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Books on the topic 'Nicotinic Acetylcholine Receptor'

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Published: 4 June 2021
Last updated: 9 March 2023

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1

Maelicke, Alfred, ed. Nicotinic Acetylcholine Receptor. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71649-2.

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2

Barrantes, Francisco J., ed. The Nicotinic Acetylcholine Receptor. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-40279-5.

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3

Li, Ming D., ed. Nicotinic Acetylcholine Receptor Technologies. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3768-4.

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4

Yamamoto, Izuru, and John E. Casida, eds. Nicotinoid Insecticides and the Nicotinic Acetylcholine Receptor. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67933-2.

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5

1928-, Yamamoto Izuru, and Casida John E. 1929-, eds. Nicotinoid insecticides and the nicotinic acetylcholine receptor. Tokyo: Springer, 1999.

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6

Alfred, Maelicke, ed. Nicotinic acetylcholine receptor: Structure and function. Berlin: Springer-Verlag, 1986.

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7

Akaike, Akinori, Shun Shimohama, and Yoshimi Misu, eds. Nicotinic Acetylcholine Receptor Signaling in Neuroprotection. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8488-1.

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8

Insect nicotinic acetylcholine receptors. New York: Springer Science+Business Media, 2010.

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9

Thany, Steeve Hervé, ed. Insect Nicotinic Acetylcholine Receptors. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6445-8.

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10

A, Selyanko A., and Derkach V. A, eds. Neuronal acetylcholine receptors. New York: Consultants Bureau, 1989.

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11

Francesco, Clementi, Gotti Cecilia, Sher Emanuele, and North Atlantic Treaty Organization. Scientific Affairs Division., eds. Nicotinic acetylcholine receptors in the nervous system. Berlin: Springer-Verlag, 1988.

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12

1944-, Barrantes Francisco J., ed. The nicotinic acetylcholine receptors: Current views and future trends. Berlin: Springer, 1998.

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13

Clementi, Francesco, Cecilia Gotti, and Emanuele Sher, eds. Nicotinic Acetylcholine Receptors in the Nervous System. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-74167-8.

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14

Slater, E. Yvonne. The effects of novel alkaloid derivatives on human nicotinic acetylcholine receptors. Oxford: Oxford Brookes University, 2000.

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15

Jackson, Charles E. P. The pharmacology of nicotinic acetylcholine receptors in Heliothis virescens and Locusta migratoria neurones in vitro. Oxford: Oxford Brookes University, 1998.

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16

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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17

A, Nordberg, and International Symposium on Nicotinic Receptors on the CNS - Their Role in Synaptic Transmission (1988 : Uppsala, Sweden), eds. Nicotinic receptors in the CNS: Their role in synaptic transmission. Amsterdam: Elsevier, 1989.

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18

1958-, Parhar Ishwar S., ed. Gonadotropin-releasing hormone: Molecules and receptors. Amsterdam: Elsevier, 2002.

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19

P, Illes, and Zimmermann Herbert 1944-, eds. Nucleotides and their receptors in the nervous system. Amsterdam: Elsevier, 1999.

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20

Masao, Norita, Bando Takehiko, and Stein Barry E, eds. Extrageniculostriate mechanisms underlying visually-guided orientation behavior. Amsterdam: Elsevier, 1996.

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21

J, Allum J. H., ed. Natural and artificial control of hearing and balance. Amsterdam: Elsevier, 1993.

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22

C, Polosa, and Weaver Lynne C. 1945-, eds. Autonomic dysfunction after spinal cord injury. Amsterdam: Elsevier, 2006.

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23

S, Martinez-Conde, and European Conference on Visual Perception (28th : 2005 : La Coruña, Spain), eds. Visual Perception. Amsterdam: Elsevier, 2006.

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24

H, Yu Albert C., ed. Neuronal-astrocytic interactions: Implications for normal and pathological CNS function. Amsterdam: Elsevier, 1992.

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25

B, Dunnett S., and Björklund Anders 1945-, eds. Functional neural transplantation II: Novel cell therapies for CNS disorders. Amsterdam: Elsevier, 2000.

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26

M, Gerrits N., Ruigrok T. J. H, and Zeeuw C. I. de, eds. Cerebellar modules: Molecules, morphology, and function. Amsterdam: Elsevier, 2000.

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27

1957-, Castellano Bernardo, and Nieto-Sampedro Manuel 1944-, eds. Glial cell function. Amsterdam: Elsevier, 2001.

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28

S, Martinez-Conde, and European Conference on Visual Perception (28th : 2005 : La Coruna, Spain), eds. Visual Perception. Amsterdam: Elsevier, 2006.

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29

1935-, Barnes Charles D., Pompeiano O, Universitá di Pisa. Dipartimento di Fisiologia e Biochimica., and Washington State University. Dept. of Veterinary and Comparative Anatomy, Pharmacology, and Physiology., eds. Neurobiology of the locus coeruleus. Amsterdam: Elsevier, 1991.

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30

G, Holstege, Bandler Richard, and Saper C. B, eds. The emotional motor system. Amsterdam: Elsevier, 1996.

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31

Jochen, Klein, Löffelholz Konrad, and International Cholinergic Symposium (9th : 1995 : Mainz, Rhineland-Palatinate, Germany), eds. Cholinergic mechanisms: From molecular biology to clinical significance. Amsterdam: Elsevier, 1996.

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32

Luigi, Aloe, and Calzà Laura, eds. NGF and related molecules in health and disease. Amsterdam: Elsevier, 2004.

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33

Nobel Symposium (76th 1989 Lidingö, Sweden). Cholinergic neurotransmission: Functional and clinical aspects : proceedings of Nobel Symposium 76. Amsterdam: Elsevier, 1990.

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34

Li, Ming D. Nicotinic Acetylcholine Receptor Technologies. Springer New York, 2018.

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35

Li, Ming D. Nicotinic Acetylcholine Receptor Technologies. Humana Press, 2016.

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36

Casida, J. E., and I. Yamamoto. Nicotinoid Insecticides and the Nicotinic Acetylcholine Receptor. Springer London, Limited, 2012.

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37

Yamamoto, I. Nicotinoid Insecticides and the Nicotinic Acetylcholine Receptor. Springer, 2013.

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38

Nicotinic Acetylcholine Receptor Signaling in Neuroprotection. Springer, 2019.

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39

Maelicke, Alfred. Nicotinic Acetylcholine Receptor: Structure and Function. Springer, 2011.

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40

Shimohama, Shun, Akinori Akaike, and Yoshimi Misu. Nicotinic Acetylcholine Receptor Signaling in Neuroprotection. Saint Philip Street Press, 2020.

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41

Nicotinic Acetylcholine Receptor: Structure and Function. Springer, 1986.

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42

Maelicke, Alfred. Nicotinic Acetylcholine Receptor: Structure and Function. Springer London, Limited, 2013.

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43

Barrantes, Francisco Jose. Nicotinic Acetylcholine Receptor: Current Views and Future Trends. Springer London, Limited, 2013.

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44

Barrantes, Francisco Jose. Nicotinic Acetylcholine Receptor: Current Views and Future Trends. Springer Berlin / Heidelberg, 1998.

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45

Maelicke, Alfred. Nicotinic Acetylcholine Receptor: Structure and Function (Nato Asi Series, Series H : Cell Biology, Vol 3). Springer, 1987.

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46

Barrantes, Francisco J. The Nicotinic Acetylcholine Receptor: Current Views and Future Trends (Biotechnology Intelligence Unit). Springer, 1998.

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47

Barrantes, Francisco Jose. The Nicotinic Acetylcholine Receptor: Current Views and Future Trends (Neuroscience Intelligence Unit). R G Landes Co, 1998.

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48

Lobel, Peter. Structural studies of a curarimimetic cobra toxin and of the nicotinic acetylcholine receptor. 1986.

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49

Lambert, David G. Mechanisms and determinants of anaesthetic drug action. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0013.

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Abstract:
This chapter is broken into two main sections: a general description of the principles of ligand receptor interaction and a discussion of the main groups of ‘targets’; and explanation of some common pharmacological interactions in anaesthesia, critical care, and pain management. Agonists bind to and activate receptors while antagonists bind to receptors and block the effects of agonists. Antagonists can be competitive (most common) or non-competitive/irreversible. The main classes of drug target are enzymes, carriers, ion channels, and receptors with examples of anaesthetic relevance interacting with all classes. There are many examples in anaesthesia where multiple interacting drugs are co-administered—polypharmacology. To give an example: neuromuscular blockade. Rocuronium is a non-depolarizing neuromuscular blocker acting as a competitive antagonist at the nicotinic acetylcholine receptor. Rocuronium competes with endogenous acetylcholine to shift the concentration–response curve for contraction to the right. The degree of contractility is less for a given concentration of acetylcholine (agonist) in the presence of rocuronium. Using the same principle, the rightward shift can be compensated by increasing the amount of acetylcholine (as long as the amount of rocuronium presented to the receptor as an antagonist remains unchanged, its action can be overcome by increased agonist). Acetylcholine at the effect site is increased by acetylcholinesterase inhibition with neostigmine. One of the side-effects of neostigmine is that it acts as an indirect parasympathomimetic. In the cardiovascular system this would lead to muscarinic receptor-mediated bradycardia; these effects are routinely reversed by the competitive muscarinic antagonist glycopyrrolate.
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50

Regulation of neuronal nicotinic acetylcholine receptor expression in embryonic chick sympathetic neurons by preganglionic-derived isform of neuregulin: NARIA. 1997.

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