Academic literature on the topic 'Osteoporosis'
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Journal articles on the topic "Osteoporosis"
Baccichetti, A., P. L. Nguyen-Thi, A. Blum, D. Mainard, F. Sirveaux, L. Nace, A. Valance, et al. "SAT0459 EVALUATION OF THE PREVALENCE AND THE MANAGEMENT OF OSTEOPOROTIC FRACTURES IN PATIENTS HOSPITALIZED AT NANCY UNIVERSITY HOSPITAL (FRANCE) IN 2017." Annals of the Rheumatic Diseases 79, Suppl 1 (June 2020): 1187.1–1187. http://dx.doi.org/10.1136/annrheumdis-2020-eular.3366.
Full textPinho, Mara Suzana. "Osteoporos e Osteoporosis." Revista Brasileira de Reumatologia 43, no. 3 (June 2003): 185–88. http://dx.doi.org/10.1590/s0482-50042003000300011.
Full textOh, Ho-Seok, Sung-Kyu Kim, and Hyoung-Yeon Seo. "Characteristics of Osteoporosis & Osteoporotic Fractures in Korea Based on Health Insurance Review and Assessment (HIRA) Database: 2009–2017." Healthcare 9, no. 3 (March 14, 2021): 324. http://dx.doi.org/10.3390/healthcare9030324.
Full textKim, Gyu Lee, Yu Hyeon Yi, Hye Rim Hwang, Jinmi Kim, Youngmin Park, Yun Jin Kim, Jeong Gyu Lee, et al. "The Risk of Osteoporosis and Osteoporotic Fracture Following the Use of Irritable Bowel Syndrome Medical Treatment: An Analysis Using the OMOP CDM Database." Journal of Clinical Medicine 10, no. 9 (May 10, 2021): 2044. http://dx.doi.org/10.3390/jcm10092044.
Full textFathala, Ahmed L., Sami Alkulaybi, Abdulrahman Khawaji, Abdelghafour Alomari, and Ahmed Almuhaideb. "The association between low bone mineral density and coronary artery calcification in osteoporotic and non-osteoporotic patients in a tertiary center in Saudi Arabia." Annals of Saudi Medicine 41, no. 2 (April 2021): 101–8. http://dx.doi.org/10.5144/0256-4947.2021.101.
Full textMaasalu, Katre, Ott Laius, Lidiia Zhytnik, Sulev Kõks, Ele Prans, Ene Reimann, and Aare Märtson. "Featured Article: Transcriptional landscape analysis identifies differently expressed genes involved in follicle-stimulating hormone induced postmenopausal osteoporosis." Experimental Biology and Medicine 242, no. 2 (November 20, 2016): 203–13. http://dx.doi.org/10.1177/1535370216679899.
Full textLaffaire, M., M. Caroline, E. Allado, E. Bauer, I. Chary Valckenaere, and D. Loeuille. "AB0948 Osteoporotic screening and prevalence of severe osteoporotic fractures in a population of psoriatic arthritis initiating a biologic treatment." Annals of the Rheumatic Diseases 81, Suppl 1 (May 23, 2022): 1602.2–1603. http://dx.doi.org/10.1136/annrheumdis-2022-eular.3871.
Full textMarchenkova, L. "POS1469-HPR THE ASSESSMENT OF FRACTURE RISK AND OSTEOPOROSIS RATE AMONG PATIENTS OVER 50 YEARS OLD UNDERGOING MEDICAL REHABILITATION." Annals of the Rheumatic Diseases 80, Suppl 1 (May 19, 2021): 1020.2–1020. http://dx.doi.org/10.1136/annrheumdis-2021-eular.4258.
Full textCampos, Wladimir Gushiken de, Gonzalo André Montesinos, Rosa Cristina Peinado Agudo, Kaisermann Costa, Luciana Munhoz, and Emiko Saito Arita. "Does type 2 diabetic osteoporotic patients present more periodontal risks than non-osteoporotic patients? An evaluation with mandibular cortical index (Klemetti)." Brazilian Journal of Oral Sciences 17 (December 11, 2018): e181211. http://dx.doi.org/10.20396/bjos.v17i0.8654217.
Full textNawrat-Szołtysik, Agnieszka, Zuzanna Miodońska, Ryszard Zarzeczny, Izabela Zając-Gawlak, Józef Opara, Alicja Grzesińska, Beata Matyja, and Anna Polak. "Osteoporosis in Polish Older Women: Risk Factors and Osteoporotic Fractures: A Cross–Sectional Study." International Journal of Environmental Research and Public Health 17, no. 10 (May 25, 2020): 3725. http://dx.doi.org/10.3390/ijerph17103725.
Full textDissertations / Theses on the topic "Osteoporosis"
Kaptoge, Stephen Kipkemoi. "Epidemiology of risk factors for osteoporosis and osteoporotic fractures." Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.615203.
Full textWilson, Aubrie. "Osteoporosis." Online version, 2009. http://www.uwstout.edu/lib/thesis/2009/2009wilsona.pdf.
Full textMisner, Scottie, and Vanessa A. Farrell. "Osteoporosis." College of Agriculture, University of Arizona (Tucson, AZ), 2017. http://hdl.handle.net/10150/625576.
Full textOsteoporosis means “porous bones.” It is a condition where the skeleton becomes fragile and results in broken bones under normal use. Osteoporosis is a “silent” condition that happens slowly over years. The rate of bone loss (“resorption”) exceeds the rate of new bone formation (“acretion”). Many times neither a person nor a doctor is aware of weakened bones until one breaks unexpectedly. Originally published: 2000
Misner, Scottie. "Osteoporosis." College of Agriculture and Life Sciences, University of Arizona (Tucson, AZ), 2011. http://hdl.handle.net/10150/146661.
Full textOriginally published: 2000
Osteoporosis means "porous bones." it is a condition where the skeleton becomes fragile and results in broken bones under normal use. Osteoporosis is a "silent" condition that happens slowly over years. This publication discusses the symptoms and the risk factors of osteoporosis, as well as how to prevent it.
Braz, Manuela Giuliani Marcondes Rocha. "Sequenciamento paralelo em larga escala de genes candidatos para fragilidade óssea em indivíduos com osteoporose grave, familiar ou idiopática." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-22102018-123623/.
Full textOsteoporosis is a highly prevalent disorder resulting in fragility fractures and incurring in great morbi-mortality and economic burden. In most cases, osteoporosis has a multifactorial etiology, with an estimated heritability of 50-85% attributable to a combination of several low-impact genetic variants. Rarely, cases of syndromic osteoporosis due to high-impact genetic defects are seen. It is therefore hypothesized that severe/idiopathic cases of otherwise inconspicuous osteoporosis may have a monoor oligogenic etiology due to genetic variants with an intermediate effect. During the past years, advances in molecular sequencing have revealed novel candidate genes for bone fragility, and have enabled simultaneous sequencing of multiple genes. In this context, the objectives of this research project were: 1) to identify candidate genes for bone fragility, as previously reported in association to Mendelian disorders with high impact on bone resistance, idiopathic or familial osteoporosis, and genome-wide association studies (GWAS) for bone mineral density and fragility fractures; and 2) to perform molecular analysis of these candidate genes in patients with severe, familial or idiopathic osteoporosis. Through a systematic review, 128 candidate genes were identified and included in a panel for massively parallel sequencing. Coding regions and 25-bp boundaries were captured and sequenced. Rare variants (allele frequency < 1%), with a predicted high impact on protein function were initially selected as variants of interest. Thirty-seven subjects (21 sporadic cases and 7 families) were included according to stringent criteria based on clinical and densitometric evaluation, excluding individuals with secondary osteoporosis. Males represented 54% of the cohort, median age at diagnosis was 44 years, and 84% of subjects had a history of fractures. Thirtythree variants of interest were identified initially. After familial segregation analysis, 5 variants were considered as benign in regard to bone fragility, resulting in 28 potentially pathogenic variants, all heterozygous, present in 71% of the cohort. Of these variants, 26 were nonsynonymous, there was one 9-bp deletion and one large deletion involving the only coding exon of candidate gene GPR68. An association of two or more variants in different genes was present in 21% of the cohort, including a young woman with severe osteoporosis and variants in WNT1, PLS3 and NOTCH2. Familial segregation in this case suggested an additive pathogenic effect of these variants. Twenty-five percent of potentially pathogenic variants were identified in well-established candidate genes (WNT1, PLS3, COL1A1, COL1A2), and 57% located to novel candidate genes initially identified by GWAS, such as NBR1 and GPR68, which have been previously associated to changes in bone remodeling in mouse models. These results support the involvement of GWAS genes in the pathophysyiology of osteoporosis, and indicate a prominent role for digenic/oligogenic interactions in cases of severe, familial or idiopathic osteoporosis. Recognition of new molecular pathways in the determination of bone fragility may lead to the development of new drugs, and the identification of pathogenic variants associated to osteoporosis may allow individualized clinical management of patients and their relatives
Clarke, Michelle Mary. "The role of B-vitamin status, homocysteine and the MTHFR 677C-T polymorphism in bone health." Thesis, Ulster University, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669658.
Full textPereira, Erika Cristina Sbrisse. "Estudo longitudinal dos efeitos da deficiência estrogênica no fêmur de ratas /." São José dos Campos : [s.n.], 2011. http://hdl.handle.net/11449/95875.
Full textAbstract: The Brazilian population has undergone a process of aging, and this phenomenon with the number of people affected by chronic diseases is increasing. Among these a disease that deserves mention is osteoporosis, since it has become a public health problem. One of the main risk factors for osteoporosis is estrogen deficiency, so to better understand how estrogen deficiency affects the bone tissue present study aimed to evaluate the effect of estrogen deficiency on bone tissue over a period of 360 days by radiographic and biomechanics analysis. For this purpose 80 rats, 3 months old and weighing about 250g were divided into 10 groups (n = 8) according to the time of sacrifice and who underwent surgery (ovariectomy and sham surgery). The rats belonging to groups OVZ and SHAM had controlled his diet (30g per animal). After the specified period (30, 60, 90, 180 and 360 days of surgery) the rats were weighed, anesthetized and sacrificed, and their femurs were removed for analysis. Data were submitted to ANOVA and Tukey tests with significance level of 5%. According to the results obtained showed that in periods of 30, 60 and 90 days the effects of estrogen deficiency on bone tissue were significant
Orientador: Rosilene Fernandes da Rocha
Coorientador: Luana Marotta Reis de Vasconcellos
Banca: Juliana Madureira de Souza Lima Alonso
Banca: Mari Eli Leonelli de Moraes
Mestre
Vanhook, Patricia M., Lynne M. Dunphy, T. South, L. Plank, and C. Luskin. "Osteoarthritis and Osteoporosis." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/etsu-works/7411.
Full textFreitas, Deborah Queiroz de 1977. "Avaliação do efeito radioprotetor do selenito de sodio na reparação de tibias de ratas ovariectomizadas." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/290143.
Full textTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
Made available in DSpace on 2018-08-10T00:43:38Z (GMT). No. of bitstreams: 1 Freitas_DeborahQueirozde_D.pdf: 863530 bytes, checksum: bf09329a36f133306020d488b7ecaa3c (MD5) Previous issue date: 2007
Resumo: A osteoporose e a irradiação são fatores que interferem no processo de reparação óssea e podem ocorrer simultaneamente, especialmente em mulheres idosas. Atualmente, várias substâncias, conhecidas como radioprotetores, têm sido estudadas, pois minimizam os efeitos deletérios da radiação; dentre eles, pode-se citar o selênio. O objetivo desse trabalho foi avaliar o efeito radioprotetor do selenito de sódio no processo de reparo ósseo em ratas ovariectomizadas submetidas à irradiação. Para isso, oitenta ratas foram submetidas à ovariectomia e divididas em quatro grupos: ovariectomizado, ovariectomizado/selênio, ovariectomizado/irradiado e ovariectomizado/selênio/irradiado. Quarenta dias após, um defeito ósseo foi confeccionado nas tíbias dos animais. Dois dias após essa cirurgia, os animais dos grupos ovariectomizado/selênio eovariectomizado/selênio/irradiado receberam 0,8 mg Se/Kg de peso corpóreo. No dia seguinte, apenas os animais pertencentes aos grupos ovariectomizado/irradiado e ovariectomizado/selênio/irradiado receberam 10 Gy de radiação X na região dos membros inferiores. Os animais foram sacrificados 7, 14, 21 e 28 dias após a cirurgia. O processo de reparação óssea foi avaliado por análise morfológica, utilizando-se a coloração pelo Tricrômico de Masson, e por análise do número de trabéculas ósseas e da birrefrigência (coloração pelo Picrosírius). Pela análise morfológica, foi possível observar um atraso no processo de reparo ósseo nos animais do grupo ovariectomizado/irradiado e similaridade entre os grupos ovariectomizado, ovariectomizado/selênio e ovariectomizado/selênio/irradiado, o que demonstrou o efeito radioprotetor do selênio sem toxicidade
Abstract: Osteoporosis and ionizing radiation affect the bone healing and people can suffer both conditions, especially older women. At the moment, antioxidant radioprotectors have been evaluated, such selenium compounds. This study aimed at evaluating the selenium protection in the bone repair process in ovariectomized rats submitted to an irradiation procedure. For this purpose, eighty ovariectomized female Wistar rats were randomly divided in four experimental groups: ovariectomized, ovariectomized/selenium, ovariectomizedlirradiated and ovariectomized/selenium/irradiated. Abone defect was made on all animals' tibias forty days after ovariectomy. Two days after surgery, only ovariectomized/selenium and ovariectomized/seleniumlirradiated rats received 0.8 mg Se/Kg. Three days after surgery, only ovariectomized irradiated and ovariectomized/selenium/irradiated rats received 10 Gy of X rays on the lower limbs region. The animals were sacrificed 7, 14, 21 and 28 days after surgery in order to assess the repair process, which was evaluated by morphologic analysis in Masson Tricromic. lt was also evaluated by analysis of trabecular bone number in Masson Tricromic and birefringence analysis in Picrosirius. It was possible to observe a delay in the bone repair process in the irradiated/ovariectomized group and similarity between ovariectomized, ovariectomized/selenium and ovariectomized/selenium/irradiated, which proved the selenium radioprotection without its toxicity
Doutorado
Radiologia Odontologica
Doutor em Radiologia Odontológica
Ferreira, Gabriel Ramalho [UNESP]. "Avaliação do reparo ósseo na interface osso/implante em ratas com osteoporose induzida tratadas com raloxifeno ou alendronato: análise histométrica, imunoistoquímica, por epifluorescência e biomecânica." Universidade Estadual Paulista (UNESP), 2014. http://hdl.handle.net/11449/127657.
Full textO objetivo deste estudo foi avaliar o reparo ósseo na interface osso/implante em ratas com osteoporose induzida. As ratas submetidas à ovariectomia bilateral foram alimentadas com uma dieta pobre em cálcio. Dois grupos receberam tratamento medicamentoso (raloxifeno [OVX RAL] ou alendronato [OVX ALE]) e outro grupo não recebeu nenhuma medicação (OVX ST). O grupo controle foi submetido à cirurgia fictícia e foi alimentado com uma dieta normal (SHAM DN). Cada animal recebeu um implante em cada tíbia. Os animais foram eutanasiados após 14 ou 42 dias. Foram realizadas as análises biomecânica (torque reverso), extensão linear de contato osso/implante (ELCOI) e dinâmica óssea periimplantar pela proporção dos fluorocromos calceína/alizarina, aplicando-se a análise de variância ANOVA e o pós-teste de Tukey (p<0,05). A imunoistoquímica marcou a precipitação de osteoprotegerina (OPG), RANKL, TRAP e osteocalcina (OC). O medicamento RAL melhorou o reparo ósseo periimplantar, em que o grupo ALE foi semelhante ao grupo OVX ST. Não houve diferenças estatisticamente significativas no torque reverso (p = 0,861), na precipitação dos fluorocromos (calceína/alizarina) e na ELCOI entre os grupos OVX RAL e grupo controle - SHAM DN (p > 0,05). As imunomarcações de OPG e RANKL foram similares para os grupos RAL e SHAM; houve moderada expressão de OC aos 14 dias. A TRAP foi marcada intensamente aos 42 dias para o grupo OVX. Portanto, o raloxifeno melhorou o reparo ósseo periimplantar de ratas osteoporóticas, sugerindo a sua indicação no tratamento da osteoporose.
The aim of this study was to evaluate the bone healing in bone/implant interface in rats with induced osteoporosis. The rats underwent bilateral ovariectomy were fed a diet low in calcium. Two groups received drug treatment (raloxifene [OVX RAL] or alendronate [OVX ALE]) and the other group received no medication (OVX NT). The control group underwent sham surgery and was fed a normal diet (SHAM ND). Each animal received an implant on the tibia. The animals were euthanized after 14 or 42 days. The biomechanical analysis (reverse torque), linear extension contact bone / implant (BIC) and bone dynamics periimplantar by the proportion of fluorochrome calcein/alizarin, applying the ANOVA and Tukey's post-test (p<0.05). Immunohistochemistry marked precipitation of osteoprotegerin (OPG), RANKL, TRAP and osteocalcin (OC). The RAL improved drug peri-implant bone repair, wherein the ALE OVX group was similar to the ST group. There were no statistically significant differences in reverse torque (p = 0.861), precipitation of fluorochromes (calcein/alizarin) and BIC between OVX RAL and control groups - SHAM ND (p> 0.05). The immunostaining of OPG and RANKL were similar to RAL and SHAM groups; there was moderate OC expression at 14 days. TRAP was marked intensely at 42 days for the OVX group. Therefore, raloxifene improved peri-implant bone repair of osteoporotic rats, suggesting its indication in the treatment of osteoporosis.
Books on the topic "Osteoporosis"
Orwoll, Eric S., and Michael Bliziotes. Osteoporosis. New Jersey: Humana Press, 2002. http://dx.doi.org/10.1385/1592592783.
Full textCusano, Natalie E., ed. Osteoporosis. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-83951-2.
Full textBartl, Reiner, and Bertha Frisch. Osteoporosis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-09163-0.
Full textAdler, Robert A., ed. Osteoporosis. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-59745-459-9.
Full textStovall, Dale W., ed. Osteoporosis. Chichester, UK: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118316290.
Full textWestendorf, Jennifer J., ed. Osteoporosis. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-104-8.
Full textLeder, Benjamin Z., and Marc N. Wein, eds. Osteoporosis. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-69287-6.
Full textRosen, Clifford J., ed. Osteoporosis. Totowa, NJ: Humana Press, 1996. http://dx.doi.org/10.1007/978-1-4612-0221-9.
Full textBartl, Reiner, and Bertha Frisch. Osteoporosis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-79527-8.
Full textFrancis, R. M., and W. Carson Dick, eds. Osteoporosis. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-010-9580-8.
Full textBook chapters on the topic "Osteoporosis"
da Silva, José António Pereira, and Anthony D. Woolf. "Osteoporotic Syndrome Osteoporosis." In Rheumatology in Practice, 261–69. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84882-581-9_26.
Full textBlain, Hubert, Tahir Masud, Finbarr C. Martin, and Stefania Maggi. "Osteoporosis." In Practical Issues in Geriatrics, 209–16. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-61997-2_22.
Full textKarnes, Jonathan M., and Colleen Watkins. "Osteoporosis." In Orthopedic Surgery Clerkship, 739–41. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-52567-9_152.
Full textYuksel, Nese, and Theresa L. Charrois. "Osteoporosis." In Patient Assessment in Clinical Pharmacy, 235–43. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11775-7_18.
Full textBarry, Maurice. "Osteoporosis." In Clinical Practice in Rheumatology, 95–97. London: Springer London, 2003. http://dx.doi.org/10.1007/978-0-85729-430-2_19.
Full textPignolo, Robert J. "Osteoporosis." In Classic Papers in Geriatric Medicine with Current Commentaries, 95–104. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-428-5_10.
Full textVeloski, Colleen. "Osteoporosis." In Women’s Health in Clinical Practice, 47–69. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-469-8_4.
Full textMiller, Paul D., and Socrates E. Papapoulos. "Osteoporosis." In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 343–44. Ames, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.part5.
Full textBaum, Thomas, Dimitrios C. Karampinos, Stefan Ruschke, Hans Liebl, Peter B. Noël, and Jan S. Bauer. "Osteoporosis." In Spinal Imaging and Image Analysis, 67–93. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-12508-4_3.
Full textAnnamalai, Aniyizhai. "Osteoporosis." In Medical Management of Psychotropic Side Effects, 289–90. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-51026-2_47.
Full textConference papers on the topic "Osteoporosis"
Paoletti, Nicola, Pietro Liò, Emanuela Merelli, and Marco Viceconti. "Osteoporosis." In the 9th International Conference. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/2037509.2037536.
Full textKasra, Mehran, Marc D. Grynpas, Rajka Soric, and Sara Arnaud. "A Clinical Evaluation of Vibration Testing in the Assessment of Osteoporosis." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2587.
Full textde Oliveira, Juliana Silva, and Alexandre dos Santos Gomes. "World of work for graduates of professional education in radiology: Distribution of bone densitometry equipment in the state of Rio de Janeiro." In VI Seven International Multidisciplinary Congress. Seven Congress, 2024. http://dx.doi.org/10.56238/sevenvimulti2024-068.
Full textKiapour, A., K. Sairyo, T. Terai, V. K. Goel, and N. A. Ebraheim. "Bilateral Laminar Hook Placements Reduces Pedicle Screw Pull Outs in Long Pedicle Screw Fixation." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19357.
Full textSalas, Christina, Meir Marmor, Thomas Chu, Paul Hansma, Amir Matityahu, and Jenni M. Buckley. "Assessment of Local Bone Quality of the Distal Radius Using a Novel Hard Tissue Diagnostic Instrument." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206823.
Full textFerreri, Suzanne, and Yi-Xian Qin. "Dynamic Mechanical Signals Delivered by Ultrasound Generate Site Specific Mediation of Bone Loss." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206219.
Full textDai, Chao. "Molecular Mechanisms of Osteoporosis: A Road Map for Osteoporosis Therapeutics." In International Conference on Health Big Data and Intelligent Healthcare. SCITEPRESS - Science and Technology Publications, 2022. http://dx.doi.org/10.5220/0011372900003438.
Full textBuckenmeyer, Laura E., Kristophe J. Karami, Ata M. Kiapour, Vijay K. Goel, Constantine K. Demetropoulos, and Teck M. Soo. "Biomechanical Effects of Lumbar Pedicle Screw Insertion Depth on Screw Loosening and Fulcrum Location." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14324.
Full textAbousleiman, Younane N., Son K. Hoang, and Minh H. Tran. "Inclined Direct Shear Testing Device: A New Tool for Bone Mechanics and Osteoporosis Research." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19156.
Full textDewi, Patria Sari. "Factors Associated with Osteoporosis." In The 5th Intenational Conference on Public Health 2019. Masters Program in Public Health, Universitas Sebelas Maret, 2019. http://dx.doi.org/10.26911/theicph.2019.01.55.
Full textReports on the topic "Osteoporosis"
su, hui, haipeng xue, ruochong wang, guoqing tan, binghan yan, and zhanwang xu. The efficacy and safety of Tanghuang Jiangu capsule in the treatment osteoporosis: A meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0031.
Full textBidwell, Joseph. Maximizing PTH Anabolic Osteoporosis Therapy. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada610951.
Full textStern, Paula H. Mechanism of Thyroid Hormone-Induced Osteoporosis. Fort Belvoir, VA: Defense Technical Information Center, October 1998. http://dx.doi.org/10.21236/ada368354.
Full textStern, Paula H. Mechanism of Thyroid Hormone-Induced Osteoporosis. Fort Belvoir, VA: Defense Technical Information Center, October 1997. http://dx.doi.org/10.21236/ada340690.
Full textStern, Paula H. Mechanism of Thyroid Hormone-Induced Osteoporosis. Fort Belvoir, VA: Defense Technical Information Center, October 1999. http://dx.doi.org/10.21236/ada391556.
Full textSarafrazi, Neda. Osteoporosis or Low Bone Mass in Older Adults: United States, 2017-2018. National Center for Health Statistics, March 2021. http://dx.doi.org/10.15620/cdc:103477.
Full textLanske, Beate. Role of Klotho in Osteoporosis and Renal Osteodystrophy. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613419.
Full textKelley, George A. Exercise and Osteoporosis: A Dose-Response Meta-Analysis. Fort Belvoir, VA: Defense Technical Information Center, May 2013. http://dx.doi.org/10.21236/ada580198.
Full textPolyakova, Yu V., L. E. Sivordova, E. A. Guryanova, B. V. Zavodovsky, and E. N. Shamitova. Is male osteoporosis a medical or social problem? Academy of Natural Knowledge, 2019. http://dx.doi.org/10.18411/2070-7428-2019-1-28556.
Full textKelley, George A. Exercise and Osteoporosis: A Dose-Response Meta-Analysis. Fort Belvoir, VA: Defense Technical Information Center, May 2011. http://dx.doi.org/10.21236/ada554471.
Full text