Relevant bibliographies by topics / Ovary

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Ovary'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 July 2024

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Journal articles on the topic "Ovary"

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Corbett, Andrea L., Pam G. Krannitz, and Lonnie W. Aarssen. "The influence of petals on reproductive success in the arctic poppy (Papaver radicatum)." Canadian Journal of Botany 70, no. 1 (January 1, 1992): 200–204. http://dx.doi.org/10.1139/b92-027.

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The flowers of the arctic poppy (Papaver radicatum L.) track the sun, and the petals of the flowers reflect light towards the ovary. This study investigated the importance of petals to reproductive success in this species. We measured light intensity, ovary temperature, seed weight, and number of seeds produced by flowers that had their petals removed versus flowers with their petals intact. Flowers without petals do not track the sun. Irradiance was greater when the photocell was held coincident with the angle of sun-tracking flowers than when facing upward. Flowers with petals had significantly higher overy temperatures both at the ovary surface and in the ovary interior than did flowers lacking petals. In pairs of flowers on the same individual plant, those with their petals removed had significantly fewer and smaller seeds than control flowers with petals intact. There were site differences in the significance of the treatment effect, but not in the magnitude of the differences between flowers with and without petals. Therefore, the presence of petals resulted in increased irradiance and temperature at and in the ovary, which affected reproductive success. Key words: heliotropism, Papaver radicatum, petals, reproductive success, temperature.
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Murmu, Dr Dasharatha, Dr Subrat Kumar Soren, Dr Kanumury Vandana, and Dr Ananta Satyanarayana M. "Fibroma of the Ovary." International Journal of Scientific Research 3, no. 4 (June 1, 2012): 351–52. http://dx.doi.org/10.15373/22778179/apr2014/124.

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Occhipinti, Kathryn A., Steven D. Frankel, and Hedvig Hricak. "THE OVARY." Radiologic Clinics of North America 31, no. 5 (September 1993): 1115–32. http://dx.doi.org/10.1016/s0033-8389(22)00359-1.

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Tripathi, Reva. "The ovary." Indian Journal of Medical Research 150, no. 3 (2019): 313. http://dx.doi.org/10.4103/ijmr.ijmr_1052_19.

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Schultze, Henrik, and Claus Fenger. "Accessory Ovary." Acta Obstetricia et Gynecologica Scandinavica 65, no. 5 (January 1986): 503–4. http://dx.doi.org/10.3109/00016348609157394.

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Hall, Janet. "The Ovary." Endocrinologist 4, no. 5 (September 1994): 400. http://dx.doi.org/10.1097/00019616-199409000-00011.

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Oktem, Ozgur, and Kutluk Oktay. "The Ovary." Annals of the New York Academy of Sciences 1127, no. 1 (April 2008): 1–9. http://dx.doi.org/10.1196/annals.1434.009.

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&NA;. "The Ovary." International Journal of Gynecological Pathology 14, no. 1 (January 1995): 94. http://dx.doi.org/10.1097/00004347-199501000-00018.

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Cheung, Vincent Y. T. "Accessory Ovary." Journal of Obstetrics and Gynaecology Canada 32, no. 2 (February 2010): 107. http://dx.doi.org/10.1016/s1701-2163(16)34420-6.

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Mahmoud, M. S. "Maldescended Ovary." Journal of Minimally Invasive Gynecology 20, no. 6 (November 2013): S100. http://dx.doi.org/10.1016/j.jmig.2013.08.331.

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Dissertations / Theses on the topic "Ovary"

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Tyndall, Victoria. "Androgens and the ovary." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5591.

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Between 10-15% of women suffer from polycystic ovary syndrome (PCOS), making it the most common cause of female infertility. Clinical features of PCOS include high circulating levels of ovarian androgens (T and A4), anovulation and obesity. The aetiology of this reproductive endocrinopathy is likely to be multifactorial, through the interplay of genetics, epigenetics and environmental factors. Primate research into sexual behaviour development noted that fetally androgenised monkeys developed symptoms like those of PCOS. There are now multiple animal models of PCOS using primates, sheep, rats and transgenic mice. The investigations described in this thesis use rodent models to examine the role of androgens in the pathogenesis of female infertility. An attempt to generate a granulosa cell specific androgen receptor knockout mouse model will first be described, followed by several studies into the developmental programming of female Wistar rat infertility and metabolism by steroid hormones. Initial investigations showed that testosterone proprionate (TP) administered to female rats during different windows of fetal and neonatal life alters the reproductive and metabolic axes of the adult animals. Fetal plus neonatal TP exposure led to complete ovarian dysgenesis, while postnatal exposure produced a PCOS-like phenotype. Animals which received TP postnatally were heavier and had an increased proportion of primordial follicles in their ovaries by postnatal day (pnd) 90 of life. Evaluation of this PCOS model showed that neonatally androgenised rats had ovarian follicles with larger antra and a greater ovarian stromal compartment. In addition, these animals were heavier when compared to controls. However, unlike human studies, neonatally androgenised rats showed no differences in circulating gonadotrophin or ovarian androgen levels. Nor did they show any programming effect of neonatal TP upon the theca interna by pnd 90. Further investigations to narrow the windows and dose of TP required to produce a PCOS phenotype showed that TP administered in an early window of neonatal life, between postnatal days (pnd) 1-6 not only led to anovulation, but potentially reprogrammed the hypothalamic-pituitary axis, as there was minimal gonadotrophin response to reduced ovarian negative feedback (inhibin B and estradiol) in these rats. Neonatal TP also affected the rat metabolic axis with adult animals becoming heavier after weaning without any change in food intake. Animals developed mesenteric and retroperitoneal obesity along with insulin resistance (IR). Increased hepatic glucocorticoid turnover and altered adipokine expression were also noted in neonatally androgenised females, possibly contributing to the pathogenesis of obesity. No effect of TP dose upon the severity of infertility or metabolic abnormalities in adult animals was observed. To delineate which features of the rat PCOS model resulted from androgenic, estrogenic or corticosteroid action, a final study used administration of different steroids during the early window of postnatal life: TP, estradiol valerate (EV), dihydrotestosterone (DHT), dehydroepiandrosterone (DHEA) and dexamethasone (DEX). The anovulatory PCO-like phenotype observed with TP was also seen in animals which received EV, but not those which received DHT, DHEA or DEX. TP and EV treatment also resulted in a reduction of ovarian follicle numbers and activated follicle proportions, with an increase in primordial follicle proportions. Although glucose tolerant, animals treated with TP and EV were highly IR. Unlike dexamethasone, DHT and DHEA also produced IR in adult animals, to a lesser extent than TP and EV. Taken collectively, the results described in this thesis demonstrate that the PCOS-like phenotype observed in the neonatally androgenised female rat is likely to be due to the estrogenic actions of testosterone, potentially through as yet unknown epigenetic mechanisms. The programming of the metabolic components described may additionally be due to the actions of androgens. Furthermore, these studies demonstrate a novel estrogenic effect of neonatal steroids upon primordial follicle populations and show that the neonatally androgenised rat may be a rational PCOS model in a poly-ovulatory species.
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Patterson, Moneka Angilene. "Polycystic Ovary Syndrome Treatment." ScholarWorks, 2017. https://scholarworks.waldenu.edu/dissertations/4319.

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Polycystic ovary syndrome (PCOS) is an endocrine system disorder that affects women of reproductive age. If not treated properly, PCOS can lead to infertility. Lack of proper treatment of PCOS may also result in medical complications such as diabetes or heart disease. The rural clinic where this project took place did not have a mandatory guideline for treatment of PCOS; therefore, no standardized method of diagnosis or treatment of PCOS existed. The purpose of this project, guided by the IOWA evidence-based practice model, was to educate providers on the evidence-based guideline for diagnosis and treatment of PCOS outlined by the Endocrine Society Taskforce. The guideline was selected after a comprehensive literature review and was used to develop an educational program that was provided to 5 nurse practitioners, the medical director and staff. A pre-test post-test design was used to determine if the participants understood the content from the guideline that was presented. Results showed that the researcher-developed test administered to participants yielded scores of 74 on the pre-test and increased after the education program with all participants scoring 100 on the post-test. The guideline used for the education was then presented to the clinic for implementation with the assistance of the medical director's support. The project provided an evidence-based guideline for diagnosing and treating PCOS and raised awareness of PCOS among all staff in a rural clinic where many patients with PCOS are treated. Positive social change may result as providers are better prepared to deliver evidence-based care for PCOS and as infertility and complications of untreated PCOS are reduced.
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Dilaver, Nafi Mehmet. "The role of anti-Mullerian hormone in the ovary : relevance to polycystic ovary syndrome." Thesis, St George's, University of London, 2017. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.719152.

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Anti-Mullerian hormone (AMH) is secreted by granulosa cells (GC) in the ovary and is produced in much higher amounts by GCs in women with polycystic ovary syndrome (PCOS). The aim of this project was to examine the role of AMH in the normal ovary and thereby understand the consequences for follicle development of greater concentrations found in women with PCOS. Human granulosa-luteal cells (GLCs), human theca tissue and KGN cells were used to investigate the effect of AMH on GC growth and apoptosis. Furthermore, the relationship between AMH and the essential gonadotrophins of folliculogenesis, i.e. FSH and LH were investigated. AMH signalling was also investigated in GLCs from women with and without PCOS. In addition, the effect of AMH on whole genome expression in GLCs from women with normal ovaries and PCOS was determined. AMH inhibited GC proliferation but did not appear to do this via apoptosis. AMH also had a differential effect on SMAD proteins in normal and PCOS cells. Interestingly, AMH increased inhibitory SMADs 6 & 7 and decreased pSMAD 1/5/8 in PCOS cells compared to basal levels. Finally, AMH had a significant differential effect on the transcriptome profile of cells from women with normal ovaries and PCOS. In summary, a differential effect of AMH in cells from women with normal ovaries and PCOS has been demonstrated, with evidence to speculate that the inhibitory role of AMH in the ovary may be due to an effect on the cell cycle.
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Wang, Ling Jia. "Cytokines and the human ovary." Title page, contents and abstract only, 1992. http://web4.library.adelaide.edu.au/theses/09PH/09phw2458.pdf.

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Bibliography: leaves 151-179. Examines aspects of the distribution of leukocyte subpopulation in human corpus luteum, cytokine determination in human preovulatory follicular fluid, as well as the effects of cytokines on human granulosalutein cells; with the aim of investigating one of the ovarian regulatory systems, which may be controlled by immune cell-derived cytokines.
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Mannan, Md Abdul. "Steroidogenesis in the developing ovary." Thesis, Royal Veterinary College (University of London), 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522681.

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Tomaszczuk-Kossowska, Katarzyna. "Stem cells in the ovary /." [S.l.] : [s.n.], 2009. http://edoc.unibas.ch/diss/DissB_8727.

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Morin-Papunen, L. (Laure). "Insulin resistance in polycystic ovary syndrome." Doctoral thesis, University of Oulu, 2000. http://urn.fi/urn:isbn:9514257405.

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Abstract The polycystic ovary syndrome, described first as the association of bilateral polycystic ovaries and amenorrhoea, oligomenorrhoea, hirsutism and obesity, was later shown to be a complex metabolic syndrome. The first purpose of this study was to investigate the occurrence of hyperinsulinaemia and the severity of insulin resistance and glucose tolerance disorders in polycystic ovary syndrome by means of the oral glucose tolerance test and the euglycaemic hyperinsulinaemic clamp. The next goal was to investigate whether women with polycystic ovary syndrome would benefit from insulin-sensitising drugs, and in particular to compare the effects of metformin and a contraceptive pill containing ethinyl oestradiol and cyproterone acetate. Altogether, 81 women with polycystic ovary syndrome and 34 healthy control subjects were involved in the study. Marked impairment of insulin sensitivity in obese subjects with polycystic ovary syndrome, including a decrease of both cellular oxidative and non-oxidative utilisation of glucose, and a slight non-significant decrease of insulin sensitivity in non-obese subjects was observed. Both non-obese and obese subjects with polycystic ovary syndrome exhibited increased abdominal obesity compared with the controls, confirming the fact that obesity, in particular abdominal obesity, is an important contributor in the development of insulin resistance in this syndrome. Metformin alleviated hyperandrogenism by essentially decreasing ovarian, but not adrenal androgen secretion. The improvement of hyperandrogenism and ovarian function seemed to be mediated by the improvement of hyperinsulinaemia, which resulted itself from subtle improvements in both hepatic insulin extraction and insulin sensitivity. Metformin decreased abdominal obesity and the release of free fatty acids from adipose tissue, and improved ovarian cyclicity and fertility. The transient decrease in serum leptin levels observed may have some role in the improvement of ovarian function. The contraceptive pill significantly improved hyperandrogenism and hirsutism, and it slightly affected glucose metabolism. Thus, it could be the treatment of choice in women with hirsutism problems and no fertility hopes. Metformin could be the drug of choice for women with polycystic ovary syndrome who wish to conceive. Because of its beneficial metabolic effects, the value of metformin in reducing the risk of cardiovascular diseases in polycystic ovary syndrome needs to be further studied.
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Singh, Jaswant. "Bovine ovary, morphologic and biochemical kinetics." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq24047.pdf.

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Cho, Li Wei. "Cardiometabolic aspects of polycystic ovary syndrome." Thesis, University of Hull, 2008. http://hydra.hull.ac.uk/resources/hull:5826.

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Polycystic ovary syndrome (PCOS) is a common endocrine disorder affecting 6-7% of the population. It is characterised by chronic anovulation and hyperandrogenism with the clinical manifestation of oligomenorrhoea, hirsutism and acne. A ten to twenty fold increased risk for type 2 diabetes in PCOS patients compared to weight matched female control subjects makes the syndrome of high socioeconomic importance. The use of differing diagnostic criteria makes the comparison of studies on PCOS difficult until harmonisation through the Rotterdam consensus in 2004. Despite being removed from the diagnostic criteria by the the Rotterdam consensus, the LH/FSH ratio is still widely used as one of the diagnostic criteria for PCOS. Therefore to determine the usefulness of the LH/FSH ratio in the diagnosis of PCOS, I have conducted a study as described in chapter two and showed that an elevated LH to FSH ratio was as commonly found in normal women as those with PCO, and therefore of no diagnostic value. In January 2004, the European Society for Human Reproduction and Embryology (ESHRE) and the American Society for Reproductive Medicine (ASRM) co-sponsored the Rotterdam polycystic ovary syndrome consensus workshop that published diagnostic guidelines, building on the consensus statement of the National Institutes of Health 1990. The Rotterdam criteria for the diagnosis of PCOS states 2 of the 3 features needs to be present to make the diagnosis, and with the exclusion of other aetiologies (congenital adrenal hyperplasia, androgen-secreting tumors, Gushing's syndrome). These features include (l)Oligo- or anovulation (2)Clinical and/or biochemical signs of hyperandrogenism and (3)Polycystic ovaries (either 12 or more follicles measuring 2-9 mm in diameter, or an ovarian volume of > 10 cm³ ). Following the introduction of this guideline, the diagnosis of PCOS in patients recruited for studies on PCOS have been standardised. However, the current biochemical test for hyperandrogenism in women is still not ideal, due to the variation in the assay at low values. All three of total testosterone, bioavailable testosterone and free androgen index (FAI) are currently used as markers of hyperandrogenism for the diagnosis of PCOS. In chapter three, I evaluated the variability of each of the three markers as well as their use in the diagnosis and monitoring patients with PCOS and found that FAI is a better diagnostic marker for hyperandrogenism in patients with PCOS, but once the diagnosis is made, all three methods are equally good in monitoring disease progression. It has now been recognised that the diagnosis of metabolic syndrome identifies patients at increased risk of developing cardiovascular disease, and attempts have been made to develop the most convenient and useful criteria for the diagnosis of this condition in clinical practice. With the pathogenesis of metabolic syndrome not well understood, central obesity and insulin resistance are acknowledged as important causative factors. Cardiovascular disease studies in PCOS had so far been inconclusive with some suggesting increased cardiac events among women with PCOS whilst other studies suggesting no increase compared with normal cycling women. This may be attributed to small sample size in studies and variation in characteristics of patients recruited as well as surrogate markers used. C-reactive protein (CRP) had been widely used as a marker of inflammation, endothelial dysfunction and risk of cardiovascular disease in general and in patients with PCOS. However, there had not been any studies on the biovailability of this marker as the potential utility of CRP as a marker of cardiovascular risk may be limited by the magnitude of this variability in both health and disease, since there can be substantial overlap between PCOS and control individuals. In chapter four, the biological variation of high sensitivity CRP in women with PCOS were compared to normal menstruating women. I found that while the mean concentration of CRP was higher in individuals with PCOS compared to healthy controls, the intraindividual variation of CRP was similarly large in both groups. Therefore, the potential utility of CRP as a marker of cardiovascular risk may be limited by the magnitude of this variability in both health and disease, since there can be substantial overlap between PCOS and control individuals. PCOS is associated with a high risk of progression to type 2 diabetes (T2DM) and impaired glucose tolerance. A history of T2DM in a first-degree relative appears to define a subset of PCOS subjects with a greater prevalence of insulin secretory defects. However, factors underlying the progression of PCOS to T2DM are unclear and may be due to either an increase in the underlying insulin resistance or the progression of beta cell failure. Chapter five described a comparison study between the insulin resistance and beta cell functions in patients with PCOS to that of diet controlled T2DM. I found that the progression from PCOS to the development of T2DM is unlikely to be due to a further increase in insulin resistance (or variability), but rather the progressive failure of pancreatic beta cells with a decrease in insulin production. Having established the effectiveness in the diagnosis of PCOS and its progression, I went on to establish the effectiveness of individual treatments of PCOS. The treatment of patients with PCOS requires that the specific goal(s) of the therapy be first established. Individual goals may include fertility, treatment for hirsutism and/or acne, achieving a regular menstrual cycle, weight reduction and the prevention of the long term consequences associated with PCOS (type 2 diabetes, dyslipidaemia and possibly cardiovascular disease) - or all of the above. Treatments aimed at modifying the long-term consequences on cardiometabolic aspect of PCOS include weight reduction strategies as well as the use of insulin sensitizers. There is currently insufficient data to suggest the superiority of one treatment over another or the use of these medications for treatment of cardiometabolic risks in patients with PCOS. Endothelial dysfunction had been recognised as an early marker for cardiovascular disease and chapter six compared the changes in endothelial function in women treated with either metformin or pioglitazone. I found that pioglitazone significantly improved endothelial function and hs-CRP whereas metformin did not produce significant improvements. Chapter seven evaluates the effects of orlistat, metformin and pioglitazone on metabolic profile and biological variability of IR in women with PCOS. The results showed that only orlistat reduced both IR and its variability significantly, though all three drugs were effective in reducing hyperandrogenism within the 12 week period of the study. These effects with orlistat were coupled with a significantly reduction in total cholesterol through a reduction in LDL. After conducting the above studies and searching through literatures, I found that studies in cardiometabolic risks in women with PCOS had so far shown conflicting results, and this may be due to the heterogeneity within the group. Chapter eight evaluates this heterogeneity particularly between anovulatory and ovulatory women with PCOS, where all subjects met the Rotterdam criteria. Women with anovulatory PCOS were found to have higher mean and biological variability of IR compared to those having an ovulatory cycle, and both were higher than women without PCOS. This suggests that the subset of patients who ovulate may be better protected against future cardiovascular consequences. In conclusion, this thesis demonstrated that LH/FSH ratio is of no use in the diagnosis of PCOS and that free androgen index appeared to be the best diagnostic marker when compared to total testosterone and bioavailable testosterone.
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Yasmin, Ephia. "Metabolic aspects of polycystic ovary syndrome." Thesis, University of Leeds, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.545722.

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Books on the topic "Ovary"

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Y, Adashi E., and Leung P. C. K, eds. The Ovary. New York: Raven Press, 1993.

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K, Leung P. C., and Adashi E. Y, eds. The ovary. 2nd ed. Amsterdam: Elsevier, 2004.

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Pal, Lubna, and David B. Seifer, eds. Polycystic Ovary Syndrome. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92589-5.

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Stouffer, Richard L., ed. The Primate Ovary. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4615-9513-7.

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Pal, Lubna, ed. Polycystic Ovary Syndrome. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8394-6.

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Dunaif, Andrea, R. Jeffrey Chang, Stephen Franks, and Richard S. Legro, eds. Polycystic Ovary Syndrome. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-108-6.

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Chang, R. Jeffrey, ed. Polycystic Ovary Syndrome. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4613-8483-0.

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Büning, Jürgen. The Insect Ovary. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-0741-9.

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HOMBURG, ROY, and MB BS. Polycystic Ovary Syndrome. Abingdon, UK: Taylor & Francis, 1988. http://dx.doi.org/10.4324/9780203426708.

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FRACOG, Kovacs Gabor MRCOG, and Norman Robert, eds. Polycystic ovary syndrome. 2nd ed. Cambridge: Cambridge University Press, 2007.

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Book chapters on the topic "Ovary"

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Heppner, John B., John B. Heppner, Minos E. Tzanakakis, Minos E. Tzanakakis, Minos E. Tzanakakis, Pauline O. Lawrence, John L. Capinera, et al. "Ovary." In Encyclopedia of Entomology, 2704. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_1909.

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Houghton, Oisin, and W. Glenn McCluggage. "Ovary." In Histopathology Specimens, 225–34. London: Springer London, 2012. http://dx.doi.org/10.1007/978-0-85729-673-3_22.

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Fahmy, Mohamed A. Baky. "Ovary." In Rare Congenital Genitourinary Anomalies, 209–27. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-43680-6_14.

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Houghton, Oisin P., and W. Glenn McCluggage. "Ovary." In Histopathology Specimens, 243–53. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57360-1_22.

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Lierse, Werner. "Ovary." In Applied Anatomy of the Pelvis, 272–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71368-2_19.

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Canepa, Carlo, and Lauren Ferrara. "Ovary." In Atlas of Handheld Ultrasound, 163–64. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73855-0_31.

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Laramore, George E., Jay J. Liao, Jason K. Rockhill, Filip T. Troicki, Jaganmohan Poli, Christin A. Knowlton, Michelle Kolton Mackay, et al. "Ovary." In Encyclopedia of Radiation Oncology, 577–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_135.

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Ruchelli, Eduardo D., Eduardo D. Ruchelli, Dale S. Huff, and Dale S. Huff. "Ovary." In Color Atlas of Fetal and Neonatal Histology, 173–80. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0019-6_14.

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Ruchelli, Eduardo D., and Dale S. Huff. "Ovary." In Color Atlas of Human Fetal and Neonatal Histology, 175–81. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11425-1_15.

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Prichard, Jeffrey, and Hanna G. Kaspar. "Ovary." In Handbook of Practical Immunohistochemistry, 371–95. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1578-1_20.

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Conference papers on the topic "Ovary"

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Mishra, Amita, Archit Pandit, Namit Kalra, and Bhawna Narula. "Primary signet ring cell mucinous carcinoma ovary: A very rare neoplasm." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685405.

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Introduction: Mucinous ovarian carcinomas are less common than serous and endometriod type, and are more frequently confined to the ovary at the time of diagnosis. But primary signet ring cell mucinous carcinomas of the ovary are extremely rare. Case Presentation: A 40 yr old patient presented with extremely rare primary signet cell mucinous carcinoma of ovary. She presented with abdominal distension and frequency of urination for one month. She was evaluated and CECT whole abdomen was s/o large left ovarian mass. All the tumor markers were with in normal range. Laparotomy frozen section of left adnexal mass was done and was reported as malignant with sheets of signet ring cells seen. Hence complete staging laparotomy including TAH with RSO with bilateral pelvic lymph node dissection with total omentectomy with para aortic lymph node dissection. Final histopathology with IHC markers were S/O primary signet ring cell carcinoma of ovary with no extracapsular invasion, no lymph nodal involvement & no metastatic spread. Conclusion: We present a very rare case of primary signet ring cell of ovary, confined to ovary itself. On literature review only 14 cases have been reported and of them very few are malignant.
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Malhotra, Vani, Smiti Nanda, Meenakshi Chauhan, and Vandana Bhuria. "Synchronous malignancy of ovary and cervix." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685373.

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Background: Synchronous primary malignancies of the female genital tract constitute 1.7% of all genital malignancies. Case: A 45-year-old para 5 woman presented with loss of appetite and abdominal distention. Provisional diagnosis of ovarian malignancy was made. Final histopathology of the specimen revealed ovarian papillary serous cystadenocacinoma with cervical leiomyosarcoma. She received chemotherapy. Results: Patient is on regular follow-up. Conclusion: The coexistence of primary neoplasms in the ovary and cervix are rare. A normal appearing organ may have a hidden malignancy. So, every surgical specimen should be subjected to detailed histopathological examination. Also, the possibility of synchronous malignancy elsewhere in body should be kept in mind while working on a genital malignancy.
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Utzinger, Urs, Nathaniel D. Kirkpatrick, Rebekah A. Drezek, and Molly A. Brewer. "Endogenous fluorescence emission of the ovary." In Biomedical Optics 2005, edited by Alexander V. Priezzhev and Gerard L. Cote. SPIE, 2005. http://dx.doi.org/10.1117/12.591378.

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Iqbal, S. A., H. Shukla, V. Jain, S. Giri, R. Sekhon, and S. Rawal. "Synchronous primary ovarian sex cord tumor and endometrial cancer." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685384.

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Synchronous primary tumors of female genital tract are rare with a rate of about 0.7-1.8% of all gynaecological tumours. Most common primary tumours presenting as synchronous lesions are ovary and endometrium. However, sex cord stromal tumors are rare variety of primary ovarian tumor and synchronous with endometrium is even much rarer. These tumors are detected usually in younger, overweight, nulliparous and perimenopausal female. Synchronous primary tumors of endometrium and ovary have a better prognosis than the either of above alone because these are usually low grade and diagnosed at early stage. We present a report of four cases of synchronous endometrial and sex cord stromal tumors of ovary.
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Kumar, H. Prasanna, and S. Srinivasan. "Segmentation of polycystic ovary in ultrasound images." In 2014 2nd International Conference on Current Trends in Engineering and Technology (ICCTET). IEEE, 2014. http://dx.doi.org/10.1109/icctet.2014.6966294.

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Metzger, N. K., P. R. T. Jess, L. Paterson, E. M. Wright, and K. Dholakia. "Optical binding of Chinese hamster ovary cells." In Optics & Photonics 2005, edited by Kishan Dholakia and Gabriel C. Spalding. SPIE, 2005. http://dx.doi.org/10.1117/12.618745.

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Gupta, Kajal, and Rajesh Prasad. "Polycystic Ovary Syndrome Detection using Deep Learning." In 2023 6th International Conference on Contemporary Computing and Informatics (IC3I). IEEE, 2023. http://dx.doi.org/10.1109/ic3i59117.2023.10397615.

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Vasavi, Reddy Richa, Seguri Priscilla Prathibha, Himabindu Valiveti, Srinidhi Maringanti, and Akhila Parsa. "Polycystic Ovary Syndrome Monitoring using Machine Learning." In 2023 International Conference on Intelligent Data Communication Technologies and Internet of Things (IDCIoT). IEEE, 2023. http://dx.doi.org/10.1109/idciot56793.2023.10052781.

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Das, Sujata. "Poster Abstract." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685400.

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Haemangiomas of the ovary are very rare neoplasms with a wide age range and present with pain lower abdomen and adenexal mass. Many a times this is an incidental finding on surgery. These neoplasms should be considered in the differential diagnosis of haemorragic ovarian lesion. A 48 yr old female presented to us with pain lower abdomen and adenexal mass. Her routine investigations were normal. Her tumour markers were S. LDH 213, CEA 1.72, CA 125 was 2.3. Ultrasound findings showed a well defined echogenic mass in left ovary measuring 6 x 3.4 cm with no ascitis. Her cervical cytological findings were with in normal limits. Staging laprotomy was done and a bilobed solid ovarian mass was identified on left side. TAH with BSO was done and specimen saved for histopathology that finally showed cavernous haemangioma of ovary. Post op recovery was uneventful with subsequent relief of pain.
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Bentabet, Chaimae, and Majda Idrissi Bendahhou. "170 Rare tumours of the ovary: a series of 65 cases of malignant non-epithelial tumours of the ovary." In ESGO 2024 Congress Abstracts. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/ijgc-2024-esgo.583.

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Reports on the topic "Ovary"

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Glintborg, Dorte, Naja Due Kolster, Pernille Ravn, and Marianne Skovsager Andersen. Prospective risk of type 2 diabetes in normal weight women with polycystic ovary syndrome. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0070.

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Review question / Objective: To investigate the risk for type 2 diabetes (T2D) in normal weight women with PCOS. The following PECOs (Population, Exposure, Comparator and Outcome) were included: Population: Pre- and postmenopausal women. Exposure: PCOS Comparator: Healthy control or background population. Outcome: T2D. Condition being studied: Polycystic ovary syndrome (PCOS) is a common endocrine disorder of reproductive-aged women with a prevalence of 15–20%. Polycystic ovary syndrome (PCOS) is most often defined according to the Rotterdam criteria, which include irregular ovulation, biochemical/clinical hyperandrogenism, and/or polycystic ovaries when other causes are excluded.
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Yang, Cai-Xia, Elane C. Wright, and Jason W. Ross. DND1 Expression and Function in the Porcine Ovary, Oocyte and Embryo. Ames (Iowa): Iowa State University, January 2012. http://dx.doi.org/10.31274/ans_air-180814-1372.

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Wang, Shuai, and Xiaoxiao Lin. The effects of intermittent fasting for Polycystic ovary syndrome : a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2023. http://dx.doi.org/10.37766/inplasy2023.10.0020.

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Yang, Yijiao, Honglin Liu, Yue Xia, Xia Peng, and Xiaorong Ni. Tanshinone for polycystic ovary syndrome: a protocol of systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2020. http://dx.doi.org/10.37766/inplasy2020.10.0017.

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Li, Li, Jianxiu Yu, and Zhongwei Zhou. Mean platelet volume and polycystic ovary syndrome: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0021.

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Wang, Yuanyuan, Zhaoning Duan, Yunpeng Wang, Xiwen He, and Zhi Cheng. Association between the CYP17A1 − 34T/C polymorphism and polycystic ovary syndrome: A meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0081.

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Holahan, Patricia K., and Martin L. Meltz. Survival of Chinese Hamster Ovary Cells Following Ultrahigh Dose Rate Electron and Bremsstrahlung Radiation. Fort Belvoir, VA: Defense Technical Information Center, April 1990. http://dx.doi.org/10.21236/ada222722.

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Shi, Yue, Liqun Wu, Zehuan Liao, and Ningning Zhang. The Comparision of Impact of Chinese Medicine and Diane-35 on Sex Hormone Level in Adolescent with Polycystic Ovary Syndrome. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0031.

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Review question / Objective: The Comparision of Impact of Chinese Medicine and Diane-35 on Sex Hormone Level in Adolescent with Polycystic Ovary Syndrome. Condition being studied: Adolescent patients who met the diagnostic criteria of PCOS. Information sources: English databases (PubMed, Embase, Web of Science, and the Cochrane Library) and Chinese databases (China National Knowledge Infrastructure(CNKI), Wanfang, the China Science and Technology Journal Database (VIP), and the Chinese Biomedical Literature Database (CBM)).
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Pan, Jing, Jianwei Zhang, Shan Xiang, Shangqian Dong, and Xiuyun Qin. A comparison of the efficacy and safety of complementary and alternative therapies for Polycystic Ovary Syndrome. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2021. http://dx.doi.org/10.37766/inplasy2021.1.0077.

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Huang, Gang, and Bojun Zhou. Intervention effects of four rehabilitation exercises on polycystic ovary syndrome: a Bayesian-based reticulated Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0058.

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