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Relevant bibliographies by topics / Opiate effects on humans

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Academic literature on the topic 'Opiate effects on humans'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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Journal articles on the topic "Opiate effects on humans"

1

PFEIFFER, A., V. BRANTL, A. HERZ, and H. M. EMRICH. "Kappa-opiate receptors mediate psychotomimetic effects in humans." Acta Endocrinologica 113, no. 1_Suppl (August 1986): S157—S158. http://dx.doi.org/10.1530/acta.0.111s157.

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Specker, S., Winai Wananukul, Dorothy Hatsukami, Kim Nolin, Lyn Hooke, Mary Jeanne Kreek, and Paul R. Pentel. "Effects of dynorphin A(1-13) on opiate withdrawal in humans." Psychopharmacology 137, no. 4 (June 17, 1998): 326–32. http://dx.doi.org/10.1007/s002130050626.

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Kabalak, Ayla A., Oytun O. Senel, and Nermin Gogus. "The Effects of Transcranial Electrical Stimulation on Opiate-Induced Analgesia in Rats." Pain Research and Management 9, no. 4 (2004): 203–6. http://dx.doi.org/10.1155/2004/416016.

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BACKGROUND AND OBJECTIVES:Recent experiments have shown that transcranial electrical stimulation significantly increases the potency and duration of the analgesic effects of opioids in humans and rats. In the present study, the influence of transcranial electrical stimulation (TCES) on the analgesic effect of remifentanil hydrochloride (HCl) in rats was determined.METHODS:Experiments were performed on 80 albino male Wistar rats. Rats were randomly assigned to four groups: remifentanil HCl, remifentanil HCl and TCES, TCES, and control (n=20/group). Remifentanil HCl was injected on the 55th minute. Analgesia was assessed using the wet tail-flick latency test.RESULTS:In the remifentanil HCl group, analgesia (10.85±1.04 s) was reached at the fifth minute, and the analgesia was high for the first 10 min. In the remifentanil HCl and TCES group, the latency time peaked (16.60±1.19 s) at the fifth minute. This peak was 150% higher than that for the remifentanil HCl group, and 251% higher than the control or TCES groups. Analgesia in the remifentanil HCl and TCES group was sustained for 20 min at a statistically higher rate than the other treatment groups (P<0.001).CONCLUSIONS:TCES markedly increased the duration and analgesic potency of remifentanil HCl in rats. This effect appeared to be related to the release of enkephalins from brain structures, thus enhancing opioid analgesia.

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Yaksh, Tony L., and Xiao-Ying Hua. "Spinal Tolerance and Dependence: Some Observations on the Role of SpinalN-Methyl-D-Aspartate Receptors and Phosphorylation in the Loss of Opioid Analgesic Responses." Pain Research and Management 5, no. 1 (2000): 33–39. http://dx.doi.org/10.1155/2000/514020.

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The continuous delivery of opiates can lead to a reduction in analgesic effects. In humans, as in other animals, some component of this change in sensitivity seems likely to have a strong pharmacodynamic component. Such loss of effect, deemed to be tolerance in the present article, can be readily demonstrated in animals with repeated bolus and continuous intrathecal infusion of mu and delta opioids and alpha-2 adrenergic agonists. Research has shown that this loss of effect can be diminished by concurrent treatment withN-methyl-D-aspartate (NMDA) receptor antagonists and by the suppression of the activity of spinal protein kinase C (PKC). This suggests in part the probable role of PKC-mediated phosphorylation in the right shift in the dose-effect curves observed with continuous opiate or adrenergic exposure. Importantly, this right shift is seen to occur in parallel with an increase in the phosphorylating activity in the dorsal horn and in the expression of several PKC isozymes. The target of this phosphorylation is not certain. Phosphorylation of the NMDA receptor enhances its functionality, while phosphorylation of the opioid receptor or associated channels seems to diminish their activity or to enhance internalization. While the focus is on several specific components, the accumulating data emphasize the biological complexity of these changes in spinal drug reactivity.

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HU, Huankai, Seiji MIYAUCHI, Christy C. BRIDGES, Sylvia B. SMITH, and Vadivel GANAPATHY. "Identification of a novel Na+- and Cl−-coupled transport system for endogenous opioid peptides in retinal pigment epithelium and induction of the transport system by HIV-1 Tat." Biochemical Journal 375, no. 1 (October 1, 2003): 17–22. http://dx.doi.org/10.1042/bj20031059.

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The endogenous opioid peptides enkephalins, dynorphins and endorphins consist of five or more amino acids. These peptides participate in a multitude of biological functions in mammalian cells by interacting with different subtypes of opiate receptors located on the plasma membrane and in the nucleus. Here we report on the identification of a new peptide transport system in the human retinal pigment epithelial (RPE) cells that transports a variety of endogenous opioid peptides with high affinity. We identified this novel, hitherto unrecognized, transport system when we were analysing the differential effects of Tat, the transacting factor encoded by HIV-1, on various transport processes in RPE cells. This transport system is markedly induced by Tat. This opioid transport system is energized by transmembrane Na+ and Cl− gradients and is distinct from any of the previously identified transport systems for opioid peptides in mammalian cells. Free amino acids, dipeptides, tripeptides and non-peptide opiate receptor antagonists are excluded by this newly identified transport system. The affinities of endogenous opioid peptides for this system are in the range of 0.4–40 μM. The identification of the high-affinity Na+- and Cl−-coupled transport system in mammalian cells that is specific for endogenous opioid peptides and is induced by HIV-1 Tat is of significance not only to the biology of opioid peptides but also to the pathology of HIV-1 infection in humans.

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Tabibi, Zahra, David C. Schwebel, Abolfazl Mohammadzadeh Moghaddam, Javad Salehi Fadardi, and Sara Mirzaei Feizabadi. "Differential effects of stimulant versus opiate drugs on driving performance." Accident Analysis & Prevention 150 (February 2021): 105885. http://dx.doi.org/10.1016/j.aap.2020.105885.

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Murphy, D. B., J. A. Sutton, L. F. Prescott, and M. B. Murphy. "Opioid-induced Delay in Gastric Emptying." Anesthesiology 87, no. 4 (October 1, 1997): 765–70. http://dx.doi.org/10.1097/00000542-199710000-00008.

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Background Opioids delay gastric emptying, which in turn may increase the risk of vomiting and pulmonary aspiration. Naloxone reverses this opiate action on gastric emptying, but it is not known whether this effect in humans is mediated by central or peripheral opiate antagonism. The importance of peripheral opioid receptor antagonism in modulating opioid-induced delay in gastric emptying was evaluated using methylnaltrexone, a quaternary derivative of the opiate antagonist naltrexone, which does not cross the blood-brain barrier. Methods In a randomized, double-blind, crossover placebo-controlled study, 11 healthy volunteers were given either placebo (saline), 0.09 mg/kg morphine, or 0.09 mg/kg morphine plus 0.3 mg/kg methylnaltrexone on three separate occasions before ingesting 500 ml deionized water. The rate of gastric emptying was measured by two methods: a noninvasive epigastric bioimpedance technique and the acetaminophen absorption test. Results The epigastric bioimpedance technique was sufficiently sensitive to detect opioid-induced changes in the rate of gastric emptying. The mean +/- SD time taken for the gastric volume to decrease to 50% (t0.5) after placebo was 5.5 +/- 2.1 min. Morphine prolonged gastric emptying to (t0.5) of 21 +/- 9.0 min (P < 0.03). Methylnaltrexone given concomitantly with morphine reversed the morphine-induced delay in gastric emptying to a t0.5 of 7.4 +/- 3.0 (P < 0.04). Maximum concentrations and area under the concentration curve from 0 to 90 min of serum acetaminophen concentrations after morphine were significantly different from placebo and morphine administered concomitantly with methylnaltrexone (P < 0.05). No difference in maximum concentration or area under the concentration curve from 0 to 90 min was noted between placebo and methylnaltrexone coadministered with morphine. Conclusions The attenuation of morphine-induced delay in gastric emptying by methylnaltrexone suggests that the opioid effect is mediated outside the central nervous system. Methylnaltrexone may have the potential to decrease the side effects of opioid medications, which are mediated peripherally, while maintaining the central analgesia effect of the opioid.

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Drago, F., G. Panissidi, F. Bellomio, A. Dal Belle, E. Aguglia, and G. Gorgone. "EFFECTS OF OPIATES AND OPIOIDS ON INTRAOCULAR PRESSURE OF RABBITS AND HUMANS." Clinical and Experimental Pharmacology and Physiology 12, no. 2 (April 1985): 107–13. http://dx.doi.org/10.1111/j.1440-1681.1985.tb02312.x.

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Dahan, Albert MD, Elise Sarton, Luc Teppema, and Cees Olievier. "Sex-related Differences in the Influence of Morphine on Ventilatory Control in Humans." Anesthesiology 88, no. 4 (April 1, 1998): 903–13. http://dx.doi.org/10.1097/00000542-199804000-00009.

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Background Opiate agonists have different analgesic effects in male and female patients. The authors describe the influence of sex on the respiratory pharmacology of the mu-receptor agonist morphine. Methods The study was placebo-controlled, double-blind, and randomized. Steady-state ventilatory responses to carbon dioxide and responses to a step into hypoxia (duration, 3 min; oxygen saturation, approximately 82%; end-tidal carbon dioxide tension, 45 mmHg) were obtained before and during intravenous morphine or placebo administration (bolus dose of 100 microg/kg, followed by a continuous infusion of 30 microg x kg(-1) x h(-1)) in 12 men and 12 women. Results In women, morphine reduced the slope of the ventilatory response to carbon dioxide from 1.8 +/- 0.9 to 1.3 +/- 0.7 l x min(-1) x mmHg(-1) (mean +/- SD; P < 0.05), whereas in men there was no significant effect (control = 2.0 +/- 0.4 vs. morphine = 1.8 +/- 0.4 l x min(-1) x mmHg(-1)). Morphine had no effect on the apneic threshold in women (control = 33.8 +/- 3.8 vs. morphine = 35.3 +/- 5.3 mmHg), but caused an increase in men from 34.5 +/- 2.3 to 38.3 +/- 3 mmHg, P < 0.05). Morphine decreased hypoxic sensitivity in women from 1.0 +/- 0.5 l x min(-1) x %(-1) to 0.5 +/- 0.4 l x min(-1) x %(-1) (P < 0.05) but did not cause a decrease in men (control = 1.0 +/- 0.5 l x min(-1) x %(-1) vs. morphine = 0.9 +/- 0.5 l x min(-1) x %(-1)). Weight, lean body mass, body surface area, and calculated fat mass differed between the sexes, but their inclusion in the analysis as a covariate revealed no influence on the differences between men and women in morphine-induced changes. Conclusions In both sexes, morphine affects ventilatory control. However, we observed quantitative and qualitative differences between men and women in the way morphine affected the ventilatory responses to carbon dioxide and oxygen. Possible mechanisms for the observed sex differences in the respiratory pharmacology of morphine are discussed.

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Sallam, Hanaa S., and Jiande D. Z. Chen. "The Prokinetic Face of Ghrelin." International Journal of Peptides 2010 (February 10, 2010): 1–11. http://dx.doi.org/10.1155/2010/493614.

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This review evaluated published data regarding the effects of ghrelin on GI motility using the PubMed database for English articles from 1999 to September 2009. Our strategy was to combine all available information from previous literature, in order to provide a complete structured review on the prokinetic properties of exogenous ghrelin and its potential use for treatment of various GI dysmotility ailments. We classified the literature into two major groups, depending on whether studies were done in health or in disease. We sub-classified the studies into stomach, small intestinal and colon studies, and broke them down further into studies done in vitro, in vivo (animals) and in humans. Further more, the reviewed studies were presented in a chronological order to guide the readers across the scientific advances in the field. The review shows evidences that ghrelin and its (receptor) agonists possess a strong prokinetic potential to serve in the treatment of diabetic, neurogenic or idiopathic gastroparesis and possibly, chemotherapy-associated dyspepsia, postoperative, septic or post-burn ileus, opiate-induced bowel dysfunction and chronic idiopathic constipation. Further research is necessary to close the gap in knowledge about the effect of ghrelin on the human intestines in health and disease.

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Dissertations / Theses on the topic "Opiate effects on humans"

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Hand, C. W. "Studies on the human pharmacology of opiates." Thesis, University of Oxford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.379946.

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Blackburn, Thomas P. "Renal, vascular and endocrine effects of Kappa opioid agonists." Thesis, University of Manchester, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.237570.

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Park, Sung Jun. "Social facilitation effects of virtual humans." Thesis, Available online, Georgia Institute of Technology, 2006, 2006. http://etd.gatech.edu/theses/available/etd-07102006-132005/.

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Gold, Ann Elizabeth. "Cerebral effects of hypoglycaemia in humans." Thesis, University of Edinburgh, 1994. http://hdl.handle.net/1842/19801.

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The effect of acute insulin-induced hypoglycaemia on cognitive function in humans was examined. The principal studies were performed: i) - to examine whether cerebral adaptation to acute neuroglycopenia occurs in normal subjects, ii) - to examine whether the degree of impairment of cognitive function during acute neuroglycopenia is related to the level of cognitive ability of the subject and iii) - to establish whether patients with insulin-treated diabetes, who have impaired awareness of hypoglycaemia exhibit a more severe degree of cognitive impairment during modest hypoglycaemia. In addition to cognitive function testing in patients with impaired awareness, studies of cerebral blood flow during hypoglycaemia were undertaken using Single Photon Emission Tomography (SPET). Mood changes during acute hypoglycaemia were also examined in non-diabetic subjects. During 60 minutes of exposure to moderate hypoglycaemia there was no evidence of short term cerebral adaptation to neuroglycopenia in normal subjects. Cognitive ability did appear to have some influence on cognitive dysfunction during hypoglycaemia: those subjects of lower cognitive ability appeared to suffer a smaller degree of cognitive dysfunction during hypoglycaemia. During acute hypoglycaemia subjects were observed to have marked changes in mood: there was a decrease in hedonic tone, an increase in tense arousal and a decrease in energetic arousal. Diabetic patients with impaired awareness of hypoglycaemia suffered greater and more prolonged cognitive dysfunction during hypoglycaemia than patients with normal awareness.

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Hall, Chris. "An investigation of the effects of opiate withdrawal syndrome on interrogative suggestibility." Thesis, University of Surrey, 1998. http://epubs.surrey.ac.uk/901/.

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Yubero, Lahoz Samanta 1985. "MDMA pharmacology in humans and serotonergic effects." Doctoral thesis, Universitat Pompeu Fabra, 2013. http://hdl.handle.net/10803/145481.

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3,4-methylenedioxymethamphetamine (MDMA, ecstasy) is one of the most abused recreational drugs in the world. It has been extensively reported that this drug inhibits its own metabolism by inhibiting a polymorphic liver enzyme, CYP2D6, which is responsible for the clearance of one quarter of drugs used in therapeutics. This phenomenon has important clinical implications, since MDMA users display a higher prevalence of psychopathology, particularly of mood disorders, compared to control population. Importantly, these psychiatric diseases are treated with drugs most of them substrate of this enzyme. In addition, it is not elucidated how MDMA is still metabolically cleared even after repeated drug doses. Therefore, the first part of this thesis was focused on studying the metabolic autoinhibition by MDMA assessing several liver enzyme activities in men and women. Although MDMA pharmacology is well established, is it still not clear which is the mechanism of action of the drug. MDMA interacts with the serotonergic system at several levels, but nowadays is technically difficult to study the serotonergic function in living human brain. Imaging methods are limited by a number of factors. Thus, it would be advantageous to have a reliable peripheral index of the serotonergic activity in the blood. The second part of this thesis presents the development of several approaches aimed to assess whether the serotonin transporter in platelets can be used as a peripheral biomarker for central serotonergic activity, and determine if it plays any role in drug mechanism of action.
La 3,4-metilendioximetanfetamina (MDMA, èxtasi) és una de les drogues més consumides al món. Aquesta droga inhibeix el seu propi metabolisme, inhibint un enzim polimòrfic del fetge, el CYP2D6, que és el responsable de l’eliminació d’una quarta part dels medicaments. Aquest fet té implicacions clíniques rellevants, ja que els consumidors de MDMA presenten una prevalença de psicopatologia més alta respecte a la població no consumidora, i moltes de la patologies psiquiàtriques es tracten amb fàrmacs substrats d’aquest enzim. A més, encara no s’ha discernit com aquesta droga pot ser eliminada de l’organisme, inclús després d’haver-ne consumit dosis de manera repetida. Així doncs, la primera part d’aquesta tesi es centra en estudiar l’autoinhibició de la MDMA determinant l’activitat de diferents enzims del fetge, en homes i dones. Encara que la farmacologia de la MDMA està descrita a fons, no està del tot clar quin és el seu mecanisme d’acció. La MDMA interactua amb el sistema serotonèrgic de diverses maneres, però avui en dia és molt difícil estudiar tècnicament el sistema serotonèrgic en el cervell humà. Les tècniques d’imatge estan limitades per molts factors, i per tant, seria molt útil tenir un índex perifèric a la sang de l’activitat serotonèrgica al sistema nerviós central. La segona part d’aquest tesi s’enfoca en el desenvolupament de tècniques per determinar a diferents nivells si el transportador de la serotonina a les plaquetes podria ser un bon biomarcador perifèric de la seva activitat al cervell, i d’aquesta manera veure si el sistema serotonèrgic està implicat en el mecanisme d’acció de la MDMA.

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Kingsley, Michael Ian Charles. "Effects of phosphatidylserine supplementation on exercising humans." Thesis, Swansea University, 2006. https://cronfa.swan.ac.uk/Record/cronfa42268.

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The aim of this research was to investigate the effects of oral soy-derived phosphatidylserine (S-PtdSer) supplementation on the physiological responses to exercise, exercise performance/capacity and recovery following exercise. The results from this research provide evidence that short-term supplementation with 750 mg.d-1 S-PtdSer: 1. does not influence concentrations of cortisol or adrenocorticosteroid hormone in the circulation in young active male subjects. These data suggest that the current supplementation regime does not affect exercise-induced changes in the hypothalamo-pituitary-adrenal (HPA) axis. 2. is ineffective in attenuating the deleterious effects of exercise on perceived soreness, muscle damage, inflammation and oxidative stress in young active male subjects following eccentric exercise with a relatively low metabolic demand. 3. improved exercise capacity following intermittent cycling and tended to improve performance during prolonged intermittent running. These findings suggest that S-PtdSer might possess potential ergogenic properties. However, the mechanism(s) responsible for these findings remain to be determined.

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Spreekmeester, Emma S. "The effects of specific opiate receptor antagonists on the habituation of novelty-induced analgesia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ29791.pdf.

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Watson, Anthony. "The psychopharmacological effects of blackcurrant phytochemicals in humans." Thesis, Northumbria University, 2014. http://nrl.northumbria.ac.uk/34139/.

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Self-medication of plant based foods and food extracts which have ostensible therapeutic benefits has considerably increased within non-clinical populations over the last two decades. The overarching reason for this rise in consumption is to improve health and cognitive performance. One such group of foods are flavonoid-rich berry fruits. In the literature, there has been a recent rise in evidence showing that the consumption of flavonoid-rich berries can modulate aspects of behaviour, especially memory, in animal models and in aged humans. Physiological parameters such as blood-flow and glucose levels; and biological mechanisms such as monoamine oxidase (MAO) activity and nitric oxide (NO) synthesis, which have the potential to impact human behaviour, are also shown to be manipulated by flavonoid compounds. The dark purple fruits of the blackcurrant (Ribes nigrum) are naturally high in flavonoids, however no literature assessing cognitive effects of their consumption is available. The main focus of this thesis was to assess the impact of standardised flavonoid-rich blackcurrant extracts upon cognitive performance and mood in healthy human participants. Two extracts were examined within the thesis, a freeze dried powder extract fortified to contain 30% anthocyanins (DelcyanTM) and a fresh from frozen cold pressed juice extract (Blackadder cultivar, Plant and Food Research Ltd). Utilising a series of randomised, between subjects, double bind studies, measures of memory, attention, executive function and psychomotor performance were implemented during the course of the thesis at various post-dose time points. Throughout the investigational chapters of the thesis, physiological parameters and potential mechanisms driving any behavioural changes were measured. Such measures included measures of central and peripheral haemodynamics, MAO inhibition, monoaminergic tone, prolactin secretion and post-prandial glucose profiles. Single doses of each of the blackcurrant extracts used in this thesis yielded positive results with effects of post-harvest extraction technique evident. Although no clear pattern of behavioural modulation was found after consumption of the blackcurrant extracts, there was some evidence to show increases in attention processes during cognitively demanding paradigms in young participants. No positive effects were evident upon any other cognitive paradigm. Physiological effects of acute blackcurrant supplementation included a modulation of post-prandial glucose profile and hemispheric dependent modulations of cerebral blood flow. Most strikingly, a pharmaceutical level inhibition of both monoamine oxidase isoforms and reductions in blood plasma prolactin were found. The findings of this thesis may have implications for enhancement of cognitive performance, attenuation in natural cognitive decline over the lifespan, and potentially, clinical applications in the treatment of neurological diseases.

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Upreti, Rita. "Metabolic effects of 5α-reductase inhibition in humans." Thesis, University of Edinburgh, 2013. http://hdl.handle.net/1842/11745.

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5α-reductases (5αRs) catalyse reduction of 4-pregnene steroids, most notably the androgen testosterone to its more potent metabolite dihydrotestosterone (DHT). Well-characterised isozymes of 5αR are designated 5αR1 and 5αR2. Inhibitors of 5αR, finasteride (a 5αR2 inhibitor) and dutasteride (a dual 5αR1 and 5αR2 inhibitor), are utilised in conditions where a reduction in androgen action is desired, including benign prostatic hyperplasia. Although 5αR2 is predominantly expressed in reproductive tissues, both isozymes, but particularly 5αR1, are expressed in metabolic tissues including liver and adipose and both metabolise glucocorticoids as well as androgens; therefore inhibition of 5αR may have consequences for metabolic health. This thesis addresses the hypotheses that 5αR1 inhibition with dutasteride decreases insulin sensitivity and causes dysregulation of the HPA axis in humans. Metabolism and the HPA axis were studied in men prior to and following 3 months of dutasteride (0.5 mg daily; n=16), finasteride (5 mg daily; n=16) or control (tamsulosin MR; 0.4 mg daily; n=14). Glucose disposal during hyperinsulinaemia was the primary endpoint, measured during a hyperinsulinaemic euglycaemic clamp, with d2-glucose and d5-glycerol tracers. Peripheral insulin sensitivity for both glucose uptake and NEFA suppression decreased with dutasteride versus both finasteride and control, while hepatic insulin sensitivity was preserved. Body fat increased with dutasteride, though was not accompanied by changes in metabolic or inflammatory gene transcript abundance in subcutaneous adipose biopsies, nor any differences in abdominal adipose depots on post-treatment MRI. Subtle dysregulation of the HPA axis was evident with both 5αR inhibitors, though to a greater degree with dutasteride and changes were largely compensated for. In support of this study, this thesis also describes the development, validation and application of two novel liquid chromatography tandem mass spectrometry assays; establishing compliance by measuring serum drug levels, and demonstrating effects of 5αR inhibitors on androgen metabolism and adrenal steroidogenesis by measurement of testosterone, DHT and androstenedione. In conclusion, 5αR1 inhibition with dutasteride, but not finasteride, induces peripheral insulin resistance and increases body fat. Findings presented may have important implications for patients prescribed dutasteride for benign prostatic hyperplasia.

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Books on the topic "Opiate effects on humans"

1

Luis, Stinus, and Koob George F, eds. Neurobiological mechanisms of opiate withdrawal. New York: Springer, 1996.

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Maldonado, Rafael. Neurobiological mechanisms of opiate withdrawal. New York: Springer, 1996.

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Mast, Barbara J. van der. The immunomodulating effects of blood transfusion in humans. Leiden: University of Leiden, 1998.

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Sindermann, Carl J. Coastal pollution: Effects on living resources and humans. Boca Raton, FL: CRC/Taylor & Francis, 2006.

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Woodward, Kevin N. Toxicological effects of veterinary medicinal products in humans. Cambridge: Royal Society of Chemistry, 2013.

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Ocean pollution: Effects on living resources and humans. Boca Raton: CRC Press, 1996.

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Hunter, Judith. Segmental effects of spinal cord stimulators in humans. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1992.

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J, Sindermann Carl, ed. Coastal pollution: Effects on living resources and humans. Boca Raton, FL: Taylor & Francis, 2005.

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Jackson, Anne. The effects of kappa opiate agonists on feeding and related activities in the rat. Birmingham: University of Birmingham, 1986.

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Prenn, U. L. Introduction to biological radiation effects: An overview of terrestrial and space radiation effects on humans. 2nd ed. Carlsborg, Wash: Systems, 1994.

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Book chapters on the topic "Opiate effects on humans"

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Nyland, Susan Bell, Steven Specter, Jeong Im-Sin, and Kenneth E. Ugen. "Opiate Effects on In Vitro Human Retroviral Infection." In Advances in Experimental Medicine and Biology, 91–100. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5347-2_11.

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Mendelson, Jack H., and Nancy K. Mello. "Endocrine Effects of Opioid Antagonists." In Opiate Receptors and Antagonists, 581–604. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-197-0_31.

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Gehrke, Brenda J., and GehrkeToni S. Shippenberg. "Effects of Opioid Antagonists on the Abuse-Related Effects of Psychomotor Stimulants and Nicotine." In Opiate Receptors and Antagonists, 273–98. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-197-0_15.

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Bodnar, Richard J. "Preclinical Effects of Opioid Antagonists on Feeding and Appetite." In Opiate Receptors and Antagonists, 387–406. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-197-0_20.

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Kanas, Nick. "Positive Effects of Space." In Humans in Space, 61–67. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-18869-0_6.

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Smith, C. W. "Electromagnetic Effects in Humans." In Biological Coherence and Response to External Stimuli, 205–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73309-3_12.

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Petrakis, Ismene. "Pharmacotherapeutic Effects of Opioid Antagonists in Alcohol-Abusing Patients with Schizophrenia." In Opiate Receptors and Antagonists, 473–83. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-197-0_25.

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Fox, Susan H., Tom H. Johnston, and Jonathan M. Brotchie. "Effects of Opioid Antagonists on l-DOPA-Induced Dyskinesia in Parkinson's Disease." In Opiate Receptors and Antagonists, 569–80. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-197-0_30.

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Somerville, Keith. "Domestication, settlement and the effects on lions." In Humans and Lions, 29–52. Milton Park, Abingdon, Oxon ; New York, NY : Routledge, 2019. | Series: Routledge environmental humanities: Routledge, 2019. http://dx.doi.org/10.4324/9781315151182-3.

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Negus, S. Stevens. "Opioid Antagonist Effects in Animal Models Related to Opioid Abuse: Drug Discrimination and Drug Self-Administration." In Opiate Receptors and Antagonists, 201–26. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-197-0_11.

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Conference papers on the topic "Opiate effects on humans"

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Larrabee, David A. "Teaching Electromagnetic Radiation effects on Humans." In 2006 International Conference on Software in Telecommunications and Computer Networks. IEEE, 2006. http://dx.doi.org/10.1109/softcom.2006.329716.

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Yazicioglu, Suheyla. "TOXIC EFFECTS OF PESTICIDES TO HUMANS AND ENVIROMENT." In 13th SGEM GeoConference on ECOLOGY, ECONOMICS, EDUCATION AND LEGISLATION. Stef92 Technology, 2013. http://dx.doi.org/10.5593/sgem2013/be5.v1/s20.145.

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Radin, Dean I. "Psychophysiological Evidence of Possible Retrocausal Effects in Humans." In FRONTIERS OF TIME: Retrocausation - Experiment and Theory. AIP, 2006. http://dx.doi.org/10.1063/1.2388755.

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Destephe, Matthieu, Martim Brandao, Tatsuhiro Kishi, Massimiliano Zecca, Kenji Hashimoto, and Atsuo Takanishi. "Emotional gait: Effects on humans' perception of humanoid robots." In 2014 RO-MAN: The 23rd IEEE International Symposium on Robot and Human Interactive Communication. IEEE, 2014. http://dx.doi.org/10.1109/roman.2014.6926263.

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Schmutzler, Luis O. F. "A Trap for Humans: The Challenges of the “Guillotine Effects”." In 1995 SAE Brasil. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1995. http://dx.doi.org/10.4271/952207.

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IWASHITA, Motoki, and Daisuke KATAGAMI. "Psychological Effects of Compliment Expressions by Communication Robots on Humans." In 2020 International Joint Conference on Neural Networks (IJCNN). IEEE, 2020. http://dx.doi.org/10.1109/ijcnn48605.2020.9206898.

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Van Loock, Walter. "Elementary effects in humans exposed to electromagnetic fields and radiation." In 2009 5th Asia-Pacific Conference on Environmental Electromagnetics (CEEM 2009). IEEE, 2009. http://dx.doi.org/10.1109/ceem.2009.5304780.

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Anin, Johnson, Hongzhi Guo, and Kyo D. Song. "Simulations of radiation effects of mobile cell phone on humans." In Nano-, Bio-, Info-Tech Sensors and 3D Systems, edited by Jaehwan Kim. SPIE, 2020. http://dx.doi.org/10.1117/12.2557521.

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Stamper, David A., David J. Lund, Jerome W. Molchany, and Bruce E. Stuck. "Effects of bilateral and unilateral laser ocular exposure in humans." In Biomedical Optics 2003, edited by Bruce E. Stuck and Michael Belkin. SPIE, 2003. http://dx.doi.org/10.1117/12.488648.

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Afshan, Amber, Jody Kreiman, and Abeer Alwan. "Speaker Discrimination in Humans and Machines: Effects of Speaking Style Variability." In Interspeech 2020. ISCA: ISCA, 2020. http://dx.doi.org/10.21437/interspeech.2020-3004.

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Reports on the topic "Opiate effects on humans"

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Vakali, Eleni, Dimitrios Rigopoulos, Andres Carrillo, Andreas Flouris, and Petros Dinas. The effects of alpha-lipoic acid supplementation on diabetic nephropathy in humans. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0095.

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Graham, C., and H. Cohen. Immunological and biochemical effects of 60-Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/5077901.

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Kues, Henry. Effects of Microwave Radiation on Humans. Monkeys Exposed to 1.25 GHZ pulsed Microwaves. Fort Belvoir, VA: Defense Technical Information Center, March 1992. http://dx.doi.org/10.21236/ada249997.

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Fotopoulos, S., and C. Graham. Immunological and biochemical effects of 60 Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), February 1985. http://dx.doi.org/10.2172/6831463.

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Graham, C., and H. Cohen. Immunological and biochemical effects of 60-Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/6831450.

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Graham, C., and S. Fotopoulos. Immunological and biochemical effects of 60-Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), December 1985. http://dx.doi.org/10.2172/7004201.

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Graham, C., and H. Cohen. Immunological and biochemical effects of 60-Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), July 1986. http://dx.doi.org/10.2172/7004224.

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Graham, C., and H. Cohen. Immunological and biochemical effects of 60-Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), May 1987. http://dx.doi.org/10.2172/7004207.

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Graham, C., and H. Cohen. Immunological and biochemical effects of 60-Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), April 1986. http://dx.doi.org/10.2172/7189405.

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Graham, C., and H. Cohen. Immunological and biochemical effects of 60-Hz electric and magnetic fields in humans. Office of Scientific and Technical Information (OSTI), April 1987. http://dx.doi.org/10.2172/7098317.

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