Relevant bibliographies by topics / Paraplegia

Academic literature on the topic 'Paraplegia'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 August 2024

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Journal articles on the topic "Paraplegia"

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Rodenbaugh, David W., Heidi L. Collins, Dustin G. Nowacek, and Stephen E. DiCarlo. "Increased susceptibility to ventricular arrhythmias is associated with changes in Ca2+ regulatory proteins in paraplegic rats." American Journal of Physiology-Heart and Circulatory Physiology 285, no. 6 (December 2003): H2605—H2613. http://dx.doi.org/10.1152/ajpheart.00319.2003.

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Paraplegia may increase susceptibility to ventricular arrhythmias by altering the autonomic control of the heart. Altered cardiac autonomic control has been documented to change the expression of genes that encode cardiac Ca2+ regulatory proteins. Therefore, we tested the hypothesis that paraplegia alters cardiac electrophysiology with concomitant changes in Ca2+ regulatory proteins in a manner that increases the susceptibility to ventricular arrhythmias. To test this hypothesis, intact ( n = 10) and paraplegic ( n = 6) male Wistar rats were chronically instrumented to measure atrioventricular (AV) interval, sinus cycle length, sinus node recovery time (SNRT), SNRT corrected for spontaneous sinus cycle (cSNRT), Wenckebach cycle length (WCL), and the electrical stimulation threshold to induce ventricular arrhythmias. In addition, relative protein abundance and mRNA expression for sarco(endo)plasmic reticulum Ca2+ ATPase (SERCA), phospholamban, and the Na/Ca exchanger were determined in intact ( n = 8) and paraplegic ( n = 8) rats. Paraplegia significantly ( P < 0.05) reduced AV interval (–25%), sinus cycle length (–24%), SNRT (–28%), cSNRT (–53%), WCL (–19%), and the electrical stimulation threshold to induce ventricular arrhythmia (–48%). Paraplegia significantly increased the relative protein abundances of SERCA (45%) and the Na/Ca exchanger (40%) and decreased phospholamban levels (–28%). In contrast, only the relative mRNA expression of the Na/Ca exchanger was increased (25%) in paraplegic rats. These data demonstrate that paraplegia enhances cardiac electrophysiological properties and alters Ca2+ regulatory proteins in a manner that increases susceptibility to ventricular arrhythmias.
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Lin, Xiang, Hui-Zhen Su, En-Lin Dong, Xiao-Hong Lin, Miao Zhao, Can Yang, Chong Wang, et al. "Stop-gain mutations in UBAP1 cause pure autosomal-dominant spastic paraplegia." Brain 142, no. 8 (June 15, 2019): 2238–52. http://dx.doi.org/10.1093/brain/awz158.

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Abstract Hereditary spastic paraplegias refer to a heterogeneous group of neurodegenerative disorders resulting from degeneration of the corticospinal tract. Clinical characterization of patients with hereditary spastic paraplegias represents progressive spasticity, exaggerated reflexes and muscular weakness. Here, to expand on the increasingly broad pools of previously unknown hereditary spastic paraplegia causative genes and subtypes, we performed whole exome sequencing for six affected and two unaffected individuals from two unrelated Chinese families with an autosomal dominant hereditary spastic paraplegia and lacking mutations in known hereditary spastic paraplegia implicated genes. The exome sequencing revealed two stop-gain mutations, c.247_248insGTGAATTC (p.I83Sfs*11) and c.526G>T (p.E176*), in the ubiquitin-associated protein 1 (UBAP1) gene, which co-segregated with the spastic paraplegia. We also identified two UBAP1 frameshift mutations, c.324_325delCA (p.H108Qfs*10) and c.425_426delAG (p.K143Sfs*15), in two unrelated families from an additional 38 Chinese pedigrees with autosomal dominant hereditary spastic paraplegias and lacking mutations in known causative genes. The primary disease presentation was a pure lower limb predominant spastic paraplegia. In vivo downregulation of Ubap1 in zebrafish causes abnormal organismal morphology, inhibited motor neuron outgrowth, decreased mobility, and shorter lifespan. UBAP1 is incorporated into endosomal sorting complexes required for transport complex I and binds ubiquitin to function in endosome sorting. Patient-derived truncated form(s) of UBAP1 cause aberrant endosome clustering, pronounced endosome enlargement, and cytoplasmic accumulation of ubiquitinated proteins in HeLa cells and wild-type mouse cortical neuron cultures. Biochemical and immunocytochemical experiments in cultured cortical neurons derived from transgenic Ubap1flox mice confirmed that disruption of UBAP1 leads to dysregulation of both early endosome processing and ubiquitinated protein sorting. Strikingly, deletion of Ubap1 promotes neurodegeneration, potentially mediated by apoptosis. Our study provides genetic and biochemical evidence that mutations in UBAP1 can cause pure autosomal dominant spastic paraplegia.
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Giacomini, Leonardo, Roger Neves Mathias, Andrei Fernandes Joaquim, Mateus Dal Fabbro, Enrico Ghizoni, and Helder Tedeschi. "Is there a right time for surgery in paraplegic patients secondary to non traumatic spinal cord compression?" Einstein (São Paulo) 10, no. 4 (December 2012): 508–11. http://dx.doi.org/10.1590/s1679-45082012000400020.

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Paraplegia is a well-defined state of complete motor deficit in lower limbs, regardless of sensory involvement. The cause of paraplegia usually guides treatment, however, some controversies remain about the time and benefits for spinal cord decompression in nontraumatic paraplegic patients, especially after 48 hours of the onset of paraplegia. The objective of this study was to evaluate the benefits of spinal cord decompression in such patients. We describe three patients with paraplegia secondary to non-traumatic spinal cord compression without sensory deficits, and who were surgically treated after more than 48 hours of the onset of symptoms. All patients, even those with paraplegia during more than 48 hours, had benefits from spinal cord decompression like recovery of gait ability. The duration of paraplegia, which influences prognosis, is not a contra-indication for surgery. The preservation of sensitivity in this group of patients should be considered as a positive prognostic factor when surgery is taken into account.
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Elegbe, Oloruntoba, Mirdhu Wickremaratchi, and Martyn Hinchcliffe. "The Patient with Acute Paraplegia: A Problem-Based Review." Acute Medicine Journal 10, no. 1 (January 1, 2011): 40–44. http://dx.doi.org/10.52964/amja.0462.

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Acute paraplegia is an emergency requiring immediate assessment by the acute medical team because of the need to rule out compressive lesions of the cord for which intervention may preserve neurological function and limit persistent disability. In addition acute paraplegia could be complicated by life-threatening problems. These require prompt recognition and treatment to prevent further deterioration. The following clinical scenario, based on a real case of acute paraplegia seen by the authors is aimed at providing a problem-based approach to the management of patients presenting with acute paraplegic weakness.
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Ashry, Ahmed, Ayman Tarek Mahmoud, and Mohamed Gabr. "Delayed recovery from paraplegia following resections of thoracic meningiomas." Surgical Neurology International 11 (October 2, 2020): 321. http://dx.doi.org/10.25259/sni_575_2020.

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Background: In this retrospective study, we evaluated the patterns of postoperative recovery for patients who were initially paraplegic before the excision of thoracic spine meningiomas. We also determined how the various prognostic factors impacted outcomes. Methods: Twenty patients with paraplegia underwent surgical excision of thoracic spine meningiomas at 2016– 2019. Patients’ demographics, clinical, radiological data, operative details, histopathology, and postoperative complications were recorded; patients were reassessed at 6 months and 1 year postoperatively. Results: Fourteen patients improved postoperatively, becoming, ambulatory with/without assistance; only six remained paraplegic. Poor prognostic factors for postoperative motor recovery included larger tumor size, longer duration of preoperative symptoms/paraplegia, and greater severity of sensory loss. Conclusion: For 6/20 patients with thoracic meningiomas, poor postoperative recovery of motor function correlated with larger tumor size, longer duration of preoperative symptoms/paraplegia, and more severe sensory loss.
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Wecht, Jill M., Ronald E. De Meersman, Joseph P. Weir, Ann M. Spungen, and William A. Bauman. "Cardiac homeostasis is independent of calf venous compliance in subjects with paraplegia." American Journal of Physiology-Heart and Circulatory Physiology 284, no. 6 (June 1, 2003): H2393—H2399. http://dx.doi.org/10.1152/ajpheart.01115.2002.

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The purpose of this study was to examine cardiac hemodynamics during acute head-up tilt (HUT) and calf venous function during acute head-down tilt (HDT) in subjects with paraplegia compared with sedentary nondisabled controls. Nineteen paraplegic males (below T6) and nine age-, height-, and weight-matched control subjects participated. Heart rate, stroke volume, and cardiac output were assessed using the noninvasive acetylene uptake method. Venous vascular function of the calf was assessed using venous occlusion plethysmography. After supine measurements were collected, the table was moved to 10° HDT followed by the three levels of HUT (10, 35, and 75°) in random order. Cardiac hemodynamics were similar between the groups at all positions. Calf circumference was significantly reduced in the paraplegic group compared with the control group ( P < 0.001). Venous capacitance and compliance were significantly reduced in the paraplegic compared with control group at supine and HDT. Neither venous capacitance ( P = 0.37) nor compliance ( P = 0.19) increased from supine with 10° HDT in the paraplegic group. A significant linear relationship was established between supine venous compliance and supine cardiac output in the control group ( r = 0.80, P < 0.02) but not in the paraplegic group. The findings of reduced calf circumference and similar venous capacitance at supine rest and 10° HDT in the paraplegic group imply that structural changes may have limited venous dispensability in individuals with chronic paraplegia. Furthermore, the lack of a relationship between supine venous compliance and supine cardiac output suggests that cardiac homeostasis does not rely on venous compliance in subjects with paraplegia.
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Olmez, Akgun, and Haluk Topaloglu. "HEREDITARY SPASTIC PARAPLEGIA: PATHOGENESIS AND PATHOPHYSIOLOGY." National Journal of Neurology 1, no. 05 (July 30, 2014): 10–22. http://dx.doi.org/10.61788/njn.v1i14.01.

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- Hereditary spastic paraplegias constitute a larger group of disorders than expected. - Autosomal dominant types are mainly composed of SPAST, Atlastin (SPG3A) and REEP1 mutations. Genetic testing is suggested mainly for these genes. - The most common autosomal recessive type is SPG11, hereditary spastic paraplegia with thin corpus callosum, but SPG15 shares the same clinical features with SPG11. Genetic testing should be done for both if thin corpus callosum is present in patients. - How different genes with many different biological functions, including axonal transport, mitochondrial functions, fatty acid and cholesterol pathways and DNA repair defects, cause hereditary spastic paraplegia is still unknown.
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Mussa, S., S. Kakar, and G. Bentley. "Total Hip Arthroplasty for Late Hip Dislocation in Paraplegia." HIP International 12, no. 3 (July 2002): 338–41. http://dx.doi.org/10.1177/112070000201200310.

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Late hip dislocation is uncommon, particularly in the context of paraplegia. We report a case in which total hip arthroplasty with a semi-constrained acetabular component was a successful treatment for this condition. A review of the literature revealed that this method of treatment had not been previously described in paraplegics. For patients with late hip dislocation in spastic paraplegia, total hip arthroplasty with a semi-constrained acetabular component, combined with adequate adductor release and obturator neurectomy is recommended.
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Mesquita Junior, Nelson, Flavia Natalia Marques Kingerski, Giovana Liz Marioto, Fabio Alex Fonseca Viegas, Suzelaine Fidelis da Silva Mesquita, and Sonia Perreto. "Prevalence of deep vein thrombosis in patients with paraplegia caused by traumas." Jornal Vascular Brasileiro 12, no. 4 (October 21, 2013): 271–77. http://dx.doi.org/10.1590/jvb.2013.051.

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BACKGROUND: Deep vein thrombosis is a common disease among people who are immobilized. Immobility is inherent to paraplegia and leads to venous stasis, which is one of the factors covered by Virchow's triad describing its development. Trauma is the primary cause of paraplegia and is currently increasing at a rate of 4% per year. OBJECTIVE: To determine the prevalence of deep vein thrombosis in paraplegic patients whose paraplegia was caused by traumas, using color Doppler ultrasonography for diagnosis. METHODS: This was a cross-sectional observational study of 30 trauma-induced paraplegia patients, selected after analysis of medical records at the neurosurgery department of a University Hospital in Curitiba, Brazil, and by a proactive survey of associations that care for the physically disabled. The prevalence of deep vein thrombosis was analyzed using 95% confidence intervals. RESULTS: Spinal cord trauma was the cause of paraplegia in 29 patients. The most common cause of trauma was gunshot wounding, reported by 17 patients. Deep vein thrombosis was diagnosed by color Doppler ultrasonography in 14 patients in the sample. The most often affected vein was the posterior tibial, in 11 patients. The left lower limb was involved three times more often than the right. Edema was observed in 25 individuals, cyanosis in 14, ulcers in 8 and localized increase in temperature in 13. CONCLUSIONS: Deep vein thrombosis was prevalent, occurring in 46.7% of the patients.
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Komachali, Sajad Rafiee, Zakieh Siahpoosh, and Mansoor Salehi. "Two novel mutations in ALDH18A1 and SPG11 gene found by whole-exome sequencing in spastic paraplegia disease patients in Iran." Genomics & Informatics 20, no. 3 (September 30, 2022): e30. http://dx.doi.org/10.5808/gi.22030.

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Hereditary spastic paraplegia is a not common inherited neurological disorder with heterogeneous clinical expressions. ALDH18A1 (located on 10q24.1) gene-related spastic paraplegias (SPG9A and SPG9B) are rare metabolic disorders caused by dominant and recessive mutations that have been found recently. Autosomal recessive hereditary spastic paraplegia is a common and clinical type of familial spastic paraplegia linked to the SPG11 locus (locates on 15q21.1). There are different symptoms of spastic paraplegia, such as muscle atrophy, moderate MR, short stature, balance problem, and lower limb weakness. Our first proband involves a 45 years old man and our second proband involves a 20 years old woman both are affected by spastic paraplegia disease. Genomic DNA was extracted from the peripheral blood of the patients, their parents, and their siblings using a filter-based methodology and quantified and used for molecular analysis and sequencing. Sequencing libraries were generated using Agilent SureSelect Human All ExonV7 kit, and the qualified libraries are fed into NovaSeq 6000 Illumina sequencers. Sanger sequencing was performed by an ABI prism 3730 sequencer. Here, for the first time, we report two cases, the first one which contains likely pathogenic NM_002860: c.475C>T: p.R159X mutation of the ALDH18A1 and the second one has likely pathogenic NM_001160227.2: c.5454dupA: p.Glu1819Argfs Ter11 mutation of the SPG11 gene and also was identified by the whole-exome sequencing and confirmed by Sanger sequencing. Our aim with this study was to confirm that these two novel variants are direct causes of spastic paraplegia.
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Dissertations / Theses on the topic "Paraplegia"

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Souza, Lúcia Inês Macedo de. "Investigação genética de duas novas doenças neurodegenerativas: síndrome de Spoan (Spastic Paraglegia with Optic Atrophy and Neuropathy) e SPG34." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/41/41131/tde-06112008-164924/.

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Estudamos duas grandes famílias com manifestações de doenças neurodegenerativas. Uma delas é originária do alto oeste do estado do Rio Grande do Norte e a outra, da região de São José do Rio Preto, SP. A primeira, uma extensa família com tradição de casamentos consangüíneos, apresenta 68 indivíduos afetados pela síndrome a qual nomeamos Spoan (Spastic Paraplegia, Optic Atrophy, Neuropathy). A mesma é uma doença neurodegenerativa de herança autossômica recessiva, caracterizada por atrofia óptica congênita, espasticidade, polineuropatia periférica axonal sensitivo-motora, sobressaltos à estimulação sonora, deformidades articulares e da coluna e disartria. Estes resultados foram publicados em 2005 no Ann Neurol. 57(5):730-7. Dando continuidade ao estudo, selecionamos 23 genes que tiveram todos os exons seqüenciados. Nenhuma mutação foi observada. Amostras de 65 afetados e seus parentes foram estudados para seis marcadores de microsatélite, totalizando 149 indivíduos genotipados. Cinqüenta SNPs foram investigados, o que nos permitiu reduzir a região candidata de 4.8 para 2.3Mb em 11q13, entre o SNP rs1939212 e o microssatélite D11S987. Para o marcador D11S1889, com alelos em homozigose para todo os pacientes, foi obtido um lod score máximo de 27 em .=0.0. Os resultados deste estudo se encontram em fase de submissão. A segunda família foi estudada pela equipe da Dra. Mayana Zatz há alguns anos. Nela, investigamos 12 indivíduos afetados e 12 normais. Dentre estes, sete, com idades entre 30 e 60 anos, foram clinicamente avaliados. A idade de início foi a partir da terceira década de vida, sendo a paraplegia espástica o único sintoma. Para o marcador DXS8057 localizado em Xq25 foi obtido um lod score máximo de 4.13 em .=0.0. Com o estudo de marcadores moleculares, delimitamos uma região candidata entre os marcadores DXS1001 e DXS8033, de cerca de 14Mb, e demonstramos a existência de um novo loco gênico no cromossomo X, por nós denominado SPG34. Os resultados deste estudo estão publicados no Neurogenet on line em 08/05/200
We studied two large families with expressions of neurodegenerative diseases. One is from the high west of the state of Rio Grande do Norte and the other from São José do Rio Preto region, in São Paulo. The first, an extended family with a tradition of consanguineous marriages, has 68 individuals affected by the syndrome named by us Spoan (Spastic Paraplegia, Optic Atrophy, Neuropathy). The Spoan syndrome is a neurodegenerative disease, autosomal recessive, characterized by congenital Optic Atrophy, spasticity, axonal polyNeuropathy peripheral sensory-motor, shocks to the sound stimuli, joint and spine deformities, and dysarthria. These results were published in 2005 in Ann Neurol. 57 (5):730-7. Latter we analyzed 23 genes that were entirely sequenced. No mutation was observed. Samples of 65 affected and their relatives were studied for six microsatellite markers, totaling 149 individuals genotiped. Fifty single nucleotide polymorphisms (SNPs), located in the critical region, were also investigated, which allowed us to reduce the region for the SPOAN gene from 4.8 to 2.3 Mb, between the SNP rs1939212 and microsatellite D11S987 in 11q13. All patients are homozygous only at D11S1889, which two-point lod score with a Zmax of 27 at .=0.0 was obtained. The results of this study are being submitted. The second family was studied by Dr. Mayana Zatz group a few years ago. We investigated 12 affected and 12 normal relatives. Among these, seven patients, aged between 30 and 60 years, were clinically evaluated. The age of onset was from the third decade of life and disease showed behaviour very uniform, all affected showed Spastic Paraplegia as the only symptom. For the marker DXS8057, in Xq25, was obtained a maximum lod score of 4.13 at .=0.0. The candidate region was maped between the markers DXS1001 and DXS8033, about 14Mb and demonstrate the existence of a new gene locus on chromosome X, named by us SPG34. The results of this study were published in Neurogenet on line on may.08.2008.
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Berry, Helen Russell. "Characterisation of cardiorespiratory responses to electrically stimulated cycle training in paraplegia." Thesis, Connect to e-thesis. Edited version, 2008. http://theses.gla.ac.uk/386/.

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Thesis (Ph.D.) - University of Glasgow, 2008.
PhD. theses submitted to the Department of Mechanical Engineering, Faculty of Engineering, University of Glasgow. Edited version of thesis available, uncleared 3rd party copyright material removed. Includes bibliographical references. Print version also available.
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MANCUSO, GIUSEPPE. "Dissecting the pathogenesis of hereditary spastic paraplegia linked to SPG4 and SPG7 genes." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2011. http://hdl.handle.net/10281/20207.

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The aim of this thesis is to analyze and characterize the function of two genes involved in Hereditary Spastic Paraplegia, SPG4 and SPG7, to dissect their role in the pathogenesis of the disease. SPG4 encodes for Spastin, a microtubule severing protein involved in cytoskeletal dynamics and subcellular trafficking. On the other hand, SPG7 encodes for Paraplegin, a subunit of the m-AAA protease complex. This protease plays a key role in inner membrane protein quality control and in specific substrate maturation. Studying two genes with different function can shed light on common pathogenetic mechanisms in an etiologically complex disease such as Hereditary Spastic Paraplegia.
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Mungovan, Sean F., and n/a. "The Effect of Elevation and Venous Occlusion Pressure on Cardiovascular Function in Physically Active Men Who Are Paraplegic." Griffith University. School of Physiotherapy and Exercise Science, 2004. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20040917.084824.

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The purpose of the present investigation was to: 1) Determine the relationship between cardiac output (estimated using the acetylene rebreathing methodology) and oxygen consumption in a homogeneous group of men who are paraplegic. 2) Investigate whether lower limb elevation increases stroke volume and decreases heart rate at rest and during submaximal arm exercise. 3) Investigate whether the application of constant circumferential pneumatic pressure applied to dependent lower limbs increases stroke volume and decreases heart rate at rest and during submaximal arm exercise.
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Mungovan, Sean F. "The Effect of Elevation and Venous Occlusion Pressure on Cardiovascular Function in Physically Active Men Who Are Paraplegic." Thesis, Griffith University, 2004. http://hdl.handle.net/10072/365190.

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The purpose of the present investigation was to: 1) Determine the relationship between cardiac output (estimated using the acetylene rebreathing methodology) and oxygen consumption in a homogeneous group of men who are paraplegic. 2) Investigate whether lower limb elevation increases stroke volume and decreases heart rate at rest and during submaximal arm exercise. 3) Investigate whether the application of constant circumferential pneumatic pressure applied to dependent lower limbs increases stroke volume and decreases heart rate at rest and during submaximal arm exercise.
Thesis (Masters)
Master of Philosophy (MPhil)
School of Physiotherapy and Exercise Science
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Oteyza, Andrés de [Verfasser], and Ludger [Akademischer Betreuer] Schöls. "Gene identification in Hereditary Spastic Paraplegias and characterization of Spastic Paraplegia type 58 (SPG58) / Andrés de Oteyza ; Betreuer: Ludger Schöls." Tübingen : Universitätsbibliothek Tübingen, 2016. http://d-nb.info/1165236532/34.

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Silva, Gelson Aguiar da. "Funcional independence of individuals With paraplegia in a rehabilitation program: results and associated factors." Universidade Federal do CearÃ, 2006. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=389.

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Spinal cord injury can convey constraints to individuals, but a rehabilitation program which evalueates the functional gain allows outpatient care during the rehabilitation process. The aim of the present research was to evaluate results obtained trough the administration of Functional Independence Measure (FIM scale) in paraplegic petients within rehabilitation programs, by connecting such results with the variables: age, gender, time, level and etiology of the lesion, classification of lesion according to the ASIA (American Spinal Injury Association) criterion, time of hosptalization, educational status and complications (pressure sores, heterotopic ossofication, spasticity and neuropathic pain). For such, a transversal quantitative retrospective descriptive study was proceeded by analyzing medical records and the scoresobtained with FIM scale. The investigations were done using spedific tests, through the SPOSS software (statistical Package for the Social Science), version 13 for Windws. Results demonstrated that among the variables there studied variables there is a direct correlation between age, time of lesion, motor level, time of hospitalization and hospitalization wich a companion and the independence gain (P< 0.05). Besides there is an inverse relation between the lession classification (AIS A, B, C, D, or E) and the functional gain (P< 0.05). Functional evaluation of people with spinal cord injury though MIF scale allows to fallow up functional gain in people within a rehabilitation program.
A lesÃo medular pode trazer limitaÃÃes ao indivÃduo, mas um programa de reabilitaÃÃo que avalie o ganho funcional permite o acompanhamento, ao longo do decurso de reabilitaÃÃo. O objetivo desta pesquisa foi avaliar os resultados obtidos com a aplicaÃÃo da Medida de IndependÃncia Funcional (MIF) em pessoas portadoras de paraplegia em programa de reabilitaÃÃo; associando tais achados com as variÃveis: idade, sexo, tempo, nÃvel e etiologia da lesÃo, classificaÃÃo da lesÃo segundo o critÃrio da ASIA (American Spinal Injury Association), tempo de hospitalizaÃÃo, escolaridade e complicaÃÃes (Ãlcera de pressÃo, ossificaÃÃo heterotÃpica, espasticidade e dor neuropÃtica). Para isso foi realizado um estudo quantitativo transversal, de natureza retrospectiva, de carÃter descritivo, com anÃlise de 228 prontuÃrios e da pontuaÃÃo obtida mediante a Escala MIF. As anÃlises foram feitas sob testes especÃficos com auxÃlio do software SPSS (Statistical Package for the Social Science), versÃo 13 para Windows. Os resultados mostraram que, dentre as variÃveis estudadas, hà uma relaÃÃo direta entre a idade, o tempo de lesÃo, nÃvel motor, tempo de internaÃÃo e a internaÃÃo com acompanhante e o ganho de independÃncia (p< 0,05). TambÃm hà uma relaÃÃo inversa entre a classificaÃÃo da lesÃo (AIS A, B, C, D ou E) e o ganho funcional (p< 0,05). A avaliaÃÃo funcional em pessoas portadoras de lesÃo medular, por meio da Escala de Medida de IndependÃncia Funcional, permite o acompanhamento do ganho funcional em pessoas submetidas a um programa de reabilitaÃÃo.
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Porto, Isabela dos Passos. "Esporte e sexualidade em homens com paraplegia adquirida." Universidade do Estado de Santa Catarina, 2015. http://tede.udesc.br/handle/handle/311.

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This study aimed to investigate the factors associated with sexual adjustment paraplegic men controlling the physical variables (functional independence) and psychological (estimated body and sexual and resilience), and even compare the sexual adjustment, estimates physical and sexual, sexual behavior and sexual function between practitioners men and not sport practitioners. It is a quantitative and analytical nature of research. Participated in the study, 60 men with paraplegia, 30 sports practitioners and 30 non-sports practitioners. To obtain the necessary data, we used a questionnaire with information about sociodemographic questions (age, education, injury time); questionnaire of physical and sexual esteem, resilience questionnaire; male sexual function questionnaire (SQ-M); Functional Independence Measure Questionnaire (MIF), semistructured questionnaire about sexual frequency, sexual desire and satisfaction, self perception of physical and psychological adjustment and sexual behavior. The data were analyzed through the use the Statistical Package program for Social Sciences (SPSS) version 20.0. The form of presentation of data consisted of descriptive statistics (absolute and relative frequencies, mean, median, standard deviation) and inferential (chi-square test, U of Mann Whitney test, of Spearman correlation test and analysis of multiple linear regression. ). It was adopted the p value <0.05. The average age of study participants was 34.8 years (SD = 8.5). It was observed that the sexual adjustment was significantly higher among the sport practitioner group (p = .001) as well as the variables of body esteem and sex (p = .002), resilience (p = .026) Functional Independence Measure (p = .014); QS-M scores (p = 0.001). With regard to sexual practices (vaginal, p = .001, anal sex, p = .001) frequency (p = .001) desire (p = .001) and sexual satisfaction (p = .001), also values were obtained higher among sports practitioners. The same result was found in the variables related to erection (p = .001) and orgasm (p = .009). There was a correlation between sex fit with the time of injury (r = 305, p <.005), resilience (r = .541, p <.005), body and sex estimates (r = .633, p <. 005), education (r = .254, p <.005) and functional Independence Measure (r = .322, p <.005). We note that the score of the body and sexual esteem questionnaire was positively correlated with the time of injury (r = .292, p <.005) vaginal sexual practice (r = .490, p <.005) sexual frequency (r =. 429, p <.005), sexual satisfaction (r = .528, p <.005), erection (r = .382, p <.005), ejaculation (r = .477, p <.005), orgasm (r = .492, p <.005), QS-M score (r = .627, p <.005). Sexual satisfaction was correlated with sexual modalities (vaginal sex r = .523, p <.005; anal sex r = .279, p <.005), sexual frequency (r = .602, p <.005), desire sexual (r = .477, p <.005), erection (r = .323, p <.005), ejaculation (r = .279, p <.005), orgasm (r = .306, p <.005 ) and QS-M score (r = .528, p <.005) sexual desire correlated with oral sex (r = .410, p <.005), and QS-M score (r = .292 p <.005). Multiple linear regression analysis showed the variables that were associated with sexual adjustment were resilience (p = .003), body esteem and sex (p = .001) and the sports practice (p = .002). The sports practice was the best predictor of sexual adjustment (R2 = .508).
Este estudo objetivou investigar os fatores associados ao ajuste sexual de homens paraplégico controlando as variáveis físicas (independência funcional) e psicológicas (estima corporal e sexual e resiliência), e ainda comparar o ajuste sexual, estima corporal e sexual, comportamento sexual e função sexual entre os homens praticantes e não praticantes de esporte. Trata-se de uma pesquisa de natureza quantitativa e analítica. Participaram do estudo, 60 homens com paraplegia, sendo 30 praticantes de esporte e 30 não praticantes de esporte. Para a obtenção dos dados necessários, utilizou-se um questionário contendo informações acerca das questões sociodemográficas (idade, escolaridade, tempo de lesão); questionário de estima corporal e sexual, questionário de resiliência; questionário de função sexual masculino (QS-M); questionário de medida de independência funcional (MIF), questionário semiestruturado sobre frequência sexual, desejo e satisfação sexual, auto percepção de ajuste físico e psicológico e comportamento sexual. Os dados foram analisados mediante o uso do programa Statistical Package for the Social Sciences (SPSS) versão 20.0. A forma de apresentação dos dados consistiu em estatística descritiva (frequências absolutas e relativas, média, mediana, desvio padrão) e inferencial (teste de qui-quadrado, teste U de Mann Whitney, teste de correlação de Spearman e análise de regressão linear múltipla.). Foi adotado o valor de p<0,05. A média de idade dos participantes deste estudo foi de 34,8 anos (dp=8,5). Observou-se que o ajuste sexual foi significativamente maior entre o grupo praticante de esporte (p=.001) assim como as variáveis de estima corporal e sexual (p=.002), resiliência (p=.026) medida de independência funcional (p=.014); escore QS-M (p=0,001). Com relação às práticas sexuais (sexo vaginal, p=.001; sexo anal, p=.001) frequência (p=.001) desejo (p=.001) e satisfação sexual (p=.001), também foram obtidos valores maiores entre os praticantes de esporte. O mesmo resultado foi encontrado nas variáveis referente à ereção (p=.001) e orgasmo (p=.009). Observou-se correlações entre o ajuste sexual com o tempo de lesão (r=305, p<.005), resiliência (r=.541, p<.005), estima corporal e sexual (r=.633, p<.005), escolaridade (r=.254, p<.005) e medida de independência funcional (r=.322, p<.005). Observamos que o escore do questionário de estima corporal e sexual se correlacionou positivamente com o tempo de lesão (r=.292, p<.005) prática sexual vaginal (r=.490, p<.005) frequência sexual (r=.429, p<.005), satisfação sexual (r=.528, p<.005), ereção (r=.382, p<.005), ejaculação (r=.477, p<.005), orgasmo (r=.492, p<.005), escore QS-M (r=.627, p<.005). A satisfação sexual se correlacionou com as modalidades sexuais (sexo vaginal r=.523, p<.005; sexo anal r=.279, p<.005), frequência sexual (r=.602, p<.005), desejo sexual (r=.477, p<.005), ereção (r=.323, p<.005), ejaculação (r=.279, p<.005), orgasmo (r=.306, p<.005) e escore QS-M (r=.528, p<.005) O desejo sexual se correlacionou com a prática de sexo oral (r=.410, p<.005), e escore QS-M (r=.292, p<.005). A análise de regressão linear múltipla apresentou as variáveis que estiveram associadas com o ajuste sexual foram a resiliência (p=.003) , estima corporal e sexual (p=.001) e a prática de esporte (p=.002). A prática de esporte foi o melhor preditor do ajuste sexual (R2=.508). Conclui-se que a prática de esporte influenciou no ajuste sexual, estimas corporal e sexual, função e comportamento sexual de homens com paraplegia adquirida.
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Parodi, Livia. "Identification of genetic modifiers in Hereditary Spastic Paraplegias due to SPAST/SPG4 mutations Spastic paraplegia due to SPAST mutations is modified by the underlying mutation and sex Hereditary spastic paraplegia: More than an upper motor neuron disease." Thesis, Sorbonne université, 2019. http://www.theses.fr/2019SORUS317.

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Les Paraplégies Spastiques Héréditaires (PSHs) sont un groupe de maladies neurodégénératives rares qui surviennent suite à la dégénérescence progressive des voies corticospinales, entraînant une spasticité des membres inférieurs, signe distinctif de la pathologie. Elles se caractérisent par une extrême hétérogénéité qui concerne à la fois les facteurs génétiques et cliniques, ainsi que d’autres aspects de la maladie, tels que l’âge d’apparition et la sévérité des signes. Cette variabilité est typiquement observée chez les patients porteurs de mutations pathogènes dans SPAST, le gène le plus fréquemment muté dans les PSHs. Après avoir réuni une cohorte de 842 patients mutés dans SPAST, nous avons utilisé une combinaison de différentes approches de Séquençage de Nouvelle Génération (NGS) afin de mieux comprendre les causes de l’hétérogénéité observée chez les patients, afin d’identifier des facteurs génétiques responsables de variations de l’âge au début de la maladie. Les données résultantes du génotypage de l’ensemble du génome ont ainsi été utilisées pour effectuer des analyses d’association et de liaison qui, combinées aux données de séquençage de l’ARN, ont permis d’identifier différents variantes/gènes candidats, potentiellement impliqués comme facteurs modificateurs de l’âge de début des SPAST-PSHs
Hereditary Spastic Paraplegias (HSPs) are a group of rare, inherited, neurodegenerative disorders that arise following the progressive degeneration of the corticospinal tracts, leading to lower limbs spasticity, the disorder hallmark. HSPs are characterized by an extreme heterogeneity that encompasses both genetic and clinical features, extending to additional disorder’s features, such as age of onset and severity. This phenotypic variability is typically observed among HSP patients carrying pathogenic mutations in SPAST, the most frequently mutated HSP causative gene. After assembling a cohort of 842 SPAST-HSP patients, a combination of different Next Generation Sequencing approaches was used to dig deeper into the causes of the observed heterogeneity, especially focusing on the identification of age of onset genetic modifiers. Sequencing data resulting from Whole Genome Genotyping were used to perform both association and linkage analysis that, combined with RNA sequencing expression data, allowed to identify different candidate variants/genes, potentially acting as SPAST-HSP age of onset modifiers
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Azevedo, Eliza Regina Ferreira Braga Machado de. "Análise cinética e cinemática da marcha de indivíduos paraplégicos com e sem órtese de pé e tornozelo (AFO)." [s.n.], 2011. http://repositorio.unicamp.br/jspui/handle/REPOSIP/313761.

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Orientador: Alberto Cliquet Júnior
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: A lesão medular tem como consequência a perda da mobilidade e sensibilidade abaixo do nível da lesão, gerando, portanto, a impossibilidade de andar. Essa perda de movimentos traz inúmeras consequências aos pacientes lesados medulares como osteoporose, atrofia muscular, espasticidade, infecções urinárias de repetição entre outras. E muitas destas consequências podem ser minimizadas com o treinamento de marcha realizado com a utilização de estimulação elétrica neuromuscular (EENM). Dessa forma, o objetivo da pesquisa foi descrever a marcha de paraplégicos completos utilizando EENM e avaliar os efeitos da ankle foot orthoses (AFO) do tipo rígida na marcha destes indivíduos. Dezesseis indivíduos, sendo dez participantes controle e seis paraplégicos completos realizaram avaliação cinética e cinemática da marcha com e sem AFO. Ambos os grupos realizaram as avaliações em duas etapas: com AFO e sem AFO. Sendo que o grupo de pacientes realizou a marcha sempre utilizando EENM nos músculos quadríceps e no nervo fibular. Foi utilizado sempre o mesmo calcado durante os testes. Foram utilizados como parâmetros espaço-temporais a velocidade média (m/s), a cadência (passos/min), o comprimento de passo (m) e a porcentagem da fase de apoio. Todas as variáveis apresentaram diferenças significativas entre grupos e uso ou não da AFO, com exceção da porcentagem da fase de apoio, que apenas apresentou diferença entre os grupos (p<0,001). Entre as variáveis cinemáticas do tornozelo no contato inicial houve diferença entre os grupos (p=0,026), na amplitude de movimento (ADM) no apoio a diferença foi significativa no uso ou não da AFO (p=0,01), entre grupos (p=0,02) e na interação entre os grupos e o uso ou não da AFO (p=0,008), e na ADM no balanço a diferença ocorreu entre o uso e não da AFO (p=0,002) e na interação entre grupos e uso ou não da AFO (p=0,007). As variáveis do joelho que apresentarão valores significativos entre o uso ou não da AFO foram o ângulo no contato inicial (p=0,002), a ADM no apoio (p=0,0001) e a flexão no impulso (p=0,018), entre os grupos a flexão no impulso (p<0,0001) e a máxima flexão no balanço (p<0,001) apresentaram valores significativos. No quadril as diferenças significativas ocorreram apenas entre os grupos para o contato inicial (p<0,0001), máxima extensão no apoio (p=0,002), a extensão no impulso (p=0,005) e a máxima flexão no balanço (p=0,001). Nas variáveis cinéticas do tornozelo foram avaliados o momento no choque de calcanhar e momento máximo flexor plantar, ambos apresentaram valores significativos entre grupos (p=0,012 e p=0,014) e apenas o momento no choque de calcanhar apresentou diferença entre o uso ou não da AFO (p=0,015). No joelho e quadril foram avaliados os momentos máximos flexor e extensor. Sendo que no joelho apenas o máximo momento extensor apresentou diferença entre os grupos (p=0,0002). No quadril os valores de momento flexor máximo foram significativos entre grupos (p<0,0001). Os resultados espaço-temporais sugerem que a marcha com AFO é mais eficaz para os paraplégicos completos. Além disso, a AFO promoveu uma maior proteção ao joelho do desses indivíduos e permitiu uma maior descarga mecânica no quadril podendo assim, prevenir a perda de massa óssea
Abstract: The spinal cord injury leads to the loss of mobility and sensibility below the injury level, causing the inability to walk. This loss of movement causes consequences for spinal cord injured patients such as osteoporosis, muscle atrophy, spasticity, repetitive urinary infections and others. Many of these consequences can be minimized by gait training with neuromuscular electrical stimulation (NMES). Thus, the objective of the research was to describe paraplegic gait with NMES and assess the influence of rigid AFO on these individuals gait. Sixteen individuals, ten control participants and six complete paraplegics went through kinetics and kinematics gait evaluation with and without AFO. The patients group performed the gait using NMES in quadriceps muscles and peroneal nerve. Both groups used the same shoes during the tests. Spatiotemporal variables assessed were velocity (m/s), cadence (steps per minute), step length (m) and percentage of stance time. All variables except for the percentage of stance time showed significant differences between groups and with and without AFO. In ankle joint kinematics, the initial contact was different between groups (p=0,026), range of motion (ROM) in stance was significant difference with and without AFO (p=0,01), between groups (p=0,02) and in the interaction between groups and with and without AFO (p=0,008), ROM in balance was different between with and without AFO (p=0,002) and in the interaction between groups and with and without AFO (p=0,007). The knee kinematics displayed significant changes between with and without AFO in initial contact (p=0,002), ROM in stance (p=0,0001) and flexion at toe-off (p=0,018), between groups difference was noted in flexion at toe-off (p<0,0001) and maximum flexion in balance (p<0,0001). The hip significant differences were only observed between groups in initial contact (p<0,0001), maximum extension in balance (p=0,002), extension at toe-off (p=0,005) and in maximum flexion in balance (p=0,001). Ankle joint kinetic variables assessed were the moment at load response and maximum plantar flexor moment, both displayed significant changes between groups (p=0,012 and p=0,014), only the moment at load response was different between with and without AFO (p=0,015). Knee and hip were assessed maximum flexor and extensor moments. Only the maximum knee extensor moment was significant different between groups (p=0,0002). Hip flexor moment values were significant between groups (p<0,0001). The spatiotemporal results suggest that the gait with AFO is more effective for complete paraplegics individuals. Furthermore, the AFO allowed a greater knee protection to these individuals and also yielded a higher mechanical loading on the hip, which can prevent the loss bone mass
Mestrado
Fisiopatologia Cirúrgica
Mestre em Ciências
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Books on the topic "Paraplegia"

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1937-, Rogers Michael A., ed. Living with paraplegia. London: Faber and Faber, 1986.

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Bromley, Ida. Tetraplegia and paraplegia: A guide for physiotherapists. 4th ed. Edinburgh: Churchill Livingstone, 1991.

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Curran, John. Just my luck! Trá Lí, Co. Chiarraí: Inné Teo, 1993.

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Antonio, Pedotti, Ferrarin Maurizio, and Commission of the European Communities., eds. Restoration of walking for paraplegics: Recent advances and trends. Milano: Pro Juventute, 1992.

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Alleyne, Cargill Herley. Clinical and seroepidemiological case-control study of tropical spastic paraparesis and multiple sclerosis in Barbados. [New Haven: s.n.], 1991.

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Ana Cristina G. Duarte Vasconcellos. Resiliência: Um estudo sobre famílias com portadores de paraplegia. Curitiba: Juruá Editora, 2010.

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Institute, Pennsylvania Bar. The catastrophic injury case: Quadriplegia, paraplegia & spinal injury cases. Mechanicsburg, Pennsylvania: Pennsylvania Bar Institute, 2015.

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Institute, Pennsylvania Bar. The catastrophic injury case: Quadriplegia, paraplegia and spinal injury cases. [Mechanicsburg, Pa.]: Pennsylvania Bar Institute, 2012.

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Robert, Kimberly. Everlasting. Baltimore, Md: America House Book Publshers, 2000.

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Lavekar, G. S. Clinical studies of certain ayurvedic formulations in the management of Paraplegia (Paṅgu). Edited by Pāḍhī Ema Ema and Central Council for Research in Ayurveda and Siddha (India). New Delhi: Central Council for Research in Ayurveda and Siddha, Dept. of AYUSH, Ministry of Health & Family Welfare, 2010.

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Book chapters on the topic "Paraplegia"

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Wilson, J. Frank, and Kevin Murray. "Paraplegia." In Practical Approaches to Cancer Invasion and Metastases, 16–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-84885-8_4.

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Baker, Julien S., Fergal Grace, Lon Kilgore, David J. Smith, Stephen R. Norris, Andrew W. Gardner, Robert Ringseis, et al. "Paraplegia." In Encyclopedia of Exercise Medicine in Health and Disease, 690. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2834.

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Liberski, Pawel P., and Craig Blackstone. "Hereditary Spastic Paraplegia." In Neurodegeneration, 161–78. Oxford, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118661895.ch15.

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Lee, T. T., and B. A. Green. "Trauma and Paraplegia." In Spinal Cord Disease, 545–83. London: Springer London, 1997. http://dx.doi.org/10.1007/978-1-4471-0911-2_31.

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Rodríguez-Leyva, Ildefonso. "Hereditary Spastic Paraplegia." In International Neurology, 205–7. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444317008.ch54.

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Tucci, K. A., H. J. Landy, B. A. Green, and F. J. Eismont. "Trauma and Paraplegia." In Clinical Medicine and the Nervous System, 409–27. London: Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-3353-7_24.

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Haberl, Roman, Dennis G. Vollmer, and Werner Hacke. "Tetraplegia and Paraplegia." In Neurocritical Care, 292–306. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-87602-8_28.

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Rodríguez-Leyva, Ildefonso. "Hereditary spastic paraplegia." In International Neurology, 451–55. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118777329.ch107.

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Eltorai, Ibrahim M. "Paraplegia Following Chemonucleolysis." In Rare Diseases and Syndromes of the Spinal Cord, 129–30. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-45147-3_41.

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Lee, T. T., and B. A. Green. "Trauma and Paraplegia." In Spinal Cord Disease, 545–83. London: Springer London, 1997. http://dx.doi.org/10.1007/978-1-4471-0569-5_31.

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Conference papers on the topic "Paraplegia"

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Katti, Vikram, and William Durfee. "Preliminary Design and Testing of a Muscle-Powered Walking Exoskeleton for People With Spinal Cord Injury." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6889.

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There are 275,000 people in the US who have spinal cord injury (SCI) and there are about 12,500 new cases each year [1]. Among these 18.5% have incomplete paraplegia and 23% have complete paraplegia [1]. The number of patients experiencing mobility impairment caused by SCI is increasing because of accidents and disease [2,3]. Among people with SCI, 51% identified walking as first choice for a technology application [4].
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Lazarin, Gabriela Bazzanella, and Eliane Pinheiro. "MODA INCLUSIVA: VESTUÁRIO PARA MULHERES COM PARAPLEGIA." In 16° Ergodesign – Congresso Internacional de Ergonomia e Usabilidade de Interfaces Humano Tecnológica. São Paulo: Editora Blucher, 2017. http://dx.doi.org/10.5151/16ergodesign-0167.

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Guanais, Luan, Patrícia Pontes Cruz, Aline Rocha Anibal, and Emília Katiane Embiruçu. "Early-onset hereditary spastic paraplegia: case report." In SBN Conference 2022. Thieme Revinter Publicações Ltda., 2023. http://dx.doi.org/10.1055/s-0043-1774578.

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Antônia Andrade Rangel, Maria, Larissa Alvarenga Pereira de Souza, Lucas Arêas Soares, Bruno Gama Linhares, and Juliana Siqueira Pessanha. "Redução do score de risco cardiovascular em pessoas com paraplegia através de um programa de reabilitação física." In Semana Científica da Faculdade de Medicina de Campos. Faculdade de Medicina de Campos, 2023. http://dx.doi.org/10.29184/anaisscfmc.v22023p35.

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Introdução: As doenças cardiovasculares são a principal causa de morte de pessoas que envelhecem com lesão medular, uma das medidas necessárias para a prevenção e redução dessas patologias está no bom condicionamento físico, promovido por atividades físicas regulares. Objetivos: Realizar um programa de reabilitação física com finalidade de reduzir o score do risco cardiovascular em indivíduos com lesão medular e paraplegia em Campos dos Goytacazes. Métodos: Essa é uma pesquisa experimental do tipo ensaio clínico randomizado aberto controlado de dois braços, com início no período de novembro de 2022, tendo como público alvo pacientes com paraplegia e lesão medular. O histórico médico dos participantes está sendo coletado de seus prontuários no Hospital Escola Álvaro Alvim (HEAA). Em seguida, esses indivíduos passarão por uma avaliação cardiológica para ratificar se estão aptos a participar desse ensaio clínico, onde serão solicitados exames como bioquímica laboratorial e eletrocardiograma. A intervenção será pautada no protocolo de Farrow, Maher e Thompson (2021) para ensaios clínicos em pacientes com paraplegia. Após as 6 semanas de intervenção, os participantes do grupo intervenção passarão pela última consulta cardiológica, a fim de compará-los com o grupo controle. Projeto aprovado pelo Comitê de Ética em Pesquisa em Seres Humanos, n°: 5.792.311. Resultados: Foi iniciada a seleção dos participantes pelo sistema de prontuários online do HEAA, através de 231 números de atendimentos, 9 participantes foram identificados com CID 10 G82.2 de paraplegia não especificada, no entanto somente 2 prontuários estavam com os dados completos, entre esses, 1 cumpriu o requisito. A etapa de seleção está em andamento, no momento aguarda-se a liberação do HEAA do acesso aos prontuários físicos para o recrutamento dos demais participantes. Discussão: Dando seguimento ao recrutamento e a intervenção, almeja-se a melhora no condicionamento físico, frequência respiratória e cardíaca e taxas metabólicas, além de reduzir o risco de complicações cardiovasculares. Conclusão: Nesse seguimento, espera-se redução do score de risco cardiovascular, de forma a ratificar que a atividade física disciplinada por profissionais de saúde devidamente capacitados é uma intervenção contra as doenças cardiovasculares em pessoas com paraplegia.
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Azimee, M., A. Chakravarty, and S. Anand. "Subarachnoid haemorrhage and paraplegia in coarctation of aorta." In 18th Annual Conference of Indian Society of Neuroanaesthesiology and Critical Care (ISNACC 2017). Thieme Medical and Scientific Publishers Private Ltd., 2017. http://dx.doi.org/10.1055/s-0038-1646203.

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Chang, Sarah R., Mark J. Nandor, Lu Li, Kevin M. Foglyano, John R. Schnellenberger, Rudi Kobetic, Roger D. Quinn, and Ronald J. Triolo. "A stimulation-driven exoskeleton for walking after paraplegia." In 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7592185.

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Kapula, Prabhakara Rao, I. A. Pasha, B. Anil Kumar, and V. Sowmya. "Wearable Lower Limb Exoskeletons for paraplegia: A Review." In 2021 International Conference on Technological Advancements and Innovations (ICTAI). IEEE, 2021. http://dx.doi.org/10.1109/ictai53825.2021.9673413.

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Braga, Vinícius Lopes, Wladimir Bocca Vieira de Rezende Pinto, Bruno de Mattos Lombardi Badia, José Marcos Vieira de Albuquerque Filho, Igor Braga Farias, Paulo Victor Sgobbi de Souza, and Acary Souza Bulle Oliveira. "Spastic paraplegia type 73: expanding phenotype of the first two Brazilian families." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.552.

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Abstract:
Context: Hereditary spastic paraplegias (HSPs) represent an expanding group of neurodegenerative diseases characterized mainly by progressive spastic paraparesis of the lower limbs. More than 80 different genetic loci have been associated with HSPs. In 2015, heterozygous pathogenic variants in the CPT1C gene were first associated with SPG73, not yet described in Brazilian patients. Objective: We present clinical, neuroimaging and genetic features of three Brazilian patients with SPG73. Cases reports: We report one male and two female patients, age range 36 to 78 years old. Case 1 presented with a 4-year-history of spasticity, predominantly crural tetraplegia, bladder incontinence, dysphagia and dysphonia. Family history disclosed a sister with epilepsy. Whole-exome sequencing (WES) disclosed a heterozygosis variant c.863G>A (p.Arg288His) in exon 9 of the CPT1C. Cases 2 and 3 are first degree relatives (mother and son). Both presented with long-standing slowly progressive spastic paraplegia. Case 3 presented bladder incontinence, constipation, dysphagia and dysphonia at late stages. Cases 2 and 3 WES disclosed the heterozygosis variant c.196T>G (p.Phe66Val) in exon 4 of the CPT1C. Discussion: Previous literature described six patients from an Italian family with pure HSPs phenotype and the pathogenic variant c.109C>G (p.Arg3. 7Cys) in CPT1C gene. Another group described three patients associated with pure HSPs phenotype and the pathogenic variant (c.226C>T) in the CPT1C gene. All previous reported cases had benign clinical course and bulbar involvement was not described before. One of our cases presented with a de novo variant and rapidly progressive motor and bulbar compromise. Conclusion: our cases expand the current knowledge about SPG73, including a rapidly progressive phenotype with bulbar involvement and cognitive compromise at late stages of disease course.
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Bakar, Norazhar Abu, and Abdul Rahim Abdullah. "Dynamic simulation of sit to stand exercise for paraplegia." In 2011 IEEE International Conference on Control System, Computing and Engineering (ICCSCE). IEEE, 2011. http://dx.doi.org/10.1109/iccsce.2011.6190506.

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Kosaka, Manabu, Duncan Wood, and Ian Swain. "FES Control Design for Paraplegia Using Indoor Rrowing Machine." In 2009 WRI Global Congress on Intelligent Systems. IEEE, 2009. http://dx.doi.org/10.1109/gcis.2009.168.

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Reports on the topic "Paraplegia"

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Purdy, Allison. The Effects of Yoga Therapy on the Quality of Life for a Paraplegic Individual. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.342.

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