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Books on the topic 'Ocular motor'

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Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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1

Linde, Lucille M. J. Effects of motor-perceptual training on academic achievement and ocular pursuit ability. Greeley, Colo: L.M.J. Linde, 1992.

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2

Linde, Lucille M. J. Manual for the Lucille Linde ocumeter: Ocular pursuit measuring instrument. Stockton, Calif: L.M.J. Linde, 1992.

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3

Jennings, David Wallace. A PC based data acquisition system for monitoring the response of the ocular motor system. S.l: The Author, 1997.

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4

Why red doesn't sound like a bell: Understanding the feel of consciousness. New York: Oxford University Press, 2011.

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5

A, Berthoz, ed. Multisensory control of movement. Oxford [England]: Oxford University Press, 1993.

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6

Peterson, Michael J. Development of a method for inactivation of cerebellar flocculus region neurons using the AMPA-kainate antagonist CNQX and the effects of inactivation on motor memory in the vestibulo-ocular reflex. Ottawa: National Library of Canada, 2003.

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7

Sánchez, Emilio. Arriortúa: "Superlopez" y la guerra oculta entre General Motors y Volkswagen. Madrid: Ediciones Temas de Hoy, 1993.

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8

G, Gale Alastair, Applied Vision Association (Great Britain), Ergonomics Society (Great Britain), and Association of Optometrists (Great Britain), eds. Vision in vehicles-IV. Amsterdam: North-Holland, 1993.

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9

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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10

Kennard, Christopher. Ocular motor disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0274.

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This chapter discusses motor disorders of the eye. The first part of the chapter describes the proper examination of eye movements to facilitate identification of ocular motor disorder pathology. The effects of nerve palsies on ocular motor function are then described.Eye movement disorders can also have their cause in the central nervous system; both the brainstem, and cerebellum have been implicated as causal factors in some eye movement disorders. Disorders of the pupil, which affect the pupillary light reflex, can be caused by lesions to central, afferent and efferent pupillary pathways as well as sympathetic pathways lesions.Finally, this chapter describes diseases of the eye orbits, including dysthyroid eye disease, idiopathic orbital inflammation, orbital tumours, vascular disorders and orbital infections.
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11

Ocular motor disorders of the brain stem. London: Baillière, 1992.

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12

Strupp, Michael, and Thomas Brandt. Ocular Motor Disorders and Vertigo: Diagnosis and Management. Springer, 2023.

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13

Chung, Ji Yeon Jenni. Consolidation of motor memory in the vestibulo-ocular reflex. 2006.

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14

Buttner, U., and Th Brandt. Ocular Motor Disorders of the Brain Stem (Bailliere's Clinical Neurology). Elsevier, 1992.

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15

Developing Ocular Motor and Visual Perceptual Skills: An Activity Workbook. Slack Incorporated, 2005.

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16

Rucker, Janet, and David Zee. Basic and Clinical Ocular Motor and Vestibular Research, Volume 1233. Wiley & Sons, Limited, John, 2011.

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17

Shaikh, Aasef G., and Alessandra Rufa, eds. Ocular Motor and Vestibular Function in Neurometabolic, Neurogenetic, and Neurodegenerative Disorders. Frontiers Media SA, 2018. http://dx.doi.org/10.3389/978-2-88945-563-8.

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18

Zee, David S., Albert F. Fuchs, and Ulrich Buettner. Contemporary Ocular Motor and Vestibular Research: A Tribute to David A. Robinson. Thieme Publishing Group, 1994.

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19

Leigh, R. John, Stefano Ramat, and Aasef G. Shaikh. Mathematical Modelling in Motor Neuroscience: State of the Art and Translation to the Clinic. Ocular Motor Plant and Gaze Stabilization Mechanisms. Elsevier Science & Technology Books, 2019.

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20

Mathematical Modelling in Motor Neuroscience: State of the Art and Translation to the Clinic. Ocular Motor Plant and Gaze Stabilization Mechanisms. Elsevier, 2019. http://dx.doi.org/10.1016/s0079-6123(19)x0005-9.

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21

Wong, Agnes Ming-Fong. Three dimensional disorders of gaze and binocular alignment after brainstem and ocular motor nerve lesions. 2001.

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22

F, Fuchs Albert, and Robinson, D. A. 1925 Dec. 9-, eds. Contemporary ocular motor and vestibular research: A tribute to David A. Robinson : international meeting Eibsee, 1993. Stuttgart, Germany: Georg Thieme Verlag, 1994.

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23

Stevens, George Thomas. A Treatise On The Motor Apparatus Of The Eyes: Embracing An Exposition Of The Anomalies Of The Ocular Adjustments And Their Treatment. Kessinger Publishing, LLC, 2007.

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24

Stevens, George Thomas. A Treatise On The Motor Apparatus Of The Eyes: Embracing An Exposition Of The Anomalies Of The Ocular Adjustments And Their Treatment. Kessinger Publishing, LLC, 2007.

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25

Kennard, Christopher. Abnormal vision. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0255.

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Neuro-ophthalmology is a discipline comprising a wide variety of disorders that overlap the fields of neurology, ophthalmology and general medicine. Diagnosis in this field requires a thorough knowledge of the anatomy and physiology of the visual pathways and ocular motor system, as well as the ability to carry out a thorough neuro-ophthalmological examination. The combination of the history and any abnormalities identified by the examination should enable a detailed differential diagnosis to be reached, leading to appropriate investigations if required, and a final diagnosis.
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26

Shaibani, Aziz. Fatigability. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0025.

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Undue fatigability is common in neuromuscular clinics, but nonneuromuscular causes are much more common than neuromuscular causes. Generalized fatigue is commonly caused by anemia, hypothyroidism, obstructive sleep apnea, depression, chronic fatigue syndrome (CFS), uremia, chronic obstructive pulmonary disease (COPD), and other diseases. Physiological fatigue is accentuated by neuromuscular disorders. Most strikingly, myasthenia gravis (MG) causes undue fatigue of the ocular, chewing, swallowing, and breathing muscles. However, amyotrophic lateral sclerosis (ALS), myopathies, and motor neuropathies are also associated with abnormal fatigue. Central causes like multiple sclerosis are notorious for fatigue. It is hard to measure fatigue because it is subjective and varies with the psychological status. Myasthenia hardly causes isolated fatigue.
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27

Shaibani, Aziz. Fatigability. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0025.

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Undue fatigability is common in neuromuscular clinics but non-neuromuscular causes are much more common than neuromuscular causes. Generalized fatigue is commonly caused by anemia, hypothyroidism, obstructive sleep apnea, depression, chronic fatigue syndrome, uremia, COPD, etc. Physiological fatigue is accentuated by neuromuscular disorders. Most strikingly, myasthenia gravis causes undue fatigue of the ocular, chewing, swallowing, and breathing muscles. However, ALS, myopathies, and motor neuropathies are also associated with abnormal fatigue. Myasthenia rarely causes isolated fatigue. Examination for fatigability should be part of neuromuscular evaluation and is conducted by inducing repetitive or sustained contraction of the suspected muscles (typically extraocular muscles) for a minute and reevaluation after 2 minutes of rest of the tested muscles.
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28

Fletcher, Nicholas. Tremor, ataxia, and cerebellar disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0898.

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Tremors are characterized by rhythmic oscillations of one or more body parts. Although typically seen in the upper limbs, almost any area may be involved, including the trunk, head, facial muscles, and legs. Sometimes, tremor is not visible at all but may be heard or palpated, for example, in vocal or orthostatic tremor, respectively. In neurological practice, the diagnosis and treatment of tremor is an everyday problem. A common scenario is the distinction between essential tremor and Parkinson’s disease. In this chapter, the wide range of tremors are discussed, with their aetiolology, pathophysiology, diagnosis and management described.Ataxia is a term used to describe a wide range of neurological disorders affecting muscle coordination, speech and balance that reflect dysfunction of a part of the central nervous system involved in motor function. Many of ataxias have a cerebellar pathology as root cause, although it must be remembered that ataxia, clumsiness, disordered ocular motility, dysarthria, and even kinetic or intention tremor are not always caused by cerebellar disease. This chapter describes the wide range of cerebellar disorders and ataxias, as are non-cerebellar ataxias such as Friedreich’s ataxia.
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29

Reddy, Ugan, and Nicholas Hirsch. Diagnosis, assessment, and management of myasthenia gravis and paramyasthenic syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0244.

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Diseases that affect the neuromuscular junction (NMJ) interfere with normal nerve transmission and cause weakness of voluntary muscles. The two most commonly encountered are acquired myasthenia gravis (MG) and the Lambert–Eaton myasthenic syndrome (LEMS). Acquired MG is an autoimmune disease in which antibodies are directed towards receptors at the NMJ. In 85% of patients, IgG antibodies against the postsynaptic acetylcholine receptor (AChR) are found (seropositive MG). The thymus gland appears to be involved in the production of these which cause an increase rate of degradation of AChR resulting in a decreased receptor density resulting in a reduced postsynaptic end-plate potential following motor nerve stimulation and leading to muscle weakness. Although all voluntary muscles can be affected, ocular, bulbar, respiratory, and proximal limb weakness predominates. In the majority of seronegative patients, an antibody directed towards a NMJ protein called muscle specific tyrosine kinase (MUSK) is found. Anti-MUSK MG is characterized by severe bulbar and respiratory muscle weakness. Diagnosis of MG requires a high degree of clinical suspicion coupled with pharmacological and electrophysiological testing, and detection of the various causative antibodies. Treatment of MG involves enhancing neuromuscular transmission with long-acting anticholinesterase agents and immunosuppression. Acute exacerbations are treated with either plasma exchange or intravenous immunoglobulin. Myasthenic crisis is associated with severe muscle weakness that necessitates tracheal intubation and mechanical ventilation. LEMS is an autoimmune disease in which IgG antibodies are directed towards the pre-synaptic voltage-gated calcium channels at the NMJ. It is often associated with malignant disease (usually small cell carcinoma of the lung). Autonomic dysfunction is prominent and patients show abnormal responses to neuromuscular blocking drugs.
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30

Innovación, motor de la competividad agroalimentaria: Políticas y estrategias para que en México ocurra. Chapingo, Estado de México: Universidad Autónoma Chapingo--CIESTAAM/PIIAI, 2007.

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31

Manrrubio, Muñoz Rodríguez, and Universidad Autónoma Chapingo. Programa Interdepartamental Integración Agricultura-Industria., eds. Innovación, motor de la competividad agroalimentaria: Políticas y estrategias para que en México ocurra. Chapingo, Estado de México: Universidad Autónoma Chapingo--CIESTAAM/PIIAI, 2007.

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32

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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