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Books on the topic 'Non-imaging fiber optic system'

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Published: 30 May 2022
Last updated: 31 May 2022

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1

Conference, on Optical Fiber Communication (2003 Atlanta Ga ). Optical Fiber Communications Conference (OFC): Postconference digest. Washington, DC: Optical Society of America, 2003.

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2

Handbook of biomedical optics. Boca Raton: CRC Press, 2011.

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3

Conference on Optical Fiber Communication (1997 Dallas, Tex.). Conference on Optical Fiber Communications: Technical digest, February 16-21, 1997, Dallas Conference Center, Dallas, Texas. Washington, DC: Optical Society of America, 1997.

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4

1961-, Hooper Brett Andrew, ed. An introduction to biomedical optics. New York: Taylor & Francis, 2007.

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5

1947-, Wootton John, ed. Electro-optical systems peformance modeling. Boston: Artech House, 1993.

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6

N, Sivarajan Kumar, ed. Optical networks: A practical perspective. 2nd ed. San Francisco: Morgan Kaufmann Publishers, 2002.

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7

Ramaswami, Rajiv. Optical networks: A practical perspective. San Francisco: Morgan Kaufmann Publishers, 1998.

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8

(Editor), L. S. Grattan, and B. T. Meggitt (Editor), eds. Optical Fiber Sensor Technology - Volume 3: Applications and Systems (Optoelectronics, Imaging and Sensing). Springer, 1999.

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9

Yeow, John Tze-Wei. Optical microelectromechanical system development for biomedical imaging and fibre-optic switches. 2003.

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10

Keoghane, Stephen, and Mark Sullivan. The principles of endourology. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0032.

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The chapter discusses the principles of endourology outlining the principles behind the sub-specialty. Topics disused include endoscopes, cystoscopes, rigid and semi-rigid ureteroscopes, flexible ureterorenoscopes, rigid and flexible nephroscopes, disposable equipment, integrated operating theatres, narrow band imaging (NBI), theatre ergonomics, and antibiotics. Urologic endoscopes are generally of two optical designs: the rigid, rod lens system described by Hopkins, while fibre-optic imaging bundles are used in both rigid and flexible endoscopes. The rod lens system consists of a series of glass rods with polished ends with the key feature of air gaps that act as a lens. Light is carried efficiently along the rod, resulting in a clear and bright image.
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11

(Editor), Francesco Baldini, Nathan I. Croitoru (Editor), Martin Frenz (Editor), Ingemar Lundstrom (Editor), and Mitsunobu Miyagi (Editor), eds. Biomedical Sensors, Fibers and Optical Delivery Systems (Progress in Biomedical Optics Series). SPIE-International Society for Optical Engine, 1999.

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12

Optical Society of America. Optical Fiber Communications Conference (Ofc): Postconference Digest. Institute of Electrical & Electronics Enginee, 2003.

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13

United States. National Aeronautics and Space Administration., ed. Fiber optic probes for laser light scattering: Ground based evaluation for micgrogravity flight experimentation and Integrated coherent imaging fiber optic systems for laser light scattering and other applications. Stony Brook, NY: State University of New York, Dept. of Electrical Engineering, 1994.

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14

L, Antos Ronald, Krisiloff Allen J, Society of Photo-optical Instrumentation Engineers. Western New York Chapter., Society of Photo-optical Instrumentation Engineers., and Canadian-American Eastern Regional Conference on Applications of Optical Engineering (1st : 1990 : Rochester, N.Y.), eds. Can-Am Eastern '90: Proceedings, 4-5 October 1990, Rochester, New York. Bellingham, Wash., USA: SPIE, 1991.

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15

Francesco, Baldini, Society of Photo-optical Instrumentation Engineers., European Optical Society, European Laser Association, and Scandinavian Society for Laser Therapy., eds. Proceedings of biomedical sensors, fibers, and optical delivery systems: 8-10 September 1998, Stockholm, Sweden. Bellingham, Wash., USA: SPIE, 1999.

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16

Handbook of Biomedical Optics. Taylor & Francis Group, 2016.

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17

ICO20: Biomedical optics : 21-26 August, 2005, Changchun, China. Bellingham, Wash: SPIE, 2006.

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18

Abraham, Katzir, Society of Photo-optical Instrumentation Engineers., and Harvard University--MIT Division of Health Sciences and Technology., eds. Optical fibers in medicine II: 17-19 September 1986, Cambridge, Massachusetts. Bellingham, Wash., USA: The Society, 1987.

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19

Abraham, Katzir, American Academy of Otolaryngology--Head and Neck Surgery., California College of Medicine, Society of Photo-optical Instrumentation Engineers., and University of California Irvine, eds. Optical fibers in medicine III: 13-16 January 1988, Los Angeles, California. Bellingham, Wash., USA: SPIE--The International Society for Optical Engineering, 1988.

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20

Optical Fibers in Medicine Ii/713: 17-19 September 1986, Cambridge, Massachusetts (Proceedings of Spie--the International Society for Optical Engineering, V. 713). Society of Photo Optical, 1986.

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21

Optical Society of America. Conference on Optical Fiber Communication (Nineteen Ninety-Seven Technical Digest Series Vol. 6). Optical Society of America, 1997.

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22

J, Landry Robert, Sliney David H, Scott Robert, and Society of Photographic Instrumentation Engineers., eds. Optical and laser technology in medicine: 23-24 January 1986, Los Angeles, California. Bellingham, Wash., USA: SPIE--the International Society for Optical Engineering, 1986.

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23

1956-, Daum Werner, ed. POF: Polymer optical fibers for data communication. Berlin: Springer, 2002.

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24

Daum, Werner, Olaf Ziemann, Jürgen Krauser, and Peter E. Zamzow. POF - Polymer Optical Fibers for Data Communication. Springer, 2002.

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25

Chang, William S. C. 1931-, ed. RF photonic technology in optical fiber links. Cambridge: Cambridge University Press, 2002.

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26

Wootton, John, and Gary Waldman. Electro-Optical Systems Performance Modeling (Artech House Optoelectronics Library). Artech House Publishers, 1992.

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27

Coppola, Silvia, and Franco Valenza. Inhalation injury in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0107.

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Inhalation injury represents one of the most serious associated injuries complicating the care of thermally-injured patient. It can result in severe respiratory failure and acute respiratory distress syndrome (ARDS) by three mechanisms—thermal or chemical injury, and impairment of systemic oxygen supply. Thermal injury can cause erythema, ulceration, and progressive, life-threatening oedema, particularly of the upper airways. Chemical injury is due to irritants or cytotoxic compounds, and depends on the material burned, the temperature of the fire, and the amount of oxygen present in the fire environment. It is responsible for irritation, ulceration, and oedema of the mucosal surface, and the initiation of a lung inflammatory reaction when small particles reach the alveoli. Moreover, the increased vascular permeability, and the reduced surfactant production carry a significant risk in the development of pneumonia and ARDS. Bronchospasm and upper airway oedema can occur rapidly, while lower airway oedema can be asymptomatic for up to 24 hours. Lung imaging techniques may not reveal injured areas for the first 24–48 hours. Fibre optic bronchoscopy is considered to be the most direct diagnostic method for the definitive diagnosis of inhalation injury. The patient management includes airways assessment, adequate fluid resuscitation, and mechanical ventilation when required. All victims of smoke inhalation should be always evaluated for cyanide and carbon monoxide poisoning.
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28

Sivarajan, Kumar, and Rajiv Ramaswami. Optical Networks: A Practical Perspective (Second Edition) (The Morgan Kaufmann Series in Networking). 2nd ed. Morgan Kaufmann, 2001.

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29

Compston, Alastair. Multiple sclerosis and other demyelinating diseases. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0871.

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The oligodendrocyte–myelin unit subserves saltatory conduction of the nerve impulse in the healthy central nervous system. At one time, many disease processes were thought exclusively to target the structure and function of myelin. Therefore, they were designated ‘demyelinating diseases’. But recent analyses, based mainly on pathological and imaging studies, (re)emphasize that axons are also directly involved in these disorders during both the acute and chronic phases. Another ambiguity is the extent to which these are inflammatory conditions. Here, distinctions should be made between inflammation, as a generic process, and autoimmunity in which rather a specific set of aetiological and mechanistic conditions pertain. And there are differences between disorders that are driven primarily by immune processes and those in which inflammation occurs in response to pre-existing tissue damage.With these provisos, the pathological processes of demyelination and associated axonal dysfunction often account for episodic neurological symptoms and signs referable to white matter tracts of the brain, optic nerves, or spinal cord when these occur in young people. This is the clinical context in which the possibility of ‘demyelinating disease’ is usually considered by physicians and, increasingly, the informed patient. Neurologists will, with appropriate cautions, also be prepared to diagnose demyelinating disease in older patients presenting with progressive symptoms implicating these same pathways even when there is no suggestive past history. Both in its typical and atypical forms multiple sclerosis remains by far the commonest demyelinating disease. But acute disseminated encephalomyelitis, the leucodystrophies, and central pontine myelinolysis also need to be considered in particular circumstances; and multiple sclerosis itself has a differential diagnosis in which the relapsing-remitting course is mimicked by conditions not associated with direct injury to the axon–glial unit. Since our understanding of the cause, pathogenesis and features of demyelinating disease remains incomplete, classification combines aspects of the aetiology, clinical features, pathology, and laboratory components. Whether the designation ‘multiple sclerosis’ encapsulates one or more conditions is now much debated. We anticipate that a major part of future studies in demyelinating disease will be further to resolve this question of disease heterogeneity leading to a new taxonomy based on mechanisms rather than clinical empiricism. But, for now, the variable ages of onset, unpredictable clinical course, protean clinical manifestations, and non-specific laboratory investigations continue to make demyelinating disease one of the more challenging diagnostic areas in clinical neurology.
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