Relevant bibliographies by topics / Neuromuscular function

Academic literature on the topic 'Neuromuscular function'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 April 2024

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Journal articles on the topic "Neuromuscular function"

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Beemer, G. H., and P. Rozental. "Postoperative Neuromuscular Function." Anaesthesia and Intensive Care 14, no. 1 (February 1986): 41–45. http://dx.doi.org/10.1177/0310057x8601400110.

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One hundred patients who received a competitive neuromuscular blocking agent during anaesthesia were randomly selected for evaluation of neuromuscular function immediately on their arrival in the recovery room. The anaesthetist was not aware that the patient would be evaluated in the recovery room. Neuromuscular function was assessed by a train-of-four (TOF) ratio, and in conscious and co-operative patients by a series of bedside tests of neuromuscular function. Twenty-one patients had a TOF ratio of less than 0.70 and seven patients a TOF ratio of less than 0.60. Bedside tests of neuromuscular function did not reliably detect this defect in neuromuscular transmission. It is concluded that a relatively large number of patients have a defect in neuromuscular transmission on their arrival in the recovery room, and suggested that this reflects the inadequacy of clinical methods used for the administration and antagonism of competitive neuromuscular blocking agents at this institution.
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Ortega, Rafael, Sorin J. Brull, Richard Prielipp, Alexander Gutierrez, Rossemary De La Cruz, and Christopher M. Conley. "Monitoring Neuromuscular Function." New England Journal of Medicine 378, no. 4 (January 25, 2018): e6. http://dx.doi.org/10.1056/nejmvcm1603741.

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Suh, Jae Hyeun. "Neuromuscular Function Monitoring." Korean Journal of Anesthesiology 24, no. 2 (1991): 219. http://dx.doi.org/10.4097/kjae.1991.24.2.219.

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Engbaek, J. "Monitoring neuromuscular function." Current Opinion in Anaesthesiology 2, no. 4 (August 1989): 479–83. http://dx.doi.org/10.1097/00001503-198908000-00020.

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Kopman, Aaron F. "Monitoring Neuromuscular Function." ASA Refresher Courses in Anesthesiology 39, no. 1 (2011): 72–79. http://dx.doi.org/10.1097/asa.0b013e3182284668.

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Monti, Elizabeth J., Mary E. Kerr, and Catherine Bender. "Monitoring Neuromuscular Function." Journal of Neuroscience Nursing 27, no. 4 (August 1995): 252–57. http://dx.doi.org/10.1097/01376517-199508000-00011.

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ALI, HASSAN H. "Monitoring Neuromuscular Function." Anesthesiology 64, no. 4 (April 1, 1986): 532. http://dx.doi.org/10.1097/00000542-198604000-00034.

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KOPMAN, AARON F. "Monitoring Neuromuscular Function." Anesthesiology 64, no. 4 (April 1, 1986): 532. http://dx.doi.org/10.1097/00000542-198604000-00035.

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Feldman, Stanley A., and Adrian J. England. "Neuromuscular function and block." Current Opinion in Anaesthesiology 8, no. 4 (August 1995): 351–55. http://dx.doi.org/10.1097/00001503-199508000-00015.

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Wareham, Anthony C. "Neuromuscular function and transmission." Anaesthesia & Intensive Care Medicine 6, no. 6 (June 2005): 203–5. http://dx.doi.org/10.1383/anes.6.6.203.65787.

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Dissertations / Theses on the topic "Neuromuscular function"

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PELLEGRINO, GIULIA MICHELA. "LUNG FUNCTION AND DYSPNEA IN NEUROMUSCULAR DISEASES." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/842435.

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Dyspnea is a common source of suffering for patients affected by cardiorespiratory or neuromuscular diseases. The symptom is complex and encompasses different sensory qualities with distinct intensities. The Multidimensional Dyspnea Profile (MDP) is an instrument specifically developed to assess the multidimensional dimensions of the symptom, and it is applicable in both the research and clinical setting. In order to allow its use for Italian speaking populations, we aimed to provide a linguistically validated, Italian translation of the MDP. We conducted a structured translation and linguistic validation of the MDP questionnaire in accordance to the international guidelines and in cooperation with a specialized company (MAPI SAS, Language Services Unit, Lyon, France). Cognitive interviews on 8 patients were conducted in order to test clarity and understandability of the questionnaire. The multistep process was enriched by several quality checks which led to a translation conceptually equivalent to the original version (American English). A final certified copy linguistically validated Italian translation of the MDP is now available. It measures the intensity of the breathing discomforts in five sensory qualities and assess its intensity and potential reactions. W e here provide an Italian translation and linguistic validation of the MDP. This instrument, allows the assessment of dyspnea in both its sensory and emotional aspects, therefore representing a valuable method for research and therapy purposes.
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Kalmar, Jayne M. "The effects of caffeine on human neuromuscular function." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ33488.pdf.

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Weinberg, Jan. "Studies on ventilatory function in chronic neuromuscular disorders /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3936-5/.

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Auburn, Richard Peter. "Screening for genes involved in Drosophila neuromuscular function." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621081.

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Penney, Jay. "Retrograde regulation of synaptic function at «Drosophila» neuromuscular junctions." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=119497.

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Neuronal growth and synaptic function are key determinants of nervous system behaviour. Understanding the molecular mechanisms that regulate these processes is central to comprehending nervous system function, both in health and during disease states. My thesis has focused on trying to better understand the mechanisms controlling glutamatergic neuronal growth, synaptic function and synaptic plasticity using the Drosophila melanogaster larval neuromuscular junction (NMJ) as a model system. This work has concentrated on the role that protein synthesis regulators play in modulating NMJ function and plasticity, and has revealed important roles for multiple regulators of cap-dependent translation in these processes. Importantly, genetic manipulations predicted to enhance cap-dependent translation in postsynaptic muscle can induce retrograde enhancements in synaptic function. In each case, these manipulations are without effect on neuronal growth, the number of synaptic sites or glutamate receptor levels. Instead, enhanced postsynaptic cap-dependent translation leads to increased presynaptic release probability. This retrograde pathway plays a key role in the expression of homeostatic synaptic plasticity at NMJ synapses. Glutamate receptor subunit IIA (GluRIIA) mutants exhibit reduced postsynaptic receptor function which is compensated for by enhanced presynaptic release; cap-dependent translation, under control of the TOR (target of rapamycin) pathway, is essential for this retrograde homeostatic response. Furthermore, we find that Parkinson's disease related protein LRRK2 (leucine rich repeat kinase 2), and its Drosophila orthologue dLRRK, can regulate synaptic function at the NMJ via cap-dependent translation. Importantly, however, mutant forms of LRRK2 are defective in their ability to regulate synaptic strength. Together, these findings further our understanding of the molecular mechanisms regulating glutamatergic synaptic function and plasticity.
La croissance neuronale et la fonction synaptique sont des constituants clés dans le comportement du système nerveux. Il est donc essentiel de comprendre les mécanismes moléculaires régulant ces processus à l'état normal ainsi qu'à l'état pathologique. Ma thèse a pour objectif une meilleure compréhension des mécanismes contrôlant la croissance neuronale, ainsi que la fonction et la plasticité synaptiques engendrés par la transmission glutamatergique, en utilisant comme modèle la jonction neuromusculaire (JNM) de la drosophile, Drosophila melanogaster. Cet ouvrage porte spécifiquement sur l'impact de régulateurs de la synthèse protéique sur la fonction et la plasticité synaptiques dans la JNM, et a identifié des rôles importants pour de multiples régulateurs de traduction coiffe-dépendante dans ces processus. Des manipulations génétiques menant à une augmentation de la traduction coiffe-dépendante dans le muscle post-synaptique induisent une augmentation rétrograde de la fonction synaptique. Dans chaque cas, ces manipulations n'affectent ni la croissance neuronale, ni le nombre de synapses, ni le niveau de récepteurs glutamatergiques. Plutôt, une traduction coiffe-dépendante post-synaptique conduit vers une augmentation de la probabilité de relâche pré-synaptique. Cette voie signalétique joue un rôle important dans l'expression de la plasticité homéostatique des synapses neuromusculaires. Des mutants du récepteur glutamatergique IIA (GluRIIA) montrent une réduction dans la fonction post-synaptique, qui est compensée par une augmentation de la probabilité de relâche pré-synaptique. La traduction coiffe-dépendante, sous le contrôle de la voie signalétique TOR (target of rapamycin), est essentielle pour cette réponse homéostatique rétrograde. De plus, la protéine reliée à la maladie de Parkinson, LRRK2 (leucine rich repeat kinase 2), ainsi que son orthologue chez la drosophile, dLRRK, peuvent réguler la fonction synaptique de la JNM via la traduction coiffe-dépendante. Cependant, les formes mutantes de LRRK2 sont défectueuses dans leur habileté à réguler la fonction synaptique. Globalement, ces trouvailles nous aident à comprendre les mécanismes moléculaires régulant la fonction et la plasticité synaptiques glutamatergiques.
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El-Kabir, Desiree Ruth. "An assessment of ventilatory function and sleep in neuromuscular disease." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.440537.

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Benatar, Michael G. "Presynaptic function in development and disease at the neuromuscular junction." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.388959.

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Dhesi, Jugdeep. "Vitamin D and neuromuscular function in older people who fall." Thesis, King's College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416021.

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Voegtle, Tracy. "The effect of an acute ankle joint injury on neuromuscular function." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ58098.pdf.

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Blunk, Aline D. (Aline Dorret). "Regulation of synaptic structure and function at the Drosophila neuromuscular junction." Thesis, Massachusetts Institute of Technology, 2013. http://hdl.handle.net/1721.1/84874.

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Thesis (Ph. D. in Neuroscience)--Massachusetts Institute of Technology, Department of Brain and Cognitive Sciences, 2013.
Cataloged from PDF version of thesis.
Includes bibliographical references.
Neuronal communication requires a spatially organized synaptic apparatus to coordinate neurotransmitter release from synaptic vesicles and activation of postsynaptic receptors. Structural remodeling of synaptic connections can strengthen neuronal communication and synaptic efficacy during development and behavioral plasticity. Here, I describe experimental approaches that have revealed how the actin cytoskeleton participates in transynaptic signaling to control synapse assembly. I also describe my studies on how regulation of endocytic trafficking controls synaptic growth during neuronal development. To identify regulators of synapse assembly, I carried out a large-scale EMS mutagenesis screen of the second chromosome. From this screen I identified a mutation in actin 57B that disrupts synaptic morphology and presynaptic active zone organization. Actin 57B is one of six actin genes in Drosophila and is expressed in body wall muscle during larval development. The isolated allele harbors a point mutation disrupting a highly conserved amino acid present throughout the actin family. Homozygous mutant larvae show impaired alignment and spacing of presynaptic active zones. Additionally, disruption of the organization of the postsynaptic density is observed, with mislocalization of the Spectrin cytoskeleton and the PSD-homolog Disc-Large. Phallodin staining reveals a severe disruption of postsynaptic actin surrounding presynaptic boutons, with the formation of aberrant large actin swirls. Based on these results, we hypothesize that the loss of a synaptic interaction mediated by actin 57B leads to disruption of postsynaptic cytoskeletal organization and dysregulation of signals required to organize presynaptic active zones. Additionally, I present data that provide new insights into the mechanisms controlling synaptic growth signaling during transit through the endocytic pathway. Nervous Wreck (Nwk) is a presynaptic F-BAR/SH3 protein that regulates synaptic growth signaling in Drosophila. Here, I show that Nwk acts through a physical interaction with Sorting Nexin 16 (SNX16). SNX16 promotes synaptic growth signaling by activated BMP receptors, and live imaging in neurons reveals that SNX16-positive early endosomes undergo transient interactions with Nwkcontaining recycling endosomes. We identify an alternative signal termination pathway in the absence of Snx16 that is controlled by ESCRT-mediated internalization of receptors into the endosomal lumen. Our results define a presynaptic trafficking pathway mediated by SNX116, NWK and the ESCRT complex that functions to control synaptic growth signaling at the interface between endosomal compartments. Together, these experiments have expanded our understanding of the molecular mechanisms that control synaptic growth and assembly, highlighting the role of the postsynaptic actin cytoskeleton and the presynaptic endosomal trafficking pathway as key regulators.
by Aline D. Blunk.
Ph.D.in Neuroscience
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Books on the topic "Neuromuscular function"

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Pharmacology of neuromuscular function. 2nd ed. London: Wright, 1990.

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Atherton, Philip J., and Daniel J. Wilkinson, eds. Neuromuscular Assessments of Form and Function. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3315-1.

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Vivian, Budnik, and Ruiz-Cãnada Catalina, eds. The Fly neuromuscular junction: Structure and function. 2nd ed. San Diego, Calif: Elsevier/Academic Press, 2006.

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Budnik, Vivian. The fly neuromuscular junction: Structure and function. 2nd ed. San Diego, Calif: Elsevier/Academic Press, 2006.

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Skeletal muscle: Form and function. Champaign, IL: Human Kinetics, 1996.

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Andrea, Melandri Bruno, Milazzo Giulio, Blank Martin 1933-, North Atlantic Treaty Organization. Scientific Affairs Division., and International School of Biophysics (20th : 1991 : Erice, Italy), eds. Bioelectrochemistry IV: Nerve muscle function : bioelectrochemistry, mechanisms, bioenergetics, and control. New York: Plenum Press, 1994.

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Raoul, Tubiana, ed. Restoration of function in upper limb paralyses and muscular defects. New York, NY: Informa Healthcare, 2008.

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Hans-Werner, Bothe, Samii Madjid, Eckmiller Rolf 1942-, and International Workshop on Neurobionics (1st : 1992 : Goslar, Germany), eds. Neurobionics: An interdisciplinary approach to substitute impaired functions of the human nervous system. Amsterdam: North-Holland, 1993.

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R, Dimitrijevic Milan, and Eccles, John C. Sir, 1903-, eds. Upper motor neuron functions and dysfunctions. Basel: Karger, 1985.

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Investigations, United States Congress House Committee on Veterans' Affairs Subcommittee on Oversight and. Applications of functional muscular stimulation: Hearing before the Subcommittee on Oversight and Investigations of the Committee on Veterans' Affairs, House of Representatives, Ninety-ninth Congress, second session, February 19, 1986. Washington: U.S. G.P.O., 1986.

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Book chapters on the topic "Neuromuscular function"

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Green, J. Hilary. "Neuromuscular function." In The Autonomic Nervous System and Exercise, 56–71. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-2919-8_3.

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Mellies, Uwe, and Christian Dohna-Schwake. "Neuromuscular Disorders." In Paediatric Pulmonary Function Testing, 233–46. Basel: KARGER, 2005. http://dx.doi.org/10.1159/000083621.

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Bassotti, Gabrio, Carolina Pellegrini, and Nunzia Bernardini. "Neuromuscular Function Abnormalities." In Colonic Diverticular Disease, 31–39. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-93761-4_4.

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Savastano, Fabio. "The Ideal Function Is Linked to Ideal Swallow." In Neuromuscular Orthodontics, 129–54. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-41295-0_8.

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Marín, Pedro J. "Modulation of Neuromuscular Function." In Manual of Vibration Exercise and Vibration Therapy, 203–11. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43985-9_14.

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Woerlee, G. M. "Disorders Affecting Neuromuscular Function." In Common Perioperative Problems and the Anaesthetist, 334–39. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1323-3_60.

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Roberts, Michael D., and Jason M. Defreitas. "Measures of Neuromuscular Function." In Neuromethods, 85–103. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3315-1_5.

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Piasecki, Mathew, and Daniel W. Stashuk. "Neuromuscular Function: Intramuscular Electromyography." In Neuromethods, 125–35. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3315-1_7.

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Agrafiotis, Michalis, Stavros Tryfon, Afroditi Boutou, and Athanasia Pataka. "Chronic Neuromuscular Disorders." In Pulmonary Function Measurement in Noninvasive Ventilatory Support, 95–103. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-76197-4_13.

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Chémali, Kamal R., and Thomas C. Chelimsky. "Testing of Autonomic Function." In Neuromuscular Disorders in Clinical Practice, 201–21. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6567-6_10.

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Conference papers on the topic "Neuromuscular function"

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Jones, R. S., D. L. Preece, J. L. Murfin, and A. W. Preece. "On-line analysis of neuromuscular function." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.94759.

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Andres, R. O. "Balance biomechanics as a probe for neuromuscular function." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.94825.

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Andrade Costa, B., and J. M. Lemos. "Neuromuscular blockade model identification based on the Hill function inversion." In Automation (MED 2010). IEEE, 2010. http://dx.doi.org/10.1109/med.2010.5547866.

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Zhao, Weifeng, Ronald J. Triolo, Michael Wibowo, and Niloy Bhadra. "Surgical Simulation of Tendon Transfers to Augment Lower Extremity Function With Functional Neuromuscular Stimulation." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0153.

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Abstract A musculoskeletal model was used to study tendon transfer surgery in order to decouple hip extension and knee flexion in lower extremity FNS applications. Effects of tendon transfer location and shortening length were quantitatively examined based on the resulting moment-generating capacity. Simulation results have shown that moment arm is not sensitive to the transfer locations. Appropriate shortening is the key to counteract the tendon slack caused by the transplantation and to achieve maximal hip extension strength.
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Layne, Charles S. "Using foot pressure to maintain neuromuscular function during long-duration spaceflight." In HADRONS AND NUCLEI: First International Symposium. AIP, 2000. http://dx.doi.org/10.1063/1.1302486.

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Wang, Ping, Yuanheng Li, Zijun Zhang, Yifeng Lin, Zhendong Jiang, Xiaofang Ding, and Lin Yang. "Effects of functional electrical stimulation on neuromuscular function after targeted muscle reinnervation surgery in rats*." In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9175836.

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Luo, J., Y. Qiao, X. Wang, L. Su, J. Guo, R. Huang, and Y. Xiao. "Pulmonary Function and Polysomnography Characteristics in 3 Cases of Neuromuscular Disease with Hypoventilation." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7615.

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Ueda, Jun, Moiz Hyderabadwala, Vijaya Krishnamoorthy, and Minoru Shinohara. "Motor task planning for neuromuscular function tests using an individual muscle control technique." In the Community (ICORR). IEEE, 2009. http://dx.doi.org/10.1109/icorr.2009.5209522.

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Emmanuel, Babatunde S. "A Study of the Effectiveness of Monophasic Electrical Stimulation in Enhancing Neuromuscular Tissue Function." In 2022 5th Information Technology for Education and Development (ITED). IEEE, 2022. http://dx.doi.org/10.1109/ited56637.2022.10051226.

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Williams, Glenn N., Peter J. Barrance, Lynn Snyder-Mackler, and Thomas S. Buchanan. "Effect of Anterior Cruciate Ligament Reconstruction With an Autologous Semitendinosus-Gracilis Graft on Neuromuscular Function." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43031.

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The quadrupled semitendinosus-gracilis (ST-G) graft is rapidly becoming the graft of choice for orthopaedic surgeons when reconstructing the anterior cruciate ligament (ACL). During this procedure orthopaedic surgeons harvest the distal semitendinosus and gracilis tendons and use them to replace the ruptured ACL. Although people who undergo this procedure have good functional outcomes over the short-term, we do not know the effect that harvesting these two tendons has on neuromuscular function. The purpose of this study was to examine the effect that ACL reconstruction with an autologous ST-G had on musculotendinous morphology. The methods used in the study included digital reconstruction of knee musculature from magnetic resonance images (MRI). Marked reductions in muscle volume, cross-sectional area, and length were observed in the semitendinosus and gracilis when reassessed approximately 6 months following surgery (after the subjects had returned to sports participation). The subjects appeared to compensate for the diminished medial knee flexor function with the biceps femoris (a lateral muscle) and semimembranosus muscles. These findings may have important implications for joint loading, the long term health of the knee, and surgical decision-making.
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Reports on the topic "Neuromuscular function"

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Gurevitz, Michael, Michael Adams, and Eliahu Zlotkin. Insect Specific Alpha Neurotoxins from Scorpion Venoms: Mode of Action and Structure-Function Relationships. United States Department of Agriculture, June 1996. http://dx.doi.org/10.32747/1996.7613029.bard.

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This study was motivated by the need to develop new means and approaches to the design of future, environmentally-safe, insecticides. Utilization of anti-insect selective toxins from scorpion venoms and clarification of the molecular basis for their specificity, are a major focus in this project and may have an applicative value. Our study concentrated on the highly insecticidal toxin, LqhaIT, and was devoted to: (I) Characterization of the neuropharmacological and electrophysiological features of this toxin. (II) Establishment of a genetic system for studying structure/activity relationships of the toxin. (III) Analysis of the insecticidal efficacy of an entomopathogenic baculovirus engineered and expressing LqhaIT. The results obtained in this project suggest that: 1) The receptor binding site of LqhaIT on insect sodium channels differs most likely from its analogous receptor site 3 on vertebrate sodium channels. 2) The effects of LqhaIT are presynaptic. Hyperexcitation at the neuromuscular results from dramatic slowing of sodium channel inactivation and enhanced peak sodium currents causes by LqhaIT. 3) The putative toxic surface of LqhaIT involves aromatic and charged amino acid residues located around the C-terminal region and five-residue-turn of the toxin (unpublished). 4) The anti-insect/anti-mammalian toxicity ratio can be altered by site-directed mutagenesis (publication 8). This effect was partly shown at the level of sodium channel function. 5) The insecticidal efficacy of AcNPV baculovirus increased to a great extent when infection was accompanied by expression of LqhaIT (publication 5).
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Jalil, Yorschua, and Ruvistay Gutierrez. Myokines secretion and their role in critically ill patients. A scoping review protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0048.

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Review question / Objective: 1-How and by which means stimulated muscle from critically ill patients can liberate myokines?, 2-Which are the main characteristics of the critically ill population studied and if some of these influenced myokine´s secretion?, 5-Can myokines exert local or distant effects in critically ill patients?, 5-Which are the potential effects of myokines in critically ill patients? Eligibility criteria: Participants and context: We will include primary studies (randomized or non-randomized trials, observational studies, case series or case report) that consider hospitalized critically ill adult patients (18 years or older) in risk for developing some degree of neuromuscular disorders such as ICU-AW, diaphragmatic dysfunction, or muscle weakness, therefore the specific setting will be critical care. Concept: This review will be focused on studies regarding the secretion or measure of myokines or similar (exerkines, cytokines or interleukin) by any mean of muscle activation or muscle contraction such as physical activity, exercise or NMES, among others. The latter strategies must be understood as any mean by which muscle, and there for myocytes, are stimulated as result of muscle contraction, regardless of the frequency, intensity, time of application and muscle to be stimulated (upper limb, lower limb, thoracic or abdominal muscles). We also will consider myokine´s effects, local or systemic, over different tissues in terms of their structure or function, such as myocytes function, skeletal muscle mass and strength, degree of muscle wasting or myopathies, among others.
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Gular, Kumar, S. Viwanathan, Khalid Alahmari, and Jaya Shanker Tedla. Neuromuscular interventions in improving upper extremity functions in post-stroke subjects - A systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0070.

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Corscadden, Louise, and Anjali Singh. Grip Strength Test In Rodents. ConductScience, January 2023. http://dx.doi.org/10.55157/cs2023109.

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Abstract:
The grip strength test is one of the most commonly applied tests in animal laboratories to measure neuromuscular functions or disorders. It was first developed in the 1970s. Today a wide range of techniques are available to study muscle strength in rodents. These methods are categorized into two categories:[2] Invasive method: In situ and in vitro measurements of muscle force are invasive methods. Non-invasive method: This method only includes in vivo measurement tests to analyze muscle force such as treadmill tests, wire hang tests, swimming endurance, vertical pole test, and grip strength tests. The most convenient technique of all tests is the grip strength test. It’s most convenient and causes less stress to animals. The grip test has been widely used in order to investigate the phenotypes of transgenic mice with neuromuscular disease and evaluate potential compounds involved in the motor functioning of organisms. The tests have been serving the purpose for 30 years either alone or in combination with other tests.
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