations on the dawn and dusk sides of the magnetosphere inside geosynchronous orbit, while poloidal mode oscillations occur predominantly along the dusk side, consistent with high azimuthal mode number excitation by ring current ions. Pc-5 pulsations following Storm Sudden Commencements (SSCs) were examined separately. The spatial distribution of modes for the SSC events was consistent with the statistical study for the lifetime of CRRES. The toroidal fundamental (and harmonic) resonances are the dominant mode seen on the dawn-side of the magnetosphere following SSCs. Power is mixed in all three components. In the 21 dusk side SSC events there were only a few examples of purely compressional (two) or radial (one) power in the CRRES study, a few more examples of purely toroidal modes (six), with all three components predominant in about half (ten) of the events. Key words. Magnetospheric physics (MHD waves and instabilities; magnetospheric configuration and dynamics) – Space plasma physics (waves and instabilities)
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Sakurai, Tohru, Yutaka Tonegawa, Takuya Kitagawa, Kiyofumi Yumoto, Tatsundo Yamamoto, Susumu Kokubun, Toshifumi Mukai, and Koichiro Tsuruda. "Dayside magnetopause Pc 3 and Pc 5 ULF waves observed by the GEOTAIL Satellite." Earth, Planets and Space 51, no. 9 (September 1999): 965–78. http://dx.doi.org/10.1186/bf03351567.

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11

Vaghasiya, Bhavesh. "A Study Paper on 5 Pen PC Technology." INTERNATIONAL JOURNAL OF RESEARCH IN ADVANCE ENGINEERING 1, no. 1 (January 27, 2015): 5. http://dx.doi.org/10.26472/ijrae.v1i1.7.

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12

Madhusudhana, H. K., and Kripa Shanker. "FMS simulation model using PC-SIMSCRIPT-II.5." Computer Integrated Manufacturing Systems 3, no. 3 (August 1990): 150–56. http://dx.doi.org/10.1016/0951-5240(90)90127-z.

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13

Chen, Shih-Hsiung, Shiow-Shyung Lin, Dong-Jang Chang, and Jing-Song Chang. "Gas transport properties of CoAlPO4-5/PC membranes." Journal of Applied Polymer Science 77, no. 1 (July 5, 2000): 89–95. http://dx.doi.org/10.1002/(sici)1097-4628(20000705)77:1<89::aid-app12>3.0.co;2-5.

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14

Liu, Xin-Hua, Bao-An Song, Pinaki S. Bhadury, Hai-Liang Zhu, Ping Cui, Ke-Ke Hou, and Hong-Li Xu. "Novel 5-(3-(Substituted)-4,5-dihydroisoxazol-5-yl)-2-methoxyphenyl Derivatives: Synthesis and Anticancer Activity." Australian Journal of Chemistry 61, no. 11 (2008): 864. http://dx.doi.org/10.1071/ch07395.

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Thirty novel 5-(3-(substituted phenyl)-4,5-dihydroisoxazol-5-yl)-2-methoxyphenyl derivatives were synthesized and evaluated for their antitumour activity. The bioassays showed that the 2-fluorobenzoyl derivative 6ai, the 4-trifluoromethylbenzoyl derivative 6ah, and the 3-trifluoromethyl isoxazole derivatives (6ch and 6ci) were highly effective against PC-3 cells. The IC50 values of 6ah and 6ai against PC-3 cells were 1.5 and 1.8 μg mL–1, respectively.
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15

Buathong, Darinnat, Jarin Buakaew, and Chutarat Nukfon. "Comparison of Platelet Concentrates Prepared from Platelet Rich Plasma-Platelet Concentrate and Buffy Coat Poor-Platelet Concentrate on Storage Days 1 and 5." Songklanagarind Medical Journal 35, no. 2 (October 7, 2016): 5. http://dx.doi.org/10.31584/smj.2017.35.1.617.

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Objective: The purpose of this study is to assess the quality of platelet concentrates on storage days 1 and 5 prepared by platelet rich plasma-platelet concentrate (PRP-PC) and buffy coat poor-platelet concentrate (BC-PC) methods comparing to the American Association of Blood Banks (AABB) recommendations.Material and Method: Totally of 120 platelet concentrates (PC) units on storage days 1 and 5 (60 of PRP-PC triple blood bag and 60 of BC-PC quadruple AS-5 blood bag) were separated from whole blood donations at Songklanagarind Hospital. The prepared PC were assessed with 5 parameters such as volume, platelet count, white blood cell count per unit, pH, swirling phenomenon score and hypotonic shock response. The independent t-tests, paired Student’s t-tests and SPSS program were utilized in statistical analysis step.Results: The mean±standard deviation (S.D.) of each parameter were as follow : (1) Volume of PRP-PC and BC-PC met the standard (40-70 ml). (2) All of the platelet concentrates met the standard (≥ 5.5x1010/unit). The mean±S.D.: PRP-PC and BC-PC (day 1) were 6.820±1.480 x1010 and 7.010±1.300 x1010/unit (p-value=0.260), while PRP-PC and BC-PC (day 5) were 6.620±1.160x1010 and 6.720± 1.150x1010/unit (p-value=0.040). (3) The white blood cell in platelet concentrates met the standard (<0.2x1010/unit). The mean±S.D.: PRP-PC and BC-PC (day 1) were 0.030±0.017 x1010and 0.026±0.019x1010/unit (p-value=0.040), while PRP-PC and BC-PC (day 5) were0.033±0.013x1010 and 0.027±0.019x1010/unit (p-value= 0.580). (4) The pH of all units (PRP-PC and BC-PC) met the standard (≥6.2). The mean±S.D.: PRP-PC and BC-PC (day 1) were 7.430±0.330 and 7.750±0.160 (p-value=0.006), while PRP-PC and BC-PC (day 5) were 7.590±0.350 and 7.620±0.280 (p-value=0.710). The swirling phenomenon score and hypotonic shock response were the same as standard AABB and were not statistically difference.Conclusion: The quality of PRP-PC and BC-PC after storing on days 1 and 5 as follow (1) Volume of PRP-PC and BC-PC met the standard. (2) The platelet count per unit of PRP-PC and BC-PC (day 1), PRP-PC and BC-PC (day 5) were not statistically difference. (3) The white blood cell count per unit of PRP-PC and BC-PC (day 1) were statistically difference, while PRP-PC and BC-PC (day 5) were not statistically difference. (4) The pH of PRP-PC and BC-PC (day 1) were statistically difference, while PRP-PC and BC-PC (day 5) were not statistically difference. The swirling phenomenon score and hypotonic shock response of PRP-PC and BC-PC were not statistically difference. Platelet concentrates of both method storing on days 1 and 5 fulfilled the quality guideline of AABB.
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16

Radai, Yisrael. "The Israeli PC virus." Computers & Security 8, no. 2 (April 1989): 111–13. http://dx.doi.org/10.1016/0167-4048(89)90065-5.

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17

Höhl, Rebekka. "Praxis-PC vor Datendieben schützen." hautnah dermatologie 30, no. 6 (November 2014): 52. http://dx.doi.org/10.1007/s15012-014-1939-5.

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18

Gaskin, F. Spencer, Kazuhiro Kamada, Mozow Yusof, and Ronald J. Korthuis. "5′-AMP-activated protein kinase activation prevents postischemic leukocyte-endothelial cell adhesive interactions." American Journal of Physiology-Heart and Circulatory Physiology 292, no. 1 (January 2007): H326—H332. http://dx.doi.org/10.1152/ajpheart.00744.2006.

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Preconditioning (PC) with nitric oxide (NO) donors or agents that increase endothelial NO synthase (eNOS) activity 24 h before ischemia-reperfusion (I/R) prevents postischemic leukocyte rolling (LR) and stationary leukocyte adhesion (LA). Since 5′-AMP-activated protein kinase (AMPK) phosphorylates eNOS at Ser1177, resulting in activation, we postulated that AMPK activation may trigger the development of a preconditioned anti-inflammatory phenotype similar to that induced by NO donors. Wild-type (WT) C57BL/6J and eNOS−/− mice were treated with the AMPK agonist 5-aminoimidazole-4-carboxamide 1-β-d-furanoside (AICAR) 30 min (early AICAR PC) or 24 h (late AICAR PC) before I/R; LR and LA were quantified in single postcapillary venules in the jejunum using intravital microscopy. I/R induced comparable marked increases in LR and LA in WT and eNOS−/− mice relative to sham-operated (no ischemia) animals. Late AICAR PC prevented postischemic LR and LA, whereas early AICAR PC prevented LA in WT mice. Late AICAR PC was ineffective in preventing I/R-induced LR but not LA in the eNOS−/− mice, and the same pattern was seen in WT animals treated with the NOS inhibitor Nω-nitro-l-arginine. Early AICAR PC remained effective in preventing LA in eNOS−/− mice. Our results indicate that both early and late PC with an AMPK agonist produces an anti-inflammatory phenotype in postcapillary venules. Since the protection afforded by late AICAR PC on postischemic LR was prevented by NOS inhibition in WT mice and absent in eNOS-deficient mice, it appears that eNOS triggers this protective effect. In stark contrast, antecedent AMPK activation prevented I/R-induced LA by an eNOS-independent mechanism.
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19

Plyasova-Bakounina, T. A., J. Kangas, K. Mursula, O. A. Molchanov, and A. W. Green. "Pc 1-2 and Pc 4-5 pulsations observed at a network of high-latitude stations." Journal of Geophysical Research: Space Physics 101, A5 (May 1, 1996): 10965–73. http://dx.doi.org/10.1029/95ja03770.

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20

Lathuillere, C., F. Glangeaud, and Z. Y. Zhao. "Ionospheric ion heating by ULF Pc 5 magnetic pulsations." Journal of Geophysical Research 91, A2 (1986): 1619. http://dx.doi.org/10.1029/ja091ia02p01619.

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21

Tian, M., T. K. Yeoman, M. Lester, and T. B. Jones. "Statistics of Pc 5 pulsation events observed by SABRE." Planetary and Space Science 39, no. 9 (September 1991): 1239–47. http://dx.doi.org/10.1016/0032-0633(91)90037-b.

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22

Konarev, Dmitri V., Alexey V. Kuzmin, Sergey V. Simonov, Salavat S. Khasanov, and Rimma N. Lyubovskaya. "Structure and optical properties of fullerene C60 complex with dipyridinated iron(II) phthalocyanine [Fe(II)Pc(C5H5N)2]·C60·4C6H4Cl2: First structure of bisaxially coordinated iron(II) phthalocyanine complex with acetonitrile Fe(II)Pc(CH3CN)2." Journal of Porphyrins and Phthalocyanines 18, no. 01n02 (January 2014): 87–93. http://dx.doi.org/10.1142/s1088424613500624.

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The complex of fullerene C 60 with dipyridinated iron(II) phthalocyanine [ Fe ( II ) Pc ( C 5 H 5 N )2]· C 60·4 C 6 H 4 Cl 2 (1) has been obtained as single crystals. According to the IR- and UV-visible-NIR spectra, 1 is molecular solid with no charge transfer from Fe ( II ) Pc ( C 5 H 5 N )2 to C 60· C 60 molecules form closely packed linear columns in 1 along the b axis with a uniform interfullerene center-to center distance of 9.99 Å and multiple short van der Waals (vdW) C … C contacts between fullerenes of 3.10–3.18 Å. Totally each Fe ( II ) Pc ( C 5 H 5 N )2 unit is surrounded by four C 60 molecules two of which form short vdW C … C contacts with the phthalocyanine plane locating near two adjacent phenylene substituents of Fe ( II ) Pc . The Fe ( II ) Pc ( C 5 H 5 N )2 geometry remains almost unchanged as compared with that of fullerene free Fe ( II ) Pc ( C 5 H 5 N )2. The iron(II) atoms are located exactly in the Pc plane, the Fe - N ( C 5 H 5 N ) bond length is 2.038(3) Å and the averaged of the Fe - N ( Pc ) bond length is 1.935(3) Å. Bisaxially coordinated iron(II) phthalocyanine complex with acetonitrile Fe ( II ) Pc ( CH 3 CN )2 does not cocrystallize with C 60. Nevertheless, good quality crystals of [ Fe ( II ) Pc ( CH 3 CN )2]·2 C 6 H 4 Cl 2 (2) were isolated in this synthesis. That is the first structure of bisaxially coordinated metal phthalocyanine complex with nitrile containing solvent. Acetonitrile unusually strongly coordinates to Fe ( II ) Pc with the Fe – N ( CH 3 CN ) bond length of 1.938(1) Å. The iron(II) atoms are located in the Pc plane and the averaged length of the Fe - N ( Pc ) bonds is 1.934(1) Å.
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23

Milenković, Čedomir, Dušan Starčević, and Biljana Mučibabić. "PC-based multimedia messaging systems." Microprocessing and Microprogramming 21, no. 1-5 (August 1987): 119–26. http://dx.doi.org/10.1016/0165-6074(87)90027-5.

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24

Bohr, H. "Supercomputer accelerator card for PC." Microprocessing and Microprogramming 25, no. 1-5 (January 1989): 51–54. http://dx.doi.org/10.1016/0165-6074(89)90172-5.

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25

Miki, Takayuki, Tetsuji Miura, Rolf Bünger, Katsuo Suzuki, Jun Sakamoto, and Kazuaki Shimamoto. "Ecto-5′-nucleotidase is not required for ischemic preconditioning in rabbit myocardium in situ." American Journal of Physiology-Heart and Circulatory Physiology 275, no. 4 (October 1, 1998): H1329—H1337. http://dx.doi.org/10.1152/ajpheart.1998.275.4.h1329.

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This study tested the hypothesis that cardiac ecto-5′-nucleotidase (ecto-5′-NT) activity during ischemic preconditioning (PC) contributes to augmented tolerance against ischemia, thereby reducing infarct size in the rabbit heart in situ. The effects of α,β-methylene-adenosine diphosphate (AOPCP), a selective inhibitor of ecto-5′-NT, on cardiovascular responses to AMP were measured to establish in vivo activities of the enzyme and its inhibitor. Left atrial infusion of AOPCP (0.75 mg ⋅ kg−1⋅ min−1) raised AOPCP plasma levels to 138 μM; under these conditions negative chronotropic and inotropic effects of AMP were blocked, demonstrating essentially full inhibition of ecto-5′-NT in the heart in situ. This AOPCP-blocked heart in situ model was used to examine the proposed contribution of ecto-5′-NT in ischemic PC. Myocardial infarction caused by 30-min ischemia was followed by 3-h reperfusion. Infarct size (IS) was measured and expressed as a percentage of the size of the area at risk (%IS/AR). In untreated controls, %IS/AR was 38.1 ± 3.8%; PC (5-min ischemia, 5-min reperfusion) markedly reduced %IS/AR to 10.0 ± 2.0%. Essentially identical IS reductions by PC were observed in AOPCP-blocked animals (%IS/AR = 13.8 ± 2.2 and 13.3 ± 1.8% in rabbits receiving AOPCP at 0.75 and 1.50 mg ⋅ kg−1⋅ min−1, respectively); here plasma AOPCP levels were established before and during PC but not during the subsequent prolonged ischemia. As expected, AOPCP also did not affect %IS/AR in non-PC controls (%IS/AR = 35.5 ± 3.7%). In contrast but as predicted, adenosine-receptor blockade by 8-phenyltheophylline (10 mg/kg iv) substantially attenuated IS reduction by PC in both AOPCP-blocked and control hearts (%IS/AR = 25.2 ± 4.3 and 21.8 ± 2.2%, respectively; P < 0.05 vs. PC alone). The results demonstrate that cardiac ecto-5′-NT is not required for ischemic PC against infarction in the rabbit.
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26

Perez-Andres, Martin, Bruno Paiva, Leandro Thiago, Nico A. Bos, Dirk Hose, Bernard Klein, Surinder S. Sahota, et al. "Immunophenotypic Analysis of Myeloma Precursors: Antigens for Therapeutic Targeting." Blood 116, no. 21 (November 19, 2010): SCI—5—SCI—5. http://dx.doi.org/10.1182/blood.v116.21.sci-5.sci-5.

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Abstract Abstract SCI-5 Multiple myeloma (MM) is a malignant disorder characterized by the (mono)clonal expansion of terminally-differentiated plasma cells (M-PC) in the bone marrow (BM) that produce and secrete a monoclonal immunoglobulin (Ig), detectable in the serum and/or urine. Infiltration by the expanded M-PC is easily identified in the involved tissues, through conventional morphology and immunophenotyping. However, the possibility exists that rather than M-PC, a less differentiated B-cell that represents a minor fraction of all tumor cells and retains self-renewal properties, is responsible for the outgrowth of the more differentiated M-PC compartment. In B-cell disorders, the idiotypic Ig produced by tumor cells and defined by its CDR3 sequence, acts as a genetic fingerprint for clonally-related B-cells. In MM, the specificity of the idiotype is further enhanced through modification of V genes by somatic hypermutation (SHM) at the germinal center (GC), since M-PC display extensively mutated VH genes which are stable throughout the disease. This suggests that in MM, malignant transformation could occur in a post-GC B-cell. Based on the CDR3 sequences of the Ig genes of M-PC, preliminary studies have identified tumor-associated circulating peripheral blood (PB) CD19+ B-cells, whose malignant/clonogenic potential remained to be demonstrated. More recently, the Matsui group has reported that while in MM cell lines both CD138− and CD138+ cells retain clonogenic capacity after in vitro serial plating clonogenic assays, in primary MM samples, such (ex vivo and in vivo) ability would be restricted to the CD34−/CD138− compartment. Of note, the clonogenic growth of these later cells significantly decreased after depletion by CD19, CD22, CD20 and CD45 antibodies, and it was associated with an in vitro multidrug-resistant functional phenotype (restricted to CD19+/CD27+, CD138− cells but not CD138+ cells) and the Hedgehog (Hh) stem cell-associated signaling pathway. These results point out the potential existence of a CD19+, CD20+, CD138− pre-PC compartment responsible for the expansion of M-PC in MM. In turn, evidence also exists in both the SCID-Hu model and in Rituximab treated MM patients, which suggests that plasmablasts/PC -but not pre-plasmablasts-, could act as MM “stem” cells, the precise characteristics of such cells remaining to be precisely defined. Alternatively, it could also be possible that both cell cellular components coexist and are relevant to MM progression through appropriate interaction with the BM stroma. Independently of all the above, trafficking of such cells through PB to BM niches could also play a key role in the spread of the tumor and its malignant behavior. In this regard, we recently confirmed that a relatively high percentage of MM patients (and a substantial fraction of all MGUS cases) show circulating PB PC with i) tumor-related clonal VH gene rearrangements and ii) an aberrant immunophenotypic profile which largely overlaps with that of BM M-PC from the same subjects; the only minor differences consisted of a significantly lower expression of CD38 and CD138, smaller size and internal complexity, features that indicate a slightly more immature plasmablastic/PC profile. Noteworthy, this was the only PB B-cell compartment for which clonally-related B-cells were detected with a sensitivity of <1 cell/50μ L in all cases investigated. In summary, accumulating evidence suggest the existence of a clonal hierarchy in MM but uncertainties remain as regards the precise immunophenotypic features of those cells responsible for tumor growth in primary patient samples, that could be of help in developing new targeted therapies. Disclosures: Sonneveld: Millennium: Consultancy; Celgene: Consultancy. Orfao:Becton/Dickinson Biosciences Europe: Patents & Royalties, Research Funding; Cytognos SL: Patents & Royalties; Alexion: Membership on an entity's Board of Directors or advisory committees; Vivia Biotech: Research Funding; Mundipharma: Research Funding.
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27

Tran, Ngoc-Thien, and Nga Thi-Hong Pham. "Investigation of the Effect of Polycarbonate Rate on Mechanical Properties of Polybutylene Terephthalate/Polycarbonate Blends." International Journal of Polymer Science 2021 (August 14, 2021): 1–7. http://dx.doi.org/10.1155/2021/7635048.

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Polybutylene terephthalate (PBT) is a brittle polymer with the disadvantage of low impact toughness, so it is not easy to meet the requirements of both high tensile strength, flexural strength, and high impact strength. In this study, PBT/polycarbonate (PC) blends at different ratios of 95/5, 90/10, 85/15, and 80/20 are investigated. Tensile strength, flexural strength, and unnotched Izod impact strength are studied according to the ASTM D638, ASTM D790, and ASTM D256 standards. The results show that tensile strength, which increased with increasing PC content, is 53.00, 62.34, 60.59, 62.98, and 64.46 MPa for 0, 5, 10, 15, and 20% PC samples. Flexural strength and elastic flexural testing of PBT/PC blends are higher than neat PBT. In addition, the unnotched Izod impact strength of PBT/PC is also higher than PBT. However, when PC content increases, impact strength tends to decrease. Impact strength is 44.82, 80.46, 68.82, 50.45, and 48.05 kJ/m2 corresponds to 0, 5, 10, 15, and 20% PC, in which 5% PC sample is twice as high as the impact strength of PBT. Microstructure of the blends has shown that PC has become dispersed phase in PBT matrix. The size and quantity of dispersed PC particles increase with increasing PC rate in the blend. Thus, when adding PC, PBT/PC all meet the requirements of high tensile strength, flexural strength, and high impact strength. The PBT/5% PC model gives the highest impact strength while still ensuring durability, which potential application for making car door handles.
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28

HOUWELING, Martin, Wil KLEIN, and Math J. H. GEELEN. "Regulation of phosphatidylcholine and phosphatidylethanolamine synthesis in rat hepatocytes by 5-aminoimidazole-4-carboxamide ribonucleoside (AICAR)." Biochemical Journal 362, no. 1 (February 8, 2002): 97–104. http://dx.doi.org/10.1042/bj3620097.

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The present study was undertaken to study the role of AMP-activated kinase (AMPK) in the biosynthesis of two major membrane phospholipids, phosphatidylcholine (PC) and phosphatidylethanolamine (PE). Incubation of rat hepatocytes with 5-aminoimidazole-4-carboxamide ribonucleoside (AICAR), an activator of AMPK, produced dose-dependent inhibition of the incorporation of [3H]choline and [3H]ethanolamine into PC and PE, respectively. Determination of the cellular uptake of choline and ethanolamine showed that the reduced synthesis of PC and PE did not result from impaired uptake of these two precursors. The decreased synthesis of PC was not mirrored by a reduction in the activities of the enzymes of the CDP-choline pathway. The diminution of PE biosynthesis, however, was paralleled by a depressed activity of CTP:phosphoethanolamine cytidylyltransferase (ET), the pace-setting enzyme of the CDP-ethanolamine pathway. AICAR treatment of hepatocytes stimulated the conversion of choline into betaine, indicating that reduced PC synthesis most probably resulted from a decrease in the availability of choline. In addition, AICAR induced a 50% reduction in the cellular level of diacylglycerols, which may further impair the synthesis of PC and PE. The results thus indicate that AICAR inhibits the biosynthesis of PC and PE and that the effect is exerted at different sites in the two pathways. Increased oxidation of choline to betaine is the main target of AICAR in the PC pathway, whereas inhibition of ET activity is the locus of AICAR action in the PE pathway.
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29

Torres, F., P. Gil, S. T. Puente, J. Pomares, and R. Aracil. "Automatic PC disassembly for component recovery." International Journal of Advanced Manufacturing Technology 23, no. 1-2 (January 1, 2004): 39–46. http://dx.doi.org/10.1007/s00170-003-1590-5.

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30

Jida, M., H. Yamamoto, T. Kubo, T. Hayashi, H. Otani, Y. Sano, and S. Toyooka. "Establishment and molecular characteristics of PC-9 derived erlotinib resistant cell lines." Journal of Clinical Oncology 27, no. 15_suppl (May 20, 2009): e22193-e22193. http://dx.doi.org/10.1200/jco.2009.27.15_suppl.e22193.

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e22193 Background: EGFR mutation is closely associated with tumor responsiveness to EGFR tyrosine kinase inhibitors (TKIs), gefitinib or erlotinib. However, many non-small cell lung cancer (NSCLC) cases with EGFR mutation that had responded to EGFR-TKIs finally acquire resistance to EGFR-TKIs. It has been reported that T790M mutation and MET amplification caused resistance to EGFR-TKIs . Recent study suggested that overexpression of HGF was also related to resistance to EGFR-TKIs. However, the whole mechanism of acquired resistance to TKIs is still not fully known. Methods: We established 5 clones of elrotinib resistant PC-9 cell line (harboring EGFR exon 19 deletion) (designated as PC-9ER1–5) by exposing PC-9 to low-dose erlotinib. The protein expression of EGFR-related molecules was examined by Western blotting. Array comparative genomic hybridization (aCGH) or real time quantitative PCR (qPCR) assay was performed to examine copy numbers of EGFR-related genes. Results: In MTS assay, the IC50 value for the parental PC-9 cells was 0.02 μmol/L. By contrast, those for PC-9ER1–5 were more than 33μmol/L. All 5 resistant cell lines retained exon19 deletions and did not obtain T790M mutation. The qPCR assay and aCGH showed no MET amplification in PC-9 or PC-9ER1–5. HGF protein was not overexpressed in PC-9ER1–5 by ELISA. The protein expressions of several molecules in the EGFR-Akt signaling pathway were examined by western blotting after erlotinib treatment. Although phospho-EGFR was suppressed in both PC-9 and PC-9ER1–5 with 2-μmol/L erlotinib, phospho-Akt was not suppressed in PC-9ER1–5. PTEN expression was not down-regulated in PC-9ER1–5. The combination of erlotinib and PHA-665752, MET tyrosine kinase inhibitor, did not suppress phospho-Akt or cell proliferation in PC-9ER1–5. Conclusions: As common molecular features of our EGFR-TKI resistant cell lines, phospho-Akt was not suppressed with exposure to erlotinib. These cell lines did not show previously reported features of resistant cell lines including T790M mutation, MET amplification or HGF overexpression. Our results indicated that other mechanisms leading to Akt activation caused resistance to EGFR-TKIs. No significant financial relationships to disclose.
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31

Takano, Hitoshi, Xian-Liang Tang, and Roberto Bolli. "Differential role of KATP channels in late preconditioning against myocardial stunning and infarction in rabbits." American Journal of Physiology-Heart and Circulatory Physiology 279, no. 5 (November 1, 2000): H2350—H2359. http://dx.doi.org/10.1152/ajpheart.2000.279.5.h2350.

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The role of ATP-sensitive potassium (KATP) channels in the late phase of ischemic preconditioning (PC) remains unclear. Furthermore, it is unknown whether KATP channels serve as end effectors both for late PC against infarction and against stunning. Thus, in phase I of this study, conscious rabbits underwent a 30-min coronary occlusion (O) followed by 72 h of reperfusion (R) with or without ischemic PC (6 4-min O/4-min R cycles) 24 h earlier. Late PC reduced infarct size ∼46% versus controls. The KATPchannel blocker 5-hydroxydecanoic acid (5-HD), given 5 min before the 30-min O, abrogated the infarct-sparing effect of late PC but did not alter infarct size in non-PC rabbits. In phase II, rabbits underwent six 4-min O/4-min R cycles for 3 consecutive days ( days 1, 2, and 3). In controls, the total deficit of systolic wall thickening (WTh) after the sixth reperfusion was reduced by 46% on day 2 and 54% on day 3compared with day 1, indicating a late PC effect against myocardial stunning. Neither 5-HD nor glibenclamide, given on day 2, abrogated late PC. The KATP channel opener diazoxide, given on day 1, attenuated stunning, and this effect was completely blocked by 5-HD. Thus the same dose of 5-HD that blocked the antistunning effect of diazoxide failed to block the antistunning effects of late PC. Furthermore, when diazoxide was administered in PC rabbits on day 2, myocardial stunning was further attenuated, indicating that diazoxide and late PC have additive anti-stunning effects. We conclude that KATP channels play an essential role in late PC against infarction but not in late PC against stunning, revealing an important pathogenetic difference between these two forms of cardioprotection.
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32

Feigenson, Gerald W., and Jeffrey T. Buboltz. "Ternary Phase Diagram of Dipalmitoyl-PC/Dilauroyl-PC/Cholesterol: Nanoscopic Domain Formation Driven by Cholesterol." Biophysical Journal 80, no. 6 (June 2001): 2775–88. http://dx.doi.org/10.1016/s0006-3495(01)76245-5.

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33

Ohnuma, Yoshito, Tetsuji Miura, Takayuki Miki, Masaya Tanno, Atsushi Kuno, Akihito Tsuchida, and Kazuaki Shimamoto. "Opening of mitochondrial KATP channel occurs downstream of PKC-ε activation in the mechanism of preconditioning." American Journal of Physiology-Heart and Circulatory Physiology 283, no. 1 (July 1, 2002): H440—H447. http://dx.doi.org/10.1152/ajpheart.00434.2001.

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We examined whether the mitochondrial ATP-sensitive K channel (KATP) is an effector downstream of protein kinase C-ε (PKC-ε) in the mechanism of preconditioning (PC) in isolated rabbit hearts. PC with two cycles of 5-min ischemia/5-min reperfusion before 30-min global ischemia reduced infarction from 50.3 ± 6.8% of the left ventricle to 20.3 ± 3.7%. PC significantly increased PKC-ε protein in the particulate fraction from 51 ± 4% of the total to 60 ± 4%, whereas no translocation was observed for PKC-δ and PKC-α. In mitochondria separated from the other particulate fractions, PC increased the PKC-ε level by 50%. Infusion of 5-hydroxydecanoate (5-HD), a mitochondrial KATP blocker, after PC abolished the cardioprotection of PC, whereas PKC-ε translocation by PC was not interfered with 5-HD. Diazoxide, a mitochondrial KATP opener, infused 10 min before ischemia limited infarct size to 5.2 ± 1.4%, but this agent neither translocated PKC-ε by itself nor accelerated PKC-ε translocation after ischemia. Together with the results of earlier studies showing mitochondrial KATP opening by PKC, the present results suggest that mitochondrial KATP-mediated cardioprotection occurs subsequent to PKC-ε activation by PC.
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34

JIANG, JIANZHUANG, JINGLEI XIE, MICHAEL T. M. CHOI, YAN YAN, SIXIU SUN, and DENNIS K. P. NG. "Double-decker Yttrium(III) Complexes with Phthalocyaninato and Porphyrinato Ligands." Journal of Porphyrins and Phthalocyanines 03, no. 04 (April 1999): 322–28. http://dx.doi.org/10.1002/(sici)1099-1409(199904)3:4<322::aid-jpp137>3.0.co;2-6.

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A series of new yttrium(III) double-decker complexes with the same and different phthalocyaninato and porphyrinato ligands has been prepared. The homoleptic complexes Y ( P )2 ( P = Pc *(C7 H 15)8, Pc *( OC 5 H 11)8; Pc * = 2,3,9,10,16,17,23,24-octasubstituted phthalocyaninate) have been synthesized by treating yttrium(III) acetylacetonate ( Y ( acac )3· H 2 O ) with the phthalonitriles C 6 H 2 R 2( CN )2 ( R = C 7 H 15, OC 5 H 11) in the presence of 1,8-diazabicyclo[5.4.0]undec-7-ene (DBU). The heteroleptic analogues Y ( P )( Pc ) ( P = Pc *( C 7 H 15)8, Pc *( OC 5 H 11)8; Pc = unsubstituted phthalocyaninate) and Y ( P )( Por ) ( P = Pc , Pc *( C 7 H 15)8; Por = meso-tetraphenylporphyrinate (TPP), meso-tetra(4-pyridyl)porphyrinate (TPyP)) have been prepared by the base-catalysed tetramerization of the corresponding phthalonitriles using Y ( P )( acac ) ( P = Pc , TPP , TPyP ) as templates. The syntheses along with the spectroscopic and electrochemical properties of these novel double-deckers are described.
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35

Bertolini, F., F. Marangoni, A. Loy, M. Marconi, D. Almini, P. Rebulla, and G. Sirchia. "Single-donor Platelet Concentrates Stored in Synthetic Medium. In Vitro and in Vivo Studies." International Journal of Artificial Organs 16, no. 5_suppl (May 1993): 135–38. http://dx.doi.org/10.1177/039139889301605s27.

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Single-donor platelet concentrates (PC) were prepared in 80-120 ml plasma and stored in two polyolefin bags after addition of 250 ml plasmalyte, a simple, glucose-free synthetic medium that was previously used for platelet storage; when compared to PC stored in plasma, PC stored in plasmalyte, showed similar platelet quality, morphology and function after 5 days of storage. In vivo incrementes observed after transfusion of PC stored for 5 days in plasmalyte were similar to those observed after transfusion of 1-2 day old PC stored in plasma. Moreover, transfusion of 5-day old PC stored in plasmalyte was associated with correction of prolonged bleeding times in all 3 of the 3 patients evaluated. It is concluded that plasmalyte seems to be promising as a medium for single-donor PC storage.
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36

Grandin, Ulf. "PC-ORD version 5: A user-friendly toolbox for ecologists." Journal of Vegetation Science 17, no. 6 (February 24, 2006): 843–44. http://dx.doi.org/10.1111/j.1654-1103.2006.tb02508.x.

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37

Zhang, Peng, Hung-Yen Chou, Pan Zheng, and Yang Liu. "PC-5 En masse discovery of human anti-cancer antibodies." JAIDS Journal of Acquired Immune Deficiency Syndromes 81 (April 2019): 59. http://dx.doi.org/10.1097/01.qai.0000558008.69257.ab.

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38

Takahashi, K., L. J. Zanetti, T. A. Potemra, and M. H. Acuña. "A model for the harmonic of compressional Pc 5 waves." Geophysical Research Letters 14, no. 4 (April 1987): 363–66. http://dx.doi.org/10.1029/gl014i004p00363.

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39

Waite, J. H., D. L. Gallagher, M. O. Chandler, R. C. Olsen, R. H. Comfort, J. F. E. Johnson, C. R. Chappell, W. K. Peterson, D. Weimer, and S. D. Shawhan. "Plasma and field observations of a Pc 5 wave event." Journal of Geophysical Research 91, A10 (1986): 11147. http://dx.doi.org/10.1029/ja091ia10p11147.

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40

Zhang, Yaping, Dong Wang, Ronglan Zhang, Jianshe Zhao, and Ying Zheng. "ZSM-5-Ln(Pc)2 catalyzed oxygen oxidation of thiophene." Catalysis Communications 29 (December 2012): 21–23. http://dx.doi.org/10.1016/j.catcom.2012.09.023.

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41

Cave, A. C., and P. B. Garlick. "Ischemic preconditioning and intracellular pH: a 31P NMR study in the isolated rat heart." American Journal of Physiology-Heart and Circulatory Physiology 272, no. 1 (January 1, 1997): H544—H552. http://dx.doi.org/10.1152/ajpheart.1997.272.1.h544.

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The aim of these studies was to investigate whether manipulation of intracellular pH affects preconditioning in isolated buffer-perfused rat hearts. Control and preconditioned [PC; 3 min of ischemia (I) + 3 min of reperfusion (R) + 5 min of I + 5 min of R or 4 x (5 min of I + 5 min of R)] hearts were subjected to two different protocols expected to alter intracellular pH during the sustained ischemic insult: 1) increased extracellular buffering capacity with the addition of 25 mM N-2-hydroxyethylpiperazine-N'-2-ethanesulfonic acid (HEPES) to the buffer to alleviate acidosis and 2) increased preischemic glycogen content to exacerbate acidosis. All hearts were subjected to 40 min of I + 40 min of R. 31P nuclear magnetic resonance was used to measure ATP, phosphocreatine, Pi, and intracellular pH. Despite a significantly better recovery of function in all PC groups, there were no significant differences in intracellular pH (rate-pressure product = 60 +/- 5, 66 +/- 10, 42 +/- 5, and 57 +/- 8% of baseline in PC, 4 x 5 PC, PC + HEPES, and PC fasted hearts, respectively, compared with 36 +/- 9, 17 +/- 7, and 20 +/- 10% of baseline in control, control + HEPES, and control fasted hearts, respectively; pH at end ischemia: control, 6.31 +/- 0.02; PC, 6.35 +/- 0.03; 4 x 5 PC, 6.35 +/- 0.04; control + HEPES, 6.40 +/- 0.10; PC + HEPES, 6.56 +/- 0.07: control fasted, 6.46 +/- 0.03; PC fasted, 6.43 +/- 0.01). No significant differences were observed among groups in ATP, phosphocreatine, or Pi on reperfusion. Thus the mechanism of preconditioning in glucose-perfused hearts does not depend on an alleviation of intracellular acidosis during the sustained ischemic period. Furthermore, under the conditions of this study, intracellular pH during ischemia did not predict functional recovery on reperfusion.
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42

Bloom, Ronald M., and Howard J. Singer. "Diurnal trends in geomagnetic noise power in the Pc 2 through Pc 5 bands at low geomagnetic latitudes." Journal of Geophysical Research 100, A8 (1995): 14943. http://dx.doi.org/10.1029/95ja01332.

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43

Konarev, Dmitri V., Alexey V. Kuzmin, Salavat S. Khasanov, and Rimma N. Lyubovskaya. "The molecular structure of high-spin (S = {\bf 5} \over {\bf 2}) manganese(II) phthalocyanine in tetrabutylammonium bromido(phthalocyaninato)manganese(II)." Acta Crystallographica Section C Structural Chemistry 70, no. 5 (April 10, 2014): 449–51. http://dx.doi.org/10.1107/s2053229614007475.

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The title complex salt, (C16H36N)[MnBr(C32H16N8)] or (TBA)[MnIIBr(Pc)] (TBA is tetrabutylammonium and Pc is phthalocyaninate), has been obtained as single crystals by the diffusion technique and its crystal structure was determined using X-ray diffraction. The high-spin (S = 5\over 2) [MnIIBr(Pc)]− macrocycle has a concave conformation, with an average equatorial Mn—N(Pc) bond length of 2.1187 (19) Å, an axial Mn—Br bond length of 2.5493 (7) Å and with the MnII cation displaced out of the 24-atom Pc plane by 0.894 (2) Å. The geometry of the MnIIN4 fragment in [MnIIBr(Pc)]− is similar to that of the high-spin (S = 5\over 2) manganese(II) tetraphenylporphyrin (TPP) in [MnII(1-MeIm)(TPP)] (1-MeIm is 1-methylimidazole).
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44

Petyaev, Ivan M., Pavel Y. Dovgalevsky, Natalia E. Chalyk, Victor A. Klochkov, Nigel H. Kyle, and Yuriy K. Bashmakov. "Reduction of Liver Span and Parameters of Inflammation in Nonalcoholic Fatty Liver Disease Patients Treated with Lycosome Formulation of Phosphatidylcholine: A Preliminary Report." International Journal of Chronic Diseases 2018 (2018): 1–7. http://dx.doi.org/10.1155/2018/4549614.

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Twenty-nine newly diagnosed individuals with Nonalcoholic Fatty Liver Disease (NAFLD) remaining on habitual dietary regimen were supplemented with regular or lycosome formulations of phosphatidylcholine (PC) during a pilot, randomized, double-blinded clinical study. After two months of oral PC intake (450 mg daily) the liver size as well as serum levels of hepatic enzymes and markers of inflammation were evaluated by ultrasonography and biochemical analysis. It was shown that there was a statistically significant reduction of medians for the Mid-Clavicular liver size from 16.0 cm (95/5% CI: 17.1/15.5) to 15.1 cm (95/5% CI: 17.2/14.4, P=0.021) in participants ingesting the lycosome-formulated PC (L-PC) whereas regular formulation of PC (R-PC) had only a marginal effect on this parameter (P=0.044). A similar tendency was observed in the Mid-Sternal liver size. Moreover, there was a reduction of medians for ALT values at the end point of the study (P=0.026) after ingestion of L-PC, while R-PC had no statistically significant effect. On the other hand, ingestion of both formulations was accompanied by reductions in values for Inflammatory Oxidative Damage (IOD) and oxidized LDL in serum. However, L-PC had superior activity in these terms, presumably due to the presence of lycopene, a powerful antioxidant, in the L-PC-Lycosome structure. C-reactive protein level was moderately decreased (reduction of medians from 6.5 [95/5% CI: 7.7/5.8] mg/L to 5.1 [95/5% CI: 5.6/4.3] mg/L) only after ingestion of L-PC. The greater efficacy of L-PC seen in NAFLD volunteers may reflect improved bioavailability of PC owing to better protection of the microencapsulated PC from gastrointestinal enzymes and possibly enhanced hepatic delivery of L-PC particles.
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45

Loto'aniu, T. M., B. J. Fraser, and C. L. Waters. "The modulation of electromagnetic ion cyclotron waves by Pc 5 ULF waves." Annales Geophysicae 27, no. 1 (January 7, 2009): 121–30. http://dx.doi.org/10.5194/angeo-27-121-2009.

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Abstract. The modulation of electromagnetic ion cyclotron (EMIC) waves by longer-period ULF waves has been proposed as a method for producing pearl structured Pc 1–2 EMIC waves. This study examines frequency and phase relationship between Pc 1 EMIC wavepacket envelopes and simultaneously occurring Pc 5 ULF waves using magnetic data measured by the CRRES spacecraft. Intervals from three days in 1991 where CRRES observed pearls are presented along with simple statistics for 58 EMIC wavepackets. The observations were dominated by EMIC waves propagating away from the equatorial region. Comparisons between pearl wavepacket envelopes and Pc 5 waves show excellent agreement. The pearl wavepacket duration times, τdur, were statistically correlated with Pc 5 wave periods, TPc5, resulting in a correlation coefficient of R=0.7 and best fit equation τdur=0.8·TPc5+6 s. In general, phase differences varied although time intervals of constant in-phase or anti-phase correlation were observed. Anti-phase modulation may be explained by a decreasing background magnetic field due to the negative cycle of the ULF wave decreasing Alfvén velocity and minimum resonant energy. In-phase modulation could be the result of adiabatic modulation of temperature anisotropy in-phase with variations in the background field. Non-adiabatic processes may contribute to intervals that showed varying phase differences with time. Results suggest that future theoretical developments should take into account the full range of possible wave particle interactions inside the magnetosphere.
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Pan, Xin, Sihua Wu, Deshan Yao, Lian Liu, Lina Zhang, Zixuan Yao, Yan Pan, Siyuan Chang, and Bingfeng Li. "Efficient biotransformation of 5-hydroxymethylfurfural to 5-hydroxymethyl-2-furancarboxylic acid by a new whole-cell biocatalyst Pseudomonas aeruginosa PC-1." Reaction Chemistry & Engineering 5, no. 8 (2020): 1397–404. http://dx.doi.org/10.1039/d0re00018c.

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47

Tian, Juan, Hua Wen, Xing Lu, Wei Liu, Fan Wu, Chang-Geng Yang, Ming Jiang, and Li-Juan Yu. "Dietary phosphatidylcholine impacts on growth performance and lipid metabolism in adult Genetically Improved Farmed Tilapia (GIFT) strain of Nile tilapia Oreochromis niloticus." British Journal of Nutrition 119, no. 1 (December 11, 2017): 12–21. http://dx.doi.org/10.1017/s0007114517003063.

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AbstractThis study aimed to determine the effects of supplementing the diet of adult Nile tilapia Oreochromis niloticus with phosphatidylcholine (PC) on growth performance, body composition, fatty acid composition and gene expression. Genetically Improved Farmed Tilapia fish with an initial body weight of 83·1 (sd 2·9) g were divided into six groups. Each group was hand-fed a semi-purified diet containing 1·7 (control diet), 4·0, 6·5, 11·5, 21·3 or 41·0 g PC/kg diet for 68 d. Supplemental PC improved the feed efficiency rate, which was highest in the 11·5 g PC/kg diet. Weight gain and specific growth rate were unaffected. Dietary PC increased PC content in the liver and decreased crude fat content in the liver, viscera and body. SFA and MUFA increased and PUFA decreased in muscle with increasing dietary PC. Cytoplasmic phospholipase A2 and secreted phospholipase A2 mRNA expression were up-regulated in the brain and heart in PC-supplemented fish. PC reduced fatty acid synthase mRNA expression in the liver and visceral tissue but increased expression in muscle. Hormone-sensitive lipase and lipoprotein lipase expression increased in the liver with increasing dietary PC. Growth hormone mRNA expression was reduced in the brain and insulin-like growth factor-1 mRNA expression in liver reduced with PC above 6·5 g/kg. Our results demonstrate that dietary supplementation with PC improves feed efficiency and reduces liver fat in adult Nile tilapia, without increasing weight gain, representing a novel dietary approach to reduce feed requirements and improve the health of Nile tilapia.
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McCarthy, Michael. "Do “PC” MDs threaten health care?" Lancet 358, no. 9289 (October 2001): 1246. http://dx.doi.org/10.1016/s0140-6736(01)06387-5.

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49

Earnshaw, Rae A., and Basem El-Haddadeh. "PC graphics: What to look for." Physics World 7, no. 5 (May 1994): 47–52. http://dx.doi.org/10.1088/2058-7058/7/5/30.

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50

Vogel, Mark. "Review of SPSS/PC + statistical package." Computational Statistics & Data Analysis 6, no. 1 (January 1988): 71–84. http://dx.doi.org/10.1016/0167-9473(88)90065-5.

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