Academic literature on the topic 'No mutation identified'
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Journal articles on the topic "No mutation identified"
Hutton, Michael. "?Missing? tau mutation identified." Annals of Neurology 47, no. 4 (April 2000): 417–18. http://dx.doi.org/10.1002/1531-8249(200004)47:4<417::aid-ana1>3.0.co;2-b.
Full textPawlik, Timothy M., Darrell R. Borger, Yuhree Kim, David Cosgrove, Sorin Alexandrescu, Ryan Thomas Groeschl, Vikram Deshpande, et al. "Genomic profiling of intrahepatic cholangiocarcinoma: Refining prognostic determinants and identifying therapeutic targets." Journal of Clinical Oncology 32, no. 3_suppl (January 20, 2014): 210. http://dx.doi.org/10.1200/jco.2014.32.3_suppl.210.
Full textDutta, Ravi Kumar, Thomas Arnesen, Anette Heie, Martin Walz, Piero Alesina, Peter Söderkvist, and Oliver Gimm. "A somatic mutation in CLCN2 identified in a sporadic aldosterone-producing adenoma." European Journal of Endocrinology 181, no. 5 (November 2019): K37—K41. http://dx.doi.org/10.1530/eje-19-0377.
Full textLowstuter, Katrina, Carin R. Espenschied, Duveen Sturgeon, Charité Ricker, Rachid Karam, Holly LaDuca, Julie O. Culver, et al. "Unexpected CDH1 Mutations Identified on Multigene Panels Pose Clinical Management Challenges." JCO Precision Oncology, no. 1 (November 2017): 1–12. http://dx.doi.org/10.1200/po.16.00021.
Full textZhu, Xiaoqiong, Xingnong Ye, Chen DAN, and Jian Huang. "Uncommon Hpgd Mutation Identified in Familial Erythrocytosis." Blood 138, Supplement 1 (November 5, 2021): 4627. http://dx.doi.org/10.1182/blood-2021-145881.
Full textBradbury, Jane. "Canine epilepsy gene mutation identified." Lancet Neurology 4, no. 3 (March 2005): 143. http://dx.doi.org/10.1016/s1474-4422(05)01004-5.
Full textBRADBURY, J. "Canine epilepsy gene mutation identified." Lancet Neurology 4, no. 3 (March 2005): 143. http://dx.doi.org/10.1016/s1474-4422(05)70010-7.
Full textShi, Zhongxun, Bing Li, Tiejun Qin, Zefeng Xu, Lijuan Pan, Shiqiang Qu, Gang Huang, and Zhijian Xiao. "Clonal Architecture Analysis of TET2 Identified Distinct Origins in Myelodysplastic Syndromes." Blood 136, Supplement 1 (November 5, 2020): 18. http://dx.doi.org/10.1182/blood-2020-139329.
Full textClaes, Kathleen, Eva Machackova, Michel De Vos, Bruce Poppe, Anne De Paepe, and Ludwine Messiaen. "Mutation Analysis of the BRCA1 and BRCA2 Genes in the Belgian Patient Population and Identification of a Belgian Founder Mutation BRCA1 IVS5+3A>G." Disease Markers 15, no. 1-3 (1999): 69–73. http://dx.doi.org/10.1155/1999/241046.
Full textPercy, Melanie J., F. G. C. Jones, T. R. J. Lappin, and M. F. McMullin. "Mutations in the VHL Gene Are the Major Identified Cause of Inherited Erythrocytosis." Blood 106, no. 11 (November 16, 2005): 569. http://dx.doi.org/10.1182/blood.v106.11.569.569.
Full textDissertations / Theses on the topic "No mutation identified"
Kozusko, Kristina. "Molecular mechanisms of Perilipin-1 action : characterisation of a novel PLIN1 mutation identified in patients with familial partial lipodystrophy." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709005.
Full textChen, Tao [Verfasser]. "Identification and functional characterization of a de novo point mutation identified in a patient with non-syndromal microcephaly and intellectual disability / Tao Chen." Berlin : Freie Universität Berlin, 2016. http://d-nb.info/1108270913/34.
Full textPERON, ANGELA. "TUBEROUS SCLEROSIS COMPLEX: IDENTIFICATION OF THE GENETIC CAUSE IN PATIENTS WITH NO MUTATION DETECTED, AND ANALYSIS OF MOSAICISM." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/885842.
Full textLi, Jia. "Identifier les variations conduisant au cancer dans le génome non codant et du transcriptome." Thesis, Université Paris-Saclay (ComUE), 2015. http://www.theses.fr/2015SACLS161/document.
Full textFunctional annotation of somatic mutations have been a consistent hotspot of cancer genomics studies. In the past, researchers preferentially focused on mutations in the coding fraction of the genome, for which ample bioinformatics tools were developed to distinguish cancer-driver mutations from neutral ones. In recent years, as an increasing number of variants were being identified as disease-associated in the non-coding genome, interpreting non-coding cancer mutations has become an urgent task. The completion of large scale projects such as ENCODE, has made functional interpretation of cancer variants achievable, and several programs were produced based on this functional information. However, there still exists some limitations as to these prediction tools, such as low prediction accuracy, lack of cancer mutation information and significant ascertainment bias. In chapter 2 of this thesis, in order to functionally interpret non-coding mutations in cancer, we developed two independent random forest models, referred to as SNP and SOM. Given a combination of features at a given genome positions, the SNP model predicts the expected fraction of rare SNPs (a measure of negative selection), and the SOM model predicts the expected mutation density at this position. We applied our two models to score these non-coding disease-associated clinvariant and HGMD variants and a set of random control SNPs. Results showed that disease-associated variants were scored higher than control SNPs with the SNP model and lower than control SNPs with the SOM model, supporting our hypothesis that purifying selection as measured by fraction of rare SNPs and mutation density is informative for the evaluation of the functional impact of cancer mutations in the non-coding genome. In the past, researchers have preferentially considered protein-coding genes as critical to the initiation and progression of cancers. However, recent evidences have shown that ncRNAs, in particular lncRNAs, are actively implicated in various cancer processes. A chapter of this thesis is devoted to this class of non-coding transcripts. Similar to protein coding genes, there might be a large number of lncRNAs with cancer-driving functions. The development of bioinformatics tools to prioritize them has become a new focus of research for computational oncologists.The last part of this thesis is devoted to the implementation of methods for discovering potential cancer-driving non-coding elements in lncRNA and protein-coding genes. We applied three scoring tools, CADD, funSeq2, GWAVA, together with our SNP and SOM scoring systems to prioritize cancer-associated elements using a permutation-based algorithm. For each locus, we compute the average score of all observed variants using one of the models, and we randomly take the same number of variants and compute their average score 1 million times to form a null distribution and obtain a P value for this locus. To validate our hypothesis and permutation model, we tested this system on 61 cancer-related lncRNA and 452 cancer genes using somatic mutation data from liver cancer, lung cancer, CLL and melanoma. We observed that both cancer lncRNAs and protein-coding genes had significantly lower average P values than total lncRNAs and protein-coding genes in all cases. Applying the permutation test to lncRNAs with five different scoring systems enabled us to prioritize hundreds to thousands of cancer-related lncRNA candidates. These candidates can be used for future experimental validation
Duff, Jennifer. "Characterisation of androgen receptor mutations identified from prostate cancer patients." Thesis, University of Aberdeen, 2005. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU200888.
Full textDempsey, Nunez Laura. "Spectrum of mutations in MMAA identified by high resolution melting analysis." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=110535.
Full textLe produit génique du MMAA est nécessaire pour le métabolisme de la cobalamine intracellulaire (Cbl). Des mutations dans ce gène conduisent à la classe de maladies cblA, caractérisé par l'acidurie méthylmalonique isolée. Nous avons été concernés que les méthodes de diagnostic de cellules somatiques peuvent manquer les patients atteints phénotypes cellulaires moins sévère. Une teste de fusion à haute résolution a été développé pour balayer rapidement les exons codantes et les régions introniques adjacentes du gène MMAA pour des variantes. Nous avons testé l'ADN à partir de 96 personnes de référence qui ne sont pas touchés, 72 patients atteints de cblA confirmé par complémentation et 181 patients présentant une élévation de l'acide méthylmalonique isolée, qui ne pouvaient pas être diagnostiquée à l'aide d'analyse de complémentation. Les variantes suspectes ont été confirmées à l'aide de séquençage Sanger. Dans la cohorte cblA, l'analyse de fusion à haute résolution a correctement identifié toutes les mutations connues antérieurement, ainsi que 22 autres variantes, dont 10 n'avaient pas été signalés précédemment. Nouveaux variantes inclus une duplication (C.551dupG, p.C187LfsX3), une délétion (c.387delC, p.Y129YfsX13), une mutation du site d'épissage (c.440-2A> G, site d'épissage), 4 mutations faux-sens (c. 748G> A, p.E520K; c.820G> A, p.G274S; c.627G> T, p.R209S; c.826A> G, p.K276E), et 3 mutations non-sens (c.960G> A, p.W320X; c.1075C> T, p.E359X; c.1084C> T, p.Q362X). Toutes les variantes faux-sens nouveaux, énumérés ci-dessus, affectent des résidus hautement conservés et sont prévus pour être endommageant. L'analyse de MMAA dans les 181 échantillons non diagnostiqués a révélé un seul changement faux-sens hétérozygote (c.821G> A, p.G274D).
Illson, Margaret. "Spectrum of mutations in MMAB identified by high resolution melting analysis." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=110564.
Full textDes variantes pathogéniques dans le gène MMAB (OMIM 607958) sont responsables de la classe cblB d'acidurie méthylmalonique (AMM) respondant à la cobalamine (OMIM 251110). MMAB encode cobalamine adénosyltranférase, une enzyme mitochondriale responsable de la formation de l'adénosylcobalamine (AdoCbl). AdoCbl fonctionne par la suite en tant que cofacteur pour méthylmalonyl-CoA mutase (MCM) durant l'isomérisation de L-méthylmalonyl-CoA vers succinyl-CoA. Des analyses sur des cellules somatiques ont été utilisées pour évaluer des échantillons de patients pour des troubles reliés à la cobalamine. En raison de niveaux de base élevés d'incorporation de propionate, certains patients présentant des phénotypes biochimiques bénins d'AMM ne peuvent être diagnostiqués par analyse de complémentation. Une analyse de fusion à haute résolution (AFHR) a été développée pour balayer rapidement les exons codants et les régions introniques avoisinnantes pour des variantes dans le gène MMAB.Trois cohortes d'échantillons ont été balayées par AFHR : une population de référence non-affectée, 42 échantillons assignés au groupe cblB par analyse de complémentation et 181 patients avec une AMM isolée sans diagnostique. L'AFHR a correctement identifié toutes les mutations précédemment rapportées dans la cohorte cblB ainsi que sept variantes additionelles, incluant une nouvelle variante non-sens (c.12C>A, p.C4X). Le balayage de la cohorte avec de l'AMM isolée a identifié six échantillons contenant des variantes dans MMAB. Deux échantillons, WG3948 et WG4034, étaient des porteurs de variantes hétérozygotes composés. Ils partageaient la mutation c.572G>A (p.R191Q). WG3948, le cas index pour cette étude, était porteur du c.398C>T (p.S133F) pour la deuxième mutation et WG4034, le deuxième patient, contenait une nouvel variante c.394C>T (p.C132R). Les échantillons provenant de quatre autres patients atteints contenait une seule variante. Le c.572G>A (p.R191Q) a été trouvé dans WG3546 et WG4090. WG3759 contenait une substitution c.52C>T (p.S174L), et WG4029 contenait une nouvelle substitution c.185C>T (p.T62M).L'identification de deux patients avec des variantes hétérozygotes composées dans le gène MMAB suggère l'existence d'un phénotype rare mais distinct de cblB. Cette sous-classe est charactérisée par des niveaux d'incorporation de propionate et de synthèse d'AdoCbl dans les valeurs normales, empêchant le diagnostique par analyse des cellules somatiques.
Mori, Minako. "Pathogenic mutations identified by a multimodality approach in 117 Japanese Fanconi anemia patients." Kyoto University, 2019. http://hdl.handle.net/2433/243302.
Full textSchröder, Michael, Rainer Winnenburg, and Conrad Plake. "Improved mutation tagging with gene identifiers applied to membrane protein stability prediction." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-177379.
Full textSchröder, Michael, Rainer Winnenburg, and Conrad Plake. "Improved mutation tagging with gene identifiers applied to membrane protein stability prediction." BioMed Central, 2009. https://tud.qucosa.de/id/qucosa%3A28888.
Full textBooks on the topic "No mutation identified"
Miller-Hodges, Eve, and Christopher Mitchell. The patient with Wilms tumour. Edited by Giuseppe Remuzzi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0173_update_001.
Full textMammoser, Aaron. Infiltrative Astrocytomas. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0126.
Full textSyrris, Petros, and Alexandros Protonotarios. Arrhythmogenic right ventricular cardiomyopathy: genetics. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0359.
Full textWalsh, Richard A. Parkinson’s Disease or Essential Tremor? Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0016.
Full textQuain, Angela, and Anne M. Comi. Sturge-Weber Syndrome and Related Cerebrovascular Malformation Syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0112.
Full textMammoser, Aaron. Primary and Secondary Glioblastoma. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0127.
Full textSchwartz, Peter J., and Lia Crotti. Monogenic and oligogenic cardiovascular diseases: genetics of arrhythmias—catecholaminergic polymorphic ventricular tachycardia. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0152.
Full textHall, Andrew, and Shamima Rahman. Mitochondrial diseases and the kidney. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0340.
Full textElliott, Perry, and Alexandros Protonotarios. Arrhythmogenic right ventricular cardiomyopathy: management of symptoms and prevention of sudden cardiac death. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0361.
Full textSadleir, Lynette G., Jozef Gecz, and Ingrid E. Scheffer. Epilepsies That Occur Predominantly in Girls. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0041.
Full textBook chapters on the topic "No mutation identified"
Nakagawa, Hitoshi. "History of mutation breeding and molecular research using induced mutations in Japan." In Mutation breeding, genetic diversity and crop adaptation to climate change, 24–39. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0003.
Full textLundqvist, Udda. "Scandinavian mutation research during the past 90 years - a historical review." In Mutation breeding, genetic diversity and crop adaptation to climate change, 10–23. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0002.
Full textKumar, Arun, Binay Kumar Agarwal, Rajesh Kumar, Sanjay J. Jambhulkar, Varsha Rani, and Zille Ali Haider. "Induction of variability for yield components in Indian mustard (Brassica juncea L. Czern & Coss) under acidic soil regime of Jharkhand." In Mutation breeding, genetic diversity and crop adaptation to climate change, 258–68. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0026.
Full textJankowicz-Cieslak, Joanna, Florian Goessnitzer, Sneha Datta, Altus Viljoen, Ivan Ingelbrecht, and Bradley J. Till. "Induced mutations for generating bananas resistant to Fusarium wilt tropical race 4." In Mutation breeding, genetic diversity and crop adaptation to climate change, 366–78. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0038.
Full textSevanthi, Amitha Mithra V., Prashant Kale, Chandra Prakash, M. K. Ramkumar, Neera Yadav, V. Sureshkumar, Yugandhar Poli, et al. "National repository of EMS induced mutants of an upland rice cultivar Nagina 22: progress update on characterization and utilization." In Mutation breeding, genetic diversity and crop adaptation to climate change, 290–302. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0030.
Full textDas, Priyanka, Rajeev N. Bahuguna, Rohit Joshi, Sneh Lata Singla-Pareek, and Ashwani Pareek. "In search of mutants for gene discovery and functional genomics for multiple stress tolerance in rice." In Mutation breeding, genetic diversity and crop adaptation to climate change, 444–50. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0045.
Full textMorales, Yonis, and Rolando Grajeda. "Virulence genes of new population of coffee rust (Hemileia vastatrix) affecting coffee variety 'Lempira', in Honduras; resistant and susceptible varieties." In Mutation breeding, genetic diversity and crop adaptation to climate change, 338–43. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0035.
Full textda Luz, Viviane Kopp, Vívian Ebeling Viana, Gabriela Magalhães da Fonseca, Camila Pegoraro, Luciano Carlos da Maia, and Antonio Costa de Oliveira. "Identification of rice mutants tolerant to cold stress at the germination stage by TILLING." In Mutation breeding, genetic diversity and crop adaptation to climate change, 111–19. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0011.
Full textLiu, Lu-xiang, Yong-dun Xie, Hui-jun Guo, Lin-shu Zhao, Hong-chun Xiong, Jia-yu Gu, and Shi-rong Zhao. "New mutation techniques for crop improvement in China." In Mutation breeding, genetic diversity and crop adaptation to climate change, 47–52. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0005.
Full textJegadeesan, Souframanien, and Kandali Sreenivasulu Reddy. "Radiation-induced mutations in genetic enhancement and development of new crop varieties in black gram (Vigna mungo (L.) Hepper)." In Mutation breeding, genetic diversity and crop adaptation to climate change, 303–11. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789249095.0031.
Full textConference papers on the topic "No mutation identified"
Birch, David G., Gabriel H. Travis, Kirsten G. Locke, and Donald C. Hood. "Rod Ergs in Mice and Humans with Putative Null Mutations in the RDS Gene." In Vision Science and its Applications. Washington, D.C.: Optica Publishing Group, 1997. http://dx.doi.org/10.1364/vsia.1997.ma.4.
Full textMukhopadhyay, Asima, Nicola Curtin, and Richard Edmondson. "Evaluation of different methods to assess homologous recombination status and sensitivity to PARP inhibitors in ovarian cancer." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685289.
Full textKant, Nimita, and Perumal Senthiappan Jayaraj. "Evaluation of Telomerase Reverse Transcriptase Expression in Squamous Cell Carcinoma of the Skin." In Annual Conference of Indian Society of Medical and Paediatric Oncology (ISMPO). Thieme Medical and Scientific Publishers Pvt. Ltd., 2021. http://dx.doi.org/10.1055/s-0041-1735375.
Full textYoussoufiän, H., A. Patel, D. Phillips, H. H. Kazazian, and S. E. Antonarakis. "RECURRENT MUTATIONS AND AN UNUSUAL DELETION IN HEMOPHILIA A." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644014.
Full textBrooks, Joseph Bruno Bidin, Fábio César Prosdócimi, Lara Fenley Granzotto, and Matheus Garcez Jorge Mariani. "Camptocormia and genetic Parkinson’s disease caused by the mutation of the LRRK2 gene. Case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.183.
Full textHiguchi, M., L. Kochhan, R. Schwaab, H. H. Brackmann, H. Egli, and K. Olek. "DETECTION OF MUTATIONS IN HEMOPHILIA A." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644012.
Full textLee, Dae-Won, Yongjun Cha, Sae-Won Han, Si-Hyun Lee, Hwang-Phill Kim, Jaemyun Lyu, Hyojun Han, et al. "Abstract A72: Mutation signature associated with colorectal cancer prognosis identified by targeted next-generation sequencing." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics; November 5-9, 2015; Boston, MA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1535-7163.targ-15-a72.
Full textBurg, ED, and JX Yuan. "A Tetramerization Domain Mutation in KCNA5 Identified in Pulmonary Arterial Hypertension Patients Causes Abnormal Trafficking Patterns." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6015.
Full textFeijão, Maria Clara Tomaz, Fernanda Pimentel Arraes Maia, Mateus Coelho Gondim de Oliveira Lima, Vitória Moreira Soares, and Luiz Gonzaga Porto Pinheiro. "CONCERNING A FAMILY WITH BRCA2 MUTATION." In XXIV Congresso Brasileiro de Mastologia. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s1019.
Full textBenslimane, Fatiha M., Hebah Al Khatib, Dana Albatesh, Ola Al-Jamal, Sonia Boughattas, Asmaa A. Althani, and Hadi M. Yassine. "Nanopore Sequencing SARS-CoV-2 Genome in Qatar." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0289.
Full textReports on the topic "No mutation identified"
Ori, Naomi, and Mark Estelle. Specific mediators of auxin activity during tomato leaf and fruit development. United States Department of Agriculture, January 2012. http://dx.doi.org/10.32747/2012.7597921.bard.
Full textWhitham, Steven A., Amit Gal-On, and Tzahi Arazi. Functional analysis of virus and host components that mediate potyvirus-induced diseases. United States Department of Agriculture, March 2008. http://dx.doi.org/10.32747/2008.7591732.bard.
Full textOlszewski, Neil, and David Weiss. Role of Serine/Threonine O-GlcNAc Modifications in Signaling Networks. United States Department of Agriculture, September 2010. http://dx.doi.org/10.32747/2010.7696544.bard.
Full textAvni, Adi, and Gitta L. Coaker. Proteomic investigation of a tomato receptor like protein recognizing fungal pathogens. United States Department of Agriculture, January 2015. http://dx.doi.org/10.32747/2015.7600030.bard.
Full textMcClure, Michael A., Yitzhak Spiegel, David M. Bird, R. Salomon, and R. H. C. Curtis. Functional Analysis of Root-Knot Nematode Surface Coat Proteins to Develop Rational Targets for Plantibodies. United States Department of Agriculture, October 2001. http://dx.doi.org/10.32747/2001.7575284.bard.
Full textChen, Junping, Zach Adam, and Arie Admon. The Role of FtsH11 Protease in Chloroplast Biogenesis and Maintenance at Elevated Temperatures in Model and Crop Plants. United States Department of Agriculture, May 2013. http://dx.doi.org/10.32747/2013.7699845.bard.
Full textOhad, Itzhak, and Himadri Pakrasi. Role of Cytochrome B559 in Photoinhibition. United States Department of Agriculture, December 1995. http://dx.doi.org/10.32747/1995.7613031.bard.
Full textWeller, Joel I., Harris A. Lewin, and Micha Ron. Determination of Allele Frequencies for Quantitative Trait Loci in Commercial Animal Populations. United States Department of Agriculture, February 2005. http://dx.doi.org/10.32747/2005.7586473.bard.
Full textGelb, Jr., Jack, Yoram Weisman, Brian Ladman, and Rosie Meir. Identification of Avian Infectious Brochitis Virus Variant Serotypes and Subtypes by PCR Product Cycle Sequencing for the Rational Selection of Effective Vaccines. United States Department of Agriculture, December 2003. http://dx.doi.org/10.32747/2003.7586470.bard.
Full textWeller, Joel I., Derek M. Bickhart, Micha Ron, Eyal Seroussi, George Liu, and George R. Wiggans. Determination of actual polymorphisms responsible for economic trait variation in dairy cattle. United States Department of Agriculture, January 2015. http://dx.doi.org/10.32747/2015.7600017.bard.
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