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Books on the topic 'Paraplegic men'

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Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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1

Martin, Chris. Greenwich mean time. Southwold, Suffolk: ScreenPress Books, 1999.

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2

Proulx, Monique. Invisible man at the window. Vancouver: Douglas & McIntyre, 1994.

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3

Invisible man at the window. Vancouver: Douglas & McIntyre, 1994.

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4

Kilby, Joan. Family matters. Toronto: Harlequin, 2004.

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5

Inoue, Takehiko. Real. Edited by Werry John (Translator), Dutro Steve, Whitley Yukiko, and Nakatani Andy. San Francisco: VIZ Media, 2009.

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6

1937-, Taylor Jim, ed. Rick Hansen: Man in motion. Vancouver: Douglas & McIntyre, 1987.

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7

McFadden, Maryann. The book lover. Vienna, NJ: Three Women Press, 2012.

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8

Moyes, Jojo. Mig før dig. 2nd ed. Kbh: Cicero, 2013.

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9

Moyes, Jojo. Me Before You. London: Penguin Books Ltd, 2016.

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10

Moyes, Jojo. Me Before You. London, England: Penguin Books, 2012.

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11

Moyes, Jojo. Ein ganzes halbes Jahr. 5th ed. Reinbek bei Hamburg: Rowohlt Polaris, 2013.

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12

Moyes, Jojo. Ein ganzes halbes Jahr: Roman. Augsburg: Weltbild, 2013.

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13

Moyes, Jojo. Me before you. Rearsby: Clipper Large Print, 2012.

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14

Linker, Beth, and Whitney E. Laemmli. Half a Man. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190458997.003.0007.

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At the conclusion of World War II, more than 600,000 men returned to the United States with long-term disabilities, profoundly destabilizing male sexuality in America. This chapter excavates the contours of that change and its attendant anxieties in order to broaden scholarly interpretations of sexuality in the postwar period. Ultimately, the chapter shows that, although sexual reproduction is often coded female and sexual performance male, such a popularly held binary does not hold true when it comes to the history of paraplegic World War II veterans. To these veterans, and to the medical men who treated them, sexual reproduction became the ultimate signifier of remasculinization.
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15

Mochel, Fanny. Spastic Paraplegia Type 5. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0041.

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Spastic paraplegia type 5 (SPG5) is an autosomal recessive hereditary spastic paraplegia due to mutations in CYP7B1, which encodes oxysterol 7α‎-hydroxylase. Oxysterol 7α‎-hydroxylase is involved in the synthesis of bile acids from cholesterol. CYP7B1 mutations are responsible for rare forms of liver failure in infancy as well as lower motor neuron degeneration in adults with no obvious genotype-phenotype correlation. SPG5 is mostly characterized by spastic paraplegia with prominent posterior column sensory impairment that can lead to sensory ataxia and bladder dysfunction. SPG5 can easily be diagnosed thanks to the significant elevation of two plasma oxysterols: 27- and 25-hydroxycholesterol. Accordingly, plasma oxysterols are biomarkers that should be included in the screening of any spastic paraplegia of unknown etiology. Furthermore, the dramatic therapeutic response of a child with liver failure due to CYP7B1 mutations using chenodeoxycholic acid opens promising therapeutic perspectives for SPG5 patients, possibly as in cerebrotendinous xanthomatosis.
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16

Sr, Owen Buckley. Paraplegic Son of a Preacher Man: Book 2. Xlibris Corporation LLC, 2022.

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17

Sr, Owen Buckley. Paraplegic Son of a Preacher Man: Book 1. Xlibris Corporation LLC, 2022.

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18

Sr, Owen Buckley. Paraplegic Son of a Preacher Man: Book 1. Xlibris Corporation LLC, 2022.

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19

Sr, Owen Buckley. Paraplegic Son of a Preacher Man: Book 2. Xlibris Corporation LLC, 2022.

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20

Paganoni, Sabrina, and Nazem Atassi. Upper Motor Neuron Disorders Hereditary Spastic Paraplegia and Primary Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0032.

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Upper motor neuron (UMN) syndromes are a group of rare, degenerative neurological disorders that are classified as either hereditary spastic paraplegia (HSP) or primary lateral sclerosis (PLS). Our understanding of their underlying pathophysiology is unfortunately very limited and has been a significant barrier to the development of disease-modifying treatments. Recent advances in genetics and in vitro and in vivo disease modeling have provided new insights into disease mechanisms and hold the promise to lead to the future development of mechanism-based therapies.
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21

Kilby, Joan. Family Matters. Harlequin Mills & Boon, Limited, 2014.

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22

Taylor, Jim, and Rick Hansen. Rick Hansen : Man in Motion. Douglas & McIntyre, 1999.

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23

Reiter, Udo. Gestatten, dass ich sitzen bleibe: Mein Leben. Aufbau Verlag GmbH, 2013.

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24

A Man Like Mac. Harlequin, 2000.

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25

Rick Hansen's Man in Motion World Tour: 30 Years Lateraa Celebration of Courage, Strength, and the Power of Community. Greystone Books Ltd., 2017.

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26

Firth, Helen V., and Jane A. Hurst. Common consultations. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199557509.003.0003.

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This chapter presents some of the disorders more commonly seen in a genetics clinic from achondroplasia to autosomal dominant polycystic kidney disease, Alport syndrome, androgen insensitivity syndrome, Angelman syndrome, Beckwith–Wiedemann syndrome, congenital adrenal hyperplasia, glaucoma, haemochromatosis, hereditary spastic paraplegias, Leigh encephalopathy, Marfan’s syndrome, Noonan syndrome, and many others. It gives an explanation of the clinical approach, the history, the examination, the investigation, and the diagnosis and lists the support groups.
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27

Shaibani, Aziz. Hyperreflexia. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0018.

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Hyperactive deep tendon reflexes (DTRs) is a sign of upper motor neuron (UMN) lesions. It is also commonly seen in normal but anxious people. The proper technique of deep tendon reflex examination and experience play a major role in eliciting and categorizing DTRs. Sustained clonus is the highest degree of hyperreflexia. The most important neuromuscular disease associated with hyperreflexia is amyotrophic lateral sclerosis (ALS) due to degeneration of the cortical motor neurons. Diagnostic difficulty occurs when hyperreflexia and spasticity are the only findings. In these cases, primary lateral sclerosis (PLS), hereditary spastic paraplegia (HSP), and other causes of myelopathies should be entertained. Compressive myelopathies are easily excludable by neuroimaging.
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28

Shaibani, Aziz. Muscle Stiffness and Cramps. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0020.

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Muscle stiffness as a nonspecific term means limited muscle mobility. Muscle and joint pain may be described as stiffness. Painful, sustained muscle cramps are usually associated with muscle stiffness. A careful history is paramount. Exercise-induced muscle cramps are usually myopathic (metabolic or mitochondrial myopathy) while resting, and nocturnal cramps are neurogenic [neuropathy, motor neuron disease (MND), etc.]. Metabolic cramps are electrically silent. Focal or generalized stiffness is typically seen in stiff person syndrome (SPS). Upper motor neuron (UMN) lesions are associated with spasticity and stiffness [hereditary spastic paraplegia (HSP), primary lateral sclerosis (PLS), myelopathies, etc.]. Painful cramps and fasciculation are important clues to peripheral nerve hyperexcitability disorder, which may also present with neuromyotonia. Not unusually, no cause is found for muscle cramps and stiffness. Symptomatic treatment frequently helps.
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29

Nuwer, Marc R. Intraoperative monitoring. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0036.

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Intraoperative monitoring and testing is conducted to improve neurological outcomes from surgery that incurs risk of neurological injury. Many techniques are familiar from the outpatient neurodiagnostic laboratory, and can be applied with minor modifications to the operating room setting. Other techniques are specific to the operating room. Transcranial electrical motor evoked potentials cannot be applied to awake patients, but are commonly used under general anaesthesia. Monitoring teams understand the tactics for obtaining quality recordings and calling alarms when potentials change past preset limits. Surgeons and anaesthesiologists have a variety of tactics for responding to adverse neurodiagnostic changes beginning with easy actions. In experienced hands, intraoperative neurophysiological monitoring substantially reduces post-operative deficits. For example, in spinal cord monitoring the risk of paraplegia and paraparesis is reduced by 60%. Monitoring is carried out by a technologist in the operating room under the supervision of an experienced neurophysiologist. In straightforward cases, the neurophysiologist may remotely monitor from outside the operating room.
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30

Cassiman, David, Pascal Laforêt, and Fanny Mochel. Glycogen Storage Disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0001.

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Glucose is the body’s major energy source, and carbohydrate serves as fuel—particularly during high-intensity exercise that requires rapid energy release. A deficiency of any of the enzymes involved in the catabolism of glycogen to glucose may cause symptoms, with hypoglycemia and exercise intolerance as the most common presentations. Glycogen storage disorders (GSD) affect muscle, liver, and brain. The most common GSDs affecting muscle are GSD II (Pompe disease) and GSD V (McArdle disease). GSDs affecting mainly the liver are GSD I, III, IV, VI, IX, XI. Most liver-GSDs present during infancy, with symptoms of hypoglycemia, impressive hepatomegaly, and retarded growth. Adult presentations have been reported for GSD Ia, III, IV, and IX.Adult polyglucosan body disease (APBD) is the main GSD affecting primarily the brain and mainly characterized by spastic paraplegia, axonal neuropathy and leukodystrophy. APBD is a subtype of GSD IV and is due to a deficiency of glycogen branching enzyme (GBE). Besides GSD IV, other GSDs have been reported to have CNS effects in some patients—notably GSD II and GSD III.
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31

Finfer, Simon, and Oliver Flower. Assessment and immediate management of spinal cord injury. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0344.

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Spinal cord injury is a potentially devastating injury, which may occur in isolation, but more commonly occurs in the setting of multiple injuries. Motor vehicle accidents and falls are the most common causes. Depending on the level of the injury and its completeness, patients may be left with paraplegia or tetraplegia. The injury may be immediately obvious based on history and clinical examination, but may have to be actively excluded in multiply-injured patients. Thoracolumbar spine fractures are almost always evident on plain X-rays, whereas computed tomography (CT) or magnetic resonance imaging (MRI) is frequently required to exclude cervical spine injuries. Immediate management should be directed at the detection and treatment of life-threatening injuries. Patients should be transferred to a facility specializing in the management of spinal cord injury as soon as feasible. Acute management of the spinal injury itself is largely supportive and aimed at avoiding preventable secondary injury. Respiratory complications are common, and high thoracic or cervical injuries may lead to neurogenic shock. Early identification of the injury and appropriate management results in improved outcome, reducing disability and costs of long-term management.
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32

Morell, Lee. Mimi. Cutting Edge, 2020.

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33

Astarci, Parla, Laurent de Kerchove, and Gébrine el Khoury. Aortic emergencies. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0061.

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Acute aortic dissections account for the leading and most feared of aortic emergencies. Acute dissections are associated with a dreadful mortality rate; therefore, an accurate diagnosis and immediate treatment are mandatory. The key point of a lifesaving management strategy is the distinction between acute type A dissection, uncomplicated type B dissection, and complicated type B dissection, and those including contained ruptured aorta (severe pleural effusion) and/or malperfusion syndrome (by end-organ ischaemia: paraplegia, intestinal ischaemia, renal insufficiency, limb ischaemia). Type A generally requires urgent surgery; uncomplicated type B dissections are treated conservatively, while complicated type B dissections are currently managed by means of minimally invasive endovascular techniques, eventually associated with a tight surgical time (e.g. in the case of limb ischaemia). Surgical repair of type A dissection consists of the replacement of the ascending aorta. The repair is extended proximally towards the aortic root and valve, and distally towards the aortic arch, in function of the lesions found and the clinical presentation of the patient (haemodynamic status, age, comorbidities). The emergence of endovascular techniques and the contribution of thoracic endovascular aortic repair, with thoracic stent-grafts deployed from the proximal descending aorta to reopen the true lumen and to seal the entry tear in type B dissections, have revolutionized the surgical treatment algorithm in this pathology, and thus the patient’s immediate and medium-term survival. In the same group of acute aortic syndromes, traumatic aortic isthmic ruptures are also life-threatening conditions and account for one of the main causes of death at the time of traumatic accidents. As in the case of complicated type B dissections, the introduction of aortic stent-grafts has changed the outcome of these patients.
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34

Curtis, Cathy. Alive Still. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780190908812.001.0001.

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In 1942, at age twenty, after a vision-impaired and rebellious childhood in Richmond, Virginia, Nell Blaine decamped for New York. Operations had corrected her eyesight, and she was newly aware of modern art, so different from the literal style of her youthful drawings. In Manhattan, she met rising young artists and poets. Her life was hectic, with raucous parties in her loft, lovers of both sexes, and freelance design jobs, including a stint at the Village Voice. Initially drawn to the rigorous formalism of Piet Mondrian, she received critical praise for her jazzy abstractions. During the 1950s, she began to paint interiors and landscapes. By 1959, when the Whitney Museum purchased one of her paintings, her career was firmly established. That year, she contracted a severe form of polio on a trip to Greece; suddenly, she was a paraplegic. Undaunted, she taught herself to paint in oil with her left hand, reserving her right hand for watercolors. In her postpolio work, she achieved a freer style, expressive of the joy she found in flowers and landscapes. Living half the year in Gloucester, Massachusetts, and the other half in New York, she took special delight in painting the views from her windows and from her country garden. Critics found her new style irresistible, and she had a loyal circle of collectors; still, she struggled to earn enough money to pay the aides who made her life possible. At her side for her final twenty-nine years was her lover, painter Carolyn Harris.
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35

Moyes, Jojo. Mi bip'o yu: Me before you. 2013.

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36

Moyes, Jojo. Senden Önce Ben. Pegasus Yayınları, 2016.

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37

Moyes, Jojo. Ein ganzes halbes Jahr. Rowohlt Taschenbuch, 2016.

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38

Moyes, Jojo. Yo Antes de Ti . Penguin Random House Grupo Editorial, 2015.

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39

Moyes, Jojo. Yo Antes de Ti. ALFAGUARA, 2014.

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40

Moyes, Jojo. Yo Antes de Ti / Me Before You. Penguin Random House Grupo Editorial, 2017.

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41

inainte sa te cunosc. editura Litera, 2016.

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42

Moyes, Jojo. Me Before You: A Novel. Penguin Publishing Group, 2013.

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43

Moyes, Jojo. Me before you. 2013.

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44

Moyes, Jojo. Me Before You. Film Tie-In [Paperback] Moyes, J. Penguin Random House UK, 2013.

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45

Moyes, Jojo. Me Before You (Turtleback School & Library Binding Edition). Turtleback, 2013.

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46

Me before you. Penguin Books, 2016.

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47

Moyes, Jojo. Ein ganzes halbes Jahr. Rowohlt Taschenbuch, 2018.

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48

Moyes, Jojo. Me Before You, Level 4. Penguin Books, Limited, 2019.

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49

Moyes, Jojo. Yo antes de ti. Debolsillo, 2016.

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50

Moyes, Jojo. Yo antes de ti. 2014.

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