Academic literature on the topic 'Nystagmus'

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Journal articles on the topic "Nystagmus"

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Apaev, Aleksander V. "Clinical pathology and diagnostics of nystagmus." Russian Pediatric Ophthalmology 16, no. 4 (December 15, 2021): 37–46. http://dx.doi.org/10.17816/rpoj84461.

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Optical nystagmus is an involuntary oscillatory movement. Nystagmus can occur as an isolated nosology and lead to decreased visual functions in combination with other pathologies. A decreased visual function, including visual acuity, is observed in many patients with optic nystagmus. Patients with optical nystagmus were noted by ophthalmologists with a significant difference in the acuity of monocular and binocular vision, which is much higher in the second case. As a rule, a difference is observed in visual acuity in the straight and forced position of the head. Frequently, nystagmus is combined with refractive errors. Some authors consider astigmatism the most common form of ametropia in nystagmus, and nystagmus itself, a predisposing factor for the presence of astigmatism. An important task in diagnostics is the quantitative eye movement assessments since the tactics of treatment and the prognosis of the course of the disease depend on this. One of the problems in the examination of patients with nystagmus is the assessment of the fundal condition. Modern optical coherence tomographs are helpful in the objective diagnostics of retinal structures of the posterior pole of the eye, as well as improved software, such as the Eye Tracking system, and an increased scanning speed allowed reliable studies even with spontaneous oscillatory eye movements and a significantly decreased visual acuity. With nystagmic relative amblyopia, both photosensitivity and fixation were violated, which may indicate the presence of organic changes in the central parts of the retina and a primary fixation violation.
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Helmchen, Christoph, Wolfgang Heide, Michael Strupp, and Dominik Straumann. "Okulomotorikstörungen und Nystagmus." Nervenheilkunde 42, no. 01/02 (February 2023): 50–58. http://dx.doi.org/10.1055/a-1946-6812.

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ZUSAMMENFASSUNGDer Diagnose von Augenbewegungsstörungen und der Nystagmusformen beruht auf einer systematischen klinischen Untersuchung aller Arten von Augenbewegungen. Diese Untersuchung umfasst: Augenposition, Untersuchung auf einen Spontannystagmus, Motilität, Blickfolge, Blickhaltefunktion, Sakkaden, Vergenzreaktion, optokinetischer Nystagmus, Funktion des vestibulookulären Reflexes (VOR) sowie die Fixationssuppression des VOR. Anatomisch relevante Strukturen sind Mesenzephalon, Pons, Medulla oblongata, Zerebellum und Kortex. Topografisch anatomisch gelten die einfachen klinischen Regeln: Vertikale und torsionale Augenbewegungen werden vorwiegend im Mesenzephalon und horizontale Augenbewegungen in dem Pons generiert. Typische Zeichen einer Mittelhirnläsion sind vertikale Sakkaden- oder Blickparese, ein isolierter vertikaler Blickrichtungsnystagmus und einer Ponsläsion entsprechende horizontale Störungen. Das Zerebellum spielt eine Rolle bei praktisch allen Augenbewegungen; typische klinische Zeichen sind eine allseitige Blickfolgesakkadierung, Blickrichtungsnystagmus oder dysmetrische Sakkaden.Unter einem Nystagmus versteht man rhythmische Augenbewegungen, die in der Regel aus einem langsamen (ursächlichen bzw. pathologischen) Augendrift und einer schnellen kompensatorischen Rückstellbewegung (Rückstellsakkade) bestehen. Es lassen sich 3 einfache Kategorien unterscheiden: Spontannystagmus, d. h. ein Nystagmus, der bei Fixation in Geradeaus-Blickposition auftritt, Nystagmusformen, die nur in Abhängigkeit von der Blickrichtung auftreten und Nystagmen, die nur durch bestimmte Manöver ausgelöst werden: Kopfschütteln, Lagerung, Hyperventilation oder physikalischen Druck (z. B. Pressen). Letztere sind oft durch peripher-vestibuläre Läsionen ausgelöst, können aber auch zentralen Ursprungs sein. Viele zentrale Nystagmusformen erlauben eine genaue anatomische Lokalisation, z. B. der Downbeat-Nystagmus (DBN), der meistens auf einer Flocculus-Läsion beruht oder der Upbeat-Nystagmus (UBN) auf einer Läsion im Mesencephalon oder der Medulla oblongata. Beispiele einer Pharmakotherapie sind die Gabe von 4-Aminopyridin beim DBN und UBN, Memantin oder Gabapentin beim Fixationspendelnystagmus oder Baclofen beim periodisch-alternierenden Nystagmus.
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Spielmann, A., and A. C. Spielmann. "Nystagmus congénital. Nystagmus manifeste/latent. Nystagmus acquis." EMC - Ophtalmologie 2, no. 4 (January 2005): 1–16. http://dx.doi.org/10.1016/s0246-0343(05)40793-5.

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Spielmann, A., and A. C. Spielmann. "Nystagmus congénital. Nystagmus manifeste/latent. Nystagmus acquis." EMC - Ophtalmologie 2, no. 4 (November 2005): 282–304. http://dx.doi.org/10.1016/j.emcop.2005.08.001.

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Imai, Takao. "Infantile Nystagmus (Congenital Nystagmus)." Practica Oto-Rhino-Laryngologica 117, no. 4 (2024): 297–302. http://dx.doi.org/10.5631/jibirin.117.297.

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Seemungal, B. M., I. A. Gunaratne, I. O. Fleming, M. A. Gresty, and A. M. Bronstein. "Perceptual and nystagmic thresholds of vestibular function in yaw." Journal of Vestibular Research 14, no. 6 (December 1, 2004): 461–66. http://dx.doi.org/10.3233/ves-2004-14604.

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A technique for simultaneous measurement of conscious (perceptual) and reflex (nystagmus) thresholds of vestibular function is described. We used an automated modified binary search algorithm with simultaneous infrared oculography in determining perceptual and VOR nystagmic thresholds respectively, during discrete whole body rotations in the dark. In a young group of 14 normal subjects (mean age 23 years) angular acceleration thresholds were significantly higher for perceptual detection (1.18 deg/s/s) than for nystagmus generation (0.51 deg/s/s). Only nystagmic thresholds were slightly raised (0.87 deg/s/s) in an older group of 9 normal subjects (mean age 63 years). The finding that nystagmic thresholds are lower than perceptual ones indicates a higher sensitivity of brainstem than cortical vestibular mechanisms. This technique would be of particular value in clinical situations where a dissociation between reflex and conscious vestibular mechanisms is expected, e.g. in patients with cortical lesions or in elderly patients with falls.
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Ishikawa, Kaoru, Jiro Hozawa, Shigeki Kamata, Keiji Fukuoka, Shuji Ohta, Hiroaki Ichijo, Keiichi Ikeno, and Masaaki Tazawa. "Characteristic difference between irritative nystagmus and paralytic nystagmus - Analysis of spontaneous nystagmus and positional nystagmus." Equilibrium Research 45, no. 2 (1986): 91–100. http://dx.doi.org/10.3757/jser.45.91.

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Ohki, Masafumi. "Head-shaking nystagmus test." Equilibrium Research 76, no. 6 (2017): 698–701. http://dx.doi.org/10.3757/jser.76.698.

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Kates, Malcolm M., and Casey J. Beal. "Nystagmus." JAMA 325, no. 8 (February 23, 2021): 798. http://dx.doi.org/10.1001/jama.2020.3911.

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Jacobson, Daniel M., and James J. Corbett. "Nystagmus." Seminars in Ophthalmology 2, no. 3 (January 1987): 183–208. http://dx.doi.org/10.3109/08820538709068803.

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Dissertations / Theses on the topic "Nystagmus"

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Spiegel, Rainer. "Downbeat nystagmus." Diss., lmu, 2011. http://nbn-resolving.de/urn:nbn:de:bvb:19-140001.

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Swart, Wayne. "Nystagmus and eye reflex sensor." Thesis, Stellenbosch : Stellenbosch University, 2011. http://hdl.handle.net/10019.1/18019.

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Thesis (MScEng)--Stellenbosch University, 2011.
ENGLISH ABSTRACT: Nystagmus is an eye movement pattern that consists of a drifting gaze component, known as the slow phase, followed by a corrective quick phase component. The presence of nystagmus or the lack thereof under certain conditions can be used for various diagnostic purposes including the diagnosis of physiological, pathological and neurological conditions. The angular velocity of the quick phase can make the detection of nystagmus a challenging task for the untrained eye, since the quick phases are usually comparable with saccadic eye motions. The goal is thus to develop a fully automated diagnostic tool that can identify the presents of nystagmus in a patient’s eye motions. In this thesis, an appropriate eye tracking method was selected from a number of eye tracking methods that are commonly implemented in the literature. A video-oculography goggle concept was chosen based on criteria such as invasiveness, sampling rate, accuracy and telemedicine capability, amongst other nystagmus related necessities. A binocular video-oculography concept was chosen that satisfied the technical requirements and provided a cost-effective design. An automated analysis algorithm was developed for automatic nystagmus identification from eye motion data. The algorithm was validated by testing the performance of the algorithm on an optokinetic nystagmus signal. It proved to provide a reliable automatic identification of nystagmus beats, even in signals that contained nystagmus as well as random motion components. A statistical analysis showed that the algorithm provided a sensitivity of 91.8% and a specificity of 96.5% for pure nystagmus signals, and a sensitivity and specificity of 87.8% and 91.1% respectively for mixed signals.
AFRIKAANSE OPSOMMING: Nystagmus is ’n oogbewegingspatroon wat bestaan uit ’n dwalende tuurkomponent, wat die stadige fase genoem word, gevolg deur ’n vinnige korrigereringsbeweging wat bekend staan as die vinnige fase. Die teenwoordigheid van nystagmus, of afwesigheid daarvan in sekere gevalle, kan gebruik word in ’n verskeidenheid diagnostiese toepassings, onder andere die diagnose van fisiologiese-, patalogiese- en neurologiese kwale. Die hoeksnelheid van die vinnige fase lei daartoe dat nystagmus dikwels moeilik is om te bespeur vir ongeöefende oë, aangesien dit vergelykbaar is met saccade bewegings. Die doel van hierdie navorsing is dus die ontwikkeling van ’n stelsel wat ’n volledige automatiese identifisering van nystagmus kan behartig. ’n Gepaste oogvolgtegniek was gekies vanuit ’n aantal verskillende oogvolgmetodes wat dikwels in die praktyk gebruik word. Die finale keuse was ’n skermbril, video-oogvolgmetode wat gekies was op grond van kriteria soos onder andere, invallendheid, meetfrekwensie, akkuraatheid en geskiktheid vir telemedisyne toepassings. Die ontwikkelde brilkonsep bied ’n koste-effektiewe oplossing, met die moontlikheid om albei oë te volg en bevredig al die bogenoemde tegniese spesifikasies. ’n Geoutomatiseerde nystagmus identifiseringsalgoritme is ontwikkel. Die algoritme se effektiwiteit is getoets op optokinetiese nystagmusseine. Betroubare resultate is vekry vanaf die algoritme, selfs in die geval van gemengde seine wat bestaan uit arbritrêre- en nystagmus komponente. Statistiese analiese het gewys dat die algoritme ’n sensitiwiteit van 91.8% en ’n spesifisiteit van 96.5% kon behaal vir seine met slegs nystagmus inhoud. Vir gemengde inhoud seine het die algoritme ’n sensitiwiteit van 87.8% en spesifisiteit van 91.1% behaal.
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Harrison, James J. "Volition and automaticity in the interactions of optokinetic nystagmus, infantile nystagmus, saccades and smooth pursuit." Thesis, Cardiff University, 2014. http://orca.cf.ac.uk/59168/.

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Volitional target-selecting eye movements, such as saccades or smooth pursuit, are frequently considered distinct and separate from automatic gaze-stabilising eye movements like optokinetic nystagmus or the vestibulo-ocular reflex. This difference is regularly mapped onto brain anatomy, with distinctions made between subcortical, automatic processes; and cortical, volitional ones. However gaze-stabilising and target-selecting eye movements must work together when a moving observer views natural scenes. Yet such co-ordination would not be possible if automatic and volitional actions are sharply divided. This thesis focuses upon interactions between gaze-stabilising and target-selecting eye movements, and how these interactions can aid our understanding of the relationship between automatic and volitional processes. For a saccade executed during optokinetic nystagmus to accurately land on target, it must compensate for the ongoing optokinetic movement. It was found that targeting saccades can partially compensate for concomitant optokinetic nystagmus. The degree of compensation during optokinetic nystagmus was indistinguishable from compensation due to voluntary smooth pursuit displacements. A subsequent experiment found that locations are similarly misperceived during optokinetic nystagmus and smooth pursuit. Furthermore, saccade end-points are subject to the same perceptual mislocalisations. The next experiment established that fast-phases of optokinetic nystagmus can act like competitive saccades and cause curvature in targeting saccades. Moreover, optokinetic nystagmus fast-phases are delayed by irrelevant visual distractors in the same way as saccades (the saccadic inhibition effect). Lastly, it was established that the fast-phases of Infantile Nystagmus Syndrome also show the saccadic inhibition effect. In conclusion, target-selecting and gaze-stabilising eye movements show substantial co-ordination. Furthermore these results demonstrate considerable commonalties between ‘automatic’ and ‘volitional’ eye movements. Such commonalities provide further evidence there is no sharp distinction between automatic and volitional processes. Instead it is likely there are substantial interconnections between automatic and volitional mechanisms, and volition has a graded influence upon behaviour.
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Robert, Matthieu. "Étiopathogénie des nystagmus verticaux du nourrisson." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB008/document.

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L’étude des nystagmus du nourrisson est difficile pour des raisons méthodologiques. La plupart des études sont donc réalisées chez des adultes et a posteriori. Ces études chez l’adulte ont permis la révision des classifications des nystagmus infantiles, avec la consolidation des concepts de syndrome du nystagmus précoce et de nystagmus de type latent. La séméiologie et l’étiopathogénie des nystagmus du nourrisson – notamment des formes comportant un élément vertical et des formes transitoires – reste donc à étudier. Dans ce but : 1. Nous avons développé de nouvelles techniques d’enregistrement des mouvements oculaires chez les nourrissons, utilisables dans le cadre d’une consultation. Elles incluent de nouveaux stimuli, un appareil d’enregistrement des mouvements oculaires spécialement conçu pour le nourrisson et de nouvelles méthodes d’analyse statistique du signal. Nous avons vérifié la faisabilité de ces enregistrements chez 28 nourrissons atteints de nystagmus. 2. Nous avons étudié systématiquement 32 cas de nystagmus de type spasmus nutans, classiquement considéré comme une entité bénigne idiopathique, avec un examen clinique complet, une imagerie cérébrale, une électrophysiologie visuelle et des enregistrements oculo-moteurs. Dans 53,1% des cas, le spasmus nutans était le symptôme d’une autre maladie : neurologique (34,3%), notamment des gliomes du chiasma (21,9%), ou rétinienne (12,5%). Une atteinte des voies visuelles antérieures est probablement en cause dans la physiopathologie des spasmus nutans. 3. Huit cas de nystagmus ayant conduit au diagnostic de gliome des voies optiques (GVO) ont été également enregistrés et étudiés. L’âge d’apparition du nystagmus allait de 2,5 à 10 mois. Le GVO était toujours chiasmatique et constituait une sous-population spécifique. Le nystagmus était toujours de type spasmus nutans. Les enregistrements oculo-moteurs montraient : une fréquence entre 2,7 et 5 Hz, une morphologie sinusoïdale du nystagmus, une dissociation et une dysconjugaison particulière, avec une opposition de phase (180°) entre les oscillations des deux yeux dans le plan horizontal mais une correspondance de phase dans le plan vertical, à l’origine d’un mouvement semblable à un mouvement de convection. Rarement et brièvement, le rapport de phase changeait. Ces caractéristiques orientent vers des oscillations dans le système des vergences, possiblement la conséquence d’une atteinte des afférences sensorielles des centres du contrôle vergentiel dans le tronc cérébral, secondaire au GVO et survenant pendant la période sensible du développement visuel. 4. Cinq cas de nystagmus upbeat chez des nourrissons avec des rétines et une imagerie cérébrale normale ont été étudiés. Le nystagmus était observé en décubitus et électivement déclenché par des rotations de la tête en position allongée. Dans tous les cas, une résolution spontanée était observée après quelques mois d’évolution. Les caractéristiques de ce type de nystagmus suggèrent une participation du système otolithique, suivie d’une recalibration secondaire des circuits vestibulo-oculaires. En conclusion, le développement de techniques d’enregistrement des mouvements oculaires adaptées aux nourrissons aide à la compréhension de l’étiopathogénie de variétés mal décrites de nystagmus, notamment dans les cas comportant un élément vertical et dans les cas transitoires. Les processus de maturation des voies visuelles antérieures et des centres de contrôle de l’oculomotricité semblent jouer un rôle central dans les mécanismes de ces nystagmus
Studying infantile nystagmus during infancy is difficult for methodological reasons. Most such studies have been performed in adults and a posteriori. These studies in adults allowed for an improvement in the existing classifications, with now robust knowledge about the two most frequent varieties of infantile nystagmus: infantile nystagmus syndrome and fusion maldevelopment nystagmus syndrome. The characteristics and pathophysiology of nystagmus in infants–notably varieties of nystagmus with a vertical component and transitory nystagmus– need further study. For this reason: 1. We developed new techniques for the recording of eye movements in infants in the setting of a clinic. They include new stimuli, the use of specially-designed infrared photo-oculography eyetrackers and new statistical analysis paradigms. We assessed these techniques in a population of 28 infants with a nystagmus. 2. We systematically studied 32 cases of spasmus nutans, classically considered an idiopathic entity, with comprehensive clinical examination, brain imaging, electrophysiology, nystagmus recording. In 53.1% of cases, it led to the diagnosis of another condition: a neurological disease (34.3%), including cases of chiasmal gliomas (21.9%), or a retinal dysfunction (12.5%). Anterior visual pathway dysfunction is likely involved in the pathophysiology of spasmus nutans. 3. Eight cases of nystagmus having led to a diagnosis of optic pathway glioma (OPG) were also recorded and studied. Age at nystagmus onset was 2.5-10 months. The associated OPG always involved the chiasm, and represent a specific subpopulation of OPG. Clinically, the nystagmus was always classified as spasmus nutans type. Oculographic recordings showed frequencies of 2.7-5 Hz, sinusoidal waveforms, dissociation and a special type of disconjugacy, with a 180° horizontal phase shift and no vertical phase shift, exhibiting a “convection-like” movement pattern. Rarely and for short periods of time, the phase shift could change. These characteristics point towards oscillations in the vergence system, which could possibly result from the specific disruption of the vergence centres afferences in the brainstem, induced by the OPG during the sensitive period of visual development. 4. Five cases of upbeat nystagmus in infants with normal retinas and normal brain imaging were studied. The nystagmus mostly occurred in supine position and could be triggered by head rotations in the supine position. All resolved spontaneously. The characteristics of this nystagmus suggest an involvement of the otolithic system, with a secondary recalibration of the vestibulo-ocular pathways. In conclusion, the development of infant-friendly devices for eye-movements recording helps providing new insights on the pathophysiology of poorly described varieties of nystagmus, including nystagmus with a vertical component and transitory nystagmus. The maturation process of both the anterior visual pathways and the oculomotor pathways appears to be central in the mechanisms of these nystagmus
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Gadaud, Antoine Élie. "Étude sur le nystagmus thèse pour le doctorat en médecine présentée et soutenue le 29 avril 1869 /." Paris : BIUM, 2003. http://www.bium.univ-paris5.fr/histmed/medica/cote?TPAR1869x097.

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Radinsky, Iliya. "Improved algorithm for classification of nystagmus." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=81561.

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An improved algorithm for classification of nystagmus was designed allowing the sorting of response segments even in severely non-linear patients and subjects with abnormally large phase shifts. The algorithm employs a model-based approach that was developed by Rey and Galiana (1991). The improved classification algorithm consists of two essential stages. In the first stage the eye velocity response is classified to obtain best possible estimates of the slow phase eye velocity intervals. In the second stage, the slow phase estimates are used to identify a response phase shift and non-linearity, and compensate for their effects. Multiple tests on simulated data and experimental data obtained from clinical subjects are presented. The results of the tests demonstrate that the algorithm is able to analyze the patient data with a high accuracy even in the presence of noise, eye-blinks and other artifacts.
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Waddington, Jonathan. "Human optokinetic nystagmus : a stochastic analysis." Thesis, University of Plymouth, 2012. http://hdl.handle.net/10026.1/1040.

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Optokinetic nystagmus (OKN) is a fundamental gaze-stabilising response in which eye movements attempt to compensate for the retinal slip caused by self-motion. The OKN response consists of a slow following movement made in the direction of stimulus motion interrupted by fast eye movements that are primarily made in the opposite direction. The timing and amplitude of these slow phases and quick phases are notably variable, but this variability is poorly understood. In this study I performed principal component analysis on OKN parameters in order to investigate how the eigenvectors and eigenvalues of the underlying components contribute to the correlation between OKN parameters over time. I found three categories of principal components that could explain the variance within each cycle of OKN, and only parameters from within a single cycle contributed highly to any given component. Differences found in the correlation matrices of OKN parameters appear to reflect changes in the eigenvalues of components, while eigenvectors remain predominantly similar across participants, and trials. I have developed a linear and stochastic model of OKN based on these results and demonstrated that OKN can be described as a 1st order Markov process, with three sources of noise affecting SP velocity, QP triggering, and QP amplitude. I have used this model to make some important predictions about the optokinetic reflex: the transient response of SP velocity, the existence of signal dependent noise in the system, the target position of QPs, and the threshold at which QPs are generated. Finally, I investigate whether the significant variability within OKN may represent adaptive control of explicit and implicit parameters. iii
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Sarvananthan, Nagini. "Epidemiology and clinical study of nystagmus." Thesis, University of Leicester, 2012. http://hdl.handle.net/2381/10383.

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Nystagmus is a repetitive to and fro movement of the eyes and can affect vision and involve individuals of all ages. Previous research into the pathophysiology of this disease has been based on case series or small numbers of patients. Improvements in and standardisation of electrodiagnostics and eye movement recordings have enabled scientists to diagnose and characterise the different nystagmus types more accurately. Purpose The research into nystagmus carried out at the University of Leicester had several aims. The first population-based study on the prevalence of nystagmus was carried out within the county of Leicestershire. The second study was aimed at examining the clinical features of patients with different types of infantile and neurological nystagmus in order to characterise any specific features associated with this groups of patients. The final study was carried out with the aim of investigating the distribution of refractive errors in patients with nystagmus and to determine if the process of emmetropization in ocular development is influenced by the presence of nystagmus. Methods Ethical approval was obtained. Patients were recruited for the epidemiological study from both the community and hospitals within Leicestershire. Patients for the clinical and refractive error studies were additionally recruited from outside the county. A further 602 normal subjects volunteered to participate in the refraction study. Results The epidemiological study estimates the prevalence of nystagmus to be 16.6 per 10 000 (under 18 population) and 26.5 per 10000 (over 18 population). The clinical study showed differences in visual acuity, stereopsis, anomalous head posture and conjugacy of nystagmus amongst different clinical groups. Finally, the refractive errors study suggests that the process of emmetropization is influenced by the presence of nystagmus. Conclusion These studies provide previously unknown data about nystagmus and provide a platform for further research into this condition.
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Worfolk, Ralph. "Ocular-motor control in congenital nystagmus." Thesis, University of Manchester, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310353.

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Wiblishauser, Kathrin. "Untersuchungen zum auslösenden Rezeptor des vibrationsinduzierten Nystagmus." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=969343574.

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Books on the topic "Nystagmus"

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Royal National Institute for the Blind. Understanding nystagmus. London: RNIB, 2000.

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Hertle, Richard W. Nystagmus in infancy and childhood: Current concepts in mechanisms, diagnoses, and management. New York: Oxford University Press, 2012.

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D, Harris Christopher Ph, and Nystagmus Network, eds. Early-onset nystagmus: A parents' guide. London: Nystagmus Network, 1998.

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Mora, Clare. Congenital Nystagmus in infants and children. Manchester: UMIST, 1998.

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S, Lessard Charles, and United States. National Aeronautics and Space Administration., eds. Characterization of slow and fast phase nystagmus. College Station, TX: Industrial Engineering Dept., Texas A&M University, 1991.

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Whittle, J. The nature of head postures in congenital nystagmus. Manchester: UMIST, 1990.

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Brodsky, Michael C. The Evolutionary Basis of Strabismus and Nystagmus in Children. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-62720-1.

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A, Sharpe James, and Barber Hugh O, eds. The Vestibulo-ocular reflex and vertigo. New York: Raven Press, 1993.

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John, Leigh R., and Devereaux Michael W, eds. Advances in understanding mechanisms and treatment of infantile forms of nystagmus. Oxford: Oxford University Press, 2008.

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Ronald, Hinchcliffe, and Stephens S. D. G, eds. Measurement in hearing and balance: Papers in honour of Ronald Hinchcliffe. Basel: Karger, 1988.

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Book chapters on the topic "Nystagmus"

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Irani, Farzin, and Ketharini Sivasegaran. "Nystagmus." In Encyclopedia of Clinical Neuropsychology, 1800–1802. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1385.

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Irani, Farzin, and Ketharini Sivasegaran. "Nystagmus." In Encyclopedia of Clinical Neuropsychology, 1–4. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1385-2.

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Mammen, Alex, Sirikishan Shetty, and Dilip A. Thomas. "Nystagmus." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 1886–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_877.

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Irani, Farzin, and Ketharini Sivasegaran. "Nystagmus." In Encyclopedia of Clinical Neuropsychology, 2486–88. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1385.

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Henn, Volker. "Nystagmus." In Sensory System I, 37. Boston, MA: Birkhäuser Boston, 1988. http://dx.doi.org/10.1007/978-1-4899-6647-6_19.

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Ehrt, O. "Nystagmus." In Pädiatrische Differenzialdiagnostik, 261–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-29798-4_63.

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Shetty, Shashikant, and Anshulee Sood. "Nystagmus." In Neuro-ophthalmic Disorders, 85–101. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8522-3_6.

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von Albert, Hans-Henning. "Nystagmus." In Vom neurologischen Symptom zur Diagnose, 180–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56278-5_57.

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Tian, Guohong, Xinghuai Sun, and Chaoyi Feng. "Nystagmus." In Neuro-Ophthalmology, 491–507. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-4668-4_21.

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von Albert, Hans-Henning. "Nystagmus." In Vom neurologischen Symptom zur Diagnose, 187–90. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-96923-2_57.

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Conference papers on the topic "Nystagmus"

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Clement, Richard A. "Periodic Forcing of Congenital Nystagmus." In EXPERIMENTAL CHAOS: 6th Experimental Chaos Conference. AIP, 2002. http://dx.doi.org/10.1063/1.1487529.

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Coleman, Chase, Matthew Jenkins, William Roberts, Charlie Thomas, William Westerkamp, Rod MacDonald, and Ahmad Salman. "Horizontal Gaze Nystagmus Transmission Interlock System." In 2023 Systems and Information Engineering Design Symposium (SIEDS). IEEE, 2023. http://dx.doi.org/10.1109/sieds58326.2023.10137888.

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Usanova, Tatjana B., Anatoli V. Skripal, Dmitry A. Usanov, and Anton V. Abramov. "Computer video diagnostics of eye nystagmus." In Biomedical Optics 2005, edited by Valery V. Tuchin. SPIE, 2005. http://dx.doi.org/10.1117/12.596872.

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Yun-shan Chen, Hui-bin Gao, Wan-xin Su, and Min Wang. "Grayscale control algorithm based on optokinetic nystagmus." In 2010 3rd International Conference on Advanced Computer Theory and Engineering (ICACTE 2010). IEEE, 2010. http://dx.doi.org/10.1109/icacte.2010.5579197.

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Ben Slama, Amine, Aymen Mouelhi, Mohamed Ali Cherni, Sondes Manoubi, Chiraz Mbarek, Hedi Trabelsi, and Mounir Sayadi. "Features extraction for medical characterization of nystagmus." In 2016 2nd International Conference on Advanced Technologies for Signal and Image Processing (ATSIP). IEEE, 2016. http://dx.doi.org/10.1109/atsip.2016.7523094.

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Ximenes, Marcelo Tognato, Dan Mohamed Salman, Francisco Tomaz Meneses de Oliveira, and Rubens José Gagliardi. "Downbeat Nystagmus Secondary to Chiari I Malformation." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.374.

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Context: Downbeat nystagmus are characterized by the rapid phase in the down direction in primary position of the eyes, in the most cases represents a cerebellar dysfunction, typically with injuries involving vestibulocerebellum (flocculus, paraflocculus, nodule and uvula), although most cases are due to primary lesions in the brain stem, usually involving paramedian tracts. This type of nystagmus is characteristic of craniocervical abnormalities, such as Chiari 1 malformation, also being seen in a wide variety of cerebellar diseases, including degenerative etiologies, toxic-metabolic and ischemic injury. Case report: A 45-year-old woman, who presented with dizziness started 6 years ago, related to rapid head movement, with progressive worsening, leading to difficulty in walking and a tendency to fall to the right, associated with mild to moderate intensity occipitonuchal headache, with improvement at rest and horizontal decubitus. Neurological examination showed downbeat nystagmus, intent tremor in the finger-to-nose-test, worse on the right arm, Romberg test presentes with anteroposterior and lateral instability, in addition to Tanden with bilateral lateropulsion, without other relevant changes. The magnetic resonance imaging showed platybasia and vertebrobasilar invagination, Chiari type 1 malformation with tonsillar herniation, without signs of cervical hydrosyringomyelia, signs of atrophy of both cerebellar hemispheres, a nonspecific nodule in the subcortical region of the left precuncle. Conclusion: The case described above is relevant to demonstrate that even in cases of acute changes in adulthood, screening for anatomical malformations in the posterior fossa should be considered.
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Currie, Debra C., and Harold E. Bedell. "The Influence of Simulated Nystagmus Parameters on Visual Acuity." In Noninvasive Assessment of the Visual System. Washington, D.C.: Optica Publishing Group, 1990. http://dx.doi.org/10.1364/navs.1990.thb1.

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In persons with congenital idiopathic nystagmus, a target's image undergoes substantial retinal motion. Although visual acuity is often reduced in these individuals, the magnitude of the reduction is typically fairly modest. In an attempt to determine what accounts for the visual loss, and therefore what the potential for improvement is, researchers have looked at the relationship between acuity and various parameters of the nystagmus eye movements.
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Reinhardt, Sophia, Joshua Schmidt, Michael Leuschel, Christiane Schüle, and Jörg Schipper. "VertiGo - Pilot project for nystagmus detection via webcam." In Abstract- und Posterband – 91. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Welche Qualität macht den Unterschied. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1711239.

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Haghighi, Hamed, Farzaneh Abdollahi, Sahar Babaei, and Shahriar Gharibzadeh. "Compensation of downbeat nystagmus with a modular controller." In 2014 21th Iranian Conference on Biomedical Engineering (ICBME). IEEE, 2014. http://dx.doi.org/10.1109/icbme.2014.7043935.

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Suaste-Gomez, Ernesto, Lorenzo S. Leija, and Humberto J. Sossa-Azuela. "Measurement with fiber optics of nystagmus eye movements." In Photonics West '95, edited by James A. Harrington, David M. Harris, and Abraham Katzir. SPIE, 1995. http://dx.doi.org/10.1117/12.208429.

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Reports on the topic "Nystagmus"

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Clancy, Noah, Jane Vennik, Jay Self, and Helena Lee. Measuring visual and quality of life outcomes in nystagmus; a scoping review protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2024. http://dx.doi.org/10.37766/inplasy2024.5.0002.

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