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Relevant bibliographies by topics / Papillomaviruses

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Academic literature on the topic 'Papillomaviruses'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 July 2024

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Journal articles on the topic "Papillomaviruses"

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White, Elizabeth A. "Manipulation of Epithelial Differentiation by HPV Oncoproteins." Viruses 11, no. 4 (April 22, 2019): 369. http://dx.doi.org/10.3390/v11040369.

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Papillomaviruses replicate and cause disease in stratified squamous epithelia. Epithelial differentiation is essential for the progression of papillomavirus replication, but differentiation is also impaired by papillomavirus-encoded proteins. The papillomavirus E6 and E7 oncoproteins partially inhibit and/or delay epithelial differentiation and some of the mechanisms by which they do so are beginning to be defined. This review will outline the key features of the relationship between HPV infection and differentiation and will summarize the data indicating that papillomaviruses alter epithelial differentiation. It will describe what is known so far and will highlight open questions about the differentiation-inhibitory mechanisms employed by the papillomaviruses.

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Lange, Christian E., Mathias Ackermann, Claude Favrot, and Kurt Tobler. "Entire Genomic Sequence of Novel Canine Papillomavirus Type 13." Journal of Virology 86, no. 18 (August 23, 2012): 10226–27. http://dx.doi.org/10.1128/jvi.01553-12.

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Papillomaviruses are associated with benign and malignant neoplasias of the skin and mucous membranes. The sequence of a novel canine papillomavirus was determined from DNA detected in the oral cavity of a dog. The sequence of the novel virus canine papillomavirus type 13 (CPV13) shares the highest levels of similarity with the Tau papillomaviruses CPV2 and CPV7.

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Antonsson, Annika, and Nigel A. J. McMillan. "Papillomavirus in healthy skin of Australian animals." Journal of General Virology 87, no. 11 (November 1, 2006): 3195–200. http://dx.doi.org/10.1099/vir.0.82195-0.

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Papillomaviruses are a group of ubiquitous viruses that are often found in normal skin of humans, as well as a range of different vertebrates. In this study, swab samples collected from the healthy skin of 225 Australian animals from 54 species were analysed for the presence of papillomavirus DNA with the general skin papillomavirus primer pair FAP59/FAP64. A total of five putative and potential new animal papillomavirus types were identified from three different animal species. The papillomaviruses were detected in one monotreme and two marsupial species: three from koalas, and one each from an Eastern grey kangaroo and an echidna. The papillomavirus prevalence in the three species was 14 % (10/72) in koalas, 20 % (1/5) in echidnas and 4 % (1/23) in Eastern grey kangaroos. Phylogenetic analysis was performed on the putative koala papillomavirus type that could be cloned and it appears in the phylogenetic tree as a novel putative papillomavirus genus. The data extend the range of species infected by papillomaviruses to the most primitive mammals: the monotremes and the marsupials.

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McPhillips, M. G., J. G. Oliveira, J. E. Spindler, R. Mitra, and A. A. McBride. "Brd4 Is Required for E2-Mediated Transcriptional Activation but Not Genome Partitioning of All Papillomaviruses." Journal of Virology 80, no. 19 (October 1, 2006): 9530–43. http://dx.doi.org/10.1128/jvi.01105-06.

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ABSTRACT Bromodomain protein 4 (Brd4) has been identified as the cellular binding target through which the E2 protein of bovine papillomavirus type 1 links the viral genome to mitotic chromosomes. This tethering ensures retention and efficient partitioning of genomes to daughter cells following cell division. E2 is also a regulator of viral gene expression and a replication factor, in association with the viral E1 protein. In this study, we show that E2 proteins from a wide range of papillomaviruses interact with Brd4, albeit with variations in efficiency. Moreover, disruption of the E2-Brd4 interaction abrogates the transactivation function of E2, indicating that Brd4 is required for E2-mediated transactivation of all papillomaviruses. However, the interaction of E2 and Brd4 is not required for genome partitioning of all papillomaviruses since a number of papillomavirus E2 proteins associate with mitotic chromosomes independently of Brd4 binding. Furthermore, mutations in E2 that disrupt the interaction with Brd4 do not affect the ability of these E2s to associate with chromosomes. Thus, while all papillomaviruses attach their genomes to cellular chromosomes to facilitate genome segregation, they target different cellular binding partners. In summary, the E2 proteins from many papillomaviruses, including the clinically important alpha genus human papillomaviruses, interact with Brd4 to mediate transcriptional activation function but not all depend on this interaction to efficiently associate with mitotic chromosomes.

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Antonsson, Annika, and Bengt Göran Hansson. "Healthy Skin of Many Animal Species Harbors Papillomaviruses Which Are Closely Related to Their Human Counterparts." Journal of Virology 76, no. 24 (December 15, 2002): 12537–42. http://dx.doi.org/10.1128/jvi.76.24.12537-12542.2002.

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ABSTRACT Papillomaviruses associated with clinical symptoms have been found in many vertebrate species. In this study, we have used an L1 gene consensus PCR test designed to detect a broad spectrum of human skin papillomaviruses to analyze swab samples from healthy skin of 111 animals belonging to 19 vertebrate species. In eight of the species, papillomavirus DNA was found with the following prevalences: chimpanzees, 9 of 11 samples positive; gorillas, 3 of 4; long-tailed macaques, 14 of 16; spider monkeys, 2 of 2; ruffed lemurs, 1 of 2; cows, 6 of 10; European elks, 4 of 4; aurochs, 1 of 1. In total, 53 new putative animal papillomavirus types were found. The results show that skin papillomaviruses can be detected in healthy skin from many different animal species and are sufficiently related genetically to their human counterparts to be identified by a human skin papillomavirus primer set (FAP59 and FAP64).

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Varsani, Arvind, Simona Kraberger, Scott Jennings, Elizabeth L. Porzig, Laurel Julian, Melanie Massaro, Annie Pollard, Grant Ballard, and David G. Ainley. "A novel papillomavirus in Adélie penguin (Pygoscelis adeliae) faeces sampled at the Cape Crozier colony, Antarctica." Journal of General Virology 95, no. 6 (June 1, 2014): 1352–65. http://dx.doi.org/10.1099/vir.0.064436-0.

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Papillomaviruses are epitheliotropic viruses that have circular dsDNA genomes encapsidated in non-enveloped virions. They have been found to infect a variety of mammals, reptiles and birds, but so far they have not been found in amphibians. Using a next-generation sequencing de novo assembly contig-informed recovery, we cloned and Sanger sequenced the complete genome of a novel papillomavirus from the faecal matter of Adélie penguins (Pygoscelis adeliae) nesting on Ross Island, Antarctica. The genome had all the usual features of a papillomavirus and an E9 ORF encoding a protein of unknown function that is found in all avian papillomaviruses to date. This novel papillomavirus genome shared ~60 % pairwise identity with the genomes of the other three known avian papillomaviruses: Fringilla coelebs papillomavirus 1 (FcPV1), Francolinus leucoscepus papillomavirus 1 (FlPV1) and Psittacus erithacus papillomavirus 1. Pairwise identity analysis and phylogenetic analysis of the major capsid protein gene clearly indicated that it represents a novel species, which we named Pygoscelis adeliae papillomavirus 1 (PaCV1). No evidence of recombination was detected in the genome of PaCV1, but we did detect a recombinant region (119 nt) in the E6 gene of FlPV1 with the recombinant region being derived from ancestral FcPV1-like sequences. Previously only paramyxoviruses, orthomyxoviruses and avian pox viruses have been genetically identified in penguins; however, the majority of penguin viral identifications have been based on serology or histology. This is the first report, to our knowledge, of a papillomavirus associated with a penguin species.

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Carrai, Maura, Kate Van Brussel, Mang Shi, Ci-Xiu Li, Wei-Shan Chang, John S. Munday, Katja Voss, et al. "Identification of a Novel Papillomavirus Associated with Squamous Cell Carcinoma in a Domestic Cat." Viruses 12, no. 1 (January 20, 2020): 124. http://dx.doi.org/10.3390/v12010124.

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Papillomaviruses infect the skin and mucosal surfaces of diverse animal hosts with consequences ranging from asymptomatic colonization to highly malignant epithelial cancers. Increasing evidence suggests a role for papillomaviruses in the most common cutaneous malignancy of domestic cats, squamous cell carcinoma (SCC). Using total DNA sequencing we identified a novel feline papillomavirus in a nasal biopsy taken from a cat presenting with both nasal cavity lymphoma and recurrent squamous cell carcinoma affecting the nasal planum. We designate this novel virus as Felis catus papillomavirus 6 (FcaPV6). The complete FcaPV6 7453 bp genome was similar to those of other feline papillomaviruses and phylogenetic analysis revealed that it was most closely related to FcaPV3, although was distinct enough to represent a new viral type. Classification of FcaPV6 in a new genus alongside FcaPVs 3, 4 and 5 is supported. Archived excisional biopsy of the SCC, taken 20 months prior to presentation, was intensely positive on p16 immunostaining. FcaPV6, amplified using virus-specific, but not consensus, PCR, was the only papillomavirus detected in DNA extracted from the SCC. Conversely, renal lymphoma, sampled at necropsy two months after presentation, tested negative on FcaPV6-specific PCR. In sum, using metagenomics we demonstrate the presence of a novel feline papillomavirus in association with cutaneous squamous cell carcinoma.

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Terai, Masanori, Rob DeSalle, and Robert D. Burk. "Lack of Canonical E6 and E7 Open Reading Frames in Bird Papillomaviruses: Fringilla coelebs Papillomavirus and Psittacus erithacus timneh Papillomavirus." Journal of Virology 76, no. 19 (October 1, 2002): 10020–23. http://dx.doi.org/10.1128/jvi.76.19.10020-10023.2002.

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ABSTRACT Determination and analyses of the complete sequence of Fringilla coelebs papillomavirus and Psittacus erithacus timneh papillomavirus indicate that they represent a distinct and distant lineage of papillomaviruses. The lack of canonical E6-E7 open reading frames suggests that they serve adaptive functions during papillomavirus evolution.

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Nordin, Peter, Bengt Göran Hansson, Carita Hansson, Ingemar Blohmè, Olle Larkö, and Kristin Andersson. "Human Papilloma Virus in Skin, Mouth and Uterine Cervix in Female Renal Transplant Recipients With or Without a History of Cutaneous Squamous Cell Carcinoma." Acta Dermato-Venereologica 87, no. 3 (February 23, 2007): 219–22. http://dx.doi.org/10.2340/00015555-0235.

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Some human papillomaviruses are thought to be associated with skin cancer. In this pilot study, 21 female renal transplant carriers, 10 with a history of skin squamous cell carcinoma and 11 without, together with 9 age-matched healthy women were investigated for human papillomavirus DNA in sun-exposed (forehead) and less sun-exposed (buttock) skin, mouth and uterine cervix. Paraffin-embedded tumours from 9 of the patients with a history of squamous cell carcinoma were analysed. Healthy skin from both the healthy and the immunosuppressed individuals harboured a wide variety of papillomaviruses. In the healthy individuals, samples from less sun-exposed skin showed a lower prevalence of human papillomavirus DNA than corresponding samples from the immunosuppressed patients (4/9 and 7/9, respectively). Among the immunosuppressed patients, human papillomavirus DNA was found as frequently in buttock samples (17/21) as in forehead samples (17/20). There was no increased prevalence of human papillomavirus in the cervix or mouth samples from the immunosuppressed patients.

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White, Elizabeth A., Rebecca E. Kramer, Justin H. Hwang, Arun T. Pores Fernando, Nana Naetar, William C. Hahn, Thomas M. Roberts, Brian S. Schaffhausen, David M. Livingston, and Peter M. Howley. "Papillomavirus E7 Oncoproteins Share Functions with Polyomavirus Small T Antigens." Journal of Virology 89, no. 5 (December 24, 2014): 2857–65. http://dx.doi.org/10.1128/jvi.03282-14.

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ABSTRACTMany of the small DNA tumor viruses encode transforming proteins that function by targeting critical cellular pathways involved in cell proliferation and survival. In this study, we have examined whether some of the functions of the polyomavirus small T antigens (ST) are shared by the E6 and E7 oncoproteins of two oncogenic papillomaviruses. Using three different assays, we have found that E7 can provide some simian virus 40 (SV40) or murine polyomavirus (PyV) ST functions. Both human papillomavirus 16 (HPV16) and bovine papillomavirus (BPV1) E7 proteins are capable of partially substituting for SV40 ST in a transformation assay that also includes SV40 large T antigen, the catalytic subunit of cellular telomerase, and oncogenic Ras. Like SV40 ST, HPV16 E7 has the ability to override a quiescence block induced by mitogen deprivation. Like PyV ST, it also has the ability to inhibit myoblast differentiation. At least two of these activities are dependent upon the interaction of HPV16 E7 with retinoblastoma protein family members. For small T antigens, interaction with PP2A is needed for each of these functions. Even though there is no strong evidence that E6 or E7 share the ability of small T to interact with PP2A, E7 provides these functions related to cellular transformation.IMPORTANCEDNA tumor viruses have provided major insights into how cancers develop. Some viruses, like the human papillomaviruses, can cause cancer directly. Both the papillomaviruses and the polyomaviruses have served as tools for understanding pathways that are often perturbed in cancer. Here, we have compared the functions of transforming proteins from several DNA tumor viruses, including two papillomaviruses and two polyomaviruses. We tested the papillomavirus E6 and E7 oncoproteins in three functional assays and found that E7 can provide some or all of the functions of the SV40 small T antigen, another well-characterized oncoprotein, in two of these assays. In a third assay, papillomavirus E7 has the same effect as the murine polyomavirus small T protein. In summary, we report several new functions for the papillomavirus E7 proteins, which will contribute new insights into the roles of viruses in cancer and the cellular pathways they perturb in carcinogenesis.

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Dissertations / Theses on the topic "Papillomaviruses"

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Jolly, Carol Elizabeth. "The effects of leptomycin B on HPV-infected cells /." St Andrews, 2008. http://hdl.handle.net/10023/900.

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Tidy, John Anthony. "Human papillomaviruses and cervical neoplasia." Thesis, Imperial College London, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267104.

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Matsha, Tandi Edith. "Human Papillomaviruses in oesophageal cancer." Doctoral thesis, University of Cape Town, 2003. http://hdl.handle.net/11427/3140.

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Fothergill, Thomas. "Receptor signalling and internalisation of papillomaviruses /." [St. Lucia, Qld.], 2006. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19808.pdf.

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Brestovac, Brian. "Human papillomavirus and cervical cancer in Western Australia." University of Western Australia. School of Biomedical and Chemical Sciences, 2005. http://theses.library.uwa.edu.au/adt-WU2006.0037.

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Smith, Lisa G. "HPV knowledge of college females and their intention to receive the HPV vaccination." Virtual Press, 2008. http://liblink.bsu.edu/uhtbin/catkey/1399190.

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The purpose of this study was to determine the HPV knowledge of college women and their intentions to receive the HPV vaccination.An original data collection instrument was created. This instrument consisted of nine HPV knowledge questions, one intention to receive the vaccine stage question, and three demographic questions. Data were collected during Fall semester of 2007. The participants of this study were females (n=361) who were enrolled in an introductory personal health course. There was a statistically significant difference in intentions to receive the vaccine stage and HPV knowledge level. Those females who had higher mean knowledge scores were more likely to have made a decision about receiving the vaccine or they are still trying to decide. Those females who had lower mean knowledge scores were more likely to be unaware of the vaccine or have not thought about receiving the vaccine.
Department of Physiology and Health Science

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Wong, Weng-man Valerie. "Prevalence, genotypes and risk factors of human papillomavirus infection among women in Macao a cross-sectional study /." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B42998013.

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Cheung, Yim-hing. "Rare types and polymorphic variants of HPV in Hong Kong." Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25176559.

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Pakarian, Farzin Bouzorgmehr. "Perinatal transmission and persistence of human papillomaviruses." Thesis, University of Leicester, 1998. http://hdl.handle.net/2381/29553.

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This thesis investigates whether human papillomaviruses are transmitted from mothers to their infants. Cervical/vaginal swabs were taken from 69 pregnant women. Buccal and genital samples were taken from their infants (all delivered vaginally) at 24 h (n=70; one set of twins), six weeks (n-49) and six months (n=19). All samples were examined for HPV-6, -11, -16, -18, -31 and -33 us polymerase chain reaction (PCR). Thirty seven (54%) women had detectable detectable in sixteen (23%) infants at 24 h and seven (14%) at six weeks; a perinatal transmission rate of 23% (6/37) and persistence of 14% (7/37). All infants tested at six months were HPV negative. At 24h, HPV-6 was demonstrated in 9 mother-infant pairs, HPV-18 in 1 mother-infant pair, HPV-11 in 1 mother-infant pair, dual infection with HPV type 16 and 18 in 3 mother-infant pairs. Two infants with HPV-18 were delivered to mothers in HPV-16/18. At 6 weeks, 5 infants remained HOV-16 positive, one infant HPV-18 positive, whilst one infant who was HPV 16/18 positive was now HPV-16 positive. To examine whether HPVs were acquired in utero or intrapartum, genital swabs were collected from 33 women who had amniotic fluid collected either at caesarean section (n=29) or at amniocentesis (n=4). Analysis of these samples demonstrated genital HPV in 10 (10%) of the women and none of the amniotic fluid samples. To demonstrate persistence and source of infectivity at 2 years, the same group sequenced a 521 bp segment of the upstream regulatory (URR) of HPV-16 DNA isolated from 13 maternal samples, samples taken from their infants. In conclusion, HPVs can be transmitted perinatally. HPV DNA persisted up to 2 years of age. In addition women with a high genital HPV load were more likely to transmit the virus.

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Shah, S. D. "Understanding the evolutionary history of the papillomaviruses." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1366951/.

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This thesis focuses on the evolutionary history of the papillomaviruses (PVs) using phylogenetic approaches. Two aspects have been examined: the first is the level of phylogenetic compatibility among PV genes and the second is determining the ancestral diversification mechanisms of the PVs in order to explain the origin of the observed associations with host species. Bayesian phylogenetic analysis has been used to make evolutionary inferences. The existence of phylogenetic compatibility among genes was examined by estimating constrained and unconstrained phylogenies for pairs of PV genes. The Bayes' factor statistic derived from comparison of the constrained and unconstrained models indicated significant evidence against identical phylogenies between any of the 6 PV genes investigated and may indicate the existence of ancestral recombination events. The formation of new host-virus associations can occur via a process of 'codivergence', where, following host speciation, the ancestral virus association is effectively inherited by the descendant host species; 'prior divergence' of the virus, which results in multiple virus associations with the host; and 'host transfer', in which the virus lineage is transferred between contemporaneous host species. To distinguish between these mechanisms of virus diversification, an approach based on temporal comparisons of host and virus divergence times was devised. Difficulties associated with the direct estimation of PV divergence times led to the incorporation of a biased sampling approach into Bayesian phylogenetic estimation. This allowed for viral divergence events to be biased in favour of codivergence but allowed sampling of times that violate this assumption and therefore indicate either prior divergence or host transfer. Statistical evaluation of the proportion of violations at each viral divergence identified significant evidence of prior divergence events behind many of the observed PV-host associations and one ancestral host transfer event.

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Books on the topic "Papillomaviruses"

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M, Steinberg Bettie, Brandsma Janet L, and Taichman Lorne B, eds. Papillomaviruses. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1987.

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David, Evered, and Clark Sarah, eds. Papillomaviruses. Chichester: n Wiley, 1986.

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Charles, Lacey, and Leeds Medical Information, eds. Papillomavirus reviews: Current research on papillomaviruses. Leeds: Leeds Medical Information, 1996.

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Symposium on Papillomaviruses (1985 : Ciba Foundation), ed. Papillomaviruses. Chichester: J. Wiley, 1986.

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Davy, Clare, and John Doorbar. Human Papillomaviruses. New Jersey: Humana Press, 2005. http://dx.doi.org/10.1385/1592599826.

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IARC, Working Group on the Evaluation of Carcinogenic Risks to Humans (1995 Lyon France). Human papillomaviruses. [Lyon, France?]: International Agency for Research on Cancer, 1995.

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Garcea, Robert L., and Daniel DiMaio, eds. The Papillomaviruses. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7.

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J, Syrjänen Kari, Gissmann L. 1949-, and Koss Leopold G, eds. Papillomaviruses and human disease. Berlin: Springer-Verlag, 1987.

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Jane, Sterling, and Tyring Stephen K, eds. Human papillomaviruses: Clinical and scientific advances. London: Arnold, 2001.

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zur Hausen, Harald, ed. Human Pathogenic Papillomaviruses. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78487-3.

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Book chapters on the topic "Papillomaviruses"

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Pfister, Herbert. "Papillomaviruses." In The Papovaviridae, 1–38. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4757-0584-3_1.

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Klein, George, and Daniel DiMaio. "Principles of Human Tumor Virology." In The Papillomaviruses, 1–11. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_1.

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Münger, Karl, Peter Howley, and Daniel DiMaio. "Human Papillomavirus E6 and E7 Oncogenes." In The Papillomaviruses, 197–252. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_10.

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Lambert, Paul F., and Anne E. Griep. "In Vivo Models for the Study of Animal and Human Papillomaviruses." In The Papillomaviruses, 253–75. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_11.

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Egelkrout, Erin M., and Denise A. Galloway. "The Humoral Immune Response to Human Papillomavirus." In The Papillomaviruses, 277–312. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_12.

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Lyons, Gretchen Eiben, Michael I. Nishimura, and Martin W. Kast. "Cell-Mediated Immune Responses to Human Papillomavirus." In The Papillomaviruses, 313–35. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_13.

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Schiller, John. "Papillomavirus Vaccines." In The Papillomaviruses, 337–69. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_14.

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Crum, Christopher P., and Ralph M. Richart. "Clinical Assessment, Therapies, New Tests, and Algorithms." In The Papillomaviruses, 371–86. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_15.

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Pagliusi, Sonia R., M. Teresa Aguado, and D. Maxwell Parkin. "Possible Worldwide Impact of Prevention of Human Papillomavirus Infection." In The Papillomaviruses, 387–413. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_16.

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Lowy, Douglas R. "History of Papillomavirus Research." In The Papillomaviruses, 13–28. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-36523-7_2.

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Conference papers on the topic "Papillomaviruses"

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Tommasino, Massimo. "Abstract IA15: Role of human papillomaviruses in carcinogenesis." In Abstracts: AACR Special Conference on the Microbiome, Viruses, and Cancer; February 21-24, 2020; Orlando, FL. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.mvc2020-ia15.

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Asante, Jessica, Destini McMillan, and Juan Peticco. "Protein association and nucleotide sequence similarities among human alpha-papillomaviruses." In 2016 IEEE Integrated STEM Education Conference (ISEC). IEEE, 2016. http://dx.doi.org/10.1109/isecon.2016.7457565.

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Münger, Karl. "Abstract IA27: Perturbation of host cellular regulatory networks by human papillomaviruses." In Abstracts: AACR Precision Medicine Series: Cancer Cell Cycle - Tumor Progression and Therapeutic Response; February 28 - March 2, 2016; Orlando, FL. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.cellcycle16-ia27.

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Pu, Wang, and Xiao Xuan. "Predicting the Risk Type of Human Papillomaviruses Based on Sequence-Derived Features." In 2011 5th International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2011. http://dx.doi.org/10.1109/icbbe.2011.5779985.

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Faizuloev, E. B., A. N. Kaira, T. R. Uzbekov, A. A. Poromov, E. A. Volynskaya, O. A. Svitich, and V. V. Zverev. "GENETIC STRUCTURE OF HIGH AND LOW RISK HUMAN PAPILLOMAVIRUSES CIRCULATING IN THE RUSSIAN FEDERATION." In Molecular Diagnostics and Biosafety. Federal Budget Institute of Science 'Central Research Institute for Epidemiology', 2020. http://dx.doi.org/10.36233/978-5-9900432-9-9-123.

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Xiao, Xuan, and Pu Wang. "A new approach using geometric moments of distance matrix image for risk type prediction of human papillomaviruses." In 2011 International Conference on Electronics, Communications and Control (ICECC). IEEE, 2011. http://dx.doi.org/10.1109/icecc.2011.6067633.

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"The study of the cross-interaction of antibodies with antigens of different types of human papillomaviruses (HPV)." In Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Novosibirsk ICG SB RAS 2021, 2021. http://dx.doi.org/10.18699/plantgen2021-193.

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Farzan, Shohreh F., Tim Waterboer, Jiang Gui, Heather Nelson, Zhongze Li, Kristina Michael, Ann Perry, et al. "Abstract 2289: Cutaneous alpha, beta and gamma human papillomaviruses in relation to basal cell and squamous cell carcinoma." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2289.

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Idoko, John, Mohammed Faruk, Jigo Yaro, James Enemari, and Peter Akpulu. "Abstract 5339: Molecular phylogenetic classification of human papillomaviruses and cancer associated genes: In correlation with cervical cancer manifestation." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5339.

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Moreno, Ana Carolina Ramos, Bruna F. M. M. Porchia, Patrícia da Cruz Souza, Roberta Liberato Pagni, Rafael Pegoraro, and Luís Carlos de Souza Ferreira. "Abstract B55: Targeting indoleamine 2,3 dioxygenase to improve the efficacy of a therapeutic vaccine against human papillomaviruses-associated tumors." In Abstracts: AACR International Conference held in cooperation with the Latin American Cooperative Oncology Group (LACOG) on Translational Cancer Medicine; May 4-6, 2017; São Paulo, Brazil. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.tcm17-b55.

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Reports on the topic "Papillomaviruses"

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Clements, Adrienne M. Structural Studies of the pRB Tumor Suppressor Complexed with Human Papillomavirus E7. Fort Belvoir, VA: Defense Technical Information Center, June 1998. http://dx.doi.org/10.21236/ada354361.

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Jomah, Arwa, and Afnan Albokhary. The Impact of Human Papillomavirus Vaccine on Women’s Health: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2023. http://dx.doi.org/10.37766/inplasy2023.12.0029.

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Villarroel, Maria, Adena Galinksy, Peng-Jun Lu, Cassandra Pingali, and Claudia Valenzuela. Human Papillomavirus Vaccination Coverage in Children Ages 9–17 Years: United States, 2022. National Center for Health Statistics (U.S.), February 2024. http://dx.doi.org/10.15620/cdc:145593.

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van de Sande, Anna, Malika Kengsakul, Margot Koeneman, Marta Jozwiak, Cornelis Gerestein, Arnold-Jan Kruse, Edith van Esch, et al. Imiquimod in cervical dysplasia: a review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0046.

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Review question / Objective: To determine the efficacy of topical imiquimod in treatment of high-grade CIN (defined as regression CIN 1 or less), and to determine the clearance rate of high-risk human papillomavirus (hr-HPV), compared to surgical treatment and placebo. Condition being studied: Women with an untreated, histologically proven, CIN2-3 lesion or women who were persistent high-risk HPV positive. Eligibility criteria: Studies that evaluated the efficacy of imiquimod treatment in intraepithelial lesions or malignancy of other organs, and studies published as conference abstract, narrative review, editorial, letter, or short communication were excluded.

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Li, Yanhui. Prevalence of human papillomavirus DNA and p16INK4apositivity in vaginal cancer and vaginal intraepithelial neoplasia: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2023. http://dx.doi.org/10.37766/inplasy2023.11.0022.

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Li, Yanhui. Efficacy of non-invasive photodynamic therapy for female lower reproductive tract diseases associated with HPV infection: a comprehensive meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0092.

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Review question / Objective: The critical point of this study was to comprehensively evaluate the curative effect of Photodynamic therapy (PDT) in diseases of female lower reproductive tract associated with the human papillomavirus (HPV) infection. Condition being studied: Traditional clinical recommendations for treating diseases of the female lower reproductive tract include topical therapy with drugs, surgery, intravaginal radiation, carbon dioxide (CO2) laser, etc. Although medication is easy to administer, it has a high recurrence rate and adverse effects such as burning sensation, pain, and dyspareunia. The other traditional treatment method is usually invasive, repeated operation of vaginal perforation, scar, easy recurrence, fertility decline, and other shortcomings. At present, the treatment strategy for cervical squamous intraepithelial lesion, vaginal squamous intraepithelial lesion, condyloma acuminatum, and vulvar lichen sclerosis are to protect the normal organ structure and function as much as possible, reduce recurrence, prevent disease progression and carcinogenesis, and preserve female reproductive function.

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Aidibai, Simayi, and Jin Hui. Association between human papillomavirus vaccination and serious adverse events in females aged 9 to 26 years: a systematic review and meta analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2022. http://dx.doi.org/10.37766/inplasy2022.2.0040.

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Wang, Huanhuan, Yuyu Zhang, Wei Bai, Bin Wang, Jinlong Wei, Ji Rui, Ying Xin, Lihua Dong, and Xin Jiang. Feasibility of immunohistochemical p16 staining in the diagnosis of human papillomavirus infection in patients with squamous cell carcinoma of the head and neck: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2020. http://dx.doi.org/10.37766/inplasy2020.7.0068.

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Gidengil, Courtney, Matthew Bidwell Goetz, Margaret Maglione, Sydne J. Newberry, Peggy Chen, Kelsey O’Hollaren, Nabeel Qureshi, et al. Safety of Vaccines Used for Routine Immunization in the United States: An Update. Agency for Healthcare Research and Quality (AHRQ), May 2021. http://dx.doi.org/10.23970/ahrqepccer244.

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Objective. To conduct a systematic review of the literature on the safety of vaccines recommended for routine immunization in the United States, updating the 2014 Agency for Healthcare Research and Quality (AHRQ) report on the topic. Data sources. We searched MEDLINE®, Embase®, CINAHL®, Cochrane CENTRAL, Web of Science, and Scopus through November 9, 2020, building on the prior 2014 report; reviewed existing reviews, trial registries, and supplemental material submitted to AHRQ; and consulted with experts. Review methods. This report addressed three Key Questions (KQs) on the safety of vaccines currently in use in the United States and included in the Centers for Disease Control and Prevention’s (CDC) recommended immunization schedules for adults (KQ1), children and adolescents (KQ2), and pregnant women (KQ3). The systematic review was supported by a Technical Expert Panel that identified key adverse events of particular concern. Two reviewers independently screened publications; data were extracted by an experienced subject matter expert. Studies of vaccines that used a comparator and reported the presence or absence of adverse events were eligible. We documented observed rates and assessed the relative risks for key adverse events. We assessed the strength of evidence (SoE) across the existing findings from the prior 2014 report and the new evidence from this update. The systematic review is registered in PROSPERO (CRD42020180089). Results. A large body of evidence is available to evaluate adverse events following vaccination. Of 56,608 reviewed citations, 189 studies met inclusion criteria for this update, adding to data in the prior 2014 report, for a total of 338 included studies reported in 518 publications. Regarding vaccines recommended for adults (KQ1), we found either no new evidence of increased risk for key adverse events with varied SoE or insufficient evidence in this update, including for newer vaccines such as recombinant influenza vaccine, adjuvanted inactivated influenza vaccine, and recombinant adjuvanted zoster vaccine. The prior 2014 report noted a signal for anaphylaxis for hepatitis B vaccines in adults with yeast allergy and for tetanus, diphtheria, and acellular pertussis vaccines. Regarding vaccines recommended for children and adolescents (KQ2), we found either no new evidence of increased risk for key adverse events with varied SoE or insufficient evidence, including for newer vaccines such as 9-valent human papillomavirus vaccine and meningococcal B vaccine. The prior 2014 report noted signals for rare adverse events—such as anaphylaxis, idiopathic thrombocytopenic purpura, and febrile seizures—with some childhood vaccines. Regarding vaccines recommended for pregnant women (KQ3), we found no evidence of increased risk for key adverse events with varied SoE among either pregnant women or their infants following administration of tetanus, diphtheria, and acellular pertussis vaccines during pregnancy. Conclusion. Across this large body of research, we found no new evidence of increased risk since the prior 2014 report for key adverse events following administration of vaccines that are routinely recommended. Signals from the prior report remain unchanged for rare adverse events, which include anaphylaxis in adults and children, and febrile seizures and idiopathic thrombocytopenic purpura in children. There is no evidence of increased risk of adverse events for vaccines currently recommended in pregnant women. There remains insufficient evidence to draw conclusions about some rare potential adverse events.

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Cancers Associated with Human Papillomavirus by State—2010–2014. United States Cancer Statistics (USCS), March 2018. http://dx.doi.org/10.15620/cdc:56915.

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