Academic literature on the topic 'Overnight oxygen saturation'
Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles
Contents
Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Overnight oxygen saturation.'
Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.
You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.
Journal articles on the topic "Overnight oxygen saturation"
Lario, Bonifacio Alvarez, José Luis Alonso Valdivielso, Javier Alegre López, Carlos Martel Soteres, José Luis Viejo Bañuelos, and Angel Marañón Cabello. "Fibromyalgia Syndrome: Overnight Falls in Arterial Oxygen Saturation." American Journal of Medicine 101, no. 1 (July 1996): 54–60. http://dx.doi.org/10.1016/s0002-9343(96)00067-8.
Full textKobayashi, Tadaharu, Akinori Funayama, Daichi Hasebe, Yusuke Kato, Michiko Yoshizawa, and Chikara Saito. "Changes in overnight arterial oxygen saturation after mandibular setback." British Journal of Oral and Maxillofacial Surgery 51, no. 4 (June 2013): 312–18. http://dx.doi.org/10.1016/j.bjoms.2012.07.004.
Full textvan Geyzel, Lisa, Michele Arigliani, Baba Inusa, Bethany Singh, Wanda Kozlowska, Subarna Chakravorty, Cara J. Bossley, Gary Ruiz, David Rees, and Atul Gupta. "Higher oxygen saturation with hydroxyurea in paediatric sickle cell disease." Archives of Disease in Childhood 105, no. 6 (December 23, 2019): 575–79. http://dx.doi.org/10.1136/archdischild-2019-317862.
Full textJohnson, Mark C., Fenella J. Kirkham, Susan Redline, Carol L. Rosen, Yan Yan, Irene Roberts, Jeanine Gruenwald, Jan Marek, and Michael R. DeBaun. "Left ventricular hypertrophy and diastolic dysfunction in children with sickle cell disease are related to asleep and waking oxygen desaturation." Blood 116, no. 1 (July 8, 2010): 16–21. http://dx.doi.org/10.1182/blood-2009-06-227447.
Full textClark, M., B. Cooper, S. Singh, M. Cooper, A. Carr, and R. Hubbard. "A survey of nocturnal hypoxaemia and health related quality of life in patients with cryptogenic fibrosing alveolitis." Thorax 56, no. 6 (June 1, 2001): 482–86. http://dx.doi.org/10.1136/thx.56.6.482.
Full textAl Rajeh, Ahmed M., Yousef Saad Aldabayan, Abdulelah Aldhahir, Elisha Pickett, Shumonta Quaderi, Jaber S. Alqahtani, Swapna Mandal, Marc CI Lipman, and John R. Hurst. "Once Daily Versus Overnight and Symptom Versus Physiological Monitoring to Detect Exacerbations of Chronic Obstructive Pulmonary Disease: Pilot Randomized Controlled Trial." JMIR mHealth and uHealth 8, no. 11 (November 13, 2020): e17597. http://dx.doi.org/10.2196/17597.
Full textDillon, Richard, Patrick Murphy, Craig Davidson, Adrian Williams, Kate Brignall, Sean Higgins, Dawn Evans, Nicholas Hart, and Jo Howard. "Prevalence of Nocturnal Hypoxia and Its Association with Disease Severity in Adults with Sickle Cell Disease." Blood 114, no. 22 (November 20, 2009): 261. http://dx.doi.org/10.1182/blood.v114.22.261.261.
Full textTerrill, Philip Ian, Carolyn Dakin, Ian Hughes, Maggie Yuill, and Chloe Parsley. "Nocturnal oxygen saturation profiles of healthy term infants." Archives of Disease in Childhood 100, no. 1 (July 25, 2014): 18–23. http://dx.doi.org/10.1136/archdischild-2013-305708.
Full textHollocks, Matthew J., Tessa B. Kok, Fenella J. Kirkham, Johanna Gavlak, Baba P. Inusa, Michael R. DeBaun, and Michelle de Haan. "Nocturnal Oxygen Desaturation and Disordered Sleep as a Potential Factor in Executive Dysfunction in Sickle Cell Anemia." Journal of the International Neuropsychological Society 18, no. 1 (November 24, 2011): 168–73. http://dx.doi.org/10.1017/s1355617711001469.
Full textLiguoro, Ilaria, Michele Arigliani, Bethany Singh, Lisa Van Geyzel, Subarna Chakravorty, Cara Bossley, Maria Pelidis, David Rees, Baba P. D. Inusa, and Atul Gupta. "Beneficial effects of adenotonsillectomy in children with sickle cell disease." ERJ Open Research 6, no. 4 (October 2020): 00071–2020. http://dx.doi.org/10.1183/23120541.00071-2020.
Full textDissertations / Theses on the topic "Overnight oxygen saturation"
Johnson, Pamela Lesley. "Sleep and Breathing at High Altitude." University of Sydney, 2008. http://hdl.handle.net/2123/3531.
Full textThis thesis describes the work carried out during four treks, each over 10-11 days, from 1400m to 5000m in the Nepal Himalaya and further work performed during several two-night sojourns at the Barcroft Laboratory at 3800m on White Mountain in California, USA. Nineteen volunteers were studied during the treks in Nepal and seven volunteers were studied at White Mountain. All subjects were normal, healthy individuals who had not travelled to altitudes higher than 1000m in the previous twelve months. The aims of this research were to examine the effects on sleep, and the ventilatory patterns during sleep, of incremental increases in altitude by employing portable polysomnography to measure and record physiological signals. A further aim of this research was to examine the relationship between the ventilatory responses to hypoxia and hypercapnia, measured at sea level, and the development of periodic breathing during sleep at high altitude. In the final part of this thesis the possibility of preventing and treating Acute Mountain Sickness with non-invasive positive pressure ventilation while sleeping at high altitude was tested. Chapter 1 describes the background information on sleep, and breathing during sleep, at high altitudes. Most of these studies were performed in hypobaric chambers to simulate various high altitudes. One study measured sleep at high altitude after trekking, but there are no studies which systematically measure sleep and breathing throughout the whole trek. Breathing during sleep at high altitude and the physiological elements of the control of breathing (under normal/sea level conditions and under the hypobaric, hypoxic conditions present at high altitude) are described in this Chapter. The occurrence of Acute Mountain Sickness (AMS) in subjects who travel form near sea level to altitudes above 3000m is common but its pathophysiology not well understood. The background research into AMS and its treatment and prevention are also covered in Chapter 1. Chapter 2 describes the equipment and methods used in this research, including the polysomnographic equipment used to record sleep and breathing at sea level and the high altitude locations, the portable blood gas analyser used in Nepal and the equipment and methodology used to measure each individual’s ventilatory response to hypoxia and hypercapnia at sea level before ascent to the high altitude locations. Chapter 3 reports the findings on the changes to sleep at high altitude, with particular focus on changes in the amounts of total sleep, the duration of each sleep stage and its percentage of total sleep, and the number and causes of arousals from sleep that occurred during sleep at increasing altitudes. The lightest stage of sleep, Stage 1 non-rapid eye movement (NREM) sleep, was increased, as expected with increases in altitude, while the deeper stages of sleep (Stages 3 and 4 NREM sleep, also called slow wave sleep), were decreased. The increase in Stage 1 NREM in this research is in agreement with all previous findings. However, slow wave sleep, although decreased, was present in most of our subjects at all altitudes in Nepal; this finding is in contrast to most previous work, which has found a very marked reduction, even absence, of slow wave sleep at high altitude. Surprisingly, unlike experimental animal studies of chronic hypoxia, REM sleep was well maintained at all altitudes. Stage 2 NREM and REM sleep, total sleep time, sleep efficiency and spontaneous arousals were maintained at near sea level values. The total arousal index was increased with increasing altitude and this was due to the increasing severity of periodic breathing as altitude increased. An interesting finding of this research was that fewer than half the periodic breathing apneas and hypopneas resulted in arousal from sleep. There was a minor degree of upper airway obstruction in some subjects at sea level but this was almost resolved by 3500m. Chapter 4 reports the findings on the effects on breathing during sleep of the progressive increase of altitude, in particular the occurrence of periodic breathing. This Chapter also reports the results of changes to arterial blood gases as subjects ascended to higher altitudes. As expected, arterial blood gases were markedly altered at even the lowest altitude in Nepal (1400m) and this change became more pronounced at each new, higher altitude. Most subjects developed periodic breathing at high altitude but there was a wide variability between subjects as well as variability in the degree of periodic breathing that individual subjects developed at different altitudes. Some subjects developed periodic breathing at even the lowest altitude and this increased with increasing altitude; other subjects developed periodic breathing at one or two altitudes, while four subjects did not develop periodic breathing at any altitude. Ventilatory responses to hypoxia and hypercapnia, measured at sea level before departure to high altitude, was not significantly related to the development of periodic breathing when the group was analysed as a whole. However, when the subjects were grouped according to the steepness of their ventilatory response slopes, there was a pattern of higher amounts of periodic breathing in subjects with steeper ventilatory responses. Chapter 5 reports the findings of an experimental study carried out in the University of California, San Diego, Barcroft Laboratory on White Mountain in California. Seven subjects drove from sea level to 3800m in one day and stayed at this altitude for two nights. On one of the nights the subjects slept using a non-invasive positive pressure device via a face mask and this was found to significantly improve the sleeping oxyhemoglobin saturation. The use of the device was also found to eliminate the symptoms of Acute Mountain Sickness, as measured by the Lake Louise scoring system. This finding appears to confirm the hypothesis that lower oxygen saturation, particularly during sleep, is strongly correlated to the development of Acute Mountain Sickness and may represent a new treatment and prevention strategy for this very common high altitude disorder.