Relevant bibliographies by topics / Normal pressure hydrocephalus

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Normal pressure hydrocephalus'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Normal pressure hydrocephalus"

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De Lucca, Maria Eduarda Turczyn, Leonardo Cesar Ferreira Antunes, Carlos Arteaga Rodriguez, and Otto Jesus Hernández Fustes. "Normal pressure hydrocephalus." JMPHC | Journal of Management & Primary Health Care | ISSN 2179-6750 13 (December 2, 2021): e017. http://dx.doi.org/10.14295/jmphc.v13.1130.

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Normal pressure hydrocephalus – NPH is one of the potentially reversible causes of dementia. In this work, we provide the clinical tools for the management of NPH to the Primary Health Care – PHC physicians. The PubMed and Cochrane databases were reviewed, and 40 articles were chosen, which include randomized clinical trials, metanalysis and systematic reviews. The symptoms of the classic triad are nonspecific and found in the elderly with other diseases. Gait apraxia is the earliest symptom, followed by cognitive impairment and urinary incontinence. The concomitance of the three symptoms occurs in about 60% of the cases in the advanced stages. Apraxia of gait is presented with short steps, without sensorial, vestibulocerebellar or muscle strength changes, which allows it to be distinguished from other pathological marches. Cognitive impairment is subcortical – psychomotor slowliness, inattention, loss of working memory and executive function and preservation of episodic memory – unlike cortical dementias, such as Alzheimer's disease. Urinary incontinence must be distinguished from overflow by prostatism and stress incontinence in women. The PHC physician, when faced with the triad, should request a CT scan or brain MRI and, in the presence of one or two symptoms, rule out other diagnoses. Patients with NPH seek help from the physician in the PHC who must know how to identify and differentiate their symptoms. In the case of suspicion, the study of neuroimaging is mandatory and referral to neurosurgery or neurology is recommended.
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Liang, Kyle, and Puja Chebrolu. "Normal-pressure hydrocephalus." Journal of the American Academy of Physician Assistants 35, no. 2 (February 2022): 35–38. http://dx.doi.org/10.1097/01.jaa.0000800300.26131.ef.

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Micchia, Katia, Caterina Formica, Simona De Salvo, Nunzio Muscarà, Placido Bramanti, Fabrizia Caminiti, Silvia Marino, and Francesco Corallo. "Normal pressure hydrocephalus." Medicine 101, no. 9 (March 4, 2022): e28922. http://dx.doi.org/10.1097/md.0000000000028922.

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Conn, Harold O. "Normal pressure hydrocephalus." Clinical Medicine 7, no. 4 (August 1, 2007): 416.3–416. http://dx.doi.org/10.7861/clinmedicine.7-4-416b.

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Byrd, Christine. "Normal Pressure Hydrocephalus." Nurse Practitioner 31, no. 7 (July 2006): 28???35. http://dx.doi.org/10.1097/00006205-200607000-00006.

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&NA;. "Normal Pressure Hydrocephalus." Nurse Practitioner 31, no. 7 (July 2006): 36???37. http://dx.doi.org/10.1097/00006205-200607000-00007.

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Tsakanikas, Diamanto, and Norman Relkin. "Normal Pressure Hydrocephalus." Seminars in Neurology 27, no. 1 (February 2007): 058–65. http://dx.doi.org/10.1055/s-2006-956756.

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Billek-Sawhney, Barbara, and Nicholas A. Jackson. "Normal Pressure Hydrocephalus." Journal of Acute Care Physical Therapy 3, no. 2 (2012): 182–88. http://dx.doi.org/10.1097/01592394-201203020-00002.

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Billek-Sawhney, Barbara, and Nicholas A. Jackson. "Normal Pressure Hydrocephalus." Journal of Acute Care Physical Therapy 3, no. 2 (2013): 182–88. http://dx.doi.org/10.1097/01592394-201303020-00002.

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Graff-Radford, Neill R., and David T. Jones. "Normal Pressure Hydrocephalus." CONTINUUM: Lifelong Learning in Neurology 25, no. 1 (February 2019): 165–86. http://dx.doi.org/10.1212/con.0000000000000689.

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Dissertations / Theses on the topic "Normal pressure hydrocephalus"

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Owler, Brian Kenneth. "Pathophysiology of normal pressure hydrocephalus." Thesis, The University of Sydney, 2004. http://hdl.handle.net/2123/685.

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Normal pressure hydrocephalus (NPH), a CSF circulation disorder, is important as a reversible cause of gait and cognitive disturbance in an aging population. The inconsistent response to CSF shunting is usually attributed to difficulties in differential diagnosis or co-morbidity. Improving outcome depends on an increased understanding of the pathophysiology of NPH. Specifically, this thesis examines the contribution of, and inter-relationship between, the brain parenchyma and CSF circulation in the pathophysiology of NPH. Of the four core studies of the thesis, the first quantifies the characteristics of the CSF circulation and parenchyma in NPH using CSF infusion studies to measure the resistance to CSF absorption and brain compliance. The second study assesses cerebral blood flow (CBF) was using O15-labelled positron emission tomography (PET) with MR co-registration. By performing CSF infusion studies in the PET scanner, CBF at baseline CSF pressure and at a higher equilibrium pressure is measured. Regional changes and autoregulatory capacity are assessed. The final study examines the microstructural integrity of the parenchyma using MR diffusion tensor imaging. These studies confirm the importance of the inter-relationship of the brain parenchyma and CSF circulation. NPH symptomatology and its relationship to the observed regional CBF reductions in the basal ganglia and thalamus are discussed. Regional CBF reductions with increased CSF pressure and the implications for autoregulatory capacity in NPH are considered. The reduction in CBF when CSF was increased was most striking in the periventricular regions. In addition, periventricular structures demonstrated increased diffusivity and decreased anisotropy. The relationship between these changes and mechanisms such as transependymal CSF passage are reviewed. The findings of this thesis support a role of both the CSF circulation and the brain parenchyma in the pathophysiology of NPH. The results have implications for the approach to the management of patients with NPH.
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Owler, Brian Kenneth. "Pathophysiology of normal pressure hydrocephalus." University of Sydney. Surgery, 2004. http://hdl.handle.net/2123/685.

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Normal pressure hydrocephalus (NPH), a CSF circulation disorder, is important as a reversible cause of gait and cognitive disturbance in an aging population. The inconsistent response to CSF shunting is usually attributed to difficulties in differential diagnosis or co-morbidity. Improving outcome depends on an increased understanding of the pathophysiology of NPH. Specifically, this thesis examines the contribution of, and inter-relationship between, the brain parenchyma and CSF circulation in the pathophysiology of NPH. Of the four core studies of the thesis, the first quantifies the characteristics of the CSF circulation and parenchyma in NPH using CSF infusion studies to measure the resistance to CSF absorption and brain compliance. The second study assesses cerebral blood flow (CBF) was using O15-labelled positron emission tomography (PET) with MR co-registration. By performing CSF infusion studies in the PET scanner, CBF at baseline CSF pressure and at a higher equilibrium pressure is measured. Regional changes and autoregulatory capacity are assessed. The final study examines the microstructural integrity of the parenchyma using MR diffusion tensor imaging. These studies confirm the importance of the inter-relationship of the brain parenchyma and CSF circulation. NPH symptomatology and its relationship to the observed regional CBF reductions in the basal ganglia and thalamus are discussed. Regional CBF reductions with increased CSF pressure and the implications for autoregulatory capacity in NPH are considered. The reduction in CBF when CSF was increased was most striking in the periventricular regions. In addition, periventricular structures demonstrated increased diffusivity and decreased anisotropy. The relationship between these changes and mechanisms such as transependymal CSF passage are reviewed. The findings of this thesis support a role of both the CSF circulation and the brain parenchyma in the pathophysiology of NPH. The results have implications for the approach to the management of patients with NPH.
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Peterson, Katie Ann. "Cognition and apathy in normal pressure hydrocephalus." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/274898.

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Normal pressure hydrocephalus (NPH) is characterised by a build-up of cerebrospinal fluid (CSF) in the brain despite apparently normal CSF pressure at lumbar puncture. In addition to movement and urinary symptoms, patients commonly display cognitive decline and apathy. NPH is recognised as an important cause of cognitive decline as it is thought be reversible with surgical CSF diversion (e.g. shunt surgery). However, this remains controversial and the neuropsychology of NPH is relatively poorly understood. Further, despite being the most commonly reported neuropsychiatric symptom in NPH, the significance of the symptom of apathy has not yet been elucidated. This thesis aims to expand on the neuropsychological and neuropsychiatric research in NPH, with the main objectives being to investigate neuropsychological outcome, and the significance of the symptom of apathy in NPH. In order to investigate neuropsychological outcome following shunt surgery in NPH, a systematic review and meta-analysis was conducted (Chapter 2). The findings from studies which used a battery of neuropsychological tests to assess cognitive outcome in NPH were combined. Meta-analyses were conducted on pre-operative and difference scores for the most commonly used neuropsychological tests. These were seven tests which spanned global cognitive function, learning and memory, executive function and psychomotor speed. Results for all tests were significant in the direction of improvement. However, the significance of the results for two measures of executive function were deemed not to be robust. This is discussed in line with previous research which suggests that executive function may be less likely to improve following shunt surgery than other neuropsychological domains. Next, the thesis focuses on the symptom of apathy. Chapter 3 investigated whether apathy in NPH relates to cognitive outcome and to a measure of ventricular enlargement. A reduction in apathetic symptoms following treatment was associated with better performance in a measure of global cognitive function. Further, larger ventricles (which may indicate greater disease severity/ brain damage) was associated with more severe apathy. A structural MRI study was then conducted to expand on these findings and to define brain structural correlates of apathy in NPH (Chapter 4). Results from this study suggested a potential role of the caudate nuclei in apathetic symptoms in NPH. Finally, consideration is given to the assessment of apathy in NPH. Since apathy is rarely investigated in patients with NPH it is unclear which assessment method is most appropriate for this patient group. Chapter 5 presents findings from a feasibility study of a novel reward learning task to determine whether it might be useful as an objective measure of motivation and apathy in NPH.
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Toma, Ahmed Kassem Abbass. "Conservative versus treatment of idiopathic normal pressure hydrocephalus." Thesis, University of London, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558383.

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Objectives: Idiopathic normal pressure hydrocephalus (INPH) is a condition that affects the elderly population with clinical presentation of gait and balance impairment, cognitive impairment and urinary incontinence. Brain imaging shows -. ventriculomegaly in the absence of raised intracranial pressure. Shunt insertion is the standard method of treatment. However; there is a lack of good quality evidence of its effectiveness. As a result, many physicians and even few surgeons are sceptic about the existence of this condition as a separate entity. The aim of this thesis is to provide evidence supporting or refuting the role of surgical management in INPH, through a prospective randomized controlled double blinded study. Methods: Fourteen probable INPH patients who showed clinical improvement following extended lumbar drainage were randomized to have ventriculoperitoneal shunt insertion with an open or closed adjustable valve. Closed shunts were opened after three months. Primary endpoint was improvement in walking speed. Secondary endpoints were improvement in cognitive function, urinary symptoms and incidence of complications. Results: There was a statistically significant advantage of the open shunt in improving walking speed compared with the closed shunt at the study endpoint (43% vs. 0% p=0.02). In addition, the prospectively collected data has shown that both groups improved in terms of mobility one year following shunt insertion (47% and 46.7% respectively). There was a trend suggesting that delay in opening the shunt has delayed improvement beyond the 3 months hypothesized in study design. However; this delay was not statistically significant (p=0.08). 4 us.
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Tarnaris, A. "Biomarkers in patients with idiopathic normal pressure hydrocephalus." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1360351/.

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Idiopathic normal pressure hydrocephalus (iNPH) is a condition affecting a small percentage of the elderly population; however it is the only known treatable cause of dementia. Surgical cerebrospinal fluid (CSF) diversion is the only known treatment for the condition today. However, such a procedure is not to be offered lightly and any expected benefit has to balance the associated surgical risks. The prognosis of a favourable surgical outcome has been problematic since the conception of the syndrome. None of current prognostic tests reaches 100% sensitivity or specificity and it is felt that there might be a need for a combination of tests, rather than a single one to maximize the chances of selecting the right patients to offer a surgical CSF diversion procedure. Biomarkers are biological substances that may act as surrogate markers of response to a treatment or to characterise a disease’s progression over time. The aim of this study was to identify CSF markers of favourable surgical outcome in patients with iNPH undergoing the insertion of a ventriculoperitoneal shunt (VPS). We first describe the effects of external lumbar drainage (ELD) on the CSF biochemistry of these patients. Correlations are made with imaging data obtained from volumetric analysis and neuropsychological tests in order to obtain a complete profile of these patients. The rostrocaudal gradients of the CSF markers examined are reported showcasing the need to understand that commonly reported values from lumbar CSF do not necessarily reflect pathological changes occurring at cerebral level. Finally, we report on the individual as well as combined prognostic value of 7 CSF markers on surgical outcomes at 6 months. The pathophysiological significance of these markers is discussed individually. It is concluded that the combined power of total tau and Aβ 1-42 may be useful in predicting favourable surgical outcomes at 6 months; further studies applying the findings in a larger cohort and correlating findings with longer outcomes are warranted to enhance the clinical application. The biochemical profile of patients with iNPH appears unique and different than patients with Alzheimer’s dementia or control subjects.
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Lenfeldt, Niklas. "The search for reversibility of idiopathic normal pressure hydrocephalus : aspects on intracranial pressure measurements and advanced MRI techniques in combination with CSF volume alteration /." Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1422.

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Solomon, Daniel L. "Evaluation of normal pressure hydrocephalus with diffusion tensor imaging." Thesis, Boston University, 2013. https://hdl.handle.net/2144/12226.

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Thesis (M.A.)--Boston University
Purpose: Normal Pressure Hydrocephalus (NPH) is a clinical diagnosis with no formal definition. Textbooks describe NPH as a clinical triad of gait abnormality, dementia, and urinary incontinence. Few patients present with all three symptoms, forcing the clinician to rely on a “preponderance of evidence” approach, which involves weighing triad symptoms with radiological findings, Cerebrospinal Fluid (CSF) opening pressure, response to Tap Test, external lumbar CSF drainage, lumbar infusion, and finally shunting. Radiological findings in NPH are limited to enlarged ventricles out of proportion to sulcal atrophy, callosal angles greater than 40 degrees, and ventricles with Evan’s ratios greater than 0.3. When radiologists evaluate suspected NPH patients they are limited to excluding disease, as opposed to searching for any particular finding. In this study we used Diffusion Tensor Imaging (DTI) to determine if differences can be identified on a group basis between NPH and normal groups to see if DTI (including tractography) can be a useful tool for understanding disease morphology and laying the groundwork for future clinical use of DTI for identification of NPH. Materials and Methods: A retrospective study of patients who underwent brain MRI imaging with a Philips 3T magnet. NPH patients were classified as “definite” or “probable NPH” by their referring physicians. Normal subjects were patients found to have no anatomical brain abnormality. DTI and tractography data were acquired using Philips Fibertrak software, and post-processing was done using Tract Based Spacial Statistics (TBSS). Conclusion: NPH patients were found to have higher Fractional Anisotropy (FA) values in the upper corticospinal tract, lower FA values in the Corpus Callosum and mixed results in the internal capsule, to p ≤ 0.05 levels, consistent with previous reports. NPH tractography was also characterized with a distinct “heart-shaped” sign. Possible uses for tractography for patients under suspicion of NPH will be discussed.
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Gelling, Leslie. "The illness experiences of people with idiopathic normal pressure hydrocephalus." Thesis, Anglia Ruskin University, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.442143.

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Lundin, Fredrik. "Idiopathic Normal Pressure Hydrocephalus : Aspects on Pathophysiology, Clinical Characteristics and Evaluation Methods." Doctoral thesis, Linköpings universitet, Neurologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-84243.

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Introduction. Idiopathic normal pressure hydrocephalus (iNPH) is a condition with enlargement of the cerebral ventricular system and an intracranial pressure (ICP) within normal limits. Cerebrospinal fluid circulation is disturbed but the mechanisms behind the symptoms: gait and balance difficulties, cognitive dysfunction and micturition problems, are as yet mostly unexplained. Aim. In Studies I and II the aim was to investigate cerebral metabolism in the frontal deep white matter (FDWM) and the thalamus in iNPH using Magnetic Resonance Spectroscopy (MRS) before and after shunt surgery and to compare this with healthy individuals (HI). In Study III the aim was, by use of actigraphy, to measure motor function, energy expenditure and resting/sleeping time in iNPH patients before and after shunt surgery, in comparison with HI. In Study IV the aim was to study postural function using computerised dynamic posturography (CDP) before and after shunt surgery as well as in comparison with HI. Patients and Methods. In all studies the patients had a neurological examination and baseline bedside assessments of motor, balance and cognitive function were performed. Motor function was assessed using a motor score (MOS) consisting of the following items: 10 metre walk time in seconds and number of steps and TUG time in seconds and number of steps. MOS was considered significant if there was an increase of 5% or more. The HI were also tested for motor, balance and cognitive function. In Study I the patients (n=16) and the HI (n=15) were examined with MRS (absolute quantification) with voxels placed in the thalamus and in FDWM and compared with one another. In Studies III and IV the preoperative results of actigraphy and CDP respectively in patients (Study III n=33; study IV n=35) were compared with the HI: Study III, n=17; Study IV, n=16. The HI performed these examinations twice and the average was calculated. In Study II, 14 patients, and in Studies III and IV, 20 patients underwent shunt surgery and new MRS/actigraphy/CDP examinations were performed three months postoperatively and compared with the preoperative results. Results. In the patients decreased total N-acetyl compounds (tNA) and N-acetyl aspartate (NAA) were found in the thalamus compared to the HI. No metabolic differences were seen in the FDWM between the groups. Postoperatively there were no metabolic changes in the thalamus but an increased total Choline (tCho) and a borderline significant decrease in myo-inositol (mIns).During the day the patients took fewer steps and had also lower total energy expenditure (TEE) than the HI. There was no difference concerning resting/sleeping time between patients and the HI. Postoperatively there were no differences of either number of steps, TEE or time spent resting or sleeping compared with the preoperative state. Postural function was worse in the patients compared to the HI, this difference being more pronounced in tests measuring vestibular function, where loss of balance (LOB) was frequent. There was only a slight improvement in balance after shunt surgery. A positive response to the shunt operation was seen in 86% in Study II, 85% in Study III and 90% in Study IV. Conclusions. Our results suggest that the thalamus may be involved in the pathogenesis of iNPH. In contrast to others, we did not find any metabolic abnormalities in the FDWM, nor detect an increment of tNA or NAA postoperatively in the thalamus. The postoperative increase in tCho and borderline decrease in mIns in the FDWM might reflect a state of metabolic recovery since high tCho, a major component of the cell membrane, may be a sign of increased membrane turnover, and a decrease in mIns may indicate diminished gliosis. The low gait capacity seen in the iNPH patients was not surprising but well that time spent resting/sleeping did not differ from the HI. Another unexpected finding was the unchanged ambulatory activity after shunt surgery despite improvement in a point test to determine capacity to walk a short distance. We believe this could be due to strong habits that are difficult to break and/or shortage of rehabilitation after surgery. There was a profound postural dysfunction in the patients with many falls, especially in test situations intended to measure vestibular function. This implies that there is a central vestibular disturbance. The discrete improvement in postural function postoperatively was lower than previously reported.
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Balamurali, Gopal. "Predicting the response to ventriculoperitoneal shunt in patients with normal pressure hydrocephalus." Thesis, University of Central Lancashire, 2011. http://clok.uclan.ac.uk/2391/.

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The diagnosis and management of normal pressure hydrocephalus (NPH) remains controversial. The aim of this prospective study was to assess the usefulness of clinical and radiological criteria together with supplemental neuropsychological and gait tests, cerebrospinal fluid (CSF) hydrodynamic studies and external lumbar drainage (ELD) in identifying those who may respond to a shunt and to compare the outcomes with the non-shunted patients at one year. Method: Forty patients with a clinical diagnosis of NPH were prospectively studied according to a fixed management protocol. Resistance to CSF (Rcsf) was measured using a lumbar infusion study and an ELD was used to determine improvement in neuropsychological and gait tests following CSF drainage. Based on specific criteria those who showed improvement were shunted. Clinical and radiological outcomes were assessed at one year in all patients. Results: Twenty three (57.5%) patients were shunted. Improvement was observed in 74% of shunted patients, while 17% did not improve and 9% deteriorated following surgery. Age, etiology, presentation, duration of symptoms and presence of co-morbid factors were unrelated to outcome. Improvement was found in 63% of shunted patients with RCSF of 12 mmHg/ml/min or higher. The sensitivity was 64% with a positive predictive value of 68%. Both Rcsf testing and ELD enhanced the positive predictive outcome of shunt operation. Using the non-shunted patients as controls, the mean difference between the two groups over time differed significantly in all the neuropsychological tests and some gait test. Conclusions: No single test was able to predict overall success with shunting but the results showed that a high percentage of improvement could be achieved by using continuous ELD and a rigorous protocol. Greater improvements were observed in cognitive and gait function than in sphincter control. An Rcsf of 12mmHg/ml/min or more was related to better outcomes. Consideration was given to the significant limitations in the study design and outcome measures.
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Books on the topic "Normal pressure hydrocephalus"

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Normal-pressure hydrocephalus: Pathology, pathophysiology, diagnostics, therapeutics and clinical course. Ratingen: PVV Science Publications, 2003.

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Young, Harold, and Anthony Marmarou. The estimated incidence of normal pressure hydrocephalus in assisted living and extended care facilities for the senior Virginia residents: Report of the Division of Neurological Surgery at the Medical College of Virginia of Virginia Commonwealth University to the Governor and the General Assembly of Virginia. Richmond, Va: Commonwealth of Virginia, 2005.

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Meier, Ullrich, Michael J. Fritsch, and Uwe Kehler. Normal Pressure Hydrocephalus: Pathophysiology, Diagnosis, Treatment. Thieme Medical Publishers, Incorporated, 2013.

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Mednick, Adam S. Normal Pressure Hydrocephalus: From Diagnosis to Treatment. Addicus Books, 2013.

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Mednick, Adam S. Normal Pressure Hydrocephalus: From Diagnosis to Treatment. Addicus Books, 2013.

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Adam S. Mednick MD PhD. Normal Pressure Hydrocephalus: From Diagnosis to Treatment. Addicus Books, 2013.

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Beller, Jerry, and Beller Health. 2019 Normal Pressure Hydrocephalus: This Dementia Strikes All Ages! Independently Published, 2019.

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Briggs, John, Jerry Beller, Beller Health, and Brain Research. Normal Pressure Hydrocephalus: Guide for Doctors, Nurses, Patients, Families, and Caregivers. Independently Published, 2020.

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Whittle, Ian. Raised intracranial pressure, cerebral oedema, and hydrocephalus. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0604.

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The brain is protected by the cranial skeleton. Within the intracranial compartment are also cerebrospinal fluid, CSF, and the blood contained within the brain vessels. These intracranial components are in dynamic equilibrium due to the pulsations of the heart and the respiratory regulated return of venous blood from the brain. Normally the mean arterial blood pressure, systemic venous pressure, and brain volume are regulated to maintain physiological values for intracranial pressure, ICP. There are a range of very common disorders such as stroke, and much less common, such as idiopathic intracranial hypertension, that are associated with major disturbances of intracranial pressure dynamics. In some of these the contribution to pathophysiology is relatively minor whereas in others it may be substantial and be a major contributory factor to morbidity or even death.Intracranial pressure can be disordered because of brain oedema, disturbances in CSF flow, mass lesions, and vascular engorgement of the brain. Each of these may have variable causes and there may be interactions between mechanisms. In this chapter the normal regulation of intracranial pressure is outlined and some common disease states in clinical neurological practice that are characterized by either primary or secondary problems in intracranial pressure dynamics described.
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Institute, Brain, John Briggs, and Beller Health. Neglected DEMENTIAS: Normal Pressure Hydrocephalus, Huntington's Disease, Korsakoff Syndrome, Creutzfeldt-Jakob Disease, & Amyotrophic Lateral Sclerosis. Independently Published, 2020.

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Book chapters on the topic "Normal pressure hydrocephalus"

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Missori, Paolo, Antonio Daniele, and Carlo Colosimo. "Normal Pressure Hydrocephalus." In Movement Disorders in Dementias, 231–52. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6365-7_14.

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Malec, James F. "Normal Pressure Hydrocephalus." In Encyclopedia of Clinical Neuropsychology, 2478. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_9171.

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Malec, James F. "Normal Pressure Hydrocephalus." In Encyclopedia of Clinical Neuropsychology, 1–2. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_9171-1.

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Oh, Dennis S., and Peter McL. Black. "Normal Pressure Hydrocephalus." In Pathy's Principles and Practice of Geriatric Medicine, 643–54. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781119952930.ch56.

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Franceschi, Ana M., and Lev Bangiyev. "Normal Pressure Hydrocephalus." In PET/MR Imaging, 335–36. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-65106-4_136.

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Kovanlikaya, Ilhami, and Gloria C. Chiang. "Normal Pressure Hydrocephalus." In Hybrid PET/MR Neuroimaging, 459–68. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-82367-2_38.

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Rovira, Alex. "Communicating Hydrocephalus. Normal Pressure Hydrocephalus." In Clinical Neuroradiology, 1–30. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-61423-6_11-1.

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Rovira, Àlex. "Communicating Hydrocephalus: Normal Pressure Hydrocephalus." In Clinical Neuroradiology, 489–517. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-68536-6_11.

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Tamaki, N., T. Nagashima, K. Ehara, T. Shirakuni, and S. Matsumoto. "Hydrocephalic Oedema in Normal-Pressure Hydrocephalus." In Brain Edema VIII, 348–50. Vienna: Springer Vienna, 1990. http://dx.doi.org/10.1007/978-3-7091-9115-6_118.

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Ammar, Ahmed, Faisal Abbas, Wisam Al Issawi, Fatima Fakhro, Layla Batarfi, Ahmed Hendam, Mohammed Hasen, Mohammed El Shawarby, and Hosam Al Jehani. "Idiopathic Normal-Pressure Hydrocephalus Syndrome: Is It Understood? The Comprehensive Idiopathic Normal-Pressure Hydrocephalus Theory (CiNPHT)." In Hydrocephalus, 67–82. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-61304-8_5.

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Conference papers on the topic "Normal pressure hydrocephalus"

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Shrinivasan, Anup, Maïté Brandt-Pearce, Adam Barth, and John Lach. "Analysis of gait in patients with normal pressure hydrocephalus." In the First ACM Workshop. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/2064942.2064947.

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Tully, Brett, and Yiannis Ventikos. "Modelling Normal Pressure Hydrocephalus as a ‘Two-Hit’ Disease Using Multiple-Network Poroelastic Theory." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19135.

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The evolution of many cerebral diseases such as Alzheimer’s and Parkinson’s Disease, Hydrocephalus, Cerebral Oedema, Stroke, and Tumour are strongly correlated to a change in the transport properties of fluid in the brain. This research proposes a novel application of Multiple-Network Poroelastic Theory (MPET) to investigate cerebral hydrodynamics through a detailed investigation of multiscalar, spatio-temporal transport of fluid between the cerebral blood, cerebrospinal fluid (CSF) and brain parenchyma. Specifically, MPET is used to interrogate the clinical markers of Normal Pressure Hydrocephalus (NPH).
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Chen, Shanshan, Adam T. Barth, Jeffery T. Barth, Bradford C. Bennett, Maïté Brandt-Pearce, Donna K. Broshek, Jason R. Freeman, Hillary L. Samples, and John Lach. "Aiding diagnosis of normal pressure hydrocephalus with enhanced gait feature separability." In the conference. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2448096.2448099.

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Tully, B., J. Byrne, and Y. Ventikos. "Is Normal Pressure Hydrocephalus more than a mechanical disruption to CSF flow?" In 2010 32nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC 2010). IEEE, 2010. http://dx.doi.org/10.1109/iembs.2010.5627772.

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Fialho, Hugo, Alice Hung, Dane Moran, Sharif Vakili, Eric Sankey, Ignacio Jusué-Torres, Benjamin Elder, et al. "Predictors of Ventriculoperitoneal Shunt Revision in Patients with Idiopathic Normal Pressure Hydrocephalus." In XXXII Congresso Brasileiro de Neurocirurgia. Thieme Revinter Publicações Ltda, 2018. http://dx.doi.org/10.1055/s-0038-1672418.

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Neri, Lorena Aparecida Teixeira, Lorraine Evangelista Rocha, and Ana Paula Oliveira Borges. "Gait speed and self-efficacy in falls in individuals with normal pressure hydrocephalus." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.639.

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Background: Normal pressure hydrocephalus (PNH) is manifested by the triad apraxia of gait, dementia and urinary incontinence. The fear of falling and the loss of a sense of self-efficacy in body balance and mobility results in the restriction of social participation. Objective: to verify the relationship between gait speed and self-efficacy for falls in individuals with PNH. Methodology: This is an observational and cross-sectional study, carried out at the Clinical School of Physiotherapy at UNIFRAN, with CEP approval (CAAE 83164918.2.0000.5495). The convenience sample consisted of 3 individuals with PNH, with DVP, age 72.5 (± 3.6), who presented independent gait without using a walking aid device and MMSE 23.6 (± 1.2) and a history of more than 2 falls in the past year. They were assessed for the sense of self-efficacy for falls using the Falls Efficacy Scale - International (FES-I) questionnaire. The speed of normal and fast gait was calculated by the time taken to cover 10 meters. To verify the relationship between FES-I and gait speed, association and correlation tests were applied. Descriptive analysis was performed and the paired t test and the Pearson correlation test were used. Results: The FEI-S average was 31.3 ± 4.8 points. The average normal walking speed was 0.62 ± 0.24m / s and the average fast speed was 0.77 ± 0.32m / s. The participants were not able to significantly modify the gait speed (p = 0.073). There was a positive correlation between FES-I and gait speed (r = 0.68; p = 0.027). Conclusion: The low self-efficacy for falls interferes with the ability to change the gait pattern through speed. Understanding the fear of falling and walking speed can help in the mapping of cases that deserve, in addition to conventional rehabilitation, a behavioral intervention to increase the sense of self-efficacy, and thereby improve walking independence.
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Galeano, M., A. Calisto, A. Bramanti, F. Angileri, G. Campobello, S. Serrano, and B. Azzerboni. "Classification of morphological features extracted from intracranial pressure recordings in the diagnosis of Normal Pressure Hydrocephalus (NPH)." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6090758.

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Swim, Benjamin M., Julie A. Reyer, Martin J. Morris, and Julian J. Lin. "Development of an Apparatus for the Testing of Hydrocephalic Shunts." In ASME 2006 Frontiers in Biomedical Devices Conference. ASMEDC, 2006. http://dx.doi.org/10.1115/nanobio2006-18025.

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This abstract summarizes the development of a new apparatus designed to test hydrocephalic shunts. Hydrocephalus is a medical condition most commonly characterized by above-normal intracranial pressure (ICP) that occurs when a patient’s head cannot properly regulate cerebrospinal fluid (CSF) volume in the head. The condition is generally caused by a blockage to flow of CSF in the normal biological pathways. This can result from a birth defect, trauma, or disease. In a hydrocephalic patient, excess fluid builds up in the ventricles resulting in increased mechanical stress and physical deformation of the brain. Untreated, this condition can be quite severe and can lead to brain damage or death. Standard treatment involves implanting an artificial shunt to drain the ventricle and bypass the blockage. The CSF is normally routed to the abdominal cavity. Reducing fluid volume alleviates high ICP and mechanical stress on the brain. Shunting improves the survival rate from 30 to 60 percent for untreated patients to 65 to 95 percent for patients with shunt systems installed [1].
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Collante, MT, and C. Bernal. "130 Normal-pressure hydrocephalus in a nine year old filipino female with systemic lupus erythematosus." In LUPUS 2017 & ACA 2017, (12th International Congress on SLE &, 7th Asian Congress on Autoimmunity). Lupus Foundation of America, 2017. http://dx.doi.org/10.1136/lupus-2017-000215.130.

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Calisto, A., A. Bramanti, M. Galeano, F. Angileri, G. Campobello, S. Serrano, and B. Azzerboni. "A preliminary study for investigating idiopatic normal pressure hydrocephalus by means of statistical parameters classification of intracranial pressure recordings." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5335371.

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