Academic literature on the topic 'Obésité sarcopénique'
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Journal articles on the topic "Obésité sarcopénique"
Tardif, Nicolas, Jérôme Salles, Christelle Guillet, Emilie Gadéa, Yves Boirie, and Stéphane Walrand. "Obésité sarcopénique et altérations du métabolisme protéique musculaire." Nutrition Clinique et Métabolisme 25, no. 3 (September 2011): 138–51. http://dx.doi.org/10.1016/j.nupar.2011.07.004.
Full textBoirie, Yves, Christelle Guillet, Mélanie Pouget, Stéphane Walrand, and Alexandre Pinel. "Obésité sarcopénique : des étapes décisives et des perspectives prometteuses." Nutrition Clinique et Métabolisme 37, no. 2 (December 2023): 2S34–2S39. http://dx.doi.org/10.1016/s0985-0562(24)00009-8.
Full textWalrand, S., and Y. Boirie. "Obésité sarcopénique: « parle à mon gras, mon muscle est malade »!" Obésité 2, no. 4 (December 2007): 331–38. http://dx.doi.org/10.1007/s11690-007-0088-y.
Full textMillet, A., A. Greil, F. Costes, Y. Boirie, N. Farigon, E. Gentes, B. Pereira, and D. Caillaud. "Étude de la sarcopénie dans le syndrome obésité-hypoventilation. Une comparaison cas témoins (SOH+/−)." Revue des Maladies Respiratoires Actualités 14, no. 1 (January 2022): 56. http://dx.doi.org/10.1016/j.rmra.2021.11.541.
Full textDissertations / Theses on the topic "Obésité sarcopénique"
Zambon, Azevedo Vittoria. "Mise au point d’un nouvel outil diagnostique de l’obésité sarcopénique : relations avec la dysfonction du tissu adipeux, l’insulinorésistance et la sévérité de l’atteinte hépatique." Electronic Thesis or Diss., Sorbonne université, 2024. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2024SORUS170.pdf.
Full textSarcopenic obesity (SO) is a condition characterized by the coexistence of obesity and sarcopenia, the latter defined as a reduction in muscle mass and function. Diagnosing SO is highly complex due to the lack of universally accepted diagnostic criteria, leading to imprecise diagnoses and highly variable prevalence estimates. Given this scenario, this thesis aimed to develop an empirical diagnostic tool for SO using artificial intelligence, based on the analysis of body composition. We developed the AIM-SO score in a population of patients with overweight/obesity and tested it in two other populations: patients with severe obesity undergoing bariatric surgery (BS) and the general population of the UK Biobank. A longitudinal study with a one-year follow-up was conducted in subjects who underwent BS. We examined clinical correlations, particularly cardiometabolic and hepatic, including perioperative histological findings in the bariatric cohort. The prevalence of SO was similar across these three cohorts. SO diagnosed by the AIM-SO score was associated with multiple cardiometabolic comorbidities and more severe inflammatory and fibrosing liver damage. Despite weight loss, the metabolic benefit (remission of comorbidities) after BS was lower in patients with SO. Preliminary analyses of the UK Biobank cohort showed a significant association between SO diagnosed by the AIM-SO score and parameters of muscular functionality, particularly muscle strength. We propose this new diagnostic tool to standardize the SO diagnosis and identify patients with obesity and sarcopenia who exhibit more severe cardiometabolic and hepatic phenotype. Diagnosing SO could also inform the expected benefit of various weight loss interventions, thus contributing to personalized medical management
Masgrau, Aurélie. "Caractérisation du métabolisme protéique musculaire au cours de l'obésité et lors de la perte de poids." Thesis, Clermont-Ferrand 1, 2012. http://www.theses.fr/2012CLF1MM07.
Full textObesity - characterized by lipid accumulation in adipose tissue and in peripheral tissues such as liver and skeletal muscles - leads to metabolic dysfunction of these tissues. In the long term, although it is frequently reported an increase in lean mass, obesity is accompanied by a loss of muscle mass. Weight loss has a positive impact on comorbidities associated with obesity. However, when it was induced by dietary restriction, it may be associated with muscle mass loss. The association of physical activity to food restriction may limit muscle mass loss. Metabolically, muscle mass depends essentially on proteins turnover, i.e. protein synthesis and breakdown. Therefore, the aim of the thesis work was to characterize changes in muscle protein metabolism, especially changes in protein synthesis, during obesity development and weight loss induced by a low-fat-diet with or without endurance exercise. The first study has shown that there are two distinct phases in the development of obesity in rats. The first is associated with body weight and muscle mass gains and an increase in myofibrillar and mitochondrial proteins synthesis rate (FSR), specifically in glycolytic muscle tibialis anterior, in postabsorptive state. Oxidative muscle soleus was not affected. The second phase is associated with body weight stabilization, reduced muscle mass and a decrease in the mitochondrial proteins FSR in the tibialis anterior. The second study has shown that isocaloric low-fat-diet or the practice of regular endurance exercise do not prevent muscle mass loss induced by obesity, unlike the combination of both treatments. Exercise alone or associated with high-fat diet stimulates the FSR of myofibrillar proteins actin in tibialis anterior muscle, but exercise stimulates the FSR of myofibrillar and mitochondrial proteins in the oxidative muscle soleus only when it is associated with lipid restriction. In conclusion, this study has shown firstly that muscle protein synthesis in postabsorptive state and muscle mass are differently affected depending on the stage of obesity development, and, secondly that muscle protein synthesis in postabsorptive state is differently affected depending on muscle typology. On the other hand, exercise has a beneficial effect on muscle mass and protein synthesis, but this "anabolic" effect is limited by the high-fat, high-sucrose diet. To transpose these data in humans, a clinical study that examines the effect of weight loss induced by bariatric surgery on muscle protein metabolism has been established and is currently underway
Rolland, Yves. "Activité physique, performances fonctionnelles et santé chez la personne âgée." Toulouse 3, 2005. http://www.theses.fr/2005TOU30004.
Full textEffective interventions such as physical activity leading to disability prevention in the elderly are a public health priority. Disability outcome measures should ideally include objective measures such as physical performance tests. This work demonstrates the test-retest reliability of a new walking test (the 400-m usual pace walk test), the association between calf circumference and muscle-related disability and the predictive value of the short physical performance battery on mortality. Physical activity appears to be an effective strategy to delay disability. This work adds further evidence, especially in the frail elderly population (patients with Alzheimer's disease, obesity or malnutrition) of its health benefits
Book chapters on the topic "Obésité sarcopénique"
Boirie, Y. "Obésité sarcopénique." In Les Obésités, 371–74. Elsevier, 2021. http://dx.doi.org/10.1016/b978-2-294-76753-1.00067-9.
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