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Books on the topic 'Obstructive'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

O’Reilly, P. H., ed. Obstructive Uropathy. London: Springer London, 1986. http://dx.doi.org/10.1007/978-1-4471-1380-5.

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2

Carter, Rick. Courage and information for life with chronic obstructive pulmonary disease: The handbook for patients, families, and care givers managing COPD (emphysema, asthmatic bronchitis, or chronic bronchitis). Onset, MA: New Technology Pub., 1999.

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3

A, Stockley Robert, ed. Chronic obstructive pulmonary disease. Malden, Mass: Blackwell Pub., 2005.

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4

Carr, Michele M. Pediatric obstructive sleep apnea. Alexandria, VA: American Academy Of Otolaryngology--Head and Neck Surgery Foundation, 2007.

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5

Currie, Graeme P. Chronic obstructive pulmonary disease. Oxford: Oxford University Press, 2009.

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6

Nakamura, Hiroyuki, and Kazutetsu Aoshiba, eds. Chronic Obstructive Pulmonary Disease. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-0839-9.

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7

Nici, Linda, and Richard ZuWallack, eds. Chronic Obstructive Pulmonary Disease. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-673-3.

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8

Stockley, Robert A., Stephen I. Rennard, Klaus Rabe, and Bartolome Celli, eds. Chronic Obstructive Pulmonary Disease. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470755976.

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9

Calverley, P. M. A., and N. B. Pride, eds. Chronic Obstructive Pulmonary Disease. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9.

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10

Celli, Bartolome R., and Stephen I. Rennard. Chronic obstructive pulmonary disease. Philadelphia, Pennsylvania: Saunders, an imprint of Elsevier, Inc., 2012.

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11

A, Calverley P. M., ed. Chronic obstructive pulmonary disease. 2nd ed. London: Arnold, 2003.

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12

R, Celli Bartolome, ed. Pharmacotherapy in chronic obstructive pulmonary disease. New York: Marcel Dekker, 2004.

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13

name, No. Snoring and obstructive sleep apnea. 3rd ed. Philadelphia, PA: Lippincott Willians & Wilkins, 2003.

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14

Blackler, Laura, Christine Jones, and Caroline Mooney, eds. Managing Chronic Obstructive Pulmonary Disease. West Sussex, England: John Wiley & Sons Ltd, 2007. http://dx.doi.org/10.1002/9780470697603.

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15

Kim, Ki Beom, Reza Movahed, Raman K. Malhotra, and Jeffrey J. Stanley, eds. Management of Obstructive Sleep Apnea. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-54146-0.

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16

O'Reilly, Patrick H. Obstructive Uropathy. Springer, 2012.

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17

1947-, O'Reilly P. H., ed. Obstructive uropathy. Berlin: Springer-Verlag, 1985.

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18

Sobol, Julia, and Jack Louro. Obstructive Shock. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0012.

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In the perioperative period, various mechanisms can lead to the development of shock. The type of shock needs to be rapidly elucidated and initial management steps initiated to minimize the time of tissue hypoperfusion. Obstructive shock is caused by physical obstruction of circulation either into or out of the heart. The mechanisms that lead to obstructive shock either prevent blood from entering the right heart during diastole such as a tension pneumothorax or pericardial tamponade, or prevent the heart from ejecting the blood due to a physical obstruction, as in the case of pulmonary embolism or left ventricular outflow obstruction. While supportive care with volume resuscitation and inotropes to maintain cardiac output is crucial, early determination of the cause with prompt treatment is needed to prevent circulatory collapse. This chapter reviews the pathophysiologic mechanisms leading to obstructive shock and management steps to stabilize the patient and treat the underlying cause.

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19

O'reilly, Patrick H. Obstructive Uropathy. Springer, 2012.

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20

Obstructive Calculous Jaundice. Beta Medical Arts, 2001.

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21

1960-, Kushida Clete Anthony, ed. Obstructive sleep apnea. New York, NY: Informa Healthcare USA, 2007.

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22

A, Dosman James, and Cockcroft Donald W, eds. Obstructive lung disease. Philadelphia: Saunders, 1990.

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23

A, Dosman James, and Cockcroft Donald W, eds. Obstructive lung diseases. Philadelphia: Saunders, 1996.

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24

Barnes. Chronic Obstructive Pulmonary. Taylor & Francis, 1997.

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25

A, Dosman James, and Cockcroft Donald W. 1946-, eds. Obstructive lung diseases. Philadelphia: W.B. Saunders, 1996.

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26

Johnson, Jonas T., Jack L. Gluckman, and Mark H. Sanders. Obstructive Sleep Apnoea. Informa Healthcare, 2001.

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27

Obstructive Airway Diseases. Taylor & Francis Group, 2011.

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28

Johnson, Jonas T., Jack L. Gluckman, and Mark H. Sanders. Obstructive Sleep Apnoea. Taylor & Francis Group, 2003.

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29

Kushida, Clete A. Obstructive Sleep Apnea. Taylor & Francis Group, 2007.

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30

Johnson, Jonas T., Jack Gluckman, and Jack Sanders. Obstructive Sleep Apnoea. Taylor & Francis Group, 2001.

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31

Craig, Sonya, and Sophie West. Obstructive sleep apnoea. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0136.

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Obstructive sleep apnoea (OSA) is caused by the repetitive closure of the pharynx during sleep, leading to sleep fragmentation and, often, daytime somnolence. Traditionally, it is defined as either the number of apnoeas (complete cessation of breathing for longer than 10 seconds) or hyponoeas (reduction in air flow by >50%) per hour in an overnight sleep study. However, it must be remembered that this definition is arbitrary, and OSA is better viewed as a spectrum with trivial snoring at one end and severe, almost continuous obstruction at the other. In addition to the sleep-study findings, if the patient is sleepy during the day, as defined by the Epworth Sleepiness Scale, then this condition is termed ‘obstructive sleep apnoea syndrome’. This distinction is important, as patients with this syndrome usually warrant treatment.

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32

Squire, Peter. Obstructive Sleep Apnea. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0012.

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Adenotonsillectomy has become first-line treatment for obstructive sleep apnea (OSA) and it is increasingly performed as a day-case procedure. A diagnosis of OSA increases the risk for postoperative respiratory morbidity from 1% to approximately 20% and unfortunately, the clinical history may be unreliable at distinguishing which children are at greatest risk. The gold standard investigation is overnight polysomnography (PSG), but this is a scarce resource considering the number of procedures performed. Fortunately, overnight home pulse oximetry also provides a useful stratification of severity and may predict postoperative problems. Children with OSA have a respiratory drive and airway tone that may be exquisitely sensitive to anesthetic and analgesic agents. Accordingly, the anesthesiologist needs to identify which patients are most at risk, and therefore which patients can be managed as “day cases,” what is an appropriate anesthetic regimen, and how best to monitor these patients postoperatively.

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33

Rogula, Tomasz G., Adriana Martin, and Ivan Alberto Zepeda Mejia. Obstructive Sleep Apnea. Edited by Tomasz Rogula, Philip Schauer, and Tammy Fouse. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190608347.003.0018.

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Obstructive sleep apnea (OSA) is highly prevalent in morbidly obese patients, although it is surprisingly underdiagnosed and undertreated. OSA can increase the risk of serious and life-threating complications in the perioperative period of bariatric surgery. Nevertheless, this potential risk can be minimized with adequate preoperative screening and perioperative management. The perioperative management of patients with OSA will affect the preparation for surgery, airway management, anesthetic selection, and monitoring. This chapter discusses and presents the best evidences available for the management of patients with OSA in order to decrease both the prevalence of undiagnosed patients and the morbidity associated with bariatric surgery.

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34

Ryan, Laura, and Paul Hopkins. Obstructive Sleep Apnea. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0011.

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Adenotonsillectomy is one of the most commonly performed surgeries in children and is the mainstay treatment for obstructive sleep apnea (OSA). Children with OSA have a higher risk of perioperative respiratory morbidity. Diagnosis of OSA is made by overnight polysomnography, but this resource is rare and expensive so children at risk for OSA must be identified based on parental history. Patients with risk factors for postoperative respiratory complications may need to be monitored in the hospital overnight. Anesthetic challenges associate with adenotonsillectomy include perioperative analgesia, prevention and treatment of postoperative nausea and vomiting, risk of airway fire, and management of airway obstruction.

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35

A, Dosman James, and Cockcroft Donald W, eds. Obstructive lung diseases. Philadelphia: Saunders, 1996.

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36

Barbé, Ferran, and Jean-Louis Pépin, eds. Obstructive Sleep Apnoea. European Respiratory Society, 2015. http://dx.doi.org/10.1183/2312508x.erm6715.

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37

Kushida, Clete A. Obstructive Sleep Apnea. CRC Press, 2007. http://dx.doi.org/10.1201/9781420061819.

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38

Obstructive Sleep Apnea. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03936-079-6.

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39

Kushida, Clete A. Obstructive Sleep Apnea. Taylor & Francis Group, 2007.

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40

1960-, Kushida Clete Anthony, ed. Obstructive sleep apnea. New York, NY: Informa Healthcare USA, 2007.

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41

Kreit, John W. Severe Obstructive Lung Disease. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0013.

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Although chronic obstructive lung disease, asthma, bronchiectasis, and bronchiolitis have very different causes, clinical features, and therapies, they share the same underlying pathophysiology. They are referred to as obstructive lung diseases because airway narrowing causes increased resistance and slowing of expiratory gas flow. Mechanical ventilation of patients with severe obstructive lung disease often produces two problems that must be recognized and effectively managed: over-ventilation and dynamic hyperinflation. Severe Obstructive Lung Disease reviews these two major adverse consequences of mechanical ventilation in patients with severe air flow obstruction. The chapter explains how to detect and correct both of these problems and provides guidelines for managing patients with respiratory failure caused by severe obstructive lung disease.

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42

1932-, Petty Thomas L., ed. Chronic obstructive pulmonary disease. 2nd ed. New York: Dekker, 1985.

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43

Rennard, Stephen I., Robert A. Stockley, Klaus Rabe, and Bartolome Celli. Chronic Obstructive Pulmonary Disease. Wiley & Sons, Incorporated, John, 2008.

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44

National Heart, Lung, and Blood Institute. Division of Lung Diseases and National Heart, Lung, and Blood Institute. Office of Prevention, Education, and Control, eds. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: National Institutes of Health, National Heart, Lung, and Blood Institute, 1993.

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45

(Editor), William Macnee, and Norbert F. Voelkel (Editor), eds. Chronic Obstructive Lung Disease. BC Decker Inc., 2002.

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46

F, Voelkel Norbert, and MacNee William, eds. Chronic obstructive lung diseases. Hamilton, Ont: BC Decker, 2002.

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47

1939-, Hodgkin John E., ed. Chronic obstructive pulmonary disease. Philadelphia: W. B. Saunders, 1990.

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48

National Heart, Lung, and Blood Institute, ed. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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49

Rennard, Stephen I., Robert A. Stockley, Klaus Rabe, and Bartolome Celli. Chronic Obstructive Pulmonary Disease. Wiley & Sons, Incorporated, John, 2008.

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50

National Heart, Lung, and Blood Institute., ed. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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