Journal articles: 'Oxygen uptake'

1

McConnell, Timothy R. "Oxygen Uptake." Journal of Cardiopulmonary Rehabilitation 23, no. 3 (May 2003): 190–92. http://dx.doi.org/10.1097/00008483-200305000-00005.

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SALTIN, BENGT, and S??REN STRANGE. "Maximal oxygen uptake." Medicine & Science in Sports & Exercise 24, no. 1 (January 1992): 30???37. http://dx.doi.org/10.1249/00005768-199201000-00007.

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3

Manns, P. J., C. R. Tomczak, and R. G. Haennel. "OXYGEN UPTAKE KINETICS." Journal of Cardiopulmonary Rehabilitation and Prevention 29, no. 5 (September 2009): 333. http://dx.doi.org/10.1097/01.hcr.0000361192.80278.bb.

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4

Jackson, D. M. "Estimating oxygen uptake." Anaesthesia 58, no. 6 (May 20, 2003): 615. http://dx.doi.org/10.1046/j.1365-2044.2003.03207_24.x.

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5

Meyer, T., J. Scharhag, and W. Kindermann. "Peak oxygen uptake." Zeitschrift f�r Kardiologie 94, no. 4 (April 2005): 255–64. http://dx.doi.org/10.1007/s00392-005-0207-4.

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6

Poole, David C., and Russell S. Richardson. "Determinants of Oxygen Uptake." Sports Medicine 24, no. 5 (November 1997): 308–20. http://dx.doi.org/10.2165/00007256-199724050-00003.

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7

Erickson, Howard. "Equine maximal oxygen uptake." Journal of Equine Veterinary Science 23, no. 7 (July 2003): 289. http://dx.doi.org/10.1053/jevs.2003.83.

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Erickson, Howard. "Equine Maximal Oxygen Uptake." Journal of Equine Veterinary Science 23, no. 7 (July 2003): 289. http://dx.doi.org/10.1016/s0737-0806(03)01003-7.

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9

Ribeiro, Jorge P., Ricardo Stein, and Gaspar R. S. Chiappa. "Beyond Peak Oxygen Uptake." Journal of Cardiopulmonary Rehabilitation 26, no. 2 (March 2006): 63–71. http://dx.doi.org/10.1097/00008483-200603000-00001.

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10

Vincent, J. L. "The relationship between oxygen demand, oxygen uptake, and oxygen supply." Intensive Care Medicine 16, S2 (February 1990): S145—S148. http://dx.doi.org/10.1007/bf01785244.

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11

VINCENT, J. L., and D. BACKER. "Oxygen uptake/oxygen supply dependency: Fact or fiction?" Acta Anaesthesiologica Scandinavica 39 (September 1995): 229–37. http://dx.doi.org/10.1111/j.1399-6576.1995.tb04364.x.

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12

Mines, Richard O. "Oxygen transfer parameters and oxygen uptake rates revisited." Journal of Environmental Science and Health, Part A 55, no. 4 (November 28, 2019): 345–53. http://dx.doi.org/10.1080/10934529.2019.1694817.

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13

Wolf, H. R. D., and S. Witte. "Blood rheology and oxygen uptake." Clinical Hemorheology and Microcirculation 10, no. 4 (December 9, 2016): 393–99. http://dx.doi.org/10.3233/ch-1990-10407.

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14

Xu, Fan, and Edward C. Rhodes. "Oxygen Uptake Kinetics During Exercise." Sports Medicine 27, no. 5 (1999): 313–27. http://dx.doi.org/10.2165/00007256-199927050-00003.

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15

HOWLEY, EDWARD T., DAVID R. BASSETT, and HUGH G. WELCH. "Criteria for maximal oxygen uptake." Medicine & Science in Sports & Exercise 27, no. 9 (September 1995): 1292???1301. http://dx.doi.org/10.1249/00005768-199509000-00009.

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16

Akkerman, Moniek, Marco van Brussel, Erik Hulzebos, Luc Vanhees, Paul J. M. Helders, and Tim Takken. "The Oxygen Uptake Efficiency Slope." Journal of Cardiopulmonary Rehabilitation and Prevention 30, no. 6 (2010): 357–73. http://dx.doi.org/10.1097/hcr.0b013e3181ebf316.

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17

Tamura, T., K. Sato, and T. Togawa. "Ambulatory oxygen uptake measurement system." IEEE Transactions on Biomedical Engineering 39, no. 12 (1992): 1274–82. http://dx.doi.org/10.1109/10.184703.

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18

Whaley, M. H., T. T. Tjepkema, L. A. Kaminsky, C. J. Grossman, K. D. Ryder, G. B. Dwyer, and L. H. Gelchell. "939 PREDICTING MAXIMAL OXYGEN UPTAKE." Medicine & Science in Sports & Exercise 25, Supplement (May 1993): S167. http://dx.doi.org/10.1249/00005768-199305001-00942.

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19

Neil, R. MacIntyre. "Oxygen Uptake and Mechanical Ventilation." Chest 96, no. 2 (August 1989): 446. http://dx.doi.org/10.1378/chest.96.2.446-a.

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20

Rolf, D. Hubmayr, M. Loosbrock Lynn, J. Gillespie Delmar, and R. Rodarte Joseph. "Oxygen Uptake and Mechanical Ventilation." Chest 96, no. 2 (August 1989): 446–47. http://dx.doi.org/10.1378/chest.96.2.446-b.

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21

Lawley, Sean D., Michael C. Reed, and H. Frederik Nijhout. "Spiracular fluttering increases oxygen uptake." PLOS ONE 15, no. 5 (May 20, 2020): e0232450. http://dx.doi.org/10.1371/journal.pone.0232450.

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22

Di Castro, Andrea, Sabrina Demarie, Carlo Minganti, Claudio Gallozzi, Roberto Tamburri, and Antonio Gianfelici. "Oxygen Uptake Efficiency Slope (oues)." Medicine & Science in Sports & Exercise 47 (May 2015): 120. http://dx.doi.org/10.1249/01.mss.0000476737.80351.d4.

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23

Wolf, H. R. D., and S. Witte. "Blood rheology and oxygen uptake." Biorheology 27, no. 6 (December 1, 1990): 913–19. http://dx.doi.org/10.3233/bir-1990-27612.

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24

Goodkin, David A., and James Stray-Gundersen. "Peak Oxygen Uptake in ESRD." American Journal of Kidney Diseases 57, no. 5 (May 2011): 803. http://dx.doi.org/10.1053/j.ajkd.2011.03.004.

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25

Zytner, Richard G., Ziyad G. Rahmé, and Michael Labocha. "Oxygen uptake at Parshall flumes." Canadian Journal of Civil Engineering 25, no. 4 (August 1, 1998): 769–76. http://dx.doi.org/10.1139/l98-010.

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Parshall flumes are frequently used to measure flow at municipal wastewater treatment plants. With the flume causing turbulent flow, there is the potential for the emission of volatile organic compounds. To assess the extent of the emissions, laboratory and field measurements at Parshall flumes were completed, using oxygen as a surrogate. The laboratory measurements showed that the most important parameters influencing oxygen uptake were total energy loss and downstream water depth. Satisfactory results were also obtained using drop height. Field results from three municipal wastewater treatment plants showed that oxygen uptake correlated strongly with drop height and only slightly with discharge rate. This is beneficial as downstream water depth is difficult to measure in the field. Findings suggest that the use of an appropriate weir model would allow the estimation of oxygen uptake and volatile organic compounds stripping at Parshall flumes.Key words: oxygen uptake, volatile organic compounds, flumes, wastewater.

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26

Midgley, Adrian W., Sean Carroll, David Marchant, Lars R. McNaughton, and Jason Siegler. "Evaluation of true maximal oxygen uptake based on a novel set of standardized criteria." Applied Physiology, Nutrition, and Metabolism 34, no. 2 (April 2009): 115–23. http://dx.doi.org/10.1139/h08-146.

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In this study, criteria are used to identify whether a subject has elicited maximal oxygen uptake. We evaluated the validity of traditional maximal oxygen uptake criteria and propose a novel set of criteria. Twenty athletes completed a maximal oxygen uptake test, consisting of an incremental phase and a subsequent supramaximal phase to exhaustion (verification phase). Traditional and novel maximal oxygen uptake criteria were evaluated. Novel criteria were: oxygen uptake plateau defined as the difference between modelled and actual maximal oxygen uptake >50% of the regression slope of the individual oxygen uptake–workrate relationship; as in the first criterion, but for maximal verification oxygen uptake; and a difference of ≤4 beats·min–1 between maximal heart rate values in the 2 phases. Satisfying the traditional oxygen uptake plateau criterion was largely an artefact of the between-subject variation in the oxygen uptake–workrate relationship. Secondary criteria, supposedly an indicator of maximal effort, were often satisfied long before volitional exhaustion, even at intensities as low as 61% maximal oxygen uptake. No significant mean differences were observed between the incremental and verification phases for oxygen uptake (t = 0.4; p = 0.7) or heart rate (t = 0.8; p = 0.5). The novel oxygen uptake plateau criterion, maximal oxygen uptake verification criterion, and maximal heart rate verification criterion were satisfied by 17, 18, and 18 subjects, respectively. The small individual absolute differences in oxygen uptake between incremental and verification phases observed in most subjects provided additional confidence that maximal oxygen uptake was elicited. Current maximal oxygen uptake criteria were not valid and novel criteria should be further explored.

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27

James, David V. B., Leigh E. Sandals, Stephen B. Draper, and Dan M. Wood. "Relationship between maximal oxygen uptake and oxygen uptake attained during treadmill middle-distance running." Journal of Sports Sciences 25, no. 8 (June 2007): 851–58. http://dx.doi.org/10.1080/02640410600875226.

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28

Sousa, Ana, Ferran A. Rodríguez, Leandro Machado, J. Paulo Vilas-Boas, and Ricardo J. Fernandes. "Exercise modality effect on oxygen uptake off-transient kinetics at maximal oxygen uptake intensity." Experimental Physiology 100, no. 6 (May 20, 2015): 719–29. http://dx.doi.org/10.1113/ep085014.

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29

Milley, J. R. "Uptake of exogenous substrates during hypoxia in fetal lambs." American Journal of Physiology-Endocrinology and Metabolism 254, no. 5 (May 1, 1988): E572—E578. http://dx.doi.org/10.1152/ajpendo.1988.254.5.e572.

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Fetal uptakes of oxygen, glucose, lactate, and alpha-amino-nitrogen containing substrates were measured in eight near-term fetal lambs by simultaneously drawing blood samples from the umbilical vein and descending aorta, then measuring umbilical blood flow using the microsphere method. These procedures were repeated after 3 h of hypoxia induced by lowering maternal inspired oxygen concentration. On the next day the experiment was repeated, except the ewes were first made hypoxic then allowed to breathe room air. These conditions decreased the delivery of oxygen, but not the delivery of other metabolic substrates to the fetus. During hypoxia, fetal oxygen uptake was 82% of normal (mean of both days); fetal glucose and amino-nitrogen uptakes were 74 and 23% of normal, respectively, and fetal lactate uptake became insignificant. These data indicate that endogenous rather than exogenous substrates are used to support fetal oxidative metabolism during hypoxia. Also, because exogenous uptake of amino-nitrogen is less than normal nitrogen accretion rates, fetal growth must be reduced as a consequence of 3-4 h of hypoxia.

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30

Milley, J. R., J. S. Papacostas, and B. K. Tabata. "Effect of insulin on uptake of metabolic substrates by the sheep fetus." American Journal of Physiology-Endocrinology and Metabolism 251, no. 3 (September 1, 1986): E349—E356. http://dx.doi.org/10.1152/ajpendo.1986.251.3.e349.

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To measure the effect of fetal hyperinsulinemia on fetal oxidative metabolic rate and the uptake of fetal oxidative substrates, we operated on 12 near-term ewes under spinal anesthesia and placed catheters in the fetus under local anesthesia. Four days after surgery, we began an 18-h insulin infusion, at the end of which we drew blood samples for analysis of oxygen, glucose, lactate, amino-nitrogen concentrations, blood gases, pH, hematocrit, and plasma insulin concentrations, then injected radiolabeled microspheres to measure umbilical blood flow. Three to five infusions were given to each fetus. Fetal plasma insulin concentrations varied from 0.3 to 60 microU/ml. As fetal plasma insulin concentration rose, the blood concentrations of oxygen, glucose, lactate, and amino-nitrogen fell, but the fetal uptakes of oxygen, glucose, and amino-nitrogen rose. The rise of fetal oxygen uptake occurred by increasing oxygen extraction, resulting in arterial hypoxemia. The increase of the glucose uptake was sufficient to account for an increased fraction of oxidative metabolism, allowing the increased uptake of amino acids to be used for either synthetic or oxidative purposes.

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31

Carlson, John S., and Geraldine A. Naughton. "An Examination of the Anaerobic Capacity of Children Using Maximal Accumulated Oxygen Deficit." Pediatric Exercise Science 5, no. 1 (February 1993): 60–71. http://dx.doi.org/10.1123/pes.5.1.60.

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The purpose of this study was to determine the anaerobic capacity of children using the maximal accumulated oxygen deficit technique (AOD). Eighteen healthy children (9 boys, 9 girls) with a mean age of 10.6 years volunteered as subjects. Peak oxygen uptake and submaximal steady-state oxygen uptake tests were conducted against progressive constant work rates on a Cybex cycle ergometer. Supramaximal work rates were predicted from the linear regression of submaximal steady-state work rates and oxygen uptakes to equal 110, 130, and 150% of peak oxygen uptake. Results indicated a significant interaction in the responses of both sexes when the accumulated oxygen deficit data were expressed in both absolute and relative terms. The profile of accumulated oxygen deficits across the three intensities indicated a downward shift in the girls responses between the 110 and 150% supramaximal tests. This trend was not evident in the boys’ responses. Intraclass correlations conducted on test-retest data indicated that compared to the boys, the reliability of the girls in the accumulated oxygen deficits in liters and ml·kg−1 was poorer.

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32

Forster, Margaret A., Gary R. Hunter, Donna J. Hester, Donna Dunaway, and Kathy Shuleva. "Aerobic Capacity and Grade-Walking Economy of Children 5–9 Years Old: A Longitudinal Study." Pediatric Exercise Science 6, no. 1 (February 1994): 31–38. http://dx.doi.org/10.1123/pes.6.1.31.

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Maximal oxygen uptake (VO2max), submaximal grade-walking oxygen uptake, and anthropometric measures were measured in a group of 19 children in 1988 and in 1992. The children were 5.2 ± .9 years old in 1988 and 9.2 ± 1.0 in 1992. The VO2max did not change relative to body weight over the 4 years (44.6 ml·kg−1·min−1 in 1988 versus 43.3 ml·kg−1·min−1 in 1992). Lower specific weight-relative oxygen uptakes were seen at the submaximal work levels in 1992 than in 1988, indicating an improvement in grade-walking economy.

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33

Kendall, S. C., T. Moritani, G. H. Hartung, and C. Pratt. "MAXIMAL OXYGEN UPTAKE AND OXYGEN KINETICS IN CARDIAC PATIENTS." Medicine & Science in Sports & Exercise 18, supplement (April 1986): S61. http://dx.doi.org/10.1249/00005768-198604001-00303.

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34

Weissman, Charles, and Marcia Kemper. "The Oxygen Uptake-Oxygen Delivery Relationship During ICU Interventions." Chest 99, no. 2 (February 1991): 430–35. http://dx.doi.org/10.1378/chest.99.2.430.

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35

Hughson, R. L., D. D. O'Leary, A. C. Betik, and H. Hebestreit. "Kinetics of oxygen uptake at the onset of exercise near or above peak oxygen uptake." Journal of Applied Physiology 88, no. 5 (May 1, 2000): 1812–19. http://dx.doi.org/10.1152/jappl.2000.88.5.1812.

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We tested the hypothesis that kinetics of O2 uptake (V˙o 2) measured in the transition to exercise near or above peakV˙o 2(V˙o 2 peak) would be slower than those for subventilatory threshold exercise. Eight healthy young men exercised at ∼57, ∼96, and ∼125%V˙o 2 peak. Data were fit by a two- or three-component exponential model and with a semilogarithmic transformation that tested the difference between required V˙o 2 and measuredV˙o 2. With the exponential model, phase 2 kinetics appeared to be faster at 125% V˙o 2 peak[time constant (τ2) = 16.3 ± 8.8 (SE) s] than at 57%V˙o 2 peak(τ2 = 29.4 ± 4.0 s) but were not different from that at 96%V˙o 2 peakexercise (τ2 = 22.1 ± 2.1 s).V˙o 2 at the completion of phase 2 was 77 and 80%V˙o 2 peak in tests predicted to require 96 and 125%V˙o 2 peak. WhenV˙o 2 kinetics were calculated with the semilogarithmic model, the estimated τ2 at 96%V˙o 2 peak (49.7 ± 5.1 s) and 125%V˙o 2 peak (40.2 ± 5.1 s) were slower than with the exponential model. These results are consistent with our hypothesis and with a model in which the cardiovascular system is compromised during very heavy exercise.

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36

Carr�, Fran�ois, Josette Dassonville, Jocelyne Beillot, Jean-Yves Prigent, and Pierre Rochcongar. "Use of oxygen uptake recovery curve to predict peak oxygen uptake in upper body exercise." European Journal of Applied Physiology and Occupational Physiology 69, no. 3 (May 1994): 258–61. http://dx.doi.org/10.1007/bf01094798.

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37

Kizaki, Z., and R. G. Thurman. "Stimulation of oxygen uptake by glucagon is oxygen dependent in perfused rat liver." American Journal of Physiology-Gastrointestinal and Liver Physiology 256, no. 2 (February 1, 1989): G369—G376. http://dx.doi.org/10.1152/ajpgi.1989.256.2.g369.

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Livers from well-fed female Sprague-Dawley rats (100-150 g) were perfused at flow rates of 4 or 8 ml.g liver-1.min-1 to deliver O2 to the organ at various rates. During perfusion at normal flow rates (4 ml.g-1.min-1), glucagon (10 nM) increased O2 uptake in perfused liver by approximately 40 mumol.g-1.h-1. In contrast, glucagon increased O2 uptake by nearly 100 mumol.g-1.h-1 when livers were perfused at high flow rates. Increase in O2 uptake was directly proportional to flow rate and was blocked partially by infusion of phorbol myristate acetate (100 nM) before glucagon. Increase in O2 uptake due to elevated flow was not due to enhanced glucagon delivery, since infusion of 120 nM glucagon at normal flow rates only increased O2 uptake by approximately 40 mumol.g-1.h-1. On the other hand, when O2 tension in the perfusate was manipulated at normal flow rates, the stimulation of O2 uptake by glucagon increased proportional to the average O2 tension in the liver. Infusion of 8-bromo-adenosine 3',5'-cyclic monophosphate (BrcAMP; 25 microM) also increased O2 uptake more than twice as much at high compared with normal flow rates. In the presence of angiotensin II (5 nM), a hormone that increases intracellular calcium, glucagon increased O2 uptake by nearly 100 mumol.g-1.h-1 at normal flow rates. Infusion of glucagon or BrcAMP into livers perfused at normal flow rates increased state 3 rates of O2 uptake of subsequently isolated mitochondria significantly by approximately 25%. In contrast, perfusion with glucagon or BrcAMP at high flow rates increased mitochondrial respiration by 50-60%. Glucagon addition acutely to suspensions of mitochondria, however, had no effect on O2 uptake. These data are consistent with reports that glucagon administration in vivo or treatment of intact cells with glucagon increases O2 uptake of subsequently isolated mitochondria, a phenomenon that can account for the observed increase in O2 uptake in livers perfused at high flow rates with glucagon. Furthermore, these results are consistent with the hypothesis that the effect of glucagon on mitochondria is O2 dependent in the perfused liver. This is most likely due to an effect of intracellular calcium on a mechanism mediated via cAMP.(ABSTRACT TRUNCATED AT 250 WORDS)

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38

Kilo, Martin, Sathya Swaroop, and Martin Lerch. "Oxygen Uptake and Diffusion in Mayenite." Defect and Diffusion Forum 289-292 (April 2009): 511–16. http://dx.doi.org/10.4028/www.scientific.net/ddf.289-292.511.

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Oxygen uptake and oxygen diffusion in Mayenite (Ca12Al14O33) were investigated using the stable tracer 18O2. Mayenite contains one intrinsic, highly mobile oxygen anion. It was shown that for high temperatures (above 700 °C), the diffusion goes through a interstitialcy mechanism, where the interstitial oxygen anion knocks out a lattice oxygen anion. The activation enthalpy for this process is around 0.9 eV, suggesting that the ionic migration is very fast.

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39

Schneider, Justine, Kathrin Schlüter, Joachim Wiskemann, and Friederike Rosenberger. "Do we underestimate maximal oxygen uptake in cancer survivors? Findings from a supramaximal verification test." Applied Physiology, Nutrition, and Metabolism 45, no. 5 (May 2020): 486–92. http://dx.doi.org/10.1139/apnm-2019-0560.

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Cancer survivors demonstrate a reduced maximal oxygen uptake, which is clinically relevant in terms of overall survival. However, it remains uncertain whether they attain their “true maximal oxygen uptake” in a cardiopulmonary exercise test (CPET). In the present study, a supramaximal verification bout (Verif) was applied in cancer survivors to confirm attainment of maximal oxygen uptake. Seventy-five participants (age, 61 ± 12 years; n = 43 females with breast cancer and n = 32 males with prostate cancer, 6–52 weeks after primary therapy) performed a CPET on a cycle ergometer and a Verif at 110% peak power output. As verification criterion, maximal oxygen uptake in Verif should not exceed maximal oxygen uptake in CPET by >3%. On average, maximal oxygen uptake was significantly lower in Verif compared with CPET (1.60 ± 0.38 L·min–1 vs. 1.65 ± 0.36 L·min–1, p = .023). On the individual level, n = 51 (68%) satisfied the verification criterion, whereas n = 24 (32%) demonstrated a higher maximal oxygen uptake in Verif. n = 69 (92%) fulfilled ≥2 secondary criteria for maximal exhaustion in the CPET. While maximal oxygen uptake was not underestimated in the CPET on average, one-third of cancer survivors did not attain their true maximal oxygen uptake. Verif appears feasible and beneficial to confirm true maximal oxygen uptake in this population. Furthermore, it might be more reliable than secondary criteria for maximal exhaustion. Novelty In about one-third of cancer survivors, maximal oxygen uptake is underestimated by a CPET. This underestimation of maximal oxygen uptake is not necessarily indicated by secondary criteria for maximal exhaustion. A supramaximal verification bout appears feasible and helpful for the determination of maximal oxygen uptake in cancer survivors.

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40

Canário-Lemos, Rui, José Vilaça-Alves, Tiago Moreira, Rafael Peixoto, Nuno Garrido, Fredric Goss, Hélio Furtado, and Victor Machado Reis. "Are Heart Rate and Rating of Perceived Exertion Effective to Control Indoor Cycling Intensity?" International Journal of Environmental Research and Public Health 17, no. 13 (July 4, 2020): 4824. http://dx.doi.org/10.3390/ijerph17134824.

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Indoor cycling’s popularity is related to the combination of music and exercise leading to higher levels of exercise intensity. It was our objective to determine the efficacy of heart rate and rating of perceived exertion in controlling the intensity of indoor cycling classes and to quantify their association with oxygen uptake. Twelve experienced males performed three indoor cycling sessions of 45 min that differed in the way the intensity was controlled: (i) oxygen uptake; (ii) heart rate; and (iii) rating of perceived exertion using the OMNI-Cycling. The oxygen uptake levels were significantly higher (p = 0.007; μp2 = 0.254) in oxygen uptake than heart rate sessions. Oxygen uptake related to body mass was significantly higher (p < 0.005) in the oxygen uptake sessions compared with other sessions. Strong correlations were observed between oxygen uptake mean in the oxygen uptake and rating of perceived exertion sessions (r =0.986, p < 0.0001) and between oxygen uptake mean in the oxygen uptake and heart rate sessions (r = 0.977, p < 0.0001). Both heart rate and rating of perceived exertion are effective in controlling the intensity of indoor cycling classes in experienced subjects. However, the use of rating of perceived exertion is easier to use and does not require special instrumentation.

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41

Barker, Alan R., Emily Trebilcock, Brynmor Breese, Andrew M. Jones, and Neil Armstrong. "The effect of priming exercise on O2 uptake kinetics, muscle O2 delivery and utilization, muscle activity, and exercise tolerance in boys." Applied Physiology, Nutrition, and Metabolism 39, no. 3 (March 2014): 308–17. http://dx.doi.org/10.1139/apnm-2013-0174.

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This study used priming exercise in young boys to investigate (i) how muscle oxygen delivery and oxygen utilization, and muscle activity modulate oxygen uptake kinetics during exercise; and (ii) whether the accelerated oxygen uptake kinetics following priming exercise can improve exercise tolerance. Seven boys that were aged 11.3 ± 1.6 years completed either a single bout (bout 1) or repeated bouts with 6 min of recovery (bout 2) of very heavy-intensity cycling exercise. During the tests oxygen uptake, muscle oxygenation, muscle electrical activity and exercise tolerance were measured. Priming exercise most likely shortened the oxygen uptake mean response time (change, ±90% confidence limits; –8.0 s, ±3.0), possibly increased the phase II oxygen uptake amplitude (0.11 L·min−1, ±0.09) and very likely reduced the oxygen uptake slow component amplitude (–0.08 L·min−1, ±0.07). Priming resulted in a likely reduction in integrated electromyography (–24% baseline, ±21% and –25% baseline, ±19) and a very likely reduction in Δ deoxyhaemoglobin/Δoxygen uptake (–0.16, ±0.11 and –0.09, ±0.05) over the phase II and slow component portions of the oxygen uptake response, respectively. A correlation was present between the change in tissue oxygenation index during bout 2 and the change in the phase II (r = –0.72, likely negative) and slow component (r = 0.72, likely positive) oxygen uptake amplitudes following priming exercise, but not for muscle activity. Exercise tolerance was likely reduced (change –177 s, ±180) following priming exercise. The altered phase II and slow component oxygen uptake amplitudes in boys following priming exercise are linked to an improved localised matching of muscle oxygen delivery to oxygen uptake and not muscle electrical activity. Despite more rapid oxygen uptake kinetics following priming exercise, exercise tolerance was not enhanced.

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42

Niemeyer, Max, Renate Leithaeuser, and Ralph Beneke. "Oxygen uptake plateau occurrence depends on oxygen kinetics and oxygen deficit accumulation." Scandinavian Journal of Medicine & Science in Sports 29, no. 10 (June 24, 2019): 1466–72. http://dx.doi.org/10.1111/sms.13493.

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43

WALSH, L. Michael. "Possible Mechanisms of Oxygen Uptake Kinetics." Annals of physiological anthropology 11, no. 3 (1992): 215–23. http://dx.doi.org/10.2114/ahs1983.11.215.

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44

MARUYAMA, Hitoshi, Akihiro NAKAYAMA, Kazuo KUROSAWA, and Taizo SHIOMI. "Maximal Oxygen Uptake and Motor Performance." Japanese journal of ergonomics 29, Supplement (1993): 540–41. http://dx.doi.org/10.5100/jje.29.supplement_540.

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45

Uchida, Katsuo. "Unit of oxygen uptake efficiency slope." Journal of Physical Fitness and Sports Medicine 7, no. 3 (May 25, 2018): 171–75. http://dx.doi.org/10.7600/jpfsm.7.171.

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46

WELLS, M. J., and J. WELLS. "Ventilation and Oxygen Uptake by Nautilus." Journal of Experimental Biology 118, no. 1 (September 1, 1985): 297–312. http://dx.doi.org/10.1242/jeb.118.1.297.

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The shell of Nautilus prevents the mantle from playing any part in creating the ventilatory stream. This is generated instead by movements of the collar and funnel folds which fuse to form flaps (the ‘wings’), overlapping below and joined to the head above. The gills lie horizontally, dividing the space enclosed by the wings into three cavities, two lateral and prebranchial, on either side of the head, and a common ventral postbranchial space. Water is drawn in above, behind the eyes, and expelled forward through the siphon of the funnel, which is used both for ventilation and jet propulsion. The pressures driving the ventilatory stream are small (of the order of 0.1 kPa), but the complex movement of the wings, described below, is such that there is (very nearly) always a pressure differential and a water flow across the gills, despite a pulsed intake and outward jet. Oxygen extraction is low by the standards of other cephalopods, only 5–10%, falling during jet propulsion and rising (exceptionally to 40%) at rest after exercise. Ventilation frequency, 35 min−1 at 16°C, rises with temperature. Ventilation stroke volumes ranged from 5 to 22 ml for an animal of 395 g. At 17 °C Nautilus can regulate its oxygen uptake down to a Po2 of about 75 mmHg. Uptake at rest ranged from 0.22 to 0.46 ml kg−1 min−1 (exceptionally 0.75 ml kg−1 min−1 after feeding) for animals of 351–395 g. In terms of flesh weight, this yields an average of 0.50mlkg−1 min−1, half to one-third of the uptake one would expect from coleoids of similar weights and at similar temperatures.

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Hillman, K. "Oxygen uptake by porcine colon contents." Proceedings of the British Society of Animal Science 1998 (1998): 163. http://dx.doi.org/10.1017/s1752756200598159.

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Since the gut environment is generally assumed to be anaerobic, studies of the monogastric intestinal microflora have concentrated on the anaerobic microorganisms. However, the intestinal epithelium constitutes a very high surface area in relation to the volume of the gut contents, and all of this surface area is richly supplied with blood. Appreciable dissolved oxygen concentrations can be detected in the intestines of anaesthetised piglets (Hillman et al, 1993), although this is never detectable in the intestines of killed animals. It was considered important to investigate the persistence of intestinal oxygen after death, in order to determine the accuracy of postmortem measurements of this component of the intestinal environment. This report demonstrates the rapidity of removal of oxygen by porcine colon contents, and indicates that, since the oxygen supply to the intestine stops when the blood circulation stops, accurate measurements of dissolved oxygen can never be made in the intestines of killed animals.Colon contents were obtained from three freshly slaughtered 20 kg piglets and diluted to 20% (w/v) slurries in quarter-strength Ringer's solution (Unipath, UK). The slurries were transferred (25 ml) to 50 ml conical flasks, stirred by magnetic follower (approx 400 rpm) and surrounded by a water jacket to maintain a temperature of 39°C in the vessel contents. A polarographic oxygen electrode was inserted so that the membrane was submerged. Gas flow through the headspace was provided by compressed air and nitrogen, mixed via a pair of needle valves to provide a constant flow rate of 600 ml min-1. Calibration of the system was performed in sterile Ringer's solution using air and oxygen-free nitrogen: calibration was repeated between each experiment.

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Rapp, D., J. Scharhag, S. Wagenpfeil, and J. Scholl. "Reference values for peak oxygen uptake." Deutsche Zeitschrift für Sportmedizin 2018, no. 6 (June 1, 2018): 199–205. http://dx.doi.org/10.5960/dzsm.2018.329.

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HIGUCHI, TAKANAO, TOSHIYO TAMURA, and TATSUO TOGAWA. "EVALUATION OF AMBULATORY OXYGEN UPTAKE MONITOR." Japanese Journal of Physical Fitness and Sports Medicine 40, no. 2 (1991): 195–201. http://dx.doi.org/10.7600/jspfsm1949.40.195.

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Armstrong, N., J. R. Welsman, and B. J. Kirby. "PEAK OXYGEN UPTAKE AND MATURATION 7." Medicine &amp Science in Sports &amp Exercise 29, Supplement (May 1997): 2. http://dx.doi.org/10.1097/00005768-199705001-00007.

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Journal articles on the topic 'Oxygen uptake'

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