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Books on the topic 'Neuropathic pain'

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Author: Grafiati
Published: 4 June 2021
Last updated: 20 July 2024

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1

Toth, Cory, and Dwight Moulin, eds. Neuropathic Pain. Cambridge: Cambridge University Press, 2013. http://dx.doi.org/10.1017/cbo9781139152211.

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2

I, Bennett Michael, ed. Neuropathic pain. Oxford: Oxford University Press, 2006.

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3

Misha-Miroslav, Backonja, ed. Neuropathic pain syndromes. Philadelphia: Saunders, 1998.

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4

Peripheral neuropathy & neuropathic pain: Into the light. Shrewsbury, England: TFM Publishing Ltd, 2015.

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5

Smith, Daryl I., and Hai Tran, eds. Pathogenesis of Neuropathic Pain. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-91455-4.

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6

Chemotherapy induced neuropathic pain. Boca Raton: CRC Press, 2012.

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7

Galer, Bradley S. A clinical guide to neuropathic pain. [New York]: McGraw Hill, Healthcare Information Programs, 2000.

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8

1966-, Malmberg Annika B., and Chaplan Sandra R, eds. Mechanisms and mediators of neuropathic pain. Basel: Birkhäuser Verlag, 2002.

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9

de Castro, Jeimylo, and Yasser El Miedany, eds. Advances in Chronic and Neuropathic Pain. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-10687-3.

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10

Malmberg, Annika B., and Sandra R. Chaplan, eds. Mechanisms and Mediators of Neuropathic Pain. Basel: Birkhäuser Basel, 2002. http://dx.doi.org/10.1007/978-3-0348-8129-6.

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11

Galer, Bradley S. A clinical guide to neuropathic pain. [New York]: McGraw Hill, Healthcare Information Programs, 2000.

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12

Neuropathic pain: Mechanisms, diagnosis, and treatment. New York: Oxford University Press, 2012.

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13

Galer, Bradley S. A clinical guide to neuropathic pain. [New York]: McGraw Hill, Healthcare Information Programs, 2000.

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14

Boulton, Andrew J. M., and Loretta Vileikyte. Managing Neuropathic Pain in the Diabetic Patient. Tarporley: Springer Healthcare Ltd., 2009. http://dx.doi.org/10.1007/978-1-908517-16-6.

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15

Bennett, Michael Ian. The development of a pain scale for identifying neuropathic pain. Birmingham: University of Birmingham, 1999.

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16

Gunn, C. Chan. Treating myofascial pain: Intramuscular stimulation (IMS) for muofascial pain syndromes of neuropathic origin. Seattle, WA: Multidisciplinary Pain Center, University of Washington Medical School, 1989.

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17

Spicher, Claude J., Tara L. Packham, Nadège Buchet, Isabelle Quintal, and Pierre Sprumont. Atlas of Cutaneous Branch Territories for the Diagnosis of Neuropathic Pain. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-45964-2.

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18

R, Jadad Alejandro, McMaster University. Evidence-based Practice Center., and United States. Agency for Healthcare Research and Quality., eds. Management of chronic central neuropathic pain following traumatic spinal cord injury. Rockville, Md: Agency for Healthcare Research and Quality, 2001.

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19

(Austin, Tex ). PBG Consulting. The neuropathic pain market: A strategic commercial opportunity 2001 : a market analysis. Austin, Tex: PBG Consulting, 2002.

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20

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Neuropathic pain. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0005.

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This chapter on neuropathic pain discusses the classification, clinical features, and evidence-based management of major neuropathic pain syndromes (painful polyneuropathy, diabetic neuropathy, post-herpetic neuralgia, HIV neuropathy, cancer neuropathic pain, phantom pain, traumatic neuropathic pain, chronic radiculopathy, central neuropathic pain, and trigeminal neuralgia).
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21

Scadding, John. Neuropathic pain. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0386.

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Pain signalled by a normal sensory system, nociceptive pain, serves a vital protective function. The peripheral and central nervous somatosensory systems permit rapid localization and identification of the nature of painful stimuli, prior to appropriate action to minimize or avoid potentially tissue damaging events. A reduction or absence of pain resulting from neurological disease emphasizes the importance of this normal protective function of pain. For example, tissue destruction occurs frequently in peripheral nerve diseases which cause severe sensory loss such as leprosy, and in central disorders such as syringomyelia. Neuropathic pain results from damage to somatosensory pathways and serves no protective function. This chapter provides an overview of neuropathic pain, considering its context, clinical features, pathophysiology, and treatment.In the peripheral nervous system, neuropathic pain is caused by conditions affecting small nerve fibres, and in the central nervous system by lesions of the spinothalamic tract and thalamus, and rarely by subcortical and cortical lesions. The clinical feature common to virtually all conditions leading to the development of neuropathic pain is the perception of pain in an area of sensory impairment, an apparently paradoxical situation. The exception is trigeminal neuralgia.Neuropathic pain is heterogeneous clinically, aetiologically, and pathophysiologically. Within a given diagnostic category, whether defined clinically or aetiologically, there are wide variations in reports of pain by patients. This heterogeneity poses one of the greatest challenges in understanding the mechanisms of neuropathic pain. Knowledge of the pathophysiology is an obvious pre-requisite to the development of effective treatments. The goal of a pathophysiologically based understanding of the symptoms and signs of neuropathic pain is, of course, just such a rational and specific approach to treatment. While this is not yet achievable, clinical-pathophysiological correlations have led to some recent advances in treatment.
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22

Bennett, Michael, ed. Neuropathic Pain. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199563678.001.0001.

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23

Khursheed, Faraz, and Marc O. Maybauer. Neuropathic Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0012.

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Neuropathic pain is a common condition that arises from injury anywhere along the somatosensory axis. Although the presentation may vary based on mechanisms and locations of injury, most patients have characteristic burning, shocklike, lancinating pain, most often in the distribution of peripheral and spinal nerves or distal extremities. Various peripheral and central processes aggravate pain through abnormal impulse generation, modulation, and processing. Common conditions include complex regional pain syndrome, diabetic neuropathy, postherpetic neuralgia, spondylotic radiculopathy, and central pain syndromes. A detailed history and physical examination will aid in differentiating various neuropathic pain conditions. Neuropathic pain is best managed using a true multidisciplinary approach.
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24

Cheng, Jianguo, ed. Neuropathic Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190298357.001.0001.

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Neuropathic pain is a category of chronic pain disorders that are most common, debilitating, costly, and difficult to treat. It is a significant challenge to individuals suffering from it, healthcare providers, and society at large. This book is written by expert clinicians and investigators from multiple disciplines to provide the most comprehensive and updated information on neuropathic pain disorders that are commonly encountered in clinical practice. It strives to reflect the current understanding of the concepts, classification, mechanisms, assessment, diagnosis, and treatment of neuropathic pain. Following chapters addressing these topics in general terms are chapters devoted to specific neuropathic pain disorders consequent to lesions or diseases of the central and peripheral nervous systems. These chapters take a case-based format to stimulate situation-guided thinking, predicting, and learning. The textbook serves to inform best practices and stimulate innovative investigations to advance patient care, as well as the science behind it.
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25

Udeagha, Cyprian Chukwunonye, ed. Neuropathic Pain. InTech, 2012. http://dx.doi.org/10.5772/2381.

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26

Fowler, Ian M., Robert J. Hackworth, and Erik P. Voogd. Neuropathic Pain. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190217518.003.0024.

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Neuropathic pain encompasses a vast number of clinical conditions that share the common characteristic of pain resulting from nerve injury or damage. Upon injury, pathophysiologic changes in the peripheral nervous system occur, including hyperexcitability and the spontaneous generation of impulses (ectopia). As a result of these peripheral changes, alterations in signal processing and intrinsic changes within the central nervous system occur. All of these changes contribute to the generation of neuropathic pain. This chapter attempts to capture the essence of the objectives and goals set forth by the International Association for the Study of Pain’s Core Curriculum for Professional Education in Pain for the topic of neuropathic pain. The questions cover topics including definitions, common clinical conditions, uncommon clinical conditions, therapeutic interventions, pathophysiological mechanisms, and current investigations.
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27

Dworkin, Robert H. Neuropathic Pain. Edited by David M. Simpson and Justin C. McArthur. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780195394702.001.0001.

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28

Bennett, Michael. Neuropathic Pain. Oxford University Press, 2012.

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29

Attal, Nadine, and Didier Bouhassira. Neuropathic Pain. Oxford University Press, Incorporated, 2023.

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30

Bennett, Michael. Neuropathic Pain. Oxford University Press, Incorporated, 2010.

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31

Neuropathic Pain. Oxford University Press, 2010.

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32

Pranav, Heena, and Dalia H. Elmofty. Postmastectomy Neuropathic Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0015.

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Neuropathic pain can burden patients in multiple domains. It is a complex pain syndrome that remains difficult to treat. Detailed history and physical examination would reveal patients with neuropathic conditions complaining of burning, shocklike pain. There are a variety of neuropathic pain conditions that can be considered in a differential diagnosis. Because patients with neuropathic pain fail to obtain satisfactory relief from pharmacologic agents alone, a multidisciplinary approach that includes preventative and interventional options is recommended. Interventional treatment options may offer relief to patients with refractory neuropathic pain. Success in the treatment of neuropathic pain depends on evidence-based medicine and individualized patient care.
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33

Wong, Stacy N., and Line G. Jacques. Neuropathic Groin Pain. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0017.

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Chronic neuropathic groin pain may be iatrogenic or posttraumatic and can be disabling or even crippling in some individuals. Patients may have significant sleep disturbances and may experience psychosocial effects along with significant physical limitations. A combination of pharmacologic treatments with physical therapy and local infiltrations may be useful. Neurostimulation techniques, including spinal cord stimulation, peripheral nerve stimulation, and dorsal root ganglion stimulation, have shown promising results in the treatment of chronic neuropathic pain. In certain cases, surgical approaches, including selective neurectomy, can be effective; in other cases, the pain will remain chronic and intractable despite all interventional measures. In summary, patients with neuropathic groin pain can be treated after a thorough pretreatment investigation. Dorsal root ganglion stimulation is a viable option and should be considered when treating this challenging patient population.
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34

Donaldson, Katelyn, and Ahmet Höke. Animal Models of Peripheral Neuropathy and Neuropathic Pain. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0119.

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There are numerous types of peripheral neuropathies and conditions that cause neuropathic pain with varying symptoms and different mechanisms of pathogenesis. Therefore, it is not surprising that many different animal models of peripheral neuropathies and neuropathic pain have been developed with varying degrees of fidelity to recapitulate the human disease. Nevertheless, these models are useful in a deconstructive manner to examine role of specific molecular pathways in pathogenesis of different types of peripheral neuropathies and test potential new drugs.
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35

Bennett, Michael. Neuropathic Pain (Oxford Pain Management Library). Oxford University Press, USA, 2006.

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36

Raffa, Robert B., Richard Langford, Ronald J. Tallarida, Frank Porreca, and Joseph V. Pergolizzi Jr. Chemotherapy-Induced Neuropathic Pain. Taylor & Francis Group, 2012.

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37

Challenging Neuropathic Pain Syndromes. Elsevier, 2018. http://dx.doi.org/10.1016/c2016-0-01358-x.

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38

Raffa, Robert B., Richard Langford, Ronald J. Tallarida, Frank Porreca, and Jr Pergolizzi. Chemotherapy-Induced Neuropathic Pain. Taylor & Francis Group, 2019.

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39

Simpson, David, and Kathryn Elliot. HIV and Neuropathic Pain. Oxford University Press, 2010.

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40

Raffa, Robert B., Richard Langford, Ronald J. Tallarida, Frank Porreca, and Joseph V. Pergolizzi Jr. Chemotherapy-Induced Neuropathic Pain. Taylor & Francis Group, 2012.

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41

Raffa, Robert B. Chemotherapy-Induced Neuropathic Pain. Taylor & Francis Group, 2012.

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42

Teeters, Brandon. Neuropathic Pain: Detailed Steps to Easy Understanding and Treatment of Neuropathic Pain. Independently Published, 2019.

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43

Central neuropathic pain: Focus on poststroke pain. Seattle: IASP Press, 2007.

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44

Bennett, Michael. Neuropathic Pain (Oxford Pain Management Library S.). Oxford University Press, USA, 2006.

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45

Central neuropathic pain: Focus on poststroke pain. Seattle, WA: IASP Press, 2007.

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46

James L, Ph.D. Henry (Editor), Akba Panju (Editor), and Kiran, Ph.D. Yashpal (Editor), eds. Central Neuropathic Pain: Focus on Poststroke Pain. Intl Assoc for the Study of Pain, 2007.

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47

Kastten, Mary J. CBD Oil for Neuropathic Pain. Independently Published, 2019.

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48

Wainger, Brian J. Drug Discovery and Neuropathic Pain. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0117.

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Pain is one of the most common causes of physician visits and disability. Pain has been classified into specific subtypes. We refer to baseline or nociceptive pain as pain that results from an ongoing, high-threshold stimulus acting on an unenhanced somatosensory system. Inflammatory pain refers to pain in the setting of tissue damage and specifically the release of inflammatory molecules that activate and sensitize the nociceptive machinery. Hyperalgesia, or increased pain in response to a noxious stimulus, results from nociceptor sensitization whereas neuropathic pain results from a lesion or disease of the somatosensory system. Pain can have spontaneous, stimulus-independent components as well as evoked components such as hyperalgesia or allodynia, pain that is elicited by a normally innocuous stimulus. This chapter describes the research strategy for discovering new drugs to relieve these different kinds of pain.
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49

Mowafak, Abdelghani. Botox analgesia for neuropathic pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0072.

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The landmark paper discussed in this chapter is ‘Botulinum toxin type A induces direct analgesic effects in chronic neuropathic pain’, published by Ranoux et al. in 2008. The muscle-relaxant properties of botulinum toxin type A (BTX-A) have long been known and used therapeutically. However, BTX-A actions independent of those affecting neuromuscular function had previously only been alluded to, via the observation that BTX-A injections alleviated pain more than would have been expected through the treatment of muscle spasm alone. This paper was one of the first to specifically examine the efficacy of BTX-A in neuropathic pain and was a catalyst for further investigation into BTX-A as a novel analgesic.
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50

Saberski, Ean, and Lloyd Saberski. Management of Neuropathic Postoperative Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457006.003.0019.

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Pain following surgery is routine and unavoidable but expected to resolve with time. In some cases, postoperative pain persists as the result of a neuropathic process such as a neuroma or nerve entrapment. Postoperative neuropathic pain is physiologically distinct from acute pain, but the mechanisms by which pain is transduced, transmitted, decoded, and modulated are shared. Effective treatment regimens for postoperative neuropathic pain employ a deliberate strategy to disrupt the aberrant nociceptive signal. Some surgeries are high risk for chronic postoperative pain with postherniorrhaphy pain syndrome and persistent pain following breast cancer surgery existing as well described entities in the literature.
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