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Relevant bibliographies by topics / Pain diagnosis

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Academic literature on the topic 'Pain diagnosis'

Author: Grafiati

Published: 4 June 2021

Last updated: 19 February 2022

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Journal articles on the topic "Pain diagnosis"

1

Fučík, Tomáš, and Jaromír Mašata. "Pelvic neuropathic pain (differential diagnosis)." Česká gynekologie 86, no. 4 (August 30, 2021): 279–83. http://dx.doi.org/10.48095/cccg2021279.

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Summary: Objective: General practitioners, surgeons, neurologists, urologists and gynecologists all encounter patients suffering from neurogenic pelvic pain. Correct management demands knowledge from all above mentioned specialties. The primary goal is to help patients suffering from chronic or acute pelvic pain coupled with functional disorders like dysuria, urgency, dyspareunia, mobility disorders orhypoesthesia. Neurogenic defects are not the most common etiology for either of listed symptoms. However, after exclusion of the more common ones and failure to respond to basic therapeutic methods such as physiotherapy or analgotheraphy doctors tend to mark the illness as idiopathic and incurable. The goal of this review is to show the most common nosological units and a robust diagnostic algorithm to describe the type and level of the damage. Methods: Review of literature using databases Pubmed, Science direct, Medline and sources of the international school of neuropelveology. Conclusion: Over a lifetime, one in seven women will suffer from chronic pelvic pain. Outside of the cases where a clear postoperative etiology is established, the time to make a correct dia gnosis is often long for the unspecific and varied symptomatology. Neuropelveological diagnostic algorithm is demonstrably efficient in shortening the time to diagnosis and more importantly to the treatment.

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Feinberg, Joseph H. "Hip Pain: Differential Diagnosis." Journal of Back and Musculoskeletal Rehabilitation 4, no. 3 (July 1, 1994): 154–73. http://dx.doi.org/10.3233/bmr-1994-4306.

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Breivik, Harald. "Physical Diagnosis of Pain." Scandinavian Journal of Pain 17, no. 1 (October 1, 2017): 447. http://dx.doi.org/10.1016/j.sjpain.2017.08.016.

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Seager, Lewis H. "Diagnosis of chest pain." Postgraduate Medicine 97, no. 2 (February 1995): 131–45. http://dx.doi.org/10.1080/00325481.1995.11945963.

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Woolf, Clifford J., and Mitchell B. Max. "Mechanism-based Pain Diagnosis." Anesthesiology 95, no. 1 (July 1, 2001): 241–49. http://dx.doi.org/10.1097/00000542-200107000-00034.

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Kuo, Peter T. "Diagnosis of Chest Pain." JAMA: The Journal of the American Medical Association 261, no. 13 (April 7, 1989): 1983. http://dx.doi.org/10.1001/jama.1989.03420130153046.

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Curtin, Catherine. "Pain Examination and Diagnosis." Hand Clinics 32, no. 1 (February 2016): 21–26. http://dx.doi.org/10.1016/j.hcl.2015.08.006.

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Shaw, David B. "Diagnosis of chest pain." International Journal of Cardiology 28, no. 3 (September 1990): 387. http://dx.doi.org/10.1016/0167-5273(90)90328-3.

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Hogans, Beth B., Bernadette C. Siaton, Michelle N. Taylor, Leslie I. Katzel, and John D. Sorkin. "Low Back Pain and Substance Use: Diagnostic and Administrative Coding for Opioid Use and Dependence Increased in U.S. Older Adults with Low Back Pain." Pain Medicine 22, no. 4 (February 17, 2021): 836–47. http://dx.doi.org/10.1093/pm/pnaa428.

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Abstract Objective Low back pain (LBP) is a leading cause of pain and disability. Substance use complicates the management of LBP, and potential risks increase with aging. Despite implications for an aging, diverse U.S. population, substance use and LBP comorbidity remain poorly defined. The objective of this study was to characterize LBP and substance use diagnoses in older U.S. adults by age, gender, and race. Design Cross-sectional study of a random national sample. Subjects Older adults including 1,477,594 U.S. Medicare Part B beneficiaries. Methods Bayesian analysis of 37,634,210 claims, with 10,775,869 administrative and 92,903,649 diagnostic code assignments. Results LBP was diagnosed in 14.8±0.06% of those more than 65 years of age, more in females than in males (15.8±0.08% vs. 13.4±0.09%), and slightly less in those more than 85 years of age (13.3±0.2%). Substance use diagnosis varied by substance: nicotine, 9.6±0.02%; opioid, 2.8±0.01%; and alcohol, 1.3±0.01%. Substance use diagnosis declined with advancing age cohort. Opioid use diagnosis was markedly higher for those in whom LBP was diagnosed (10.5%) than for those not diagnosed with LBP (1.5%). Most older adults (54.9%) with an opioid diagnosis were diagnosed with LBP. Gender differences were modest. Relative rates of substance use diagnoses in LBP were modest for nicotine and alcohol. Conclusions Older adults with LBP have high relative rates of opioid diagnoses, irrespective of gender or age. Most older adults with opioid-related diagnoses have LBP, compared with a minority of those not opioid diagnosed. In caring for older adults with LBP or opioid-related diagnoses, health systems must anticipate complexity and support clinicians, patients, and caregivers in managing pain comorbidities. Older adults may benefit from proactive incorporation of non-opioid pain treatments. Further study is needed.

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Ketenci, Ayşegül. "Head and Neck Pain, Differential Diagnosis, Referred Pain." Türkiye Fiziksel Tıp ve Rehabilitasyon Dergisi 56, supp 1 (April 18, 2010): 34–37. http://dx.doi.org/10.4274/tftr.56.34.

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Dissertations / Theses on the topic "Pain diagnosis"

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Nakamura, Ryogo. "Diagnosis of Ulnar Wrist Pain." Nagoya University School of Medicine, 2001. http://hdl.handle.net/2237/5370.

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Ohnmeiss, Donna D. "Pain drawings in the evaluation of lumbar disc-related pain /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4069-X/.

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Miller, Amanda Jeannine. "Gender Disparities in Diagnosis and Pain Management." Master's thesis, Temple University Libraries, 2018. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/491860.

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Urban Bioethics
M.A.
The proliferation of social media and other online forums has allowed female patients to share their experiences in the healthcare system. Female patients and women’s health advocates can more easily speak out about instances of gender bias in medicine, which impact women’s access to equitable healthcare and positive healthcare experiences. Although there are some medical studies addressing gender disparities in various aspects of medicine, the impacts of gender bias on healthcare remain understudied and poorly understood. Patient narratives therefore provide an essential insight into the state of gender bias in medicine today. This paper aims to explore these narratives for common themes, to determine whether the current medical literature supports the presence of gender-based disparities, and to highlight the biological, psychological, and sociocultural factors impacting any disparities. Patient narratives frequently cite frustrations with diagnostic errors or delays and inadequate pain management, and the medical literature generally supports women’s accounts of gender disparities in these areas. Several studies of diagnostic disparities show that women more frequently experience delays in diagnosis, missed diagnoses, and incorrect psychiatric diagnoses. Multiple pain management studies have found that women face longer delays in care, lower rates of analgesic administration (particularly opiates), and fewer referrals for nonpharmacologic management strategies. Explanations for these disparities are likely multifactorial, and include provider ignorance of female-specific presentations and diseases, prevalence of understudied diseases in women, misattribution of symptoms to psychogenic causes, communication differences, normalization of female pain, and misconceptions about pain tolerance.
Temple University--Theses

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Buckley, David A. "Improving the diagnosis and treatment of chronic neuropathic pain." Thesis, University of Huddersfield, 2018. http://eprints.hud.ac.uk/id/eprint/34551/.

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Chronic neuropathic pain (CNP) occurs as a consequence of injury to the nervous system. Despite recent advances, CNP lacks objective diagnostic criteria, is often unrelenting and refractory to treatment. The primary aims of this thesis are twofold; the identification of CNP biomarkers using both human cohorts and an animal model (spinal nerve ligation; SNL) of neuropathic pain, and to provide clarity on the role of GTP cylcohydrolase I (GCH1) in CNP. Analysis of GCH1 and related genes and metabolites was conducted. As biomarkers, nitrite/nitrate and neopterin did not differentiate controls from CNP patients. However, significant differences were observed with biopterins, whilst correlations were observed between GCH1, nitrite/nitrate and neopterin, which were notably stronger in patients than controls. Analysis in human cohorts and in the SNL model also inferred that downregulation of GCHFR may contribute to BH4 synthesis. In order to provide clarity on the role of the GCH1 pain protective haplotype, reporter gene assays were used. This demonstrated a potential regulatory role for the GCH1 5’ SNP (rs8007267). In silico prediction of transcription factor binding sites suggested that this may be mediated by the aryl hydrocarbon nuclear translocator. The use of electrophoretic mobility shift assays showed strong specific binding with probe pertaining to the major allele. Further analysis is required to elucidate transcription factor binding, potentially facilitated by 2D-PAGE and mass spectrometry. In order to further elucidate potential CNP biomarkers, microarray analysis and qRT-PCR were performed using blood obtained from CNP patients. Data refinement led to the isolation of 27 potential CNP biomarkers, of which several cross-validated between cohorts. Microarray data, literature evidence, and correlations with previous microarrays provided evidence suggestive of a role for TIMP1. Multiple other genes, including CASP5, TLR4, TLR5, MC1R and CX3CR1, were differentially regulated in CNP. Genes surviving microarray data refinement were subsequently analysed in the dorsal horn of Sprague Dawley and Wistar Kyoto rats after SNL. Several genes, including Dpp3, Mc1r and Timp1, were similarly differentially expressed in the rodent SNL model, which suggests that these genes may be involved in the pathophysiological mechanisms of CNP, and may also function as potential translational biomarkers of CNP. This work provides multiple avenues for expansion and further investigation. Clearly, the challenges associated with biomarker discovery in CNP states are considerable, though it is hoped that this thesis provides valuable insight and the necessary foundation for future work.

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Tidwell, Irene Donna 1956. "NURSING DIAGNOSIS--ALTERATION IN COMFORT-PAIN: VALIDATION OF THE DEFINING CHARACTERISTICS." Thesis, The University of Arizona, 1986. http://hdl.handle.net/10150/291287.

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Torstensson, Thomas. "Chronic Pelvic Pain Persisting after Childbirth : Diagnosis and Implications for Treatment." Doctoral thesis, Uppsala universitet, Allmänmedicin och preventivmedicin, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-211847.

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Objectives: To explore the pain mechanism and the origin of the pain and to evaluate a short-term pain relief treatment in women suffering from CPP persisting after childbirth in order to enable physiotherapeutic intervention. Material and methods: Thirty-six parous women with chronic pelvic pain persisting after childbirth were recruited at the Department of Physiotherapy, SundsvallHospital and by advertisements in newspapers and 29 parous women without chronic pelvic pain were recruited from an organized gynaecological screening at a midwifery surgery. All women were provoked by intra-pelvic palpation of 13 predetermined intra-pelvic landmarks. The provoked pain distribution was expressed in pain drawings and the pain intensity verbally on a Likert scale.Also, in a randomised controlled trial the 36 women with chronic pelvic pain were allocated to bilateral injection treatment with either triamcinolone or saline solutions, given once on the ischial spine with follow-up after four weeks. Results: Referred pain provoked on intra-pelvic landmarks follows a specific pattern. In general, pain provoked by palpation of the posterior intra-pelvic landmarks was mostly referred to the sacral region and pain provoked by palpation of the ischial and pubic bones was mostly referred to the groin and pubic regions. In women with chronic pelvic pain the provoked pain distribution area and pain intensity were magnified as compared to women without chronic pelvic pain. In the clinical trial decreased pain intensity, decreased distribution of pain and improved physical function was achieved among the triamcinolone treatment group as compared to the saline treatment group. Also, a positive correlation was shown between reduced pain intensity and improved function. Conclusions: Referred pain patterns provoked on intra-pelvic landmarks in women with chronic pelvic pain persisting after childbirth are consistent with sclerotomal sensory innervations and indicates allodynia and central sensitisation. This suggests that pain mapping can be used to evaluate and confirm the pain experience and contribute to diagnosis. Also, the pain intensity provoked by stimulation of the intra-pelvic landmarks is suggested to be useful to differentiate women with chronic pelvic pain from those without. Corticosteroid treatment to the ischial spine resulted in decreased pain and increased function.

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Tse, Yuk-hang Jessica, and 謝毓衡. "Application of surface electromyography topography in low back pain rehabilitation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208612.

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The management of low back pain (LBP) has long been a challenge as it is a highly prevalent disease worldwide due to the lack of an apparent etiology and a conclusive therapeutic treatment. Heterogeneous groups of LBP patients with diverse medical backgrounds also complicate the problem. A clinical threshold is demanded to match the patients and treatments appropriately in order to maximize the treatment success rate. Besides, the assessments of disability and pain perception due to LBP made by self-evaluated questionnaires in current clinical setting are highly susceptible to subjective feeling and the memory of patients. LBP and spasm are closely related but little is known for the underlying physiology, especially the musculature of LBP patients with spasm. These problems exacerbate the difficulty in LBP rehabilitation further. Surface electromyography (sEMG) topography is a cutting- edge technology to assess the lumbar muscle in vivo non-invasively by illustrating the distribution of global muscle activity visually. sEMG topography has the potential application as an objective assessment tool for LBP rehabilitation. In present study, sEMG topography was used to address 1) the prognostic value of sEMG topography on LBP rehabilitation, 2) the establishment and validation of a clinical classification threshold for identification of LBP patients who are responsive to exercise therapy and 3) the investigation of sEMG topography in accordance with the physiological outcomes (functional disability, pain perception and spasm). Forty-five healthy subjects and fifty patients with chronic non-specific LBP were enrolled to the study. sEMG test was conducted to every subject under the motions of lumbar flexion and extension in order to gather the myoelectric signals by a 16-channel sEMG. Various sEMG topographic parameters (sEMG parameters) were developed for quantitative analysis of sEMG topography. They were Root-Mean-Square-Difference of Relative Area (RMSD RA), Relative Width (RMSD RW) and Relative Height (RMSD RH) at flexion and extension. Results showed that sEMG parameters were of significant prognostic value for LBP patients towards exercise therapy. A clinical threshold of 0.21 was proposed and validated based on the geometric calculation of RMSD RA and RMSD RW at flexion and extension. The threshold was substantiated to increase the success rate of exercise therapy from 46% to 86% when the value measured by sEMG topography was below 0.21. sEMG parameters were found significantly associated with disability and pain perception in a positive manner. Severer disability and pain perception were represented by larger values of sEMG parameters. sEMG topography demonstrated symmetric patterns for patients with or without spasms on bilateral sides of lumbar muscles. The symmetry in sEMG topography evinced the consistency of the musculature of bilateral lumbar muscles while the lost of symmetry might indicate malfunction of lumbar muscles unilaterally. To conclude, this study corroborated versatile roles of sEMG topography in LBP rehabilitation as a prognosis, clinical threshold, and objective measurement. The findings of this study have paved the way of sEMG topography for future application in clinical setting. A study of larger scale would be recommended to complement the present findings.
published_or_final_version
Orthopaedics and Traumatology
Master
Master of Philosophy

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Vickers, Edward Russell. "Neuropathic orofacial pain: a review and guidelines for diagnosis and management." University of Sydney. Anaesthesia and Pain Management, 2001. http://hdl.handle.net/2123/806.

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Neuropathic pain is defined as "pain initiated or caused by a primary lesion or dysfunction in the nervous system". In contrast to physiological pain that warns of noxious stimuli likely to result in tissue damage, neuropathic pain serves no protective function. Examples of neuropathic pain states include postherpetic neuralgia (shingles) and phantom limb / stump pain. This pain state also exists in the orofacial region, with the possibility of several variants including atypical odontalgia and burning mouth syndrome. There is a paucity of information on the prevalence of neuropathic pain in the orofacial region. One study assessed patients following endodontic treatment and found that approximately 3 to 6percent of patients reported persistent pain. Patients predisposed to the condition atypical odontalgia (phantom tooth pain) include those suffering from recurrent cluster or migraine headaches. Biochemical and neurobiological processes leading to a neuropathic pain state are complex and involve peripheral sensitisation, and neuronal plasticity of the central and peripheral nervous systems. Subsequent associated pathophysiology includes regional muscle spasm, sympathetic hyperfunction, and centralisation of pain. The relevant clinical features of neuropathic pain are: (i) precipitating factors such as trauma or disease (infection), (ii) pain that is frequently described as having burning, paroxysmal, and lancinating or sharp qualities, and (iii) physical examination may indicate hyperalgesia, allodynia and sympathetic hyperfunction. The typical patient complains of persistent, severe pain, yet there are no clearly identifiable clinical or radiographic abnormalities. Often, due to the chronicity of the problem, afflicted patients exhibit significant distress and are poor pain historians, thus complicating the clinician's task of obtaining a detailed and relevant clinical and psychosocial history. An appropriate analgetic blockade test for intraoral sites of neuropathic pain is mucosal application of topical anaesthetics. Other, more specific, tests include placebo controlled lignocaine infusions for assessing neuropathic pain, and placebo controlled phentolamine infusions for sympathetically maintained pain. The treatment and management of neuropathic pain is multidisciplinary. Medication rationalisation utilises first-line antineuropathic drugs including tricyclic antidepressants, and possibly an anticonvulsant. Topical applications of capsaicin to the gingivae and oral mucosa are a simple and effective treatment. Neuropathic pain responds poorly to opioid medication. Psychological assessment is often crucial in developing strategies for pain management. Psychological variables include distress, depression, expectations of treatment, motivation to improve, and background environmental factors. To enable a greater understanding of neuropathic pain, thereby leading to improved treatments, high-performance liquid chromatography-mass spectrometry is one analytical technique that has the potential to contribute to our knowledge base. This technique allows drugs and endogenous substances to be assayed from one sample in a relatively short time. The technique can identify, confirm, and measure the concentrations of multiple analytes from a single sample.

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Vickers, E. R. "Neuropathic orofacial pain a review and guidelines for diagnosis and management /." Connect to full text, 2001. http://hdl.handle.net/2123/806.

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Thesis (M. Sc. Med.)--University of Sydney, 2001.
Title from title screen (viewed Apr. 23, 2008). Submitted in fulfilment of the requirements for the degree of Master of Science in Medicine to the Dept. of Anaesthesia and Pain Management, Faculty of Medicine. Includes bibliography. Also available in print form.

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Stynes, Siobhán Margaret. "The diagnosis and classification of low back-related leg pain." Thesis, Keele University, 2017. http://eprints.keele.ac.uk/3344/.

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Low back-related leg pain (LBLP) is clinically diagnosed as referred leg pain or sciatica. The clinical task of differentiating sciatica from referred leg pain can be challenging but is important for the purpose of treatment choices. There is currently no agreement on which clinical criteria best identify sciatica in clinical or research settings and the spectrum of clinical presentation in patients with LBLP is variable. This thesis aimed to identify diagnostic criteria for sciatica and explore and describe clusters of LBLP patients using cross-sectional data from 609 primary care LBLP consulters. A systematic literature search of LBLP classification systems showed very few systems specifically addressed LBLP classification. Within the systems, there was wide variation in definitions and clinical features of sciatica, with most systems based on clinical opinion. Reliability was merely fair (kappa = 0.35) amongst clinicians diagnosing sciatica but at higher levels of confidence in diagnosis (≥80%), reliability improved (kappa =0.68). Using high confidence clinical diagnosis as a reference standard, with and without confirmatory MRI findings, diagnostic models for sciatica were developed and compared. A simple scoring tool based on the best performing model was devised showing the probability of having sciatica based on results from five clinical items (subjective sensory changes, below knee pain, leg pain worse than back pain, positive neural tension, neurological deficit). Latent class analysis identified five classes of LBLP patients. One class was clearly a referred leg pain group, the other four classes seemed to represent sciatica with varying clinical profiles. This thesis provides a diagnostic tool for sciatica with potential application in clinical and research settings. It also reveals clusters of LBLP patients which could represent more homogenous groups amenable to different treatment approaches. This thesis has provided a strong basis for future work to further explore the clinical utility of the findings.

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Books on the topic "Pain diagnosis"

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Physical diagnosis of pain. Philadelphia, PA: W.B. Saunders, 2006.

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T, Upton Mark, ed. Diagnosis of chest pain. New York: Raven Press, 1988.

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Waldman, Steven D. Imaging of pain. Philadelphia, PA: Saunders/Elsevier, 2010.

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Mense, Siegfried, and Robert Gerwin. Muscle pain: Diagnosis and treatment. Heidelberg: Springer, 2010.

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Diagnosis of acute abdominal pain. 2nd ed. Edinburgh: Churchill Livingstone, 1991.

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Mense, Siegfried, and Robert D. Gerwin, eds. Muscle Pain: Diagnosis and Treatment. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-05468-6.

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Fitzgibbon, Dermot R. Cancer pain: Assessment, diagnosis, and management. Philadelphia, PA: Lippincott Williams & Wilkins, 2009.

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Fitzgibbon, Dermot R. Cancer pain: Assessment, diagnosis, and management. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2010.

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Fitzgibbon, Dermot R. Cancer pain: Assessment, diagnosis, and management. Philadelphia, PA: Lippincott Williams & Wilkins, 2009.

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Loeser, John D. (John David), 1935-, ed. Cancer pain: Assessment, diagnosis, and management. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2010.

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Book chapters on the topic "Pain diagnosis"

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Rosenberg, Paul A. "Diagnosis." In Endodontic Pain, 1–29. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54701-0_1.

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Canavero, Sergio, and Vincenzo Bonicalzi. "Diagnosis." In Central Pain Syndrome, 201–7. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-56765-5_7.

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Glass, Allison, Sanoj Punnen, and Katsuto Shinohara. "Pain Prevention." In Prostate Cancer Diagnosis, 133–45. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-188-2_12.

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Patten, John Philip. "Facial Pain." In Neurological Differential Diagnosis, 372–79. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-58981-2_21.

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Sandler, Gerald, and John Fry. "Chest Pain." In Early Clinical Diagnosis, 1–24. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4147-2_1.

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Govind, Jayantilal. "Neuralgia, Diagnosis." In Encyclopedia of Pain, 2041–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_2622.

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Auci, Alessio. "Radiologic Diagnosis." In Groin Pain Syndrome, 25–42. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-41624-3_4.

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Govind, Jayantilal. "Radicular Pain, Diagnosis." In Encyclopedia of Pain, 3344–47. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_3710.

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Vivian, David. "Central Pain, Diagnosis." In Encyclopedia of Pain, 537–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_615.

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Derbyshire, Stuart W. G. "Fetal Pain." In Prenatal and Preimplantation Diagnosis, 119–30. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-18911-6_6.

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Conference papers on the topic "Pain diagnosis"

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FIGUEIREDO, RAISSA VELASQUES DE, MARIA LÚCIA LEMOS LOPES, TATIANA FREITAS TOURINHO, MARIA ODETE ESTEVES HILÁRIO, GILBERTO SCANAGATTA, RAFAEL CORADIN, LUANA RIBEIRO CARLOS, THIAGO WILLERS, RAQUEL DOS SANTOS RAMOS, and TATIANE ANDRESSA GASPARETTO. "ERYTHROMELALGIA: A DIFFERENTIAL DIAGNOSIS OF CHRONIC PAIN." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-100.

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Lee, Y. C., O. Schieir, M. F. Valois, S. J. Bartlett, G. Boire, B. Haraoui, C. Hitchon, et al. "OP0349 Remaining pain and widespread pain distribution during the first 12 months after ra diagnosis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.3343.

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Das, Sumit, Manas Kumar Sanyal, and Debamoy Datta. "Intelligent Approaches for the Diagnosis of Low Back Pain." In 2019 Amity International Conference on Artificial Intelligence (AICAI). IEEE, 2019. http://dx.doi.org/10.1109/aicai.2019.8701266.

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"TOWARDS AN AUTOMATIC DIAGNOSIS SYSTEM FOR ACUTE ABDOMINAL PAIN - Support Vector Machines for the Diagnosis of Diverticulitis and Non-specific Abdominal Pain." In International Conference on Health Informatics. SciTePress - Science and and Technology Publications, 2009. http://dx.doi.org/10.5220/0001546200510057.

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Ashraf, Ali, and John Unterborn. "Shoulder Pain Plus An Incidental Finding Equals A Rare Diagnosis." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5669.

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Dullen, D., L. Zieske, and M. Mohanty. "Observational Study Monitoring Pain in 25 Dogs Comparing Novel Non-Invasive Device and Veterinary Diagnosis to Differentiate Location and Magnitude of Pain." In Pain in Animals Workshop 2017: Abstracts. Schattauer GmbH, 2018. http://dx.doi.org/10.1055/s-0038-1660879.

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Bounds, Lloyd, Mathew, and Waddell. "A multilayer perceptron network for the diagnosis of low back pain." In Proceedings of 1993 IEEE International Conference on Neural Networks (ICNN '93). IEEE, 1988. http://dx.doi.org/10.1109/icnn.1988.23963.

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Belova, Eva, and Andre Sotelo. "Difficult Diagnosis Of Abdominal Pain In The Patient With Infective Endocarditis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4587.

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Li, Ting, and Nanxi Li. "Noninvasive optical diagnosis of low back pain with the aid of Chinese cupping procedure." In Optical Diagnostics and Sensing XVIII: Toward Point-of-Care Diagnostics, edited by Gerard L. Coté. SPIE, 2018. http://dx.doi.org/10.1117/12.2287070.

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Brandstätter, J., U. Raab, S. Shah Hauschild, and G. Baier. "Differential diagnosis of newly occurring pain in a curatively treated tumor patient." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685968.

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Reports on the topic "Pain diagnosis"

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O'Kelly, John, and Frank O'Brien. Aetiology and diagnosis of bacterial chronic prostatitis (Type II) and chronic pelvic pain syndrome (CPPS) Type III. BJUI Knowledge, January 2020. http://dx.doi.org/10.18591/bjuik.0059.

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Huang, Jin-Feng. What is the best management for low back pain? Evidence mapping of recommendations on diagnosis and management for low back pain: an international review of 15 guidelines. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0104.

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Fisherkeller, Karen, Cindy Burgess-Russotti, and Dale Hamilton. Documentation for the Computer Assisted Diagnostic Program for Dental Pain. Fort Belvoir, VA: Defense Technical Information Center, April 1989. http://dx.doi.org/10.21236/ada573903.

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Harrington, Cherise B. Patterns of diagnostic care in nonspecific low back pain: Relation to patient satisfaction and perceived health. Fort Belvoir, VA: Defense Technical Information Center, November 2006. http://dx.doi.org/10.21236/ad1013990.

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