Relevant bibliographies by topics / Orofacial pain

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Orofacial pain'

Author: Grafiati
Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "Orofacial pain"

1

Belok, Gregory. "Orofacial Pain." Journal of the American Dental Association 152, no. 4 (April 2021): 257–58. http://dx.doi.org/10.1016/j.adaj.2021.02.006.

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Abdelatti, M. O. "Orofacial Pain." British Journal of Anaesthesia 105, no. 2 (August 2010): 241–42. http://dx.doi.org/10.1093/bja/aeq175.

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Oomens, Marjolijn. "Orofacial pain." Journal of Dentomaxillofacial Science 1, no. 1 (June 20, 2016): 96. http://dx.doi.org/10.15562/jdmfs.v1i1.38.

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Kadambande, S. "Orofacial Pain." Anaesthesia 66, no. 4 (March 2, 2011): 328–29. http://dx.doi.org/10.1111/j.1365-2044.2011.06677.x.

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Morgan, O. "Orofacial pain." British Dental Journal 208, no. 7 (April 2010): 323–24. http://dx.doi.org/10.1038/sj.bdj.2010.305.

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Warfield, Carol A., and Cynthia H. Kahn. "Orofacial Pain." Hospital Practice 24, no. 2 (February 15, 1989): 247–71. http://dx.doi.org/10.1080/21548331.1989.11703669.

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Rodd, H., and F. Boissonade. "Orofacial pain." Journal of Pain 5, no. 3 (April 2004): S41. http://dx.doi.org/10.1016/j.jpain.2004.02.129.

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Morgan, C., H. Rodd, N. Clayton, and F. Boissonade. "Orofacial pain." Journal of Pain 5, no. 3 (April 2004): S41. http://dx.doi.org/10.1016/j.jpain.2004.02.130.

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Fall-Dickson, J., C. Picco, C. Kasten-Sportes, K. Castro, X. Wang, and S. Gordon. "Orofacial pain." Journal of Pain 5, no. 3 (April 2004): S41. http://dx.doi.org/10.1016/j.jpain.2004.02.131.

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Haegerstam, G., and M. Allerbring. "Orofacial pain." Journal of Pain 5, no. 3 (April 2004): S41. http://dx.doi.org/10.1016/j.jpain.2004.02.132.

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Dissertations / Theses on the topic "Orofacial pain"

1

McCormick, Emma, and Magdalena Sjöwall. "Central sensitization in orofacial pain." Thesis, Malmö högskola, Odontologiska fakulteten (OD), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-19833.

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Syfte. Att retrospektivt undersöka relationen mellan central sensitisering i det orofacialaområdet och refererad smärta, som kliniskt fynd, samt psykosociala faktorer hos patienter medDC/TMD-muskeldiagnosen myofasciell smärta med refererad smärta (MPR). Studien syftadeäven till att undersöka skillnader gällande psykosociala faktorer mellan patienter somdiagnostiserats med DC/TMD muskeldiagnoserna myofasciell smärta med refererad smärta(MPR), lokal myalgi (LM) och patienter med orofacial smärta eller käkdysfunktion men ejkäkmuskeldiagnos (WMD) som kontrollgrupper.Material och metod. Information från 85 patienters DC/TMD-undersökning utförd påOrofaciala smärtenheten vid Malmö högskola mellan september 2012 till årsslutet 2013insamlades retrospektivt. Undersökta variabler inkluderade smärtintensitet, smärt-relateraddysfunktion, psykosociala faktorer (depression, ångest och stress) samt refererad smärta.Patienterna indelades i grupper baserade på muskeldiagnos enligt DC/TMD samt utbredning avsmärta. Non-parametrisk statistik användes och P < 0,05 betraktades som signifikant.Resultat. Patienter med MPR uppvisade en signifikant korrelation mellan totala antaletrefererade smärtlokalisationer och smärt-relaterad dysfunktion (rs = 0,43, n = 49, p = 0,002),depression (rs = 0,32, n = 49, p = 0,023) och stress (rs = 0,39, n = 49, p = 0,006). Patienter meden generell smärtutbredning uppvisade en signifikant högre grad av stress (p = 0,020) samt flerantal refererade smärtlokalisationer (p = 0,019) jämfört med patienter med lokal och/ellerregional orofacial smärta.Konklusion. Studien indikerar att grad av central sensitisering kan bedömas med hjälp avutbredningen av refererad smärta, undersökt enligt DC/TMD, hos patienter med diagnosenmyofasciell smärta med refererad smärta i det orofaciala området. Studien kunde inte påvisaskillnader gällande psykosociala faktorer mellan de undersökta grupperna.
Objective. The aim of this study was to retrospectively investigate the relation between referredpain, as a clinical finding, and psychosocial factors versus central sensitization in patients withmyofascial pain with referral (MPR) as assessed according to DC/TMD. The study also aimedto investigate differences regarding psychosocial factors between patients demonstratingmyofascial pain with referral (MPR) and patients diagnosed with the DC/TMD muscle diagnoselocal myalgia (LM) as well as OFP/TMD patients without masticatory muscular diagnose(WMD) as control patients.Material and methods. Patients’ medical records of 85 patients examined at the Orofacial PainUnit at Malmö University during September 2012 till the end of 2013 were retrospectivelyexamined for DC/TMD data. Examined variables included pain intensity, pain-related disability,psychosocial factors (depression, anxiety and stress) and referred pain. The patients weredivided into groups based on DC/TMD muscle diagnosis as well as extension of pain. Nonparametricstatistics were used and a probability level of P < 0.05 was considered as significant.Results. Patients with MPR demonstrated significant correlations between the total number ofreferred pain sites and disability score (rs = 0.43, n = 49, p = 0.002), depression (rs = 0.32, n =49, p = 0.023) as well as stress (rs = 0.39, n = 49, p = 0.006). Patients with generalized paindistribution demonstrated a significantly higher degree of stress (p = 0.020) as well as highernumber of referred pain sites (p = 0.019) than patients with local and/or regional orofacial pain.Conclusion. This study indicates that the degree of central sensitization can be estimated bythe extent of referred pain, as assessed according to DC/TMD, in patients with myofascial painwith referred pain in the orofacial region. This study could not detect a difference inpsychosocial factors between the three groups, myofascial pain with referral (MPR), localmyalgia (LM) and no masticatory muscle diagnosis (WMD).
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Mills, Emily. "Pain-modulation neural circuits underlying chronic orofacial pain." Thesis, The University of Sydney, 2019. https://hdl.handle.net/2123/21813.

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We have all experienced short-term pain that results from a brief noxious (harmful) stimulus. When we encounter noxious stimuli, the intensity of our pain response is modulated through the activation of pain-modulation neural circuits in the brain. Specifically, systems within the brainstem can both inhibit and facilitate nociceptive (i.e. pain-related) information when it first enters the central nervous system, either in the spinal cord dorsal horn or in the spinal trigeminal nucleus (SpV) in the brainstem for information relating to orofacial (face and mouth) regions. Pain-modulation systems serve a clear biological purpose – in some situations, pain inhibition is highly beneficial as it allows us to focus on escape or defensive behaviours; in other circumstances, pain facilitation can be helpful as it encourages us to rest and recover from potential tissue damage. Despite many of us experiencing short-term pain that is associated with an injury, for reasons that are unclear, some individuals experience long-lasting chronic pain that persists for months or years after the initial injury has healed. There is growing evidence from experimental animal investigations to suggest that the functioning of the pain-modulation circuits, particularly those centred around the rostral ventromedial medulla (RVM) and locus coeruleus in the brainstem, is compromised in chronic pain conditions. These preclinical studies suggest that both neuropathic pain (i.e. pain related to somatosensory nervous system damage) and non-neuropathic pain (i.e. pain related to non-nervous tissue injury) are associated with a functional shift in the pain-modulation system such that it favours an overall facilitation of pain processing. This may contribute to the maintenance of long-term pain in some individuals even after the initial injury has resolved. In humans, there is emerging evidence from psychophysical studies to suggest that some chronic pain conditions, including orofacial conditions, are associated with altered pain-modulation capacities. However, to date, pain-modulation circuit functioning has not been directly explored in individuals with chronic pain. The overall aim of this thesis was to use resting-state functional magnetic resonance imaging (fMRI) to explore the ongoing function of brainstem pain-modulation neural circuits in humans with chronic neuropathic and non-neuropathic orofacial pain. The first investigation (Chapter 2) aimed to determine whether a neuropathic pain condition, painful trigeminal neuropathy (PTN), is associated with altered ongoing functioning in RVM and locus coeruleus pain-modulation pathways. We performed functional connectivity analyses to investigate whether there is an altered coupling of fMRI signals between the RVM, locus coeruleus and other pain-modulation regions in PTN patients compared with pain-free controls. We identified that individuals with PTN display enhanced functional connectivity (signal coupling) between the RVM and other pain-modulation sites, including the midbrain periaqueductal gray (PAG), locus coeruleus and subnucleus reticularis dorsalis (SRD). Additionally, we found that PTN patients display enhanced RVM functional connectivity with the SpV, the region that first receives nociceptive information from orofacial regions. Together, these results show that PTN is associated with functional alterations within the brainstem pain-modulation network and the SpV. Considering the existing experimental animal literature, it is likely that this represents an ongoing and enhanced engagement of brainstem pain-facilitating processes that may contribute to persistent pain in individuals with neuropathic pain conditions. The second investigation (Chapter 3) aimed to explore whether painful temporomandibular disorder (TMD), a non-neuropathic pain condition characterised by pain around the temporomandibular joint, is also associated with alterations in signal coupling between the RVM and other brainstem pain-modulation regions. In this investigation, we employed two functional connectivity techniques to explore the coupling of fMRI signals averaged over the entire scan (“static” functional connectivity) in addition to changes in signal coupling over the course of the scan (“dynamic” functional connectivity) to explore pain-modulation circuit function in TMD patients compared with pain-free controls. We identified that, compared to controls, TMD patients display enhanced RVM static and dynamic connectivity with the SpV and SRD, and no change in RVM connections with the PAG and locus coeruleus. These findings show that TMD is associated with functional alterations within specific brainstem pain-modulation circuits that regulate nociceptive processing at the SpV. Consistent with the findings from preclinical studies, and similar to neuropathic conditions, these ongoing functional changes in TMD likely reflect an enhanced descending facilitation of nociceptive processing at the SpV that contributes to the maintenance of pain in these individuals. The first two investigations (Chapters 2 and 3) revealed that individuals with both neuropathic and non-neuropathic pain display functional alterations within pain-modulation circuits that likely contribute to the presence of ongoing pain. In addition to persistent pain, many patients with neuropathic pain report spontaneous fluctuations in their pain intensity. It is possible that moment-to-moment variations in pain-modulation system functioning can contribute to these spontaneous fluctuations in chronic pain intensity. As such, the third investigation (Chapter 4) aimed to determine whether, within PTN individuals, there are differences in RVM functional connectivity strengths with other brainstem regions during scan periods in which patients experience high versus low pain. We found that PTN patients displayed stronger RVM connectivity strengths with both the PAG and SpV during the period of highest compared with lowest reported pain. These findings show that moment-to-moment fluctuations in spontaneous neuropathic pain intensity are associated with functional changes in the pain-modulation system. Given that this system can both facilitate and inhibit nociception, these findings may reflect short-term variations in descending pain-modulation output at the SpV that contribute to short-term changes in spontaneous pain intensity. Overall, this series of investigations reveals that both neuropathic and non-neuropathic orofacial pain conditions are associated with ongoing functional changes in the brainstem pain-modulation circuits. Additionally, in neuropathic pain, short-term variations in brainstem system functioning are associated with spontaneous fluctuations in ongoing pain intensity. Together, these findings suggest that functional alterations within the pain-modulation neural circuits are associated with the presence and intensity of ongoing chronic orofacial pain in humans. Considering these data alongside the existing experimental animal research, it is likely that, following injury, some individuals experience a change in pain-modulation circuit functioning such that it favours the facilitation of nociceptive processing. This may contribute to the maintenance and intensity of persistent orofacial pain in some individuals following injury.
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Maulina, Tantry. "ASSOCIATIONS BETWEEN COMMUNITY OROFACIAL PAIN AND EXPERIMENTAL OROFACIAL PAIN WITH PHYSICAL, SOCIAL AND/OR PSYCHOLOGICAL VARIABLES." Thesis, The University of Sydney, 2013. http://hdl.handle.net/2123/9789.

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Chronic orofacial pain can influence the quality of life of the sufferer by interfering with physical, social, and psychological functions. The aim of the study was to investigate the effect of orofacial pain on muscle activity and its association with physical and social functioning as well as psychological variables. The study was conducted in two consecutive phases. The first phase of the study was an epidemiological study that involved 700 participants and that was conducted in Indonesia. The second phase of the study was an experimental orofacial pain study in 14 participants, and this was conducted in Australia. The results of the first phase of the study revealed that there is a high prevalence of orofacial pain in an Indonesian patient sample with a high level of interference in social functioning and several physical activities including mastication. The results of the second phase of the study revealed some significant effects of experimental muscle pain on jaw muscle activity and some of these effects correlated with some psychological variables. In conclusion, this study has demonstrated the biopsychosocial effects of orofacial pain through study of a community based patient sample and through a study of experimental pain.
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Galli, Ursula. "Stress and pain (dys)regulation in chronic orofacial pain." Göttingen Cuvillier, 2008. http://d-nb.info/99103158X/04.

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Öjstedt, Erik, and Simon Pankalla. "Clinical Assessment of Disturbed Central Pain Modulation in Orofacial Pain." Thesis, Malmö universitet, Odontologiska fakulteten (OD), 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-19798.

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Syfte. Studiens syfte var att retrospektivt undersöka vilka kliniska variabler, bedömda under specialistundersökning av orofacial smärta, som kan förutsäga närvaro av en störd central smärtmodulering (DCPM). Material och metod. DC/TMD-data hämtades ur patientjournaler från 86 patienter som undersökts på Orofaciala smärtenheten på Malmö Universitet under perioden september 2012 till och med december 2013. Undersökta variabler omfattade smärtintensitet, smärtutbredning, smärtrelaterad nedsatthet, psykosociala variabler, refererad smärta samt kliniska fynd under somatosensoriska undersökningar. Baserat på denna data delades patienterna upp i en DCPM-grupp och en grupp utan DCPM. Allodyni, hyperalgesi, dysestesi, wind-up, regional/generell smärtutbredning samt eftersensation ansågs vara markörer för DCPM. Icke-parametriska statistiska analyser användes och en sannolikhetsnivå på P<0,05 ansågs vara signifikant. Resultat. Graden av ospecifika fysiska symptom och antalet refererande smärtor var signifikant högre i DCPM-gruppen. Den multivariata logistiska regressionen visade att ospecifika fysiska symptom, stress, smärtduration, smärtintensitet, smärtrelaterad nedsatthet, antalet refererande smärtpunkter, maximal gapning med och utan smärta, ångest samt antalet smärtinducerande käkrörelser var signifikanta marörer för DCPM (LR Chi2 = 26.89, p = 0.003, Pseudo R2 = 0.29). Slutsats. Denna studie indikerar att stress, ångest, smärtduration, smärtintensitet, smärtrelaterad nedsatthet, antalet refererande smärtpunkter, maximal gapning med och utan smärta samt antalet smärtinducerande käkrörelser är associerat med DCPM hos patienter med orofacial smärta.
Objective. To retrospectively investigate clinical variables that can predict the presence of disturbed central pain modulation (DCPM). Material and methods Medical records of 86 patients examined at the Orofacial Pain Unit at Malmö University from September 2012 to December 2013 were examined regarding pain intensity, pain distribution, pain-related disability, psychosocial variables, referred pain as well as somatosensory changes. Based on these variables, the patients were divided into a disturbed central pain modulation (DCPM) group and a non-DCPM group. Allodynia, hyperalgesia, dysesthesia, increased wind-up, regional/general pain distribution and aftersensation were considered as markers for DCPM. Non-parametric statistics were used and a probability level of P<0.05 was considered as significant. Results. The degree of unspecific physical symptoms and the number of sites eliciting pain referral were significantly higher in the DCPM group. In the multivariate regression model, the independent variables physical symptoms, stress, pain duration, characteristic pain intensity, pain-related disability, number of sites with referred pain, maximum mouth opening with and without pain, anxiety, and number of pain eliciting jaw movements significantly predicted DCPM (LR Chi2 = 26.89, p = 0.003, Pseudo R2 = 0.29). Conclusion. This study indicates that stress, anxiety, orofacial pain and its consequences, unspecific physical symptoms and jaw dysfunction are clinical signs of DCPM in patients with orofacial pain. Also, high number of palpations sites with referred pain over the masseter and temporal muscles and the TMJ indicate presence of DCPM.
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Hampf, Göran. "Somatic and psychic aspects of orofacial dysaesthesia." Helsinki : [s.n.], 1987. http://catalog.hathitrust.org/api/volumes/oclc/15667812.html.

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Thesis--University of Helsinki, 1987.
At head of title: Department of Oral and Maxillofacial Surgery, University of Helsinki ... Department of Psychiatry of Helsinki University Central Hospital. Also published in: Proceedings of the Finnish Dental Society, 1986, Vol. 83, Suppl. II. Includes bibliographical references (p. 63-72).
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Vickers, Edward Russell. "Clinical and pharmacological studies of orofacial pain." University of Sydney. Anaesthesia and Pain Management, 2000. http://hdl.handle.net/2123/845.

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For pain research, the orofacial region is unique in a number of ways. The region has complex local anatomy, including substantial sensory innervation from neural pathways, and muscles of facial expression that convey important information concerning pain intensity and associated psychological traits. Although chronic orofacial pain conditions appear prevalent, useful documentation on pain intensity ratings using well established instruments is sparse. In particular, two conditions, atypical facial pain and atypical odontalgia, are poorly understood in aetiology so that definitive treatment modalities are severely limited. The region's local biofluid, saliva, has been used to diagnose various local and systemic disease states, and to quantitate drug concentrations. However, recent studies indicate that saliva also contains some of the same peptides, e.g. bradykinin, that are involved in pain mechanisms. It may be that pharmacological-pharmacokinetic studies of these peptides could shed more information on thesignificance of their presence in saliva. This thesis consists of four major sections. Section 1 comprises of three clinical studies investigating orofacial pain. Section 2 deals with clinical laboratory studies of saliva. Section 3 is concerned with the development of chromatographic methods to assay bradykinin and its pharmacokinetics in saliva. Section 4 uses chromatography for the identification of novel salivary peptides. This thesis, then, presents clinical studies of orofacial pain and pharmacological investigations of saliva as the local biofluid.
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Bonathan, C. J. "How do patients understand chronic orofacial pain?" Thesis, University College London (University of London), 2011. http://discovery.ucl.ac.uk/1329456/.

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Part one of this volume presents a review of the literature on the relationship between chronic pain and socioeconomic status. It examines the evidence supporting the association and considers the psychological meaning of the findings. Part two consists of a qualitative empirical paper which samples patients with chronic orofacial pain to explore their understanding of their pain and their beliefs and fears about the causes and maintenance of their pain, both before and after an initial consultation at a specialist pain clinic. The final section is a critical appraisal of conducting this thesis. It contains a personal reflection of conducting both the literature review and empirical paper and describes some of the obstacles encountered during the process.
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Vickers, E. R. "Clinical and pharmacological studies of orofacial pain." Connect to full text, 1999. http://hdl.handle.net/2123/845.

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Thesis (Ph. D.)--University of Sydney, 1999.
Title from title screen (viewed Apr. 21, 2008). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the Dept. of Anaesthesia and Pain Management, Faculty of Medicine. Includes bibliography. Also available in print form.
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Vickers, Edward Russell. "Clinical and pharmacological studies of orofacial pain." Thesis, The University of Sydney, 1999. http://hdl.handle.net/2123/845.

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For pain research, the orofacial region is unique in a number of ways. The region has complex local anatomy, including substantial sensory innervation from neural pathways, and muscles of facial expression that convey important information concerning pain intensity and associated psychological traits. Although chronic orofacial pain conditions appear prevalent, useful documentation on pain intensity ratings using well established instruments is sparse. In particular, two conditions, atypical facial pain and atypical odontalgia, are poorly understood in aetiology so that definitive treatment modalities are severely limited. The region's local biofluid, saliva, has been used to diagnose various local and systemic disease states, and to quantitate drug concentrations. However, recent studies indicate that saliva also contains some of the same peptides, e.g. bradykinin, that are involved in pain mechanisms. It may be that pharmacological-pharmacokinetic studies of these peptides could shed more information on thesignificance of their presence in saliva. This thesis consists of four major sections. Section 1 comprises of three clinical studies investigating orofacial pain. Section 2 deals with clinical laboratory studies of saliva. Section 3 is concerned with the development of chromatographic methods to assay bradykinin and its pharmacokinetics in saliva. Section 4 uses chromatography for the identification of novel salivary peptides. This thesis, then, presents clinical studies of orofacial pain and pharmacological investigations of saliva as the local biofluid.
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Books on the topic "Orofacial pain"

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Clark, Glenn T., and Raymond A. Dionne, eds. Orofacial Pain. West Sussex, UK: John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118704851.

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Vadivelu, Nalini, Amarender Vadivelu, and Alan David Kaye, eds. Orofacial Pain. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-01875-1.

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Goulet, Jean-Paul, and Ana Miriam Velly, eds. Orofacial Pain Biomarkers. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-53994-1.

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Sharav, Yair, and Rafael Benoliel. Orofacial pain and headache. Chicago: Quintessence Publishing Co, Inc, 2015.

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Yair, Sharav, and Benoliel Rafael, eds. Orofacial pain and headache. Edinburgh: Mosby, 2008.

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E, Bell Welden, ed. Bell's Orofacial pains. 5th ed. Chicago: Quintessence Pub. Co., 1995.

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Kaye, Alan David, Nalini Vadivelu, and Amarender Vadivelu. Orofacial pain: A clinician's guide. Cham: Springer, 2014.

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Moule, Alex J., and M. Lamar Hicks, eds. Diagnosing Dental and Orofacial Pain. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119155218.

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R, Fricton James, and Dubner R. 1934-, eds. Orofacial pain and temporomandibular disorders. New York: Raven Press, 1994.

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Peter, Selvaratnam, Niere Ken, and Zuluaga Maria 1957-, eds. Headache, orofacial pain and bruxism. Edinburgh: Churchill Livingstone, 2009.

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Book chapters on the topic "Orofacial pain"

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Sessle, Barry J., Lene Baad-Hansen, and Peter Svensson. "Orofacial Pain." In Clinical Pain Management, 258–66. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444329711.ch31.

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Day, Miles, Kathryn Glynn, Ryan McKenna, Bhargav Mudda, and Katrina von-Kriegenbergh. "Orofacial Pain." In Academic Pain Medicine, 317–25. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-18005-8_41.

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Lewis, Michael A. O., and Philip-John Lamey. "Orofacial Pain." In BDJ Clinician’s Guides, 145–68. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-15432-5_8.

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Vettore, Mario Vianna, and Gabriela de Almeida Lamarca. "Orofacial Pain." In Textbooks in Contemporary Dentistry, 107–20. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50123-5_6.

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Nasri-Heir, Cibele, Rafael Benoliel, Riva Touger-Decker, Joel B. Epstein, and Eli Eliav. "Orofacial Pain." In Nutrition and Oral Medicine, 313–31. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-60761-490-6_17.

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Eckmann, Maxim S., and Antoun Nader. "Orofacial Pain." In Chronic Pain Management in General and Hospital Practice, 341–53. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-2933-7_20.

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Elsharydah, Ahmad. "Orofacial Pain." In Pain Management for Clinicians, 183–91. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39982-5_8.

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Sessle, Barry J., and Jonathan O. Dostrovsky. "Orofacial Pain." In Encyclopedia of Pain, 2535–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_2997.

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Jay, Gary W. "Orofacial pain." In The Headache Handbook, 101–6. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9781003076032-10.

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Vadivelu, Nalini, Yili Huang, Peter Mancini, Shaun Gruenbaum, Amarender Vadivelu, and Susan Dabu-Bondoc. "The Neurobiology of Orofacial Pain." In Orofacial Pain, 1–8. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-01875-1_1.

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Conference papers on the topic "Orofacial pain"

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Santos, Alcylene Carla de Jesus dos, Amanda dos Santos de Oliveira, Cintia Moraes Gutierrez, Fernanda Cardoso Nakamoto, Letícia Neves Mode, Margarete Nobilo Leonardis, Lais Silva Ferreira, Taisi Antunes da Cunha, and Daniela Aparecida Biasotto-Gonzalez. "Effect of telerehabilitation in patients with orofacial pain during the COVID-19 pandemic." In III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-142.

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The COVID-19 pandemic has impacted public and private health services, replacing face-to-face care with tele-rehabilitation as an alternative to assisting the population. Despite studies on the influence of self-efficacy on adherence, implementation of the therapeutic plan and health promotion behaviors, there is little national scientific data on the impact of telerehabilitation on self-efficacy and health care for patients with orofacial pain. Thus, the objective of this study was to evaluate the effect of telerehabilitation on self-efficacy and health care of patients with orofacial pain during the COVID-19 pandemic1,2.​
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Santos, Alcylene Carla de Jesus dos, Amanda dos Santos de Oliveira, Cintia Moraes Gutierrez, Fernanda Cardoso Nakamoto, Letícia Neves Mode, Margarete Nobilo Leonardis, Lais Silva Ferreira, Taisi Antunes da Cunha, and Daniela Aparecida Biasotto-Gonzalez. "Effect of telerehabilitation on patients with orofacial pain during the COVID-19 pandemic." In III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-268.

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The COVID-19 pandemic has impacted public and private health services, replacing face-to-face consultations with telerehabilitation as an alternative to assist the population. Despite studies on the influence of self-efficacy on adherence, completion of the therapeutic plan and health promotion behaviors, there is little national scientific data about the impact of telerehabilitation on self-efficacy and health care of patients with orofacial pain. Thus, the objective of this study was to evaluate the effect of telerehabilitation on the self-efficacy and health care of patients with orofacial pain during the COVID-19 pandemic 1,2.
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Vardasca, R., M. Clemente, A. Pinto, and J. Gabriel. "The outcomes of thermal symmetry after orofacial pain acupuncture treatment." In 2016 Quantitative InfraRed Thermography. QIRT Council, 2016. http://dx.doi.org/10.21611/qirt.2016.143.

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Oliveira, Marcos Fernando dos Santos, Andréa Rico Cabra, Taísa Maria Mendes Matuiama Machado, Antônio Sérgio Guimarães, and Luciane Lacerda Franco Rocha Rodrigues. "Phonophoresis therapy for TMD pain control." In II INTERNATIONAL SEVEN MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/homeinternationalanais-109.

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Abstract Temporomandibular dysfunction (TMD) according to Okeson, (1998) is the term used to describe a comprehensive condition of clinical changes in the orofacial region, temporomandibular joint (TMJ) and adjacent structures, where it is most commonly observed the painful symptomatology present in the dysfunctions of the masticatory muscles and cervical muscles. A non-invasive therapeutic option for TMD control is phonophoresis, a technique that associates therapeutic ultrasound with a drug, described by Skauen and Zentner (1984), as drug movement through intact living skin and soft tissues under the influence of an ultrasonic disturbance, enabling greater spreading, penetration and absorption of the drugs used.
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Nicoll, Steven B., Christopher K. Hee, Martin B. Davis, and Beth A. Winkelstein. "A Rat Model of Osteoarthritic Temporomandibular Joint Pain: Mechanically-Induced Behavioral Hypersensitivity and Histologic Modifications." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176520.

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Orofacial pain associated with osteoarthritis (OA) in the temporomandibular joint (TMJ) is a significant clinical problem [1]. The pathophysiologic and cellular mediators that underlie the development of such chronic orofacial pain are not well understood, nor has a relationship to mechanical loading been defined. Several experimental models have been developed to examine causative factors in TMJ OA progression and joint pathology. Such models often involve intra-articular injections or surgical manipulation of tissue structures in order to alter joint kinematics and stability [2–6]. For example, severing of the discal attachments followed by anterior displacement of the disc has been employed in a rabbit model, while disc perforation and scraping of the condylar surface have been used in sheep models to induce OA symptoms [2,3]. A limitation of the above approaches is that they introduce artificial damage to the joint structures and do not approximate the clinical disorder of mechanically-induced TMJ OA. Therefore, the goal of this pilot study was to develop a novel model of TMJ OA via non-invasive and mechanically relevant methods that could produce behavioral hypersensitivity (mechanical allodynia) suggestive of pain symptoms and histological changes in the TMJ consistent with osteoarthritic pathology.
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Daniela Craciun, Maria, and Adriana Sabau. "Means of evaluation and treatment in the orofacial myogenic pain for rheumatic patients." In 2015 E-Health and Bioengineering Conference (EHB). IEEE, 2015. http://dx.doi.org/10.1109/ehb.2015.7391437.

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Dinculescu, Adrian, Cosmin Dugan, Cristian Vizitiu, and Ioanina Parlatescu. "Study on Choice Reaction Time as a Complementary Method in Idiopathic Orofacial Pain." In 2021 International Conference on e-Health and Bioengineering (EHB). IEEE, 2021. http://dx.doi.org/10.1109/ehb52898.2021.9657580.

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Mode, Leticia Neves, Fernanda Cardoso Nakamoto, Lais Silva Ferreira, Alcylene Carla dos Santos, Margarete Nobilo, Taisi Antunes da Cunha, Cintia Moraes Gutierrez, and Daniela Aparecida Biasotto-Gonzalez. "Effect of oculomotor exercises added to the treatment of temporomandibular dysfunction: case report." In III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-144.

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Temporomandibular Dysfunction (TMD) is of multifactorial etiology, considered the most common chronic musculoskeletal cause of orofacial pain and with an estimated prevalence in the world population of approximately 31% in adults and the elderly and about 11% in children and adolescents1. Convergence insufficiency (CI) is characterized by the inability of the eyes to perform the adduction movement together, to focus on a nearby object2. The literature shows that there is anatomical and physiological evidence of a connection between the oculomotor apparatus and the stomatognathic system, and there are significant associations between convergence insufficiency, pain and severity of TMD2. The aim of the study was to identify the effects of oculomotor treatment added to TMD treatment on decreasing muscle pain, increasing mandibular range of motion and improving ocular convergence.
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Gutierrez, Cintia Moraes, Fernanda Cardoso Nakamoto, Leticia Neves Mode, Margarete Nobilo, Lais Silva Ferreira, Taisi Antunes da Cunha, Alcylene Carla dos Santos, and Daniela Aparecida Biasotto-Gonzalez. "Mandibular exercises with hyperboloid decrease pain in individuals with temporomandibular dysfunction: randomized, blinded clinical trial." In III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-147.

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Temporomandibular dysfunction (TMD) is characterized by a set of clinical alterations, which may present articular, muscular or mixed components1. The most common and limiting clinical manifestation is orofacial pain. Exercise has been proposed as a tool in the treatment of TMD, being effective in reducing pain1,2. The Hyperboloid is a proprioceptive device, created and developed to assist in the treatment of conditions involving the stomatognathic system2. This myofunctional device is widely used for the treatment of patients with temporomandibular dysfunction (TMD), however the literature is still scarce regarding its use2, 3. Thus, this pilot study aimed to evaluate and compare the effects and clinical difference of proprioceptive treatment with hyperboloid associated with tongue-on-palate exercise versus proprioceptive treatment with hyperboloid on pain intensity in individuals with TMD.
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Kodji, X., J. D. S. Valente, F. Lundy, I. El Karim, and S. D. Brain. "THU0545 Characterising a mouse model of temporomandibular joint (TMJ) arthritis to study orofacial pain and inflammation." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.5876.

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Reports on the topic "Orofacial pain"

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Hawkins, James M. Body Pain Reporting in Tricare Eligible Beneficiaries with Orofacial Pain. Fort Belvoir, VA: Defense Technical Information Center, May 2015. http://dx.doi.org/10.21236/ad1012704.

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Devito, Karina, and Karina D. Devito Americano. Can infrared thermography replace other methods for evaluating the presence and intensity of neurogenic and musculoskeletal orofacial pain in adult patients? A systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2023. http://dx.doi.org/10.37766/inplasy2023.3.0091.

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Review question / Objective: To investigate the question "Can infrared thermography replace other methods for evaluating the presence and intensity of neurogenic and musculoskeletal orofacial pain in adult patients?", the following PECO question was formulated: P – Adult patients with a history of neurogenic and musculoskeletal orofacial pain E – Subjected to infrared thermography C – Submitted to other evaluative methods of presence and intensity of orofacial pain O – Correlation of infrared thermography with other evaluation methods of presence and intensity of orofacial pain.
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