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Relevant bibliographies by topics / Paraventricular nucleus

Academic literature on the topic 'Paraventricular nucleus'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 April 2022

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Journal articles on the topic "Paraventricular nucleus"

1

Badoer, Emilio. "Role of the hypothalamic PVN in the regulation of renal sympathetic nerve activity and blood flow during hyperthermia and in heart failure." American Journal of Physiology-Renal Physiology 298, no. 4 (April 2010): F839—F846. http://dx.doi.org/10.1152/ajprenal.00734.2009.

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The hypothalamic paraventricular nucleus is a key integrative area in the brain involved in influencing sympathetic nerve activity and in the release of hormones or releasing factors that contribute to regulating body fluid homeostasis and endocrine function. The endocrine and hormonal regulatory function of the paraventricular nucleus is well studied, but the regulation of sympathetic nerve activity and blood flow by this region is less clear. Here we review the critical role of the paraventricular nucleus in regulating renal blood blow during hyperthermia and the evidence pointing to an important pathophysiological role of the paraventricular nucleus in the elevated renal sympathetic nerve activity that is a characteristic of heart failure.

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Fraser, Kurt M., and Patricia H. Janak. "Stressing the other paraventricular nucleus." Nature Neuroscience 21, no. 7 (June 25, 2018): 901–2. http://dx.doi.org/10.1038/s41593-018-0178-1.

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Uribe, Rosa Maria, Patricia Joseph-Bravo, and Jean-Louis Charli. "Pups removal enhances thyrotropin-releasing hormone mRNA in the hypothalamic paraventricular nucleus." European Journal of Endocrinology 133, no. 3 (September 1995): 354–60. http://dx.doi.org/10.1530/eje.0.1330354.

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Uribe RM, Joseph-Bravo P, Charli J-L. Pups removal enhances thyrotropin-releasing hormone mRNA in the hypothalamic paraventricular nucleus. Eur J Endocrinol 1–60. ISSN 0804–4643 Previous studies have shown that lactation and suckling alter thyrotropin-releasing hormone (TRH) biosynthesis in hypothalamic paraventricular neurons. The amounts of paraventricular TRH mRNA and mediobasal hypothalamus (MBH) TRH were determined following removal of the pups to examine whether paraventricular TRH neuron activity is altered during the transition from lactation to estrous cycle. Paraventricular TRH mRNA and MBH TRH levels were determined by Northern blot analysis and radioimmunoassay, respectively. We had shown previously that after an 8-h withdrawal of the pups at mid-lactation the MBH TRH and paraventricular TRH mRNA levels are not modified. This condition was compared to one where pups were removed for 56 h, finding a significant decrease (46%, p < 0.005) of MBH TRH and a significant increase (156%, p < 0.02) of paraventricular TRH mRNA. The effect observed in the paraventricular TRH mRNA was correlated negatively with the serum corticosterone levels, a potential negative regulator of paraventricular TRH mRNA. The results were similar if a 1-h suckling period was introduced 8 h after withdrawal of the pups to induce a transient increase of corticosterone levels. The pattern of TRH mRNA was specific to the paraventricular nucleus because there was no enhancement in the preoptic area-anterior hypothalamus. In summary, our data suggest that TRH biosynthesis in paraventricular neurons is slowly adjusted after withdrawal of the pups, possibly to prepare TRH neurons to the new secretory demands of the estrous cycle. JL Charli, Instituto de Biotecnologia, UNAM, AP 510-3, Cuernavaca, Mor. 62271, México

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Pyner, Susan. "The paraventricular nucleus and heart failure." Experimental Physiology 99, no. 2 (January 6, 2014): 332–39. http://dx.doi.org/10.1113/expphysiol.2013.072678.

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Adams, E. "The paraventricular nucleus modulates immune function." Journal of Neuroimmunology 16, no. 1 (September 1987): 11. http://dx.doi.org/10.1016/0165-5728(87)90144-5.

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Yamaguchi, Ken'ichi, Hitoshi Hama, Kazuo Watanabe, and Chieko Adachi. "Effect of dopamine injection into the anteroventral third ventricular region and the paraventricular nucleus on vasopressin secretion in conscious rats." Acta Endocrinologica 127, no. 5 (November 1992): 420–24. http://dx.doi.org/10.1530/acta.0.1270420.

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To investigate the role of dopamine receptors situated in the paraventricular nucleus and the anteroventral third ventricular region in regulating vasopressin release, responses of plasma AVP and its controlling factors to injections of dopamine into these regions and the lateral cerebral ventricle were examined in conscious rats. The injections of 156 nmol (30 μg) dopamine into the cerebral ventricle produced transient rises in plasma AVP 5 min later. When the dose of dopamine was reduced to 26 nmol (5 μg), the increase in plasma AVP was not provoked any more. However, injections of 26 nmol dopamine into the paraventricular nucleus greatly augmented plasma AVP 5 and 15 min later. This dose of dopamine was without effect on plasma AVP when injected into the anteroventral third ventricular region, including the organum vasculosum lamina terminalis, median preoptic nucleus, medial preoptic area and the periventricular preoptic nucleus. These dopamine administrations in the cerebral ventricle, paraventricular nucleus and the anteroventral third ventricular region did not significantly change AVP-controlling factors such as plasma osmolality, sodium and arterial pressure. On the basis of these results, we conclude that dopamine receptors in the paraventricular nucleus may function to facilitate AVP secretion, whereas those in the anteroventral third ventricular region may not play an important role in the regulation of AVP release.

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Csáki, Ágnes, Katalin Köves, Zsolt Boldogkői, Dóra Tombácz, and Zsuzsanna E. Tóth. "The Same Magnocellular Neurons Send Axon Collaterals to the Posterior Pituitary and Retina or to the Posterior Pituitary and Autonomic Preganglionic Centers of the Eye in Rats." NeuroSci 2, no. 1 (January 20, 2021): 27–44. http://dx.doi.org/10.3390/neurosci2010002.

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In rats, some parvocellular paraventricular neurons project to spinal autonomic centers. Using the virus tracing technique, we have demonstrated that some magnocellular paraventricular neurons, but not supraoptic neurons, also project to autonomic preganglionic centers of the mammary gland, gingiva, or lip. A part of these neurons has shown oxytocin immunoreactivity. In the present experiment, we have examined whether the same magnocellular neuron that sends fibers to the retina or autonomic preganglionic centers of the eye also projects to the posterior pituitary. Double neurotropic viral labeling and oxytocin immunohistochemistry were used. After inoculation of the posterior pituitary and the eye with viruses, spreading in a retrograde direction and expressing different fluorescence proteins, we looked for double-labeled neurons in paraventricular and supraoptic nuclei. Double-labeled neurons were observed in non-sympathectomized and cervical-sympathectomized animals. Some double-labeled neurons contained oxytocin. After the optic nerve was cut, the labeling did not appear in the supraoptic nucleus; however, it could still be observed in the paraventricular nucleus. In the paraventricular nucleus, the double-labeled cells may be the origin of centrifugal visual fibers or autonomic premotor neurons. In the supraoptic nucleus, all double-labeled neurons are cells of origin of centrifugal visual fibers.

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Castren, E., and J. M. Saavedra. "Lack of vasopressin increases hypothalamic atrial natriuretic peptide binding sites." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 257, no. 1 (July 1, 1989): R168—R173. http://dx.doi.org/10.1152/ajpregu.1989.257.1.r168.

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Atrial natriuretic peptide (ANP) binding sites were measured by quantitative autoradiography in the supraoptic and paraventricular nuclei and in the subfornical organ of hypophysectomized, adrenalectomized, and genetically vasopressin-deficient (Brattleboro) rats. Hypophysectomized and Brattleboro rats had significantly higher numbers of ANP binding sites in the supraoptic nucleus and in the magnocellular subdivision of the paraventricular nucleus than their respective controls. ANP binding density was also increased in the parvocellular subdivision of the paraventricular nucleus in hypophysectomized rats and in the subfornical organ of homozygous Brattleboro rats. When homozygous Brattleboro rats were treated with vasopressin, the density of ANP binding sites was restored to control level in the subfornical organ but not in the supraoptic or paraventricular nuclei. Adrenalectomy did not influence ANP binding in the brain areas studied. Increased ANP binding density in Brattleboro rats and after hypophysectomy in the nuclei in which vasopressin neurons are located suggest that ANP binding sites may represent physiologically active receptors and may mediate the inhibitory action of ANP on vasopressin secretion.

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Ceccatelli, Sandra, and Catello Orazzo. "Effect of different types of stressors on peptide messenger ribonucleic acids in the hypothalamic paraventricular nucleus." Acta Endocrinologica 128, no. 6 (June 1993): 485–92. http://dx.doi.org/10.1530/acta.0.1280485.

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Using in situ hybridization we have studied the effects of different types of stressors, such as ether, immobilization, cold and swimming, on the expression of several peptide messenger ribonucleic acids (mRNAs) in the hypothalamic paraventricular nucleus of adult male rats. Paraventricular nucleus sections were hybridized using synthetic oligonucleotide probes complementary to mRNA for corticotropin-releasing hormone, neurotensin, enkephalin and thyrotropin-releasing hormone. A clear upregulation of neurotensin mRNA was seen after ether and, to a lesser extent, after immobilization stress, whereas after the two other stressors neurotensin mRNA was undetectable, as in control rats. An increase in enkephalin mRNA was observed in a selective region of the dorsal part of the medioparvocellular subdivision of the paraventricular nucleus only after ether and immobilization stress. No significant changes were seen in corticotropin-releasing hormone and thyrotropin-releasing hormone mRNA levels in any of the experimental paradigms. The present results show selective changes for various peptide mRNAs in the paraventricular nucleus after various types of stress. Significant effects could be demonstrated only on neurotensin and enkephalin mRNA after ether and immobilization stress. This suggests that adaptive changes in the rate of synthesis, processing and transport of the peptide may develop over a longer period of time.

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Babovic, Sinisa, Dejan Ivanov, Ljilja Mijatov-Ukropina, Takashi Toyonaga, Ivan Dimitrijevic, and Milena Djordjevic. "Cytoarchitecture of the human paraventricular hypothalamic nucleus." Medical review 62, no. 9-10 (2009): 417–20. http://dx.doi.org/10.2298/mpns0910417b.

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Introduction. The significance of this research in terms of structure and biochemical processes in PVN contributes to further understanding of vital physiological processes from delivery and stress to delicate chemical processes that keep the hypothalamo-hypophysial axis in balance. Conclusion. Comparative studies of the human hypothalamus with the hypothalamus of other mammals enable further research, especially pharmacological and physiological ones. These are made possible with the aid of highly sophisticated equipment for examination of neurophysiological features of the brain.

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Dissertations / Theses on the topic "Paraventricular nucleus"

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Davison, Ian. "Integration by the hypothalamic paraventricular nucleus of stressful stimuli." Thesis, University of Bristol, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260655.

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Verkuyl, Jan Maarten. "Stress, corticosterone and GABAergic inhibition in the rat paraventricular nucleus." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2003. http://dare.uva.nl/document/70710.

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Cao, Xiao Yan. "Excitatory actions of orexins in rat paraventricular nucleus of thalamus." Thesis, University of Ottawa (Canada), 2006. http://hdl.handle.net/10393/27232.

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The midline thalamic paraventricular nucleus (PVT) receives a unique orexinergic innervation. To address possible function, this investigation used patch clamp recordings in rat brain slice preparations to evaluate intrinsic properties of PVT neurons and neuronal responses to bath-applied orexin peptides (A and/or B). PVT neurons displayed distinct state-dependent burst or tonic firing patterns, time dependent and time-independent inward rectification, T-type currents and low threshold spikes, action potential after-hyperpolarizations, spike frequency adaptation and spike broadening. A majority responded to both orexin peptides with slowly rising and prolonged membrane depolarizations, and inward currents that involved closure of potassium channels and/or opening of nonselective cationic channels. These data imply that endogenously released orexins likely act at both types of orexin receptors that can engage two conductances to modulate and increase neuronal excitability in PVT, a role that may be important for 'arousal' and neurotransmission within this midline thalamic nucleus.

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Ho, Sze-ngar Sara. "Synaptic modulation by 5-hydroxytryptamine in the rat hypothalamic paraventricular nucleus." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B3194341X.

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Ho, Sze-ngar Sara, and 何思雅. "Synaptic modulation by 5-hydroxytryptamine in the rat hypothalamic paraventricular nucleus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B3194341X.

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Follwell, Matthew J. "Effects of orexin A on hypothalamic paraventricular nucleus neurons of the rat." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/MQ63303.pdf.

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Borduas, Jean-Francois. "Modulation of Voltage-Gated Calcium Channels by Group II Metabotropic Glutamate Receptors in the Paraventricular Nucleus of the Thalamus." Thèse, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/19988.

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Compounds that interact with Group II metabotropic glutamate receptors (mGluRs) have antipsychotic effects in animal models. These drugs have also shown efficacy in the treatment of schizophrenia in humans. The mechanism of action is believed to arise from a reduction of glutamatergic transmission in limbic and forebrain regions commonly associated with this disorder. Previous anatomical tracer and lesion studies have revealed that neurons of the paraventricular nucleus of the thalamus (PVT) are an important source of the glutamatergic drive to these specific regions. However, the function of Group II mGluRs in the PVT remains to be determined. Whole-cell recordings from PVT neurons reveal that activation of these receptors has two interesting effects; it reduces calcium entry through voltage-gated calcium channels and it causes neurons to hyperpolarize. These two effects may contribute to affect the excitability of PVT neurons, an action that may underlie the effectiveness of Group II mGluR-activating compounds.

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Clark, Andrew J. M. "On the action of noradrenaline microinjected into the paraventricular nucleus of rat hypothalamus." Thesis, University of St Andrews, 1990. http://hdl.handle.net/10023/14704.

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The microinjection of noradrenaline (NA) into the hypothalamic paraventricular nucleus (PVN) of the rat results in feeding. This response was shown; contrary to previous reports; to be mediated through both a-1 and a-2 NA receptors. Selective blockade of these two receptor sub-types, in conjunction with re-uptake blockade was used to examine the individual contributions of each receptor type to the whole response. It is suggested that the previously reported a-2 receptor specificity of the response to microinjected NA is a result of the location of these receptors. The post-synaptic a-1 receptor being located close to the pre-synaptic re-uptake mechanism, whilst the post-synaptic a-2 receptor is located outside the synapse and thus away from the re-uptake mechanism. The re-uptake mechanism acts to create a concentration difference of microinjected NA between the two receptor sub-types, resulting in a higher concentration and thus a preferential action at a-2 receptors. The involvement of the paraventricular NA system in stress induced eating was examined using a tail pinch procedure. Microinjection of NA antagonists into PVN prior to the onset of the pinch had no effect on the duration or latency of the eating response, thus there was no evidence for the involvement of this system in tail pinch elicited feeding. Further to the suggestion that the NA a-2 receptor is extra-synaptic whilst a-1 is intrasynaptic, the actions of NA were examined at a second site. NA microinjected into the ventral striatum elicited a vigorous locomotor response, although the origins of this showed a clear priming effect. However, this response was unaffected by prior microinjection of NA a-antagonists, preventing an analysis of receptor involvement comparable with that performed in PVN.

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Bains, Jaideep S. "Nitric oxide driven inhibitory neurotransmission in the paraventricular nucleus, evidence for ultra-short-loop feedback." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/nq22443.pdf.

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Mueller, Heather. "Depressor and diuretic effects of imidazoline receptor stimulation in the paraventricular nucleus of the hypothalamus." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape9/PQDD_0007/MQ41750.pdf.

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Books on the topic "Paraventricular nucleus"

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Deering, Jane Amanda. Discharge patterns in sympathetic nerves mediating changes in cardiovascular functions of the hypothalamic paraventricular nucleus. Birmingham: University of Birmingham, 1997.

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Brown, Christina Margaret. Effects of serotonin on neuropeptide Y-induced feeding when injected into the paraventricular nucleus and the perifornical hypothalamus. Ottawa: National Library of Canada, 1993.

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Yang, Zhuo. The influence of the paraventricular nucleus of the hypothalamus on cardiovascular neurones in the ventrolateral medulla in normotensive and hypertensive rats. Birmingham: University of Birmingham, 1999.

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4

Jensen, Laurie Lynn. The role of the paraventricular nucleus of the hypothalamus in blood pressure regulation and dipsogenicity. 1990.

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Schulkin, Jay. Conservation of CRF in Brains and its Regulation by Adrenal Steroids. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780198793694.003.0003.

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The regulation of the HPA axis has been categorized as the classical mechanism of slow-acting genomic regulation of gene products, but this has given way to both slow and fast regulation of the HPA axis. We do not know how cortisol restrains the production of CRF in the paraventricular nucleus, thereby directly decreasing ACTH and, subsequently, cortisol; we know the classical negative-feedback regulatory system, which provides a mechanism, but how it works, well, that is another thing. Glucocorticoids restrain the HPA axis, but not other regions of the brain, such as the central nucleus of the amygdala and bed nucleus of the amygdala. But we now know that both chemically and electrically, these regions are not the same (equal).

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Herman, James P. Limbic Pathways to Stress Control. Edited by Israel Liberzon and Kerry J. Ressler. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.003.0008.

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Appropriate control of the HPA (hypothalamo-pituitary-adrenocortical axis) is required for adaptation to physiological and environmental challenges. Inadequate control is linked to numerous stress-related pathologies, including PTSD, highlighting its importance in linking physiological stress responses with behavioral coping strategies. This chapter highlights neurocircuit mechanisms underlying HPA axis adaptation and pathology. Control of the HPA stress response is mediated by the coordinated activity of numerous limbic brain regions, including the prefrontal cortex, hippocampus, and amygdala. In general, hippocampal output inhibits anticipatory HPA axis responses, whereas amygdala subnuclei participate in stress activation. The prefrontal cortex plays an important role in inhibition of context-dependent stress responses. These regions converge on subcortical structures that relay information to paraventricular nucleus corticotropin-releasing hormone neurons, controlling the magnitude and duration of HPA axis stress responses. The output of these neural networks determines the net effect on glucocorticoid secretion, both within the normal adaptive range and in pathological circumstances.

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Book chapters on the topic "Paraventricular nucleus"

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Sawchenko, Paul E. "Neuropeptides, the Paraventricular Nucleus, and the Integration of Hypothalamic Neuroendocrine and Autonomic Function." In Neuropeptides and Stress, 73–91. New York, NY: Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4612-3514-9_7.

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Stern, Javier. "Cellular Properties of Autonomic-Related Neurons in the Paraventricular Nucleus of the Hypothalamus." In Neural Mechanisms of Cardiovascular Regulation, 147–61. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-1-4419-9054-9_7.

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Li, Yi-Fan, Yu Wang, Keith M. Channon, Harold D. Schultz, Irving H. Zucker, and Kaushik P. Patel. "Manipulation of Neuronal Nitric Oxide Synthase Within the Paraventricular Nucleus Using Adenovirus and Antisense Technology." In Molecular Cardiology, 59–79. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-879-x:059.

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Lechan, Ronald M., and Ildikó Kakucska. "Feedback Regulation of Thyrotropin-Releasing Hormone Gene Expression by Thyroid Hormone in the Hypothalamic Paraventricular Nucleus." In Novartis Foundation Symposia, 144–64. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514283.ch10.

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Chang, Simon, and Jan M. Deussing. "Corticotropin-Releasing Hormone in the Paraventricular Nucleus of the Hypothalamus—Beyond Hypothalamic–Pituitary–Adrenal Axis Control." In Masterclass in Neuroendocrinology, 231–50. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-86630-3_9.

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Chang, Sulie L., Velga Kenigs, Roberta L. Moldow, and James E. Zadina. "Chronic Treatment with Morphine and Ethanol, But Not Cocaine, Attenuates IL-1β Activation of FOS Expression in the Rat Hypothalamic Paraventricular Nucleus." In The Brain Immune Axis and Substance Abuse, 201–8. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1951-5_28.

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Brody, Michael J., Timothy P. O’Neill, and James P. Porter. "Role of Paraventricular and Arcuate Nuclei in Cardiovascular Regulation." In Central and Peripheral Mechanisms of Cardiovascular Regulation, 443–64. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-9471-0_14.

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Krstić, R. V. "Neurosekretorische oder Neuroendokrine Nervenzellen. Beispiel: Zellen der Nuclei Supraoptici et Paraventriculares." In Die Gewebe des Menschen und der Säugetiere, 332–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-61380-7_162.

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Tasker, Jeffrey G., Cherif Boudaba, and Laura A. Schrader. "Local Glutamatergic and GABAergic Synaptic Circuits and Metabotropic Glutamate Receptors in the Hypothalamic Paraventricular and Supraoptic Nuclei." In Advances in Experimental Medicine and Biology, 117–21. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4871-3_11.

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Bernstein, H. G., A. Stanarius, D. Krell, G. Northoff, P. Danos, H. Henning, B. Baumann, and B. Bogerts. "Reduced Number of Nitric Oxide Synthase Immunoreactive Neurons in the Nucleus Paraventricularis Hypothalami of Depressed Patients and Schizophrenics." In Neuroendocrinology, 107–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60915-2_9.

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Conference papers on the topic "Paraventricular nucleus"

1

Dzambazova, Elena, Boycho Landzhov, Adriana Bocheva, and Anastasia Bozhilova-Pastirova. "Stress-induced nitric oxide activity in rat's paraventricular nucleus was affected by kyotorphin and its synthetic analogue." In XIth Conference Biologically Active Peptides. Prague: Institute of Organic Chemistry and Biochemistry, Academy of Sciences of the Czech Republic, 2009. http://dx.doi.org/10.1135/css200911028.

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Chen Guan, Shaomin Zhang, Yi Sun, and Xiaoxiang Zheng. "Assess the effect of ciliary neurotrophic factor on extracellular level of neuropeptide Y in paraventricular nucleus using microdialysis." In 2005 IEEE Engineering in Medicine and Biology 27th Annual Conference. IEEE, 2005. http://dx.doi.org/10.1109/iembs.2005.1617262.

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Diaz Martinez, Myriam, Masoud Ghamari-Langroudi, Aliya Gifford, Roger Cone, and E. B. Welch. "Automated pipeline to analyze non-contact infrared images of the paraventricular nucleus specific leptin receptor knock-out mouse model." In SPIE Medical Imaging, edited by Barjor Gimi and Robert C. Molthen. SPIE, 2015. http://dx.doi.org/10.1117/12.2082102.

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