Journal articles: 'Neuromuscular fatigue'

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Sharp, C. "Neuromuscular Fatigue." British Journal of Sports Medicine 28, no. 3 (September 1, 1994): 208–9. http://dx.doi.org/10.1136/bjsm.28.3.208-a.

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Williams, Jay H. "Neuromuscular Fatigue." Medicine & Science in Sports & Exercise 26, no. 7 (July 1994): 934. http://dx.doi.org/10.1249/00005768-199407000-00021.

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van Dijk, J. G. "Neuromuscular fatigue." Journal of the Neurological Sciences 120, no. 1 (December 1993): 121. http://dx.doi.org/10.1016/0022-510x(93)90038-z.

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Deffieux, X., K. Hubeaux, E. Lapeyre, M. Jousse, S. Sheikh Ismael, P. Thoumie, and G. Amarenco. "Perineal neuromuscular fatigue." Annales de Réadaptation et de Médecine Physique 49, no. 6 (July 2006): 413–17. http://dx.doi.org/10.1016/j.annrmp.2006.03.010.

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Collins, Brandon W., Gregory E. P. Pearcey, Natasha C. M. Buckle, Kevin E. Power, and Duane C. Button. "Neuromuscular fatigue during repeated sprint exercise: underlying physiology and methodological considerations." Applied Physiology, Nutrition, and Metabolism 43, no. 11 (November 2018): 1166–75. http://dx.doi.org/10.1139/apnm-2018-0080.

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Neuromuscular fatigue occurs when an individual’s capacity to produce force or power is impaired. Repeated sprint exercise requires an individual to physically exert themselves at near-maximal to maximal capacity for multiple short-duration bouts, is extremely taxing on the neuromuscular system, and consequently leads to the rapid development of neuromuscular fatigue. During repeated sprint exercise the development of neuromuscular fatigue is underlined by a combination of central and peripheral fatigue. However, there are a number of methodological considerations that complicate the quantification of the development of neuromuscular fatigue. The main goal of this review is to synthesize the results from recent investigations on the development of neuromuscular fatigue during repeated sprint exercise. Hence, we summarize the overall development of neuromuscular fatigue, explain how recovery time may alter the development of neuromuscular fatigue, outline the contributions of peripheral and central fatigue to neuromuscular fatigue, and provide some methodological considerations for quantifying neuromuscular fatigue during repeated sprint exercise.

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Féasson, L., J. P. Camdessanché, L. El Mhandi, P. Calmels, and G. Y. Millet. "Fatigue and neuromuscular diseases." Annales de Réadaptation et de Médecine Physique 49, no. 6 (July 2006): 375–84. http://dx.doi.org/10.1016/j.annrmp.2006.04.016.

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Piponnier, Enzo, Vincent Martin, Bastien Bontemps, Emeric Chalchat, Valérie Julian, Olivia Bocock, Martine Duclos, and Sébastien Ratel. "Child-adult differences in neuromuscular fatigue are muscle dependent." Journal of Applied Physiology 125, no. 4 (October 1, 2018): 1246–56. http://dx.doi.org/10.1152/japplphysiol.00244.2018.

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The aim of the present study was to compare the development and etiology of neuromuscular fatigue of the knee extensor (KE) and plantar flexor (PF) muscles during repeated maximal voluntary isometric contractions (MVICs) between children and adults. Prepubertal boys ( n = 21; 9–11 yr) and men ( n = 24; 18–30 yr) performed two fatigue protocols consisting of a repetition of 5-s isometric MVIC of the KE or PF muscles interspersed with 5-s passive recovery periods until MVIC reached 60% of its initial value. The etiology of neuromuscular fatigue of the KE and PF muscles was investigated by means of noninvasive methods, such as the surface electromyography, single and doublet magnetic stimulation, twitch interpolation technique, and near-infrared spectroscopy. The number of repetitions performed was significantly lower in men (15.4 ± 3.8) than boys (38.7 ± 18.8) for the KE fatigue test. In contrast, no significant difference was found for the PF muscles between boys and men (12.1 ± 4.9 and 13.8 ± 4.9 repetitions, respectively). Boys displayed a lower reduction in potentiated twitch torque, low-frequency fatigue, and muscle oxygenation than men whatever the muscle group considered. In contrast, voluntary activation level and normalized electromyography data decreased to a greater extent in boys than men for both muscle groups. To conclude, boys experienced less peripheral and more central fatigue during repeated MVICs than men whatever the muscle group considered. However, child-adult differences in neuromuscular fatigue were muscle-dependent since boys fatigued similarly to men with the PF muscles and to a lower extent with the KE muscles. NEW & NOTEWORTHY Child-adult differences in neuromuscular fatigue during repeated maximal voluntary contractions are specific to the muscle group since children fatigue similarly to adults with the plantar flexor muscles and to a lower extent with the knee extensor muscles. Children experience less peripheral fatigue and more central fatigue than adults, regardless of the muscle group considered.

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Millet, Guillaume Y., Vincent Martin, Nicola A. Maffiuletti, and Alain Martin. "Neuromuscular Fatigue After a Ski Skating Marathon." Canadian Journal of Applied Physiology 28, no. 3 (June 1, 2003): 434–45. http://dx.doi.org/10.1139/h03-032.

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The aim of this study was to characterize neuromuscular fatigue in knee extensor muscles after a marathon skiing race (mean ± SD duration = 159.7 ± 17.9 min). During the 2 days preceding the event and immediately after, maximal percutaneous electrical stimulations (single twitch, 0.5-s tetanus at 20 and 80 Hz) were applied to the femoral nerve of 11 trained skiers. Superimposed twitches were also delivered during maximal voluntary contraction (MVC) to determine maximal voluntary activation (%VA). EMG was recorded from the vastus lateralis muscle. MVC decreased with fatigue from 171.7 ± 33.7 to 157.3 ± 35.2 Nm (-8.4%; p < 0.005) while %VA did not change significantly. The RMS measured during MVC and peak-to-peak amplitude of the compound muscle action potential (PPA) from the vastus lateralis decreased with fatigue by about 30% (p < 0.01), but RMS•PPA−1was similar before and after the ski marathon. Peak tetanus tension at 20 Hz and 80 Hz (P020 and P080, respectively) did not change significantly, but P020•P080−1 increased (p < 0.05) after the ski marathon. Data from electrically evoked single twitches showed greater peak mechanical response, faster rate of force development, and shorter contraction time in the fatigued state. From these results it can be concluded that a ski skating marathon (a) alters slightly but significantly maximal voluntary strength of the knee extensors without affecting central activation, and (b) induces both potentiation and fatigue. Key words: low- and high-frequency electrical stimulation, central activation, potentiation

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Izquierdo, M., J. Ibañez, J. Calbet, M. González-Izal, I. Navarro-Amézqueta, C. Granados, A. Malanda, et al. "Neuromuscular Fatigue after Resistance Training." International Journal of Sports Medicine 30, no. 08 (April 20, 2009): 614–23. http://dx.doi.org/10.1055/s-0029-1214379.

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Lou, Jau-Shin. "Approaching Fatigue in Neuromuscular Diseases." Physical Medicine and Rehabilitation Clinics of North America 16, no. 4 (November 2005): 1063–79. http://dx.doi.org/10.1016/j.pmr.2005.08.012.

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Girard, Olivier, and Grégoire P. Millet. "Neuromuscular Fatigue in Racquet Sports." Physical Medicine and Rehabilitation Clinics of North America 20, no. 1 (February 2009): 161–73. http://dx.doi.org/10.1016/j.pmr.2008.10.008.

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Girard, Olivier, and Grégoire P. Millet. "Neuromuscular Fatigue in Racquet Sports." Neurologic Clinics 26, no. 1 (February 2008): 181–94. http://dx.doi.org/10.1016/j.ncl.2007.11.011.

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Wadden, Katie P., Duane C. Button, Armin Kibele, and David G. Behm. "Neuromuscular fatigue recovery following rapid and slow stretch–shortening cycle movements." Applied Physiology, Nutrition, and Metabolism 37, no. 3 (June 2012): 437–47. http://dx.doi.org/10.1139/h2012-020.

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The purpose of this study was to investigate underlying mechanisms and neuromuscular recovery patterns following rapid and slow stretch–shortening cycle (SSC) movements performed to fatigue. Fourteen (10 moderately trained (MT) and four highly trained (HT)) subjects completed rapid and slow SSC movements to fatigue. The rapid SSC movement consisted of continuous drop jumps from a 30 cm platform until a predetermined jump height was no longer maintained, and the slow SSC movement consisted of continuous squats to 90° of knee flexion at a load of 65% of subject’s one-repetition maximum until no further repetitions could be completed. Although blood lactate measures were significantly (p < 0.002) higher after the rapid SSC condition versus after the slow SSC condition, the recovery of neuromuscular properties (maximum voluntary contractions, twitch force, muscle compound action potential) following the two conditions to fatigue did not differ. The duration of the rapid SSC movement was dependent on the training status of the subject; HT subjects performed the rapid SSC longer (68.2%) than the MT subjects until fatigued. Thus, the neuromuscular fatigue recovery patterns were independent of the type of SSC movement, condition duration, and subject training status. Because rapid and slow SSC exercises induce similar fatigue patterns, training programs incorporating rapid SSC exercises can be developed similar to that prescribed in traditional slow SSC resistance training programs.

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Cunningham, David A. "Noninvasive brain stimulation enhances sustained muscle contractions by reducing neuromuscular fatigue: implications for rehabilitation." Journal of Neurophysiology 117, no. 3 (March 1, 2017): 1215–17. http://dx.doi.org/10.1152/jn.00468.2016.

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Neuromuscular fatigue is due, in part, to central processes that involve failure of the nervous system to drive muscles maximally during exercise. A recent study by Abdelmoula, Baudry, and Duchateau ( Neuroscience 322: 94–103, 2016) showed that noninvasive brain stimulation can mitigate neuromuscular fatigue, however, does not rely on enhanced corticospinal excitability of the primary motor cortex. These findings are of high clinical importance because rehabilitative therapies are necessary to mitigate neuromuscular fatigue for patients with central nervous system disorders.

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Mäestu, Jarek, Antonio Cicchella, Priit Purge, Sergio Ruosi, Jaak Jürimäe, and Toivo Jürimäe. "Electromyographic and Neuromuscular Fatigue Thresholds as Concepts of Fatigue." Journal of Strength and Conditioning Research 20, no. 4 (2006): 824. http://dx.doi.org/10.1519/r-18275.1.

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MÄESTU, JAREK, ANTONIO CICCHELLA, PRIIT PURGE, SERGIO RUOSI, JAAK JÜRIMÄE, and TOIVO JÜRIMÄE. "ELECTROMYOGRAPHIC AND NEUROMUSCULAR FATIGUE THRESHOLDS AS CONCEPTS OF FATIGUE." Journal of Strength and Conditioning Research 20, no. 4 (November 2006): 824–28. http://dx.doi.org/10.1519/00124278-200611000-00016.

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Gorham-Rowan, M., and R. Morris. "Exploring the effect of laryngeal neuromuscular electrical stimulation on voice." Journal of Laryngology & Otology 130, no. 11 (November 2016): 1022–32. http://dx.doi.org/10.1017/s0022215116009038.

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AbstractObjective:This study was conducted to explore the potential use of neuromuscular electrical stimulation as an adjunctive treatment for muscle tension dysphonia.Methods:Voice data and ratings of fatigue and soreness were obtained for two experiments. Experiment one examined the vocal effects of neuromuscular electrical stimulation applied to the neck for 15 minutes. Experiment two examined the recovery effect of laryngeal neuromuscular electrical stimulation following a vocal loading task among normophonic women.Results:No significant differences in vocal function following 15 minutes of laryngeal neuromuscular electrical stimulation were found. Six of 11 participants receiving laryngeal neuromuscular electrical stimulation exhibited improved recovery following the vocal loading task.Conclusion:A short session of laryngeal neuromuscular electrical stimulation may be beneficial in reducing muscle fatigue for some individuals. Further investigation is warranted to determine the applicability of laryngeal neuromuscular electrical stimulation in voice therapy.

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Allen, Matti D., Kurt Kimpinski, Timothy J. Doherty, and Charles L. Rice. "Decreased muscle endurance associated with diabetic neuropathy may be attributed partially to neuromuscular transmission failure." Journal of Applied Physiology 118, no. 8 (April 15, 2015): 1014–22. http://dx.doi.org/10.1152/japplphysiol.00441.2014.

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Diabetic polyneuropathy (DPN) can cause muscle atrophy, weakness, contractile slowing, and neuromuscular transmission instability. Our objective was to assess the response of the impaired neuromuscular system of DPN in humans when stressed with a sustained maximal voluntary contraction (MVC). Baseline MVC and evoked dorsiflexor contractile properties were assessed in DPN patients ( n = 10) and controls ( n = 10). Surface electromyography was used to record tibialis anterior evoked maximal compound muscle action potentials (CMAPs) and neuromuscular activity during MVCs. Participants performed a sustained isometric dorsiflexion MVC for which task termination was determined by the inability to sustain ≥60% MVC torque. The fatigue protocol was immediately followed by a maximal twitch, with additional maximal twitches and MVCs assessed at 30 s and 2 min postfatigue. DPN patients fatigued ∼21% more quickly than controls ( P < 0.05) and featured less relative electromyographic activity during the first one-third of the fatigue protocol compared with controls ( P < 0.05). Immediately following fatigue, maximal twitch torque was reduced similarly (∼20%) in both groups, and concurrently CMAPs were reduced (∼12%) in DPN patients, whereas they were unaffected in controls ( P > 0.05). Twitch torque and CMAP amplitude recovered to baseline 30 s postfatigue. Additionally, at 30 s postfatigue, both groups had similar (∼10%) reductions in MVC torque relative to baseline, and MVC strength recovered by 2 min postfatigue. We conclude DPN patients possess less endurance than controls, and neuromuscular transmission failure may contribute to this greater fatigability.

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Rice, C. L., N. K. Vollestad, and B. Bigland-Ritchie. "DISSOCIATION OF FATIGUE-RELATED NEUROMUSCULAR EVENTS." Medicine & Science in Sports & Exercise 24, Supplement (May 1992): S56. http://dx.doi.org/10.1249/00005768-199205001-00333.

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Garrandes, F., S. S. Colson, M. Pensini, and P. Legros. "Neuromuscular fatigue kinetics are sport specific." Computer Methods in Biomechanics and Biomedical Engineering 8, sup1 (September 2005): 113–14. http://dx.doi.org/10.1080/10255840512331388515.

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Ross, Emma Z., Warren Gregson, Karen Williams, and Keith George. "Neuromuscular Fatigue Following Repetitive Endurance Cycling." Medicine & Science in Sports & Exercise 41 (May 2009): 195. http://dx.doi.org/10.1249/01.mss.0000355152.72245.20.

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Shortz, Ashley E., and Ranjana K. Mehta. "Cognitive challenges, aging, and neuromuscular fatigue." Physiology & Behavior 170 (March 2017): 19–26. http://dx.doi.org/10.1016/j.physbeh.2016.11.034.

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&NA;. "A-27 Thematic Poster - Neuromuscular Fatigue." Medicine & Science in Sports & Exercise 46 (May 2014): 5–8. http://dx.doi.org/10.1249/01.mss.0000451094.33228.0c.

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Weavil, Joshua C., and Markus Amann. "Neuromuscular fatigue during whole body exercise." Current Opinion in Physiology 10 (August 2019): 128–36. http://dx.doi.org/10.1016/j.cophys.2019.05.008.

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Aldrich, T. K., A. Shander, I. Chaudhry, and H. Nagashima. "Fatigue of isolated rat diaphragm: role of impaired neuromuscular transmission." Journal of Applied Physiology 61, no. 3 (September 1, 1986): 1077–83. http://dx.doi.org/10.1152/jappl.1986.61.3.1077.

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We compared the contributions of impaired neuromuscular transmission (transmission fatigue) and impaired muscle contractility (contractile fatigue) to fatigue of the isolated rat diaphragm. To make this comparison, we measured the differences in active tension elicited by direct muscle stimulation and by indirect (phrenic nerve) stimulation before and after fatigue induced by indirect supramaximal stimulation at varying frequencies and durations. Transmission fatigue was observed after all experimental protocols. Although significant contractile fatigue was not demonstrated after brief periods of low-frequency stimulation (6 min, 15 Hz, 25% duty cycle), it was present after longer or higher frequency stimulation. We repeated the direct stimulation in the presence of neuromuscular blockade with 6 microM d-tubocurarine to demonstrate that a reduced response to stimulation of intramuscular branches of the phrenic nerve during direct stimulation was not responsible for the apparent contractile fatigue. Since we found significant decreases in the response to direct stimulation even after neuromuscular blockade, we could verify the presence of contractile fatigue. We conclude that both contractile and transmission fatigue can occur in the isolated rat diaphragm and that transmission fatigue is a much more important factor after brief periods of fatiguing contractions.

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Miras Moreno, Sergio. "La altura del salto en contramovimiento como instrumento de control de la fatiga neuromuscular. Revisión sistemática (Counter-movement Jump height as a means to monitor neuromuscular fatigue. Systematic Review)." Retos, no. 37 (October 8, 2019): 820–26. http://dx.doi.org/10.47197/retos.v37i37.73302.

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Las múltiples ventajas que aporta un buen control de la carga y la fatiga tanto en deportes de equipo como individuales son conocidas, aunque es importante determinar si un mismo método de control puede ser utilizado para cualquier tipo de deporte, ya que cada uno posee unas demandas metabólicas y neuromusculares diferentes. El objetivo de esta revisión fue recopilar conocimiento acerca de la fiabilidad de la altura del salto en contramovimiento (CMJ, por sus siglas en inglés) como instrumento de control de la carga y la fatiga neuromuscular en atletas de diferentes deportes. El objetivo complementario fue relacionar el CMJ con otras variables metabólicas sensibles a la fatiga neuromuscular. Dos bases de datos (PubMed y WebOfScience) fueron utilizadas para la búsqueda de un total de 1051 artículos científicos. La calidad metodológica para cada artículo se llevó a cabo mediante la escala de la base de datos sobre Fisioterapia Basada en la Evidencia (PEDro). Un total de 12 artículos siguieron los criterios de inclusión y exclusión. Se encontraron numerosas correlaciones con otras variables metabólicas como el nivel de cortisol en saliva, el amonio y lactato en sangre. La altura del CMJ es una herramienta fiable para medir tanto como la fatiga neuromuscular a lo largo de una temporada como para medir la fatiga aguda después de una sesión de entrenamiento, no obstante, es recomendable establecer un protocolo adecuado de medición y que se relacione con otros instrumentos o parámetros metabólicos para aumentar su fiabilidad dentro de los distintos deportes.Abstract: The many advantages of good load and fatigue control in team and individual sports are known, although it is important to determine if the same control method can be used for any type of sport, for each one has different metabolic and neuromuscular demands. To analyze the reliability of counter-movement jump height (CMJ) as an instrument for load and neuromuscular fatigue control in athletes of different sports. The secondary aim was to relate vertical jump height (VJH) with other metabolic variables sensitive to neuromuscular fatigue. Two databases (PubMed and Web Of Science) were used to search a total of 1051 scientific journals. The methodological quality for each article was carried out using the scale of the database on Evidence-Based Physiotherapy (PEDro). A total of 12 papers matched the inclusion criteria. Numerous correlations were found with other metabolic variables such as the level of cortisol in saliva, ammonium and lactate in the blood. The height of the CMJ is a reliable tool to measure neuromuscular fatigue over a season and acute fatigue after a training session; however, it is advisable to establish an adequate measurement protocol and compare it with other instruments to increase their reliability within different sports.

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Alba-Jiménez, Claudia, Daniel Moreno-Doutres, and Javier Peña. "Trends Assessing Neuromuscular Fatigue in Team Sports: A Narrative Review." Sports 10, no. 3 (February 28, 2022): 33. http://dx.doi.org/10.3390/sports10030033.

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Neuromuscular fatigue is defined as a reduction induced by exercise in the maximal voluntary force that a muscle or group of muscles can generate. An accumulation of work or an incomplete force restoration can significantly influence the neuromuscular performance in both the short and long terms. Thus, fatigue management is essential for controlling the training adaptations of athletes and reducing their susceptibility to injury and illness. The main individualized monitoring tools used to describe fatigue are questionnaires and subjective assessments of fatigue, biochemical markers, sprint tests, and vertical jump tests. Among the subjective measures, the rating of the perceived exertion has been widely used because of its simplicity and high validity. In terms of the objective measures, one of the more frequently employed tools by practitioners to assess neuromuscular fatigue is the countermovement jump. Because of its high validity and reliability, it is accepted as the reference standard test in sports, in general, and particularly in team sports. Our review aims to clarify how all these indicators, as well as several devices, can help coaches in different sports contexts to monitor neuromuscular fatigue, and how these procedures should be used to obtain data that can be used to make decisions in complex environments.

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Morris, Amanda J., and Anita D. Christie. "The Effect of Mental Fatigue on Neuromuscular Function is Similar in Young and Older Women." Brain Sciences 10, no. 4 (March 25, 2020): 191. http://dx.doi.org/10.3390/brainsci10040191.

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The purpose of this study was to examine the effect of a mentally fatiguing task on neuromuscular function in young and older women. Neuromuscular measures were obtained prior to and following 20 min of a mentally fatiguing task. Maximal force output significantly decreased after the mental fatigue task (p = 0.02) and this was not different between age groups (p = 0.32). Increases in cortical silent period duration approached significance in both young and older groups (p = 0.06), suggesting that mental fatigue may cause increased cortical inhibition. Measures of peripheral neuromuscular function (contractile properties of the muscle, M-wave) did not change (p ≥ 0.09), suggesting that changes in force production with mental fatigue are more likely due to supraspinal than peripheral mechanisms. These findings provide further evidence of an interaction between mental fatigue and physical function.

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Hogarth, Luke W., Brendan J. Burkett, and Mark R. McKean. "Neuromuscular and Perceptual Fatigue Responses to Consecutive Tag Football Matches." International Journal of Sports Physiology and Performance 10, no. 5 (July 2015): 559–65. http://dx.doi.org/10.1123/ijspp.2014-0355.

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Purpose:To examine the neuromuscular and perceptual fatigue responses to consecutive tag football matches played on the same day and determine the relationship between fatigue and match running performance.Methods:Neuromuscular and perceptual fatigue responses of 15 national tag football players were assessed before and during the 2014 State of Origin tournament. Global positioning systems (GPS) provided data on players’ match running performance, and a vertical-jump test and subjective questionnaire were used to assess players’ neuromuscular and perceptual fatigue, respectively.Results:There were small to moderate reductions in the majority of match-running-performance variables over consecutive matches, including distance (ES = −0.81), high-speed-running (HSR) distance (ES = −0.51), HSR efforts (ES = −0.64), and maximal accelerations (ES = −0.76). Prematch vertical jump was initially below baseline values before the first match (ES = 0.68−0.88). There were no substantial reductions in vertical-jump performance from baseline values over consecutive matches, although there was a small decline from after match 2 to after match 3 (3.3%; ES = −0.45 ± 0.62). There were progressive reductions in perceived well-being scores after matches 1 (ES = −0.38), 2 (ES = −0.70), and 3 (ES = −1.14). There were small to moderate associations between changes in fatigue measures and match running performance.Conclusions:Perceptual fatigue accumulates over consecutive tag football matches, although there were only marginal increases in neuromuscular fatigue. However, both neuromuscular and perceptual fatigue measures were found to contribute to reduced match running performance in the final match.

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Bilodeau, Martin, Tara K. Henderson, Brian E. Nolta, Paul J. Pursley, and Gretchen L. Sandfort. "Effect of aging on fatigue characteristics of elbow flexor muscles during sustained submaximal contraction." Journal of Applied Physiology 91, no. 6 (December 1, 2001): 2654–64. http://dx.doi.org/10.1152/jappl.2001.91.6.2654.

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The purpose of this study was to compare fatigue-related measures of central and peripheral mechanisms between young and elderly subjects for a task performed with elbow flexor muscles. Ten young and nine elderly subjects performed a sustained submaximal fatigue task at 35% of their maximum voluntary contraction torque. Measures of neuromuscular function, reflecting changes in neuromuscular propagation, voluntary activation, excitation-contraction-relaxation processes, and metabolite buildup, were taken before, during, and after the fatigue task. The main results were the absence of neuromuscular propagation failure in either young or elderly subjects, the presence of central fatigue at the end of the fatigue task in 7 of 9 elderly but only 3 of 10 young subjects, and lesser changes in twitch torque contraction-relaxation variables and electromyographic median frequency in elderly compared with young subjects. The lesser fatigue-related changes in twitch contraction speed and median frequency in elderly compared with young subjects could reflect the increase in type I-to-type II fiber area reported with old age. The presence of significant central fatigue can apparently minimize some of the potential differences present in peripheral fatigue sites.

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Froyd, Christian, Fernando Beltrami, and Timothy Noakes. "Neuromuscular Fatigue at Task Failure and During Immediate Recovery after Isometric Knee Extension Trials." Sports 6, no. 4 (November 28, 2018): 156. http://dx.doi.org/10.3390/sports6040156.

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We asked whether the level of peripheral fatigue would differ when three consecutive exercise trials were completed to task failure, and whether there would be delayed recovery in maximal voluntary contraction (MVC) force, neuromuscular activation and peripheral fatigue following task failure. Ten trained sport students performed three consecutive knee extension isometric trials (T1, T2, T3) to task failure without breaks between trials. T1 and T2 consisted of repeated 5-s contractions followed by 5-s rests. In T1, contractions were performed at a target force at 60% pre-exercise MVC. In T2, all contractions were MVCs, and task failure occurred at 50% MVC. T3 was a sustained MVC performed until force fell below 15% MVC. Evoked force responses to supramaximal electrical femoral nerve stimulation were recorded to assess peripheral fatigue. Electromyography signals were normalized to an M-wave amplitude to assess neuromuscular activation. Lower levels of evoked peak forces were observed at T3 compared with T2 and T1. Within 5 s of task failure in T3, MVC force and neuromuscular activation recovered substantially without any recovery in evoked peak force. Neuromuscular activation 5–10 s after T3 was unchanged from pre-exercise values, however, evoked peak forces were substantially reduced. These results challenge the existence of a critical peripheral fatigue threshold that reduces neuromuscular activation. Since neuromuscular activation changed independently of any change in evoked peak force, immediate recovery in force production after exercise is due to increased central recruitment and not to peripheral mechanisms.

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El-Aloul, B., Y. Wei, K. Speechley, and C. Campbell. "P.073 Factors associated with fatigue in children and adolescents with Duchenne muscular dystrophy: A Canada-wide cross-sectional survey." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 44, S2 (June 2017): S31—S32. http://dx.doi.org/10.1017/cjn.2017.157.

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Background: Fatigue is frequent and disabling in adults with neuromuscular disorders, but not well characterized in paediatric neuromuscular disorders. Recently, fatigue was reported to be associated with poor health-related quality of life in children with Duchenne muscular dystrophy (DMD). Determinants of fatigue—a modifiable symptom—have not been studied in DMD. Our objective was to explore risk factors for fatigue in children with DMD. Methods: Patients aged 4–17 years identified via the Canadian Neuromuscular Disease Registry received mailed questionnaires. Fatigue was assessed using the PedsQLTM Multidimensional Fatigue Scale (patient- and parent-report). Standardized measures for depressive symptoms, sleep disturbances, functional ability and physical activity were used. Spearman’s correlations and Wilcoxon rank-sum tests were computed. Results: Of 194 eligible patients, 64 have responded to date. DMD patients reported greater fatigue than healthy controls from published data. Depressive symptoms were associated with greater fatigue, by patient-report (ρ=-0.44, P<0.001) and parent-report (ρ=-0.40, P=0.002). Sleep disturbances were associated with greater fatigue, by patient-report (ρ=-0.41, P=0.007) and parent-report (ρ=-0.51, P<0.001). Greater functional ability was associated with less fatigue, by parent-report (ρ=0.30, P=0.02). Physical activity and ambulatory status were not associated with fatigue. Conclusions: Fatigue is a significant issue in DMD. Depressive symptoms and sleep disturbances are associated with fatigue, warranting attention in therapeutic strategies to reduce fatigue.

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Sadiye, Gumusyayla, Vural Gonul, Barakli Serdar, Yildiz Rezzan, Deniz Orhan, Hasanoglu Imran, and Guner Hatice Rahmet. "Evaluation of neuromuscular junction functions with single fiber electromyography in individuals with persistent fatigue after Coronavirus disease 2019." Journal of Neurology, Neurological Science and Disorders 10, no. 1 (February 29, 2024): 015–20. http://dx.doi.org/10.17352/jnnsd.000059.

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Purpose: Post COVID Syndrome (PCS) is one of the most intriguing topics related to coronavirus disease 2019 (COVID-19). Fatigue is one of the most prevalent and disabling symptoms of PCS. In this study, we aimed to investigate the neuromuscular junction functions in people who have had long-term fatigue due to COVID-19 and to investigate whether the neuromuscular junction is the cause of fatigue. Methods: 37 patients who had COVID-19 at least 12 weeks ago and continued to complain of fatigue and 37 healthy individuals were included in the study. The Fatigue Severity Scale (FSS) and Fatigue Impact Scale (FIS) questionnaires were applied to people with fatigue. Single Fiber Electromyography (SFEMG) was applied to all individuals and the obtained data were compared between the two groups. Results: 70.3% of the patients with Post Viral Fatigue Syndrome (PVFS) had increased fatigue severity. 45.9% of the patients experienced mild fatigue, 35.1% of them experienced moderate fatigue, 13.5% experienced significant fatigue and 5.4% of them had severe fatigue according to FIS. The number of fibers with pathologically increased jitter and the mean jitter values of patients with PVFS were significantly higher than those of healthy individuals. 59.4% of patients with PVFS had moderate motor end plate dysfunction on SFEMG and 13.6% had apparent motor end plate dysfunction. Conclusion: Patients with PVFS caused by COVID-19 have neuromuscular junction dysfunction. It is unclear why these individuals are affected, but abnormal immune responses can lead to this dysfunction.

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Rosario, Martin G., Alexandre Costet, and Logan Berlof. "The Influence of Energy Drinks on Lower Limb Muscle Fatigue and Recovery in Healthy Young Adults." Journal of Sports Research 9, no. 2 (October 5, 2022): 101–11. http://dx.doi.org/10.18488/90.v9i2.3153.

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Many studies have researched the influences of energy drinks (Edrinks) on the central nervous system, balance, and neuromuscular control. Understanding the neuromuscular adaptations that occur after drinking Celsius Edrinks on muscle fatigue and balance can be helpful when advising the use of Edrinks during physical activity. This study aims to understand the impact Celsius Edrinks has on neuromuscular adaptations in lower limb musculature during muscle fatigue, and balance in young, healthy adults. Two males and eighteen females were recruited for this study. The tibialis anterior, gastrocnemius, and Gluteus Medius neuromuscular data were captured with surface EMG electrodes. The number of single-leg heel raises repetitions and time to fatigue were recorded. Subjects were asked to perform single-leg heel raises to fatigue and single-leg balance activities pre-energy drink consumption, and the same tasks were repeated on the opposite not tested limb (post Edrinks consumption) to equate data points. Neuromuscular timing data suggested modifications on leg musculature. Although not significant, the amount of single-leg heel raises performed pre, and post-drink intake increased post-energy drink consumption. We infer that the ingredients in Celsius energy drinks, including caffeine, Taurine, and Guarana, alter nerve conduction velocity in lower limb musculature during exercise and balance activities. Future studies should research the influence of Celsius energy drinks in different scenarios and populations.

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Silva, Cristiano Rocha da, Danilo de Oliveira Silva, Deisi Ferrari, Rúben de Faria Negrão Filho, Neri Alves, and Fábio Mícolis de Azevedo. "Exploratory study of electromyographic behavior of the vastus medialis and vastus lateralis at neuromuscular fatigue onset." Motriz: Revista de Educação Física 20, no. 2 (June 2014): 213–20. http://dx.doi.org/10.1590/s1980-65742014000200012.

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This study aimed to determine and analyze the neuromuscular fatigue onset by median frequency (MDF) and the root mean square (RMS) behavior of an electromyographic signal (EMG). Eighteen healthy men with no prior knee problems initially performed three maximum voluntary isometric contractions (MVIC). After two days of MVIC test, participants performed a fatiguing protocol in which they performed submaximal knee-extension contractions at 20% and 70% MVIC held to exhaustion. The MDF and RMS values from the EMG signals were recorded from the vastus medialis (VM) and the vastus lateralis (VL). Analysis of the MDF and RMS behavior enabled identification of neuromuscular fatigue onset for VM and VL muscles in 20% and 70% loads. Alterations between the VM and VL in the neuromuscular fatigue onset, at 20% and 70% MVIC, were not significant. These findings suggest that the methodology proposal was capable of indicating minute differences sensible to alterations in the EMG signals, allowing identification of the moment when the MDF and the RMS showed significant changes in behavior. The methodology used was also a viable one for describing and identifying the neuromuscular fatigue onset by means of the analysis of EMG signals.

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Armatas, Vasilios, Eleni Bassa, Dimitrios Patikas, Ilias Kitsas, Georgios Zangelidis, and Christos Kotzamanidis. "Neuromuscular Differences Between Men and Prepubescent Boys During a Peak Isometric Knee Extension Intermittent Fatigue Test." Pediatric Exercise Science 22, no. 2 (May 2010): 205–17. http://dx.doi.org/10.1123/pes.22.2.205.

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The aim of this study was to examine the fatigue and recovery in boys and men during a maximal intermittent isometric fatigue test of the knee extensor muscles, by evaluating the electromyogram of vastus lateralis, vastus medialis and biceps femoris. Thirteen boys (10.0 ± 0.8yrs) and 13 men (26.1 ± 4.2yrs) were fatigued until torque reached 50% of its initial value. Three and 6 min after, a maximal isometric knee extension test was assessed. Men had faster torque decline during fatigue and slower torque recovery compared with boys. Agonist activity declined in both groups during fatigue but men had greater extent of reduction. After 6 min boys recovered fully in respect to agonist EMG, whereas this was not the case for the men. The lower level of fatigue and faster recovery in boys could be attributed to the limited inhibition that was observed in the boys’ agonist muscles, whereas the antagonist activity does not seem to play a role in the fatigue or recovery differences between the groups.

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Jammes, Yves, and Frédérique Retornaz. "Understanding neuromuscular disorders in chronic fatigue syndrome." F1000Research 8 (November 28, 2019): 2020. http://dx.doi.org/10.12688/f1000research.18660.1.

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Muscle failure has been demonstrated in patients with myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS). Neurophysiological tools demonstrate the existence of both central and peripheral fatigue in these patients. Central fatigue is deduced from the reduced amplitude of myopotentials evoked by transcranial magnetic stimulation of the motor cortex as well as by the muscle response to interpolated twitches during sustained fatiguing efforts. An impaired muscle membrane conduction velocity assessed by the reduced amplitude and lengthened duration of myopotentials evoked by direct muscle stimulation is the defining feature of peripheral fatigue. Some patients with ME/CFS show an increased oxidative stress response to exercise. The formation of lipid hydroperoxides in the sarcolemma, which alters ionic fluxes, could explain the reduction of muscle membrane excitability and potassium outflow often measured in these patients. In patients with ME/CFS, the formation of heat shock proteins (HSPs) is also reduced. Because HSPs protect muscle cells against the deleterious effects of reactive oxygen species, the lack of their production could explain the augmented oxidative stress and the consecutive alterations of myopotentials which could open a way for future treatment of ME/CFS.

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Knorr, Svetlana, Charles L. Rice, and S. Jayne Garland. "Perspective on neuromuscular factors in poststroke fatigue." Disability and Rehabilitation 34, no. 26 (June 5, 2012): 2291–99. http://dx.doi.org/10.3109/09638288.2012.683233.

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Montes, Jacqueline, Michelle Blumenschine, Sally Dunaway, Aliza S. Alter, Kristin Engelstad, Ashwini K. Rao, Claudia A. Chiriboga, Douglas M. Sproule, and Darryl C. De Vivo. "Weakness and Fatigue in Diverse Neuromuscular Diseases." Journal of Child Neurology 28, no. 10 (July 11, 2013): 1277–83. http://dx.doi.org/10.1177/0883073813493663.

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Lou, Jau-Shin. "Techniques in Assessing Fatigue in Neuromuscular Diseases." Physical Medicine and Rehabilitation Clinics of North America 23, no. 1 (February 2012): 11–22. http://dx.doi.org/10.1016/j.pmr.2011.11.003.

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Knorr, S., T. D. Ivanova, T. J. Doherty, J. A. Campbell, and S. J. Garland. "The origins of neuromuscular fatigue post-stroke." Experimental Brain Research 214, no. 2 (August 17, 2011): 303–15. http://dx.doi.org/10.1007/s00221-011-2826-5.

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Vitry, Florian, Alain Martin, and Maria Papaiordanidou. "Impact of stimulation frequency on neuromuscular fatigue." European Journal of Applied Physiology 119, no. 11-12 (October 11, 2019): 2609–16. http://dx.doi.org/10.1007/s00421-019-04239-x.

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Wohlfarth, K., R. Dengler, M. Schubert, J. Elek, and S. Zierz. "Testing neuromuscular fatigue in controls and patients." Electroencephalography and Clinical Neurophysiology 87, no. 2 (August 1993): S104. http://dx.doi.org/10.1016/0013-4694(93)91340-7.

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Schillings, M. L., J. S. Kalkman, H. M. H. A. Janssen, B. G. M. van Engelen, G. Bleijenberg, and M. J. Zwarts. "Experienced and physiological fatigue in neuromuscular disorders." Clinical Neurophysiology 118, no. 2 (February 2007): 292–300. http://dx.doi.org/10.1016/j.clinph.2006.10.018.

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JUBEAU, MARC, THOMAS RUPP, JOHN TEMESI, STÉPHANE PERREY, BERNARD WUYAM, GUILLAUME Y. MILLET, and SAMUEL VERGES. "Neuromuscular Fatigue during Prolonged Exercise in Hypoxia." Medicine & Science in Sports & Exercise 49, no. 3 (March 2017): 430–39. http://dx.doi.org/10.1249/mss.0000000000001118.

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Lou, Jenny W. H., Austin J. Bergquist, Abdulaziz Aldayel, Jennifer Czitron, and David F. Collins. "Interleaved neuromuscular electrical stimulation reduces muscle fatigue." Muscle & Nerve 55, no. 2 (November 23, 2016): 179–89. http://dx.doi.org/10.1002/mus.25224.

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Medysky, Mary E., John Temesi, Shu J. Fan, S. Nicole Culos-Reed, and Guillaume Y. Millet. "Comparing Indices of Neuromuscular Fatigue with Subjective Fatigue in Cancer Survivors." Medicine & Science in Sports & Exercise 48 (May 2016): 334. http://dx.doi.org/10.1249/01.mss.0000486011.64680.fa.

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Lattier, G., G. Y. Millet, A. Martin, and V. Martin. "Fatigue and Recovery After High-Intensity Exercise Part I: Neuromuscular Fatigue." International Journal of Sports Medicine 25, no. 6 (August 2004): 450–56. http://dx.doi.org/10.1055/s-2004-820939.

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Binder-Macleod, Stuart A., and Lynn Snyder-Mackler. "Muscle Fatigue: Clinical Implications for Fatigue Assessment and Neuromuscular Electrical Stimulation." Physical Therapy 73, no. 12 (December 1, 1993): 902–10. http://dx.doi.org/10.1093/ptj/73.12.902.

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Torri, Francesca, Piervito Lopriore, Vincenzo Montano, Gabriele Siciliano, Michelangelo Mancuso, and Giulia Ricci. "Pathophysiology and Management of Fatigue in Neuromuscular Diseases." International Journal of Molecular Sciences 24, no. 5 (March 5, 2023): 5005. http://dx.doi.org/10.3390/ijms24055005.

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Fatigue is a major determinant of quality of life and motor function in patients affected by several neuromuscular diseases, each of them characterized by a peculiar physiopathology and the involvement of numerous interplaying factors. This narrative review aims to provide an overview on the pathophysiology of fatigue at a biochemical and molecular level with regard to muscular dystrophies, metabolic myopathies, and primary mitochondrial disorders with a focus on mitochondrial myopathies and spinal muscular atrophy, which, although fulfilling the definition of rare diseases, as a group represent a representative ensemble of neuromuscular disorders that the neurologist may encounter in clinical practice. The current use of clinical and instrumental tools for fatigue assessment, and their significance, is discussed. A summary of therapeutic approaches to address fatigue, encompassing pharmacological treatment and physical exercise, is also overviewed.

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Journal articles on the topic 'Neuromuscular fatigue'

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