Dissertations / Theses on the topic 'Paediatric traumatic brain injury'
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Trenchard, Sian Olivia. "Traumatic brain injury in a paediatric population." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/traumatic-brain-injury-in-a-paediatric-population(cf299afa-75ff-4a58-9684-6b332d25715e).html.
Full textRohlwink, Ursula Karin. "Paediatric traumatic Brain Injury: The relationship between Intracranial Pressure and Brain Oxygenation." Master's thesis, University of Cape Town, 2009. http://hdl.handle.net/11427/2889.
Full textCoullie, Charis Blythe. "Adult Psychiatric and Offending Outcomes of Paediatric Mild Traumatic Brain Injury." Thesis, University of Canterbury. Psychology, 2013. http://hdl.handle.net/10092/7802.
Full textWard, Heather Jean, and n/a. "Prospective Memory: Early Developmental Trajectory and Effects of Paediatric Traumatic Brain Injury on its Functioning." Griffith University. School of Psychology, 2005. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20050804.154501.
Full textKilchenmann, Nadine M. "Theory of mind following paediatric traumatic brain injury : a comparative study of South African children." Master's thesis, University of Cape Town, 2011. http://hdl.handle.net/11427/12656.
Full text“Theory of mind” (ToM) refers to the ability to understand and make inferences about other people’s intentions, feelings and beliefs. The fact that previous research shows an impairment in social competence following traumatic brain injury (TBI) alludes to a potential relationship between TBI and ToM. Although the relationship between paediatric TBI (pTBI) and ToM ability is relatively unexplored, especially within a South African context, previous research on TBI and ToM suggests social impairment following TBI. The current research project was aimed to investigate exactly that. The study reported here investigated this relationship, and specifically focused on the effects of pTBI on ToM ability in 9-15 year old children.
Mogere, Edwin. "Impact of secondary insults on the outcome of paediatric traumatic brain injury : a retrospective cross sectional study at the Red Cross Children’s Hospital, Cape Town." Master's thesis, University of Cape Town, 2013. http://hdl.handle.net/11427/13970.
Full textSecondary insults in severe traumatic brain injury (TBI) may worsen outcome; however, these are poorly characterized in children. For example, despite the known association between intracranial pressure (ICP) and poor outcome, there are few large paediatric series on the subject, definitions vary, functional outcome is often not assessed, and the best measures to assess ICP for statistical analysis are unknown. We aimed to document the frequency of secondary insults, and the association of various ICP measures, with outcome in a large cohort of paediatric patients with severe TBI. A retrospective analysis of 5-year prospectively collected data was examined for the frequency of hypoxia, hypotension, raised ICP, and low cerebral perfusion pressure (CPP). ICP parameters included initial ICP, mean ICP in the first 24 hours, mean ICP overall, peak ICP, mean ICP over 20 mmHg, and episodes of ICP over 20 mmHg. Hypotension was defined by age †and height †adjusted mean arterial pressure ranges, and hypoxia was defined as arterial partial pressure of oxygen (PaO2) less than 8kPa or pulse oximetry less than 90%. We examined for univariate and multivariate associations with mortality and the Extended Paediatric Glasgow Outcome Score.
Bellesi, Giulia. "The association between paediatric traumatic brain injury and antisocial behaviour in adulthood : a longitudinal study using the ALSPAC data." Thesis, King's College London (University of London), 2018. https://kclpure.kcl.ac.uk/portal/en/theses/the-association-between-paediatric-traumatic-brain-injury-and-antisocial-behaviour-in-adulthood-a-longitudinal-study-using-the-alspac-data(f3e3b936-e780-4331-adca-1af9df57e5d4).html.
Full textFigaji, Anthony A. "Multimodality monitoring in paediatric severe traumatic brain injury : the contributions of brain oxygen, transcranial doppler and autoregulation monitoring to conventional methods on monitoring." Doctoral thesis, University of Cape Town, 2008. http://hdl.handle.net/11427/2882.
Full textIncludes bibliographical references (p. 175-218).
Traumatic brain injury (TBI) is a highly complex clinical condition in the most complex organ of the body. The foundation of care of the patient with severe TBI is the prevention of secondary insults to the brain. This relies on conventional monitoring tools to identify patients at risk, but often these may fail to detect important secondary insults. Moreover, the therapies that are used commonly in the critical care environment all have potential adverse effects, many of which may not be evident. TBI treatment in children is further complicated by changing thresholds with age, and the much smaller evidence base compared to their adult counterparts.
Force, Lisa Marie. "Traumatic brain injury and acidosis /." view abstract or download text of file, 2006. http://hdl.handle.net/1794/3913.
Full textSingh, Rajiv K. "Depression after traumatic brain injury." Thesis, University of Sheffield, 2017. http://etheses.whiterose.ac.uk/18730/.
Full textPerel, Pablo Andraes. "Prognosis in traumatic brain injury." Thesis, London School of Hygiene and Tropical Medicine (University of London), 2009. http://researchonline.lshtm.ac.uk/1635515/.
Full textRowe, Rachel K. "POST-TRAUMATIC SLEEP FOLLOWING DIFFUSE TRAUMATIC BRAIN INJURY." UKnowledge, 2013. http://uknowledge.uky.edu/neurobio_etds/7.
Full textKeller, Kristen Jo. "Challenges to Secondary Brain Injury Prevention in Severe Traumatic Brain Injury." Diss., The University of Arizona, 2014. http://hdl.handle.net/10150/338712.
Full textBainbridge, Stephen. "Parental coping with paediatric encephalitic brain injury." Thesis, University of Leicester, 2005. http://hdl.handle.net/2381/31205.
Full textHocking, Sian Elin. "Parental adjustment in paediatric acquired brain injury." Thesis, University of Exeter, 2015. http://hdl.handle.net/10871/18329.
Full textTyerman, Emma. "Family experience after paediatric acquired brain injury." Thesis, Lancaster University, 2015. http://eprints.lancs.ac.uk/76531/.
Full textAl-Hasani, Omer Hussain. "Traumatic brain injury with particular reference to diffuse traumatic axonal injury subpopulations." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5569.
Full textMalhotra, Rajiv. "GENE EXPRESSION FOLLOWING TRAUMATIC BRAIN INJURY." VCU Scholars Compass, 1998. http://scholarscompass.vcu.edu/etd/5082.
Full textDavies, Suzanne. "Personality change following traumatic brain injury." Thesis, University of Birmingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397519.
Full textMacqueen, Ruth. "Masculine identity after traumatic brain injury." Thesis, University of East Anglia, 2016. https://ueaeprints.uea.ac.uk/60949/.
Full textHelmy, Adel Ezzat. "Neuro-inflammation in traumatic brain injury." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610114.
Full textNagulavancha, Sruthi. "Traumatic brain injury options web application." Kansas State University, 2010. http://hdl.handle.net/2097/4626.
Full textDepartment of Computing and Information Sciences
Daniel A. Andresen
According to the Division of Injury Response, Centers for Disease Control and Prevention, approximately 1.4 million Americans sustain a traumatic brain injury each year. The aim of the project is to create a web interface to link survivors, family members, and caregivers of individuals suffering from traumatic brain injuries (TBI) to potentially helpful agencies or service centers within their local communities. Often the TBI service centers located in the remote places are difficult to trace hence this website mainly concentrates on small rural centers which are located in Kansas State. The portal will offer two-dimensional and basic information about traumatic brain injury centers and specifically about access of resources. Within the portal, a link to an interactive map will be provided. A form for data entry helps the service centers to publish about their presence and the regions they serve. A search distance feature is also added into the website which interactively searches the nearest latitude, longitude values (TBI service center) to the user’s location by using the haversine formula.
LeMay, Carrie C., and Jill D. Stinson. "Sex Offenders With Traumatic Brain Injury." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/7906.
Full textOsterstock, Guillaume. "Hypothalamic defaults after traumatic brain injury." Thesis, Montpellier 1, 2012. http://www.theses.fr/2012MON1T017/document.
Full textThe works of this thesis were interested in the control of the hypothalamic GHRH neurons in physiological and pathological conditions. The goal was to clarify the molecular and cellular mechanisms involved in the control or impairments of GHR neuronal network functions. These neurons are the main stimulators of the GH release. We first showed that the hypothalamic growth axis could be regulated independently from the feeding network. Indeed, GHRH neurons are directly stimulated by ghrelin, which is the only hormone produced by the gastrointestinal tract known to stimulate the GH release through acting mainly on GHRH neurons. These effects are independent from its orexigenic effects exerted on the neighbourings NPY neurons. In addition, ghrelin and GHS (synthetic ghrelin receptor agonists) don’t change neither the firing rate of GHRH neurons, nor synchronize them. These effects are not gender-dependant; by contrast, Somatostatin, the major GH axis inhibitor, generates a sexual dimorphic and rhythmic inhibition of the GHRH neurons electrical activity mediated by its SST1 and SST2 receptors subtypes. These effects are so time-dependant direct and indirect effects and can probably be involved in the generation of the ultradian rhythm of the GH release. After a traumatic brain injury, we found an early and sustained deficiency of the GH release, like those observed in human. No pathological changes are visible in the pituitary gland. Inflammation occurs at the arcuate nucleus, and mainly at the median eminence levels; it involves a strong astrocyte reaction, tanycytes, and microglial and (or) infiltrated immune cells activations. These changes elicit morpho-functional impairments of the median eminence, permeability and leakage of the tanycyte barrier between the blood, CSF and Arc; at the opposite, nothing occur at the periventricular level, where are located SST neurons. Neither the number of GHRH neurons, neither their passive electrophysiological properties changed. Impairments of the activities of the GHRH nerve terminals, maybe associated to impairments of their regulated activity, must explain a GH deficiency
Moazzez, Lesko Mehdi. "Prognosis in traumatic brain injury (TBI)." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/prognosis-in-traumatic-brain-injury-tbi(8b69e340-7ecd-4890-9746-863089bf55f5).html.
Full textArietta, Luca. "Clinical Data Mining: Traumatic Brain Injury." Master's thesis, Alma Mater Studiorum - Università di Bologna, 2012. http://amslaurea.unibo.it/3897/.
Full textAkin, Faith W. "Vestibular Evaluation of Traumatic Brain Injury." Digital Commons @ East Tennessee State University, 2009. https://dc.etsu.edu/etsu-works/2448.
Full textYin, Terry. "Neuroprotective strategies for traumatic brain injury." Diss., University of Iowa, 2015. https://ir.uiowa.edu/etd/1811.
Full textSavicki, Laura Elizabeth. "Collaborative referencing in traumatic brain injury." Thesis, University of Iowa, 2012. https://ir.uiowa.edu/etd/2977.
Full textAkin, Faith W., and Owen D. Murnane. "Vestibular Consequences of Mild Traumatic Brain Injury (Blast Injury)." Digital Commons @ East Tennessee State University, 2008. https://dc.etsu.edu/etsu-works/1940.
Full textLalani, Sanam Jivani. "Effects of Traumatic Brain Injury on Pediatric Brain Volume." BYU ScholarsArchive, 2018. https://scholarsarchive.byu.edu/etd/6924.
Full textHånell, Anders. "Plasticity and Inflammation following Traumatic Brain Injury." Doctoral thesis, Uppsala universitet, Neurokirurgi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-146551.
Full textVan, der Merwe Jó-Marié. "Family needs following adult traumatic brain injury." Thesis, University of Port Elizabeth, 2004. http://hdl.handle.net/10948/335.
Full textCrawford, Maria Anne, and n/a. "Speed of retrieval after traumatic brain injury." University of Otago. Department of Psychology, 2005. http://adt.otago.ac.nz./public/adt-NZDU20060830.115029.
Full textKruijk, Jelle de. "Mild traumatic brain injury intervention and prognosis /." [Maastricht] : Maastricht : UPM, Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 2001. http://arno.unimaas.nl/show.cgi?fid=7622.
Full textIsraelsson, Charlotte. "Molecular Characterization of Experimental Traumatic Brain Injury." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7087.
Full textJohnson, Victoria E. "Progressive neuopathological changes following traumatic brain injury." Thesis, University of Glasgow, 2012. http://theses.gla.ac.uk/3162/.
Full textCraddock, Holly L. "Nursing interventions utilized in traumatic brain injury." Honors in the Major Thesis, University of Central Florida, 1998. http://digital.library.ucf.edu/cdm/ref/collection/ETH/id/27.
Full textBachelors
Health and Public Affairs
Nursing
Bradshaw, Douglas Robert Saunders. "Linear wave propagation in traumatic brain injury." Thesis, University of Southampton, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341646.
Full textWilliams, Claire. "Emotion deficit disorders following traumatic brain injury." Thesis, Swansea University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678429.
Full textHerbert, Camilla. "Grief and loss following traumatic brain injury." Thesis, University of Sheffield, 1998. http://etheses.whiterose.ac.uk/10277/.
Full textKhadr, Sophie N. "Neuroendocrine consequences of childhood traumatic brain injury." Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/29194.
Full textDonnelly, Joseph. "Intracranial monitoring after severe traumatic brain injury." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/271422.
Full textWalk, Alexandra Elizabeth. "Traumatic Brain Injury: Teacher Knowledge and Skills." University of Dayton / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=dayton1312572728.
Full textKinkela, Jessica H. "Diagnosis Threat in Mild Traumatic Brain Injury." Ohio University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1223597555.
Full textSzabó, Borbála L. "Families of adolescents with traumatic brain injury /." The Ohio State University, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488196234911166.
Full textWagner, Michael R. "The Nitroxidative Response to Traumatic Brain Injury." Ohio University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1584179902783766.
Full textLaRoux, Charlene I. 1979. "Executive function deficits in traumatic brain injury." Thesis, University of Oregon, 2010. http://hdl.handle.net/1794/11063.
Full textThe short and long term pathophysiology of traumatic brain injury (TBI) has not been fully elucidated. Individuals recently suffering a mild TBI (mTBI) or having a history of TBI frequently suffer deficits in their ability to maintain and allocate attention within and between tasks. This dissertation examines the influence of mild and chronic TBI on performance of task switching. We employed spatial and numerical task switching paradigms to assess the behavioral deficits in mTBI, and we used an internally generated switching and an externally cued switching task along with functional Magnetic Resonance Imaging (fMRI) to assess the long term deficits in executive function resulting from chronic TBI. In the first experiment, individuals with mTBI were identified and tested within the first 48 hours of injury and then at a set interval 5, 14, and 28 days post injury. In the second investigation, individuals with chronic TBI were tested at least 12 months after their most recent injury. Healthy gender, age, and education matched controls were also tested in both studies. This research demonstrated that mTBI subjects display deficits in switching behavior within 48 hours of injury that failed to resolve a month post-injury; however, these costs did not generalize across the switching task types. Chronic TBI subjects performed internally generated and externally cued switching paradigms with a degree of success equivalent to that of healthy controls but displayed larger amounts of activation and recruited more areas of the brain at lower levels of difficulty and did not increase recruitment in a stepwise fashion at higher levels of difficulty. Mild TBI causes significant deficits in task switching, but there is specificity in these deficits. Chronic TBI patients performed at a level equivalent to that of controls but displayed different patterns and degree of activation. Taken together, these findings indicate that there may be a specific time frame during which task switching shows behavioral deficits, after which the subject may compensate for these deficits to produce normalized performance.
Committee in Charge: Dr. Paul van Donkelaar, Chair; Dr. Li-Shan Chou; Dr. Ulrich Mayr; Dr. Marjorie Woollacott
Akin, Faith W. "Symposium: Vestibular Related Traumatic Brain Injury (TBI)." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/2428.
Full textAkin, Faith W., and Owen D. Murnane. "Vestibular Consequences of Mild Traumatic Brain Injury." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/1928.
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