Relevant bibliographies by topics / Neuromuscular fatigue

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
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Academic literature on the topic 'Neuromuscular fatigue'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 April 2024

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Journal articles on the topic "Neuromuscular fatigue"

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Sharp, C. "Neuromuscular Fatigue." British Journal of Sports Medicine 28, no. 3 (September 1, 1994): 208–9. http://dx.doi.org/10.1136/bjsm.28.3.208-a.

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Williams, Jay H. "Neuromuscular Fatigue." Medicine & Science in Sports & Exercise 26, no. 7 (July 1994): 934. http://dx.doi.org/10.1249/00005768-199407000-00021.

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van Dijk, J. G. "Neuromuscular fatigue." Journal of the Neurological Sciences 120, no. 1 (December 1993): 121. http://dx.doi.org/10.1016/0022-510x(93)90038-z.

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Deffieux, X., K. Hubeaux, E. Lapeyre, M. Jousse, S. Sheikh Ismael, P. Thoumie, and G. Amarenco. "Perineal neuromuscular fatigue." Annales de Réadaptation et de Médecine Physique 49, no. 6 (July 2006): 413–17. http://dx.doi.org/10.1016/j.annrmp.2006.03.010.

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Collins, Brandon W., Gregory E. P. Pearcey, Natasha C. M. Buckle, Kevin E. Power, and Duane C. Button. "Neuromuscular fatigue during repeated sprint exercise: underlying physiology and methodological considerations." Applied Physiology, Nutrition, and Metabolism 43, no. 11 (November 2018): 1166–75. http://dx.doi.org/10.1139/apnm-2018-0080.

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Neuromuscular fatigue occurs when an individual’s capacity to produce force or power is impaired. Repeated sprint exercise requires an individual to physically exert themselves at near-maximal to maximal capacity for multiple short-duration bouts, is extremely taxing on the neuromuscular system, and consequently leads to the rapid development of neuromuscular fatigue. During repeated sprint exercise the development of neuromuscular fatigue is underlined by a combination of central and peripheral fatigue. However, there are a number of methodological considerations that complicate the quantification of the development of neuromuscular fatigue. The main goal of this review is to synthesize the results from recent investigations on the development of neuromuscular fatigue during repeated sprint exercise. Hence, we summarize the overall development of neuromuscular fatigue, explain how recovery time may alter the development of neuromuscular fatigue, outline the contributions of peripheral and central fatigue to neuromuscular fatigue, and provide some methodological considerations for quantifying neuromuscular fatigue during repeated sprint exercise.
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Féasson, L., J. P. Camdessanché, L. El Mhandi, P. Calmels, and G. Y. Millet. "Fatigue and neuromuscular diseases." Annales de Réadaptation et de Médecine Physique 49, no. 6 (July 2006): 375–84. http://dx.doi.org/10.1016/j.annrmp.2006.04.016.

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Piponnier, Enzo, Vincent Martin, Bastien Bontemps, Emeric Chalchat, Valérie Julian, Olivia Bocock, Martine Duclos, and Sébastien Ratel. "Child-adult differences in neuromuscular fatigue are muscle dependent." Journal of Applied Physiology 125, no. 4 (October 1, 2018): 1246–56. http://dx.doi.org/10.1152/japplphysiol.00244.2018.

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The aim of the present study was to compare the development and etiology of neuromuscular fatigue of the knee extensor (KE) and plantar flexor (PF) muscles during repeated maximal voluntary isometric contractions (MVICs) between children and adults. Prepubertal boys ( n = 21; 9–11 yr) and men ( n = 24; 18–30 yr) performed two fatigue protocols consisting of a repetition of 5-s isometric MVIC of the KE or PF muscles interspersed with 5-s passive recovery periods until MVIC reached 60% of its initial value. The etiology of neuromuscular fatigue of the KE and PF muscles was investigated by means of noninvasive methods, such as the surface electromyography, single and doublet magnetic stimulation, twitch interpolation technique, and near-infrared spectroscopy. The number of repetitions performed was significantly lower in men (15.4 ± 3.8) than boys (38.7 ± 18.8) for the KE fatigue test. In contrast, no significant difference was found for the PF muscles between boys and men (12.1 ± 4.9 and 13.8 ± 4.9 repetitions, respectively). Boys displayed a lower reduction in potentiated twitch torque, low-frequency fatigue, and muscle oxygenation than men whatever the muscle group considered. In contrast, voluntary activation level and normalized electromyography data decreased to a greater extent in boys than men for both muscle groups. To conclude, boys experienced less peripheral and more central fatigue during repeated MVICs than men whatever the muscle group considered. However, child-adult differences in neuromuscular fatigue were muscle-dependent since boys fatigued similarly to men with the PF muscles and to a lower extent with the KE muscles. NEW & NOTEWORTHY Child-adult differences in neuromuscular fatigue during repeated maximal voluntary contractions are specific to the muscle group since children fatigue similarly to adults with the plantar flexor muscles and to a lower extent with the knee extensor muscles. Children experience less peripheral fatigue and more central fatigue than adults, regardless of the muscle group considered.
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Millet, Guillaume Y., Vincent Martin, Nicola A. Maffiuletti, and Alain Martin. "Neuromuscular Fatigue After a Ski Skating Marathon." Canadian Journal of Applied Physiology 28, no. 3 (June 1, 2003): 434–45. http://dx.doi.org/10.1139/h03-032.

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The aim of this study was to characterize neuromuscular fatigue in knee extensor muscles after a marathon skiing race (mean ± SD duration = 159.7 ± 17.9 min). During the 2 days preceding the event and immediately after, maximal percutaneous electrical stimulations (single twitch, 0.5-s tetanus at 20 and 80 Hz) were applied to the femoral nerve of 11 trained skiers. Superimposed twitches were also delivered during maximal voluntary contraction (MVC) to determine maximal voluntary activation (%VA). EMG was recorded from the vastus lateralis muscle. MVC decreased with fatigue from 171.7 ± 33.7 to 157.3 ± 35.2 Nm (-8.4%; p < 0.005) while %VA did not change significantly. The RMS measured during MVC and peak-to-peak amplitude of the compound muscle action potential (PPA) from the vastus lateralis decreased with fatigue by about 30% (p < 0.01), but RMS•PPA−1was similar before and after the ski marathon. Peak tetanus tension at 20 Hz and 80 Hz (P020 and P080, respectively) did not change significantly, but P020•P080−1 increased (p < 0.05) after the ski marathon. Data from electrically evoked single twitches showed greater peak mechanical response, faster rate of force development, and shorter contraction time in the fatigued state. From these results it can be concluded that a ski skating marathon (a) alters slightly but significantly maximal voluntary strength of the knee extensors without affecting central activation, and (b) induces both potentiation and fatigue. Key words: low- and high-frequency electrical stimulation, central activation, potentiation
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Izquierdo, M., J. Ibañez, J. Calbet, M. González-Izal, I. Navarro-Amézqueta, C. Granados, A. Malanda, et al. "Neuromuscular Fatigue after Resistance Training." International Journal of Sports Medicine 30, no. 08 (April 20, 2009): 614–23. http://dx.doi.org/10.1055/s-0029-1214379.

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Lou, Jau-Shin. "Approaching Fatigue in Neuromuscular Diseases." Physical Medicine and Rehabilitation Clinics of North America 16, no. 4 (November 2005): 1063–79. http://dx.doi.org/10.1016/j.pmr.2005.08.012.

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Dissertations / Theses on the topic "Neuromuscular fatigue"

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St, Clair Gibson Alan. "Fatigue, aging and the neuromuscular system." Master's thesis, University of Cape Town, 2001. http://hdl.handle.net/11427/3205.

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Bibliography: p. 400-445.
The aim of this thesis was to investigate the relationship between chronic exercise activity, aging, the neuromuscular system and the symptom of fatigue in a series of studies. The hypothesis of the thesis was that in contrast to the accepted dogma that exercise is beneficial to an individual, increasing longevity and improving quality of life, excessive or chronic exercise activity may accelerate the aging process, lead to neuromuscular damage, and cause the development of pathological symptoms or levels of fatigue.
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Taylor, Kristie-Lee. "Monitoring neuromuscular fatigue in high performance athletes." Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2012. https://ro.ecu.edu.au/theses/581.

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With improving professionalism of sports around the world, the volume and frequency of training required for competitive performances at the elite level has increased concurrently. With this amplification in training load comes an increased need to closely monitor the associated fatigue responses, since maximising the adaptive response to training is also reliant on avoiding the negative consequences of excessive fatigue. The rationale for the experimental chapters in this thesis was established after considering survey responses regarding current best practice for monitoring fatigue in high performance sporting environments (Chapter 3). On the basis of the results, vertical jump assessments were selected for further investigation regarding their utility in determining neuromuscular fatigue responses. Outcomes from the subsequent series of studies aimed to provide practitioners working in high performance sport with guidelines for using vertical jumps to monitor athletic fatigue. The results from Chapter 4 indicate using the mean value of at least six jumps enhances the ability to detect small but practically important changes in performance from week to week. This study also highlighted large differences (4-6%) in morning and afternoon performance, indicating that the time of day performance is assessed needs to be accounted for when monitoring changes in jump performance. Chapter 5 explored the theory that the time of day effect observed in Chapter 4 can be explained by internal temperature differences. This theory was supported by demonstrating that an extended warm-up period can negate differences in jump performance in the morning and the afternoon. Researchers who are unable to standardise the time of day that assessment occurs are able, therefore, to control for performance differences by manipulating the warm-up protocols. The third study examined changes in vertical jump performance over a three month training period and produced several novel outcomes. A major finding was that unloaded jumps were more sensitive to neuromuscular fatigue during intensive training than loaded jumps (Chapter 6). Furthermore, this set of results showed that all subjects changed their jump technique via a reduction in the amplitude of the countermovement when they were highly fatigued. Using the same data, an analysis was performed to quantify individual differences in within-subject variation (Chapter 7) during normal and intensive training. These results provided the first indication that within-subject variability in vertical jump performance is substantially different between individuals and between different training phases, an important consideration for interpreting the practical importance of performance changes. In Chapter 8 the relationship between vertical jump performance and electrically elicited force of the knee extensors was examined to better understand the mechanism(s) of changes in jump performance associated with neuromuscular fatigue during intensive overload training. The results showed that the fatigue assessed by vertical jump performance was likely not only peripheral in origin as previously suggested by other authors. Further research is required to further understand the mechanisms of reduced performance during overload training, although the preliminary evidence presented implicates central mechanisms. To conclude the thesis, the findings presented in the experimental chapters are summarised, with a series of practical recommendations for using vertical jumps to monitor athletic fatigue presented.
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Lin, Zengyuan. "Neuromuscular fatigue following a singles badminton match." Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2014. https://ro.ecu.edu.au/theses/1279.

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A typical badminton singles match involves numerous intense and high impact movements. Lunges were accounted for approximately 15% of overall movements and were believed to presumably induce significant muscle damage following a match. However, no previous study has investigated changes in knee extensor muscle function after a badminton match.The present study investigated changes in knee extensor neuromuscular function and muscle soreness after a simulated 1-h badminton singles match in relation to the number of lunges performed in the match. Ten state-level male badminton players were recruited (n=10), with each player played a total of eight simulated 1-h matches under the International Badminton World Federation rules. However, each participant was required to play against the same opponent twice and only one participant was fitted with the equipment at any one session, thus the total number of matches analysed was 40. The number of lunges performed by each player in a game was obtained from video analysis. Heart rate (HR) and core body temperature were recorded during the matches, and blood lactate (BL) was measured before and immediately post match. Both femoral nerve and muscle electrical stimulations were used in the present study. Maximal voluntary isometric contraction (MVC) torque of the knee extensors and flexors, voluntary activation during the knee extension MVC (VA), torque generated by a doublet (DT), and 20 Hz (T20) and 80 Hz stimulation (T80) and the ratio (T20/T80) for the knee extensors, and muscle soreness of knee extensor muscles by a 100-mm visual analog scale (VAS) were measured before, immediately (8 - 10 min post-match), 1-h and 24-h after a match. Pearson product-moment correlations were computed to examine relationships between variables using ANOVA.Average (± SD) match HR was 162.0 ± 11.0 bpm, post-match BL was 7.2 ± 1.3 mM.L-1, and 194 ± 18 lunges were performed per match per player. Core body temperature increased from 36.5 ± 0.5 oC to 39.4 ± 0.5 oC immediately post match. Knee extension MVC torque was lower than baseline (278.4 ± 50.8 Nm) at immediately (-11%) and 1 h (-14%) post match (P Moderate muscle soreness developed after 1-h simulated badminton matches, but muscle function returned to baseline by 24 hour post match, indicating moderate muscle fibre damage. Since VA was decreased without changes in T20/T80, and knee flexion MVC torque also showed similar changes to those of knee extension MVC torque that was thought to be affected by lunges, the decrease in MVC torque appeared to be associated with central rather than peripheral fatigue or muscle damage. With moderate muscle soreness developing after 1-h simulated badminton matches and muscle function returning to baseline by 24 hour post-game, suggesting minimal muscle fibre damage. It was concluded that both central and peripheral factors contributed to alterations in neuromuscular fatigue and that muscle damage was moderate after the singles matches in which the game intensity and physiological characteristics were close to those in competitive tournaments.
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Alahmari, Sami Khaloufah M. "Muscle force production and neuromuscular fatigue responses to neuromuscular electrical stimulation and tendon vibration." Thesis, Queensland University of Technology, 2022. https://eprints.qut.edu.au/236174/1/Sami_Alahmari_Thesis.pdf.

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Neuromuscular electrical stimulation is an intervention that produces artificial muscle contractions and is used to rehabilitate people with motor impairments. However, a typical problem is the occurrence of rapid fatigue, which limits the training stimulus. This thesis investigated whether the addition of tendon vibration and concurrent activation of upper limb muscles could minimise fatigue and maximise torque produced by the calf muscles during neuromuscular electrical stimulation. The experiments in this thesis showed that superimposition of tendon vibration can increase the amount of torque produced by calf muscles, and that the addition of sustained handgrip contraction could further increase this torque.
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Lloyd, Alex. "Neuromuscular fatigue, muscle temperature and hypoxia : an integrative approach." Thesis, Loughborough University, 2016. https://dspace.lboro.ac.uk/2134/21766.

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Real world exposures to physiologically and/or psychologically stressful environments are often multifactorial. For example, high-altitude typically combines exposure to hypobaric hypoxia, solar radiation and cold ambient temperatures, while sea level thermal stress is often combined with supplementary or transient stressors such as rain, solar radiation and wind. In such complex environments, the effect of one stressor on performance may be subject to change, simply due to the presence of another independent stressor. Such differential influences can occur in three basic forms; additive, antagonistic and synergistic, each term defining a fundamental concept of inter-parameter interactions. As well as the natural occurrence of stressors in combination, understanding interactions is fundamental to experimentally modelling how multiple physiological strains integrate in their influence on or regulation of - exercise intensity. In this thesis the current literature on neuromuscular fatigue and the influence of thermal and hypoxic stress is reviewed (Chapter 1). This is followed by an outline of the methodological developments used in the subsequent experiments (Chapter 2). In the first experimental study (Chapter 3) a novel approach was adopted to investigate the combined effect of muscle cooling and hypoxia on neuromuscular fatigue in humans. The results showed that the neuromuscular system s maximal force generating capacity declined by 8.1 and 13.9% during independent cold and hypoxic stress compared to control. Force generation decreased by 21.4% during combined hypoxic-cold compared to control, closely matching the additive value of hypoxia and cold individually (22%). This was also reflected in the measurement of mechanical fatigue (electromechanical ratio), demonstrating an additive response during combined hypoxic-cold. From this study, it was concluded that when moderate hypoxia and cold environmental temperatures are combined during low intensity exercise, the level of fatigue increases additively with no interaction between these stressors. Before conducting a more complex investigation on combined stressors, a better understanding of the role of muscle temperature on central fatigue - i.e. voluntary muscle activation via the afferent signalling pathways was sought. The focus of Chapter 4 was to quantify the relationship between muscle temperature and voluntary muscle activation (central fatigue) across a wide range of temperatures. The primary finding was that different muscle temperatures can induce significant changes in voluntary activation (0.5% reduction per-degree-centigrade increase in muscle temperature) when neural drive is sustained for a prolonged effort (e.g. 120-s); however this effect is not exhibited during efforts that are brief in duration (e.g. 3-s). To further explore this finding, Chapter 5 investigated the effect of metaboreceptive feedback at two different muscle temperatures, using post-exercise muscle ischemia, on voluntary activation of a remote muscle group. The results showed that at the same perceived mental effort, peripheral limb discomfort was significantly higher with increasing muscle temperature (2% increase per-degree-centigrade increase). However any influence of increased muscle temperature on leg muscle metaboreceptive feedback did not appear to inhibit voluntary muscle activation - i.e. central control - of a remote muscle group, as represented by an equal force output and voluntary activation in the thermoneutral, contralateral leg. In Chapter 6, the psycho-sensory effects of changes in muscle temperature on central fatigue during dynamic exercise were investigated. During sustained dynamic exercise, fatigue development appeared to occur at a faster rate in hot muscle (4% increase per-degree-centigrade increase) leading to a nullification of the beneficial effects of increased muscle temperature on peak power output after a period of ~60-s maximal exercise. In support of previous studies using isometric exercise (Chapter 4 and 6), participants reported significantly higher muscular pain and discomfort in hot muscle compared to cooler muscle during dynamic exercise (2 and 1% increase per-degree-centigrade increase respectively), however this did not result in a lower power output. From Chapters 4, 5 and 6 it was concluded that in addition to faster rates of metabolite accumulation due to cardiovascular strain, it is possible that a direct sensitisation of the metaboreceptive group III and IV muscle afferents occurs in warmer muscle. This likely contributes to the reduction in voluntary muscle activation during exercise in the heat, while it may attenuate central fatigue in the cold. It was also interpreted that muscle afferents may have a similar signalling role to cutaneous sensory afferents; the latter of which are recognised for their role in providing thermal feedback to the cognitive-behavioural centres of the brain and aiding exercise regulation under thermal stress. The impact of body core and active muscle temperature on voluntary muscle activation represented a similar ratio (5 to 1 respectively) to the temperature manipulated (single leg) to non-temperature manipulated mass (rest of body) in Chapters 4, 5 and 6. This indicates that voluntary muscle activation may also be regulated based on a central meta-representation of total body heat content i.e. the summed firing rates of all activated thermoreceptors in the brain, skin, muscle, viscera and spine. Building on the initial findings of Chapter 3, Chapter 7 investigated the causative factors behind the expression of different interaction types during exposure to multi-stressor environments. This was achieved by studying the interaction between thermal stress and hypoxia on the rate of peripheral and central fatigue development during a high intensity bout of knee extension exercise to exhaustion. The results showed that during combined exposure to moderate hypoxia and mild cold, the reductions in time to exhaustion were additive of the relative effects of hypoxia and cold independently. This differs from the findings in Chapter 3, in which fatigue was additive of the absolute effects of cold and hypoxia. In contrast, combining moderate hypoxia with severe heat stress resulted in a significant antagonistic interaction on both the absolute and relative reductions in time to exhaustion i.e. the combined effect being significantly less than the sum of the individual effects. Based on the results in Chapter 7, a quantitative paradigm for understanding of systematic integration of multifactorial stressors was proposed. This is, that the interaction type between stressors is influenced by the impact magnitude of the individual stressors effect on exercise capacity, whereby the greater the stressors impact, the greater the probability that one stressor will be cancelled out by the other. This is the first study to experimentally model the overarching principles characterising the presence of simultaneous physiological strains, suggesting multifactorial integration be subject to the worst strain takes precedence when the individual strains are severe.
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Yang, Qi. "BIO-SIGNAL ANALYSIS IN FATIGUE AND CANCER RELATED FATIGUE: WEAKENING of CORTICOMUSCULAR FUNCTIONAL COUPLING." Cleveland, Ohio : Cleveland State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=csu1213888265.

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Thesis (D.Eng.)--Cleveland State University, 2008.
Abstract. Title from PDF t.p. (viewed on Oct. 8, 2008). Includes bibliographical references (p. 91-99). Available online via the OhioLINK ETD Center. Also available in print.
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Reed, Georgina. "The effect of neuromuscular training on fatigue resistance in female footballers." Thesis, University of Gloucestershire, 2017. http://eprints.glos.ac.uk/5285/.

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ACL injury is predominant in female footballers largely due to a combination of kinetic and neuromuscular risk factors. The majority of ACL injuries in football occur during an unanticipated cutting manoeuvre, and the risk of this injury is heightened during the final 30 minutes of each half of match-play. Due to an increased injury incidence towards the end of match-play, it is possible that fatigue might serve as a risk factor for ACL injury. However, there currently exists limited research examining the effects of fatigue on a variety of kinetic and electromyographic variables in female footballers during an unanticipated cutting manoeuvre. Neuromuscular training programmes have been utilised in injury prevention studies and proven effective in reducing injury incidence by improving certain kinetic and neuromuscular ACL injury risk factors. The overall aim of this thesis is to examine the effectiveness of neuromuscular training on the fatigue resistance of ACL injury risk factors in female footballers. Study one of this thesis examined the reliability of a combination of kinetic and electromyographic measures in female footballers performing an unanticipated cutting manoeuvre. There were no significant differences in mean values and large to nearly perfect correlations (ICC = 0.49 - 0.96) for all kinetic variables. The majority of kinetic variables displayed a CV of less than 10%, with the exception of loading rates and time to peak force (CV% = 17.33 - 24.51). In comparison to previous research, electromyographic variables displayed a greater range of typical error (CV% = 17.6 - 129.2); however, the majority of electromyographic variables displayed a large, very large or nearly perfect correlation (ICC = 0.26 – 0.91) and no significant differences in the mean score. In line with previous research, standards of reliability, and anticipated changes in response to acute fatigue, all kinetic and electromyographic variables were deemed acceptable to use in subsequent studies. Kinetic variables showed better reliability than electromyographic variables, which was to be expected due to electromyographic measures being a physiological measure. Previous research has established that 16 - 18 year old female footballers are at highest risk of ACL injury, and it is most commonly caused during performance of an unanticipated cutting manoeuvre. It has also been established that the majority of injuries in female football are sustained in the last 30 minutes of each half when fatigue is present. Therefore, acute fatigue appears to be an influential risk factor for ACL injury. Study two of this thesis examined the effects of acute fatigue on the electromyographic and kinetic ACL injury risk factors in 16 - 18 year old female footballers, when performing an unanticipated cutting manoeuvre. A modified SAFT90 protocol was used to mimic football match-play. Data showed that during the unanticipated cutting manoeuvre following the SAFT90, participants produced greater GRF (vGRF; possibly, apGRF very likely), lower GCT (very likely), increased background hamstring activation (0 - 30 ms; very likely) and increased short-latency feedback activation of the hamstrings (31 - 60 ms; likely and possibly). Results suggested that following a simulated match-play protocol, female footballers experienced greater force absorption while utilising a safer muscle recruitment strategy. Therefore, injury prevention training should seek to improve a player’s aility to tolerate ground reaction forces when experiencing acute fatigue, with a large emphasis on enhancing neuromuscular control within the hamstrings muscle group.
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Cormack, Stuart J. "Neuromuscular fatigue and endocrine responses in elite Australian rules football players." Connect to thesis, 2008. http://portal.ecu.edu.au/adt-public/adt-ECU2008.0010.html.

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BERETTA-PICCOLI, MATTEO CARLOFELICE. "EVALUATION OF NEUROMUSCULAR FATIGUE THROUGH INNOVATIVE SURFACE EMG PARAMETERS IN HUMANS." Doctoral thesis, Università degli studi di Pavia, 2020. http://hdl.handle.net/11571/1371980.

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The surface electromyogram (EMG) undergoes several changes during an isometric fatiguing muscle contraction. Amplitude and spectral parameters, as well as muscle fiber conduction velocity and fractal dimension of the surface EMG signal, may be used as indirect fatigability indices to monitor these changes. The aims of this thesis were to determine whether surface EMG is a reliable tool for estimating conduction velocity, through a systematic review of the literature; to determine the relationship between muscle force and the selected fatigability indices; and to study the behavior of the fatigability indices in patients with facioscapulohumeral muscular dystrophy (FSHD) during a fatiguing task, with respect to healthy controls. It was hypothesized that the fractal dimension was not related to the intensity of muscle contraction, and that FSHD patients would have shown significant differences in the considered fatigability indices. In the first study (p. 32) high reliability was reported in eight studies and was, in general, associated with using the initial or mean conduction velocity value, using several electrodes (3 to 8), ensuring appropriate electrode positioning, and evaluating muscles with fibers that run parallel to the skin. It was next demonstrated in the second study (p. 52), that the values of fractal dimension and mean frequency of the power spectrum increased with force unless a plateau was reached at 30% maximal voluntary contraction. Finally, the third study (p. 61) showed that FSHD patients presented significantly less steeper slopes of mean frequency of the power spectrum, conduction velocity and fractal dimension, compared to the controls. The results of this thesis demonstrated firstly, that sEMG is suitable for use when investigating conduction velocity, which is sensible to peripheral aspects affecting performance fatigability; secondly, the use of fractal dimension, as index of central factors affecting performance fatigability, may be considered above a certain level of force, regardless of muscle contraction intensity; and lastly, that impaired neuromuscular function caused patients with FSHD to exert a smaller force, yield a longer endurance time and experience lower levels of performance fatigability compared to healthy participants. In conclusion, the use of the fractal dimension of the sEMG signal to infer central aspects of performance fatigability should be promoted; in particular, in those muscles were motor unit decomposition techniques are limited by anatomical constraints.
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Castronovo, Anna Margherita <1984&gt. "Techniques and Methods for a multi-scale analysis of neuromuscular fatigue." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2014. http://amsdottorato.unibo.it/6274/7/Castronovo_AnnaMargherita_tesi.pdf.

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This thesis proposes an integrated holistic approach to the study of neuromuscular fatigue in order to encompass all the causes and all the consequences underlying the phenomenon. Starting from the metabolic processes occurring at the cellular level, the reader is guided toward the physiological changes at the motorneuron and motor unit level and from this to the more general biomechanical alterations. In Chapter 1 a list of the various definitions for fatigue spanning several contexts has been reported. In Chapter 2, the electrophysiological changes in terms of motor unit behavior and descending neural drive to the muscle have been studied extensively as well as the biomechanical adaptations induced. In Chapter 3 a study based on the observation of temporal features extracted from sEMG signals has been reported leading to the need of a more robust and reliable indicator during fatiguing tasks. Therefore, in Chapter 4, a novel bi-dimensional parameter is proposed. The study on sEMG-based indicators opened a scenario also on neurophysiological mechanisms underlying fatigue. For this purpose, in Chapter 5, a protocol designed for the analysis of motor unit-related parameters during prolonged fatiguing contractions is presented. In particular, two methodologies have been applied to multichannel sEMG recordings of isometric contractions of the Tibialis Anterior muscle: the state-of-the-art technique for sEMG decomposition and a coherence analysis on MU spike trains. The importance of a multi-scale approach has been finally highlighted in the context of the evaluation of cycling performance, where fatigue is one of the limiting factors. In particular, the last chapter of this thesis can be considered as a paradigm: physiological, metabolic, environmental, psychological and biomechanical factors influence the performance of a cyclist and only when all of these are kept together in a novel integrative way it is possible to derive a clear model and make correct assessments.
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Books on the topic "Neuromuscular fatigue"

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J, Sargeant A., and Kernell D, eds. Neuromuscular fatigue. Amsterdam: North-Holland, 1993.

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Advanced neuromuscular exercise physiology. Champaign, IL: Human Kinetics, 2011.

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C, Gandevia Simon, ed. Fatigue: Neural and muscular mechanisms. New York: Plenum Press, 1995.

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1949-, Enoka Roger M., and Society for Neuroscience Meeting, eds. Neural and neuromuscular aspects of muscle fatigue: Miami, Florida, November 10-13, 1994. New York: Wiley, 1996.

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Brooks, Barbara. CFIDS, an "owner's manual". 2nd ed. Silver Spring, MD: BBNS Publishers, 1990.

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Ratel, Sébastien, and Craig A. Williams. Neuromuscular fatigue. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0009.

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Scientific evidence supports the proposition that prepubertal children fatigue less than adults when performing whole-body dynamic activities like maximal cycling, running bouts, and maximal voluntary isometric/isokinetic muscle contractions. Although the mechanisms underpinning differences in fatigue between children and adults are not all fully understood, there is a consensus that children experience less peripheral fatigue (i.e. muscular fatigue) than their older counterparts. Central factors may also account for the lower fatigability in children. Some studies report a higher reduction of muscle voluntary activation during fatiguing exercise in prepubertal children compared to adults. This could reflect a strategy of the central nervous system aimed at limiting the recruitment of motor units, in order to prevent any extensive peripheral fatigue. Further studies are required to clarify this proposition.
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Stuart, Douglas G., Christine K. Thomas, Alan J. McComas, Simon C. Gandevia, Roger M. Enoka, and Patricia A. Pierce. Fatigue: Neural and Muscular Mechanisms. Springer, 2013.

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Quinlivan, Ros, and Pascal Laforêt. Chronic Fatigue and Acute Rhabdomyolysis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0068.

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Chronic fatigue syndrome is usually idiopathic, management involves a multi-disciplinary approach, advice on pacing activities and cognitive behavioral therapy. Metabolic myopathies that cause exercise intolerance may lead to a fatigue syndrome due to deconditioning, other neuromuscular disorders presenting with paroxysmal fatigue and weakness such as the myasthenic syndromes and channelopathies can occasionally be mistaken for a metabolic disorder. Acute rhabdomyolysis, a potentially life-threatening complication, has many causes both acquired and genetic. Urgent treatment is required and prevention of future episodes requires a careful search for an underlying genetic cause.
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Shaibani, Aziz. Fatigability. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0025.

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Undue fatigability is common in neuromuscular clinics but non-neuromuscular causes are much more common than neuromuscular causes. Generalized fatigue is commonly caused by anemia, hypothyroidism, obstructive sleep apnea, depression, chronic fatigue syndrome, uremia, COPD, etc. Physiological fatigue is accentuated by neuromuscular disorders. Most strikingly, myasthenia gravis causes undue fatigue of the ocular, chewing, swallowing, and breathing muscles. However, ALS, myopathies, and motor neuropathies are also associated with abnormal fatigue. Myasthenia rarely causes isolated fatigue. Examination for fatigability should be part of neuromuscular evaluation and is conducted by inducing repetitive or sustained contraction of the suspected muscles (typically extraocular muscles) for a minute and reevaluation after 2 minutes of rest of the tested muscles.
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Shaibani, Aziz. Fatigability. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0025.

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Undue fatigability is common in neuromuscular clinics, but nonneuromuscular causes are much more common than neuromuscular causes. Generalized fatigue is commonly caused by anemia, hypothyroidism, obstructive sleep apnea, depression, chronic fatigue syndrome (CFS), uremia, chronic obstructive pulmonary disease (COPD), and other diseases. Physiological fatigue is accentuated by neuromuscular disorders. Most strikingly, myasthenia gravis (MG) causes undue fatigue of the ocular, chewing, swallowing, and breathing muscles. However, amyotrophic lateral sclerosis (ALS), myopathies, and motor neuropathies are also associated with abnormal fatigue. Central causes like multiple sclerosis are notorious for fatigue. It is hard to measure fatigue because it is subjective and varies with the psychological status. Myasthenia hardly causes isolated fatigue.
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Book chapters on the topic "Neuromuscular fatigue"

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Kernell, D. "Neuromuscular Frequency-Coding and Fatigue." In Advances in Experimental Medicine and Biology, 135–45. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-1016-5_11.

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Sieck, G. C., and Y. S. Prakash. "Fatigue at the Neuromuscular Junction." In Advances in Experimental Medicine and Biology, 83–100. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-1016-5_7.

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Tarata, M., W. Wolf, D. Georgescu, D. Alexandru, and M. Serbanescu. "Monitoring Neuromuscular Fatigue – A Noninvasive Approach." In IFMBE Proceedings, 144–47. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-22586-4_30.

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Zhou, Lan. "A 40-Year-Old Man with Muscle Pain and Fatigue." In A Case-Based Guide to Neuromuscular Pathology, 213–20. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-25682-1_18.

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Jones, D. A. "Muscle Fatigue Due to Changes Beyond the Neuromuscular Junction." In Novartis Foundation Symposia, 178–96. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470715420.ch11.

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Barber-Westin, Sue, and Frank R. Noyes. "Effect of Fatigue and Gender on Lower Limb Neuromuscular Function." In ACL Injuries in the Female Athlete, 259–73. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-56558-2_14.

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Mugnosso, Maddalena, Francesca Marini, Luca Doglio, Chiara Panicucci, Claudio Bruno, Paolo Moretti, Pietro Morasso, and Jacopo Zenzeri. "Quantitative Muscle Fatigue Assessment in Neuromuscular Disorders: A Pilot Study on Duchenne Pediatric Subjects." In Converging Clinical and Engineering Research on Neurorehabilitation III, 459–63. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-01845-0_92.

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Downey, R., R. Kamalapurkar, N. Fischer, and W. Dixon. "Compensating for Fatigue-Induced Time-Varying Delayed Muscle Response in Neuromuscular Electrical Stimulation Control." In Recent Results on Nonlinear Delay Control Systems, 143–61. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-18072-4_7.

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Tarata, M. T. "Monitoring Neuromuscular Fatigue through the Area/Amplitude Ratio Computed on the SEMG and MMG." In IFMBE Proceedings, 215–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-04292-8_47.

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Damit, D. N. Filzah P., S. M. N. Arosha Senanayake, Owais A. Malik, and Nor Jaidi Tuah. "Neuromuscular Fatigue Analysis of Soldiers Using DWT Based EMG and EEG Data Fusion During Load Carriage." In Intelligent Information and Database Systems, 602–12. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54430-4_58.

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Conference papers on the topic "Neuromuscular fatigue"

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Sayenko, Dimitry G., Milos R. Popovic, and Kei Masani. "Spatially distributed sequential stimulation reduces muscle fatigue during neuromuscular electrical stimulation." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6610325.

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Burbano, Maria Jose, Yecid Moreno, Gabriel Patti Sanches Coelho, Felix Mauricio Escalante Ortega, and Adriano Siqueira. "Assessment of neuromuscular fatigue, torque reduction, and velocity performance following FES intervention." In 27th Brazilian Congress of Thermal Sciences and Engineering. ABCM, 2023. http://dx.doi.org/10.26678/abcm.cobem2023.cob2023-1958.

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Araújo, Tiago, Ana Anjos, Neuza Nunes, Pedro Rebelo, and Hugo Gamboa. "Quadriceps Muscle Fatigue and Comfort Generated by Neuromuscular Electrical Stimulation with Current Modulated Waveforms." In International Conference on Bio-inspired Systems and Signal Processing. SCITEPRESS - Science and and Technology Publications, 2015. http://dx.doi.org/10.5220/0005267400920098.

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Sharma, N., P. M. Patre, C. M. Gregory, and W. E. Dixon. "Nonlinear Control of NMES: Incorporating Fatigue and Calcium Dynamics." In ASME 2009 Dynamic Systems and Control Conference. ASMEDC, 2009. http://dx.doi.org/10.1115/dscc2009-2642.

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Neuromuscular electrical stimulation (NMES) is a promising technique that has the potential to restore functional tasks in persons with movement disorders. Clinical and commercial NMES products exist for this purpose, but a pervasive problem with current technology is that overstimulation of the muscle (among other factors) leads to muscle fatigue. The objective of the current effort is to develop a NMES controller that incorporates the effects of muscle fatigue through an uncertain function of the calcium dynamics. A neural network-based estimate of the fatigue model mismatch is incorporated in a nonlinear controller through a backstepping based method to control the human quadriceps femoris muscle undergoing non-isometric contractions. The developed controller is proven to yield uniformly ultimately bounded stability for an uncertain nonlinear muscle model in the presence of bounded nonlinear disturbances (e.g., spasticity, delays, changing load dynamics).
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Mehrabi, Naser, and John McPhee. "Steering Feel Improvement for Different Driver Types Using Model-Based Control." In ASME 2014 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/detc2014-34521.

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A realistic driver model can support the development of new steering technologies by reducing the time-consuming trial and error process of designing products. A neuromuscular driver model, by offering physiologically realistic steering maneuvers can provide insights into the task performance and energy consumption of the driver, including fatigue and muscle co-contraction. Here, two muscles are used in a simplified neuromuscular driver model. To study the effect of driver’s characteristics such as age, gender and physical ability on steering, the muscle parameters are adjusted to represent a particular population. Then, this modified driver model is used to to tune the Electric Power Steering (EPS) assist curves for that particular population.
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Pinto, Wladimir Bocca Vieira de Rezende, Paulo Victor Sgobbi de Souza, Paulo Roberto Abrão Ferreira, Igor Braga Farias, José Marcos Vieira de Albuquerque Filho, Roberta Ismael Lacerda Machado, Bruno de Mattos Lombardi Badia, Márcio Luiz Escórcio Bezerra, Marcus Vinícius Magno Gonçalves, and Acary Souza Bulle Oliveira. "Post-COVID-19 mononeuritis multiplex: a potential complication in severe SARS-CoV- 2 infection survivors." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.030.

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Background: Neuromuscular involvement has been identified in acute and early stages of severe COVID-19. Guillain-Barre syndrome and variants, rhabdomyolysis and prone position-related neuropathy represent early complications. Mononeuritis multiplex is rarely a post-infectious complication. Objectives: Characterization of patients with mononeuritis multiplex after severe COVID-19. Methods, design and setting: We performed a retrospective observational study of clinical, laboratorial and neurophysiological aspects of nine Brazilian patients with mononeuritis multiplex after severe COVID-19 at the Division of Neuromuscular Diseases, Federal University of São Paulo (UNIFESP), São Paulo, Brazil. Results: Nine patients (4 male, 5 female) had mean age at diagnosis of 60.6 years. 78% had at least one risk factor for severe COVID-19. Dyspnea, cough, fever, headache, anosmia, odynophagia, and myalgia were the most common SARS-CoV-2 symptoms. Most patients had large length of stay in intensive care (35.8 days), with orotracheal intubation and the need of prone positioning and tracheostomy. 44% had venous or arterial thromboembolic complications. Mononeuritis multiplex symptoms started after 45.7 days (23-71) of first COVID-19 symptoms. Sensorimotor multifocal axonal mononeuritis multiplex was the most common pattern (78%) with moderate to severe (89%) and lower limb-dominant compromise (67%). 33% with LANSS pain scale >12 and 67% with high fatigue scores on Fatigue Severity Scale. Two patients developed moderate titles of positive antinuclear antibody for nuclear membrane compounds (titin) during diagnostic work-up. Three patients were treated with oral corticosteroids with moderate disease control. Conclusions: Mononeuritis multiplex may be a late neuromuscular complication after severe COVID-19. Vasculitis and endotheliopathy seem to mediate its pathophysiology.
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Abitante, Thomas J., Seward B. Rutkove, Kevin R. Duda, and Dava J. Newman. "Muscle Fatigue During Neuromuscular Electrical Stimulation is Dependent on Training: A Basis for Microgravity Musculoskeletal Countermeasure Design." In ASCEND 2020. Reston, Virginia: American Institute of Aeronautics and Astronautics, 2020. http://dx.doi.org/10.2514/6.2020-4010.

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Marillier, M., A. C. Bernard, D. E. O'Donnell, O. Moran Mendoza, S. Verges, and J. A. Neder. "Exertional Oxygen Supplementation Improves Leg Muscle Oxygenation and Neuromuscular Fatigue in Patients with Fibrotic Interstitial Lung Disease." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2396.

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Sheng, Zhiyu, Kang Kim, and Nitin Sharma. "An Ultrasound Imaging Based Observer for Estimating NMES-Induced Muscle Fatigue: Theory and Simulation." In ASME 2020 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/dscc2020-3196.

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Abstract Neuroprosthetic devices that use transcutaneous neuromuscular electrical stimulation (NMES) are potential interventions to restore skeletal muscle function in people with neurological disorders. As commonly noted, how to assess the NMES-induced muscle fatigue is a critical problem. This is because the capability of fatigue assessment is a necessary precursor for optimally modulating the NMES dosage to improve the control performance of a neuroprosthesis and ensure user’s safety. To effectively estimate the NMES-induced muscle fatigue, this paper proposes a novel state observer that combines a mathematical predictive fatigue model and intermittent feedback from ultrasound-derived strain images. The strain images quantify muscle contractility during NMES. Principal component regression (PCR) is used to derive a relationship between the strain images and instantaneous muscle force production. Lyapunov stability analysis was performed to obtain the convergence property of the designed observer. A globally uniformly ultimately bounded (GUUB) result was obtained. Simulations based on pre-recorded data from a human experiment were also conducted to demonstrate the performance of the designed observer.
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Costa, Maria Alice Dias da, Danielle Antoniazzi Costa Sousa, Igor Roque de Paula, Marcela Ferreira de Andrade Rangel, and Mariana Asmar Alencar. "Fatigue presence in amiotrophic lateral sclerosis and related clinical and functional aspects." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.481.

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Background: Some symptoms are reported in Amyotrophic Lateral Sclerosis (ALS), such as fatigue, which, despite having an impact on the individual, is still poorly investigated, seeming to have a multifactorial cause and manifesting itself as tiredness. Therefore, it is necessary to investigate this symptom and check the factors associated with this condition. Objective: To investigate the presence of ALS fatigue and the correlated clinical and functional factors. Objectives: 65 people with ALS attended at the Neuromuscular Diseases Clinic of HC / UFMG participated. Demographic, clinical, functional aspects were assessed and the Fatigue Severity Scale (FSS), Functional Scale (ALSRFSR), Quality of Life Questionnaire (ALSAQ-40) and mobility were used. Results: Participants (56.6 ± 11.0 years), about 44.6% (n = 29) presented fatigue. The average intensity was 5.4 ± 1.2 and 10.4% used specific medication. The fatigue group differed from the fatigue group in terms of: gender (male n = 48.3% vs female n = 72.2% (p = 0.049)); the presentation of greater pain intensity (p= 0.026); the worst results in functionality and independence in ALSFRS-R (p =0.004); worse perception of Quality of Life (QOL) (p = 0.000). Fatigue correlated with pain (r = 0.425; p = 0.001), overall muscle strength (r = -0.356; p = 0.004), functionality (r = -0.363; p = 0.003) and QOL (r = 0.481; p = 0.000). Conclusions: Fatigue is a symptom that is correlated with worse functionality and QOL, therefore, it is necessary to investigate this symptom and adopt treatment.
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Reports on the topic "Neuromuscular fatigue"

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Sánchez-Ramírez, Celso, Claudia Gohurdet-Silva, and Enzo Bosco-Bau. Acute effects of trail running on neuromuscular fatigue in lower limbs. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2023. http://dx.doi.org/10.37766/inplasy2023.8.0099.

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Rebelo, André, João R. Pereira, Paulo Cunha, Manuel J. Coelho-e-Silva, Lauren B. Sherar, and João Valente-dos-Santos. Training Load, Neuromuscular Fatigue and Well-Being in Volleyball: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2022. http://dx.doi.org/10.37766/inplasy2022.7.0059.

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Review question / Objective: This systematic review aims to compile and order all the training load measures, all fatigue assessments, and all well-being questionnaires used in volleyball training/match monitoring, systematizing them. Condition being studied: Training load: cumulative amount of stress placed on an individual from multiple sessions and games over a period of time. Neuromuscular fatigue: A response that is less than the expected or anticipated contractile response, for a given stimulation. Well-being: A continuous, active process, which is geared towards balancing one’s physical, emotional, social, intellectual and spiritual wellness in order to enhance one’s life quality.5 In sport science the subjective measurement of the response to training and competition are used through the athlete self-report measures (ASRMs). In practice, these often comprise brief, single-item checklists derived from validated questionnaires that are intended to be completed daily.
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C. Uy, Genevieve, Raymond L. Rosales, and Satish Khadilkar. Myopathies in Clinical Care: A Focus on Treatable Causes. Progress in Neurobiology, February 2024. http://dx.doi.org/10.60124/j.pneuro.2024.10.01.

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Myopathies present a wide range of clinical symptoms that affect the skeletal muscles, including weakness, fatigue, and pain. While acquired myopathies receive significant attention due to the availability of treatment options, it is important to note that some inherited myopathies can also be effectively managed. These myopathies can be classified based on their underlying causes, such as infectious agents, autoimmune disorders leading to muscle inflammation, granulomatous inflammation, metabolic abnormalities within the muscle cells, skeletal muscle channel dysfunctions, prolonged ICU stay, and inherited conditions such as Duchenne muscular dystrophy. In this review, we initially present a clinical approach to neuromuscular diseases and subsequently place specific emphasis on myopathies, particularly to those that have treatment options available.
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MALDONADO, KARELYS, JUAN ESPINOZA, DANIELA ASTUDILLO, and WILSON BRAVO. Fatigue and fracture resistance and survival of occlusal veneers of composite resin and ceramics blocks in posterior teeth with occlusal wear: A protocol for a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0036.

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Review question / Objective: The aim of this systematic review is to synthesize the scientific evidence that evaluates fatigue and fracture resistance, survival, and stress distribution, of composite resin CAD/CAM and ceramic CAD/CAM occlusal veneers in posterior teeth with severe occlusal wear. Condition being studied: Currently there is an increase in cases of dental wear, due to several factors such as: excessive consumption of carbonated drinks, a diet high in acids, gastric diseases, anorexia, bulimia, dental grinding, use of highly abrasive toothpastes, or a combination of these(9) (10) (11) (12); which affect the patient in several aspects: loss of vertical dimension, sensitivity due to the exposure of dentin, esthetics, affectation of the neuromuscular system(11) (13) (14). With the advent of minimally invasive dentistry, occlusal veneers have been found to be a valid option to rehabilitate this type of cases and thus avoid greater wear of the dental structure with full coverage restorations. Sometimes when performing a tabletop it is not necessary to perform any preparation, thus preserving the maximum amount of dental tissue(3) (6) (15). Due to the masticatory load either in patients without parafunction where the maximum masticatory force is approximately 424 N for women and 630 N for men or in those who present parafunction where the maximum bite force can vary from 780 to 1120N(7), it is necessary that the occlusal veneers support that load which makes indispensable a compilation of studies investigating both fatigue and fracture resistance and the survival rate of occlusal veneers in different materials and thicknesses.
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