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Journal articles on the topic 'Ontogeny'

To see the other types of publications on this topic, follow the link: Ontogeny.

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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1

Gabryel, Nasser Suleiman. "Ontogeny." Anuac 3, no. 1 (June 28, 2015): 75–84. http://dx.doi.org/10.7340/anuac2239-625x-152.

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The ontogeny of the anthropology consists in returning to the history of the discipline, of its formation. It involves studying its institutional and intellectual development.The philosophical anthropology is an essential matrix of the current anthropology; its study allows us to understand the historic shape of intellectualization of the concepts and the work of definition which notably begins in the XVI century with Montaigne and in the XVIII century with Kant. As a consequence, the philosophy was the armature of the ontogeny of the anthropology and allowed to propose a universal frame of analysis on the world and men.

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2

Brown, Caleb, Robert Holmes, and Phillip Currie. "A subadult individual of Styracosaurus albertensis (Ornithischia: Ceratopsidae) with comments on ontogeny and intraspecific variation in Styracosaurus and Centrosaurus." Vertebrate Anatomy Morphology Palaeontology 8 (May 11, 2020): 67–95. http://dx.doi.org/10.18435/vamp29361.

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Styracosaurus albertensis is an iconic centrosaurine horned dinosaur from the Campanian of Alberta, Canada, known for its large spike-like parietal processes. Although described over 100 years ago, subsequent discoveries were rare the last few decades, during which time several new skulls, skeletons, and bonebeds were found. Here we described an immature individual, the smallest known for the species, represented by a complete skull and fragmentary skeleton. Although ~80% maximum size, it possesses a suite of characters associated with immaturity, and is regarded as a subadult. The ornamentation is characterized by a small, recurved, but fused nasal horncore; low, rounded postorbital horncores; and short, triangular, and flat parietal processes. Using this specimen, and additional skulls and bonebed material, the cranial ontogeny of Styracosaurus is described, and compared to Centrosaurus. Styracosaurus shows a similar early ontogeny of the nasal horncore, starting thin, recurved, and unfused, but retains the recurved morphology into large adult size, and never develops the procurved morphology common in Centrosaurus. The postorbital horncores of Styracosaurus are lower and more rounded than those of Centrosaurus throughout ontogeny, and show greater resorption later in ontogeney. The length and thickness of the parietal processes increase drastically through ontogeny, but their position and orientation are static across the size series. Several diagnostic Styracosaurus albertensis specimens now preserve medially orientated P3 spikes, causing issues for the diagnosis of S. ovatus. Variability in parietal ornamentation, either expression of P1 and P2 parietal processes, or other cranial ornamentations, does not appear to correlate with stratigraphy.

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3

Nadel, B., S. Tehranchi, and A. J. Feeney. "Coding end processing is similar throughout ontogeny." Journal of Immunology 154, no. 12 (June 15, 1995): 6430–36. http://dx.doi.org/10.4049/jimmunol.154.12.6430.

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Abstract During the recombination process, extensive processing of the coding ends provides tremendous potential diversity to the joint of any two gene segments. However, the diversity of the newborn B and T cell repertoires is greatly reduced compared with that of the adult. At the mechanistic level, this difference is primarily due to the absence of terminal deoxynucleotidyltransferase expression until the first week after birth. Additionally, one direct consequence of the lack of N regions early in ontogeny is the more frequent occurrence of homology-directed recombination, reducing even further the potential of diversity. Other enzymatic factors could also contribute to this ontogenic difference. However, the use of the homology-directed recombination pathway early in life obscures the analysis of the coding end processing. In this study we compared the coding end processing throughout ontogeny, in normal and terminal deoxynucleotidyltransferase -/- mice in the presence of minimal homology-directed recombination. The analysis of partial D-J joints allowed us to avoid potential bias by early selection events. Our results show that the extent of nucleotide deletion of a given end is consistent throughout ontogeny in the presence or absence of terminal deoxynucleotidyltransferase. However, a distinctive processing pattern is observed for each coding end.

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4

Bunn, H. Franklin. "Reversing Ontogeny." New England Journal of Medicine 328, no. 2 (January 14, 1993): 129–31. http://dx.doi.org/10.1056/nejm199301143280210.

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5

Potter, V. "Blocked ontogeny." Science 237, no. 4818 (August 28, 1987): 964. http://dx.doi.org/10.1126/science.3616628.

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6

Ng, K. W. "Osteoblast ontogeny." Bone 27, no. 4 (October 2000): 8. http://dx.doi.org/10.1016/s8756-3282(00)80023-x.

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7

BLACKMORE, STEPHEN. "CELLULAR ONTOGENY." Cladistics 2, no. 3 (June 1986): 358–62. http://dx.doi.org/10.1111/j.1096-0031.1986.tb00458.x.

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8

Bennett, S. Christopher. "Ontogeny andArchaeopteryx." Journal of Vertebrate Paleontology 28, no. 2 (June 12, 2008): 535–42. http://dx.doi.org/10.1671/0272-4634(2008)28[535:oaa]2.0.co;2.

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9

Buckley, William R. "Computational Ontogeny." Biological Theory 3, no. 1 (March 2008): 3–6. http://dx.doi.org/10.1162/biot.2008.3.1.3.

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10

Bovet, Pierre. "Functional ontogeny." Behavioural Processes 14, no. 2 (April 1987): 229–30. http://dx.doi.org/10.1016/0376-6357(87)90048-9.

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11

Nanthakumar, N. N., and S. J. Henning. "Distinguishing normal and glucocorticoid-induced maturation of intestine using bromodeoxyuridine." American Journal of Physiology-Gastrointestinal and Liver Physiology 268, no. 1 (January 1, 1995): G139—G145. http://dx.doi.org/10.1152/ajpgi.1995.268.1.g139.

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Exogenous glucocorticoids administered during the first two postnatal weeks are capable of eliciting precocious maturation of the rat intestine. However, it is not known whether this represents an alternative developmental pathway or is essentially an advancement of normal ontogeny. The goal of the present study was to address this question using the thymidine analogue 5-bromo-2'-deoxyuridine (BrdU), which is known to selectively inhibit differentiation in a number of tissues. Intestinal development was assessed by following changes in sucrase, trehalase, glucoamylase, and lactase activities. The first experiment assessed whether BrdU has any influence on the cellular differentiation that occurs continuously along the crypt-villus axis. After administration of BrdU to suckling and mature animals, there was no effect on lactase and sucrase activities, respectively. Thus BrdU does not inhibit crypt-villus differentiation in either the suckling or mature jejunum. In the second experiment, dexamethasone was used to induce precocious maturation in the rat jejunum on day 10. BrdU treatment significantly inhibited glucocorticoid-induced elevation of sucrase, trehalase, and glucoamylase but had no effect on the lactase activity. In contrast, treatment with BrdU during normal development significantly accelerated the ontogenic rise of sucrase and trehalase as well as the ontogenic decline of lactase. The acceleration of development was also seen in adrenalectomized rats, indicating that it is the glucocorticoid-independent component of normal intestinal ontogeny that is activated by BrdU. The opposite effect of BrdU on glucocorticoid-induced precocious maturation suggests that such maturation involves different molecular mediators than normal ontogeny.

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12

Kovtun, M. F., and H. V. Sheverdyukova. "Ontogeny And Phylogeny. To The Problem Of The Relation Of Individual And Historical Development In Organisms." Vestnik Zoologii 49, no. 4 (August 1, 2015): 291–98. http://dx.doi.org/10.1515/vzoo-2015-0030.

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Abstract The theory of filembriogenesis is only an introduction to the problem’s development of ontogeny’ and phylogeny’ relation (hereinafter — «relation»). Discussions as to whether ontogeny creates phylogeny, or vice versa, are devoid of meaning. The opinion of O. Hertwig (Hertwig, 1906) that the ontogeny and phylogeny are two parallel and independent developmental processes is valid only in the first part; thesis about independence distorts the essence of «relation.» According to the authors, one of the essential characteristics of the «relation» is that ontogeny gives the material for phylogeny, and phylogeny renews ontogeny, leading away ontogeny from inbreeding; that ontogeny ensures the life continuity and phylogeny — its differentiation, that is, creates biodiversity; that ontogeny and phylogeny can exist and function only in conjunction or in parallel, changing places (in terms of priority) in the life evolution.

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13

Palosaari, Jedidiah J. "Ontogeny vs. Phylogeny." Science News 156, no. 4 (July 24, 1999): 51. http://dx.doi.org/10.2307/4011639.

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14

Takeuchi, Satoshi, and Masutaka Furue. "Dendritic Cells—Ontogeny—." Allergology International 56, no. 3 (2007): 215–23. http://dx.doi.org/10.2332/allergolint.r-07-149.

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15

Crowell, David H., Lee J. Brooks, Michael Corwin, Sally Davidson-Ward, Carl E. Hunt, Linda E. Kapuniai, Michael R. Neuman, et al. "Ontogeny of Arousal." Journal of Clinical Neurophysiology 21, no. 4 (July 2004): 290–300. http://dx.doi.org/10.1097/01.wnp.0000141754.03598.dc.

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16

Palis, James. "Ontogeny of erythropoiesis." Current Opinion in Hematology 15, no. 3 (May 2008): 155–61. http://dx.doi.org/10.1097/moh.0b013e3282f97ae1.

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17

Bruce Rosenstock. "Phylogeny Recapitulates Ontogeny:." Soundings: An Interdisciplinary Journal 96, no. 1 (2013): 25. http://dx.doi.org/10.5325/soundings.96.1.0025.

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18

Naimark, Elena. "Ontogeny of Agnostida." Palaeoworld 15, no. 3-4 (August 2006): 315–27. http://dx.doi.org/10.1016/j.palwor.2006.10.011.

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19

Zelazo, Philip David, and J. Steven Reznick. "Ontogeny and intentionality." Behavioral and Brain Sciences 13, no. 4 (December 1990): 631–32. http://dx.doi.org/10.1017/s0140525x0008064x.

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20

Cramer, Steven C., and Michael Chopp. "Recovery recapitulates ontogeny." Trends in Neurosciences 23, no. 6 (June 2000): 265–71. http://dx.doi.org/10.1016/s0166-2236(00)01562-9.

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21

Love, Alan C., and Michael Travisano. "Microbes modeling ontogeny." Biology & Philosophy 28, no. 2 (February 12, 2013): 161–88. http://dx.doi.org/10.1007/s10539-013-9363-5.

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22

Kluge, A. G., and R. E. Strauss. "Ontogeny and Systematics." Annual Review of Ecology and Systematics 16, no. 1 (November 1985): 247–68. http://dx.doi.org/10.1146/annurev.es.16.110185.001335.

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23

NELSON, GARETH. "OUTGROUPS AND ONTOGENY." Cladistics 1, no. 1 (December 1985): 29–45. http://dx.doi.org/10.1111/j.1096-0031.1985.tb00409.x.

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24

Deutsch, Carol. "Potassium Channel Ontogeny." Annual Review of Physiology 64, no. 1 (March 2002): 19–46. http://dx.doi.org/10.1146/annurev.physiol.64.081501.155934.

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25

Hamburger, V. "Ontogeny of neuroembryology." Journal of Neuroscience 8, no. 10 (October 1, 1988): 3535–40. http://dx.doi.org/10.1523/jneurosci.08-10-03535.1988.

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26

Dzierzak, Elaine. "Ontogeny of Hematopoiesis." Blood 122, no. 21 (November 15, 2013): SCI—4—SCI—4. http://dx.doi.org/10.1182/blood.v122.21.sci-4.sci-4.

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Abstract The current challenge in hematopoietic transplantation and regeneration therapies is acquiring and/or producing a reliable and plentiful source of hematopoietic stem cells (HSCs). Given that HSCs from bone marrow, peripheral, or umbilical cord blood undergo only limited/no expansion ex vivo, there is a high interest in understanding how the adult cohort of multipotent self-renewing HSCs are generated and expanded during embryonic development. The development of HSCs in vertebrate embryos begins in the major vasculature. HSCs are generated in a short window of developmental time starting at embryonic day E10.5 until E12 in the mouse embryo, and from gestational weeks four to six in the human embryo. The first HSCs, which are as potent as bone marrow HSCs in transplantation procedures, are generated in the aorta-gonad-mesonephros (AGM) region. HSCs are found in the major vasculature – aorta, vitelline artery, and umbilical artery – subsequent to the appearance of hematopoietic cell clusters closely associated with the lumenal walls of these vessels. The relationship of HSCs to these clusters and the identification of the precursors to HSCs have been recently established through genetic, phenotypic, and real-time imaging studies. Remarkably, HSCs and hematopoietic progenitors arise directly from a subset of endothelial cells (hemogenic endothelial cells) in a natural transdifferentiation event. They are made through a process called endothelial to hematopoietic cell transition (EHT). EHT and HSC generation is in part regulated through ventral-derived developmental signals and a group of pivotal (core) transcription factors, including Runx1 and Gata2. Conditional knockout strategies show that these transcription factors are required for the generation of vascular hematopoietic clusters and HSCs, suggesting a role in hematopoietic fate induction and/or cell expansion. Interestingly, whereas both Runx1 and Gata2 are required for HSC generation, only Gata2 remains essential in HSCs after their production. We are profiling hemogenic endothelial and HSCs by RNA sequencing so as to understand the complete genetic program that leads to generation of HSCs. These results will be discussed in the context of developmental signaling pathways (BMP4, Hedgehog, etc.) that appear to impact HSC generation and expansion, and the localized dynamic expression and function of Gata2 and Runx1 in vascular endothelial and hematopoietic cluster cells. Disclosures: No relevant conflicts of interest to declare.

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27

Vermeij, Geerat J. "Gould’s intellectual ontogeny." Journal of Biosciences 27, no. 5 (September 2002): 451–52. http://dx.doi.org/10.1007/bf02705038.

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28

Bowers, W. E., and E. M. Goodell. "Dendritic cell ontogeny." Research in Immunology 140, no. 9 (January 1989): 880–83. http://dx.doi.org/10.1016/0923-2494(89)90047-3.

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29

Taylor, Amanda, and Kevin Burns. "Epiphyte community development throughout tree ontogeny: an island ontogeny framework." Journal of Vegetation Science 26, no. 5 (April 11, 2015): 902–10. http://dx.doi.org/10.1111/jvs.12289.

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30

Rivera-Gaxiola, Maritza, and Annette Karmiloff-Smith. "It's a far cry from speech to language." Behavioral and Brain Sciences 19, no. 4 (December 1996): 645–46. http://dx.doi.org/10.1017/s0140525x00043454.

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AbstractWe agree with Müller's epigenetic view of evolution and ontogeny and applaud his multilevel perspective. With him, we stress the importance in ontogeny of progressive specialisation rather than prewired structures. However, we argue that he slips from “speech” to “language” and that, in seeking homologies, these two levels need to be kept separate in the analysis of evolution and ontogeny.

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31

Shen, Defeng, Ting Ting Xiao, Robin van Velzen, Olga Kulikova, Xiaoyun Gong, René Geurts, Katharina Pawlowski, and Ton Bisseling. "A Homeotic Mutation Changes Legume Nodule Ontogeny into Actinorhizal-Type Ontogeny." Plant Cell 32, no. 6 (April 10, 2020): 1868–85. http://dx.doi.org/10.1105/tpc.19.00739.

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32

Renosi, Florian, Anne Roggy, Ambre Giguelay, Lou Soret, Pierre-Julien Viailly, Meyling Cheok, Sabeha Biichle, et al. "Transcriptomic and genomic heterogeneity in blastic plasmacytoid dendritic cell neoplasms: from ontogeny to oncogenesis." Blood Advances 5, no. 5 (March 9, 2021): 1540–51. http://dx.doi.org/10.1182/bloodadvances.2020003359.

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Abstract Oncogenesis and ontogeny of blastic plasmacytoid dendritic cell neoplasm (BPDCN) remain uncertain, between canonical plasmacytoid dendritic cells (pDCs) and AXL+ SIGLEC6+ DCs (AS-DCs). We compared 12 BPDCN to 164 acute leukemia by Affymetrix HG-U133 Plus 2.0 arrays: BPDCN were closer to B-cell acute lymphoblastic leukemia (ALL), with enrichment in pDC, B-cell signatures, vesicular transport, deubiquitination pathways, and AS-DC signatures, but only in some cases. Importantly, 1 T-cell ALL clustered with BPDCN, with compatible morphology, immunophenotype (cCD3+ sCD3− CD123+ cTCL1+ CD304+), and genetics. Many oncogenetic pathways are deregulated in BPDCN compared with normal pDC, such as cell-cycle kinases, and importantly, the transcription factor SOX4, involved in B ontogeny, pDC ontogeny, and cancer cell invasion. High-throughput sequencing (HaloPlex) showed myeloid mutations (TET2, 62%; ASXL1, 46%; ZRSR2, 31%) associated with lymphoid mutations (IKZF1), whereas single-nucleotide polymorphism (SNP) array (Affymetrix SNP array 6.0) revealed frequent losses (mean: 9 per patient) involving key hematological oncogenes (RB1, IKZF1/2/3, ETV6, NR3C1, CDKN2A/B, TP53) and immune response genes (IFNGR, TGFB, CLEC4C, IFNA cluster). Various markers suggest an AS-DC origin, but not in all patients, and some of these abnormalities are related to the leukemogenesis process, such as the 9p deletion, leading to decreased expression of genes encoding type I interferons. In addition, the AS-DC profile is only found in a subgroup of patients. Overall, the cellular ontogenic origin of BPDCN remains to be characterized, and these results highlight the heterogeneity of BPDCN, with a risk of a diagnostic trap.

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33

Ivanova, T. A., S. V. Morozova, and Y. D. Reuka. "ONTOGENY OF COHERENT SPEECH." Современные проблемы науки и образования (Modern Problems of Science and Education), no. 3 2021 (2021): 66. http://dx.doi.org/10.17513/spno.30818.

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34

Zietlow, Amelia R. "Craniofacial ontogeny in Tylosaurinae." PeerJ 8 (October 20, 2020): e10145. http://dx.doi.org/10.7717/peerj.10145.

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Mosasaurs were large, globally distributed aquatic lizards that lived during the Late Cretaceous. Despite numerous specimens of varying maturity, a detailed growth series has not been proposed for any mosasaur taxon. Two taxa—Tylosaurus proriger and T. kansasensis/nepaeolicus—have robust fossil records with specimens spanning a wide range of sizes and are thus ideal for studying mosasaur ontogeny. Tylosaurus is a genus of particularly large mosasaurs with long, edentulous anterior extensions of the premaxilla and dentary that lived in Europe and North America during the Late Cretaceous. An analysis of growth in Tylosaurus provides an opportunity to test hypotheses of the synonymy of T. kansasensis with T. nepaeolicus, sexual dimorphism, anagenesis, and heterochrony. Fifty-nine hypothetical growth characters were identified, including size-dependent, size-independent, and phylogenetic characters, and quantitative cladistic analysis was used to recover growth series for the two taxa. The results supported the synonymy of T. kansasensis with T. nepaeolicus and that T. kansasensis represent juveniles of T. nepaeolicus. A Spearman rank-order correlation test resulted in a significant correlation between two measures of size (total skull length and quadrate height) and maturity. Eleven growth changes were shared across both species, neither of the ontogram topologies showed evidence of skeletal sexual dimorphism, and a previous hypothesis of paedomorphy in T. proriger was not rejected. Finally, a novel hypothesis of anagenesis in Western Interior Seaway Tylosaurus species, driven by peramorphy, is proposed here.

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35

Frederickson, Joseph A., and Allison R. Tumarkin-Deratzian. "Craniofacial ontogeny inCentrosaurus apertus." PeerJ 2 (February 13, 2014): e252. http://dx.doi.org/10.7717/peerj.252.

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36

Malueg, K. R., S. E. Schlarbaum, E. T. Graham, and R. N. Trigiano. "CORNUS FLORIDA POLLEN ONTOGENY." HortScience 30, no. 3 (June 1995): 440g—441. http://dx.doi.org/10.21273/hortsci.30.3.440g.

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Development of Cornus florida L. pollen was monitored using standard paraffin histological techniques and light microscopy. Terminal buds (putative floral buds) were collected over a 6 weeks from mature landscape trees located on The Univ. of Tennessee Agricultural Campus, Knoxville. Examination of samples taken at 3- to 7-day intervals revealed variations in development representing 1- to 2-week differences between florets in a single inflorescence, florets on the same tree, and florets from different trees. Floral initiation occurred before 19 July in the 2 years of this study. Pollen development followed typical angiosperm stages: tapetal cells were multinucleate, pollen tetrads were tetrahedral, and meiosis occurred late in the developmental period. Pollen grains appeared morphologically mature by early September in both years.

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37

STUTMAN, OSIAS. "Ontogeny of T Cells." Clinics in Immunology and Allergy 5, no. 2 (June 1985): 191–234. http://dx.doi.org/10.1016/s0260-4639(22)00126-8.

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38

Pavlovskaya, O. S., O. V. Il’ina, T. E. Putilina, N. V. Ozolina, E. V. Pradedova, and R. K. Salyaev. "Tonoplast ATPases in ontogeny." Doklady Biological Sciences 418, no. 1 (February 2008): 44–46. http://dx.doi.org/10.1134/s0012496608010158.

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39

Owens, Simon J., S. Blackmore, and R. B. Knox. "Microspores. Evolution and Ontogeny." Kew Bulletin 47, no. 2 (1992): 336. http://dx.doi.org/10.2307/4110681.

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40

Bonal, Claire, and Pedro L. Herrera. "Genes controlling pancreas ontogeny." International Journal of Developmental Biology 52, no. 7 (2008): 823–35. http://dx.doi.org/10.1387/ijdb.072444cb.

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41

Tang, Mimi L. K., and Andrew S. Kemp. "Ontogeny of IL4 production." Pediatric Allergy and Immunology 6, no. 1 (February 1995): 11–19. http://dx.doi.org/10.1111/j.1399-3038.1995.tb00251.x.

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42

Lucivero, G., V. D’Addario, N. Tannoia, A. Dell’Osso, V. Gambatesa, P. L. Lopalco, and G. Cagnazzo. "Ontogeny of Human Lymphocytes." Fetal Diagnosis and Therapy 6, no. 3-4 (1991): 101–6. http://dx.doi.org/10.1159/000263632.

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43

Taylor, Mackenzie L., Patrick J. Hudson, Jolene M. Rigg, Julie N. Strandquist, Julie Schwartz Green, Tara C. Thiemann, and Jeffrey M. Osborn. "Pollen Ontogeny inVictoria(Nymphaeales)." International Journal of Plant Sciences 174, no. 9 (November 2013): 1259–76. http://dx.doi.org/10.1086/673246.

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44

Ekblom, Peter, and Andreas Weller. "Ontogeny of tubulointerstitial cells." Kidney International 39, no. 3 (March 1991): 394–400. http://dx.doi.org/10.1038/ki.1991.51.

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45

Jankowski, M., B. Danalache, D. Wang, P. Bhat, F. Hajjar, M. Marcinkiewicz, J. Paquin, S. M. McCann, and J. Gutkowska. "Oxytocin in cardiac ontogeny." Proceedings of the National Academy of Sciences 101, no. 35 (August 17, 2004): 13074–79. http://dx.doi.org/10.1073/pnas.0405324101.

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46

Margraf, Andreas, Claudia Nussbaum, and Markus Sperandio. "Ontogeny of platelet function." Blood Advances 3, no. 4 (February 26, 2019): 692–703. http://dx.doi.org/10.1182/bloodadvances.2018024372.

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AbstractAlthough the hemostatic potential of adult platelets has been investigated extensively, regulation of platelet function during fetal life is less clear. Recent studies have provided increasing evidence for a developmental control of platelet function during fetal ontogeny. Fetal platelets feature distinct differences in reactive properties compared with adults. These differences very likely reflect a modified hemostatic and homeostatic environment in which platelet hyporeactivity contributes to prevent pathological clot formation on the one hand but still ensures sufficient hemostasis on the other hand. In this review, recent findings on the ontogeny of platelet function and reactivity are summarized, and implications for clinical practice are critically discussed. This includes current platelet-transfusion practice and its potential risk in premature infants and neonates.

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47

Wu, Chi-Guang, and David M. Sylvia. "Spore Ontogeny ofGlomus Globiferum." Mycologia 85, no. 2 (March 1993): 317–22. http://dx.doi.org/10.1080/00275514.1992.12026276.

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48

Visscher, Marty, and Vivek Narendran. "The Ontogeny of Skin." Advances in Wound Care 3, no. 4 (April 2014): 291–303. http://dx.doi.org/10.1089/wound.2013.0467.

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49

Ingold, C. T. "Chlamydospore ontogeny in Itersonilia." Transactions of the British Mycological Society 86, no. 3 (January 1986): 501–3. http://dx.doi.org/10.1016/s0007-1536(86)80200-5.

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50

Kelly, E. J., and S. J. Newell. "Gastric ontogeny: clinical implications." Archives of Disease in Childhood - Fetal and Neonatal Edition 71, no. 2 (September 1, 1994): F136—F141. http://dx.doi.org/10.1136/fn.71.2.f136.

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