Relevant bibliographies by topics / Pain epidemiology

Academic literature on the topic 'Pain epidemiology'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Pain epidemiology"

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Green, C., X. Lin, and S. Ndao-Brumblay. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S119. http://dx.doi.org/10.1016/j.jpain.2004.02.446.

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Reyes-Gibby, C., and C. Cleeland. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S119. http://dx.doi.org/10.1016/j.jpain.2004.02.447.

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Jensen, M., R. Abresch, G. Carter, and C. McDonald. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S120. http://dx.doi.org/10.1016/j.jpain.2004.02.448.

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McWilliams, L., R. Goodwin, and B. Cox. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S120. http://dx.doi.org/10.1016/j.jpain.2004.02.449.

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Roth, T., T. Mundel, S. Martin, L. LaMoreaux, and L. Hotary. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S120. http://dx.doi.org/10.1016/j.jpain.2004.02.450.

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Kessler, R., N. Brandenburg, M. Lane, and P. Stang. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S120. http://dx.doi.org/10.1016/j.jpain.2004.02.451.

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Hoffman, A., C. Warms, H. Marshall, and E. Tyler. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S121. http://dx.doi.org/10.1016/j.jpain.2004.02.452.

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Vargas, C., R. Scott, and N. Tinanoff. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S121. http://dx.doi.org/10.1016/j.jpain.2004.02.453.

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Jeon, Y., X. Wang, H. Guo, P. Jones, and C. Cleeland. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S121. http://dx.doi.org/10.1016/j.jpain.2004.02.454.

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Lan, M. "Pain epidemiology." Journal of Pain 5, no. 3 (April 2004): S121. http://dx.doi.org/10.1016/j.jpain.2004.02.455.

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Dissertations / Theses on the topic "Pain epidemiology"

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Aggarwal, Vishal R. K. "Epidemiology of chronic oro-facial pain." Thesis, University of Manchester, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.496345.

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The principal aims of the present study were to determine, in an unselected general population: (a) whether chronic oro-facial pain co-occurs with other frequently unexplained symptoms (b) whether factors associated with chronic oro-facial pain are common across symptoms. A population-based cross-sectional study was conducted using 4200 randomly selected adults who were recruited from the age-sex register of a General Medical Practice in North West, England. The study examined the prevalence and co-occurrence of chronic oro-facial pain with three other chronic symptoms that are frequently unexplained: chronic widespread pain, Irritable bowel syndrome and chronic fatigue. Validated instruments were used to measure the occurrence of symptoms and to collect information on a variety of associated factors: demographic (age and gender), psychosocial (anxiety, depression, illness behaviour, life stressors and reporting of somatic symptoms), mechanical (teeth grinding, facial trauma, missing teeth and reporting that the teeth did not fit together properly). 2505 subjects returned completed questionnaires (adjusted response rate 72%). The prevalence of Chronic Widespread Pain was 15%. Chronic Oro-facial Pain 7%. Irritable Bowel Syndrome 9% and Chronic Fatigue 8%. The study found that 587 subjects (27%) reported one or more symptoms: 404 (18%) reported one symptom, 134 (6%) reported two, 34 (2%) reported three, whilst 15 (1%) reported all four symptoms. This study has shown that chronic symptoms (including Chronic Oro-facial pain) that are frequently unexplained co-occur in the general population and share common associated factors. These findings are consistent with the hypothesis that chronic oro-facial pain may share a common aetiology with other frequently unexplained symptoms although this needs to be confirmed in a prospective study.
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Xu, Daquan 1965. "Epidemiology of pain and pain management after knee surgery : arthroplasty and arthroscopy." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=80899.

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Background. Pain after knee surgery has been reported as a common problem. It is highly ranked in terms of intensity and has important consequences on both quality of life and psychological well-being. However, assessment and management of postoperative pain remain a key clinical problem.
Objectives. To describe the occurrence of pain after total knee arthroplasty (TKA) and knee arthroscopy; identify the predictors of postoperative pain and evaluate the consequences of pain on quality of life and on depression status.
Methods. Patients were recruited from nine university and regional hospitals in the province of Quebec and were followed for three months after knee surgery. Time points of postoperative day 7 and month 3 were our prime interest. We used a prospective cohort design to investigate characteristics of postoperative pain and a case-control design to identify the impact of postoperative pain on quality of life and on depression. Both logistic regression and multiple linear regression models were used to analyze postoperative pain intensity and the impact of postoperative pain respectively. (Abstract shortened by UMI.)
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Zondervan, Krina Tynke. "The epidemiology of chronic pelvic pain in women." Thesis, University of Oxford, 1999. http://ora.ox.ac.uk/objects/uuid:7ecabb07-7d48-4da0-8a51-8b303ec08086.

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Macfarlane, Gary J. "The epidemiology of regional and widespread pain syndromes." Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394971.

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Docking, Rachael Elizabeth. "The epidemiology of back pain in older adults." Thesis, University of Aberdeen, 2012. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=203834.

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There is currently little primary data looking at the epidemiology of back pain in older people and there are few large-scale population based studies which consider the occurrence or aetiology of back pain within this age group. Some evidence suggests that while non-disabling back pain decreases in the oldest old, the prevalence of disabling back pain may continue to increase. However, the aetiology of back pain in this group remains relatively unknown. Therefore, the overall aim of the current thesis was to investigate the epidemiology of back pain in older people, to examine the occurrence of back pain and age-related patterns in prevalence and incidence (descriptive epidemiology) and to identify potential risk factors for predicting back pain onset in those ≥75 years, specifically, to determine the role of social networks (analytical epidemiology). This was done through secondary analysis of a prospective cohort study from Cambridge looking at older people ≥75 years. It has been shown that while the prevalence of non-disabling back pain did not vary significantly across age, the prevalence of disabling back pain increased with age. In addition, it has been demonstrated, firstly, that aspects and indicators of physical health and a prior history of back pain are associated and independent predictors of back pain in older people; and secondly, that while objective measures of social contact are not risk markers for back pain, low mood, feelings of social isolation and depression are more strongly associated. These findings were further verified and confirmed in a second study, a cross-sectional survey of people ≥65 years living in rural Scotland. It can therefore be concluded that back pain is a common problem in the older population and, while mild back pain may remain constant across older age, disabling back pain continues to increase into the oldest old. The risk profile for back pain in older age is multifactorial, while some risk factors evident in younger populations remain; there is greater emphasis on the impact of mood and psychosocial factors.
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Ayorinde, Abimbola. "The epidemiology of chronic pelvic pain in women." Thesis, University of Aberdeen, 2016. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229702.

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Background: Epidemiological studies on chronic pelvic pain (CPP) mainly focused on women of reproductive age so it is not clear whether there is any evidence of a distinct epidemiology at older ages. Therefore, the primary aim of this study was to establish the epidemiological characteristics of CPP whilst including women beyond the reproductive age. Methods: A cross-sectional postal survey was conducted amongst 5300 randomly selected women aged ≥25 years resident in the Grampian region, UK. Factors associated with CPP, poor health state and passive/active coping strategies were assessed using logistic regression. To identify sub-groups of CPP cases, cluster analysis was performed using variables of pain severity, psychosocial factors and pain coping strategies. Results: Of 2088 participants, 309 reported CPP (prevalence 14.8%). Prevalence was higher among women of reproductive years compared to older women (20.5% v. 9.6%). CPP was significantly associated with having multiple non-pain somatic symptoms, having fatigue and depression. CPP was more strongly associated with multiple non-pain somatic symptoms among older women compared to women of reproductive age but no other factor was found to be differentially related to CPP in the post-reproductive years. Poor health states among CPP cases were not strongly associated with pain intensity and duration but associated with depression, sleep disturbance, fatigue and passive coping. Women with CPP often engage passive coping strategy which was strongly associated with severe and constant pain, fatigue and multiple somatic symptoms. Two distinct groups of CPP cases characterised by the absence/presence of psychosocial distress were identified. Conclusion: This study provides a more accurate prevalence estimate for CPP which will aid healthcare planning and resource allocation. The associations identified for CPP are similar to those often reported for chronic pain conditions suggesting that they may have similar underlying mechanism. This study offers a basis for additional comprehensive and prospective evaluation of CPP and could inform the development of CPP management strategies.
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Flüss, Elisa. "The epidemiology of pain : improving the methodologies used in population-based studies of pain." Thesis, University of Aberdeen, 2014. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=211414.

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Introduction: Currently used methods to collect self-reported information on pain status in population studies have remained unchanged over the past decades. Decreasing response rates and the necessity to collect more detailed information to advance on understanding declare the importance of updating these instruments. The aims of this thesis were to (1) explore different strategies to increase response rates and (2) assess whether population estimates of pain change when participants' pain management is considered for the pain assessment. Methods: A cross-sectional study was conducted in the general population in Grampian, Scotland. 4600 potential participants were randomly selected from the NHS community health index register and sent a self-completion questionnaire. Collected information included people's health status (SF-36), pain status (manikins, chronic pain grade), pain management and estimated pain status without pain treatments (enhanced pain status questionnaire). The newly developed pain management and enhanced pain status questionnaire were validated within a small sample of the questionnaire respondents who attended a subsequent interview. Results: The response rate was not significantly increased when single-sided questionnaires were sent to potential participants (OR=1.13, 95%CI=0.998, 1.28) or when an option to reply via the internet was provided (OR=1.06, 95%CI=0.94, 1.20). Population estimates of pain changed significantly with the use of the enhanced pain status questionnaire: the pain prevalence increased from 50.5% to 56.2% (difference=5.7%, 95% CI=2.2, 9.2). Likewise, respondents with pain estimated their pain to be significantly higher when they considered the effect of their treatments. Conclusions: Neither an option to reply via the internet nor single-sided questionnaires are effective methods to increase response rates to population studies. Conclusions: Neither an option to reply via the internet nor single-sided questionnaires are effective methods to increase response rates to population studies. The burden of pain is currently underestimated in population-based pain studies which do not include an assessment of pain management. As a result of the findings, pain management information should routinely be collected and considered in epidemiological studies assessing pain.
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Laurell, Katarina. "Headache in Schoolchildren : Epidemiology, Pain Comorbidity and Psychosocial Factors." Doctoral thesis, Uppsala University, Neurology, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-5850.

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Headache is the most frequently reported pain in children and is associated with missed schooldays, anxiety, depressive symptoms and various physical symptoms. A secular trend of increasing headache prevalence has been suggested. Few studies have focused on tension-type headache among children from the general population.

The aims of this thesis were to describe the prevalence, incidence and prognosis of tension-type headache, migraine and overall headache in schoolchildren, to identify medical, psychological and social factors associated with these headache types, and to determine whether the prevalence of headache has increased over the last decades.

In 1997, 1850 schoolchildren aged 7-15 years from the city of Uppsala participated in a questionnaire study and 1371 (74.1%) responded. Out of these, a randomly selected, stratified sample of 131 children and their parents were interviewed. Three years later, 122 children from the interview sample replied to an identical headache questionnaire.

Compared with a similar study in 1955, a significantly lower proportion of schoolchildren reported no headache. The prevalence of tension-type headache increased with age and was significantly higher in girls than boys after the age of twelve. Similar age and gender differences were obtained for migraine. A higher proportion of girls reported frequent headache than boys. Children with headache, especially those with migraine, as well as their first-degree relatives suffered from other pains and physical symptoms more frequently than headache-free children and their first-degree relatives. Although the likelihood of experiencing the same headache diagnosis and symptoms at follow-up was high, about one fifth of children with migraine developed tension-type headache and vice versa. Female gender was a predictor of migraine and frequent headache a predictor of overall headache at follow-up. The estimated annual incidence for tension-type headache, migraine and overall headache was 81, 65 and 131 per 1000 children, respectively.

In conclusion, the results indicate that headache has become increasingly common among schoolchildren over the last decades. Prevention and treatment of headache is particularly important for girls since they have high prevalence of headache, frequent headache episodes and a poor outcome. In children with headache, diagnoses and treatment should be reassessed regularly and other pains should be asked about and treated as well.

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Harvey, Hollie. "Epidemiology and genetics of catastrophizing and pain in OA." Thesis, University of Nottingham, 2017. http://eprints.nottingham.ac.uk/40041/.

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Background: Pain is the main reason patients present with Osteoarthritis (OA). There are many risk factors for increased pain but one risk factor that is the focus of this thesis is catastrophizing. There is a discrepancy between the amount of radiographic damage to the joint and the pain intensity felt by the OA patient, which leads to the hypothesis that there must be other non-structural factors such as catastrophizing that could explain this discrepancy. Objectives: (1) To assess the effect of catastrophizing on joint and body pain in individuals who have undergone total joint replacement surgery after accounting for sleep quality, anxiety and depression in people diagnosed with large joint OA and controls. X-rays and resulting K/L grade also used to account for joint damage. (2) To identify if there are specific genes and pathways that could explain the relationship between catastrophizing and pain, also whether catastrophizing is a trait in its own right which is not dependent on depression or bad quality sleep. (3) To identify genetic factors which contribute to the effect of catastrophizing on pain intensity in OA by comparing those with symptomatic OA to those with asymptomatic OA. Methods: Pain post- Total Joint Replacement (TJR) (Chapter 3): This is a case-control study design. Cases were derived from the Nottingham genetics of OA study (NGOAS) and the Genetics of Osteoarthritis and Lifestyle Study (GOAL) cohorts that have undergone joint replacement. Controls were derived from participants in the GOAL cohort that were selected as controls for OA. 1093 individuals from the NGOAS cohort who had been diagnosed with knee or hip OA were recruited from secondary care (82% post-total joint replacement (TJR) none on waiting list) and completed a questionnaire which included items on: joint pain, neuropathic pain like symptoms (using the painDETECT questionnaire), anxiety (using the hospital anxiety and depression scale (HADS) questionnaire), depression (using the HADS questionnaire) and the 13-item pain catastrophizing scale (PCS). A similar questionnaire was sent to participants from a case control OA study known as the GOAL involving 679 cases all with hip or knee OA (59.7% post-TJR, none on waiting list) and 402 non-OA controls. TJR used as cases due to this being the end point of OA with those undergoing TJR most likely in pain from OA. High pain catastrophizing was defined as being in the top tertile of the PCS (score≥9). SPSS was then used to calculate descriptive statistics such as percentage of participants that were female, mean age, BMI. This would give a picture of the cohort being used. Then binary logistic regressions were used to calculate odds ratios (ORs) for sleep, depression, anxiety and catastrophizing whilst adjusting for confounding factors such as age, BMI, gender. Genetics (Chapter 4 and 5): Quantile-Quantile (QQ) and Manhattan plots were produced using R to quality test the genomic data. A Manhattan plot is a type of scatter plot, usually used to display data with a large number of data-points - many of non-zero amplitude, and with a distribution of higher-magnitude values, for instance in genome-wide association studies (GWAS). The statistics program R (version 3.0.2) was used to create Manhattan and QQ plots using the “ggplot2” library and “qqplot” script. A genome wide association scan (GWAS) and single nucleotide polymorphism (SNP) extractions, conducted using Plink, were carried out on a subset of 1113 post TJR cases from the NGOAS cohort and pathway analysis was then conducted based on significant SNPs found in the GWAS. In chapter 4 replication and meta-analysis were then carried out using a separate cohort of 679 participants, taken from the GOAL study. Results: Pain post- total joint replacement (TJR) (Chapter 3): The prevalence of high catastrophizing was not significantly higher in OA and post TJR cases (36.8%) than in controls (33.1%)(p<0.07), however as controls were from intravenous urography (IVU) clinics they would have higher levels of cardio-vascular disease, stroke, kidney disease for example than general population. The main factors associated with catastrophizing were older age, higher body mass index, high pain intensity, anxiety, depression and multiple regional pain. High catastrophizing was associated with increased risk of severe joint (hip or knee) pain after adjustment for sleep quality, anxiety and depression (OR= 2.99, 95% CI= 2.20-4.04, p< 1.54E-12). Similarly, high catastrophizing was associated with body pain (OR=1.47, CI=1.16-1.86, P<0.0002) and neuropathic-like pain symptoms (OR=5.35, CI=4.16-6.90, P<1.42E-35). I also explored if these associations with pain were due to the presence of comorbidities which are common in OA patients and post-TJR. I did find that pain catastrophizing scores were also associated with cardiovascular disease (CVD) after adjustment for depression and sleep quality as well as BMI, age and gender (OR= 1.44, 95% CI= 1.14-1.81, p< 0.001). Genetics (Chapter 4 and 5): Several SNPs were linked to high catastrophizing scores with a p-value <0.00005. ADRA2C was the only gene associated with catastrophizing that replicated in chapter 4 after meta-analysis. This variant was associated with OR=0.67, 95% CI= 0.56-0.80, p-value=7.8x10[-6]. It is involved in the formation of adrenergic receptors and therefore the regulation of possibly many pain pathways including the sympathetic nervous system and the hypothalamic–pituitary–adrenal axis as well as being required for normal presynaptic control of transmitter release in the heart. PRR20 was another interesting finding in chapter 4. It is involved in dopamine receptors, so possibly influencing that particular pain pathway. These data demonstrate that catastrophizing does influence pain intensity and implicates certain molecular pathways that may be involved in this relationship. LEPRE1 and COL6A1 are both implicated in chapter 5. Both of these genes are involved in collagen formation and destruction thereby could affect the progression of OA and joint structure leading to pain. LEPRE1 is further known as one of the genetic causes of Osteogenesis imperfecta type VIII another bone formation disorder. Conclusion: Pain catastrophizing is associated with joint and body pain in individuals with OA after TJR and this effect is not mediated by sleep quality, anxiety or depression. Therefore catastrophizing can be said to be a trait in its own right. Many genes and pain pathways were implicated by the genetics studies, mainly involving adrenaline and dopamine, so more focus on these neurotransmitters could be suggested for future study. Another interesting finding that could be investigated further is the link between cardiovascular disorders and catastrophizing as this is a relatively unexplored area, made more interesting by the finding of ADRA2C both indicating involvement of the sympathetic nervous system and other central mechanisms.
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Chen, Lingxiao. "Exploring the heterogeneity of musculoskeletal pain." Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/27464.

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Musculoskeletal pain often includes pain in the back, neck, knee, and hip, and is associated with a substantial financial and personal burden. Eight chapters are included in this thesis that aims to improve the understanding of the heterogeneity in treatment effects and prognosis of musculoskeletal pain. Four issues were identified: i) people with different pain phenotypes (i.e. back pain with or without neurological deficit) or with distinct underlying health conditions (e.g. pregnancy-related back pain) may respond differently to treatment strategies; ii) people with chronic back pain and presenting different radiological phenotypes may experience different course of the disease; iii) different patterns of analgesic use over time may be associated with different long term health status; iv) different types and number of sites of musculoskeletal pain may be associated with different clinical prognoses.
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Books on the topic "Pain epidemiology"

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K, Crombie I., ed. Epidemiology of pain: A report of the Task Force on Epidemiology of the International Association for the Study of Pain. Seattle: IASP Press, 1999.

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Chronic pain epidemiology: From aetiology to public health. Oxford: Oxford University Press, 2010.

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Pain in psychiatric disorders. Basel: Karger, 2015.

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Back pain and sciatica in Finnish farmers. Helsinki: Social Insurance Institution, Research Institute for Social Security, 1987.

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Page, Frederick T. Occupational groups and the occurrence of musculo-skeletal disorders in industry: "an ergonomic approach". Bournemouth, England: Anglo-European College of Chiropractic, 1985.

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Turczyn, Kathleen M. Inventory of pain data from the National Center for Health Statistics. Hyattsville, Md: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, National Center for Health Statistics, 1992.

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Wessex Institute for Health Research and Development., ed. Low back pain: Health care needs asessment : the epidemiologically based needs assessment reviews, second series. Oxford: Radcliffe Medical Press, 1997.

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Hmara, Ivan, and Viktor Strel'nikov. Environmental epidemiology and risk assessment. ru: INFRA-M Academic Publishing LLC., 2021. http://dx.doi.org/10.12737/1019063.

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The material presented in the textbook is based on modern ideas about environmental epidemiology as an interdisciplinary field of knowledge, the purpose of which is a multi — level study of the "environment-human health"system. Special attention is paid to the issues of risk assessment as an integral part of ecoepidemiological research. It corresponds to the program of the discipline "Environmental Epidemiology", approved by the Scientific and Methodological Council for Environmental Education of UMO Universities. For students in the field of training 05.03.06 "Ecology and nature management", as well as related biological, environmental and medical areas and specialists of the relevant work profiles.
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National Research Council (U.S.). Panel on Musculoskeletal Disorders and the Workplace. and Institute of Medicine (U.S.), eds. Musculoskeletal disorders and the workplace: Low back and upper extremities. Washington, D.C: National Academy Press, 2001.

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Maria dos Prazeres Cardoso Lourenço Pires Ferreira. Estudo epidemiológico e microbiológico do fluxo vaginal: Aspectos ḑa infecção pelo VIH : estudo das doenças de tranmissão sexual incluindo VIH-1 e 2 em 400 mulheres na Maternidade Lucrécia Paim durante o período de julho e agosto de 1992. Luanda: Universidade Agostinho Neto, Faculdade de Medicina, 1993.

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Book chapters on the topic "Pain epidemiology"

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Chinn, Steven Y., Elizabeth Chuang, and Karina Gritsenko. "Epidemiology." In Pain Medicine, 27–29. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-43133-8_8.

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Andreae, Michael. "Epidemiology." In Academic Pain Medicine, 47–50. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-18005-8_9.

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Kalia, Hemant, Omar Viswanath, and Alaa Abd-Elsayed. "Epidemiology." In Trigeminal Nerve Pain, 17–21. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-60687-9_3.

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Canavero, Sergio, and Vincenzo Bonicalzi. "Epidemiology." In Central Pain Syndrome, 7–53. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-56765-5_2.

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Ruutu, Mirja, Mikael Leppilahti, and Jukka Sairanen. "Epidemiology." In Bladder Pain Syndrome, 11–19. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4419-6929-3_2.

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King, Cara R., and Andrew R. King. "Anatomy and Epidemiology." In Pain, 827–29. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99124-5_176.

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Lipton, Richard B., and Marcelo E. Bigal. "Migraine Epidemiology." In Encyclopedia of Pain, 1855–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_2419.

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Reyes-Gibby, Cielito, and Karen O. Anderson. "Cancer Pain, Epidemiology." In Encyclopedia of Pain, 438–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_517.

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Graziottin, Alessandra, and Filippo Murina. "Epidemiology of Vulvar Pain." In Vulvar Pain, 9–18. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-42677-8_2.

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Nickel, J. Curtis. "Epidemiology Commentary." In Bladder Pain Syndrome – An Evolution, 23–25. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-61449-6_4.

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Conference papers on the topic "Pain epidemiology"

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Fargetti, Simone, Telma de Cassia dos Santos Nery, Salvador Celso Callia, Maria Thereza Toledo Penteado, Monize Mendonça Cruz, and Moacyr Vergara de Godoy Moreira. "O-465 Pain in healthcare workers: a perspective of multidisciplinary approach." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.89.

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Mosbah, Hibat Allah, Ines Rassas, Marouen Hayouni, Aouatef Mahfoudh, Amira Khelil, Adnene Hanchi, Mohamed Akrout, and Irtyah Merchaoui. "P-257 Work-Family Conflict as a Risk Factor of Back Pain among Nurses." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.254.

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Chaiklieng, Sunisa. "P-25 Assessment of lighting intensity at workstations and incidence of shoulder pain among electronic manufacturing workers." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.167.

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Kheder, Asma, Ben Afia Latifa, Meriem Heni, Amira Omrane, Olfa Jlassi, Nesrine Mars, Harrathi Chayma, Taoufik Khalfallah, and Lamia Bouzgarrou. "P-457 Work organization as a preventive factor of work-related low back pain in Tunisian craftsmen." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.337.

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Nicolau França Chivambo, Ivan Manuel, Fernando Feijó, Paulo Oliveira, and Daniela Monteiro. "O-474 Prevalence and risk factors for work-related neck pain in civil Servants: A Systematic review." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.93.

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Petersen, Jonathan, Charlotte Brauer, Lau Thygesen, Esben Meulengracht Flachs, Christina Bach Lund, and Jane Froelund Thomsen. "P-51 Repetitive and forceful movements of the hand as predictors for pain in the distal upper extremities." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.177.

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Feijó, Fernando, Ivan Manuel Nicolau França Chivambo, Lídia Sanduane, Eduarda Buriol, Anaclaudia Fassa, and Paulo Oliveira. "P-478 Association between workplace bullying and neck pain: a study with civil servants from a middle-income country." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.347.

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Salma, Kamoun, Olfa Jlassi, Lamia Bouzgarrou, Neila Chaari, Adel Amri, and Irtyah Merchaoui. "P-454 Low back pain in the care setting: Study among 300 healthcare staff from two university hospital in Tunisia." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.335.

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Feijó, Fernando, Neil Pearce, Neice Faria, Maitê Carvalho, Ana Laura Szortyka, Paulo Oliveira, and Anaclaudia Fassa. "RF-382 Association between workplace bullying and acute and chronic low back pain in civil servants from a middle-income country." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.380.

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Donato, Michael Di, Ting Xia, Ross Iles, Rachelle Buchbinder, and Alex Collie. "O-21 Patterns of opioid dispensing and associated wage replacement duration in workers with accepted claims for low back pain: a retrospective cohort study." In 28th International Symposium on Epidemiology in Occupational Health (EPICOH 2021). BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/oem-2021-epi.85.

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Reports on the topic "Pain epidemiology"

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Shpigel, Nahum Y., Ynte Schukken, and Ilan Rosenshine. Identification of genes involved in virulence of Escherichia coli mastitis by signature tagged mutagenesis. United States Department of Agriculture, January 2014. http://dx.doi.org/10.32747/2014.7699853.bard.

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Abstract:
Mastitis, an inflammatory response of the mammary tissue to invading pathogenic bacteria, is the largest health problem in the dairy industry and is responsible for multibillion dollar economic losses. E. coli are a leading cause of acute mastitis in dairy animals worldwide and certainly in Israel and North America. The species E. coli comprises a highly heterogeneous group of pathogens, some of which are commensal residents of the gut, infecting the mammary gland after contamination of the teat skin from the environment. As compared to other gut microflora, mammary pathogenic E. coli (MPEC) may have undergone evolutionary adaptations that improve their fitness for colonization of the unique and varied environmental niches found within the mammary gland. These niches include competing microbes already present or accompanying the new colonizer, soluble and cellular antimicrobials in milk, and the innate immune response elicited by mammary cells and recruited immune cells. However, to date, no specific virulence factors have been identified in E. coli isolates associated with mastitis. The original overall research objective of this application was to develop a genome-wide, transposon-tagged mutant collection of MPEC strain P4 and to use this technology to identify E. coli genes that are specifically involved in mammary virulence and pathogenicity. In the course of the project we decided to take an alternative genome-wide approach and to use whole genomes bioinformatics analysis. Using genome sequencing and analysis of six MPEC strains, our studies have shown that type VI secretion system (T6SS) gene clusters were present in all these strains. Furthermore, using unbiased screening of MPEC strains for reduced colonization, fitness and virulence in the murine mastitis model, we have identified in MPEC P4-NR a new pathogenicity island (PAI-1) encoding the core components of T6SS and its hallmark effectors Hcp, VgrG and Rhs. Next, we have shown that specific deletions of T6SS genes reduced colonization, fitness and virulence in lactating mouse mammary glands. Our long-term goal is to understand the molecular mechanisms of host-pathogen interactions in the mammary gland and to relate these mechanisms to disease processes and pathogenesis. We have been able to achieve our research objectives to identify E. coli genes that are specifically involved in mammary virulence and pathogenicity. The project elucidated a new basic concept in host pathogen interaction of MPEC, which for the best of our knowledge was never described or investigated before. This research will help us to shed new light on principles behind the infection strategy of MPEC. The new targets now enable prevalence and epidemiology studies of T6SS in field strains of MPEC which might unveil new geographic, management and ecological risk factors. These will contribute to development of new approaches to treat and prevent mastitis by MPEC and perhaps other mammary pathogens. The use of antibiotics in farm animals and specifically to treat mastitis is gradually precluded and thus new treatment and prevention strategies are needed. Effective mastitis vaccines are currently not available, structural components and effectors of T6SS might be new targets for the development of novel vaccines and therapeutics.
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