Relevant bibliographies by topics / Ocular pathogenesis

Academic literature on the topic 'Ocular pathogenesis'

Author: Grafiati
Published: 10 December 2022
Last updated: 29 January 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Contents

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Ocular pathogenesis.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Ocular pathogenesis"

1

Zhang, Jun, Jingsheng Tuo, Zhongfeng Wang, Aiqin Zhu, Anna Machalińska, and Qin Long. "Pathogenesis of Common Ocular Diseases." Journal of Ophthalmology 2015 (2015): 1–2. http://dx.doi.org/10.1155/2015/734527.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Deepthi, KrishnanNair Geetha, and Solai Ramatchandirane Prabagaran. "Ocular bacterial infections: Pathogenesis and diagnosis." Microbial Pathogenesis 145 (August 2020): 104206. http://dx.doi.org/10.1016/j.micpath.2020.104206.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Pavesio, C. E., and S. Lightman. "Toxoplasma gondii and ocular toxoplasmosis: pathogenesis." British Journal of Ophthalmology 80, no. 12 (December 1, 1996): 1099–107. http://dx.doi.org/10.1136/bjo.80.12.1099.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Holland, Gary N. "Reconsidering the pathogenesis of ocular toxoplasmosis." American Journal of Ophthalmology 128, no. 4 (October 1999): 502–5. http://dx.doi.org/10.1016/s0002-9394(99)00263-9.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Ponzoni, M., R. Dolcetti, S. Magnino, F. Bertoni, M. Milco D'Elios, S. Govi, E. Guerini, C. Doglioni, and A. J. M. Ferreri. "3 Pathogenesis of ocular adnexal lymphoma." European Journal of Cancer Supplements 8, no. 4 (April 2010): 3. http://dx.doi.org/10.1016/s1359-6349(10)70674-1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Pierce, Eric A., and Donald J. D'amico. "Ocular Toxoplasmosis: Pathogenesis, Diagnosis, and Management." Seminars in Ophthalmology 8, no. 1 (January 1993): 40–52. http://dx.doi.org/10.3109/08820539309060209.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Vitaly, V. Potemkin, Olga A. Marchenko, Elena V. Ageeva, and Yuliya I. Malakhova. "Ocular cicatricial pemphigoid." Ophthalmology journal 10, no. 1 (March 15, 2017): 102–6. http://dx.doi.org/10.17816/ov101102-106.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Calugaru, Dan. "Etiology, pathogenesis, and diagnosis of neovascular glaucoma." International Journal of Ophthalmology 15, no. 6 (June 18, 2022): 1005–10. http://dx.doi.org/10.18240/ijo.2022.06.20.

Full text
Abstract:
Neovascular glaucoma is defined as iris and/or anterior chamber angle neovascularization associated with increased intraocular pressure. It is a secondary glaucoma that is most frequently caused by severe retinal ischemia. The most common diseases responsible for the development of neovascular glaucoma are diabetic retinopathy, ischemic central retinal vein occlusion, and ocular ischemic syndrome. Uncommon causes include ocular radiation, ocular tumors, uveitis and other miscellaneous conditions. Vascular endothelial growth factor is an important and likely predominant agent involved in the pathogenesis of intraocular neovascularization and neovascular glaucoma. The evolution of clinical and histopathological changes from predisposing conditions to the occurrence of rubeosis iridis and neovascular glaucoma is divided into four stages: prerubeosis, preglaucoma, open angle glaucoma, and angle-closure glaucoma.
APA, Harvard, Vancouver, ISO, and other styles
9

Egwuagu, C. E., L. Sun, S. H. Kim, and I. M. Dambuza. "Ocular Inflammatory Diseases: Molecular Pathogenesis and Immunotherapy." Current Molecular Medicine 15, no. 6 (August 19, 2015): 517–28. http://dx.doi.org/10.2174/1566524015666150731095426.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Ahmed, Muna, Ghassan Zein, Faizullah Khawaja, and C. Stephen Foster. "Ocular cicatricial pemphigoid: pathogenesis, diagnosis and treatment." Progress in Retinal and Eye Research 23, no. 6 (November 2004): 579–92. http://dx.doi.org/10.1016/j.preteyeres.2004.05.005.

Full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Ocular pathogenesis"

1

Ali, Magdi Mahmoud M. "The immunologic aspects of the pathogenesis of human onchocerciasis /." Stockholm : Dept. of immunology, Wenner-Gren institute, Stockholm university, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-793.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Kelly, Helena T. "The role of T helper 1 and T helper 2 lymphocyte subsets in the pathogenesis of experimental autoimmune uveoretinitis." Thesis, University of Aberdeen, 1995. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU543992.

Full text
Abstract:
CD4+ T cells can be subdivided on the basis of their lymphokine repertoire produced on activation resulting in TH1 like and TH2 like populations. The purpose of this study was to measure the intraocular expression of cytokines as a means of defining the CD4+ lymphocyte subsets present during the development of uveoretinitis. Lewis rats were immunised with retinal antigen and pertussis toxin resulting in early signs of disease activity evident by day 9 with increasing severity evident by day 12. Extensive clinical and histological damage was observed by day 14 with a reduction in severity through to end stage disease at day 24. In this study, the failure to establish pure populations of retinal antigen specific T lymphocyte cell lines and the observation of the lack of sensitivity of Northern hybridization to signals expressed at low levels resulted in the more sensitive technique of RT-PCR being utilized. Both IL2 and IFN mRNA expression was detected at all stages of disease with highest levels being present early in uveitis. In contrast IL4 mRNA levels increased with disease progression. This study suggests a pathogenic role for TH1 like cells and a protective role for TH2 like cells in this autoimmune disease. In order to provide an insight into alternative treatment strategies of the disease, immunomodulation of EAU was carried out using the immunosuppressive drugs CsA, FK506 and rapamycin and the resultant cytokine mRNA profiles examined. The results indicated that CsA and FK506 downregulated the TH1 response having suppressive effects on the levels of IL2 and IFN mRNA respectively. In contrast rapamycin was found to modulate the TH2 response enhancing IL4 levels. From this data, a drug based strategy employing rapamycin appears to be the most favourable approach.
APA, Harvard, Vancouver, ISO, and other styles
3

"Pathogenesis of retinoic acid-induced developmental ocular defects studied using mouse models." Thesis, 2009. http://library.cuhk.edu.hk/record=b6074726.

Full text
Abstract:
As exogenously administered RA suppressed the expression of the RA synthesizing enzymes, further investigation on whether this would lead to deficiency in endogenous RA concentrations was conducted. Results showed that exogenously administered RA significantly reduced the endogenous RA level in the head region with C57 embryos showing a greater reduction than ICR embryos.
In addition, detailed morphological and histological studies were conducted to determine if RA treatment caused early embryonic changes with strain difference. When compared with ICR embryos, C57 embryos exhibited more pronounced responses to RA, including developmental retardation, underdevelopment of the anterior neural plate and absence of or smaller optic pit/optic vesicle formation. However, RA treatment did not cause abnormal apoptosis in the early stages in both strains.
Since the teratogenic effect of RA is highly developmental stage-dependent, it is possible that there is a difference in the developmental stage between these 2 mouse strains at the time of RA injection. Indeed, it was found that the developmental stage of ICR embryos was approximately 6 hours ahead of C57 embryos. However, the role that this factor plays in the differential strain susceptibility to RA can be excluded since C57 fetuses were still 3 times more susceptible to developing anophthalmia/microphthalmia than ICR fetuses that were subject to RA treatment at equivalent developmental stages. Comparison of susceptibility to RA-induced anophthalmia/microphthalmia was also made among heterozygous fetuses obtained from reciprocal matings between C57 and ICR male and female mice, and those in homozygous ICR and C57 fetuses. Results showed that the C57 strain has conferred both genetic predisposition and maternal effects in increasing the embryo's susceptibility to RA-induced ocular defects.
Since the type of RA-induced ocular defects mimic those that developed in Raldh2 null mutant embryos, the effect of RA treatment on the expression of RA synthesizing enzymes, Raldh2 and Raldh3, and the RA-inducible gene Cyp26a1, as well as some early eye development genes were examined. Exogenously administered RA reduced the mRNA expression levels of Raldh2, Raldh3 and Cyp26a1 in the head region, with C57 embryos showing a greater reduction than ICR embryos.
Taken together, results of this thesis suggest that there is a strain difference in susceptibility to RA-induced ocular defects in which exogenously applied RA suppresses the expression of RA synthesizing enzymes and leads to endogenous RA deficiency. This finding may shed light on understanding why both excess and deficiency of RA can lead to similar types of ocular defects.
To determine if there are strain differences in the susceptibility to RA-induced ocular defects, two mouse strains were used. They are C57BL/6J (C57), mice that spontaneously develop ocular defects and ICR mice, which are not prone to developing ocular defects. Detailed time and dose response studies were conducted and eye defects were examined in near-term fetuses. C57 fetuses were found to be significantly more susceptible to RA-induced anophthalmia/microphthalmia than ICR fetuses.
Vitamin A (retinol) and its most active metabolite, all- trans retinoic acid (RA) is essential for vision in the adult and for eye development in the embryo. It is well documented that in humans, excess intake or deficiency of vitamin A or RA is associated with congenital ocular defects such as microphthalmia. However, the underlying mechanism remains unclear. The aim of this study is to examine the pathogenic mechanism of RA-induced developmental ocular defects.
Lau, Wing Sze Josephine.
Source: Dissertation Abstracts International, Volume: 71-01, Section: B, page: 0240.
Thesis (Ph.D.)--Chinese University of Hong Kong, 2009.
Includes bibliographical references (leaves 186-211).
Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.
Electronic reproduction. Ann Arbor, MI : ProQuest Information and Learning Company, [200-] System requirements: Adobe Acrobat Reader. Available via World Wide Web.
Abstracts in English and Chinese.
APA, Harvard, Vancouver, ISO, and other styles
4

Kintner, Randall Lynn. "Herpes simplex virus ocular disease viral and host factors involved in pathogenesis /." 1994. http://catalog.hathitrust.org/api/volumes/oclc/32418259.html.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Kim, Bum Seok. "HSV-1 induced ocular angiogenesis antiangiogenic strategies to prevent herpetic stromal keratitis pathogenesis /." 2005. http://etd.utk.edu/2005/KimBumSeok.pdf.

Full text
Abstract:
Thesis (Ph. D.) -- University of Tennessee, Knoxville, 2005.
Title from title page screen (viewed on September 1, 2005). Thesis advisor: Barry T. Rouse. Document formatted into pages (xiii, 165 p. : ill. (some col.)). Vita. Includes bibliographical references.
APA, Harvard, Vancouver, ISO, and other styles

Books on the topic "Ocular pathogenesis"

1

Glaucoma, Meeting (1995 Basel Switzerland). Ocular blood flow: New insights into the pathogenesis of ocular diseases. Basel: Karger, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Tatham, Andrew, and Peng Tee Khaw. Glaucoma. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199672516.003.0008.

Full text
Abstract:
This chapter explores glaucoma. It starts off with an outline of optic nerve head anatomy and then describes aqueous fluid dynamics and the pathogenesis of glaucoma. It then goes on to discuss the clinical skill areas of optic nerve head assessment in glaucoma, glaucoma imaging devices, tonometry and tachymetry, gonioscopy, and perimetry. The chapter also details ocular hypertension, primary open-angle glaucoma, primary angle closure, and secondary angle closure. In addition, it discusses normal tension glaucoma, steroid-induced glaucoma, traumatic glaucoma, inflammatory glaucomas, pseudoexfoliative glaucoma, pigmentary glaucoma, and neovascular glaucoma. It then covers aqueous misdirection, iridocorneal endothelial syndrome and iridocorneal dysgenesis, ocular hypotensive agents, laser therapy for glaucoma, and glaucoma surgery.
APA, Harvard, Vancouver, ISO, and other styles
3

Read, Jennifer S. Zika Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0015.

Full text
Abstract:
Although generally asymptomatic or mildly symptomatic in the general population, infection with the Zika virus (ZIKV) during pregnancy may lead to severely adverse fetal and infant outcomes, including the congenital Zika syndrome (CZS). Characteristics of this syndrome that are unique to it or are not typically observed with other congenital infections comprise anomalies of the brain and cranial morphology, ocular anomalies, congenital contractures, and neurological sequelae. The full spectrum of outcomes of mother-to-child transmission (MTCT) of ZIKV appears to be large, ranging from asymptomatic infection at birth, with possible later manifestation of significant abnormalities, to obvious and severe abnormalities in the fetus and infant. Although our understanding of pathogenesis, rates, and manifestations of CZS has improved rapidly and dramatically, much remains unknown or poorly understood regarding this potentially devastating congenital infection. Because of this, a broad research agenda regarding ZIKV is being implemented.
APA, Harvard, Vancouver, ISO, and other styles
4

Lloyd, Sheelagh, and Eric R. Morgan. Toxocarosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0071.

Full text
Abstract:
Toxocara canis and the syndromes of visceral and ocular larva migrans (VLM, OLM), covert toxocarosis, and neurological toxocarosis are described. Other potential agents, particularly Toxocara cati and Baylisascaris procyonis , are described. The transmission dynamics of toxocarosis to humans have never been fully elucidated, but the potential roles of pet and stray dogs, foxes, cats, and the influence of their population densities, and age demographies, are discussed in relation to contamination of the environment with eggs. Routes of infection with eggs by geophagia, poor hygiene outdoors and with dogs, and fly-borne contamination of food, and meat-borne ingestion of larvae are described. The development of prolonged in vitro culture and analyses of T. canis larval excretions/secretions (TES) and surface antigens helped explain the importance of the rapid production and shedding of TES in the prolonged course of infection and pathogenesis of disease. TES also have greatly improved serodiagnosis. However, we still have insufficient understanding of differences in the aetiology of the larvae or differences in immune responses among individuals to account for development of VLM, covert toxocarosis, or OLM in different individuals. Our understanding of the immunopathological response of the host to TES has emphasized the need for anti-inflammatory therapy in treatment; unfortunately, less information is available on the true efficacy of the anthelmintics available. The complexity of the T. canis life cycle in dogs is described and therapeutic regimens to prevent excretion of eggs by pet dogs are given. This, plus adequate control or exclusion of stray or wild canids from a property could prevent most cases of VLM. Control of infection from free-ranging stray dogs, cats and foxes, will be difficult and more data are needed to clarify the importance of these and of fly-borne and meat-borne transfer of infection to humans for control.
APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Ocular pathogenesis"

1

Trebing, Dietrich. "Ocular Rosacea." In Pathogenesis and Treatment of Acne and Rosacea, 665–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-540-69375-8_88.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Sen, Seema. "Pathogenesis and Pathology of Ocular Tuberculosis." In Essentials in Ophthalmology, 7–15. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57520-9_2.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Chauhan, Balwantray C. "The Role of Ocular Blood Flow Abnormalities in the Pathogenesis of Glaucoma." In Ocular Blood Flow, 411–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-69469-4_17.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Kaur, Tarandeep, Satish Patnaik, Saurabh Kumar, and Inderjeet Kaur. "Molecular Mechanisms in the Pathogenesis of Retinopathy of Prematurity (ROP)." In Genetics of Ocular Diseases, 101–23. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-4247-0_9.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Hipsley, Ann Marie, and Brad Hall. "Influence of Ocular Rigidity and Ocular Biomechanics on the Pathogenesis of Age-Related Presbyopia." In Ocular Rigidity, Biomechanics and Hydrodynamics of the Eye, 127–46. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-64422-2_9.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Genchi, C., P. Falagiani, G. Riva, and C. Sioli. "Ocular Toxocariasis: Role of IgE in the Pathogenesis of the Syndrome and Diagnostic Implications." In Helminth Zoonoses, 175–82. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3341-5_24.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Schiefer, A. Reagan, and Rebecca S. Bahn. "Pathogenesis of Graves’ ophthalmopathy." In Ocular Disease, 433–37. Elsevier, 2010. http://dx.doi.org/10.1016/b978-0-7020-2983-7.00056-5.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

de Paiva, Cintia. "Pathogenesis and Treatment of Dry Eye Syndrome." In Ocular Surface, 157–73. CRC Press, 2012. http://dx.doi.org/10.1201/b13153-14.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

"Pathogenesis of Infection and the Ocular Immune Response." In Ocular Infection, 1–21. CRC Press, 2007. http://dx.doi.org/10.3109/9781420020434-2.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

"n1 Pathogenesis of Infection and the Ocular Immune Response." In Ocular Infection, 15–36. CRC Press, 2007. http://dx.doi.org/10.3109/9781420020434-6.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Ocular pathogenesis"

1

Chauhan, Sheetal, Seema Sen, Anjana Sharma, Shyam S. Chauhan, Seema Kashyap, Radhika Tandon, Neelam Pushker, and M. Vanathi. "Abstract A03: Epigenetic alterations of stratifin in ocular surface squamous neoplasia: Pathogenesis and prognosis." In Abstracts: Third AACR International Conference on Frontiers in Basic Cancer Research - September 18-22, 2013; National Harbor, MD. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.fbcr13-a03.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Eilaghi, Armin, Ian A. Sigal, Christian G. Olesen, Inka Tertinegg, John G. Flanagan, G. Wayne Brodland, and C. Ross Ethier. "The Effect of Nonlinear Scleral Properties on Optic Nerve Head Biomechanics." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176251.

Full text
Abstract:
Glaucoma is a group of potentially blinding ocular diseases caused by gradual and progressive damage to the optic nerve, and is usually associated with elevated intraocular pressure (IOP) [1]. This damage occurs at the optic nerve head (ONH), the site where the optic nerve axons leave the posterior eye. IOP-related biomechanical factors are hypothesized to play a key role in the pathogenesis of glaucomatous damage [2].
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Ocular pathogenesis' for particular source types:
Journal articles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography