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Relevant bibliographies by topics / Pathophysiology / Dissertations / Theses

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Dissertations / Theses on the topic 'Pathophysiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Farrán, Díaz-Cano Aina. "Pathophysiology of osteoarthritis." Doctoral thesis, Universitat de Barcelona, 2017. http://hdl.handle.net/10803/456376.

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Osteoarthritis (OA) is a multifactorial disease characterized by a progressive degeneration and eventual failure of the synovial joint functionality. Although it has been traditionally considered as an exclusive disease of the articular cartilage, nowadays it is considered as a whole joint disease. Therefore, the progression of the disease involves articular cartilage degeneration, osteochondral bone sclerosis and synovial membrane hypertrophy. Articular cartilage is a connective tissue resistant to tensile and shears strength that is composed of water (>70%) and a dense extracellular matrix (

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2

Stoodley, Marcus A. "Pathophysiology of Syringomyelia /." Title page, contents and abstract only, 1996. http://web4.library.adelaide.edu.au/theses/09PH/09phs882.pdf.

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3

Robinson, Monique Renee. "Cardiac pathophysiology of obesity." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.414224.

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4

Padera, Timothy P. (Timothy Patrick) 1975. "Lymphatic pathophysiology of tumors." Thesis, Massachusetts Institute of Technology, 2003. http://hdl.handle.net/1721.1/29591.

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Thesis (Ph. D.)--Harvard-MIT Division of Health Sciences and Technology, 2003.<br>Includes bibliographical references (leaves 146-166).<br>Lymph node metastases have a negative impact on cancer survival, but the mechanisms for lymphatic metastasis are not well understood. The universal finding in solid tumors of an absence of functional lymphatic vessels seems paradoxical, as cancer cells do travel through lymphatics in order to disseminate. In order to address some of these issues, this thesis proposes two etiologies for the absence of functional lymphatic vessels in solid tumors. The first h

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5

Fletcher, Jessica Louise. "Pathophysiology of canine fucosidosis." Thesis, The University of Sydney, 2013. http://hdl.handle.net/2123/10420.

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Pathophysiology of Canine Fucosidosis by Jessica L Fletcher Understanding processes involved in the onset and progression of fucosidosis, a childhood neurodegenerative lysosomal storage disease, is critical to the development of effective treatment. Fucosidosis is caused by deficiency of the lysosomal hydrolase α-L-fucosidase due to inherited mutations in the FUCA1 gene. Canine fucosidosis, the only preserved animal model, was used to characterise preclinical pathophysiology, to establish potential markers of disease progression and test their capacity to measure responses to early therapy. S

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6

Kirk, Calum Norman Robert. "Pathophysiology of anoctaminopathy (LGMD2L)." Thesis, University of Newcastle upon Tyne, 2017. http://hdl.handle.net/10443/3861.

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Ageing is a natural process, which is characterised by progressive decline in physiological functions and increased susceptibility to disease and death. Brain is particularly susceptible to structural and functional changes, which is more evident in disorders associated with ageing such as Alzheimer disease (AD). Copper is necessary for the protection against oxidative stress, energy production and neurotransmitter processing in the brain. However, higher copper levels can increase oxidative stress, resulting in neuronal damage. In order to avoid copper induced cytotoxicity, cells have to regu

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7

Cheng, Allen Cheuk-Seng, and allencheng@ozemail com au. "MELIOIDOSIS: EPIDEMIOLOGY, PATHOPHYSIOLOGY AND MANAGEMENT." Flinders University. Medicine, 2005. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20051121.141305.

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In under a century, melioidosis, the infection due to Burkholderia pseudomallei, has emerged from Whitmores series of glanders-like infections amongst the morphia addicts in Burma to a major cause of mortality in northeastern Thailand and northern Australia. Also endemic in other parts of south-east Asia, melioidosis may have varied presentations ranging from severe, overwhelming infection to chronic, low grade disease. Observational evidence had suggested that granulocyte colony stimulating factor (G-CSF), a naturally occurring substance produced by the body in response to infection, may h

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8

Berg, Erika L. "Endocrinology of equine metabolic pathophysiology." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4472.

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Thesis (Ph. D.) University of Missouri-Columbia, 2006.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on July 31, 2007) Includes bibliographical references.

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9

Sethia, Krishna Kumar. "The pathophysiology of detrusor instability." Thesis, University of Oxford, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.235958.

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10

Steele, Ian Conrad. "Pathophysiology of chronic cardiac failure." Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337046.

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11

Ong, Patricia Mei Lin. "Pathophysiology of acute intermittent porphyria." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318116.

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12

Rome, Susan Margaret. "On the pathophysiology of dystonia." Thesis, University of Sheffield, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.369890.

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13

Read, Simon James. "Free radical pathophysiology in migraine." Thesis, University of Hertfordshire, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391365.

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14

Yellon, Derek M. "The pathophysiology of myocardial protection." Thesis, University of Bath, 1994. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.760654.

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15

Loosemore, Michael. "The pathophysiology of boxing injuries." Thesis, University of Bath, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.687310.

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This thesis explores the prevalence, nature and pathogenesis of injuries in boxing. Following an introductory chapter and literature review (Chapters 1 and 2 respectively); Chapter 3 examines injuries in the GB boxing squad from 2005 to 2009. There were a total of 66 boxers on the squad during this period 61% were injured, a total of 297 injuries were recorded. The injury rate in competition was at least 460 times higher than in training, and most injuries were new rather than recurrent (246 v 51). The incidence of concussion is comparatively low compared to other studies in amateur boxing (5

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16

Borazjani, Ali. "Pathophysiology of Pelvic Organ Prolapse." Cleveland State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=csu1432745397.

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17

Owler, Brian Kenneth. "Pathophysiology of normal pressure hydrocephalus." Thesis, The University of Sydney, 2004. http://hdl.handle.net/2123/685.

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Normal pressure hydrocephalus (NPH), a CSF circulation disorder, is important as a reversible cause of gait and cognitive disturbance in an aging population. The inconsistent response to CSF shunting is usually attributed to difficulties in differential diagnosis or co-morbidity. Improving outcome depends on an increased understanding of the pathophysiology of NPH. Specifically, this thesis examines the contribution of, and inter-relationship between, the brain parenchyma and CSF circulation in the pathophysiology of NPH. Of the four core studies of the thesis, the first quantifies the chara

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18

Owler, Brian Kenneth. "Pathophysiology of normal pressure hydrocephalus." University of Sydney. Surgery, 2004. http://hdl.handle.net/2123/685.

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Normal pressure hydrocephalus (NPH), a CSF circulation disorder, is important as a reversible cause of gait and cognitive disturbance in an aging population. The inconsistent response to CSF shunting is usually attributed to difficulties in differential diagnosis or co-morbidity. Improving outcome depends on an increased understanding of the pathophysiology of NPH. Specifically, this thesis examines the contribution of, and inter-relationship between, the brain parenchyma and CSF circulation in the pathophysiology of NPH. Of the four core studies of the thesis, the first quantifies the chara

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19

Silva, José Pablo. "The pathophysiology of respiratory chain dysfunction /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-234-9/.

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20

McMurdo, Lorraine. "Endothelin in cardiovascular physiology and pathophysiology." Thesis, Queen Mary, University of London, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321680.

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21

McKnight, John Alexander. "The pathophysiology of atrial natriuretic factor." Thesis, Queen's University Belfast, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335972.

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22

Bowers, Keith Cyril. "Pathophysiology of ATP in single cardiomyocytes." Thesis, University of Liverpool, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316576.

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23

Wallis, William Richard James. "The cellular pathophysiology of myocardial hypertrophy." Thesis, King's College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265997.

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24

Parees, Moreno I. "Pathophysiology of functional (psychogenic) movement disorders." Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1466184/.

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This thesis describes a series of studies involving healthy subjects, carefully selected patients with functional movement disorders and organic movement disorders, in which different aspect of the mechanism underlying functional movement disorders were explored: 1. The presence of physical precipitating factors at onset of functional movement disorder by using semistructured interviews. I found that most patients with functional movement disorder have a clear physical event prior to the onset of functional symptoms. 2. The presence of a “jumping to conclusions” reasoning style that may predis

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25

Washington, Georgita T. "Student Outcomes after Pathophysiology Course Revisions." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/etsu-works/7607.

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The value of a nursing course revision for the purpose of improved student learning and outcomes is discussed in this descriptive, quasi-experimental study. An instructor of a nursing pathophysiology course devised and implemented the study to determine if outcomes would differ between two groups of first semester baccalaureate nursing students taking the required pathophysiology course. The revisions, which were applied and then evaluated, demonstrated improvement in student learning.

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26

Rymut, Sharon Marie. "Microtubule Regulation in Cystic Fibrosis Pathophysiology." Case Western Reserve University School of Graduate Studies / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=case1432730616.

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27

Simpson, Kenneth J. "Studies on cytokines in liver pathophysiology." Thesis, University of Edinburgh, 1997. http://hdl.handle.net/1842/21531.

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<I>Aims of the thesis:</I> To study the mechanisms of chemokine production that may occur during hepatic disease, the role of chemokines in hepatic injury and repair following paracetamol poisoning and hepatic expression of chemokines in patients with liver disease. In addition, the roles of tumour necrosis factor alpha (TNF) and stem cell factor (SCF) were also studied following paracetamol poisoning. <I>Results:</I> Both CXC and CC chemokines were produced during monocyte adhesion with human hepatoma cell lines. IL-8 production was dependent on proinflammatory cytokine production. In contras

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28

Busbridge, Mark. "The physiology and pathophysiology of hepcidin." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/10743.

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Hepcidin, the critical iron regulatory factor, is a small peptide produced by the hepatocytes in response to increased body iron and inflammation. Circulating hepcidin controls both intestinal iron absorption and the release of iron from macrophages into plasma via a negative iron feedback system. I developed a novel competitive immunoassay for hepcidin using a polyclonal antibody produced against synthetic hepcidin. I validated the immunoassay and determined it was able to discriminate between healthy controls and selected disease groups. I compared the immunoassay against another established

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29

Cheluvappa, Rajkumar. "Pathophysiology of Liver Sinusoidal Endothelial Cells." Thesis, The University of Sydney, 2008. http://hdl.handle.net/2123/2802.

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Owing to its strategic position in the liver sinusoid, pathologic and morphologic alterations of the Liver Sinusoidal Endothelial Cell (LSEC) have far-reaching repercussions for the whole liver and systemic metabolism. LSECs are perforated with fenestrations, which are pores that facilitate the transfer of lipoproteins and macromolecules between blood and hepatocytes. Loss of LSEC porosity is termed defenestration, which can result from loss of fenestrations and/ or decreases in fenestration diameter. Gram negative bacterial endotoxin (Lipopolysaccharide, LPS) has marked effects on LSEC morpho

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30

Cheluvappa, Rajkumar. "Pathophysiology of Liver Sinusoidal Endothelial Cells." University of Sydney, 2008. http://hdl.handle.net/2123/2802.

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Doctor of Philosophy(PhD)<br>Owing to its strategic position in the liver sinusoid, pathologic and morphologic alterations of the Liver Sinusoidal Endothelial Cell (LSEC) have far-reaching repercussions for the whole liver and systemic metabolism. LSECs are perforated with fenestrations, which are pores that facilitate the transfer of lipoproteins and macromolecules between blood and hepatocytes. Loss of LSEC porosity is termed defenestration, which can result from loss of fenestrations and/ or decreases in fenestration diameter. Gram negative bacterial endotoxin (Lipopolysaccharide, LPS) has

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31

Spratt, James Christopher Samuel. "Endothelin : cardiovascular pharmacology, physiology & pathophysiology." Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/23202.

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32

Kelly, Richard John. "The pathophysiology of paroxysmal nocturnal haemoglobinuria." Thesis, University of Leeds, 2014. http://etheses.whiterose.ac.uk/6820/.

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Paroxysmal nocturnal haemoglobinuria (PNH) is a rare, life-threatening condition caused by an expansion of a clone of haemopoietic stem cells (HSC) harboring a somatic mutation of the PIG-A gene. PNH blood cells are deficient in glycophosphatidylinositollinked (GPI) proteins rendering PNH erythrocytes susceptible to complement attack which leads to intravascular haemolysis and an increased tendency to develop thromboses. The survival of 79 consecutive patients treated with eculizumab was compared to both age and sex matched normal controls and to 30 similar patients managed in the era immediat

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33

Sulaiman, H. "Pathophysiology of post-operative peritoneal adhesions." Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1446209/.

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Post-operative peritoneal adhesions are fibrous bands of tissue joining together organs occurring in the majority of patients following laparotomy, leading to complications such as abdominal pain, infertility in women and intestinal obstruction. However the structure and pathogenesis of adhesions is still not clear. It is proposed that persistence of fibrin between damaged tissues due to impaired plasminogen activator activity induces adhesion formation. This thesis aims to: (a) assess the structure of peritoneal adhesions in humans and in an experimental murine model (b) characterise and asse

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34

Tsang, Wing-ting Andrea, and 曾詠婷. "Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208418.

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Alzheimer’s disease (AD) is a devastating neurodegenerative disease with growing prevalence in our society. Patients suffering from this debilitating disorder also develop neuropsychiatric symptoms. Depression is one of the most frequently conveyed comorbidity; moreover, depression is also a risk factor associated with AD development. There is a complex interplay between the neurobiology of depression and AD, but their concomitant disease mechanisms remain largely unknown. Retraction of axons and dendrites has been reported to be a common occurrence in both illnesses, proposing the involvement

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35

Ráez, Bravo Arián. "Pathophysiology of sarcoptic mange in Iberian ibex." Doctoral thesis, Universitat Autònoma de Barcelona, 2019. http://hdl.handle.net/10803/669361.

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La sarna sarcóptica es una enfermedad parasitaria causada por el ácaro Sarcoptes scabiei. Afecta a mamíferos de todo el mundo, incluyendo al ser humano. En la fauna salvaje se considera una enfermedad emergente, pudiendo causar graves consecuencias poblacionales. La cabra montés (Capra pyrenaica) es un ungulado de montaña endémico de la Península Ibérica. Desde finales de los años 80, las poblaciones de cabra montés del sur y del este peninsular se han visto afectadas por esta parasitosis, con mortalidades variables que han llegado a superar el 90%. La mayoría de los estudios sobre la sarna en

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36

Forcada, Yaiza. "Genetics and pathophysiology of feline diabetus mellitus." Thesis, Royal Veterinary College (University of London), 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701655.

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37

Alix, James John Philip. "The physiology and pathophysiology of central axons." Thesis, University of Leicester, 2006. http://hdl.handle.net/2381/29957.

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This thesis investigates the physiology underlying central axon development and the pathophysiology of ischaemic injury to both immature and mature central axons, using the murine optic nerve model. A central theme to the work is that voltage gated Ca2+ channels (VGCCs) are involved in both axon development and injury. First, electrophysiological and immunohistochemical techniques were used to describe the expression of L-type and P/Q-type VGCCs at early developmental time points. Following this, two strains of mice, leaner and ducky2J, in which P/Q-type VGCC function is compromised, were used

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38

Byrne, Philip John. "Pathophysiology of bacterial gill disease in trout." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2002. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/NQ65820.pdf.

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39

Prunell, dos Santos Giselle F. "Pathophysiology of subarachnoid hemorrhage in the rat /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-610-3/.

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40

Calming, Ulrika. "Langerhans cell histiocytosis : detection, monitoring and pathophysiology /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-180-2.

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41

Lomas, David Arthur. "The molecular pathophysiology of alphaâ†1-antitrypsin." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308299.

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42

Calder, James David Forbes. "The anatomical extent and pathophysiology of osteonecrosis." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368712.

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43

Williamson, G. "Studies in the Pathophysiology of Childhood Asthma." Thesis, Queen's University Belfast, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.527903.

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44

Abuderman, Abdulwahab Ali. "Pathophysiology of Helicobacter-induced gastric precancerous changes." Thesis, University of Liverpool, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539509.

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45

Maile, Charlotte Amy. "Pathophysiology of equine type1 polysaccharide storage myopathy." Thesis, Royal Veterinary College (University of London), 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618291.

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46

Doherty, G. M. "Studies on the pathophysiology of childhood wheeze." Thesis, Queen's University Belfast, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431605.

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47

Hunt, Judith Mary. "The pathophysiology of equine post-operative ileus." Thesis, Royal Veterinary College (University of London), 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309273.

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48

Bendall, Jennifer Kate. "Physiology and pathophysiology of cardiac NADPH oxidase." Thesis, King's College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289766.

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49

Moyes, Lisa Helen. "Studies of the pathophysiology of Barrett's oesophagus." Thesis, University of Glasgow, 2013. http://theses.gla.ac.uk/4741/.

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Barrett’s oesophagus is a common condition in which the normal stratified squamous oesophageal epithelium is replaced by metaplastic reflux-induced glandular (“columnar”) mucosa (Jankowski, Barr, Wang et al. 2010;Playford 2005). Over the last three decades, the incidences of oesophageal adenocarcinoma (OA) and Barrett’s oesophagus have risen to the point that OA is now common in the United Kingdom, with Scotland having one of the highest rates in the world (Jankowski, Provenzale, & Moayyedi 2002). Unfortunately most cancers present at an advanced stage with five year survival less than 30% (Ho

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50

Hall, Judith. "Physiology and pathophysiology of vitamin D metabolism." Thesis, University of Newcastle upon Tyne, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.377459.

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