Academic literature on the topic 'Osteoclastogenesis'
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Journal articles on the topic "Osteoclastogenesis"
Kitaura, Hideki, Keisuke Kimura, Masahiko Ishida, Haruka Kohara, Masako Yoshimatsu, and Teruko Takano-Yamamoto. "Immunological Reaction in TNF-α-Mediated Osteoclast Formation and Bone ResorptionIn VitroandIn Vivo." Clinical and Developmental Immunology 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/181849.
Full textFukawa, Yuki, Kou Kayamori, Maiko Tsuchiya, and Tohru Ikeda. "IL-1 Generated by Oral Squamous Cell Carcinoma Stimulates Tumor-Induced and RANKL-Induced Osteoclastogenesis: A Possible Mechanism of Bone Resorption Induced by the Infiltration of Oral Squamous Cell Carcinoma." International Journal of Molecular Sciences 24, no. 1 (December 30, 2022): 688. http://dx.doi.org/10.3390/ijms24010688.
Full textKim, Jung-Eun. "Osteoclastogenesis and Osteogenesis." International Journal of Molecular Sciences 23, no. 12 (June 15, 2022): 6659. http://dx.doi.org/10.3390/ijms23126659.
Full textLegg, Katrin. "Osteoclastogenesis inhibitor identified." Nature Reviews Rheumatology 5, no. 8 (August 2009): 413. http://dx.doi.org/10.1038/nrrheum.2009.128.
Full textLu, Weiguang, Liu Yang, and Zhuojing Luo. "Myricitrin inhibits osteoclastogenesis." Journal of Orthopaedic Translation 7 (October 2016): 95–96. http://dx.doi.org/10.1016/j.jot.2016.06.087.
Full textMaruotti, Nicola, Maria Grano, Silvia Colucci, Francesca d’Onofrio, and Francesco Paolo Cantatore. "Osteoclastogenesis and arthritis." Clinical and Experimental Medicine 11, no. 3 (November 11, 2010): 137–45. http://dx.doi.org/10.1007/s10238-010-0117-2.
Full textFeng, Wei. "Osteoclastogenesis and osteoimmunology." Frontiers in Bioscience 19, no. 5 (2014): 758. http://dx.doi.org/10.2741/4242.
Full textDou, Ce, Nan Li, Ning Ding, Chuan Liu, Xiaochao Yang, Fei Kang, Zhen Cao, et al. "HDAC2 regulates FoxO1 during RANKL-induced osteoclastogenesis." American Journal of Physiology-Cell Physiology 310, no. 10 (May 15, 2016): C780—C787. http://dx.doi.org/10.1152/ajpcell.00351.2015.
Full textPanahipour, Layla, Zahra Kargarpour, Maria Laggner, Michael Mildner, Hendrik J. Ankersmit, and Reinhard Gruber. "TGF-β in the Secretome of Irradiated Peripheral Blood Mononuclear Cells Supports In Vitro Osteoclastogenesis." International Journal of Molecular Sciences 21, no. 22 (November 13, 2020): 8569. http://dx.doi.org/10.3390/ijms21228569.
Full textHuang, Deqiu, Fujian Zhao, Wendong Gao, Xiaofeng Chen, Zhouyi Guo, and Wen Zhang. "Strontium-substituted sub-micron bioactive glasses inhibit ostoclastogenesis through suppression of RANKL-induced signaling pathway." Regenerative Biomaterials 7, no. 3 (March 30, 2020): 303–11. http://dx.doi.org/10.1093/rb/rbaa004.
Full textDissertations / Theses on the topic "Osteoclastogenesis"
Apfeldorfer, Coralie. "Lysosome biogenesis during osteoclastogenesis." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2006. http://nbn-resolving.de/urn:nbn:de:swb:14-1164801444532-19433.
Full textDay, Christopher. "Genes Involved in Osteoclastogenesis." Thesis, Griffith University, 2005. http://hdl.handle.net/10072/367814.
Full textThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Medical Science
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Ansalone, Cecilia. "Myeloid precursors, osteoclastogenesis, and Spondyloarthropathies." Thesis, University of Glasgow, 2016. http://theses.gla.ac.uk/7578/.
Full textWitte, Sara. "The effect of redoxmodulation on osteoclastogenesis." Thesis, Linköping University, Department of Physics, Chemistry and Biology, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-57263.
Full textDuring osteoclast differentiation and bone resorption the redox status in the cell display a decrease in reduction and a shift to an oxidized state. Structure, metabolism and function are some of the extensive changes that cells undergo during differentiation which alters both the extra- and intracellular redox environment. Osteoclasts express enzymes such as TRAP and NADPH oxidase which generates reactive oxygen species (ROS). ROS are molecules formed by oxygen reduction which gives these radicals at least one unpaired electron and makes them very reactive and chemically unstable. These are factors which stimulates differentiation of osteoclasts and bone resorption. RAW 264.7 cells will differentiate to osteoclasts when stimulated with RANKL and to activated macrophages when stimulated with LPS.
The aim of this project was to analyze if the redox environment is affected during differentiation of RAW 264.7 cells to osteoclasts and macrophages. The reason for this was that we aimed to se if RAW 264.7 cells could be used as an in vitro system to study the effects of redox changes in osteoclasts and macrophages and their activation.
Results from Western blot showed that protein expression of the Cysteine/Glutamate transporter xCT was up regulated with LPS and downregulated with RANKL. Results from the GSH/Cys assay show that the treatments with redox modulators did not affect the levels of GSH and Cys to a measurable extent. However the levels increased for both intracellular and extracellular GSH and Cys forms at day 4 in the control and stimulated cells. Addition of the disulfide reductant DTT affected differentiation to osteoclasts, leading to smaller osteoclasts probably due to interference with fusion of mononuclear pre-osteoclasts. Thus, down regulation of the xCT transporter could be an important mechanism to maintain a low level of free thiols shown to interfere with the differentiation to osteoclasts.
Agrawal, Ankita. "Regulation of osteoclastogenesis by purinergic signalling." Thesis, University of Sheffield, 2013. http://etheses.whiterose.ac.uk/4679/.
Full textDoonan, James Joseph. "Fc gamma receptor mediated modulation of osteoclastogenesis." Thesis, University of Glasgow, 2014. http://theses.gla.ac.uk/5579/.
Full textCaballé, Serrano Jordi. "In vitro study of osteoclastogenesis under simulated bone augmentation: The effects of bone-conditioned medium and saliva on osteoclastogenesis." Doctoral thesis, Universitat Internacional de Catalunya, 2015. http://hdl.handle.net/10803/311633.
Full textLa present tesis doctoral és un compendi de quatre publicacions ampliant el coneixement de l’osteoclastogènesis en les regeneracions òssies, més especialment sobre els efectes de la saliva i el medi condicionat ossi en l’osteoclastogènesis. La reabsorció dels empelts ossis i de l’os de l’hoste, pot ser un repte especialment quan un defecte ossi ha de ser regenerat en condicions desfavorables o grans atròfies com per exemple després de traumatismes, diverses patologies, edat avançada o extraccions múlti¬ples. En la cavitat oral, la saliva pot entrar en contacte amb superfícies mineralitzades, tot i això la relació entre saliva i reabsorció òssia és encara desconeguda. En la present tesis hem examinat si la saliva afecta el procés de l’osteoclastogènesis in vitro, possiblement afectant a la regeneració i cicatrització òssia. La regeneració òssia és un procés comú en traumatolo¬gia, periodòncia, cirurgia oral i maxil•lofacial que involucra l’ús de substituts ossis. Els empelts d’os autòleg són considerats l’estàndard d’or dels sub¬stituts ossis degut a la seva trinitat de propietats: osteoconductivitat, oste¬oinducció i osteogènesis. Els factors paracrins alliberats pels empelts d’os autòleg podrien contribuir en el conjunt de processos que donen com a re-sultat la consolidació del empelts, tanmateix els mecanismes que regeixen aquest processos no són coneguts. En el present treball hem pogut carac¬teritzar un conjunt de proteïnes alliberades per partícules d’os cortical porcí en el medi condicionat ossi (BCM) per imitar l’ambient paracrí dels em¬pelts d’os cortical. Alguns dels factors alliberats pels empelts d’os autòleg podrien influenciar la reabsorció òssia explicant per què els osteoclasts es formen ràpidament a la superfície de les partícules d’os autòleg en els llocs regenerats. Tot i això els mecanismes moleculars que regeixen aquest pro¬cés, encara son desconeguts. Factors solubles alliberats pels empelts d’os autòleg in vitro tenen un impacte robust a la diferenciació de cèl•lules mes¬enquimals. En la present tesis doctoral, hem determinat si aquests factors solubles son capaços de canviar la diferenciació de cèl•lules mare hemat¬opoètiques a osteoclasts, desconegut abans de realitzar els estudis aquí presentats. Basant-nos en els resultats in vitro aquí presentats, es pot observar que la saliva suprimeix l’osteoclastogènesis i promociona el desenvolupament de cèl•lules amb un fenotip fagocític, afectant a la funció dels osteoclasts, les cèl•lules encarregades de reabsorbir l’os. La reabsorció dels empelts d’os autòleg es pot atribuir a l’efecte d’algunes de les proteïnes detecta¬des en les secrecions dels auto-empelts, anomenant aquestes secrecions Medi Condicionat d’Os (BCM). Un estudi proteòmic del BCM va mostrar que aquest medi condicionat conté més de 150 proteïnes, de les quals 43 es van caracteritzar com “secretades” i presents en la matriu extracel•lular. Vàrem descobrir que alguns dels factors continguts en el BCM com per exemple pleiotropina, galectina-1 o TGF-β1 poden afectar processos cel•lulars involucrats en la regeneració òssia. El resultats presentats en aquesta tesis sobre l’influencia del BCM en l’osteoclastogènesis demostra que el BCM termo-activat és capaç d’estimular l’osteoclastogènesis in vitro. Aquests resultats in vitro suporten la noció que la reabsorció dels auto-empelts ossis pot ser que estigui estimulada per mecanismes reguladors encara no definits. En aquesta línia, els protocols presentats sobre l’ús del BCM haurien d’animar a revelar els efectes paracrins dels empelts d’os autòleg durant el procés de regeneració òssia i obrir nous camins a investi¬gacions translacionals en l’ampli camp de la cirurgia reconstructora. Resumint-ho tot, podem concloure que la saliva afecta la reabsorció òssia promocionant el desenvolupament de cèl•lules amb un fenotip fagocític, i que no només la saliva pot afectar a la reabsorció òssia, sinó que també les se¬crecions dels injerts d’os autòleg. En aquest punt, hi ha suficient evidencia per concloure que els auto-empelts d’os no només tenen tres propietats, sinó una més: la propietat reguladora, la quarta dimensió dels empelts d’os autòleg.
Rashid, A. N. M. Mamun Or. "Modulation of in vitro osteoclastogenesis by glycated proteins." Thesis, https://doors.doshisha.ac.jp/opac/opac_link/bibid/BB13059543/?lang=0, 2017. https://doors.doshisha.ac.jp/opac/opac_link/bibid/BB13059543/?lang=0.
Full text博士(理学)
Doctor of Philosophy in Science
同志社大学
Doshisha University
Khol, Matthew Philip. "The Effects of Recombinant Osteoactivin on Murine Osteoclastogenesis." NEOMED Integrated Pharmaceutical Medicine / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ne2mh1424053380.
Full textBendixen, Amy Catherine. "INHIBITION OF OSTEOCLASTOGENESIS BY SEX STEROIDS AND OTHER NUCLEAR RECEPTOR LIGANDS." University of Cincinnati / OhioLINK, 2001. http://rave.ohiolink.edu/etdc/view?acc_num=ucin985626097.
Full textBooks on the topic "Osteoclastogenesis"
Yamaguchi, Masayoshi. Osteoclastogenesis: Research Advances and Clinical Challenges. Nova Science Publishers, Incorporated, 2017.
Find full textLebowitz, Dina P. Identifying the roles of Rac1 and Rac2 in osteoclastogenesis --in vitro. 2007, 2007.
Find full textSkiba, Grzegorz. Fizjologiczne, żywieniowe i genetyczne uwarunkowania właściwości kości rosnących świń. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_gs_2020.
Full textBook chapters on the topic "Osteoclastogenesis"
Kukita, Toshio, Akira Takahashi, Jing-Qi Zhang, and Akiko Kukita. "Membrane Nanotube Formation in Osteoclastogenesis." In Methods in Molecular Biology, 193–202. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2703-6_14.
Full textTaguchi, Yuu, Jin Gohda, and Jun-ichiro Inoue. "NF-κB Signaling in Osteoclastogenesis." In Protein Modifications in Pathogenic Dysregulation of Signaling, 197–210. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55561-2_13.
Full textLeung, Roland, and Michael Glogauer. "Rho GTPase Techniques in Osteoclastogenesis." In Methods in Molecular Biology, 167–79. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-61779-442-1_12.
Full textLee, Junwon, Seoung-Hoon Lee, Yongwon Choi, and Nacksung Kim. "The Negative Role of IDS in Osteoclastogenesis." In Advances in Experimental Medicine and Biology, 23–31. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-72009-8_3.
Full textSilvestris, Franco, Sabino Ciavarella, Sabino Strippoli, and Franco Dammacco. "Cell Fusion and Hyperactive Osteoclastogenesis in Multiple Myeloma." In Advances in Experimental Medicine and Biology, 113–28. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0782-5_5.
Full textColonna, Marco, Isaiah Turnbull, and Julia Klesney-Tait. "The Enigmatic Function of TREM-2 in Osteoclastogenesis." In Advances in Experimental Medicine and Biology, 97–105. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-72009-8_13.
Full textYasui, Tetsuro, Hisataka Yasuda, and Sakae Tanaka. "Regulation of RANKL-Induced Osteoclastogenesis by TGF-β." In Osteoimmunology, 103–7. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5366-6_11.
Full textKanzaki, Hiroyuki, Xiaozhe Han, Xiaoping Lin, Toshihisa Kawai, and Martin A. Taubman. "Is RANKL shedding involved in immune cell-mediated osteoclastogenesis?" In Interface Oral Health Science 2009, 403–5. Tokyo: Springer Japan, 2010. http://dx.doi.org/10.1007/978-4-431-99644-6_115.
Full textYang, Yajun, and Baoxue Yang. "Anti-osteoporosis Effect of Ganoderma (Lingzhi) by Inhibition of Osteoclastogenesis." In Advances in Experimental Medicine and Biology, 263–69. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9421-9_11.
Full textTsuneto, Motokazu, Toshiyuki Yamane, and Shin-Ichi Hayashi. "Methods for Investigation of Osteoclastogenesis Using Mouse Embryonic Stem Cells." In Methods in Molecular Biology, 239–53. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-962-8_16.
Full textConference papers on the topic "Osteoclastogenesis"
Massaro, Laura, Tania Colasanti, Francesca Spinelli, Fulvia Ceccarelli, Riccardo Mancini, Arbi Pecani, Enrica Cipriano, et al. "FRI0517 ANTI-CARBAMYLATED PROTEINS ANTIBODIES INDUCE OSTEOCLASTOGENESIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.6859.
Full textLee, Sang-Heon, Kyung Ann Lee, and Hae-Rim Kim. "THU0007 IL-26 PROMOTES OSTEOCLASTOGENESIS IN RHEUMATOID ARTHRITIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.6724.
Full textDegboe, Yannick, Iain Mcinnes, and Carl Goodyear. "FRI0365 PDE4 TARGETING SELECTIVELY INHIBITS INFLAMMATORY-DRIVEN OSTEOCLASTOGENESIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.1802.
Full textBrunner, JS, M. Hofmann, V. Saferding, A. Vogel, A. Lercher, P. Cheng, G. Schabbauer, and S. Blüml. "P113 Arginase I and the metabolic control of osteoclastogenesis." In 38th European Workshop for Rheumatology Research, 22–24 February 2018, Geneva, Switzerland. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2018.128.
Full textPore, Subrata K., Anuradha Sehrawat, and Shivendra V. Singh. "Abstract 826: Benzyl isothiocyanate inhibits breast cancer-induced osteoclastogenesis." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-826.
Full textFischer, A., S. Abdollahi-Rodsaz, ACY Yau, E. Lonnblom, R. Holmdahl, and G. Steiner. "P020 Toll-like receptor 9 influences inflammatory arthritis and osteoclastogenesis." In 38th European Workshop for Rheumatology Research, 22–24 February 2018, Geneva, Switzerland. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2018.45.
Full textFischer, A., S. Abdollahi-Rodsaz, A. C. Y. Yau, E. Lönnblom, B. Meyer, B. Niederreiter, R. Holmdahl, and G. Steiner. "AB0040 Toll-like receptor 9 influences inflammatory arthritis and osteoclastogenesis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.4557.
Full textGangar, Subhash Chander, Gagan Deep, and Rajesh Agarwal. "Abstract 5661: Silibinin inhibits advanced human prostate carcinoma-induced osteoclastogenesis." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5661.
Full textChen, Yu-Jen, Yu-Yawn Chen, Hui-Fen Liao, and Chih-Wen Chi. "Abstract 1661: Effect of thalidomide and liposomal doxorubicin on human osteoclastogenesis." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1661.
Full textBhaskara, Vasantha Kumar, Indra Mohanam, Jasti S. Rao, and Sanjeeva Mohanam. "Abstract 408: Intermittent hypoxia effect on osteoclastogenesis stimulated by neuroblastoma cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-408.
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