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Books on the topic 'Nitrite metabolism'

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Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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1

Khan, M. Nasir, Mohammad Mobin, Firoz Mohammad, and Francisco J. Corpas, eds. Nitric Oxide in Plants: Metabolism and Role in Stress Physiology. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-06710-0.

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2

Talbot, R. J. Biokinetics of 237Pu-citrate and nitrate in rats after the intravenous injection of only 2 pg plutonium. Oxfordshire, OX: Environmental and Medical Sciences Divison, Harwell Laboratory, 1989.

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3

Giménez, Maria Sofia. Advances in chemistry and biology of nitric oxide. Kerala, India: Research Signpost, 2007.

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4

C, Fang Ferric, ed. Nitric oxide and infection. New York: Kluwer Academic/Plenum Publishers, 1999.

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5

Nitrile oxides, nitrones, and nitronates in organic synthesis: Novel strategies in synthesis. New York, N.Y: VCH Publishers, 1988.

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6

Stanislaw, Lukiewicz, and Zweier Jay L, eds. Nitric oxide in transplant rejection and anti-tumor defense. Boston: Kluwer Academic Publishers, 1998.

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7

Bonavida, Benjamin. Nitric Oxide (NO) and Cancer: Prognosis, Prevention, and Therapy. New York, NY: Springer Science + Business Media, LLC, 2010.

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8

Kenneth, Weir E., Archer Stephen L, and Reeves John T, eds. Nitric oxide and radicals in the pulmonary vasculature. Armonk, NY: Futura Pub. Co. Inc., 1996.

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9

Koutsoukos, Georgios. The regulation of metabolic coronary dilation and reactive hyperemia by nitric oxide in the isolated rat heart. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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10

Rauschmaier, Rüdiger. Nutzung von Nukleotiden und Nukleobasen als Wasserstoff und Kohlestoffquelle für die Denitrifikation. München: R. Oldenbourg, 1987.

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11

Microbiology, American Society for, and Knovel (Firm), eds. Nitrification. Washington, DC: ASM Press, 2011.

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12

Henry, Yann A. Nitric oxide research from chemistry to biology: EPR spectroscopy of nitrosylated compounds. Austin, Tex., USA: Landes, 1997.

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13

Microbiology, American Society for, ed. Nitrification. Washington, DC: ASM Press, 2011.

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14

Yoshikawa, Toshikazu. Gas biology research in clinical practice. Basel: Karger, 2011.

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15

Lester, Packer, Hiramatsu Midori, and Yoshikawa Toshikazu, eds. Free radicals in brain physiology and disorders. San Diego: Academic Press, 1996.

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16

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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17

A, Titheradge Michael, ed. Nitric oxide protocols. Totowa, N.J: Humana Press, 1998.

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18

Lester, Packer, ed. Nitric oxide. San Diego: Academic Press, 1999.

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19

(Editor), Ernst van Faassen, and Anatoly Vanin (Editor), eds. Radicals for Life: The various forms of nitric oxide. Elsevier Science, 2007.

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20

Recent research development in chemistry and biology of nitric oxide. Trivandrum: Transworld Research Network, 2008.

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21

Gupta, Kapuganti Jagadis. Plant Nitric Oxide: Methods and Protocols. Springer New York, 2016.

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22

Gupta, Kapuganti Jagadis. Plant Nitric Oxide: Methods and Protocols. Springer New York, 2018.

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23

1942-, Wray John L., and Kinghorn James R, eds. Molecular and genetic aspects of nitrate assimilation. Oxford: Oxford University Press, 1989.

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24

1942-, Wray John L., and Kinghorn James R, eds. Molecular and genetic aspects of nitrate assimilation. Oxford [England]: Oxford Science Publications, 1989.

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25

Nitrate and Man: Toxic, Harmless or Beneficial? CABI, 2002.

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26

(Editor), P. Michael Conn, and Mahin D. Maines (Editor), eds. Nitric Oxide Synthase: Characterization and Functional Analysis, Volume 31 (Methods in Neurosciences). Academic Press, 1996.

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27

D, Maines Mahin, ed. Nitric oxide synthase: Characterization and functional analysis. San Diego: Academic Press, 1996.

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28

(Editor), P. Michael Conn, and Mahin D. Maines (Editor), eds. Nitric Oxide Synthase: Characterization and Functional Analysis, Volume 31 (Methods in Neurosciences). Academic Press, 1996.

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29

Maines, Mahin D. Nitric Oxide Synthase: Characterization and Functional Analysis. Elsevier Science & Technology Books, 1996.

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30

Mohammad, Firoz, M. Nasir Khan, Mohammad Mobin, and Francisco J. Corpas. Nitric Oxide in Plants: Metabolism and Role in Stress Physiology. Springer, 2014.

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31

Mohammad, Firoz, M. Nasir Khan, Mohammad Mobin, and Francisco J. Corpas. Nitric Oxide in Plants: Metabolism and Role in Stress Physiology. Springer, 2016.

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32

Mohammad, Firoz, M. Nasir Khan, Mohammad Mobin, and Francisco J. Corpas. Nitric Oxide in Plants: Metabolism and Role in Stress Physiology. Springer London, Limited, 2014.

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33

Zilliox, Lindsay, and James W. Russell. Diabetic and Prediabetic Neuropathy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0115.

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Impaired glucose regulation (IGR) constitutes a spectrum of impaired glucose and metabolic regulation that can result in neuropathy. Several different pathways of injury in the diabetic peripheral nervous system that include metabolic dysregulation induced by metabolic syndrome induce oxidative stress, failure of nitric oxide regulation, and dysfunction of certain key signaling pathways. Oxidative stress can directly injure both dorsal route ganglion neurons and axons. Modulation of the nitric oxide system may have detrimental effects on endothelial function and neuronal survival. Reactive oxidative species can alter mitochondrial function, protein and DNA structure, interfere with signaling pathways, and deplete antioxidant defenses. Advanced glycelation end (AGE) products and formation of ROS are activated by and in turn regulate key signal transduction pathways.
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34

Titheradge, Michael A. Nitric Oxide Protocols. Humana Press, 1997.

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35

Fang, Ferric C. Nitric Oxide and Infection. Springer London, Limited, 2007.

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36

Fang, Ferric C. Nitric Oxide and Infection. Springer, 2013.

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37

(Editor), John N. Abelson, Melvin I. Simon (Editor), and Helmut Sies (Editor), eds. Methods in Enzymology, Volume 301: Nitric Oxide, Part C: Biological and Antioxidant Activities (Methods in Enzymology). Academic Press, 1998.

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38

S, Moncada, and Higgs E. Annie, eds. Nitric oxide from L-arginine: a bioregulatory system. Excerpta Medica, 1990.

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39

Nitric Oxide Part E : Methods in Enzymology (Methods in Enzymology) (Methods in Enzymology). Academic Press, 2005.

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40

(Editor), Enrique Cadenas, and Lester Packer (Editor), eds. Nitric Oxide Part E : Methods in Enzymology (Methods in Enzymology) (Methods in Enzymology). Academic Press, 2005.

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41

Polak, Julia M., Mika V. J. Hukkanen, and Sean P. F. Hughes. Nitric Oxide in Bone and Joint Disease. University of Cambridge ESOL Examinations, 2021.

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42

Hukkanen, Mika V. J., 1962-, Polak Julia M, Hughes Sean, and Royal Postgraduate Medical School, eds. Nitric oxide in bone and joint disease. Cambridge, UK: Cambridge University Press, 1998.

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43

Polak, Julia M., Mika V. J. Hukkanen, and Sean P. F. Hughes. Nitric Oxide in Bone and Joint Disease. Cambridge University Press, 2011.

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44

Rubanyi, Gabor M. Pathophysiology and Clinical Applications of Nitric Oxide (Endothelial Cell Research). CRC, 1999.

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45

Lukiewicz, Stanislaw, and Jay L. Zweier. Nitric Oxide in Transplant Rejection and Anti-Tumor Defense. Springer London, Limited, 2012.

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46

Litell, John M., and Nathan I. Shapiro. Pathophysiology of septic shock. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0297.

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The pathophysiology of sepsis is the result of a dysregulated host response to infection. Interactions between conserved pathogenic signals and host recognition systems initiate a systemic reaction to local infection. Pro- and anti-inflammatory intermediates and associated coagulatory abnormalities lead to altered macrovascular, microvascular, and mitochondrial function. Uncorrected, these processes yield similar patterns of failure in multiple organ systems. Mortality increases with successive organ failures. Although commonly thought to be a manifestation of impaired renal circulation, septic acute kidney injury may be due primarily to non-haemodynamic factors. Pulmonary parenchymal dysfunction in sepsis also contributes to failures in other organ systems. Sepsis involves complex alterations in myocardial function, vascular tone, and capillary integrity, which are mediated by elevated concentrations of inflammatory cytokines, inducible nitric oxide, and reactive oxygen species, among others. Gut hypomotility and translocation of enteric flora likely contribute to a persistent inflammatory response. This perpetuates the pathophysiological pattern of sepsis, and can lead to the delayed onset of these features in patients with other types of critical illness. The neurological manifestations of sepsis include acquired delirium, which is also probably due to circulatory and inflammatory abnormalities, as well as alterations in cerebral amino acid metabolism. Critical illness-related corticosteroid insufficiency and derangements in glucose metabolism are among the endocrine abnormalities commonly seen in septic patients. Restoration of homeostasis requires early haemodynamic resuscitation and aggressive infectious source control.
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47

Wang, Rui. Signal Transduction and the Gasotransmitters: NO, CO, and H2S in Biology and Medicine. Humana Press, 2004.

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48

D, Wang Rui M., ed. Signal transduction and the gasotransmitters: NO, CO, and H2S in biology and medicine. Totowa, N.J: Humana Press, 2004.

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49

Hoffman, Ronald L., and Leyla Muedin. Optimum Macronutrition for Sexual Health (DRAFT). Edited by Madeleine M. Castellanos. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190225889.003.0007.

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The importance of nutrition in sexual health cannot be overemphasized. Given the prevalence of obesity in America and its associated morbidities such as metabolic syndrome, type 2 diabetes, and cardiovascular disease, sadly, sexual dysfunction is on the rise. Some—usually men —confronting a sexual problem may seek to consume commercial supplements or specific foods traditionally believed to improve sexual performance. However, the scientific evidence makes it clear that oysters, asparagus, or supplements are far less relevant than optimizing one's daily diet. Macronutrition impacts sexual function in many ways and as a consequence of specific dietary regimens, habits, and health practices. Nutritional patterns negatively affect testosterone, nitric oxide, cortisol, and inflammatory pathways and have other impacts. This chapter sets out what science says about macronutrition's key role in sexual function and dysfunction and advocates the best nutritional strategies for improved sexual function, vitality, and overall health.
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50

Henry, Yann A. Nitric Oxide Research from Chemistry to Biology: Epr Spectroscopy Of Nitrosylated Compounds. Springer, 2013.

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