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Journal articles on the topic 'NLA'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 March 2023

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1

Lei, Hao, and Yunhuo Cui. "Effects of Academic Emotions on Achievement Among Mainland Chinese Students: A Meta-Analysis." Social Behavior and Personality: an international journal 44, no. 9 (October 9, 2016): 1541–53. http://dx.doi.org/10.2224/sbp.2016.44.9.1541.

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We undertook a meta-analysis to explore the relationship between academic emotions (comprising positive high-arousal [PHA], positive low-arousal [PLA], negative high-arousal [NHA], and negative low-arousal [NLA] emotions) and academic performance among mainland Chinese students, and analyzed the moderating effects of regional location, age, achievement domain match, and gender on these associations. Included in the research were 35 primary studies with 17,548 participants. Results of the meta-analysis showed overwhelming support for the positive correlations between PHA, PLA, and academic achievement (rPHA = .312, rPLA = .376), and negative correlations between NHA, NLA, and academic achievement (rNHA = -.179, rNLA = -.371). Furthermore, moderation analyses suggested that these effects were influenced by regional location, age, achievement domain match, and gender. Practical and theoretical implications of the findings are discussed.

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2

Liao, Qiong, Tian-jiao Tang, Ting Zhou, Hai-xing Song, Ying-peng Hua, and Zhen-hua Zhang. "Integrated Transcriptional and Proteomic Profiling Reveals Potential Amino Acid Transporters Targeted by Nitrogen Limitation Adaptation." International Journal of Molecular Sciences 21, no. 6 (March 21, 2020): 2171. http://dx.doi.org/10.3390/ijms21062171.

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Nitrogen (N) is essential for plant growth and crop productivity. Organic N is a major form of remobilized N in plants’ response to N limitation. It is necessary to understand the regulatory role of N limitation adaption (NLA) in organic N remobilization for this adaptive response. Transcriptional and proteomic analyses were integrated to investigate differential responses of wild-type (WT) and nla mutant plants to N limitation and to identify the core organic N transporters targeted by NLA. Under N limitation, the nla mutant presented an early senescence with faster chlorophyll loss and less anthocyanin accumulation than the WT, and more N was transported out of the aging leaves in the form of amino acids. High-throughput transcriptomic and proteomic analyses revealed that N limitation repressed genes involved in photosynthesis and protein synthesis, and promoted proteolysis; these changes were higher in the nla mutant than in the WT. Both transcriptional and proteomic profiling demonstrated that LHT1, responsible for amino acid remobilization, were only significantly upregulated in the nla mutant under N limitation. These findings indicate that NLA might target LHT1 and regulate organic N remobilization, thereby improving our understanding of the regulatory role of NLA on N remobilization under N limitation.

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3

Gandhi, Kamal M., Taro Ohkawa, Matthew D. Welch, and Loy E. Volkman. "Nuclear localization of actin requires AC102 in Autographa californica multiple nucleopolyhedrovirus-infected cells." Journal of General Virology 93, no. 8 (August 1, 2012): 1795–803. http://dx.doi.org/10.1099/vir.0.041848-0.

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Autographa californica multiple nucleopolyhedrovirus requires nuclear actin for progeny virus production and thereby encodes viral products that ensure actin’s translocation to and retention within the nucleus. Current evidence suggests that the ie0–ie1 gene complex along with five nuclear localization of actin (NLA) genes are sufficient for NLA in transient transfection experiments. Here we report that, during infection, only one of the five NLA genes, Ac102, was essential for NLA, and that AC102 had at least one other activity critical for budded virus (BV) production. Viral deletion mutants in the other four NLA genes were viable, with only two having replication phenotypes different from that of the wild type. Infection with AcΔpe38 revealed a delay in both BV production and NLA. Infection with AcΔ152 revealed a delay in BV production, but no corresponding delay in NLA. Infection with either AcΔpe38 or AcΔ152 resulted in slightly reduced BV titres. Deletion of Ac004 or he65 had no impact on actin translocation kinetics, timing of BV production or BV titres. These results implicate AC102 as a key player in baculovirus manipulation of actin.

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4

Nandini, S., CS Prashanth, Sanju K. Somiah, and SRK Reddy. "An Evaluation of Nasolabial Angle and the Relative Inclinations of the Nose and Upper Lip." Journal of Contemporary Dental Practice 12, no. 3 (2011): 152–57. http://dx.doi.org/10.5005/jp-journals-10024-1026.

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ABSTRACT Esthetics is one of the major motivating factors for patients seeking orthodontic treatment. Hard tissue and soft tissue drape both determine the facial esthetics. The structures in this region are so variable that the nasolabial angle (NLA) has been drawn differently by various investigators. Variations can lead to erroneous conclusions in orthodontic diagnosis. Aims and objectives The study was done to evaluate a reliable method of constructing the nasolabial angle (NLA) and to correlate the soft tissue profile parameters with one another. Materials and methods Lateral cephalogram of 50 randomly selected adult patients were taken. The tracings were made and 10 copies of each tracing were randomly distributed to 10 different orthodontists to draw the NLA. Results Pearson's correlation coefficient (r) showed both N/ FH and L/FH angles to have significant p values when compared with NLA. The regression analysis showed that the nasolabial angle can be calculated for any given value of N/FH or L/FH by the formula: NLA = 80.33° + 1.02° (N/FH) and NLA = 14.2° + 1.04° (L/FH). The mean value of N/FH was 17.42° ± 8.40° and L/FH was 80.68° + 6.45° for this sample. Inter examiner reliability calculated by repeated measures of ANOVA and Dahlerg's formula showed high degree of reliability and reproducibility of the method. Clinical significance NLA can be predicted for any given value of N/FH and L/FH. NLA = 80.33° + 1.02° (N/FH) and NLA = 14.2° + 1.04° (L/FH). If an individual has either N/FH or L/FH in the normal range but not the NLA then one could calculate the correct NLA using this formula. Thereby the NLA can be brought within the normal range by altering the other nasolabial parameters by correct treatment planning. Since the nasolabial angle plays a vital role in profile esthetics of a person, the clinician should place greater emphasis in evaluating this area and plan treatment mechanics to place this angle within the accepted normal variation. How to cite this article Nandini S, Prashanth CS, Somiah SK, Reddy SRK. An Evaluation of Nasolabial Angle and the Relative Inclinations of the Nose and Upper Lip. J Contemp Dent Pract 2011;12(3):152-157.

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5

Sobchenko, S. A., O. S. Schetchikova, and N. V. Yakovleva. "Acute non-atopic late-onset asthma induced by respiratory infection." PULMONOLOGIYA, no. 1 (February 28, 2005): 53–57. http://dx.doi.org/10.18093/0869-0189-2005-0-1-53-57.

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The aim of the study was to investigate features of respiratory infection inducing acute non-atopic late-onset asthma (NLA). Virologic and microbiologic examinations of brash biopsy samples of rhinopharyngeal and bronchial mucosa and bronchial lavage fluid were performed in 116 NLA patients admitted to a hospital in autumn and winter. The leading cause of acute NLA was found to be respiratory viral infections. We noted that different clinical NLA types had different sensibility to various viruses: adenoviruses mainly caused exacerbations of aspirin-induced asthma, respiratory syncytial and influenza A viruses were prevalently determined in non-atopic asthma. Patients with posttuberculotic lesions of the lungs mostly had viral and bacterial associations. Such mixed infection resulted in more severe and prolonged exacerbations of NLA.

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6

Ivanova, Krasimira Borislavova. "NLA-Bit: A Basic Structure for Storing Big Data with Complexity O(1)." Big Data and Cognitive Computing 5, no. 1 (February 24, 2021): 8. http://dx.doi.org/10.3390/bdcc5010008.

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This paper introduces a novel approach for storing Resource Description Framework (RDF) data based on the possibilities of Natural Language Addressing (NLA) and on a special NLA basic structure for storing Big Data, called “NLA-bit”, which is aimed to support middle-size or large distributed RDF triple or quadruple stores with time complexity O(1). The main idea of NLA is to use letter codes as coordinates (addresses) for data storing. This avoids indexing and provides high-speed direct access to the data with time complexity O(1). NLA-bit is a structured set of all RDF instances with the same “Subject”. An example based on a document system, where every document is stored as NLA-bit, which contains all data connected to it by metadata links, is discussed. The NLA-bits open up a wide field for research and practical implementations in the field of large databases with dynamic semi-structured data (Big Data). Important advantages of the approach are as follow: (1) The reduction of the amount of occupied memory due to the complete absence of additional indexes, absolute addresses, pointers, and additional files; (2) reduction of processing time due to the complete lack of demand—the data are stored/extracted to/from a direct address.

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7

Clarke, Ben, Mari G. Strand Cary, Lina Shanley, and Marah Sutherland. "Exploring the Promise of a Number Line Assessment to Help Identify Students At-Risk in Mathematics." Assessment for Effective Intervention 45, no. 2 (August 10, 2018): 151–60. http://dx.doi.org/10.1177/1534508418791738.

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This manuscript presents the results from a study to investigate the technical characteristics of two versions of a number line assessment (NLA 0–20 and NLA 0–100). The sample consisted of 60 kindergarten and 46 first grade students. Both number line versions had sufficient alternate form and test–retest reliability. The NLA 0–20 had low and the NLA 0–100 had low to moderate correlations with math achievement. Results indicated that the NLA 0–100 explained a small, but unique portion of the variance in first grade mathematics performance when controlling for performance on the Assessing Student Proficiency in Early Number Sense (ASPENS) a set of early numeracy screening measures. We discuss study results related to the utility of adding number line assessment tasks to mathematics screening batteries and propose additional areas of research.

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8

Setyadi, Resad, Yusmadi Kristian, Afrijal Afrijal, Farhan Deyantoro, and Noto Indra Prakoso. "Maturity Level of ITSM Analysis Using ITIL V 3 Framework in NLAA Purbalingga." SISFORMA 8, no. 1 (May 10, 2021): 7. http://dx.doi.org/10.24167/sisforma.v8i1.3039.

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The development of communication and information technology impacts the activities and ways of thinking carried out by organizations and governments. The Purbalingga National Land Agency (NLA) is an institution that uses information technology to issue proof of land rights in the agrarian/land and spatial planning sector. This study analyzes IT service management maturity level (ITSM) at NLA Purbalingga using the Information Technology Infrastructure Library (ITIL) V.3 in the operation of domain services. Descriptive analysis method of questionnaires distributed to the community in NLA Purbalingga. The IT maturity level determination at NLA Purbalingga can be done based on the service operation domain factor. The IT maturity level used has five levels for this study. The results of the analysis show that the ITSM maturity level is at level four. Level four means that IT service management at NLA Purbalingga is appropriately handled by paying attention to the factors that are fulfilled according to procedures and taking process actions against systems that are not running effectively. This study aims to provide NLA Purbalingga to improve website maintenance by carrying out comprehensive system integration to enhance work effectiveness and quality.

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9

Barnard, M. L., B. Robertson, B. P. Watts, and J. F. Turrens. "Role of nitric oxide and superoxide anion in spontaneous lung chemiluminescence." American Journal of Physiology-Lung Cellular and Molecular Physiology 272, no. 2 (February 1, 1997): L262—L267. http://dx.doi.org/10.1152/ajplung.1997.272.2.l262.

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Inhibition of nitric oxide (.NO) synthase by nitro-L-arginine (NLA) decreased baseline chemiluminescence in a dose-dependent fashion up to 78% at 300 microM NLA. This inhibition was prevented by pretreatment with 1 mM arginine. Similarly, addition of superoxide dismutase (SOD; 200 U/ml) to the perfusion buffer inhibited spontaneous light emission by 57%. Addition of NLA after SOD or vice versa did not inhibit light emission any further, suggesting that both .NO and O2.- were precursors of the same oxidant. Production of additional extracellular O2.- by neutrophils activated with phorbol 12-myristate 13-acetate increased light emission by >200%, but this increase was insensitive to NLA. Increasing the intracellular steady-state O2.- concentration by perfusion of control lungs with the Cu and Zn-containing SOD inhibitor diethyldithiocarbamate (1 mM) stimulated light emission up to fourfold, but this spontaneous chemiluminescence was also insensitive to NLA. In experiments using cultured endothelial cells supplemented with extracellular bovine serum albumin (BSA), 5 microM of the Ca2+ ionophore A-23187 (a stimulant of .NO synthase) stimulated chemiluminescence by 40%. This increase was again SOD and NLA sensitive. Addition of NLA after SOD or vice versa did not change light emission. These results suggest that the background chemiluminescence of isolated-perfused intact lungs may result from the constant release of small amounts of O2.- and .NO by endothelial cells into the capillary lumen, which in turn react with BSA in the perfusion buffer.

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10

Jing, Fan, Weiqi Liu, Lingzhi Kong, and Chen He. "Improving the Performance of Continuous-Variable Measurement-Device-Independent Quantum Key Distribution via a Noiseless Linear Amplifier." Entropy 23, no. 12 (December 16, 2021): 1691. http://dx.doi.org/10.3390/e23121691.

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In the continuous variable measurement-device-independent quantum key distribution (CV-MDI-QKD) protocol, both Alice and Bob send quantum states to an untrusted third party, Charlie, for detection through the quantum channel. In this paper, we mainly study the performance of the CV-MDI-QKD system using the noiseless linear amplifier (NLA). The NLA is added to the output of the detector at Charlie’s side. The research results show that NLA can increase the communication distance and secret key rate of the CV-MDI-QKD protocol. Moreover, we find that the more powerful the improvement of the performance with the longer gain of NLA and the optimum gain is given under different conditions.

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11

Papadopoulou, Maria V., Michael W. Epperly, Donna S. Shields, and William D. Bloomer. "Radiosensitization and Hypoxic Cell Toxicity of NLA-1 and NLA-2, Two New Bioreductive Compounds." Japanese Journal of Cancer Research 83, no. 4 (April 1992): 410–14. http://dx.doi.org/10.1111/j.1349-7006.1992.tb00123.x.

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12

Alam, Mohammad Khursheed, Ahmed Ali Alfawzan, Fatema Akhter, Haytham Jamil Alswairki, and Prabhat Kumar Chaudhari. "Evaluation of Lip Morphology and Nasolabial Angle in Non-Syndromic Cleft Lip and/Palate and Non-Cleft Individuals." Applied Sciences 12, no. 1 (December 30, 2021): 357. http://dx.doi.org/10.3390/app12010357.

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Objective: To investigate the variation between the non-syndromic cleft lip and/or palate (NSCLP) and non-cleft (NC) subjects in relation to the lip morphology (LM) and nasolabial angle (NLA). Materials and Methods: Lateral cephalogram (Late. Ceph.) of 123 individuals (92 NSCLP [29 = bilateral cleft lip and palate (BCLP), 41 = unilateral cleft lip and palate (UCLP), 9 = unilateral cleft lip and alveolus (UCLA), 13 = unilateral cleft lip (UCL)], and 31 NC) who did not undergo any orthodontic treatment were investigated. By WebCeph, an artificial intelligence- (A.I.) driven software, 2 (two) parameters of LM, namely upper lip to E line (LM-1) and lower lip to E line (LM-2), and NLA analysis was carried out for each individual. Multiple tests were carried out for statistical analysis. Results: The mean ± SD observed for LM-1, LM-2, and NLA for NC individuals were 1.56 ± 2.98, 0.49 ± 3.51, and 97.20 ± 16.10, respectively. On the other hand, the mean ± SD of LM-1, LM-2, and NLA for NSCLP individuals were 4.55 ± 4.23, 1.68 ± 2.82, and 82.02 ± 14.66, respectively. No significant variation was observed with respect to gender and side. NSCLP (different types) and NC individuals showed significant disparities in LM-1 and NLA. Conclusion: It can be concluded that parameters of lip morphology such as LM-1, LM-2, and NLA vary among NSCLP and NC individuals.

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13

Stewart, Julian M., Indu Taneja, Neeraj Raghunath, Debbie Clarke, and Marvin S. Medow. "Intradermal angiotensin II administration attenuates the local cutaneous vasodilator heating response." American Journal of Physiology-Heart and Circulatory Physiology 295, no. 1 (July 2008): H327—H334. http://dx.doi.org/10.1152/ajpheart.00126.2008.

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The vasodilation response to local cutaneous heating is nitric oxide (NO) dependent and blunted in postural tachycardia but reversed by angiotensin II (ANG II) type 1 receptor (AT1R) blockade. We tested the hypothesis that a localized infusion of ANG II attenuates vasodilation to local heating in healthy volunteers. We heated the skin of a calf to 42°C and measured local blood flow to assess the percentage of maximum cutaneous vascular conductance (%CVCmax) in eight healthy volunteers aged 19.5–25.5 years. Initially, two experiments were performed; in one, Ringer solution was perfused in three catheters, the response to heating was measured, 2 μg/l losartan, 10 mM nitro-l-arginine (NLA), or NLA + losartan was added to perfusate, and the heat response was remeasured; in another, 10 μM ANG II was given, the heat response was measured, losartan, NLA, or NLA + losartan was added to ANG II, and the heat response was reassessed. The heat response decreased with ANG II, particularly the plateau phase (47 ± 5 vs. 84 ± 3 %CVCmax). Losartan increased baseline conductance in both experiments (from 8 ± 1 to 20 ± 2 and 12 ± 1 to 24 ± 3). Losartan increased the ANG II response (83 ± 4 vs. 91 ± 6 in Ringer). NLA decreased both angiotensin and Ringer responses (31 ± 4 vs. 43 ± 3). NLA + losartan blunted the Ringer response (48 ± 2), but the ANG II response (74 ± 5) increased. In a second set of experiments, we used dose responses to ANG II (0.1 nM to 10 μM) with and without NLA + losartan to confirm graded responses. Sodium ascorbate (10 mM) restored the ANG II-blunted heating plateau. NO synthase and AT1R inhibition cause an NO-independent angiotensin-mediated vasodilation with local heating. ANG II mediates the AT1R blunting of local heating, which is not exclusively NO dependent, and is improved by antioxidant supplementation.

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14

Quinzi, Vincenzo, Licia Coceani Paskay, Nicola D’Andrea, Arianna Albani, Annalisa Monaco, and Sabina Saccomanno. "Evaluation of the Nasolabial Angle in Orthodontic Diagnosis: A Systematic Review." Applied Sciences 11, no. 6 (March 12, 2021): 2531. http://dx.doi.org/10.3390/app11062531.

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Background: This study is a systematic literature review aiming at identifying the variation of the average nasolabial angle (NLA) in various orthodontic situations. The NLA is one of the key factors to be studied in an orthodontic diagnosis for the aesthetics of the nose and facial profile. Methods: Out of 3118 articles resulting from four search engines (PubMed, Cochrane Library, Turning Research Into Practice (TRIP) and SciELO), the final study allowed the analysis and comparison of only 26 studies. These included studies have considered the NLA in the following cases: teeth extraction, class II malocclusion, class III malocclusion, rapid palatal expansion (RPE), orthognathic surgery, and non-surgical rhinoplasty with a hyaluronic acid filler. Results: The results indicate that teeth extraction and the use of hyaluronic acid fillers significantly affect the NLA. Conclusions: This systematic review shows that a statistically significant change in NLA values occurs in: extractive treatments of all four of the first or second premolars in class I patients; in class II patients with upper maxillary protrusion; in patients with maxillary biprotrusion, except for cases of severe crowding; and in patients undergoing non-surgical rhinoplasty with a hyaluronic acid filler. Trial registration number: PROSPERO CRD42020185166

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15

"News from NLA." Journal of Clinical Lipidology 16, no. 4 (July 2022): 360. http://dx.doi.org/10.1016/j.jacl.2022.07.002.

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"News from NLA." Journal of Clinical Lipidology 14, no. 1 (January 2020): 3. http://dx.doi.org/10.1016/j.jacl.2020.02.003.

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"News from NLA." Journal of Clinical Lipidology 16, no. 6 (November 2022): 763–64. http://dx.doi.org/10.1016/j.jacl.2022.11.005.

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"News from NLA." Journal of Clinical Lipidology 16, no. 5 (September 2022): 546. http://dx.doi.org/10.1016/j.jacl.2022.09.009.

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"News from NLA." Journal of Clinical Lipidology 17, no. 1 (January 2023): 3. http://dx.doi.org/10.1016/j.jacl.2023.01.003.

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"News from the NLA." Journal of Clinical Lipidology 15, no. 1 (January 2021): 2. http://dx.doi.org/10.1016/j.jacl.2021.01.004.

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"News from the NLA." Journal of Clinical Lipidology 15, no. 5 (September 2021): 625–26. http://dx.doi.org/10.1016/j.jacl.2021.11.006.

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"News from the NLA." Journal of Clinical Lipidology 15, no. 2 (March 2021): 244. http://dx.doi.org/10.1016/j.jacl.2021.02.004.

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"News from the NLA." Journal of Clinical Lipidology 15, no. 4 (July 2021): 528–29. http://dx.doi.org/10.1016/j.jacl.2021.08.006.

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"News from the NLA." Journal of Clinical Lipidology 2, no. 6 (December 2008): 404. http://dx.doi.org/10.1016/j.jacl.2008.10.006.

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"News from the NLA." Journal of Clinical Lipidology 3, no. 2 (April 2009): 63–64. http://dx.doi.org/10.1016/j.jacl.2009.02.011.

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"News from the NLA." Journal of Clinical Lipidology 3, no. 4 (August 2009): 242–43. http://dx.doi.org/10.1016/j.jacl.2009.07.006.

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"News from the NLA." Journal of Clinical Lipidology 3, no. 5 (October 2009): 301–2. http://dx.doi.org/10.1016/j.jacl.2009.09.002.

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"News from the NLA." Journal of Clinical Lipidology 3, no. 6 (December 2009): 358–59. http://dx.doi.org/10.1016/j.jacl.2009.10.003.

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"News from the NLA." Journal of Clinical Lipidology 4, no. 1 (January 2010): 2–4. http://dx.doi.org/10.1016/j.jacl.2009.12.002.

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"News from the NLA." Journal of Clinical Lipidology 4, no. 2 (March 2010): 79–81. http://dx.doi.org/10.1016/j.jacl.2010.02.006.

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"News from the NLA." Journal of Clinical Lipidology 4, no. 3 (May 2010): 142–43. http://dx.doi.org/10.1016/j.jacl.2010.03.006.

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"News from the NLA." Journal of Clinical Lipidology 4, no. 4 (July 2010): 238–39. http://dx.doi.org/10.1016/j.jacl.2010.07.003.

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"News from the NLA." Journal of Clinical Lipidology 4, no. 5 (September 2010): 323. http://dx.doi.org/10.1016/j.jacl.2010.08.010.

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"News from the NLA." Journal of Clinical Lipidology 4, no. 6 (November 2010): 448. http://dx.doi.org/10.1016/j.jacl.2010.10.001.

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"News from the NLA." Journal of Clinical Lipidology 5, no. 1 (January 2011): 2. http://dx.doi.org/10.1016/j.jacl.2010.11.009.

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"News from the NLA." Journal of Clinical Lipidology 5, no. 2 (March 2011): 64–65. http://dx.doi.org/10.1016/j.jacl.2011.01.003.

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"News from the NLA." Journal of Clinical Lipidology 5, no. 3 (May 2011): 122–23. http://dx.doi.org/10.1016/j.jacl.2011.02.005.

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"News from the NLA." Journal of Clinical Lipidology 5, no. 4 (July 2011): 249–50. http://dx.doi.org/10.1016/j.jacl.2011.06.002.

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"News from the NLA." Journal of Clinical Lipidology 5, no. 5 (September 2011): 336–37. http://dx.doi.org/10.1016/j.jacl.2011.08.002.

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"News from the NLA." Journal of Clinical Lipidology 5, no. 6 (November 2011): 422–23. http://dx.doi.org/10.1016/j.jacl.2011.10.004.

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"News from the NLA." Journal of Clinical Lipidology 6, no. 1 (January 2012): 3–4. http://dx.doi.org/10.1016/j.jacl.2011.12.006.

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"News from the NLA." Journal of Clinical Lipidology 6, no. 2 (March 2012): 96–97. http://dx.doi.org/10.1016/j.jacl.2012.02.003.

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"News from the NLA." Journal of Clinical Lipidology 6, no. 3 (May 2012): 198. http://dx.doi.org/10.1016/j.jacl.2012.04.078.

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"News from the NLA." Journal of Clinical Lipidology 6, no. 4 (July 2012): 301–2. http://dx.doi.org/10.1016/j.jacl.2012.06.008.

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"News from the NLA." Journal of Clinical Lipidology 6, no. 5 (September 2012): 395–96. http://dx.doi.org/10.1016/j.jacl.2012.08.004.

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"News from the NLA." Journal of Clinical Lipidology 6, no. 6 (November 2012): 483. http://dx.doi.org/10.1016/j.jacl.2012.10.006.

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"News from the NLA." Journal of Clinical Lipidology 7, no. 1 (January 2013): 3. http://dx.doi.org/10.1016/j.jacl.2012.12.001.

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"News from the NLA." Journal of Clinical Lipidology 7, no. 2 (March 2013): 93. http://dx.doi.org/10.1016/j.jacl.2013.01.001.

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"News from the NLA." Journal of Clinical Lipidology 7, no. 3 (May 2013): 186. http://dx.doi.org/10.1016/j.jacl.2013.03.006.

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"News from the NLA." Journal of Clinical Lipidology 7, no. 4 (July 2013): 291. http://dx.doi.org/10.1016/j.jacl.2013.06.006.

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