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Relevant bibliographies by topics / Overload model

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Academic literature on the topic 'Overload model'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Overload model"

1

Tsay, Jaime, Zheiwei Yang, F. Patrick Ross, Susanna Cunningham-Rundles, Hong Lin, Rhima Coleman, Philipp Mayer-Kuckuk, et al. "Bone loss caused by iron overload in a murine model: importance of oxidative stress." Blood 116, no. 14 (October 7, 2010): 2582–89. http://dx.doi.org/10.1182/blood-2009-12-260083.

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AbstractOsteoporosis is a frequent problem in disorders characterized by iron overload, such as the thalassemias and hereditary hemochromatosis. The exact role of iron in the development of osteoporosis in these disorders is not established. To define the effect of iron excess in bone, we generated an iron-overloaded mouse by injecting iron dextran at 2 doses into C57/BL6 mice for 2 months. Compared with the placebo group, iron-overloaded mice exhibited dose-dependent increased tissue iron content, changes in bone composition, and trabecular and cortical thinning of bone accompanied by increased bone resorption. Iron-overloaded mice had increased reactive oxygen species and elevated serum tumor necrosis factor-α and interleukin-6 concentrations that correlated with severity of iron overload. Treatment of iron-overloaded mice with the antioxidant N-acetyl-L-cysteine prevented the development of trabecular but not cortical bone abnormalities. This is the first study to demonstrate that iron overload in mice results in increased bone resorption and oxidative stress, leading to changes in bone microarchitecture and material properties and thus bone loss.

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Stroebe, Margaret, and Henk Schut. "Overload." OMEGA - Journal of Death and Dying 74, no. 1 (September 21, 2016): 96–109. http://dx.doi.org/10.1177/0030222816666540.

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The Dual Process Model of Coping with Bereavement (DPM) was put forward as a framework to help understand reactions to the death of a loved person. Since its inception, there have been various developments and further specifications regarding the model’s parameters. A number of researchers have assessed the model’s contribution and put some of its parameters to empirical test. It has also been applied in clinical practice. Despite generally positive assessment among both scientific and applied communities, we recently discovered what we consider to be a major shortcoming. The concept of overload has been neglected. Incorporation of this feature helps explain the preponderance of mental and physical health problems beyond the previous DPM focus on complications of grief. In this article, we incorporate the phenomenon of overload within the original framework, illustrating its application, and we discuss broader implications for coping and adaptation to bereavement.

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Kidd, James M., Kamilia Abdelraouf, and David P. Nicolau. "1553. Human-Simulated Pharmacokinetic Profiles of Cefiderocol and Meropenem Are Conserved in Murine Models of Thigh Infection With or Without Iron Overload." Open Forum Infectious Diseases 6, Supplement_2 (October 2019): S567. http://dx.doi.org/10.1093/ofid/ofz360.1417.

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Abstract Background A translational murine model of thigh infection with comorbid iron overload was previously developed to study the efficacy of iron-dependent siderophore-antibiotic conjugates under conditions where the hypoferremic response of innate immunity may be compromised. Given the potential for functional organ damage from excessive tissue iron, which could alter the pharmacokinetic (PK) profiles of antibiotics being compared for efficacy using this model, the effects of iron overload on a siderophore-β-lactam conjugate, cefiderocol (CFDC), and a non-siderophore β-lactam, meropenem (MEM), were studied. Methods Female CD-1 mice received iron dextran (Fe-D) 100 mg/kg intraperitoneally for 14 days as previously shown to produce vastly supranormal iron concentrations in serum, liver, and spleen (ASM Microbe 2019 abstract HMB-373). Age-matched control mice were not dosed with Fe-D. Mice were rendered neutropenic. On day 15, both thighs of iron-overloaded and control mice were inoculated intramuscularly with Acinetobacter baumannii suspensions of 107 CFU/mL. Two hours after inoculation, mice in each model were dosed with previously developed human-simulated regimens (HSR) of CFDC or MEM simulating human PK profiles after doses of 2g q8h (3 hours infusion) for both drugs. At 4 time points per regimen, 6 mice per model were sacrificed for blood collection. Plasma total MEM and CFDC concentrations were measured with HPLC and LC-MS-MS, respectively. Free concentrations were calculated with murine protein binding. At each time point, mean free concentrations in both models were compared using Student’s t-test. Results Observed murine-free plasma concentrations ± 95% CI of CFDC and MEM are overlaid with simulated human and murine profiles in the figure. In both models, these regimens approximated human exposures after clinical doses. For all time points and both drugs, concentrations were not significantly different (P > 0.05) between models with or without iron overload. Conclusion Iron overload did not significantly alter PK profiles of a siderophore-β-lactam conjugate, CFDC, or a non-siderophore β-lactam, MEM. These data support the use of CFDC and MEM HSR for pharmacodynamic studies utilizing both iron-overloaded and standard murine thigh infection models. Disclosures All authors: No reported disclosures.

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Huang, Jiawei, Qingchun Meng, Zhixin Zhan, Weiping Hu, and Fei Shen. "Damage mechanics-based approach to studying effects of overload on fatigue life of notched specimens." International Journal of Damage Mechanics 28, no. 4 (May 9, 2018): 538–65. http://dx.doi.org/10.1177/1056789518775173.

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A continuum damage mechanics-based method is adopted to predict the fatigue life of notched specimens subjected to constant amplitude cyclic loading while containing single or multiple overloads. The residual stress and plastic damage induced by an overload are considered to be the main factors affecting the fatigue life of a specimen. The residual stress and plastic strain fields of a notched specimen are calculated using the elastic–plastic finite element method. The mean stress of the following cyclic loading is then varied by superimposing the residual stress. Meanwhile, the plastic damage is calculated based on the ductile damage model and accumulated into the total damage of the material. The quantitative effects of an overload on the damage evolution and the fatigue life are evaluated. Furthermore, the effects of the damage–overload ratio on the variation of the residual stress induced by an overload are investigated, and the effects of the occurrence time for a single overload and the occurrence frequency for multiple overloads are studied.

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Rushton, Philip A., Farid Taheri, and David C. Stredulinsky. "Fatigue Response and Characterization of 350WT Steel Under Semi-Random Loading." Journal of Pressure Vessel Technology 129, no. 3 (March 9, 2006): 525–34. http://dx.doi.org/10.1115/1.2748835.

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Novel data obtained through experimental investigation into the fatigue response of 350WT steel, subjected to semi-random loading, comprised of various combinations of intermittent tensile overloads and compressive underloads are presented. An effective model for predicting the fatigue response is also introduced. For that, the capabilities of some of the currently available models are investigated and then an exponential delay model, being capable of accounting for the effects of not only overload ratio, but also stress ratio and overload/underload ratio is introduced. Since most variable amplitude models are based on a constant amplitude model, efforts were also expended to identify a constant amplitude fatigue crack growth model that would be easy to use, requiring the calibration of few (if any) empirical curve-fitting parameters. The integrity of a selected model is examined and results are presented.

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Zou, Xiao Li, and Jian Hui Yang. "Simulation of Fatigue Crack Growth Under Random Overloads with Retardation." Key Engineering Materials 324-325 (November 2006): 923–26. http://dx.doi.org/10.4028/www.scientific.net/kem.324-325.923.

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In order to study the behavior of fatigue crack propagation under random overloads, a Monte Carlo simulation scheme is proposed. Overloads of Poisson flow with uniform distribution on base-line constant-amplitude cyclic loads are considered. The retardation effect of overload is taken into account using crack closure model and the crack opening stress level is assumed to vary linearly in the yield zone produced by the overload. The fatigue crack growth curve from initial crack size till fatigue failure is simulated step by step. Through the large number of the simulated samples, the average fatigue crack propagation life is calculated. Finally, the influence of overload intensity and magnitude on fatigue crack propagation life is studied.

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Pasipoularides, Ares, Ming Shu, Ashish Shah, Scott Silvestry, and Donald D. Glower. "Right ventricular diastolic function in canine models of pressure overload, volume overload, and ischemia." American Journal of Physiology-Heart and Circulatory Physiology 283, no. 5 (November 1, 2002): H2140—H2150. http://dx.doi.org/10.1152/ajpheart.00462.2002.

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By limiting filling, abnormalities of right ventricular (RV) diastolic function may impair systolic function and affect adaptation to disease. To quantify diastolic RV pressure-volume relations and myocardial compliance (MC), a new sigmoidal model was developed. RV micromanometric and sonomicrometric data in alert dogs at control ( n = 16) and under surgically induced subacute (2–5 wk) RV pressure overload ( n = 6), volume overload ( n = 7), and ischemia ( n = 6) were analyzed. The conventional exponential model detected no changes from control in the passive filling pressure-volume (Ppf-V) relations. The new sigmoidal model revealed significant quantifiable changes in Ppf-V relations. Maximum RV MC (MCmax), attained during early filling, is reduced from control in pressure overload ( P = 0.0016), whereas filling pressure at maximum MC (PMCmax) is increased ( P = 0.0001). End-diastolic RV MC increases significantly in volume overload ( P = 0.0131), whereas end-diastolic pressure is unchanged. In ischemia, MCmax is decreased ( P = 0.0102), with no change in PMCmax. We conclude that the sigmoidal model quantifies important changes in RV diastolic function in alert dog models of pressure overload, volume overload, and ischemia.

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Alagna, Sheryle W. "An Overload(ed) Model of Stress Effects." Contemporary Psychology: A Journal of Reviews 30, no. 6 (June 1985): 499–500. http://dx.doi.org/10.1037/023876.

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Goel, H. S., and Satish Chand. "A Fatigue Crack Growth Model for Single Overload Tests." Journal of Engineering Materials and Technology 116, no. 2 (April 1, 1994): 168–72. http://dx.doi.org/10.1115/1.2904268.

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Crack growth rate experiments are conducted by applying an intermediate single overload cycle in constant amplitude load (CAL) tests. For a particular overload ratio, three to four tests are conducted by applying the overload cycle at different crack lengths. The loads are selected in such a way that for a given overload ratio, the size of the overload and CAL monotonic plastic zones are the same at each crack length. A functional form for the crack growth during the retardation was developed that accurately describes all the tests. For comparison, the corresponding CA-load tests are also conducted separately. Finally, a crack growth rate model is developed.

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Yoshihara, Fumiki, Toshio Nishikimi, Takeshi Horio, Chikao Yutani, Noritoshi Nagaya, Hisayuki Matsuo, Tohru Ohe, and Kenji Kangawa. "Ventricular adrenomedullin concentration is a sensitive biochemical marker for volume and pressure overload in rats." American Journal of Physiology-Heart and Circulatory Physiology 278, no. 2 (February 1, 2000): H633—H642. http://dx.doi.org/10.1152/ajpheart.2000.278.2.h633.

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This study was designed to investigate the pathophysiological significance of adrenomedullin (AM) concentration in volume- and pressure-overloaded cardiac hypertrophy. We measured ventricular AM concentrations and compared them with changes of α-actin and myosin heavy chain (MHC) mRNA isoforms after the creation of an aortocaval (AC) shunt as a volume-overload model or the injection of monocrotaline (MCT) as a pressure-overload model, respectively. The left ventricular AM levels after the creation of AC shunt and the right ventricular AM levels after the injection of MCT were significantly increased and correlated with changes of the α-actin and MHC mRNA isoforms. However, the ventricular AM mRNA expressions were increased and correlated with ventricular AM concentrations only in the AC shunt model. These results suggest that the ventricular AM levels are upregulated in both the volume- and pressure-overloaded cardiac hypertrophy by differential transcriptional regulation and that the ventricular AM may be a biochemical marker for the volume and pressure overload to the ventricle.

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Dissertations / Theses on the topic "Overload model"

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Crowe, Sarah Lynn. "Cardioprotective effects of dihydropyridine antagonists in a murine model of chronic iron-overload." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2002. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/MQ65615.pdf.

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Yardimci, Ulaş Izmen. "Enough is as good as a feast : a simple model of choice overload." Master's thesis, Instituto Superior de Economia e Gestão, 2018. http://hdl.handle.net/10400.5/18013.

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Mestrado em Economia
O pressuposto fundamental da teoria económica neoclássica é a racionalidade dos agentes econômicos. No entanto, no tempo, desvios sistemáticos desta suposição foram observados. Vários desses desvios dizem respeito ao processo de tomada de decisão e um deles é a carga aparentemente contraditória que se origina do número abundante de alternativas disponíveis. O comportamento sub-ótimo causado por esse fardo que é chamado de "sobrecarga de escolha" foi observado em experimentos de campo e de laboratório e posteriormente introduzido em alguns modelos econômicos modernos. Esta dissertação fornece um modelo simples para sobrecarga de escolha. Ao dividir o fenômeno em três partes, o fator de novidade, o custo de avaliar as escolhas e o pesar antecipado, examina os elementos que constituem a sobrecarga de escolha. Posteriormente, este trabalho fornece um exemplo do papel da sobrecarga no contexto do Dilema do Prisioneiro.
The fundamental assumption of neoclassical economic theory is the rationality of economic agents. Nonetheless, in time, systematic deviations from this assumption has been observed. A number of such deviations pertains to the decision-making process and one of them is the rather seemingly contradictory burden originating from the abounding number of alternatives available. Suboptimal behavior caused by this burden that is called "choice overload" has been observed in field and laboratory experiments and subsequently introduced into some modern economic models. This dissertation provides a simple model for choice overload. By breaking the phenomenon into three parts, the novelty factor, the cost of evaluation of choices, and the anticipated regret, it examines the elements that constitute choice overload. Subsequently, this work provides an example of the role choice overload may play in the context of Prisoner's Dilemma.
info:eu-repo/semantics/publishedVersion

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Lunova, Mariia [Verfasser]. "Hepcidin knockout mice as a model of iron-overload associated liver disease / Mariia Lunova." Ulm : Universität Ulm. Medizinische Fakultät, 2013. http://d-nb.info/1045278475/34.

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Advani, Tushar M. "Brain derived neurotrophic factor deficient mouse a putative model of allostatic overload : a dissertation /." San Antonio : UTHSC, 2008. http://proquest.umi.com.libproxy.uthscsa.edu/pqdweb?did=1588771861&sid=7&Fmt=2&clientId=70986&RQT=309&VName=PQD.

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Johnson, Luke A. "Locus Coeruleus and Hippocampal Tyrosine Hydroxylase Levels in a Pressure-Overload Model of Heart Disease." Digital Commons @ East Tennessee State University, 2013. https://dc.etsu.edu/honors/288.

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Studies have indicated that approximately 30% of people with heart disease experience major depressive disorder (MDD). Despite strong clinical evidence of a link between the two diseases, the neurobiological processes involved in the relationship are poorly understood. A growing number of studies are revealing similar neuroanatomical and neurochemical abnormalities resulting from both depression and heart disease. The locus coeruleus (LC) is a group of neurons in the pons that synthesize and release norepinephrine, and that is known to play a significant role in depression pathobiology. For example, there is evidence that tyrosine hydroxylase (TH) is elevated in the LC in depression. In addition, there is evidence that the LC plays a role in cardiovascular autonomic regulation. The hippocampus is another region that exhibits abnormalities in both depression and heart disease. In this study, the levels of TH in the hippocampus and LC were examined in the guinea pig pressure-overload model of heart disease. TH levels were also measured in the pressure-overload model treated with vagal nerve stimulation, a new investigational therapeutic intervention in heart disease. This study found that there were no changes in TH levels in the LC or the hippocampus of the pressure-overload model or in the pressure-overload model treated with vagal nerve stimulation.

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Jordaan, Esaias Engelbertus. "Evaluation of isolated dorsal root ganglion cells as a model to study neural calcium overload / E.E. Jordaan." Thesis, North-West University, 2004. http://hdl.handle.net/10394/645.

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Background and motivation: The event of neural Ca2+ overload is known to have several deleterious effects resulting in cell death caused by ischaemia, hypoglycaemia, hypoxia and several neurodegenerative diseases such as Alzheimer's disease, Parkinson's disease and AIDS-related dementia. In vitro models for the investigation of the mechanisms involved in Ca2+ overload include brain slice preparations, neuronal cultures as well as acutely isolated neurons, mostly from the hippocampus and cortical brain areas. Additional models for investigating Ca2+ overload may bring about new knowledge to areas of the phenomenon that are still unresolved. Methodology: In this study, several theoretical Ca2+ overload-related interventions were combined aimed at inducing cell death in acutely isolated rat dorsal root ganglia. To elucidate the mechanism/s involved in the cell death observed following exposure to this intervention, the effects of several alterations to the intervention's composition were assessed. This examination was extended by the addition of several recognized and potential protective compounds to the intervention. Cell death was indicated by the trypan blue exclusion assay and recorded after 18 hours exposure to the interventions by counting live and dead neurons under a light microscope. Results and conclusions: The goal was to evaluate the possible application of dorsal root ganglia as a model for neural Ca2+ overload outside the brain. Since Ca2+w as required for cell death to be induced, it is concluded that the observed cell death was indeed primarily due to Ca2+ overload. Besides extracellular Ca2+, KC1-induced depolarization was also required for cell death to be induced, while the antagonists did not demonstrate significant protection against cell death. Based on the results, the mechanism of Ca2+ overload could not be defined beyond doubt, but the voltage activated Ca2+ channels are likely to be involved.
Thesis (M.Sc. (Physiology))--North-West University, Potchefstroom Campus, 2005.

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Schymura, Yves [Verfasser]. "Effects of Riociguat and Sildenafil in a murine model of chronic right ventricular pressure overload / Yves Schymura." Gießen : Universitätsbibliothek, 2013. http://d-nb.info/1065320558/34.

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Wolfberg, Adrian. "A Theory of Overload and Equivocality Effects on Learning during Knowledge Transfer within Policy Making Dyads." Case Western Reserve University School of Graduate Studies / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=case1393843187.

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Pavlaki, Nikoleta [Verfasser]. "Gene therapy with phosphodiesterases 2A and 4B in a murine model of pressure overload-induced cardiac hypertrophy / Nikoleta Pavlaki." Hamburg : Staats- und Universitätsbibliothek Hamburg Carl von Ossietzky, 2021. http://d-nb.info/1237050472/34.

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Allen, Adrian. "Modulation of contractile function through neuropeptide Y receptors during the development of cardiomyocyte hypertrophy in a model of pressure overload." Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368483.

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Books on the topic "Overload model"

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Marsh, Brian T. Overland Models-- the first 10 years. [Indiana]: B.T. Marsh, 1987.

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Wong, Tommy S. W. Overland flow and surface runoff. Hauppauge, N.Y: Nova Science Publishers, 2011.

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Kavanagh, Leonnie. Benkelman beam rebound: AC overlay model : joint C-SHRP/Manitoba Bayesian application. Ottawa: Canadian Strategic Highway Research Program, Transportation Association of Canada, 1995.

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Ovsyannikov, Evgeniy, and Tamara Gaytova. Optimal control of traction electric drives. ru: INFRA-M Academic Publishing LLC., 2021. http://dx.doi.org/10.12737/1141767.

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The monograph considers various types of traction electric drives of motor vehicles intended for operation in urban conditions. Mathematical models of these systems are proposed. On the basis of parametric optimization and graphoanalytic method, a method of joint control of electric drives according to the criteria of minimum losses and maximum overload capacity, taking into account possible restrictions on the resources of power elements, has been developed. For a wide range of readers interested in improving motor vehicles. It will be useful for students, postgraduates and teachers of engineering and technical universities.

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Oregon. Dept. of Land Conservation and Development., ed. Chronic coastal natural hazards model overlay zone. [Salem, Or.]: Oregon Dept. of Land Conservation and Development, 1998.

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Huda, Ahmed Samei. The Medical Model in Mental Health. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198807254.001.0001.

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The medical model is a biopsychosocial model assessing a patient’s problems and matching them to the diagnostic construct using pattern recognition of clinical features. Diagnostic constructs allow for researching, communicating, teaching, and learning useful clinical information to influence clinical decision-making. They also have social and administrative functions such as access to benefits. They may also help explain why problems occur. Diagnostic constructs are used to describe diseases/syndromes and also other types of conditions such as spectrums of conditions. Treatments in medicine and psychiatry have several treatment objectives including cure or reducing distress and a variety of mechanisms of action apart from reversing disease/cure. Causation of conditions in medicine and psychiatry are often complex. The medical model allows doctors to assess and offer effective treatments to large numbers of patients and provide emergency cover. Diagnostic constructs in psychiatry and general medicine overlap for attributes such as clinical utility (e.g. predicting likely outcomes) and validity (e.g. lack of boundaries between different diagnostic constructs) and importance of social factors. There is an overlap in effectiveness between psychiatric and general medicine treatments and many general medicine medications do not reverse disease processes. Different mental health classifications have particular strengths and weaknesses for clinical, research, and social functions. Mental health research into understanding causes and mechanisms may need other classifications than diagnosis. As doctors in all specialties encounter mental health problems, there will always be psychiatric diagnostic constructs compatible with their training. Mental health research and service provision will always need to address psychosocial issues.

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Edwards, Jane. Approaches and Models of Music Therapy. Edited by Jane Edwards. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199639755.013.38.

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This chapter presents eleven models and approaches in music therapy practice. The inclusion criteria is that each forms the basis, or is part, of an existing training, and at least one monograph exists. The distinction betweenmodelsand approaches is made such that models refer to developments which evolved from music therapy practice, andapproachesare music therapy techniques and methods overlaid on an existing model of therapy or theoretical principles. Music therapy training requires learning about theory within the parameters of at least one model or approach, and this engagement must be thorough and intensive. Trainees can struggle with having to do so much thinking and analyzing of their own reactions and integration of key theoretical concepts while concurrently focusing on improving their techniques and methods. Ultimately good enough training allows students to first comprehend and eventually internalize, the theoretical basis of their professional thinking in practice.

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Wolff, Nancy. A General Model of Harm in Correctional Settings. Edited by John Wooldredge and Paula Smith. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780199948154.013.33.

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The literature on inmate “harm” and inmate victimization within prison settings is reviewed with emphasis on the prevalence, predictors, and consequences associated with inmate misconduct, physical victimization, and sexual victimization in prison. The degree of overlap between “offenders” and “victims” is also discussed. The relevance of considering both inmate and facility characteristics for a more comprehensive understanding of both violent and property victimization is underscored. The potential impact of victimization on inmates’ feelings of safety is also covered. Strategies for preventing victimization and their limitations (e.g., protective custody, administrative segregation, disciplinary custody, prison transfers) are reviewed. A dyadic model of harm is developed that draws on routine activities theory and rational choice theory, to more clearly and systematically predict the effects of harm- and victim-propensity attributes of incarcerated people and correctional facilities on levels of harm.

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Keel, Pamela K., and Lauren A. Holland. Eating Disorders. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.017.

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This chapter examines patterns of comorbidity between eating disorders and mood, anxiety, and substance use disorders along with evidence regarding support for different theoretical models that may account for these patterns. Although comorbidity estimates may be inflated by reliance on treatment-seeking samples and double counting of symptoms that overlap between syndromes, evidence supports elevated risk of mood, anxiety, and substance use disorders in anorexia nervosa, bulimia nervosa, and binge eating disorder. Data from family and twin studies support that eating and anxiety disorders may have a shared diathesis, consistent with the common cause model. Data from longitudinal studies suggest that eating disorders may increase vulnerability for developing a substance use disorder, consistent with the predisposition model. In contrast, comorbidity between eating and mood disorders, such as depression, remains poorly understood. Clinical issues regarding comorbidity of depression and eating disorders along with guidelines for clinicians treating patients with comorbid depression and eating disorders are discussed.

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Ostermann, Marlies, and Ruth Y. Y. Wan. Diuretics in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0058.

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Fluid overload and chronic hypertension are the most common indications for diuretics. The diuretic response varies between different types and depends on underlying renal function. In patients with congestive heart failure, diuretics appear to reduce the risk of death and worsening heart failure compared with placebo, but their use in acute decompensated heart failure is questionable. Diuretics are also widely used in chronic kidney disease to prevent or control fluid overload, and treat hypertension. In acute kidney injury, there is no evidence that they improve renal function, speed up recovery, or change mortality. In patients with chronic liver disease and large volume ascites, paracentesis is more effective and associated with fewer adverse events than diuretic therapy, but maintenance treatment with diuretics is indicated to prevent recurrence of ascites. Mannitol has a role in liver patients with cerebral oedema and normal renal function. The use of diuretics in rhabdomyolysis is controversial and restricted to patients who are not fluid deplete. In conditions associated with resistant oedema (chronic kidney disease, congestive heart failure, chronic liver disease), combinations of diuretics with different modes of action may be necessary. Diuresis is easier to achieve with a continuous furosemide infusion compared with intermittent boluses, but there is no evidence of better outcomes. The role of combination therapy with albumin in patients with fluid overload and severe hypoalbuminaemia is uncertain with conflicting data.

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Book chapters on the topic "Overload model"

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Gahlawat, Monica, and Priyanka Sharma. "Support Vector Machine-Based Model for Host Overload Detection in Clouds." In Advances in Intelligent Systems and Computing, 369–76. Singapore: Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0129-1_39.

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Mishra, Atul. "Study and Proposal of Probabilistic Model for SIP Server Overload Control." In Communications in Computer and Information Science, 347–60. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8660-1_26.

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Abaev, Pavel, and Rostislav Valerievich Razumchik. "Queuing Model for SIP Server Hysteretic Overload Control with Bursty Traffic." In Internet of Things, Smart Spaces, and Next Generation Networking, 383–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-40316-3_34.

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Abaev, Pavel, Yuliya Gaidamaka, and Konstantin E. Samouylov. "Queuing Model for Loss-Based Overload Control in a SIP Server Using a Hysteretic Technique." In Internet of Things, Smart Spaces, and Next Generation Networking, 371–78. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-32686-8_34.

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Hübel, Hartwig. "Overlay Model." In Simplified Theory of Plastic Zones, 211–32. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29875-7_6.

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Hübel, Hartwig. "Overlay-Modell." In Vereinfachte Fließzonentheorie, 213–33. Wiesbaden: Springer Fachmedien Wiesbaden, 2015. http://dx.doi.org/10.1007/978-3-658-07922-2_6.

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Chaanine, Antoine H., and Roger J. Hajjar. "Characterization of the Differential Progression of Left Ventricular Remodeling in a Rat Model of Pressure Overload Induced Heart Failure. Does Clip Size Matter?" In Methods in Molecular Biology, 195–206. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8597-5_15.

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Eades, Susan C. "Experimental Models of Laminitis: Starch Overload." In Equine Laminitis, 54–58. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2016. http://dx.doi.org/10.1002/9781119169239.ch7.

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Pollitt, Christopher C., and Gabriel J. Milinovich. "Experimental Models of Laminitis: Oligofructose Overload." In Equine Laminitis, 59–63. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2016. http://dx.doi.org/10.1002/9781119169239.ch8.

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Hoggard, Patrick E. "Angular Overlap Model Parameters." In Structure and Bonding, 37–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/b11304.

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Conference papers on the topic "Overload model"

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Huang, Xingjian, and Cheng Tang. "Grey Markov Model Railway Overload Predictions." In 2014 International Conference of Logistics Engineering and Management. Reston, VA: American Society of Civil Engineers, 2014. http://dx.doi.org/10.1061/9780784413753.053.

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Qi, Fang, Alexander Stippich, Stefan Koschik, and Rik W. De Doncker. "Model predictive overload control of induction motors." In 2015 IEEE International Electric Machines & Drives Conference (IEMDC). IEEE, 2015. http://dx.doi.org/10.1109/iemdc.2015.7409183.

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Gutkin, Leonid, and Douglas A. Scarth. "Statistical Modeling of Resistance to Crack Initiation due to Hydrided Region Overloads at Simulated Flaws in CANDU Pressure Tubes." In ASME 2015 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/pvp2015-45997.

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CANDU(1) Zr-2.5%Nb pressure tubes are susceptible to formation of hydrided regions at the locations of stress concentration, such as in-service flaws. Hydrided region overloads occur when the applied stress acting on a flaw with an existing hydrided region exceeds the stress at which the hydrided region has been formed. The overload events may potentially result in crack initiation and its subsequent growth by the mechanism of delayed hydride cracking. Therefore, evaluating the in-service flaws in the pressure tubes for crack initiation due to hydrided region overloads is required by the Canadian Nuclear Standards, and methodology is being developed to perform such evaluations. As part of this development, the resistance of pressure tube material to crack initiation due to hydrided region overloads was modeled statistically. In the proposed modeling framework, the overload resistance is expressed as a power-law function of the material resistance to initiation of delayed hydride cracking under constant loading. This approach fundamentally relies on the concept of a dual process zone introduced by E. Smith, as discussed in the paper. Both the overload crack initiation coefficient and the overload crack initiation exponent vary with the flaw geometry. The overload crack initiation coefficient also varies with the extent of stress reduction prior to hydride formation and with the number of non-ratcheting hydride formation thermal cycles. The developed model is suitable for use as a predictive model in probabilistic assessments of CANDU reactor core, and has been proposed for implementation into the scheduled revision (2015) of the Canadian Nuclear Standard CSA N285.8.

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Ransom, Daniel L., and Randy Hamilton. "Extending motor life with updated thermal model overload protection." In 2011 64th Annual Conference for Protective Relay Engineers. IEEE, 2011. http://dx.doi.org/10.1109/cpre.2011.6035624.

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Ransom, Daniel L., and Randy Hamilton. "Extending motor life with updated thermal model overload protection." In 2012 IEEE Petroleum and Chemical Industry Technical Conference (PCIC). IEEE, 2012. http://dx.doi.org/10.1109/pcicon.2012.6549679.

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Ransom, Daniel L., and Randy Hamilton. "Extending motor life with updated thermal model overload protection." In 2011 IEEE Industry Applications Society Annual Meeting. IEEE, 2011. http://dx.doi.org/10.1109/ias.2011.6074475.

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Zhang, Jian-Hua, Luo Fan, Jing-Jing Zheng, and Yue Lin. "Assessment Model and Optimization Control to Transmission Line Overload." In 2015 International Conference on Energy, Environmental & Sustainable Ecosystem Development (EESED 2015). WORLD SCIENTIFIC, 2015. http://dx.doi.org/10.1142/9789814723008_0139.

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Jihong Cheng, Heng Li, and Youan Zhang. "Robust Low-Cost Sliding Mode Overload Control for Uncertain Agile Missile Model." In 2006 6th World Congress on Intelligent Control and Automation. IEEE, 2006. http://dx.doi.org/10.1109/wcica.2006.1712746.

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Mizutaka, Shogo, and Kousuke Yakubo. "Overload Network Failures: An Approach from the Random-Walk Model." In 2013 International Conference on Signal-Image Technology & Internet-Based Systems (SITIS). IEEE, 2013. http://dx.doi.org/10.1109/sitis.2013.103.

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Mustapar, Nur Amirah, Natrah Abdullah, and Nor Laila Md Noor. "A review towards developing a moment of information overload model." In 2016 4th International Conference on User Science and Engineering (i-USEr). IEEE, 2016. http://dx.doi.org/10.1109/iuser.2016.7857964.

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Reports on the topic "Overload model"

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Ritter, Frank E., and Steven R. Haynes. An Architectural Overlay: Modifying an Architecture to Help Cognitive Models Understand and Explain Themselves. Fort Belvoir, VA: Defense Technical Information Center, February 2006. http://dx.doi.org/10.21236/ada443755.

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Phan, Long T., Donald N. Slinn, and Shaun W. Kline. Introduction of wave set-up effects and mass flux to the sea, lake, and overland surges from hurricanes (slosh) model. Gaithersburg, MD: National Institute of Standards and Technology, 2010. http://dx.doi.org/10.6028/nist.ir.7689.

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Swallow, G., J. Drake, H. Ishimatsu, and Y. Rekhter. Generalized Multiprotocol Label Switching (GMPLS) User-Network Interface (UNI): Resource ReserVation Protocol-Traffic Engineering (RSVP-TE) Support for the Overlay Model. RFC Editor, October 2005. http://dx.doi.org/10.17487/rfc4208.

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HEFNER, Robert. IHSAN ETHICS AND POLITICAL REVITALIZATION Appreciating Muqtedar Khan’s Islam and Good Governance. IIIT, October 2020. http://dx.doi.org/10.47816/01.001.20.

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Ours is an age of pervasive political turbulence, and the scale of the challenge requires new thinking on politics as well as public ethics for our world. In Western countries, the specter of Islamophobia, alt-right populism, along with racialized violence has shaken public confidence in long-secure assumptions rooted in democracy, diversity, and citizenship. The tragic denouement of so many of the Arab uprisings together with the ascendance of apocalyptic extremists like Daesh and Boko Haram have caused an even greater sense of alarm in large parts of the Muslim-majority world. It is against this backdrop that M.A. Muqtedar Khan has written a book of breathtaking range and ethical beauty. The author explores the history and sociology of the Muslim world, both classic and contemporary. He does so, however, not merely to chronicle the phases of its development, but to explore just why the message of compassion, mercy, and ethical beauty so prominent in the Quran and Sunna of the Prophet came over time to be displaced by a narrow legalism that emphasized jurisprudence, punishment, and social control. In the modern era, Western Orientalists and Islamists alike have pushed the juridification and interpretive reification of Islamic ethical traditions even further. Each group has asserted that the essence of Islam lies in jurisprudence (fiqh), and both have tended to imagine this legal heritage on the model of Western positive law, according to which law is authorized, codified, and enforced by a leviathan state. “Reification of Shariah and equating of Islam and Shariah has a rather emaciating effect on Islam,” Khan rightly argues. It leads its proponents to overlook “the depth and heights of Islamic faith, mysticism, philosophy or even emotions such as divine love (Muhabba)” (13). As the sociologist of Islamic law, Sami Zubaida, has similarly observed, in all these developments one sees evidence, not of a traditionalist reassertion of Muslim values, but a “triumph of Western models” of religion and state (Zubaida 2003:135). To counteract these impoverishing trends, Khan presents a far-reaching analysis that “seeks to move away from the now failed vision of Islamic states without demanding radical secularization” (2). He does so by positioning himself squarely within the ethical and mystical legacy of the Qur’an and traditions of the Prophet. As the book’s title makes clear, the key to this effort of religious recovery is “the cosmology of Ihsan and the worldview of Al-Tasawwuf, the science of Islamic mysticism” (1-2). For Islamist activists whose models of Islam have more to do with contemporary identity politics than a deep reading of Islamic traditions, Khan’s foregrounding of Ihsan may seem unfamiliar or baffling. But one of the many achievements of this book is the skill with which it plumbs the depth of scripture, classical commentaries, and tasawwuf practices to recover and confirm the ethic that lies at their heart. “The Quran promises that God is with those who do beautiful things,” the author reminds us (Khan 2019:1). The concept of Ihsan appears 191 times in 175 verses in the Quran (110). The concept is given its richest elaboration, Khan explains, in the famous hadith of the Angel Gabriel. This tradition recounts that when Gabriel appeared before the Prophet he asked, “What is Ihsan?” Both Gabriel’s question and the Prophet’s response make clear that Ihsan is an ideal at the center of the Qur’an and Sunna of the Prophet, and that it enjoins “perfection, goodness, to better, to do beautiful things and to do righteous deeds” (3). It is this cosmological ethic that Khan argues must be restored and implemented “to develop a political philosophy … that emphasizes love over law” (2). In its expansive exploration of Islamic ethics and civilization, Khan’s Islam and Good Governance will remind some readers of the late Shahab Ahmed’s remarkable book, What is Islam? The Importance of Being Islamic (Ahmed 2016). Both are works of impressive range and spiritual depth. But whereas Ahmed stood in the humanities wing of Islamic studies, Khan is an intellectual polymath who moves easily across the Islamic sciences, social theory, and comparative politics. He brings the full weight of his effort to conclusion with policy recommendations for how “to combine Sufism with political theory” (6), and to do so in a way that recommends specific “Islamic principles that encourage good governance, and politics in pursuit of goodness” (8).

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Holland, Darren, and Nazmina Mahmoudzadeh. Foodborne Disease Estimates for the United Kingdom in 2018. Food Standards Agency, January 2020. http://dx.doi.org/10.46756/sci.fsa.squ824.

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In February 2020 the FSA published two reports which produced new estimates of foodborne norovirus cases. These were the ‘Norovirus Attribution Study’ (NoVAS study) (O’Brien et al., 2020) and the accompanying internal FSA technical review ‘Technical Report: Review of Quantitative Risk Assessment of foodborne norovirus transmission’ (NoVAS model review), (Food Standards Agency, 2020). The NoVAS study produced a Quantitative Microbiological Risk Assessment model (QMRA) to estimate foodborne norovirus. The NoVAS model review considered the impact of using alternative assumptions and other data sources on these estimates. From these two pieces of work, a revised estimate of foodborne norovirus was produced. The FSA has therefore updated its estimates of annual foodborne disease to include these new results and also to take account of more recent data related to other pathogens. The estimates produced include: •Estimates of GP presentations and hospital admissions for foodbornenorovirus based on the new estimates of cases. The NoVAS study onlyproduced estimates for cases. •Estimates of foodborne cases, GP presentations and hospital admissions for12 other pathogens •Estimates of unattributed cases of foodborne disease •Estimates of total foodborne disease from all pathogens Previous estimates An FSA funded research project ‘The second study of infectious intestinal disease in the community’, published in 2012 and referred to as the IID2 study (Tam et al., 2012), estimated that there were 17 million cases of infectious intestinal disease (IID) in 2009. These include illness caused by all sources, not just food. Of these 17 million cases, around 40% (around 7 million) could be attributed to 13 known pathogens. These pathogens included norovirus. The remaining 60% of cases (equivalent to 10 million cases) were unattributed cases. These are cases where the causal pathogen is unknown. Reasons for this include the causal pathogen was not tested for, the test was not sensitive enough to detect the causal pathogen or the pathogen is unknown to science. A second project ‘Costed extension to the second study of infectious intestinal disease in the community’, published in 2014 and known as IID2 extension (Tam, Larose and O’Brien, 2014), estimated that there were 566,000 cases of foodborne disease per year caused by the same 13 known pathogens. Although a proportion of the unattributed cases would also be due to food, no estimate was provided for this in the IID2 extension. New estimates We estimate that there were 2.4 million cases of foodborne disease in the UK in 2018 (95% credible intervals 1.8 million to 3.1 million), with 222,000 GP presentations (95% Cred. Int. 150,000 to 322,000) and 16,400 hospital admissions (95% Cred. Int. 11,200 to 26,000). Of the estimated 2.4 million cases, 0.9 million (95% Cred. Int. 0.7 million to 1.2 million) were from the 13 known pathogens included in the IID2 extension and 1.4 million1 (95% Cred. Int. 1.0 million to 2.0 million) for unattributed cases. Norovirus was the pathogen with the largest estimate with 383,000 cases a year. However, this estimate is within the 95% credible interval for Campylobacter of 127,000 to 571,000. The pathogen with the next highest number of cases was Clostridium perfringens with 85,000 (95% Cred. Int. 32,000 to 225,000). While the methodology used in the NoVAS study does not lend itself to producing credible intervals for cases of norovirus, this does not mean that there is no uncertainty in these estimates. There were a number of parameters used in the NoVAS study which, while based on the best science currently available, were acknowledged to have uncertain values. Sensitivity analysis undertaken as part of the study showed that changes to the values of these parameters could make big differences to the overall estimates. Campylobacter was estimated to have the most GP presentations with 43,000 (95% Cred. Int. 19,000 to 76,000) followed by norovirus with 17,000 (95% Cred. Int. 11,000 to 26,000) and Clostridium perfringens with 13,000 (95% Cred. Int. 6,000 to 29,000). For hospital admissions Campylobacter was estimated to have 3,500 (95% Cred. Int. 1,400 to 7,600), followed by norovirus 2,200 (95% Cred. Int. 1,500 to 3,100) and Salmonella with 2,100 admissions (95% Cred. Int. 400 to 9,900). As many of these credible intervals overlap, any ranking needs to be undertaken with caution. While the estimates provided in this report are for 2018 the methodology described can be applied to future years.

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