Relevant bibliographies by topics / P300

Academic literature on the topic 'P300'

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Published: 4 June 2021
Last updated: 11 March 2023

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Journal articles on the topic "P300"

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McPherson, David L., and Mimi T. Salamat. "Interactions among Variables in the P300 Response to a Continuous Performance Task in Normal and ADHD Adults." Journal of the American Academy of Audiology 15, no. 10 (November 2004): 666–77. http://dx.doi.org/10.3766/jaaa.15.10.2.

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This study investigated the effect of variable interstimulus intervals (ISIs) in a group of normal and ADHD (attention deficit hyperactivity disorder) adults on behavioral reaction time and the auditory P300 event-related potential. This study involved 20 adult subjects with no history of ADHD and 11 adult subjects diagnosed with ADHD. The subjects were instructed to respond to the common stimuli and ignore the rare stimulus. Significant differences in the latency of the P300a, P300b, the amplitude of the P300b, and in the number of false alarms and correct rejections between ISIs were observed in the normal group. The group with ADHD failed to show any significant differences between ISIs. Psychophysical measures of hits showed significant differences for the number of hits for ISI 2 (2 sec) between the two groups. False alarms and correct rejections for all ISIs showed significant differences between groups. Significant group differences were seen for latency of the P300a and P300b at each of the three ISIs, for amplitude of the P300a and P300b for ISI 1 and ISI 3, and for the amplitude of the P300b for ISI 2. There was a greater separation in the group with ADHD between the P300a and P300b suggesting a processing lag in that group.
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Mathis, Stéphane, Jean-Philippe Neau, Claudette Pluchon, Marie-Noëlle Fargeau, Stéphane Karolewicz, Anna Iljicsov, and Roger Gil. "Apathy in Parkinson’s Disease: An Electrophysiological Study." Neurology Research International 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/290513.

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In Parkinson’s disease (PD), apathy (or loss of motivation) is frequent. Nevertheless, the contribution of attentional disorders to its genesis is still not clearly known. We want to determine the relation existing between apathy and attentional disorders by using P300a (or novelty P3) as a marker of the attentional process. The study included 25 patients (13 women and 12 men) with PD for whom we have determined the relationship between automatic attention (represented by P300a) and motor status, apathy, executive dysfunction, mental flexibility, inhibitory control, and depression/anxiety. We have found a correlation between the apathy score and amplitude of novelty P300 during the ON period and also a correlation of the apathy score with a decrease in amplitude of P300 during the OFF period. In a linear regression model, changes in the P300a predicted the severity of apathy independently of any other variable. We concluded firstly that the reduction in amplitude of the P300a wave was a neurophysiological marker of apathy in PD and secondly that apathy led to both dopaminergic denervation (mesolimbic) and nondopaminergic (dorsolateral prefrontal-subcortical) dysfunction.
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Weinberg, Anna, and Greg Hajcak. "The Late Positive Potential Predicts Subsequent Interference with Target Processing." Journal of Cognitive Neuroscience 23, no. 10 (October 2011): 2994–3007. http://dx.doi.org/10.1162/jocn.2011.21630.

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The current study investigated the association between neural engagement with task-irrelevant images and subsequent interference with target processing using the Emotional Interrupt paradigm [Mitchell, D., Richell, R., Leonard, A., & Blair, R. Emotion at the expense of cognition: Psychopathic individuals outperform controls on an operant response task. Journal of Abnormal Psychology, 115, 559, 2006]. Consistent with previous studies, PCA-derived factors corresponding to the early posterior negativity, P300, and late positive potential (LPP) were enhanced for emotional (i.e., both unpleasant and pleasant) compared with neutral distracters, and the P300 elicited by targets was smaller following emotional compared with neutral pictures. In addition, RTs were increased to targets that followed emotional pictures. Within-subject analyses demonstrated that slow trials were characterized by a smaller P300 and were preceded by pictures with a larger LPP. Additionally, between-subject analyses indicate that individuals with a larger LPP also demonstrated slower RTs to targets and reduced target-elicited P300s. All results were specific to the LPP and were not observed for either the early posterior negativity or the P300 elicited by task-irrelevant pictures. By relating the LPP to subsequent behavioral and ERP interference in both within- and between-subject analyses, the current study provides direct support for the notion that LPP indexes attentional engagement with visual stimuli that is uniquely associated with subsequent interference in terms of both RT slowing and P300 reduction to targets.
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Schienle, Anne, Axel Schäfer, and Ewald Naumann. "Event-Related Brain Potentials of Spider Phobics to Disorder- Relevant, Generally Disgust- and Fear-Inducing Pictures." Journal of Psychophysiology 22, no. 1 (January 2008): 5–13. http://dx.doi.org/10.1027/0269-8803.22.1.5.

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This investigation covered the disgust and fear reactivity of patients suffering from spider phobia. We analyzed event-related brain potentials and affective responses to pictures depicting spiders, as well as generally fear-inducing, disgust-inducing, and neutral content, and compared them between 18 phobic and 18 nonphobic female participants. The patients rated the spider scenes as more fear- and disgust-inducing than the controls. This was accompanied by enhanced amplitudes of the P300 and the late positive potential (LPP). The other picture types elicited comparable electrocortical and affective responses in both groups. Separate group analyses showed that the patients were characterized by larger ERP positivity (P300, LPP) for phobic than for nonphobic material. Control subjects’ P300s were equally enhanced for spider, disgust and fear pictures relative to neutral scenes. Their LPPs were smaller for spiders compared to disgust and fear. Taken altogether, motivated attention in emotional picture processing is reflected by P300 and LPP modulation.
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Dal Seno, Bernardo, Matteo Matteucci, and Luca Mainardi. "Online Detection of P300 and Error Potentials in a BCI Speller." Computational Intelligence and Neuroscience 2010 (2010): 1–5. http://dx.doi.org/10.1155/2010/307254.

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Error potentials (ErrPs), that is, alterations of the EEG traces related to the subject perception of erroneous responses, have been suggested to be an elegant way to recognize misinterpreted commands in brain-computer interface (BCI) systems. We implemented a P300-based BCI speller that uses a genetic algorithm (GA) to detect P300s, and added an automatic error-correction system (ECS) based on the single-sweep detection of ErrPs. The developed system was tested on-line on three subjects and here we report preliminary results. In two out of three subjects, the GA provided a good performance in detecting P300 (90% and 60% accuracy with 5 repetitions), and it was possible to detect ErrP with an accuracy (roughly 60%) well above the chance level. In our knowledge, this is the first time that ErrP detection is performed on-line in a P300-based BCI. Preliminary results are encouraging, but further refinements are needed to improve performances.
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Shelley-Tremblay, John F., Joshua C. Eyer, and Benjamin D. Hill. "A Laboratory Word Memory Test Analogue Differentiates Intentional Feigning from True Responding Using the P300 Event-Related Potential." Brain Sciences 9, no. 5 (May 14, 2019): 109. http://dx.doi.org/10.3390/brainsci9050109.

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Symptom exaggeration and feigned cognitive impairment occur commonly in forensic and medicolegal evaluations. As a result, methods to detect feigned cognitive impairment are an indispensable component of neuropsychological assessments. This study reports the results of two neurophysiological experiments using a forced-choice recognition task built from the stimuli of the Word Memory Test and Medical Symptom Validity Test as well as a new linguistically informed stimulus set. Participant volunteers were instructed either to do their best or to feign cognitive impairment consistent with a mild traumatic brain injury while their brain activity was monitored using event-related potentials (ERP). Experiment 1 varied instructions across individuals, whereas Experiment 2 varied instructions within individuals. The target brain component was a positive deflection indicating stimulus recognition that occurs approximately 300 ms after exposure to a stimulus (i.e., the P300). Multimodal comparison (P300 amplitude to behavioral accuracy) allowed the detection of feigned cognitive impairment. Results indicate that, for correct responses, P300s were equivalent for the simulated malingering and good effort conditions. However, for incorrect responses, feigned impairment produced reliable but significantly reduced P300 amplitudes. Although the P300 is an automatic index of recognition—even when knowledge is hidden—its amplitude appears capable of modulation by feigning strategies. Implications of this finding are discussed for research and clinical applications.
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Poizat, Coralie, Pier Lorenzo Puri, Yan Bai, and Larry Kedes. "Phosphorylation-Dependent Degradation of p300 by Doxorubicin-Activated p38 Mitogen-Activated Protein Kinase in Cardiac Cells." Molecular and Cellular Biology 25, no. 7 (April 1, 2005): 2673–87. http://dx.doi.org/10.1128/mcb.25.7.2673-2687.2005.

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ABSTRACT p300 and CBP are general transcriptional coactivators implicated in different cellular processes, including regulation of the cell cycle, differentiation, tumorigenesis, and apoptosis. Posttranslational modifications such as phosphorylation are predicted to select a specific function of p300/CBP in these processes; however, the identification of the kinases that regulate p300/CBP activity in response to individual stimuli and the physiological significance of p300 phosphorylation have not been elucidated. Here we demonstrate that the cardiotoxic anticancer agent doxorubicin (adriamycin) induces the phosphorylation of p300 in primary neonatal cardiomyocytes. Hyperphosphorylation precedes the degradation of p300 and parallels apoptosis in response to doxorubicin. Doxorubicin-activated p38 kinases α and β associate with p300 and are implicated in the phosphorylation-mediated degradation of p300, as pharmacological blockade of p38 prevents p300 degradation. p38 phosphorylates p300 in vitro at both the N and C termini of the protein, and enforced activation of p38 by the constitutively active form of its upstream kinase (MKK6EE) triggers p300 degradation. These data support the conclusion that p38 mitogen-activated protein kinase regulates p300 protein stability and function in cardiomyocytes undergoing apoptosis in response to doxorubicin.
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Bragagnolo Júnior, Maurício Augusto, Vinícius Teodoro, Lígia Mendonça Lucchesi, Ruth Ferreira Santos, Ana Cristina de Castro Amaral Feldner, Tarsila Campanha da Rocha Ribeiro, Sérgio Tufik, and Mário Kondo. "Correlação entre a amônia e o potencial evocado relacionado a eventos (P300) em pacientes cirróticos." Revista Neurociências 17, no. 2 (January 23, 2019): 122–27. http://dx.doi.org/10.34024/rnc.2009.v17.8568.

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A amônia é a neurotoxina que melhor explica alterações cognitivas em cirróticos e o potencial evocado relacionado a eventos auditivos (P300) tem sido considerado um bom método para rastreá-las. Entretanto, é ainda desconhecido se pode ele pode ser influenciado pelas concentrações de amônia. Objetivo. avaliar a correlação entre amônia arterial e o P300 em cirróticos. Método. Cirróticos sem encefalopatia hepática foram submetidos à determinação da dosagem arterial de amônia arterial, P300 e avaliação da gravidade da doença hepática. O P300 foi anormal quando a latência de P300 foi superior ao limite superior da normalidade determinado por estudo normativo. Resultados. Avaliados 48 pacientes, com P300 anormal em 36 (75%). Aqueles com P300 anormal apresentaram amônia arterial significativamente maior do que aqueles com P300 normal (197±156 vs. 90±82 mmol/L; p <0,05). Amônia ³300 mmol/L também se associou à anormalidade do P300. Conclusão. A amônia arterial foi significativamente maior em cirróticos com P300 anormal e maiores níveis se associaram à anormalidade do exame, sugerindo que o P300 é boa ferramenta para rastrear anormalidades cognitivas em cirróticos, desde que se correlaciona com a principal substância envolvida na fisiopatogenia da encefalopatia hepática. >
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Sartorelli, V., J. Huang, Y. Hamamori, and L. Kedes. "Molecular mechanisms of myogenic coactivation by p300: direct interaction with the activation domain of MyoD and with the MADS box of MEF2C." Molecular and Cellular Biology 17, no. 2 (February 1997): 1010–26. http://dx.doi.org/10.1128/mcb.17.2.1010.

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By searching for molecules that assist MyoD in converting fibroblasts to muscle cells, we have found that p300 and CBP, two related molecules that act as transcriptional adapters, coactivate the myogenic basic-helix-loop-helix (bHLH) proteins. Coactivation by p300 involves novel physical interactions between p300 and the amino-terminal activation domain of MyoD. In particular, disruption of the FYD domain, a group of three amino acids conserved in the activation domains of other myogenic bHLH proteins, drastically diminishes the transactivation potential of MyoD and abolishes both p300-mediated coactivation and the physical interaction between MyoD and p300. Two domains of p300, at its amino and carboxy terminals, independently function to both mediate coactivation and physically interact with MyoD. A truncated segment of p300, unable to bind MyoD, acts as a dominant negative mutation and abrogates both myogenic conversion and transactivation by MyoD, suggesting that endogenous p300 is a required coactivator for MyoD function. The p300 dominant negative peptide forms multimers with intact p300. p300 and CBP serve as coactivators of another class of transcriptional activators critical for myogenesis, myocyte enhancer factor 2 (MEF2). In fact, transactivation mediated by the MEF2C protein is potentiated by the two coactivators, and this phenomenon is associated with the ability of p300 to interact with the MADS domain of MEF2C. Our results suggest that p300 and CBP may positively influence myogenesis by reinforcing the transcriptional autoregulatory loop established between the myogenic bHLH and the MEF2 factors.
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Gruber, Martina, Lavinia Ferrone, Martin Puhr, Frédéric R. Santer, Tobias Furlan, Iris E. Eder, Natalie Sampson, Georg Schäfer, Florian Handle, and Zoran Culig. "p300 is upregulated by docetaxel and is a target in chemoresistant prostate cancer." Endocrine-Related Cancer 27, no. 3 (March 2020): 187–98. http://dx.doi.org/10.1530/erc-19-0488.

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Administration of the microtubule inhibitor docetaxel is a common treatment for metastatic castration-resistant prostate cancer (mCRPC) and results in prolonged patient overall survival. Usually, after a short period of time chemotherapy resistance emerges and there is urgent need to find new therapeutic targets to overcome therapy resistance. The lysine-acetyltransferase p300 has been correlated to prostate cancer (PCa) progression. Here, we aimed to clarify a possible function of p300 in chemotherapy resistance and verify p300 as a target in chemoresistant PCa. Immunohistochemistry staining of tissue samples revealed significantly higher p300 protein expression in patients who received docetaxel as a neoadjuvant therapy compared to control patients. Elevated p300 expression was confirmed by analysis of publicly available patient data, where significantly higher p300 mRNA expression was found in tissue of mCRPC tumors of docetaxel-treated patients. Consistently, docetaxel-resistant PCa cells showed increased p300 protein expression compared to docetaxel-sensitive counterparts. Docetaxel treatment of PCa cells for 72 h resulted in elevated p300 expression. shRNA-mediated p300 knockdown did not alter colony formation efficiency in docetaxel-sensitive cells, but significantly reduced clonogenic potential of docetaxel-resistant cells. Downregulation of p300 in docetaxel-resistant cells also impaired cell migration and invasion. Taken together, we showed that p300 is upregulated by docetaxel, and our findings suggest that p300 is a possible co-target in treatment of chemoresistant PCa.
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Dissertations / Theses on the topic "P300"

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Odelade, Mobolaji. "P300 Control Matrix| A Novel Approach to P300 Speller Matrix." Thesis, North Carolina Agricultural and Technical State University, 2018. http://pqdtopen.proquest.com/#viewpdf?dispub=10976563.

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Over the years, researchers have been able to prove Brain Computer Interface (BCI) -P300 Speller as an effective communication tool. The first P300 speller was developed by Farwell and Donchin (1988), using the oddball paradigm to evoke a P300 response from a speller matrix. This P300 speller matrix has been a strong basis for studies that aimed at using BCI-P300 protocol for spelling, cursor movement, internet navigation or even control and manipulation of devices. However, application of P300 based BCI to controlling and manipulation of devices often involves the user relating with multiple interfaces. These multiple interfaces could be a distraction or have negative effects on the user (Fazel-Rezai et al. 2012) and as a consequence hinders the evoking of P300 potential and causing inaccurate classification. For this research, a novel P300 control matrix is developed by replacing the alphabets in the traditional P300 speller matrix with arrow images. Then the novel P300 control matrix was investigated to compare the P300 latency and amplitude to that of the traditional P300 speller matrix. The elements in the novel P300 control matrix were in form of arrows facing upward, left, right and downward directions, while elements in the P300 speller matrix were alphabets U, L, R and D for the upward, left, right and downward directions respectively. The participants were presented with a set of randomly sequenced directions, and each participant decides which of the arrows or letters to focus on based on the direction presented to them. Electroencephalography (EEG) was used to record the brainwaves using the international 10-20 system of electrode placement. This research is potentially a more efficient approach for controlling devices using P300-based BCI systems by eradicating the need for multiple interfaces associated with BCI-robotic control systems that are based on P300 speller.

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Iyer, N. G. "Functional characterisation of p300." Thesis, University of Cambridge, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604977.

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p300, originally discovered as an adenoviral El A binding protein, is a putative tumour suppressor gene. Mutational analysis done in our laboratory found truncating mutations in a range of cell lines and primary tumours, in some cases associated with loss of the second allele. In order to study the possible pathways leading from p300 loss to cancer, I adopted a somatic cell line knock out strategy to study the gene function in a human epithelial system. Using homologous recombination mediated gene targeting I successfully disrupted p300 in HCT116, by targeting the single expressed, truncated allele. Resultant cells were null for p300 protein. Rescue clones were generated by re-introducing full-length p300 cDNA into the knock-out clones. p300 knock-out (KO) cells were defective in proliferation, with prolonged doubling times and increased S-phase. A fraction of cells were in senescence/quiescence, and included a flat cell phenotype. These changes were reversed in the rescue clones. KO cells also showed a Gl to S-phase transition defect with early S phase entry after serum depletion and nocodazole arrest and release experiments. Rb pathways were deranged, principally by Rb phosphorylation defects, which probably led to the activation of the E2F pathway and early S-phase entry. KO cells also demonstrated abnormalities in p21 response during the cell cycle, which could have contributed to the cell cycle phenotype. In addition, KO cells were found to have reduced cell-cell adhesion, due to a decrease in E-cadherin at tight junctions and total E-cadherin levels. DNA damage experiments suggest that p300 KO cells are especially sensitive to UV mediated DNA damage with increased apoptosis seen 24 h after irradiation. This was due to abnormalities in the p53 pathway- a combination of increased stability of p53, reduced acetylation at lysine 382 and abnormal transactivation of downstream factors. In contrast, long term growth of KO cells after UV and XR irradiation showed increased resistance to DNA damage.
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Kaper, Matthias. "P300 based brain computer interfacing." [S.l.] : [s.n.], 2006. http://deposit.ddb.de/cgi-bin/dokserv?idn=979538203.

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Cota, Navin Gupta. "Advancing the P300 based BCI design." Thesis, University of Essex, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.617085.

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Brain Computer Interface (BCI) systems capture brain signals and convert them into commands without using peripheral nerves or muscles. This doctoral thesis embarks on advancing the design of P300 based BCI system using electroencephalogram (EEG) responses to visual and audio stimuli. This research work investigates techniques for pre processing, feature extraction as well as attempts some variations in paradigm design from a P300/0ddball paradigm perspective. Pre-processing is an important module in BCI systems. This thesis proposes a fully automated method (GALME-=ICA) to reduce artefacts effectively for an offline scenario. The method uses the recorded EEG channels only and does not require recorded EOG channels. The P300/0ddball systems were designed so as to reduce perceptual errors, thereby making them participant friendly. Few variations in paradigm design . reported here have not been attempted before. In one study, the spatial effect of the target stimuli location with respect to the non-target stimuli was explored. In another study limiting interference from irrelevant task related stimuli was studied. Results are discussed in terms of classification accuracies and bit rates. This thesis also proposes the usage of gamma band features with P300 time domain features for RSVP and audio based oddball paradigms for BCI based applications. An attractive integration of EEG-NIRS for monitoring participant concentration was also attempted. Preliminary results highlight the importance in selecting training datasets for good online classification results from an EEG viewpoint. There is no denying the fact that recent advances in BCI field have led researchers to explore new applications like cursor control and game control. However, there are many challenging problems which remain to be solved, before a commercial BCI system becomes a reality. Hence there is an urgent need to explore better system design and develop novel signal processing as well as machine learning algorithms. Imagination combined with practicality appears to be the key here. This Doctoral work is a step forward in this direction.
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Teufel, Daniel Paul. "The interactions of p300 with p53." Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.613972.

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Delvecchio, Manuela. "Mécanisme de régulation de l'acétyltransférase p300/CBP." Phd thesis, Université de Grenoble, 2011. http://tel.archives-ouvertes.fr/tel-00631344.

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Le p300/CBP acétyltransférase est un co-activateur transcriptionnel très important qui est impliqué dans la régulation d'un grand nombre de processus biologiques, comme la transcription d'ADN, le développement et l'immunité innée. Jusqu'à présent, le rôle de p300/CBP dans la régulation de l'expression des gènes a été largement étudiée, mais les mécanismes qui régulent son activité enzymatique sont encore peu connus. Des études ont montré que le dysfonctionnement de p300/CBP est associé à plusieurs formes de cancer et de maladies neurodégénératives. Dés lors, chaque progrès concernant les mécanismes de régulation de p300/CBP est devenu primordial pour le développement de nouvelles thérapies. Le 'noyau' de p300/CBP contient deux domaines pour la reconnaissance des modifications post-traductionnelles (MPTs), un bromodomaine et un PHD finger (le module BP), adjacent à un domaine HAT (ou domaine histone acétyltransférase). Plusieurs enzymes, modifiant la chromatine, contiennent des domaines de reconnaissance des MPTs. Fréquemment des groupements particuliers de ces domaines sont très conservés et liés, au sein de la même protéine ou du même complexe protéique, suggérant qu'ils réalisent des fonctions coordonnées. Ces domaines adjacents peuvent agir en concertation dans la reconnaissance simultanée de différents MPTs ou peuvent exercer des fonctions différentes de celles qui sont effectuées par ces deux domaines particuliers, tels que les fonctions de régulation enzymatique. Plusieurs études suggèrent que les cycles acétylation/désacétylation dans la boucle d'auto-inhibition, à l'intérieur du domaine HAT, jouent un rôle important dans la régulation de l'activité enzymatique de p300/CBP. La proximité du module BP et du domaine HAT suggère que la spécificité de liaison, appartenant au module BP, peut être intrinsèquement liée à la régulation de l'activité du domaine HAT. L'objectif de ma thèse est de déterminer le rôle du module BP dans la régulation de l'activité du domaine HAT. Je propose que le module BP soit impliqué dans la régulation de p300/CBP de deux façons. La première consiste à établir un lien avec le domaine HAT qui stabilise la conformation auto-inhibée de l'enzyme. La deuxième exige que le module BP joue un rôle dans le choix des substrats de p300/CBP. J'ai été en mesure de montrer que BP peut se lier au domaine HAT et à la chromatine modifiée et qu'il peut reconnaître les modifications effectuées par p300/CBP lui-même. Les données obtenues indiquent que le module BP peut être impliqué dans la régulation de l'activité de p300/CBP et dans son ciblage à la chromatine.
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Weston, Louise. "Functional studies on the p300 cofactor JMY." Thesis, University of Oxford, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534179.

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Santoso, Buyung. "Transcription repression by coactivator p300 in vitro /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2005. http://wwwlib.umi.com/cr/ucsd/fullcit?p3170244.

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Skinner, Tim. "Effect of Intensity Increment on P300 Amplitude." Scholar Commons, 2004. https://scholarcommons.usf.edu/etd/1251.

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The purpose of this study was to determine the effects of task difficulty on the amplitude and latency of the P300 by altering the intensity of the oddball stimulus. A P300 was obtained on 22 adult subjects ranging in age from 21 to 34 years of age (mean = 24 years) with normal hearing. The "frequent stimulus" was a 1000 Hz or 4000 Hz tone burst, gated with a rise and fall time of 10 msec and 20 msec plateau, presented at 75 dBn HL The "oddball stimulus" was a tone burst of the same frequency (1000 Hz or 4000 Hz)presented at 77, 79, or 81 dBn HL. A four-channel recording was made with linked reference electrodes and the following montages:Cz-A1+A2, Pz-A1+A2, and Fz-A1+A2. The fourth channel was used to monitor "eye blink" activity. The investigation tested the null hypothesis that changing the intensity of the oddball stimuli would not result in a significant change in either the amplitude or latency of the P300. Analyses of Variance (ANOVA) indicate that P300 latency and amplitude did not differ significantly by run, stimulus frequency, intensity of the oddball, or montage. Thus the null hypothesis was supported.
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Harpur, Timothy John. "Determining cerebral lateralisation : the use of the P300." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/25418.

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The P300 component of the average evoked potential was recorded at Pz during two divided visual field tasks. During a lexical decision task, reaction time and P300 latency were faster to stimuli in the right visual field, indicating that the latency of the P300 may be a useful measure in laterality research. A right visual field advantage was obtained for reaction time in a face perception task and the P300 latency difference showed a similar but non-significant advantage. Use of the P300 latency to assess the validity of the assumptions underlying the application of an additive factors model to divided visual field studies of cerebral assymetry was discussed. The present evidence suggests that the assumptions are valid.
Arts, Faculty of
Psychology, Department of
Graduate
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Books on the topic "P300"

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Kilpatrick, David. Pentax K1000, P30N/P3N and P30T. Hove, Sussex: Hove Foto Books, 1990.

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Kilpatrick, David. Pentax K1000 P30n/P3n and P30T. Surrey: Hove Fountain, 1990.

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Kilpatrick, David. Pentax K1000, P30N/P3N and P30T. Hove, Sussex: Hove Foto Books, 1990.

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1947-, Polich John, ed. Detection of change: Event-related potential and fMRI findings. Boston: Kluwer Academic Publishers, 2003.

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Polich, John. Neuropsychology of P300. Oxford University Press, 2011. http://dx.doi.org/10.1093/oxfordhb/9780195374148.013.0089.

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Timsit-Berthier, Martine, and Andrée Gerono. Manuel d'interprétation des potentiels évoqués endogènes (P300 et VCN). Mardaga, 1998.

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Tesar, George, Sibdas Ghosh, Steven W. Anderson, and Tom Bramorski. Strategic Technology Management. IMPERIAL COLLEGE PRESS, 2004. http://dx.doi.org/10.1142/p300.

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Ford, Greg R. The effects of simple and complex visual stimuli on the cortical distribution of P300. 1990.

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Jorg, J. Evozierte Potentiale in Klinik Und Praxis: Ein Einfuhrung in Vep, Aep, Mep, P300 Und Pap. 3rd ed. Springer-Verlag, 1995.

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(Editor), Johannes Jörg, and Horst Hielscher (Editor), eds. Evozierte Potentiale in Klinik und Praxis: Eine Einführung in VEP, SEP, AEP, MEP, P300 und PAP. 4th ed. Springer, 1997.

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Book chapters on the topic "P300"

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Zasler, Nathan D., and Jeffery Samuels. "P300." In Encyclopedia of Clinical Neuropsychology, 2541–42. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_56.

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Zasler, Nathan D., and Jeffery Samuels. "P300." In Encyclopedia of Clinical Neuropsychology, 1–2. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-56782-2_56-2.

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McAllister-Williams, R. Hamish, Daniel Bertrand, Hans Rollema, Raymond S. Hurst, Linda P. Spear, Tim C. Kirkham, Thomas Steckler, et al. "P300." In Encyclopedia of Psychopharmacology, 947. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1554.

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Samuels, Jeffery. "P300." In Encyclopedia of Clinical Neuropsychology, 1839–40. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_56.

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Reuter, B. M., and D. B. Linke. "P300 and Coma." In Topographic Brain Mapping of EEG and Evoked Potentials, 192–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-72658-3_16.

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Turnell, Andrew S. "CBP/p300 Coactivators." In Encyclopedia of Cancer, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_899-2.

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Turnell, Andrew S. "CBP/p300 Coactivators." In Encyclopedia of Cancer, 833–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_899.

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Eikmeier, G., H. M. Olbrich, E. Lodemann, D. Zerbin, C. Unger, and M. Gastpar. "P300 und schizophrene Negativsymptomatik." In Biologische Psychiatrie, 269–73. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77086-9_51.

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Demiralp, Tamer, Ahmet Ademoglu, and Erol Ba§ar. "Wavelet Analysis of Oddball P300." In Springer Series in Synergetics, 293–302. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59893-7_22.

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Casagrande, Alberto, Joanna Jarmolowska, Marcello Turconi, Francesco Fabris, and Piero Paolo Battaglini. "PolyMorph: A P300 Polymorphic Speller." In Lecture Notes in Computer Science, 297–306. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-02753-1_30.

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Conference papers on the topic "P300"

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Krusienski, Dean J., and Jerry J. Shih. "Control of a Brain-Computer Interface Using Intracranial Electrodes." In ASME 2011 6th Frontiers in Biomedical Devices Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/biomed2011-66077.

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A brain-computer interface (BCI) is a device that uses brain signals to provide a non-muscular communication channel [1], particularly for individuals with severe neuromuscular disabilities. One of the most promising signals for controlling a BCI are event-related potentials (ERPs) such as the P300. The P300 event related potential is an evoked response to an external stimulus that has been traditionally observed in scalp-recorded electroencephalography (EEG). The scalp-recorded P300 response has proven to be a reliable signal for controlling a BCI using the P300 Speller paradigm [2]. Recent studies have demonstrated that the P300 Speller has the potential to serve as an effective communication device for persons who have lost or are losing the ability to write and speak.
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Wee Lih Lee, Tele Tan, and Yee Hong Leung. "An improved P300 extraction using ICA-R for P300-BCI speller." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6611185.

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Esfahani, Ehsan Tarkesh, and V. Sundararajan. "Using Brain Computer Interfaces for Geometry Selection in CAD Systems: P300 Detection Approach." In ASME 2011 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2011. http://dx.doi.org/10.1115/detc2011-48775.

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The purpose of this paper is to explore the potential of brain-computer interfaces as user interfaces for CAD systems. The paper describes experiments and algorithms that use the BCI for selecting different surface of geometrical objects in the CAD systems using the P300 wave. The P300 (P3) wave is an event related potential (ERP) elicited by infrequent, stimuli (target faces flashing). Users wear an electroencephalogram (EEG) headset and try to select a target face of an object. Different faces of the object randomly flash which make the flashing of target face, an infrequent event. The EEG headset collects brain activity from 14 locations on the scalp. The data is analyzed with independent component analysis (ICA) and the discrete wavelet transforms (DWT) to detect the P300 component in the signal. The flashing face which causes the P300 component in the EEG signal is classified as the target face. Using a linear discriminant analysis, the target face is classified correctly with an average accuracy of 73.9%.
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Kubler, Andrea. "Quo vadis P300 BCI?" In 2017 5th International Winter Conference on Brain-Computer Interface (BCI). IEEE, 2017. http://dx.doi.org/10.1109/iww-bci.2017.7858151.

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Li, Yi, Jianhui Zhang, Yu Su, Weidong Chen, Yu Qi, Jicai Zhang, and Xiaoxiang Zheng. "P300 based BCI messenger." In 2009 ICME International Conference on Complex Medical Engineering - CME 2009. IEEE, 2009. http://dx.doi.org/10.1109/iccme.2009.4906636.

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Bernal, Sergio Lopez, Enrique Tomas Martinez Beltran, Mario Quiles Perez, Ruben Ortega Romero, Alberto Huertas Celdran, and Gregorio Martinez Perez. "Study of P300 Detection Performance by Different P300 Speller Approaches Using Electroencephalography." In 2022 IEEE 16th International Symposium on Medical Information and Communication Technology (ISMICT). IEEE, 2022. http://dx.doi.org/10.1109/ismict56646.2022.9828283.

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Lu, Zhaohua, Ruirui Zhang, and Qi Li. "Classification of P300 signals from P300 spelling system based on ASK-CNN model." In 2022 15th International Congress on Image and Signal Processing, BioMedical Engineering and Informatics (CISP-BMEI). IEEE, 2022. http://dx.doi.org/10.1109/cisp-bmei56279.2022.9980326.

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Giraudet a, Louise, Marie Berenger a, Jean-Paul Imbert b, Sébastien Tremblay c, and Mickaël Causse a. "Inattentional Deafness in Simulated Air Traffic Control Tasks: A Behavioral and P300 Analysis." In Applied Human Factors and Ergonomics Conference. AHFE International, 2019. http://dx.doi.org/10.54941/ahfe100219.

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The acoustic environment is critical in Air Traffic Control (ATC), as operators exchange information with planes and must also be aware of the occurrence of auditory alarms. In such situations, observing inattentional deafness is likely. In this study, we aimed to identify the physiological indicators of inattentional deafness through the analysis of the P300 evoked potential, known to be an indicator of attention allocation, an important step to a stimulus reaching consciousness. Based on the assumption that the high mental load generated by an ATC task may reduce the alarm detection rate, we wished to test whether this effect would be reflected in the alarm-evoked P300 amplitude. Participants had to perform simulated ATC tasks within the LABY microworld while electroencephalographic (EEG) activity was recorded. Simultaneously to the LABY tasks, participants were asked to respond to target tones (the “alarm”) and to ignore standard tones. Behavioral results showed that 4.6% of alarms were not reported. For these alarm detection failures, the EEG analysis showed a diminution of the P300 amplitude in comparison to a control condition in which participants only focused on the tones. These results suggest that the P300 amplitude seems to be a valid physiological indicator of vulnerability to inattentional deafness in complex environments. Relevant applications include the prevention of alarm omission and the assessment of warning designs.
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Yu, Moonwon, Netiwit Kaongoen, and Sungho Jo. "P300-BCI-based authentication system." In 2016 4th International Winter Conference on Brain-Computer Interface (BCI). IEEE, 2016. http://dx.doi.org/10.1109/iww-bci.2016.7457443.

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Lee, Sangmin, Yunjun Nam, and Seungjin Choi. "Self-labeling for P300 detection." In 2012 IEEE International Conference on Systems, Man and Cybernetics - SMC. IEEE, 2012. http://dx.doi.org/10.1109/icsmc.2012.6377712.

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Reports on the topic "P300"

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Deursen, Jan M. van. The Role of CBP/p300 in Breast Cancer Development. Fort Belvoir, VA: Defense Technical Information Center, September 2003. http://dx.doi.org/10.21236/ada424640.

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Trejo, Leonard J., Mark Inlow, Robert R. Stanny, William A. Morey, and Scott Makeig. The P300 Component of the Auditory Event-Related Potential: Interlaboratory Consistency and Test-Retest Reliability. Fort Belvoir, VA: Defense Technical Information Center, March 1991. http://dx.doi.org/10.21236/ada235114.

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