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1

Mills, K. B. "Ocular Hypertension." Seminars in Ophthalmology 1, no. 1 (January 1986): 41–45. http://dx.doi.org/10.3109/08820538609071474.

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2

Lundberg, Lars, Karin Wettrell, and Erik Linnér. "Ocular hypertension." Acta Ophthalmologica 65, no. 6 (May 27, 2009): 705–8. http://dx.doi.org/10.1111/j.1755-3768.1987.tb07067.x.

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3

LINNÉR, ERIK. "OCULAR HYPERTENSION." Acta Ophthalmologica 54, no. 6 (May 27, 2009): 707–20. http://dx.doi.org/10.1111/j.1755-3768.1976.tb01790.x.

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4

LINNÉR, ERIK. "OCULAR HYPERTENSION." Acta Ophthalmologica 56, no. 2 (May 27, 2009): 179–90. http://dx.doi.org/10.1111/j.1755-3768.1978.tb01344.x.

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5

You, Yu X., Chang X. Chen, Ke Ma, and Jost B. Jonas. "Ocular hypertension." Acta Ophthalmologica 91, no. 7 (May 29, 2013): e587-e589. http://dx.doi.org/10.1111/aos.12183.

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6

Mirza, Salman, Tariq Saeed, and Philip I. Murray. "Ocular Hypertension Associated with Ocular Sarcoidosis." Ocular Immunology and Inflammation 15, no. 6 (January 2007): 447–49. http://dx.doi.org/10.1080/09273940701732230.

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7

Akingbehin, Tayo. "Corticosteroid-Induced Ocular Hypertension." Journal of Toxicology: Cutaneous and Ocular Toxicology 5, no. 1 (January 1986): 45–53. http://dx.doi.org/10.3109/15569528609068362.

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8

Hovding, Gunnar, and Torstein I. Bertelsen. "Ocular hypertension versus glaucoma." Current Opinion in Ophthalmology 1, no. 2 (April 1990): 105–8. http://dx.doi.org/10.1097/00055735-199001020-00002.

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9

Hovding, Gunnar, and Torstein I. Bertelsen. "Ocular hypertension versus glaucoma." Current Opinion in Ophthalmology 1, no. 2 (April 1990): 105–8. http://dx.doi.org/10.1097/00055735-199004000-00002.

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10

Gordon, Mae O., and Michael A. Kass. "Ocular Hypertension Treatment Study." Journal of Glaucoma 2, Supplement A (1993): 24???25. http://dx.doi.org/10.1097/00061198-199300021-00011.

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11

BOSCHI, A. "Thyroid and ocular hypertension." Acta Ophthalmologica 89, s248 (September 2011): 0. http://dx.doi.org/10.1111/j.1755-3768.2011.3452.x.

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12

Tuulonen, Anja. "Treatment of ocular hypertension." Current Opinion in Ophthalmology 27, no. 2 (March 2016): 89–93. http://dx.doi.org/10.1097/icu.0000000000000233.

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13

Grasso, Cono M. "Ocular Hypertension Treatment Study." Evidence-Based Eye Care 4, no. 1 (January 2003): 16–17. http://dx.doi.org/10.1097/00132578-200301000-00007.

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14

Wilson, M. Roy, Michael Drake, and James Brandt. "Management of Ocular Hypertension." Journal of Glaucoma 13, no. 1 (February 2004): 81–83. http://dx.doi.org/10.1097/00061198-200402000-00015.

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15

Oh, Min Jin, and Soo Young Lee. "Ocular Diseases and Hypertension." Korean Journal of Medicine 83, no. 5 (2012): 591. http://dx.doi.org/10.3904/kjm.2012.83.5.591.

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16

Muñoz, Silvia, Nieves Martín, and Jorge Arruga. "Malignant Hypertension: Ocular Manifestations." Scientific World JOURNAL 6 (2006): 122–24. http://dx.doi.org/10.1100/tsw.2006.28.

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Malignant hypertension may be the first manifestation of systemic hypertension. We report a clinical case of a Caucasian 41-year-old man with no previous history of blood hypertension seen at casualty because of blurred vision. Fundus examination disclosed optic disk swelling, retinal hemorrhages and infarcts. The blood pressure was 220/130 mmHg. After the appropriate management of hypertension, optic disk and retinal edema resolved, leaving minor changes as mild optic disk pallor and hard exudates.
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17

Muñoz, Silvia, Nieves Martín, and Jorge Arruga. "Malignant Hypertension: Ocular Manifestations." TheScientificWorldJOURNAL 6 (January 26, 2006): 122–24. http://dx.doi.org/10.1100/tsw2006.28.

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18

Bhargava, M., M. K. Ikram, and T. Y. Wong. "Ocular manifestations of hypertension." Hipertensión y Riesgo Vascular 29, no. 3 (July 2012): 96–105. http://dx.doi.org/10.1016/j.hipert.2012.07.001.

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19

Higginbotham, Eve J. "Ocular Hypertension Treatment Study." Archives of Ophthalmology 127, no. 2 (February 9, 2009): 213. http://dx.doi.org/10.1001/archophthalmol.2008.599.

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20

Sommer, Alfred. "Treatment of Ocular Hypertension." Archives of Ophthalmology 128, no. 3 (March 1, 2010): 363. http://dx.doi.org/10.1001/archophthalmol.2010.13.

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21

Yablonski, Michael E. "Ocular Hypertension Treatment Study." Archives of Ophthalmology 122, no. 7 (July 1, 2004): 1088. http://dx.doi.org/10.1001/archopht.122.7.1088.

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22

Khatri, Anadi, Kinsuk Singh, Bivek Wagle, Hony K.C, Pratap Karki, and André Mermoud. "Causes and Managements of Early-Onset Ocular Hypertension Following Pars Plana Vitrectomy with Silicone Oil for Retinal Detachment and Exploration of Trabeculectomy as a Viable Alternative Management: A Pilot Study." Nepalese Journal of Ophthalmology 14, no. 1 (May 23, 2022): 39–48. http://dx.doi.org/10.3126/nepjoph.v14i1.35475.

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Introduction: This study aims to study a relatively unexplored topic about the causes and managements of early-onset ocular hypertension (OHTN) following the pars plana vitrectomy with silicone oil (PPV with SO) procedure for retinal detachment. Additionally, to explore the outcome of trabeculectomy in managing such patients. Materials and methods: This is a retrospective exploratory pilot study. We studied 23 patients who underwent the procedure then subsequently developed ocular hypertension within a month of the procedure. The probable causes for their early-onset ocular hypertension were identified and addressed with medicine, peripheral iridotomy (PI), complete or partial silicone removal. Trabeculoplasty was done in irretractable causes. This study aimed to evaluate the causes of early onset ocular hypertension after pars plana vitrectomy with silicone oil and explore the outcome of different managements including trabeculectomy. Results: Inflammation (n=11, 47.8%) was the most common cause of early-onset ocular hypertension. Other causes were overfilling/spilling of silicone oil in anterior chamber (n=5, 21.7%), pupillary block (n=4, 17.4%) and angle-recession glaucoma (n=2, 8.69%). Majority of the cases responded to intraocular pressure (IOP) lowering medications (n=11). Three eyes with persistently high intraocular pressure underwent trabeculectomy after which the intraocular pressure was controlled. Conclusion: Even though prior studies have reported that trabeculectomy does not address late-onset ocular hypertension, our study shows that the procedure might be helpful in early-onset ocular hypertension. This is probably because at the time of presentation for early-onset ocular hypertension, silicone has not emulsified, which will not be the case in late-onset ocular hypertension. If a large study also shows that trabeculectomy can correct early-onset ocular hypertension, this information can guide the practices of ophthalmologists whose patients cannot afford expensive glaucoma drainage devices.
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23

&NA;. "Timolol gel for ocular hypertension." Inpharma Weekly &NA;, no. 911 (October 1993): 20. http://dx.doi.org/10.2165/00128413-199309110-00051.

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24

Dawson, William W., J. Pete Schroeder, Judyth C. Dawson, and Paul E. Nachtigall. "CYCLIC OCULAR HYPERTENSION IN CETACEANS." Marine Mammal Science 8, no. 2 (April 1992): 135–42. http://dx.doi.org/10.1111/j.1748-7692.1992.tb00372.x.

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25

Jonas, Jost B., Gangolf Sauder, Wido M. Budde, Ulrich H. M. Spandau, Bernd A. Kamppeter, and Bjoern Harder. "Triamcinolone Acetonide–Induced Ocular Hypertension." Journal of Ocular Pharmacology and Therapeutics 22, no. 4 (August 2006): 247–50. http://dx.doi.org/10.1089/jop.2006.22.247.

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26

Schubert, Hermann D. "Ocular manifestations of systemic hypertension." Current Opinion in Ophthalmology 9, no. 6 (December 1998): 69–72. http://dx.doi.org/10.1097/00055735-199812000-00012.

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27

Kass, Michael A. "The Ocular Hypertension Treatment Study." Journal of Glaucoma 3, no. 2 (1994): 97???100. http://dx.doi.org/10.1097/00061198-199400320-00001.

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28

Cunningham, Emmett T., and Manfred Zierhut. "Uveitic Ocular Hypertension and Glaucoma." Ocular Immunology and Inflammation 25, no. 6 (November 2, 2017): 737–39. http://dx.doi.org/10.1080/09273948.2017.1415077.

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29

Lewczuk, Natalia, Alexander Zdebik, Joanna Bogusławska, Anna Turno-Kręcicka, and Marta Misiuk-Hojło. "Ocular manifestations of pulmonary hypertension." Survey of Ophthalmology 64, no. 5 (September 2019): 694–99. http://dx.doi.org/10.1016/j.survophthal.2019.02.009.

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30

Clark, C. V., and R. Mapstone. "AUTONOMIC NEUROPATHY IN OCULAR HYPERTENSION." Lancet 326, no. 8448 (July 1985): 185–87. http://dx.doi.org/10.1016/s0140-6736(85)91500-4.

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31

Mediero, Aránzazu, Pilar Alarma-Estrany, and Jesús Pintor. "New treatments for ocular hypertension." Autonomic Neuroscience 147, no. 1-2 (May 2009): 14–19. http://dx.doi.org/10.1016/j.autneu.2008.12.009.

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32

Dibas, Adnan, and Thomas Yorio. "Glucocorticoid therapy and ocular hypertension." European Journal of Pharmacology 787 (September 2016): 57–71. http://dx.doi.org/10.1016/j.ejphar.2016.06.018.

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33

Ambrosio, G., G. Arienzo, P. Aurilia, A. Colasanti, and R. Fusco. "Pattern electroretinograms in ocular hypertension." Documenta Ophthalmologica 69, no. 2 (June 1988): 161–65. http://dx.doi.org/10.1007/bf00153697.

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34

Erichev, V. P., and L. A. Panyushkina. "Modern view on ocular hypertension." Vestnik oftal'mologii 135, no. 5 (2019): 305. http://dx.doi.org/10.17116/oftalma2019135052305.

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35

Georgopoulos, G., D. Andreanos, N. Liokis, D. Papakonstantinou, J. Vergados, and G. Theodossiadis. "Risk Factors in Ocular Hypertension." European Journal of Ophthalmology 7, no. 4 (October 1997): 357–63. http://dx.doi.org/10.1177/112067219700700409.

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The aim of the study was to determine whether certain factors are related to an increased risk of developing open-angle glaucoma. A total of 345 untreated glaucoma suspects with intraocular pressure (IOP) ≥ 21 mmHg, cup to disc ratio 0.4 or less and no visual field defects, were followed up for 6 to 8 years (mean 7.3). During the follow-up 71 patients developed established glaucoma and were compared to the remaining 274 patients. The following factors were analysed: age, family history of glaucoma, IOP, Humphrey 30-2 visual fields, optic disc appearance, myopia, exfoliation, arterial hypertension and diabetes. Analysis yielded statistically significant results regarding a number of these factors in the patients who subsequently developed open-angle glaucoma. A significant association with the subsequent development of field loss in ocular hypertension (OHT) included: heredity (p < 0.001), age ≥ 60 years (p = 0.013), axial myopia (0.001 < p < 0.01) and arterial hypertension (p = 0.05). About 20% of patients with ocular hypertension developed glaucoma over a period of seven years. Risk factors such as heredity, age, myopia and arterial hypertension, among others, must be considered in the follow-up of glaucoma suspects.
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36

Lee, Anne J., and Ivan Goldberg. "Emerging drugs for ocular hypertension." Expert Opinion on Emerging Drugs 16, no. 1 (February 25, 2011): 137–61. http://dx.doi.org/10.1517/14728214.2011.521631.

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37

Erichev, V. P., A. O. Tarasenkov, and Yu S. Andreeva. "Ocular hypertension after intravitreal injections." Vestnik oftal'mologii 138, no. 5 (2022): 234. http://dx.doi.org/10.17116/oftalma2022138052234.

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38

Akarsu, Cengiz, Yasemin Karadeniz Bilgili, Birsen Ünal, Pelin Taner, Ahmet Ergin, and Simay Altan Kara. "Cerebral hemodynamics in ocular hypertension." Graefe's Archive for Clinical and Experimental Ophthalmology 243, no. 4 (October 27, 2004): 317–20. http://dx.doi.org/10.1007/s00417-004-0963-2.

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39

Robin, Alan L. "The Ocular Hypertension Treatment Study." Archives of Ophthalmology 122, no. 3 (March 1, 2004): 376. http://dx.doi.org/10.1001/archopht.122.3.376.

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40

Higginbotham, Eve J. "The Ocular Hypertension Treatment Study." Archives of Ophthalmology 122, no. 6 (June 1, 2004): 813. http://dx.doi.org/10.1001/archopht.122.6.813.

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41

Kass, Michael A. "Ocular Hypertension Treatment Study—Reply." Archives of Ophthalmology 122, no. 7 (July 1, 2004): 1089. http://dx.doi.org/10.1001/archopht.122.7.1089-a.

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42

Gordon, Mae O. "The Ocular Hypertension Treatment Study." Archives of Ophthalmology 117, no. 5 (May 1, 1999): 573. http://dx.doi.org/10.1001/archopht.117.5.573.

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43

Kass, Michael A. "The Ocular Hypertension Treatment Study." Archives of Ophthalmology 120, no. 6 (June 1, 2002): 701. http://dx.doi.org/10.1001/archopht.120.6.701.

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44

Gordon, Mae O. "The Ocular Hypertension Treatment Study." Archives of Ophthalmology 120, no. 6 (June 1, 2002): 714. http://dx.doi.org/10.1001/archopht.120.6.714.

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45

Stein, Joshua D. "MANAGEMENT OF OCULAR HYPERTENSION: A COST-EFFECTIVENESS APPROACH FROM THE OCULAR HYPERTENSION TREATMENT STUDY." Evidence-Based Ophthalmology 7, no. 4 (October 2006): 220–22. http://dx.doi.org/10.1097/01.ieb.0000212041.42945.42.

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46

Myers, J. S. "Management of Ocular Hypertension: A Cost-effectiveness Approach From the Ocular Hypertension Treatment Study." Yearbook of Ophthalmology 2007 (January 2007): 66–68. http://dx.doi.org/10.1016/s0084-392x(08)70054-6.

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47

Kymes, Steven M., Michael A. Kass, Douglas R. Anderson, J. Philip Miller, and Mae O. Gordon. "Management of Ocular Hypertension: A Cost-effectiveness Approach From the Ocular Hypertension Treatment Study." American Journal of Ophthalmology 141, no. 6 (June 2006): 997–1008. http://dx.doi.org/10.1016/j.ajo.2006.01.019.

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48

Calugaru, Dan. "Intraocular pressure modifications in patients with acute central/hemicentral retinal vein occlusions." International Journal of Ophthalmology 14, no. 6 (June 18, 2021): 931–35. http://dx.doi.org/10.18240/ijo.2021.06.20.

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Intraocular pressure (IOP) modifications in patients with acute central/hemicentral retinal vein occlusions (RVOs) consist in IOP reductions and increases. The IOP reduction is due to a transitional hyposecretory phase of the aqueous humor, that increases gradually until 3mo after the venous occlusion onset, and then finally disappears after month 4th. The IOP increases lead to the ocular hypertension and glaucoma. The possible pathogenetic correlations between ocular hypertension/glaucoma and acute central/hemicentral RVOs have been classified into three groups: 1) the venous occlusion precedes the ocular hypertension/glaucoma causing neovascular glaucoma and secondary angle-closure glaucoma without rubeosis; 2) the ocular hypertension and the glaucoma precede the venous occlusion and favor its appearance (ocular hypertension, primary angle-closure, primary angle-closure glaucoma, and open angle glaucomas); and 3) the venous occlusion and the ocular hypertension/glaucoma are mostly age dependent appearances due to common vascular and collagen alterations, lacking a causal connection between the 2 conditions.
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49

Parajuli, Sanket, Pooja Shrestha, Sadhana Sharma, and Jeevan K. Shrestha. "Prevalence of Ocular Hypertension in Patients Above 40 Years of Age." Nepalese Journal of Ophthalmology 14, no. 1 (May 23, 2022): 140–43. http://dx.doi.org/10.3126/nepjoph.v14i1.29740.

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Introduction: Ocular hypertension is a condition with elevated intraocular pressure that needs to be monitored closely to prevent glaucoma and other complications. The study aims to find out the prevalence of ocular hypertension in patients aged more than 40 years. Materials and methods: This is a hospital based cross-sectional study conducted in a community based tertiary hospital of Nepal. Results: Mean age of the patients was 47.53years. 62% of the patients were males and 38% were females. Mean intraocular pressure in the right eye was 15.8 mmHg and mean intraocular pressure in left eye was 16.2 mm Hg. Prevalence of ocular hypertension was 6%. Conclusion: All patients more than 40 years of age should undergo detailed ocular examination for early detection and treatment of ocular hypertension.
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50

Frangois, J., A. Neetens, and J. M. Collette. "EXPERIMENTAL HYPERTENSION OF THE OPHTHALMIC ARTERY AND OCULAR HYPERTENSION." Acta Ophthalmologica 37, no. 4 (May 27, 2009): 395–403. http://dx.doi.org/10.1111/j.1755-3768.1959.tb03452.x.

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