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Books on the topic 'Parastic infection'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 February 2022

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1

Lamb, Tracey J., ed. Immunity to Parasitic Infection. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781118393321.

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2

Duerden, B. I. Microbial and parasitic infection. 7th ed. London: Edward Arnold, 1993.

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3

Immunity to parasitic infections. Chichester, West Sussex, UK: John Wiley & Sons, 2012.

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4

Kaufmann, Johannes. Parasitic Infections of Domestic Animals. Basel: Birkhäuser Basel, 1996. http://dx.doi.org/10.1007/978-3-0348-7666-7.

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5

Tuli, Jagdeesh Singh. Stress and parasitic infection in laboratory mice. Birmingham: University of Birmingham, 1993.

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6

Poppa, Anna. Fungal and parasitic infections: An overview. London: Body Positive, 1995.

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7

Kreier, Julius P. Infection, resistance and immunity. 2nd ed. New York, NY: Taylor & Francis, c2002., 2002.

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8

Kreier, Julius P. Infection, resistance and immunity. 2nd ed. New York, NY: Taylor & Francis, c2002., 2002.

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9

F, Mortensen Richard, ed. Infection, resistance, and immunity. New York: Harper & Row, 1990.

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10

Björn, Beermann, and Abdi Y. A, eds. Handbook of drugs for tropical parasitic infections. London: Taylor & Francis, 1987.

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11

Lefevre, P. C. Infectious and parasitic diseases of livestock. Paris, France: Editions Tec & Doc, 2010.

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12

Kent M. Van De Graaff. Survey of infectious and parasitic diseases. Dubuque, IA: Wm. C. Brown Publishers, 1995.

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13

Pathophysiology of endoparasitic infection: Compared with ectoparasitic infestation and microbial infection. Sydney: Academic Press, 1989.

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14

Diagnostic pathology of parasitic infections with clinical correlations. 2nd ed. New York: Oxford University Press, 2000.

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15

Wakelin, Derek. Immunity to parasites: How parasitic infections are controlled. 2nd ed. Cambridge: Cambridge University Press, 1996.

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16

Parasitic infections of domestic animals: A diagnostic manual. Basel: Birkhäuser Verlag, 1996.

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17

Diagnostic pathology of parasitic infections with clinical correlations. Philadelphia: Lea & Febiger, 1990.

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18

Kaufmann, Stefan H. E., ed. T-Cell Paradigms in Parasitic and Bacterial Infections. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74983-4.

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19

Belimenko, Vladislav, Pavel Hristianovskiy, Aleksey Gulyukin, Svetlana Alekseenkova, and Aleksey Laishevcev. Current parasitic and infectious diseases of horses. ru: INFRA-M Academic Publishing LLC., 2020. http://dx.doi.org/10.12737/1074103.

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The main infectious diseases of ungulates caused by helminths, ticks, insects, protozoa, bacteria and viruses are considered. The questions of etiology, biological features and resistance of pathogens, distribution and clinical manifestations of diseases are highlighted. Attention is paid to modern methods of diagnosis, treatment and prevention of infectious diseases of horses. Part 1 is written by Professor P. I. Khristianovsky, V. V. Belimenko, A. M. Gulyukin, A. I. Laishevtsev and E. V. Novosad; part 2 is written by Professor K. P. Yurov and S. V. Alekseenkova. It is intended for students studying in the specialty "veterinary medicine", practical veterinarians and a wide range of horse lovers.

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20

Hybrid models of tropical infections. Berlin: Springer-Verlag, 1985.

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21

Feldman, Charles, and George A. Sarosi, eds. Tropical and Parasitic Infections in the Intensive Care Unit. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/b101401.

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22

Blažek, Karel. Host response to Cysticercus bovis infection. Praha: Academia, 1988.

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23

Ogunbambi, Olabambo, and Yusuf I. Patel. Parasitic infection. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0105.

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Parasitic infections remain prevalent, particularly in the developing world. However, increasing global travel poses a risk of contracting parasitic infections while travelling through endemic areas and therefore all medical practitioners should remain aware of parasitic infections and investigate for them when appropriate. Increased understanding of the relationship of parasites with the immune system has led to some progress with therapeutics but this still lags behind other infectious diseases. In this chapter we outline the musculoskeletal manifestations of parasitic infection and updated therapeutic approaches to these infections. The burden of disease may change with increasing use of potent biologic immunosuppressants and global travel, as seen in HIV-related immunosuppression, but as yet no significant increased incidence of parasitic infection has been reported within 'rheumatic diseases' cohorts around the world.

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24

B, Grieve Robert, ed. Parasitic infections. Philadelphia: Saunders, 1987.

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25

B, Grieve Robert, ed. Parasitic infections. Philadelphia: Saunders, 1987.

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26

H, Leech James, Sande Merle A. 1939-, and Root Richard K, eds. Parasitic infections. New York: Churchill Livingstone, 1988.

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27

Duerden. Microbial & Parasitic Infection. Arnold, 1993.

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28

Lamb, Tracey. Immunity to Parasitic Infection. Wiley & Sons, Incorporated, John, 2012.

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29

I, Duerden B., Jewsbury J. M, Reid T. M. S, and Turk D. C, eds. Microbial and parasitic infection. 7th ed. London: Hodder and Stoughton, 1993.

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30

Lamb, Tracey. Immunity to Parasitic Infection. Wiley & Sons, Incorporated, John, 2012.

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31

Walzer, P. Parasitic Infections in the Compromised Host (Infectious Disease and Therapy). CRC, 1988.

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32

Letter, The Medical. Drugs for Parasitic Infections. The Medical Letter, 2010.

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33

Crompton, D. W. T. 1937-, ed. Human nutrition and parasitic infection. Cambridge: Cambridge University Press, 1993.

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34

M, Behnke Jerzy, ed. Parasites, immunity and pathology: The consequences of parasitic infection in mammals. London: Taylor & Francis, 1990.

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35

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Neurological infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0015.

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This chapter reviews the clinical features and management of meningitis (community-acquired bacterial meningitis and chronic meningitis), Mycobacterium tuberculosis, encephalitis and myelitis, Lyme disease, brain abscess and parameningeal infection, neurological infections in the immunocompromised, fungal infection, parasitic infection, and bacterial neurotoxins.

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36

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Bacterial, fungal, and parasitic infections of the liver. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0059.

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Bacterial sepsis 428Spirochaetal infections 431Rickettsial infections 432Fungal infections 432Parasitic infections 434Granulomatous hepatitis 437Infectious agents can affect the liver either via direct invasion or by release of toxins. The liver's dual blood supply renders it uniquely susceptible to infection, receiving blood from the intestinal tract via the hepatic portal system, and from the systemic circulation via the hepatic artery. Because of this unique perfusion, the liver is frequently exposed to systemic or intestinal infections or the mediators of toxaemia. The biliary tree provides a further conduit for gut bacteria or parasites to access the liver parenchyma....

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37

Kotton, Camille Nelson. Infection. Edited by Jeremy R. Chapman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0284_update_001.

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The immunosuppression which makes organ transplantation possible increases the risk of infections, both ordinary and opportunistic. The accurate diagnosis and management of infection after organ transplantation reduces morbidity and improves survival. Infections can be acquired in the hospital (i.e. nosocomial infections), from the transplant itself, from the blood product donor, from reactivation of latent infection in the host or from community exposure. Although viral infections are the most common, bacterial, fungal, and parasitic infections are also seen. While the intensity of immunosuppression is at its highest for a year after solid organ transplant, most opportunistic infections occur in the first 6 months.

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38

Lever, Andrew, and Sian Coggle. Nature and demographics: Epidemiology of infective organisms. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0304.

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An infection is an interaction between a host and a parasitic microorganism, with the interaction being deleterious to the host. Its occurrence and outcome are a combination of the nature of the organism, the site at which it is found, and the competence of the host defensive (immune) system. There are around 1500 documented agents that are infectious for man. This chapter reviews the epidemiology of infective organisms.

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39

Nolan, T. J., T. B. Nutman, and G. A. Schad. Strongyloidosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0064.

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Strongyloidosis is an intestinal parasitism caused by the threadworm, Strongyloides stercoralis. The parasite, occurring in dogs, primates and man, is found throughout the moist tropics, as well as in temperate areas where poor sanitation or other factors facilitate the occurrence of faecally transmitted organisms. In some parts of the world, notably Africa and New Guinea, human infections caused by S. fülleborni have been reported. In Africa, the latter is primarily a parasite of primates, but in New Guinea, no animal host is known. S. stercoralis is unique among zoonotic nematodes, in that larvae passing in the faeces can give rise to a free-living generation of worms which, in turn, give rise to infective larvae. This life history alternative (i.e. heterogonic development) acts as an amplification mechanism, increasing the population of infective larvae in the external environment. The infective larvae are active skin penetrators; infection per os , while possible, is probably of limited importance. Because the parasitic female’s eggs hatch internally, a potential for autoinfection exists when precociously developing larvae attain infectivity while still in the host. This is another virtually unique feature of S. stercoralis infections in both its human and animal hosts. Autoinfection can occasionally escape control by the host, with massive re-penetration and larval migration. This can cause pulmonary or cerebro-spinal strongyloidosis as well as fulminant intestinal parasitism. Control of canine strongyloidosis has been achieved in kennels by strategic use of anthelmintics. Given the lack of epidemiological information community-based programs to control human strongyloidosis have not been attempted. The growing importance of human strongyloidosis depends upon the unique ability of S. stercoralis to replicate within its host and to behave as a potentially fatal opportunistic pathogen in immunocompromised hosts, particularly in those receiving corticosteroids.

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40

Crompton, D. W. T. Human Nutrition and Parasitic Infection (Parasitology). Cambridge University Press, 1994.

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41

Soulsby, E. J. L. Immune Response to Parasitic Infections. CRC, 1987.

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42

Soulsby, E. J. L. Immune Response to Parasitic Infections. CRC, 1987.

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43

Chatterjee, T. K. Chemotherapy of Tropical Parasitic Infections. Prentice-Hall of India Pvt.Ltd, 2004.

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44

Soulsby, E. J. L. Immune Response to Parasitic Infections. CRC, 1987.

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45

Jirillo, Emilio, Thea Magrone, and Giuseppe Miragliotta, eds. Immune Response to Parasitic Infections. BENTHAM SCIENCE PUBLISHERS, 2014. http://dx.doi.org/10.2174/97816080598501140201.

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46

1942-, Walzer Peter D., and Genta Robert M. 1946-, eds. Parasitic infections in the compromised host. New York: Marcel Dekker, 1989.

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47

(Editor), Edward H. Kass, and Catharina Svanborg Eden (Editor), eds. Host-Parasite Interaction in Urinary Tract Infections (Studies in Infectious Diseases Research). University Of Chicago Press, 1989.

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48

Barsoum, Rashad S. Schistosomiasis. Edited by Neil Sheerin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0181_update_001.

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AbstractSchistosomes are blood flukes that parasitize humans, apes, cattle, and other animals. In these definitive hosts they are bisexual, and lay eggs which are shed to fresh water where they complete an asexual cycle in different snails, ending in the release of cercariae which infect the definitive hosts to complete the life cycle.Seven of over 100 species of schistosomes are human pathogens, causing disease in different organs depending on the parasite species. Racial and genetic factors are involved in susceptibility, severity, and sequelae of infection.Morbidity is induced by the host’s immune response to schistosomal antigens. The latter include tegument, microsomal, gut, and oval antigens. The former are important in the process of invasion and establishment of infection, oval antigens in formation of granulomata which lead to fibrosis in different sites, and the gut antigens constitute the main circulating antigens in established infection, leading to immune-complex disease, particularly in the kidneys. The host immunological response includes innate and adaptive mechanisms, the former being the front line responsible for removing 90% of the infecting cercarial load. Adaptive immunity includes a Th1 phase, dominated by activation of an acute inflammatory response, followed by a prolonged Th2 phase which is responsible for immunity to re-infection as well as progression of tissue injury. Switching from Th1 to Th2 phases is controlled by functional and morphological change in the antigen-presenting cells, which is achieved by molecules of host as well as parasitic origin.Many cells participate in parasite killing, but also in the induction of tissue injury. The most potent of these is the eosinophil, which by binding antibodies to the parasite, particularly immunoglobulin E, facilitates parasite elimination. However, this process is complex, including agonist as well as antagonist pathways, which provide escape mechanisms for the parasite to survive, thereby achieving a delicate balance that permits schistosomes to live for decades in the infected host.

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49

Thomas, Charles F., and Sean M. Caples. Pulmonary Infections. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0619.

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Different types of infections affect the lungs. Viral infections include viral pneumonia, influenza, hantavirus, severe acute respiratory syndrome (SARS). Bacterial infections include sinusitis, otitis media, pharyngitis, bacterial pneumonia, and community-acquired pneumonia. Mycobacterial infections cause tuberculosis and nontuberculous mycobacterial disease. Fungal diseases include histoplasmosis, blastomycosis, cryptococcosis, coccidioidomycosis, and aspergillosis. Parasitic lung disease is also reviewed.

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50

Microbial and Parasitic Infection (Hodder Arnold Publication). 7th ed. A Hodder Arnold Publication, 1996.

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