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Published: 10 December 2022
Last updated: 29 January 2023

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1

Belevt͡sev, I͡Akov Nikolaevich. Metamorphogenic ore formation. Moscow: General Editorial Board for Foreign Language Publications, Nauka Publishers, 1986.

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2

Making the Mount Isa Mine, 1923-1933: The discovery ofthe giant Mount Isa silver-lead-zinc ore deposit, the formation of Mount Isa Mines Limited, and the development of the mine and township. Carlton Victoria, Australia: Australasian Institute of Mining and Metallurgy, 1996.

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3

Berkman, D. A. Making the Mount Isa Mine, 1923-1933: The discovery of the giant Mount Isa silver-lead-zinc ore deposit, the formation of Mount Isa Mines Limited, and the development of the mine and township. Carlton, Vic: Australasian Institute of Mining and Metallurgy, 1996.

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4

Iron ore deposits and banded iron formations of India. New Delhi: Daya Pub. House, 2012.

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5

Nefedʹev, M. A. Obʺemnai︠a︡ modelʹ i ot︠s︡enka perspektiv Ozerninskogo rudnogo uzla po geofizicheskim dannym: (Zapadnoe Zabaĭkalʹe). Ulan-Udė: Izdatelʹstvo Buri︠a︡tskogo nauchnogo t︠s︡entra SO RAN, 2009.

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6

Salemink, Jan. Skarn and ore formation at Seriphos, Greece, as a consequence of granodiorite intrusion =: Skarn en erts vorming te Serifos, Griekenland, ten gevolge van een granodioriet intrusie. [Utrecht: Instituut voor Aardwetenschappen der Rijksuniversiteit te Utrecht], 1985.

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7

Derkachev, A. N. Mineralogicheskie indikatory obstanovok prikontinentalʹnogo osadkoobrazovenii︠a︡ zapadnoĭ chasti Tikhogo okeana: Environmental mineralogical indicators of near-continental sediment formation within Pacific Ocean western part. Vladivostok: Dalʹnauka, 2010.

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8

Reger, R. D. Multiple glaciation and gold-placer formation, Valdez Creek Valley, western Clearwater Mountains, Alaska. Fairbanks, Alaska: State of Alaska, Dept. of Natural Resources, Division of Geological and Geophysical Surveys, 1990.

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9

Loen, Jeffrey S. Sedimentology and gold placer deposits--Cathedral Bluffs member of the Wasatch Formation, Dickie Springs-Pacific Butte Area, Fremont County, Wyoming. [Reston, Va.?]: U.S. Dept. of the Interior, Geological Survey, 1986.

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10

Loen, Jeffrey S. Sedimentology and gold placer deposits--Cathedral Bluffs member of the Wasatch Formation, Dickie Springs-Pacific Butte Area, Fremont County, Wyoming. [Reston, Va.?]: U.S. Dept. of the Interior, Geological Survey, 1986.

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11

Dickinson, Kendell A. The geology, carbonaceous materials, and origin of the epigenetic uranium deposits in the Tertiary Sespe Formation in Ventura County, California. Washington: U.S. G.P.O., 1988.

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12

Seeland, David A. Depositional systems of a synorogenic continental deposit: The Upper Paleocene and Lower Eocene Wasatch Formation of the Powder River Basin, northeast Wyoming. [Washington, D.C.]: U.S. G.P.O., 1992.

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13

Gole, Martin. The refinement of extrusive models for the genesis of nickel deposits: Implications from case studies at Honeymoon Well and the Walter Williams Formation : results of research carried out as MERIWA Project 79 in the CSIRO Division of Exploration Geoscience. East Perth, WA: Minerals and Energy Research Institute of Western Australia, 1990.

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14

Kalinin, A. A., Ye E. Savchenko, and V. Yu Prokofiev. Mineralogy and genesis of the Oleninskoe gold deposit (Kola Peninsula). FRC KSC RAS, 2021. http://dx.doi.org/10.37614/978.5.91137.446.4.

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Data on geology of the Oleninskoe deposit, and results of mineralogical and geochemical investigations of ores and altered rocks are presented. Mineralization is connected with granite porphyry sills, an end member of gabbrodiorite-diorite-granodiorite complex of minor intrusions. The main alteration processes are diopsidization and biotitization, formation of quartz-muscovite-albite, quartz-aresenopyrite-tourmaline, and quartz metasomatic rocks. More than 50 ore minerals (sulfides, sulfosalts, tellurides, and native metals) were identified in the ore, including 20 minerals of silver and gold. Mineral associations in the ore and sequence of mineral formation are defined. Five generations of gold-silver alloys are identified, its composition covers spectrum from native silver to high-grade gold. Mineralized fluids in the deposit are of high salinity (sodium and calcium chlorides), and rich in As, Sb, Pb, Cu, Zn, and Ag. The Oleninskoe deposit is classified as an epithermal metamorphosed gold deposit.The book is of interest for specialists in economic geology, mineralogy and geochemistry of ore deposits.
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15

L, Hansley Paula, and Geological Survey (U.S.), eds. Mineralogy of uranium ore from the Crow Butte uranium deposit, Oligocene Chadron Formation, northwestern Nebraska. Denver, CO: Dept. of the Interior, U.S. Geological Survey, 1989.

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16

Wauschkuhn, Arndt. Syngenesis and Epigenesis in the Formation of Mineral Deposits. Springer, 2011.

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17

Barendregt, R. W., J. V. Matthews, V. Behan-Pelletier, J. Brigham-Grette, J. G. Fyles, L. E. Ovenden, D. H. McNeil, et al., eds. Biostratigraphy, Age, and Paleoenvironment of the Pliocene Beaufort Formation on Meighen Island, Canadian Arctic Archipelago. Geological Society of America, 2021. http://dx.doi.org/10.1130/spe551.

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The Beaufort Formation records extraordinary details of Arctic environments and amplified temperatures at approximately modern levels of atmospheric CO2. It was deposited during the Neogene on the western side of what is now the Canadian Arctic Archipelago. Meighen Island is a key locality for studying this formation because marine sediments there are interbedded with terrestrial fossiliferous sands. The biostratigraphic succession, fossils from the marine beds, and paleomagnetic data from the Bjaere Bay region of the island suggest two potential ages for the studied exposures: either continuous deposition at ca. 3.0 Ma, or a sequence of deposits at ca. 4.5 Ma and 3.4 Ma. The sediments appear to encompass at least two eustatic highstands of sea level and a particularly warm climate interval of the Pliocene Arctic.
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18

Joyce, Rosemary A., and Joshua Pollard. Archaeological Assemblages and Practices of Deposition. Edited by Dan Hicks and Mary C. Beaudry. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780199218714.013.0012.

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Archaeologists routinely describe sites as composed of assemblages encountered in deposits. But what is actually meant by ‘assemblage’ and ‘deposition’? This article explores how these concepts have been developed and considers the implications of contemporary understandings of deposition and assemblage that depart significantly from conventional definitions, many still to be found in introductory text books. Conventionally, the term ‘assemblage’ is applied to a collection of artefacts or ecofacts recovered from a specific archaeological context — a site, an area within a site, a stratified deposit, or a specific feature such as a ditch, tomb, or house. This article further explains in details the histories of archaeological approaches followed by contemporary approaches. The conventional definitions of assemblage and deposition emerged from geological and processual models of archaeological ‘formation processes’ that developed from the nineteenth century through the mid-twentieth century. Analysis the contemporary approach towards archaeological assemblages finishes this article.
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19

J, Bartel Ardith, and Geological Survey (U.S.), eds. Some geochemical features of the Blackbird and Jackass zones of the Yellowjacket Formation (Middle Proterozoic) in east-central Idaho. [Denver, Colo.]: U.S. Dept. of the Interior, Geological Survey, 1991.

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20

W, Caddey Stanton, ed. The Homestake Gold Mine, an Early Proterozoic iron-formation-hosted gold deposit, Lawrence County, South Dakota. [Washington, D.C.]: U.S. G.P.O., 1991.

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21

1913-, Love J. D., and Geological Survey (U.S.), eds. Vanadium deposits in the Lower Permian Phosphoria Formation, Afton area, Lincoln County, western Wyoming. Denver, CO: U.S. Geological Survey, 2004.

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22

Winters, Mark B. An investigation of fluid inclusions and geochemistry of ore formation in the Cedar Creek breccia pipe, North Santium Mining District, Oregon. 1985.

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23

Lachmann, Helen J., and Giampaolo Merlini. The patient with amyloidosis. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0152.

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Amyloidosis is a disorder of protein folding in which normally soluble plasma proteins are deposited in the extracellular space in an abnormal insoluble fibrillar form. The process of amyloid formation and deposition causes cytotoxicity and progressive organ dysfunction. Amyloid is remarkably diverse and can be hereditary or acquired, localized or systemic, and lethal or merely an incidental finding. The most important numerically are AL amyloidosis, in which the fibrils are composed of monoclonal immunoglobulin light chains, and AA amyloidosis, in which the acute phase reactant Serum Amyloid A component forms the fibrils.The kidney is involved in 75% of patients with systemic amyloidosis. Heavy proteinuria or nephrotic syndrome is characteristic of most amyloid variants.Without treatment, systemic disease is usually fatal but measures that reduce the supply of amyloid fibril precursor proteins can result in regression of amyloid deposits, prevention of organ failure, and improved quality of life and survival. Early diagnosis, before irreversible organ damage has occurred, is the key to effective treatment. Recent advances in diagnosis and therapy have much improved the outlook of patients with AL amyloidosis, but agents with broader promise are under investigation.
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24

Geological Survey (U.S.), ed. Sedimentology and gold placer deposits--Cathedral Bluffs member of the Wasatch Formation, Dickie Springs-Pacific Butte Area, Fremont County, Wyoming. [Reston, Va.?]: U.S. Dept. of the Interior, Geological Survey, 1986.

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25

Malyukova, N. N., and T. K. Ametova. Geology. Part 1. KRSU Publishing House, 2021. http://dx.doi.org/10.36979/978-9967-19-808-1-2021.

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The study guide summarizes modern information about the structure and composition of the Earth’s crust. The descriptions of endogenous and exogenous geological processes, the most important ore and rock-forming minerals, rocks, tectonic dislocations are given. Theoretical course introduces students to the geology of oil and gas, considers hypotheses of hydrocarbons origin, physical and chemical properties of oil and gas, conditions of formation of deposits and regularities of their location in the Earth’s crust. This material contributes to solving urgent problems of mining production and training highly qualified specialists in the field of design and development of mineral deposits. For students of higher educational institutions, studying in the field of training of diplomate specialists in «Mining». The applicable nature of the Guide makes it useful and intelligible for not only students, Ph. students and teachers, but for specialists of the mining and geological survey.
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26

Structural controls and the role of fault dynamics during the formation of vein-hosted gold deposits in the Bendigo-Ballarat zone of the Lachlan fold belt. Townsville, Qld: Economic Geology Research Unit, Geology Dept., James Cook University of North Queensland, 1991.

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27

Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

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Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level of extracellular PPi and Pi is tightly regulated by several interlinked feedback mechanisms and growth factors. The relative concentration of Pi and PPi determines whether CPP or hydroxyapatite crystal is formed, with low Pi/PPi ratio resulting in CPP crystal formation, while a high Pi/PPi ratio promotes basic calcium phosphate crystal formation. CPP crystals are deposited in the cartilage matrix (preferentially in the middle layer) or in areas of chondroid metaplasia. Hypertrophic chondrocytes and specific cartilage matrix changes (e.g. high levels of dermatan sulfate and S-100 protein) are related to CPP crystal deposition and growth. CPP crystals cause inflammation by engaging with the NALP3 inflammasome, and with other components of the innate immune system, and is marked with a prolonged neutrophilic inflitrate. The pathogenesis of resolution of CPP crystal-induced inflammation is not well understood.
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28

Syngenesis and Epigenesis in the Formation of Mineral Deposits: A Volume in Honour of Professor G. Christian Amstutz on the Occasion of His 60th ... to One of His Main Scientific Interests. Springer, 2012.

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29

Kluth, C., A. Wauschkuhn, and R. A. Zimmermann. Syngenesis and Epigenesis in the Formation of Mineral Deposits: A Volume in Honour of Professor G. Christian Amstutz on the Occasion of His 60th Birthday with Special Reference to One of His Main Scientific Interests. Springer London, Limited, 2012.

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30

Grassi, Walter, Tadashi Okano, and Emilio Filippucci. Ultrasound in osteoarthritis and crystal-related arthropathies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0017.

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Ultrasonography (US) is a safe and cheap imaging technique which in experienced hands allows for a multiplanar and multisite high-resolution assessment of both morphological and structural features of bone, cartilage, and intra- or periarticular soft tissues. This chapter describes the main applications of US in patients with osteoarthritis (OA) and crystal-related arthropathies. Imaging plays a key role for diagnosis, prognosis, and follow-up in patients with OA. Although conventional radiography is still the gold standard imaging technique in daily clinical practice, US has been revealed to be capable of detecting a wide spectrum of otherwise undetectable details, including cartilage damage, joint effusion, synovial hypertrophy, osteophyte formation, and meniscal protrusion. Crystal visualization by US has the potential to change the diagnostic approach in patients with suspicion of crystal-related arthropathies. The double-contour sign, due to urate crystal deposits on the chondrosynovial interface of the hyaline cartilage, is a highly specific finding for gout as well as the hyperechoic spots within the hyaline cartilage for calcium pyrophosphate dihydrate crystal deposition disease. The potential applications of US in the management of patients with OA and crystal-related arthropathies are not only limited to diagnosis and monitoring. Finally, US guidance allows the real-time visualization of the needle moving through different tissues and reaching the target to aspirate and/or inject. The correct placement of the tip of the needle plays a key role in improving efficacy and reducing side effects of the injection.
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31

Barsoum, Rashad S. Schistosomiasis. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0194_update_001.

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The urinary system is the primary target of Schistosoma haematobium infection, which leads to granuloma formation in the lower urinary tract that heals with fibrosis and calcification. While the early lesions may be associated with distressing acute or subacute symptoms, it is the late lesions that constitute the main clinical impact of schistosomiasis. The latter include chronic cystitis, ureteric fibrosis, ureterovesical obstruction or reflux which may lead to chronic pyelonephritis. Secondary bacterial infection and bladder cancer are the main secondary sequelae of urinary schistosomiasis.The kidneys are also a secondary target of S. mansoni infection, attributed to the systemic immune response to the parasite. Specific immune complexes are responsible for early, often asymptomatic, possibly reversible, mesangioproliferative lesions which are categorized as ‘class I’. Subsequent classes (II–VI) display different histopathology, more serious clinical disease, and confounding pathogenic factors. Class II lesions are encountered in patients with concomitant salmonellosis; they are typically exudative and associated with acute-onset nephrotic syndrome. Classes III (mesangiocapillary glomerulonephritis) and IV (focal segmental sclerosis) are progressive forms of glomerular disease associated with significant hepatic pathology. They are usually associated with immunoglobulin A deposits which seem to have a significant pathogenic role. Class V (amyloidosis) occurs with long-standing active infection with either S. haematobium or S. mansoni. Class VI is seen in patients with concomitant HCV infection, where the pathology is a mix of schistosomal and cryoglobulinaemic lesions, as well as amyloidosis which seems to be accelerated by the confounded pathogenesis.Early schistosomal lesions, particularly those of the lower urinary tract, respond to antiparasitic treatment. Late urological lesions may need surgery or endoscopic interventions. As a rule, glomerular lesions do not respond to treatment with the exception of class II where dual antiparasitic and antibiotic therapy is usually curative. Patients with end-stage kidney disease may constitute specific, yet not insurmountable technical and logistic problems in dialysis or transplantation. Recurrence after transplantation is rare.
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32

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_001.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across the endothelium into the intima. Infiltrated pro-atherogenic monocytes (mainly Mon2) differentiate into macrophages which acquire a specialized phenotypic polarization (protective/M1 or harmful/M2), depending on the stage of the atherosclerosis progression. Once differentiated, macrophages upregulate pattern recognition receptors capable of engulfing modified low-density lipoprotein, leading to foam cell formation. Foam cells release growth factors and cytokines that promote vascular smooth muscle cell migration into the intima, which then internalize low-density lipoproteins via low-density lipoprotein receptor-related protein-1 receptors becoming foam cells. As the plaque evolves, the number of vascular smooth muscle cells decline, whereas the presence of fragile/haemorrhagic neovessels and calcium deposits increases, promoting plaque destabilization. Disruption of this atherosclerotic lesion exposes thrombogenic surfaces rich in tissue factor that initiate platelet adhesion, activation, and aggregation, as well as thrombin generation. Platelets also participate in leucocyte and progenitor cell recruitment are likely to mediate atherosclerosis progression. Recent data attribute to microparticles a modulatory effect in the overall atherothrombotic process and evidence their potential use as systemic biomarkers of thrombus growth. This chapter reviews our current understanding of the pathophysiological mechanisms involved in atherogenesis, highlights platelet contribution to thrombosis and atherosclerosis progression, and provides new insights into how atherothrombosis may be prevented and modulated.
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33

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0040_update_002.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the intimal layer and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles attached to the extracellular matrix suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across the endothelium into the intima. Infiltrated pro-atherogenic monocytes (mainly Mon2) differentiate into macrophages which acquire a specialized phenotypic polarization (protective/M1 or harmful/M2), depending on the stage of the atherosclerosis progression. Once differentiated, macrophages upregulate pattern recognition receptors capable of engulfing modified low-density lipoprotein, leading to foam cell formation. Foam cells release growth factors and cytokines that promote vascular smooth muscle cell migration into the intima, which then internalize low-density lipoproteins via low-density lipoprotein receptor-related protein-1 receptors becoming foam cells. As the plaque evolves, the number of vascular smooth muscle cells decline, whereas the presence of fragile/haemorrhagic neovessels and calcium deposits increases, promoting plaque destabilization. Disruption of this atherosclerotic lesion exposes thrombogenic surfaces rich in tissue factor that initiate platelet adhesion, activation, and aggregation, as well as thrombin generation. Platelets also participate in leucocyte and progenitor cell recruitment are likely to mediate atherosclerosis progression. Recent data attribute to microparticles a modulatory effect in the overall atherothrombotic process and evidence their potential use as systemic biomarkers of thrombus growth. This chapter reviews our current understanding of the pathophysiological mechanisms involved in atherogenesis, highlights platelet contribution to thrombosis and atherosclerosis progression, and provides new insights into how atherothrombosis may be prevented and modulated.
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