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Journal articles on the topic 'Neuromuscular development'

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Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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1

Orlando, Lianna. "Neuromuscular synapse development." Trends in Neurosciences 24, no. 7 (July 2001): 373. http://dx.doi.org/10.1016/s0166-2236(00)01911-1.

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2

KELLY, A. M., and N. A. RUBINSTEIN. "Development of neuromuscular specialization." Medicine & Science in Sports & Exercise 18, no. 3 (June 1986): 292–98. http://dx.doi.org/10.1249/00005768-198606000-00007.

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3

Miller, Geoffrey. "Neuromuscular development and disease." Neuromuscular Disorders 3, no. 1 (January 1993): 90. http://dx.doi.org/10.1016/0960-8966(93)90050-t.

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4

Russman, B. S. "Neuromuscular Diseases During Development." Archives of Neurology 55, no. 6 (June 1, 1998): 879. http://dx.doi.org/10.1001/archneur.55.6.879.

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5

Kleinman, Ronald E. "Complementary Feeding and Neuromuscular Development." Pediatrics 106, Supplement_4 (November 1, 2000): 1279. http://dx.doi.org/10.1542/peds.106.s4.1279a.

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6

Campagna, Jason A. "Development of the Neuromuscular Junction." International Anesthesiology Clinics 44, no. 2 (May 2006): 1–20. http://dx.doi.org/10.1097/00004311-200604420-00003.

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7

Witzemann, Veit. "Development of the neuromuscular junction." Cell and Tissue Research 326, no. 2 (July 4, 2006): 263–71. http://dx.doi.org/10.1007/s00441-006-0237-x.

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8

Aydin, Onur, Austin P. Passaro, Mohamed Elhebeary, Gelson J. Pagan-Diaz, Anthony Fan, Sittinon Nuethong, Rashid Bashir, Steven L. Stice, and M. Taher A. Saif. "Development of 3D neuromuscular bioactuators." APL Bioengineering 4, no. 1 (March 1, 2020): 016107. http://dx.doi.org/10.1063/1.5134477.

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9

Jennings, Charles G. B., and Steven J. Burden. "Development of the neuromuscular synapse." Current Opinion in Neurobiology 3, no. 1 (February 1993): 75–81. http://dx.doi.org/10.1016/0959-4388(93)90038-z.

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10

Collins, Brandon W., Gregory E. P. Pearcey, Natasha C. M. Buckle, Kevin E. Power, and Duane C. Button. "Neuromuscular fatigue during repeated sprint exercise: underlying physiology and methodological considerations." Applied Physiology, Nutrition, and Metabolism 43, no. 11 (November 2018): 1166–75. http://dx.doi.org/10.1139/apnm-2018-0080.

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Neuromuscular fatigue occurs when an individual’s capacity to produce force or power is impaired. Repeated sprint exercise requires an individual to physically exert themselves at near-maximal to maximal capacity for multiple short-duration bouts, is extremely taxing on the neuromuscular system, and consequently leads to the rapid development of neuromuscular fatigue. During repeated sprint exercise the development of neuromuscular fatigue is underlined by a combination of central and peripheral fatigue. However, there are a number of methodological considerations that complicate the quantification of the development of neuromuscular fatigue. The main goal of this review is to synthesize the results from recent investigations on the development of neuromuscular fatigue during repeated sprint exercise. Hence, we summarize the overall development of neuromuscular fatigue, explain how recovery time may alter the development of neuromuscular fatigue, outline the contributions of peripheral and central fatigue to neuromuscular fatigue, and provide some methodological considerations for quantifying neuromuscular fatigue during repeated sprint exercise.
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11

Sanes, Joshua R., and Jeff W. Lichtman. "DEVELOPMENT OF THE VERTEBRATE NEUROMUSCULAR JUNCTION." Annual Review of Neuroscience 22, no. 1 (March 1999): 389–442. http://dx.doi.org/10.1146/annurev.neuro.22.1.389.

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12

Koles, K., and V. Budnik. "Wnt Signaling in Neuromuscular Junction Development." Cold Spring Harbor Perspectives in Biology 4, no. 6 (June 1, 2012): a008045. http://dx.doi.org/10.1101/cshperspect.a008045.

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13

Nose, A., D. Van Vactor, V. Auld, and C. S. Goodman. "Development of Neuromuscular Specificity in Drosophila." Cold Spring Harbor Symposia on Quantitative Biology 57 (January 1, 1992): 441–49. http://dx.doi.org/10.1101/sqb.1992.057.01.049.

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14

Raghavendra, Thandla. "Neuromuscular Blocking Drugs: Discovery and Development." Journal of the Royal Society of Medicine 95, no. 7 (July 2002): 363–67. http://dx.doi.org/10.1177/014107680209500713.

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15

Sharma, Kamal, and Juan Carlos Izpisúa Belmonte. "Development of the limb neuromuscular system." Current Opinion in Cell Biology 13, no. 2 (April 2001): 204–10. http://dx.doi.org/10.1016/s0955-0674(00)00198-8.

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16

Sunaga, Hiroshi, and Cynthia A. Lien. "Development of New Neuromuscular Blocking Agents." Current Anesthesiology Reports 3, no. 2 (March 29, 2013): 105–13. http://dx.doi.org/10.1007/s40140-013-0016-7.

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17

Ferraro, Elisabetta, Francesca Molinari, and Libera Berghella. "Molecular control of neuromuscular junction development." Journal of Cachexia, Sarcopenia and Muscle 3, no. 1 (October 14, 2011): 13–23. http://dx.doi.org/10.1007/s13539-011-0041-7.

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18

RHEUBEN, M., M. YOSHIHARA, and Y. KIDOKOROT. "Ultrastructural Correlates of Neuromuscular Junction Development." International Review of Neurobiology 43 (1999): 69–92. http://dx.doi.org/10.1016/s0074-7742(08)60541-3.

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19

FERNANDES, J., and H. KESHISHIAN. "Development of the Adult Neuromuscular System." International Review of Neurobiology 43 (1999): 221–39. http://dx.doi.org/10.1016/s0074-7742(08)60547-4.

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20

Raghavendra, T. "Neuromuscular blocking drugs: discovery and development." JRSM 95, no. 7 (July 1, 2002): 363–67. http://dx.doi.org/10.1258/jrsm.95.7.363.

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21

Urman, RichardD, Amit Prabhakar, AlanD Kaye, MelvilleQ Wyche, OrlandoJ Salinas, and Kenneth Mancuso. "Novel drug development for neuromuscular blockade." Journal of Anaesthesiology Clinical Pharmacology 32, no. 3 (2016): 376. http://dx.doi.org/10.4103/0970-9185.188833.

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22

Fournier, Mario, Mahlet Alula, and Gary C. Sieck. "Neuromuscular transmission failure during postnatal development." Neuroscience Letters 125, no. 1 (April 1991): 34–36. http://dx.doi.org/10.1016/0304-3940(91)90124-c.

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23

Tomàs, Josep, Manel M. Santafé, Maria A. Lanuza, Neus García, Nuria Besalduch, and Marta Tomàs. "Silent synapses in neuromuscular junction development." Journal of Neuroscience Research 89, no. 1 (September 20, 2010): 3–12. http://dx.doi.org/10.1002/jnr.22494.

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24

Tintignac, Lionel A., Hans-Rudolf Brenner, and Markus A. Rüegg. "Mechanisms Regulating Neuromuscular Junction Development and Function and Causes of Muscle Wasting." Physiological Reviews 95, no. 3 (July 2015): 809–52. http://dx.doi.org/10.1152/physrev.00033.2014.

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The neuromuscular junction is the chemical synapse between motor neurons and skeletal muscle fibers. It is designed to reliably convert the action potential from the presynaptic motor neuron into the contraction of the postsynaptic muscle fiber. Diseases that affect the neuromuscular junction may cause failure of this conversion and result in loss of ambulation and respiration. The loss of motor input also causes muscle wasting as muscle mass is constantly adapted to contractile needs by the balancing of protein synthesis and protein degradation. Finally, neuromuscular activity and muscle mass have a major impact on metabolic properties of the organisms. This review discusses the mechanisms involved in the development and maintenance of the neuromuscular junction, the consequences of and the mechanisms involved in its dysfunction, and its role in maintaining muscle mass during aging. As life expectancy is increasing, loss of muscle mass during aging, called sarcopenia, has emerged as a field of high medical need. Interestingly, aging is also accompanied by structural changes at the neuromuscular junction, suggesting that the mechanisms involved in neuromuscular junction maintenance might be disturbed during aging. In addition, there is now evidence that behavioral paradigms and signaling pathways that are involved in longevity also affect neuromuscular junction stability and sarcopenia.
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25

Barbeau, Susie, Julie Tahraoui-Bories, Claire Legay, and Cécile Martinat. "Building neuromuscular junctions in vitro." Development 147, no. 22 (November 15, 2020): dev193920. http://dx.doi.org/10.1242/dev.193920.

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ABSTRACTThe neuromuscular junction (NMJ) has been the model of choice to understand the principles of communication at chemical synapses. Following groundbreaking experiments carried out over 60 years ago, many studies have focused on the molecular mechanisms underlying the development and physiology of these synapses. This Review summarizes the progress made to date towards obtaining faithful models of NMJs in vitro. We provide a historical approach discussing initial experiments investigating NMJ development and function from Xenopus to mice, the creation of chimeric co-cultures, in vivo approaches and co-culture methods from ex vivo and in vitro derived cells, as well as the most recent developments to generate human NMJs. We discuss the benefits of these techniques and the challenges to be addressed in the future for promoting our understanding of development and human disease.
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26

Miquel-Vergés, Joan, and Elena Sánchez-Trigo. "The Social Model of Translation and Its Application to Internet Search Engines Specialized in Health: The ASEM Search Engine for Neuromuscular Diseases." Meta 55, no. 2 (August 10, 2010): 374–86. http://dx.doi.org/10.7202/044246ar.

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The use of the Internet as a source of health information is greatly increasing. However, identifying relevant and valid information can be problematic. This paper firstly analyses the efficiency of Internet search engines specialized in health in order to then determine the quality of the online information related to a specific medical subdomain like that of neuromuscular diseases. Our aim is to present a model for the development and use of a bilingual electronic corpus (MYOCOR), related to the said neuromuscular diseases in order to: a) on one hand, provide a quality health information tool for health professionals, patients and relatives, as well as for translators and writers of specialized texts, and software developers, and b) on the other hand, use the same as a base for the implementation of a search engine (using keywords and semantics), like the ASEM (Federación Española Contra las Enfermedades Neuromusculares) search engine for neuromuscular diseases.
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27

Epperson, Hannah E., and Mary J. Sandage. "Neuromuscular Development in Neonates and Postnatal Infants: Implications for Neuromuscular Electrical Stimulation Therapy for Dysphagia." Journal of Speech, Language, and Hearing Research 62, no. 8 (August 15, 2019): 2575–83. http://dx.doi.org/10.1044/2019_jslhr-s-18-0502.

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Purpose The aim of the current study was to review neuromuscular development, summarize the current body of evidence describing the use of neuromuscular electrical stimulation (NMES) therapy in infants, and identify possible contraindications for the use of NMES in the neonate and young infant. Method After a review of the literature describing neuromuscular development, we created a timeline of the developmental processes. Key milestones were determined, and a literature search was conducted to identify potential effects of electrical stimulation on this process. Results Current evidence supporting the use of NMES in the pediatric population is limited and of poor quality. Contraindications of the use of NMES in the neonate and young infant were identified, including (a) inhibited expression of the neural cell adhesion molecule that is vital for neuromuscular development, (b) alteration of muscle fiber type metabolic profile away from intended muscle fiber type morphology, and (c) interruption of postsynaptic acetylcholine receptor synthesis during neuromuscular junction development. Conclusion The use of NMES for the treatment of dysphagia in the neonate and young infant may influence early neuromuscular development in a manner that is not currently well understood. Future research is needed to further understand the effects of NMES on the developing neuromuscular system.
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28

Keshishian, Haig, Te Ning Chang, and Jill Jarecki. "Precision and plasticity during Drosophila neuromuscular development." FASEB Journal 8, no. 10 (July 1994): 731–37. http://dx.doi.org/10.1096/fasebj.8.10.8050672.

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29

Bushby, Kate. "Neuromuscular diseases: milestones in development of treatments." Lancet Neurology 10, no. 1 (January 2011): 11–13. http://dx.doi.org/10.1016/s1474-4422(10)70311-2.

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30

Van Vactor, David, Carlos M. Loya, Cecilia S. Lu, and Tudor A. Fulga. "Function of microRNA during neuromuscular junction development." Developmental Biology 331, no. 2 (July 2009): 388. http://dx.doi.org/10.1016/j.ydbio.2009.05.019.

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31

Marthiens, Véronique, Julie Gavard, Mireille Lambert, and René Marc Mège. "Cadherin-based cell adhesion in neuromuscular development." Biology of the Cell 94, no. 6 (October 2002): 315–26. http://dx.doi.org/10.1016/s0248-4900(02)00005-9.

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32

Vieira, V. L. A., and I. A. Johnston. "Temperature and neuromuscular development in the tambaqui." Journal of Fish Biology 55, sa (December 1999): 66–83. http://dx.doi.org/10.1111/j.1095-8649.1999.tb01046.x.

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33

Menon, Kaushiki P., Robert A. Carrillo, and Kai Zinn. "Development and plasticity of theDrosophilalarval neuromuscular junction." Wiley Interdisciplinary Reviews: Developmental Biology 2, no. 5 (February 5, 2013): 647–70. http://dx.doi.org/10.1002/wdev.108.

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34

Kamimura, Keisuke, and Nobuaki Maeda. "Heparan sulfate proteoglycans in Drosophila neuromuscular development." Biochimica et Biophysica Acta (BBA) - General Subjects 1861, no. 10 (October 2017): 2442–46. http://dx.doi.org/10.1016/j.bbagen.2017.06.015.

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35

Hall, Zach W., and Joshua R. Sanes. "Synaptic structure and development: The neuromuscular junction." Cell 72 (January 1993): 99–121. http://dx.doi.org/10.1016/s0092-8674(05)80031-5.

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36

Wilcox, Susan R. "Corticosteroids and neuromuscular blockers in development of critical illness neuromuscular abnormalities: A historical review." Journal of Critical Care 37 (February 2017): 149–55. http://dx.doi.org/10.1016/j.jcrc.2016.09.018.

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37

Digala, Lakshmi, and Raghav Govindarajan. "ICU related neuromuscular complications." RRNMF Neuromuscular Journal 1, no. 2 (June 9, 2020): 11–19. http://dx.doi.org/10.17161/rrnmf.v1i2.13596.

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The spectrum of neuromuscular diseases encountered in the ICUs today has rapidly evolved over the last decades. Multiple predisposing factors are involved in the development of neuromuscular complications in intensive care patients. Those complications broadly classified into weakness from the preexisting neuromuscular disease exacerbated by critical illness or the complication of the critical illness itself. Patients, when unresponsive, confused, or sedated precludes careful clinical examination. A careful schematic approach that involves acquiring extensive history, any underlying infections, use of any offending medications, and the course of presenting illness will help in delineating the underlying etiology. Here in this review, we describe many causes and the pathophysiology that contribute to the development of neuromuscular weakness in the ICU. A comprehensive investigation protocol must strictly be adhered to all the cases in the ICU settings to reduce the mortality and morbidity.
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38

Schaeffer, E., J. Alder, P. Greengard, and M. M. Poo. "Synapsin IIa accelerates functional development of neuromuscular synapses." Proceedings of the National Academy of Sciences 91, no. 9 (April 26, 1994): 3882–86. http://dx.doi.org/10.1073/pnas.91.9.3882.

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39

Muntoni, Francesco, Sue Brown, Caroline Sewry, and Ketan Patel. "Muscle development genes: their relevance in neuromuscular disorders." Neuromuscular Disorders 12, no. 5 (June 2002): 438–46. http://dx.doi.org/10.1016/s0960-8966(01)00326-1.

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40

Nicoleau, Camille, Dorothée Buttigieg, Sullivan Henriques, Johannes Krupp, Jacquie Maignel, Elsa Raban, Sandra Marlin, Rémy Steinschneider, and Keith Foster. "Development of an in vitro human neuromuscular junction." Toxicon 156 (December 2018): S84. http://dx.doi.org/10.1016/j.toxicon.2018.11.204.

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41

El Mouelhi, Mohamed. "Drug Development and Challenges for Neuromuscular Clinical Trials." Journal of Molecular Neuroscience 58, no. 3 (December 21, 2015): 374–78. http://dx.doi.org/10.1007/s12031-015-0700-9.

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42

Zschüntzsch, Jana, Stefanie Meyer, Mina Shahriyari, Karsten Kummer, Matthias Schmidt, Susann Kummer, and Malte Tiburcy. "The Evolution of Complex Muscle Cell In Vitro Models to Study Pathomechanisms and Drug Development of Neuromuscular Disease." Cells 11, no. 7 (April 5, 2022): 1233. http://dx.doi.org/10.3390/cells11071233.

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Many neuromuscular disease entities possess a significant disease burden and therapeutic options remain limited. Innovative human preclinical models may help to uncover relevant disease mechanisms and enhance the translation of therapeutic findings to strengthen neuromuscular disease precision medicine. By concentrating on idiopathic inflammatory muscle disorders, we summarize the recent evolution of the novel in vitro models to study disease mechanisms and therapeutic strategies. A particular focus is laid on the integration and simulation of multicellular interactions of muscle tissue in disease phenotypes in vitro. Finally, the requirements of a neuromuscular disease drug development workflow are discussed with a particular emphasis on cell sources, co-culture systems (including organoids), functionality, and throughput.
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43

Santoso, Jeffrey W., and Megan L. McCain. "Neuromuscular disease modeling on a chip." Disease Models & Mechanisms 13, no. 7 (July 1, 2020): dmm044867. http://dx.doi.org/10.1242/dmm.044867.

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ABSTRACTOrgans-on-chips are broadly defined as microfabricated surfaces or devices designed to engineer cells into microscale tissues with native-like features and then extract physiologically relevant readouts at scale. Because they are generally compatible with patient-derived cells, these technologies can address many of the human relevance limitations of animal models. As a result, organs-on-chips have emerged as a promising new paradigm for patient-specific disease modeling and drug development. Because neuromuscular diseases span a broad range of rare conditions with diverse etiology and complex pathophysiology, they have been especially challenging to model in animals and thus are well suited for organ-on-chip approaches. In this Review, we first briefly summarize the challenges in neuromuscular disease modeling with animal models. Next, we describe a variety of existing organ-on-chip approaches for neuromuscular tissues, including a survey of cell sources for both muscle and nerve, and two- and three-dimensional neuromuscular tissue-engineering techniques. Although researchers have made tremendous advances in modeling neuromuscular diseases on a chip, the remaining challenges in cell sourcing, cell maturity, tissue assembly and readout capabilities limit their integration into the drug development pipeline today. However, as the field advances, models of healthy and diseased neuromuscular tissues on a chip, coupled with animal models, have vast potential as complementary tools for modeling multiple aspects of neuromuscular diseases and identifying new therapeutic strategies.
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44

Xing, Guanglin, Guangming Gan, Dandan Chen, Mingkuan Sun, Jukang Yi, Huihui Lv, Junhai Han, and Wei Xie. "Drosophila Neuroligin3 Regulates Neuromuscular Junction Development and Synaptic Differentiation." Journal of Biological Chemistry 289, no. 46 (September 16, 2014): 31867–77. http://dx.doi.org/10.1074/jbc.m114.574897.

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45

Bianchi, C. Paul. "Neuromuscular Development and Disease.Alan M. Kelly , Helen M. Blau." Quarterly Review of Biology 68, no. 3 (September 1993): 432. http://dx.doi.org/10.1086/418205.

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46

Bertoni, Carmen. "Therapy development for neuromuscular diseases: translating hope into promise." Future Neurology 8, no. 4 (July 2013): 399–401. http://dx.doi.org/10.2217/fnl.13.29.

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47

JELLIES, JOHN A. "Cellular Interactions in the Development of Annelid Neuromuscular Systems." American Zoologist 35, no. 6 (December 1995): 529–41. http://dx.doi.org/10.1093/icb/35.6.529.

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48

Li, H., X. Peng, and R. L. Cooper. "Development of Drosophila larval neuromuscular junctions: maintaining synaptic strength." Neuroscience 115, no. 2 (December 2002): 505–13. http://dx.doi.org/10.1016/s0306-4522(02)00380-9.

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49

Pirker, Martina E., Udo Rolle, Toko Shinkai, Masato Shinkai, and Prem Puri. "Prenatal and Postnatal Neuromuscular Development of the Ureterovesical Junction." Journal of Urology 177, no. 4 (April 2007): 1546–51. http://dx.doi.org/10.1016/j.juro.2006.11.081.

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50

Sanes, Joshua R., Elizabeth D. Apel, Robert W. Burgess, Ronald B. Emerson, Guoping Feng, Medha Gautam, David Glass, et al. "Development of the neuromuscular junction: Genetic analysis in mice." Journal of Physiology-Paris 92, no. 3-4 (June 1998): 167–72. http://dx.doi.org/10.1016/s0928-4257(98)80004-1.

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