Relevant bibliographies by topics / Nitric oxide

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Nitric oxide'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 July 2024

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Journal articles on the topic "Nitric oxide"

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Kim, Youngsun, Donghee Choi, Hosun Jang, Changsu Na, Moonhyeon Hwang, Joohyun Cho, Kyoungin Lee, Sunmin Kim, Byoungsik Pyo, and Daehwan Youn. "Effects of Acupuncture at ST41, BL60, GB38 on Changes of Nitric Oxide and Nitric Oxide Synthase in Rats." Korean Journal of Acupuncture 30, no. 2 (June 27, 2013): 97–103. http://dx.doi.org/10.14406/acu.2013.30.2.097.

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Han, Ji young, Younghwa Kim, Jeehye Sung, Yurry Um, Yi Lee, and Junsoo Lee. "Suppressive Effects of Chrysanthemum zawadskii var. latilobum Flower Extracts on Nitric Oxide Production and Inducible Nitric Oxide Synthase Expression." Journal of the Korean Society of Food Science and Nutrition 38, no. 12 (December 31, 2009): 1685–90. http://dx.doi.org/10.3746/jkfn.2009.38.12.1685.

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Ushamohan, B. P., Aravind Kumar Rajasekaran, Yamini Keshavaprasad Belur, Judu Ilavarasu, and T. M. Srinivasan. "Nitric Oxide, Humming and Bhramari Pranayama." Indian Journal Of Science And Technology 16, no. 5 (February 5, 2023): 377–84. http://dx.doi.org/10.17485/ijst/v16i5.1212.

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Burke-Martindale, CH. "Inhaled nitric oxide therapy for adult respiratory distress syndrome." Critical Care Nurse 18, no. 6 (December 30, 1998): 21–27. http://dx.doi.org/10.4037/ccn1998.18.6.21.

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The selective pulmonary vasodilatory effects of inhaled nitric oxide decrease pulmonary artery hypertension and improve arterial oxygenation in patients with ARDS without causing concomitant systemic vasodilation. Inhaled nitrix oxide therapy may decrease the prevalence of pulmonary edema, pulmonary barotrauma, and oxygen toxicity that occur with current ARDS treatment. The effect of nitric oxide on oxygenation and pulmonary artery pressure may allow more time for the lungs to recover. Initial results of clinical trials are encouraging; however, the impact of inhaled nitric oxide therapy on patients with ARDS remains unclear. Further research is needed to develop safe delivery systems and monitoring techniques for routine clinical use, to determine potential adverse and toxic effects of nitric oxide therapy on patients, and to determine the effects of long-term exposure to nitric oxide among healthcare workers. Concomitant administration of other medications with inhaled nitric oxide should also be investigated.
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yu, Hai Bo, Li Feng Ding, Ren Fei Wang, and Liu Lian. "The Research Progress of Nitric Oxides Controlling Technology." Advanced Materials Research 955-959 (June 2014): 2481–86. http://dx.doi.org/10.4028/www.scientific.net/amr.955-959.2481.

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Because of the special physic-chemical characters of the nitric oxides, controlling of the nitric oxides which shaped during combustion is a complex technology. In this paper, the advance in emission controlling technology of nitric oxide has been reviewed.Selective catalytic reaction of the nitric oxide(SCR) and plasma process in denitration of flue gas are emphasized.
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Danylovych, G. V., T. V. Bohach, and Yu V. Danylovych. "The biosynthesis of nitric oxide from L-arginine. Nitric oxide formation features and its functional role in mitochondria." Ukrainian Biochemical Journal 90, no. 1 (February 15, 2018): 3–24. http://dx.doi.org/10.15407/ubj90.01.003.

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&NA;. "Nitric oxide." Reactions Weekly &NA;, no. 547 (April 1995): 8. http://dx.doi.org/10.2165/00128415-199505470-00026.

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&NA;. "Nitric oxide." Reactions Weekly &NA;, no. 465 (August 1993): 10. http://dx.doi.org/10.2165/00128415-199304650-00043.

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&NA;. "Nitric oxide." Reactions Weekly &NA;, no. 623 (October 1996): 10. http://dx.doi.org/10.2165/00128415-199606230-00028.

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&NA;. "Nitric oxide." Reactions Weekly &NA;, no. 629 (November 1996): 9. http://dx.doi.org/10.2165/00128415-199606290-00023.

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Dissertations / Theses on the topic "Nitric oxide"

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Davies, Nathan Alun. "Nitric oxide in sepsis." Thesis, University of Essex, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285823.

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Molinari, Micol Ariella. "Nitric oxide synthase and the contribution of nitric oxide to vertebrate motor contol /." St Andrews, 2008. http://hdl.handle.net/10023/489.

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Illes, Mary. "Role of nitric oxide and nitric oxide synthases in the rice blast fungus." Thesis, University of Oxford, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670065.

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Petersson, Joel. "Nitrate, Nitrite and Nitric Oxide in Gastric Mucosal Defense." Doctoral thesis, Uppsala University, Department of Medical Cell Biology, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8624.

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The human stomach normally contains high levels of bioactive nitric oxide (NO). This NO derives from salivary nitrate (NO3-) that is converted to nitrite (NO2-) by oral bacteria and thereafter non-enzymatically reduced in the acidic gastric lumen to NO. Nitrate is a common component in vegetables, and after ingestion it is absorbed in the small intestine. Interestingly, circulating nitrate is then concentrated by the salivary glands. Hence, intake of nitrate-rich vegetables results in high levels of NO in the stomach. The physiological effects of the high concentration of NO gas normally present in the gastric lumen have been hitherto unknown, and the present investigations were therefore conducted to address this issue.

NO produced in the gastric lumen after nitrate ingestion increased gastric mucosal blood flow and the thickness of the firmly adherent mucus layer in the stomach. The blood flow and mucus layer are essential defense mechanisms that protect the mucosa from luminal acid and noxious agents. Nonsteroidal antiinflammatory drugs (NSAID) are commonly prescribed and effective drugs for treating pain and inflammation, but are associated with severe gastrointestinal side effects. We demonstrated that a nitrate-rich diet protects against NSAID-induced gastric damage, as a result of the increased formation of NO in the stomach. We also showed that the gastroprotective effect attributed to nitrate depended completely on conversion of nitrate to nitrite by the bacterial flora colonizing the tongue, and that the oral microflora is therefore important in regulating physiological conditions in the stomach.

In summary, this thesis challenges the current dogma that nitrate intake is hazardous, and on the contrary suggests that dietary nitrate plays a direct role in regulating gastric homeostasis. It is likely that a sufficient supply of nitrate in the diet together with the oral microflora is essential for preventing pathological conditions in the gastrointestinal tract.

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Madrasi, Kumpal J. "Preservation of Nitric Oxide Availability as Nitrite and Nitrosothiols." FIU Digital Commons, 2012. http://digitalcommons.fiu.edu/etd/805.

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Nitric Oxide (NO) has been known for long to regulate vessel tone. However, the close proximity of the site of NO production to “sinks” of NO such as hemoglobin (Hb) in blood suggest that blood will scavenge most of the NO produced. Therefore, it is unclear how NO is able to play its physiological roles. The current study deals with means by which this could be understood. Towards studying the role of nitrosothiols and nitrite in preserving NO availability, a study of the kinetics of glutathione (GSH) nitrosation by NO donors in aerated buffered solutions was undertaken first. Results suggest an increase in the rate of the corresponding nitrosothiol (GSNO) formation with an increase in GSH with a half-maximum constant EC50 that depends on NO concentration, thus indicating a significant contribution of ∙NO2 mediated nitrosation in the production of GSNO. Next, the ability of nitrite to be reduced to NO in the smooth muscle cells was evaluated. The NO formed was inhibited by sGC inhibitors and accelerated by activators and was independent of O2 concentration. Nitrite transport mechanisms and effects of exogenous nitrate on transport and reduction of nitrite were examined. The results showed that sGC can mediate nitrite reduction to NO and nitrite is transported across the smooth muscle cell membrane via anion channels, both of which can be attenuated by nitrate. Finally, a 2 – D axisymmetric diffusion model was constructed to test the accumulation of NO in the smooth muscle layer from reduction of nitrite. It was observed that at the end of the simulation period with physiological concentrations of nitrite in the smooth muscle cells (SMC), a low sustained NO generated from nitrite reduction could maintain significant sGC activity and might affect vessel tone. The major nitrosating mechanism in the circulation at reduced O2 levels was found to be anaerobic and a Cu+ dependent GSNO reduction activity was found to deliver minor amounts of NO from physiological GSNO levels in the tissue.
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Liu, Jia Clinical School Prince of Wales Hospital Faculty of Medicine UNSW. "Nitric oxide in airway inflammation." Publisher:University of New South Wales. Clinical School - Prince of Wales Hospital, 2009. http://handle.unsw.edu.au/1959.4/43678.

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Exhaled breath condensate (EBC) is a non-invasive method of investigating airway inflammation associated with nitric oxide (NO) and the metabolites nitrite/nitrates (NOx) in diseases such as chronic obstructive pulmonary disease (COPD), but some of the variables affecting the results are unknown. It was hypothesised that 1) EBC would be influenced by lung volumes and the type of EBC collection device; 2) fractional exhaled NO (FENO) and EBC NOx in COPD patients would be altered by smoking and glucocorticosteroids (GCS); 3) cigarette smoke could contribute to the EBC NOx concentration while it may also decrease FENO indirectly by converting airway NO to NOx. It was found that EBC volume was significantly correlated with both tidal volume and minute volume. Comparing four EBC collection devices demonstrated greater efficiency with the ECoScreen?? than siliconised glass tubes or RTube?? but it gave factitiously high NOx levels. Total EBC protein levels over a 10-minute collection were significantly higher using the ECoScreen?? than either glass or RTube?? devices. A cross-sectional study of 96 COPD patients and 80 age-matched control subjects demonstrated that FENO levels in COPD patients were significantly higher than normal subjects when comparing either the combined groups or appropriate two subgroups: ex-smokers and smokers. GCS treatment demonstrated no significant effect on either FENO levels or EBC NOx, but EBC NOx was elevated in smokers. In vitro, cigarette smoke extract (CSE) induced significantly higher NOx and asymmetric dimethylarginine (ADMA) levels in A549 cells when compared with control media. The anti-oxidant, NAC pre-treatment partially reversed the elevated NOx levels but not the ADMA levels. This thesis is the first to report FENO and EBC NOx in COPD patients in an appropriate sample size to be able to evaluate each subgroup, and the increased EBC NOx levels found in smokers in vivo was consistent with the elevated NOx level in response to CSE observed in vitro. These data indicate that smoking-related airway inflammation and activation of the NO pathway are complex with both an increase in ADMA, NO, NOx and may be regulated by oxidative stress rather than the nitric oxide synthase (NOS) pathway.
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Bescós, García Raúl. "The effect of nitric oxide donors on human performance." Doctoral thesis, Universitat de Barcelona, 2011. http://hdl.handle.net/10803/62896.

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Nitric oxide or nitrogen monoxide (NO) is a tiny free radical gas. The discovery of this intriguing molecule has revolutionized physiology and pharmacology research during the last 20 years. Currently, it is known that NO is endogenously synthesized by several molecules and tissues via two pathways: the synthase-dependent pathway and the synthase-independent pathway. In the first, the amino acid L-arginine is the main donnor of NO synthesis. In the second, inorganic nitrate is the main substrate for the synthesis of this molecule. Interestingly, both pathways are directly connected. While the synthase-dependent pathway is oxygen dependent, the synthase-independent metabolic route is greatly facilitated under hypoxia conditions. Thus, these mechanisms of NO production regulate levels of NO in the tissues. An adequate production of NO is important because it plays an essential role in mechanisms related with vasodilatation and blood flow distribution. Additionally, NO modulates other important functions in the human body such as mitochondrial respiration and immune mechanisms. For all these reasons, the interest for dietary NO donors have increased during the last years. It has been suggested that the consumption of food rich in L-arginine or in inorganic nitrate may enhance NO availability in the human body. This hypothesis has not been unnoticed in exercise physiology. In fact, it has been suggested that supplementation with NO donors may improve the cardio-respiratory response, as well as the tolerance to endurance exercise in humans. However, there is a lack of studies analyzing this issue. Therefore, the aim of this doctoral thesis was to assess the effect of L-arginine and inorganic nitrate in the cardio-respiratory and metabolic response of healthy humans. To develop this aim, three studies and one review were carried out. In the first, it was found that L-arginine supplementation during three days at several doses, between 5.5 and 20.5 g/day was not effective to increase plasma markers of NO, as well as the cardio-respiratory and metabolic response during endurance test. In the second study we found that acute dose of inorganic nitrate supplementation (10 mg/kg of body mass) raised significantly plasma levels of nitrate and nitrite. However, this effect did not report an improvement in the cardio-respiratory response at low-to-moderate intensities of exercise. However, at maximal work loads of exercise dietary nitrate induced significantly reduction of oxygen consumption (VO2peak) compared with placebo. Other cardio-respiratory parameters, as well as blood lactate concentration did not differ between nitrate and placebo. In addition, exercise performance measured as time to exhaustion during an incremental test did not increase compared with placebo. All these findings together suggested that at higher intensities of exercise energy production became more efficient after inorganic nitrate ingestion. Accordingly, in the third study it was analyzed the effect of dietary inorganic nitrate ingestion for three days during endurance exercise in a cycle ergometer at high intensity (time-trial of 40-min). Results of this study showed that nitrate supplementation did not increase significantly plasma levels of nitrite, as well as enhance performance in healthy subjects. Interestingly, a significant, negative correlation was found between change in nitrite and endurance capacity measured as VO2peak during the exercise test. These results indicated that the effect of dietary nitrate ingestion was lower in subjects with high cardiovascular capacity compared with subjects with poor tolerance capacity to endurance exercise. This fact is very important, since it is known that endurance training increase values of VO2peak in sedentary population and this fact is correlated with lower incidence of cardiovascular diseases. These and other important conclusions of these studies are included in the last work of this thesis which was a review article.
L’òxid nítric (NO) es un radical lliure alliberat per diverses molècules i teixits en l’organisme humà. El descobriment d’aquesta intrigant molècula ha revolucionat la recerca en el camp de la fisiologia i la farmacologia durant els últims 20 anys. Actualment, es coneix que la alliberació de NO per part de les cèl•lules endotelials estimula el procés de vasodilatació. A més, també es coneix que aquesta molècula es un important regulador de la respiració mitocondrial i del sistema immunològic. Totes aquestes funcions han generat un gran interès per els precursors nutricionals de NO. En l’àmbit de la fisiologia de l’exercici físic s’ha suggerit que la suplementació amb alguna d’aquestes substancies (L-arginina o nitrat inorgànic) pot millorar la tolerància a l’exercici físic de resistència. No obstant, hi ha molta controvèrsia en els resultats dels estudis que han analitzat aquesta hipòtesi. Per tant, l’objectiu principal d’aquesta tesi doctoral va ser analitzar els efectes dels principals precursors de NO, L-arginina i nitrat inorgànic, en la resposta cardiorrespiratòria i metabòlica durant l’exercici físic de caràcter aeròbic en humans. Per dur a terme aquest objectiu es van realitzar 3 estudis i una revisió bibliogràfica. Els principals resultats d’aquests estudis van mostrar que la suplementació de L-arginina en diferents dosis no va ser efectiva per augmentar el marcadors plasmàtics de NO, així com, la resposta cardiorrespiratòria i metabòlica durant un exercici físic aeròbic en intensitats moderades. En referència als nitrat inorgànic, es va observar que la suplementació augmenta els nivells d’aquests compostos en plasma. No obstant, aquest fet no es va correlacionar amb una millora de la tolerància a l’exercici físic de resistència. A més, es va observar una correlación negativa i significativa entre l’augment dels nitrits plasmàtics i la potència aeròbica màxima (VO2max). Tots aquests resultats van ser àmpliament tractats en l’últim treball (revisió bibliogràfica) d’aquesta tesi. En resum, l’ingesta nutricional de L-arginina i/o nitrat inorgànic no resulta efectiva per millorar la resposta cardiorrespiratòria i la tolerància a l’exercici físic de resistència en humans sans i entrenats físicament.
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Murrell, George Anthony Calvert St George Clinical School UNSW. "Nitric oxide and tendon healing." Awarded by:University of New South Wales. St George Clinical School, 2006. http://handle.unsw.edu.au/1959.4/31887.

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Nitric oxide is a small free radical generated by family of enzymes, the nitric oxide synthases. In a series of experiments performed over the last 15 years we showed that nitric oxide is induced by all three isoforms of nitric oxide synthase during tendon healing and that it plays a crucial beneficial role in restoring tendon function. In normal tendon we found very little nitric oxide synthase activity while in injured rat and human tendons nitric oxide synthase activity was expressed in healing fibroblasts in a temporal fashion. In healing rat Achilles tendon fibroblasts the first isoform to be expressed was endothelial nitric oxide synthase (eNOS), followed by inducible nitric oxide synthase (iNOS), and then brain or neuronal nitric oxide synthase (bNOS). Systemic inhibition of nitric oxide synthase activity decreased the cross sectional area and mechanical properties of the healing rodent Achilles tendons. Addition of nitric oxide via NO-flurbiprofen or NO-paracetamol enhanced rat Achilles tendon healing. Addition of nitric oxide to cultured human tendon cells via chemical means and via adenoviral transfection enhanced collagen synthesis, suggesting that one mechanism for the beneficial of nitric oxide on tendon healing might be via matrix synthesis. The final part of the work involved three randomized, double-blind clinical trials which evaluated the efficacy of nitric oxide donation via a patch in the management of the tendinopathy. In all three clinical trials there was a significant positive beneficial effect of nitric oxide donation to the clinical symptoms and function of patients with Achilles tendinopathy, tennis elbow and Achilles tendonitis.
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Gahm, Caroline. "Nitric oxide in brain contusion /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-392-2/.

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Ashley, Euan A. "Nitric oxide and cardiac function." Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249192.

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Books on the topic "Nitric oxide"

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Mayer, Bernd, ed. Nitric Oxide. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3.

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McCarthy, Helen O., and Jonathan A. Coulter, eds. Nitric Oxide. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61737-964-2.

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Mengel, Alexander, and Christian Lindermayr, eds. Nitric Oxide. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7695-9.

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Lester, Packer, ed. Nitric oxide. San Diego: Academic Press, 1996.

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Lester, Packer, ed. Nitric oxide. San Diego: Academic Press, 1996.

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Lester, Packer, ed. Nitric oxide. San Diego: Academic Press, 1999.

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Lester, Packer, ed. Nitric oxide. San Diego: Academic Press, 1996.

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service), ScienceDirect (Online, ed. Nitric oxide. San Diego, Calif: Elsevier/Academic Press, 2008.

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Lester, Packer, and Cadenas Enrique, eds. Nitric oxide. San Diego, Calif: Elsevier/Academic Press, 2005.

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Bruno, Tota, and Trimmer Barry, eds. Nitric oxide. Amsterdam: Elsevier, 2007.

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Book chapters on the topic "Nitric oxide"

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Moncada, S. "Introduction." In Nitric Oxide, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_1.

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Balligand, J. L. "Regulation of Cardiac Function by Nitric Oxide." In Nitric Oxide, 207–34. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_10.

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McNaughton, L., A. Radomski, G. Sawicki, and Marek W. Radomski. "Regulation of Platelet Function." In Nitric Oxide, 235–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_11.

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Garthwaite, J. "The Physiological Roles of Nitric Oxide in the Central Nervous System." In Nitric Oxide, 259–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_12.

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Martin, W. "The Role of Nitric Oxide in the Peripheral Nervous System." In Nitric Oxide, 277–313. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_13.

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Navarra, P., A. Costa, and A. Grossman. "Nitric Oxide and Neuroendocrine Function." In Nitric Oxide, 315–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_14.

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Hackenthal, E. "The Role of Nitric Oxide in Kidney Function." In Nitric Oxide, 329–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_15.

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Kojda, G. "Therapeutic Importance of Nitrovasodilators." In Nitric Oxide, 365–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_16.

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Vallance, P., D. Rees, and S. Moncada. "Therapeutic Potential of NOS Inhibitors in Septic Shock." In Nitric Oxide, 385–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_17.

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Keh, D., H. Gerlach, and K. Falke. "Inhalation Therapy with Nitric Oxide Gas." In Nitric Oxide, 399–441. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_18.

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Conference papers on the topic "Nitric oxide"

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Guzman Valderrabano, C. R., L. De la Vega-Morales, P. Troncoso-Huitron, and L. G. Gochicoa-Rangel. "Nasal Nitric Oxide Low Levels Detection Using Different Nitric Oxide Analyzers." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2358.

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Chazan, David, Vivek Balasubramanyam, Bhairavi Parikh, and Nina Peled. "The Effect Of Ambient Nitric Oxide On Exhaled Nitric Oxide Measurements." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4272.

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Solomon, Stanley C., Charles A. Barth, Penina Axelrad, Scott M. Bailey, Ronald Brown, Randal L. Davis, Timothy E. Holden, et al. "Student Nitric Oxide Explorer." In SPIE's 1996 International Symposium on Optical Science, Engineering, and Instrumentation, edited by E. Kane Casani and Mark A. Vander Does. SPIE, 1996. http://dx.doi.org/10.1117/12.255131.

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Delaney, L. "335. Health Care Workers' Exposures to Nitric Oxide, Nitrogen Dioxide, and Nitric Acid During Inhaled Nitric Oxide Therapy." In AIHce 2001. AIHA, 2001. http://dx.doi.org/10.3320/1.2765869.

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Bariatto, Marcelo, Rogerio Furlan, Koiti Arakai, and Jorge J. Santiago-Aviles. "A Simple Silicon Based Nitric Oxide Sensor." In ASME 1999 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1999. http://dx.doi.org/10.1115/imece1999-0327.

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Abstract Nitric oxide (NO) is known to mediate many beneficial physiology processes, motivating its detection in vivo as well as in vitro. Electrochemical detection provides the required cellular level determination of NO among several other techniques. In this work, electrochemical micro-sensors for both types of detection, in vivo and in vitro, were developed, exploring the silicon planar technology, which presents high yield and reliability and also permits batch fabrication. The developed in vitro sensor features eight detection sites (10 μm × 10 μm microelectrodes), for determination of nitric oxide spatial distribution or multi-species analysis. Different electrochemical methods were applied to provide sensor calibration and chemical reproducibility. For in vivo analysis, the designed structures have a needle shape (40 μm thick) and they were silicon micro-machined by using plasma etching or etch stop techniques. Different configurations were designed and implemented, containing a number of detection microelectrodes that vary from 2 to 10. The amperometric detection of both nitric oxide and nitride (NO2−) — a molecule that causes an interference — were investigated by using the in vitro micro-sensor configuration. The need of a cationic exchanger (Nafion) was demonstrated in order to provide selectivity to NO for low concentrations. Also, the developed sensor has a sensitivity of 500 A/M.cm2 and a detection limit of 10 μM.
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Parikh, Bhairavi R., Babs R. Soller, and Tal Rencus. "Reversibility of heme-nitric oxide reactions for use in an inhaled nitric oxide sensor." In BiOS '97, Part of Photonics West, edited by Tuan Vo-Dinh, Robert A. Lieberman, Gerald G. Vurek, and Abraham Katzir. SPIE, 1997. http://dx.doi.org/10.1117/12.275544.

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Wang, Wei-ding, Zi-qi Lai, Zi-cong Wu, Yi-fei Wang, and Zhi-ping Wang. "Evaluation of Nitric Oxide Scavenging and Nitric Oxide Synthases Expression of Resveratrol and Polydatin." In 2021 10th International Conference on Applied Science, Engineering and Technology (ICASET 2021). Paris, France: Atlantis Press, 2021. http://dx.doi.org/10.2991/aer.k.210817.003.

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Caton, Jerald A. "The Use of a Three-Zone Combustion Model to Determine Nitric Oxide Emissions From a Homogeneous-Charge, Spark-Ignited Engine." In ASME 2003 Internal Combustion Engine Division Spring Technical Conference. ASMEDC, 2003. http://dx.doi.org/10.1115/ices2003-0598.

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Abstract:
Nitric oxide emissions were estimated for a homogeneous-charge, spark-ignited automotive engine using a cycle simulation which employed three zones for the combustion process: (1) unburned gas, (2) adiabatic core region, and (3) boundary-layer gas. The use of the adiabatic core region has been shown to be especially necessary to capture the production of nitric oxides which are highly temperature dependent. The effects of major engine parameters such as equivalence ratio, spark timing, inlet manifold pressure, and engine speed on nitric oxide emissions are examined. In particular, the detail reasons for the effects of these engine parameters on the nitric oxide emissions are presented. Comparisons are completed between the computed values and a set of published measurements for the nitric oxide concentrations. Although not all engine parameters were known, reasonable agreement is demonstrated for most cases. In particular, the variations of nitric oxide concentrations as engine speed increased were duplicated. As an example, four operating conditions are examined in detail to help explain the measured results. Nitric oxide emissions are shown to be mainly the net result of gas temperatures, oxygen concentrations, and residence times.
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Eckel, Sandrah, Zilu Zhang, Edward Rappaport, Rima Habre, William Linn, Kiros Berhane, Bastain Theresa, Zhang Yue, and Gilliland Frank. "Indoor room nitric oxide and alveolar nitric oxide in the Southern California children's health study." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4503.

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Lopez, Raquel, Cristina Navarro, and Miguel Perpina. "Usefulness Of Alveolar Nitric Oxide And Bronchial Nitric Oxide Flux In The Diagnosis Of Asthma." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4313.

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Reports on the topic "Nitric oxide"

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Lala, Peeyush K. Nitric Oxide in Mammary Tumor Progression. Fort Belvoir, VA: Defense Technical Information Center, July 2001. http://dx.doi.org/10.21236/ada398946.

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Li, Nianzhen. Nitric Oxide in Astrocyte-Neuron Signaling. Office of Scientific and Technical Information (OSTI), January 2002. http://dx.doi.org/10.2172/803739.

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Flytzani-Stephanopoulos, M., A. F. Sarofim, and Y. Zhang. Direct catalytic decomposition of nitric oxide. Office of Scientific and Technical Information (OSTI), January 1992. http://dx.doi.org/10.2172/7045949.

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Flytzani-Stephanopoulos, M., A. F. Sarofim, and Y. Zhang. Direct catalytic decomposition of nitric oxide. Office of Scientific and Technical Information (OSTI), January 1992. http://dx.doi.org/10.2172/6930554.

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Flytzani-Stephanopoulos, M., A. F. Sarofim, and Y. Zhang. Direct catalytic decomposition of nitric oxide. Office of Scientific and Technical Information (OSTI), January 1992. http://dx.doi.org/10.2172/6788311.

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Armour, Elwood P. Nitric Oxide Gene Therapy for Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, April 2001. http://dx.doi.org/10.21236/ada395737.

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Flytzani-Stephanopoulos, M., A. F. Sarofim, and Yanping Zhang. Direct catalytic decomposition of nitric oxide. Final report. Office of Scientific and Technical Information (OSTI), June 1995. http://dx.doi.org/10.2172/111932.

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Przedborski, Serge E. Role of Nitric Oxide in MPTP Induced Dopaminergic Neuron. Fort Belvoir, VA: Defense Technical Information Center, September 2000. http://dx.doi.org/10.21236/ada384796.

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Przedborski, Serge E. Role of Nitric Oxide in MPTP-Induced Dopaminergic Neuron Degeneration. Fort Belvoir, VA: Defense Technical Information Center, September 2004. http://dx.doi.org/10.21236/ada450371.

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Witten, Mark L. Role of Nitric Oxide in a Model of Toxic Exposure. Fort Belvoir, VA: Defense Technical Information Center, August 2010. http://dx.doi.org/10.21236/ada532841.

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